The YIP1 protein of Leishmania major and its role in terbinafine resistance (2007)
- Autores:
- Autor USP: TOSI, LUIZ RICARDO ORSINI - FMRP
- Unidade: FMRP
- Assunto: LEISHMANIA (GENÉTICA)
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Abstracts
- Nome do evento: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology-SBBq
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ABNT
PINTO, M. R. e TOSI, Luiz Ricardo Orsini. The YIP1 protein of Leishmania major and its role in terbinafine resistance. 2007, Anais.. São Paulo: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2007. . Acesso em: 23 abr. 2024. -
APA
Pinto, M. R., & Tosi, L. R. O. (2007). The YIP1 protein of Leishmania major and its role in terbinafine resistance. In Abstracts. São Paulo: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Pinto MR, Tosi LRO. The YIP1 protein of Leishmania major and its role in terbinafine resistance. Abstracts. 2007 ;[citado 2024 abr. 23 ] -
Vancouver
Pinto MR, Tosi LRO. The YIP1 protein of Leishmania major and its role in terbinafine resistance. Abstracts. 2007 ;[citado 2024 abr. 23 ] - The 9-1-1 complex is involved in cell cycle checkpoint and gene amplification modulation in the protozoan Leishmania
- Isolamento e clonagem da RAB/GTPase YPT em Leishmania SPP
- LmYPT overexpression abolishes HTBF-mediated terbinafine resistance in Leishmania major
- Characterization of differences in the expression of epissomal genes in leishmania major and leishmania braziliensis
- The role of chromosome context in site-directed disruption of subtelometric targets of Leishimania major
- YIP1 of Leishmania major: characterization and possible role in terbinafine resistance
- Leishmania major nuclear protein LMHUS1 protects DNA from damage
- A transposon toolkit for gene transfer and mutagenesis in protozoan parasites
- LmHus1 is required in the DNA damage response in Leishmania major and forms a complex with an unusual Rad9 homologue
- DiCre-based conditional knockout of LMHUS1 reveals functional compartmentalization of the 9-1-1 complex in the DNA damage response of Leishmania major
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