Cellular prion protein: implications in seizure and epilepsy (2002)
- Autores:
- Autores USP: CARLOTTI JUNIOR, CARLOS GILBERTO - FMRP ; SAKAMOTO, AMERICO CEIKI - FMRP ; BRENTANI, RICARDO RENZO - FM
- Unidades: FMRP; FM
- Assunto: EPILEPSIA
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Cellular and Molecular Neurobiology
- ISSN: 0272-4340
- Volume/Número/Paginação/Ano: v. 22, n. 3, p. 249-257, 2002
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ABNT
WALZ, Roger et al. Cellular prion protein: implications in seizure and epilepsy. Cellular and Molecular Neurobiology, v. 22, n. 3, p. 249-257, 2002Tradução . . Acesso em: 14 maio 2024. -
APA
Walz, R., Castro, R. M. R. P. S., Velasco, T. R., Carlotti Júnior, C. G., Sakamoto, A. C., Brentani, R. R., & Martins, V. R. (2002). Cellular prion protein: implications in seizure and epilepsy. Cellular and Molecular Neurobiology, 22( 3), 249-257. -
NLM
Walz R, Castro RMRPS, Velasco TR, Carlotti Júnior CG, Sakamoto AC, Brentani RR, Martins VR. Cellular prion protein: implications in seizure and epilepsy. Cellular and Molecular Neurobiology. 2002 ; 22( 3): 249-257.[citado 2024 maio 14 ] -
Vancouver
Walz R, Castro RMRPS, Velasco TR, Carlotti Júnior CG, Sakamoto AC, Brentani RR, Martins VR. Cellular prion protein: implications in seizure and epilepsy. Cellular and Molecular Neurobiology. 2002 ; 22( 3): 249-257.[citado 2024 maio 14 ] - On the prognostic value of ictal EEG patterns in temporal lobe epilepsy surgery: a cohort study
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- Cognitive performance of patients with mesial temporal lobe epilepsy is not associated with human prion protein gene variant allele at codons 129 and 171
- High capacity and low cost detection of prion protein gene variant alleles by denaturing HPLC
- Atypical neuropsychological profiles and cognitive outcome in mesial temporal lobe epilepsy
- Suppression of obsessive-compulsive symptoms after epilepsy surgery
- Nasu-Hakola disease (Polycystic Lipomembranous Osteodysplasia with Sclerosing Leukoencephalopathy-PLOSL): Adementia associated with bone cystic lesions. From clinical to genetic and molecular aspects
- Human cellular prion protein gene variant alleles are associated with intractable symptomatic epilepsies
- Cortical malformations are associated with a rare polymorphism of cellular prion protein
- Discordant neuropsychological assessment predicts post-surgical declarative memory consolidation impairment, but not seizure control in mesial temporal lobe epilepsy
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