The molecular basis of focal cyst formation in human autosomal dominant polycistic kidney disease type I (1996)
- Autores:
- Autor USP: ONUCHIC, LUIZ FERNANDO - FM
- Unidade: FM
- Assunto: NEFROLOGIA
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Cell
- Volume/Número/Paginação/Ano: v. 87, n. 6, p. 979-987, december 1996
-
ABNT
QIAN, Feng et al. The molecular basis of focal cyst formation in human autosomal dominant polycistic kidney disease type I. Cell, v. 87, n. 6, p. 979-987, 1996Tradução . . Acesso em: 25 set. 2024. -
APA
Qian, F., Waitnick, T. J., Onuchic, L. F., & Germino, G. G. (1996). The molecular basis of focal cyst formation in human autosomal dominant polycistic kidney disease type I. Cell, 87( 6), 979-987. -
NLM
Qian F, Waitnick TJ, Onuchic LF, Germino GG. The molecular basis of focal cyst formation in human autosomal dominant polycistic kidney disease type I. Cell. 1996 ; 87( 6): 979-987.[citado 2024 set. 25 ] -
Vancouver
Qian F, Waitnick TJ, Onuchic LF, Germino GG. The molecular basis of focal cyst formation in human autosomal dominant polycistic kidney disease type I. Cell. 1996 ; 87( 6): 979-987.[citado 2024 set. 25 ] - Regulação do volume celular no ramo ascendente fino da alça de Henle de ratos
- Fine mapping of the autosomal recessive polycystic kidney disease locus (PKHD1) and the genes MUT, RDS, CSNK2(beta), and GSTA1 at 6p21.1-p12
- Genomic structure and exclusion of two candidate genes for the PKHD1 locus and three new polymorphic markers within the critical interval
- Direct association of full-lenght PKD1 and PKD2 proteins in mammalian cells
- Aumento regulatório do volume celular em túbulos de malpighi
- Refinement of the autosomal recessive polycystic kidney disease (PKHD1) interval and exclusion of an EF hand-containing gene as a PKHD1 candidate gene
- Expression of PKD1 and PKD2 transcripts and proteins in human embryo and during normal kidney development
- Contribuições dos Modelos Animais ao Entendimento da Patogênese das Doenças Renais Policísticas
- Recombinant and endogenous polyductin (PD1) is secreted from primary cilia
- PKHD1 mutations in autosomal recessive polycystic kidney disease (ARPKD)
Como citar
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
