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Artigo de periodico
Metabolic reprogramming represents a targetable mechanism to overcome acquired resistance to venetoclax in acute myeloid leukemia (2025)
Queiroz, Gustavo Nery de ; Lima, Keli Cristina de ; Cipelli, Marcella ; Tomaz, Victoria ; Cortez, Luiz Gustavo Ferreira; Silva, Marina de Franca Basto; Guedes, Rafael Lucas Muniz; Campregher, Paulo Vidal ; Rego, Eduardo Magalhães ; Câmara, Niels Olsen Saraiva ; Lotufo, Letícia Veras Costa ; Machado Neto, João Agostinho
Source: Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. Unidades: FM, ICB
Subjects: FARMACOLOGIA, IMUNOLOGIA
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QUEIROZ, Gustavo Nery de et al. Metabolic reprogramming represents a targetable mechanism to overcome acquired resistance to venetoclax in acute myeloid leukemia. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, v. 1872, 2025Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2025.168065. Acesso em: 27 nov. 2025.
APA
Queiroz, G. N. de, Lima, K. C. de, Cipelli, M., Tomaz, V., Cortez, L. G. F., Silva, M. de F. B., et al. (2025). Metabolic reprogramming represents a targetable mechanism to overcome acquired resistance to venetoclax in acute myeloid leukemia. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1872. doi:10.1016/j.bbadis.2025.168065
NLM
Queiroz GN de, Lima KC de, Cipelli M, Tomaz V, Cortez LGF, Silva M de FB, Guedes RLM, Campregher PV, Rego EM, Câmara NOS, Lotufo LVC, Machado Neto JA. Metabolic reprogramming represents a targetable mechanism to overcome acquired resistance to venetoclax in acute myeloid leukemia [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 2025 ; 1872[citado 2025 nov. 27 ] Available from: https://doi.org/10.1016/j.bbadis.2025.168065
Vancouver
Queiroz GN de, Lima KC de, Cipelli M, Tomaz V, Cortez LGF, Silva M de FB, Guedes RLM, Campregher PV, Rego EM, Câmara NOS, Lotufo LVC, Machado Neto JA. Metabolic reprogramming represents a targetable mechanism to overcome acquired resistance to venetoclax in acute myeloid leukemia [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 2025 ; 1872[citado 2025 nov. 27 ] Available from: https://doi.org/10.1016/j.bbadis.2025.168065
Artigo de periodico
Potency and efficacy of pharmacological PIP4K2 inhibitors in acute lymphoblastic leukemia (2024)
Lima, Keli ; Nogueira, Frederico Lisboa ; Cipelli, Marcella ; Carvalho, Maria Fernanda Lopes ; Martins, Diego Antonio Pereira ; Silva, Wellington Fernandes da ; Medeiros, Rita de Cássia Cavaglieri ; Nardinelli, Luciana ; Leal, Aline de Medeiros; Velloso, Elvira Deolinda Rodrigues Pereira ; Bendit, Israel ; Camara, Niels Olsen Saraiva ; Schuringa, Jan Jacob ; Machado Neto, João Agostinho ; Rego, Eduardo Magalhães
Source: European Journal of Pharmacology. Unidades: FM, ICB
Subjects: FARMACOLOGIA, IMUNOLOGIA, ANTINEOPLÁSICOS, SOBREVIVÊNCIA CELULAR, INIBIDORES DE ENZIMAS, PROTEÍNAS QUINASES, PROTEÍNAS PROTO-ONCOGÊNICAS, NEOPLASIAS POR TIPO HISTOLÓGICO, LEUCEMIA
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LIMA, Keli et al. Potency and efficacy of pharmacological PIP4K2 inhibitors in acute lymphoblastic leukemia. European Journal of Pharmacology, v. 977, p. 11 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2024.176723. Acesso em: 27 nov. 2025.
