Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity (2006)
- Authors:
- Autor USP: KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidade: IQ
- DOI: 10.1016/j.freeradbiomed.2005.08.041
- Subjects: RADICAIS LIVRES; BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Free Radical Biology and Medicine
- ISSN: 0891-5849
- Volume/Número/Paginação/Ano: v. 40, n. 3, p. 469-479, 2006
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
FACUNDO, Heberty Di Tarso Fernandes et al. Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity. Free Radical Biology and Medicine, v. 40, n. 3, p. 469-479, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2005.08.041. Acesso em: 19 abr. 2024. -
APA
Facundo, H. D. T. F., Carreira, R. S., Paula, J. G. de, Santos, C. X. da C. dos, Ferranti, R., Laurindo, F. R. M., & Kowaltowski, A. J. (2006). Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity. Free Radical Biology and Medicine, 40( 3), 469-479. doi:10.1016/j.freeradbiomed.2005.08.041 -
NLM
Facundo HDTF, Carreira RS, Paula JG de, Santos CX da C dos, Ferranti R, Laurindo FRM, Kowaltowski AJ. Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity [Internet]. Free Radical Biology and Medicine. 2006 ; 40( 3): 469-479.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2005.08.041 -
Vancouver
Facundo HDTF, Carreira RS, Paula JG de, Santos CX da C dos, Ferranti R, Laurindo FRM, Kowaltowski AJ. Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity [Internet]. Free Radical Biology and Medicine. 2006 ; 40( 3): 469-479.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2005.08.041 - Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
- Bioenergetic consequences of opening the ATP-sensitive `K POT. +´ channel of heart mitochondria
- Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart
- Effect of Bcl-2 overexpression on mitochondrial structure and function
- Mitochondrial ATP-sensitive 'K POT. +' channel opening decreases reactive oxygen species generation
- Mitochondrial channels that regulate cell viability
- Mitochondrial permeability transition is delayed by ATP-sensitive 'K POT. +' channel ('Mitok IND. ATP') opening
- Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress
- ATP-sensitive K+ channel (mitoKATP) opening protects against cellular damage preventing mitochondrial permeability transition
- Diet, mitochondria and energy metabolism
Informações sobre o DOI: 10.1016/j.freeradbiomed.2005.08.041 (Fonte: oaDOI API)
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