Glomerular abnormalities in long-term experimental diabetes (1992)
- Autores:
- Autores USP: FUJIHARA, CLARICE KAZUE - FM ; ZATZ, ROBERTO - FM
- Unidade: FM
- Assunto: ENDOCRINOLOGIA
- Idioma: Inglês
- Imprenta:
- Fonte:
- Título do periódico: Diabetes
- Volume/Número/Paginação/Ano: v.41, n.3 , p.286-93, mar. 1992
-
ABNT
FUJIHARA, C K e PADILHA, R M e ZATZ, Roberto. Glomerular abnormalities in long-term experimental diabetes. Diabetes, v. 41, n. 3 , p. 286-93, 1992Tradução . . Acesso em: 24 abr. 2024. -
APA
Fujihara, C. K., Padilha, R. M., & Zatz, R. (1992). Glomerular abnormalities in long-term experimental diabetes. Diabetes, 41( 3 ), 286-93. -
NLM
Fujihara CK, Padilha RM, Zatz R. Glomerular abnormalities in long-term experimental diabetes. Diabetes. 1992 ;41( 3 ): 286-93.[citado 2024 abr. 24 ] -
Vancouver
Fujihara CK, Padilha RM, Zatz R. Glomerular abnormalities in long-term experimental diabetes. Diabetes. 1992 ;41( 3 ): 286-93.[citado 2024 abr. 24 ] - Role of glomerular hypertension: glomerular hypertrophy and lipid deposition in the genesis of glomerular sclerosis (gs) of experimental diabetes (dm)
- Effects of an endothelin et receptor antagonist on experimental fk506 induced acute renal dysfunction
- An extremely high dose of losartan affords superior renoprotection in the remnant model
- Chronic inhibition of nuclear factor-kB attenuates renal injury in the 5/6 renal ablation model
- Absence of focal glomerulosclerosis in aging analbuminemic rats
- Discarding the haystack to examine the needles: the potential role of urinary exosome analysis [Editorial]
- Massive albuminuria glomerulosclerosis (gs) by chronic nitric oxide (no) blackade and sodium overload
- Sodium excess aggravates hypertension and renal parenchymal injury in rats with chronic no inhibition
- Reversão da atividade inflamatória renal no modelo de inibição crônica do óxido nítrico
- Losartan-hydrochlorothiazide association promotes lasting blood pressure normalization and completely arrests long-term renal injury in the 5/6 ablation model
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