APA
Lima, K., Nogueira, F. L., Cipelli, M., Carvalho, M. F. L., Martins, D. A. P., Silva, W. F. da, et al. (2024). Potency and efficacy of pharmacological PIP4K2 inhibitors in acute lymphoblastic leukemia. European Journal of Pharmacology, 977, 11 . doi:10.1016/j.ejphar.2024.176723
NLM
Lima K, Nogueira FL, Cipelli M, Carvalho MFL, Martins DAP, Silva WF da, Medeiros R de CC, Nardinelli L, Leal A de M, Velloso EDRP, Bendit I, Camara NOS, Schuringa JJ, Machado Neto JA, Rego EM. Potency and efficacy of pharmacological PIP4K2 inhibitors in acute lymphoblastic leukemia [Internet]. European Journal of Pharmacology. 2024 ; 977 11 .[citado 2025 nov. 27 ] Available from: https://doi.org/10.1016/j.ejphar.2024.176723
Vancouver
Lima K, Nogueira FL, Cipelli M, Carvalho MFL, Martins DAP, Silva WF da, Medeiros R de CC, Nardinelli L, Leal A de M, Velloso EDRP, Bendit I, Camara NOS, Schuringa JJ, Machado Neto JA, Rego EM. Potency and efficacy of pharmacological PIP4K2 inhibitors in acute lymphoblastic leukemia [Internet]. European Journal of Pharmacology. 2024 ; 977 11 .[citado 2025 nov. 27 ] Available from: https://doi.org/10.1016/j.ejphar.2024.176723
Artigo de periodico
Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae (2024)
Padovani, Barbara Nunes ; Fénero, Camila Ideli Morales ; Paredes, Lais Cavalieri ; Amaral, Mariana Abrantes do ; Amorocho, Omar Alberto Domínguez ; Cipelli, Marcella ; Gomes, Juliana Moreira Mendonça ; Silva, Eloisa Martins da ; Silva, Luisa Menezes ; Vieira, Raquel de Souza ; Pereira, Mariana Tominaga ; Cruz, Mario Costa ; Camara, Niels Olsen Saraiva
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: IMUNOLOGIA, ANTINEOPLÁSICOS, INSUFICIÊNCIA RENAL AGUDA, INFLAMAÇÃO, CITOCINAS, INTERLEUCINAS, TOXICIDADE EM ANIMAL, REAÇÃO EM CADEIA POR POLIMERASE, CITOMETRIA DE FLUXO, PEIXES DE ÁGUA DOCE, PEIXES TROPICAIS
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PADOVANI, Barbara Nunes et al. Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae. International Journal of Molecular Sciences, v. 25, n. 4, p. 16 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25042363. Acesso em: 27 nov. 2025.
APA
Padovani, B. N., Fénero, C. I. M., Paredes, L. C., Amaral, M. A. do, Amorocho, O. A. D., Cipelli, M., et al. (2024). Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae. International Journal of Molecular Sciences, 25( 4), 16 . doi:10.3390/ijms25042363
NLM
Padovani BN, Fénero CIM, Paredes LC, Amaral MA do, Amorocho OAD, Cipelli M, Gomes JMM, Silva EM da, Silva LM, Vieira R de S, Pereira MT, Cruz MC, Camara NOS. Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 4): 16 .[citado 2025 nov. 27 ] Available from: https://doi.org/10.3390/ijms25042363
Vancouver
Padovani BN, Fénero CIM, Paredes LC, Amaral MA do, Amorocho OAD, Cipelli M, Gomes JMM, Silva EM da, Silva LM, Vieira R de S, Pereira MT, Cruz MC, Camara NOS. Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 4): 16 .[citado 2025 nov. 27 ] Available from: https://doi.org/10.3390/ijms25042363
Artigo de periodico
Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis (2024)
Ignacio, Aline ; Cipelli, Marcella ; Takiishi, Tatiana ; Aguiar, Cristhiane Favero ; Terra, Fernanda Fernandes ; Ghirotto, Bruno ; Silva, Eloisa Martins da ; Castoldi, Angela ; Magalhães, Yuli Thamires ; Antonio, Tiago; Padovani, Barbara Nunes ; Hiyane, Meire Ioshie ; Andrade-Oliveira, Vinicius ; Forti, Fabio Luis ; Camara, Niels Olsen Saraiva
Source: Journal of Leukocyte Biology. Unidades: IQ, ICB
Subjects: IMUNOLOGIA, FÁRMACOS IMUNOSSUPRESSORES, BIOTA INTESTINAL, CÉLULAS DENDRÍTICAS, COLITE ULCERATIVA, CÉLULAS MIELOIDES, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, CITOESQUELETO, CITOCINAS, INTERLEUCINA 6
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IGNACIO, Aline et al. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis. Journal of Leukocyte Biology, v. 116, n. 4, p. 779-792, 2024Tradução . . Disponível em: https://doi.org/10.1093/jleuko/qiae084. Acesso em: 27 nov. 2025.
