Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells (1998)
- Authors:
- Autor USP: SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; BIOLOGIA MOLECULAR
- Language: Inglês
- Imprenta:
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
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ABNT
BENGTSON, M. H. e VALENTINI, S R e ARMELIN, Mari Cleide Sogayar. Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells. 1998, Anais.. São Paulo: SBBQ, 1998. . Acesso em: 24 abr. 2024. -
APA
Bengtson, M. H., Valentini, S. R., & Armelin, M. C. S. (1998). Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells. In . São Paulo: SBBQ. -
NLM
Bengtson MH, Valentini SR, Armelin MCS. Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells. 1998 ;[citado 2024 abr. 24 ] -
Vancouver
Bengtson MH, Valentini SR, Armelin MCS. Analysis of the involvement of a C-type retrovirus in the glucocorticoid-induced transformed to normal phenotypic reversion displayed by C6/ST1 cells. 1998 ;[citado 2024 abr. 24 ] - Role of polyoma t antigens in balb-c/3t3 cells growth control deregulation
- Further characterization of balb-3t3 transfectants bearing polyoma t antigen genes
- Controle e subversao da proliferacao
- Glucocorticoid-regulated gene in transformed to normal phenotypic reversion
- T antigens role in polyomavirus transformation: c-myc but not c-fos or c-jun expression is a target for middle t
- Cdna cloning and expression of growth control genes
- Fibroblast growth factor and camp induce the expression of fos and jun proto-oncogenes in pc12 cells
- Molecular bases for the transformed to normal phenotypic reversion induced by glucocorticoid hormoners in rat glioma
- Cloning of differentially expressed genes involved glucocorticoid-induced phenotypic reversion of c6 glioma cells
- Hydrazine derivative - induced proliferation superimposed on citotoxity in cultured moused fibroblasts
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