Myeloperoxidase, hydrogen peroxide and tumor necrosis factor 'alfa' production as methods to access platelet aggregating factor - like activity of oxidazed low density lipoprotein (OXLDL) (2004)
- Authors:
- USP affiliated authors: NEGRO, SONIA JANCAR - ICB ; GIDLUND, MAGNUS AKE - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Ouro Preto
- Date published: 2004
- Source:
- Título do periódico: Final Program and Abstracts
- Conference titles: Sociedade Brasileira de Imunologia
-
ABNT
JURADO, M. C. et al. Myeloperoxidase, hydrogen peroxide and tumor necrosis factor 'alfa' production as methods to access platelet aggregating factor - like activity of oxidazed low density lipoprotein (OXLDL). 2004, Anais.. Ouro Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2004. . Acesso em: 20 abr. 2024. -
APA
Jurado, M. C., Shio, M. T., Paulino, E. C., Jancar Negro, S., & Gidlund, M. A. (2004). Myeloperoxidase, hydrogen peroxide and tumor necrosis factor 'alfa' production as methods to access platelet aggregating factor - like activity of oxidazed low density lipoprotein (OXLDL). In Final Program and Abstracts. Ouro Preto: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Jurado MC, Shio MT, Paulino EC, Jancar Negro S, Gidlund MA. Myeloperoxidase, hydrogen peroxide and tumor necrosis factor 'alfa' production as methods to access platelet aggregating factor - like activity of oxidazed low density lipoprotein (OXLDL). Final Program and Abstracts. 2004 ;[citado 2024 abr. 20 ] -
Vancouver
Jurado MC, Shio MT, Paulino EC, Jancar Negro S, Gidlund MA. Myeloperoxidase, hydrogen peroxide and tumor necrosis factor 'alfa' production as methods to access platelet aggregating factor - like activity of oxidazed low density lipoprotein (OXLDL). Final Program and Abstracts. 2004 ;[citado 2024 abr. 20 ] - Oxidized low density lipoprotein inhibits LPS/IFN-induced nitric oxide production in RAW264.7 cells - effects of a PAF-antagonist
- Vascular permeability is increased by oxidized low density lipoprotein (oxLDL), size-inclusion subfractions and slelcted immunogenic apolipoprotein B (apoB) derived synthetic peptides
- OxLDL inibe a produção de óxido nítrico induzida por LPS e IFN-gamma em células RAW264.7 via receptor de PAF
- Oxidized low density lipoprotein (LDL) can increase the expression of scavenger receptor (CD36) and FCgRII by interaction with platelet-activating factor (PAF) receptor
- Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR
- Oxidized low density lipoprotein (oxLDL) and purified oxLDL products inhibits nitric oxide releasem iNOS and COX-2 expression in macrophages
- Oxidized low density lipoprotein inhibits LPS/IFN-induced nitric oxideproduction in RAW264.7 cells - effect of a PAF-antagonist
- Degree of modification of low density lipoprotein (LDL) displays different properties in size and eletric charge and induction of CD11B and CD36 in THP-1 cells
- Vascular permeability is increased by oxidized low density lopoprotein (oxLDL) and selected apolipoprotein B derived synthetic polypeptides
- Vascular permeability is increased by oxidized low density lipoprotein (oxLDL) and selected apolipoprotein B derived synthetic polypeptides
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