APA
Ignacio, A., Cipelli, M., Takiishi, T., Aguiar, C. F., Terra, F. F., Ghirotto, B., et al. (2024). Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis. Journal of Leukocyte Biology, 116( 4), 779-792. doi:10.1093/jleuko/qiae084
NLM
Ignacio A, Cipelli M, Takiishi T, Aguiar CF, Terra FF, Ghirotto B, Silva EM da, Castoldi A, Magalhães YT, Antonio T, Padovani BN, Hiyane MI, Andrade-Oliveira V, Forti FL, Camara NOS. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis [Internet]. Journal of Leukocyte Biology. 2024 ; 116( 4): 779-792.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1093/jleuko/qiae084
Vancouver
Ignacio A, Cipelli M, Takiishi T, Aguiar CF, Terra FF, Ghirotto B, Silva EM da, Castoldi A, Magalhães YT, Antonio T, Padovani BN, Hiyane MI, Andrade-Oliveira V, Forti FL, Camara NOS. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis [Internet]. Journal of Leukocyte Biology. 2024 ; 116( 4): 779-792.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1093/jleuko/qiae084
Artigo de periodico
The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism (2023)
Foresto Neto, Orestes ; Silva, Ana Ruth Paolinetti Alves da ; Cipelli, Marcella ; Santana-Novelli, Fernanda Paula Roncon Santana ; Camara, Niels Olsen Saraiva
Source: Kidney Research and Clinical Practice. Unidade: ICB
Subjects: IMUNOLOGIA, SISTEMA IMUNE, METABOLISMO CELULAR, NEFROPATIAS, BIOTA INTESTINAL, ANÓXIA
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FORESTO NETO, Orestes et al. The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism. Kidney Research and Clinical Practice, v. 42, n. 5, p. 561-578, 2023Tradução . . Disponível em: https://doi.org/10.23876/j.krcp.23.012. Acesso em: 27 nov. 2025.
APA
Foresto Neto, O., Silva, A. R. P. A. da, Cipelli, M., Santana-Novelli, F. P. R. S., & Camara, N. O. S. (2023). The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism. Kidney Research and Clinical Practice, 42( 5), 561-578. doi:10.23876/j.krcp.23.012. Epub 2023 Jun 15.
NLM
Foresto Neto O, Silva ARPA da, Cipelli M, Santana-Novelli FPRS, Camara NOS. The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism [Internet]. Kidney Research and Clinical Practice. 2023 ; 42( 5): 561-578.[citado 2025 nov. 27 ] Available from: https://doi.org/10.23876/j.krcp.23.012
Vancouver
Foresto Neto O, Silva ARPA da, Cipelli M, Santana-Novelli FPRS, Camara NOS. The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism [Internet]. Kidney Research and Clinical Practice. 2023 ; 42( 5): 561-578.[citado 2025 nov. 27 ] Available from: https://doi.org/10.23876/j.krcp.23.012
Artigo de periodico
Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis (2023)
Silva, Aline Ignacio Silvestre da; Cipelli, Marcella ; Takiishi, Tatiana ; Aguiar, Cristhiane Favero ; Terra, Fernanda Fernandes ; Nunes, Bruno Ghirotto ; Silva, Eloisa Martins da ; Castoldi, Angela ; Magalhães, Yuli Thamires ; Souza, Tiago Antonio de ; Hiyane, Meire Ioshie ; Oliveira, Vinicius Andrade de ; Forti, Fabio Luis ; Camara, Niels Olsen Saraiva
Source: bioRxiv. Unidades: ICB, IQ
Subjects: COLITE, CÉLULAS MIELOIDES
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SILVA, Aline Ignacio Silvestre da et al. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis. bioRxiv, 2023Tradução . . Disponível em: https://doi.org/10.1101/2023.04.26.537895. Acesso em: 27 nov. 2025.
APA
Silva, A. I. S. da, Cipelli, M., Takiishi, T., Aguiar, C. F., Terra, F. F., Nunes, B. G., et al. (2023). Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis. bioRxiv. doi:10.1101/2023.04.26.537895
NLM
Silva AIS da, Cipelli M, Takiishi T, Aguiar CF, Terra FF, Nunes BG, Silva EM da, Castoldi A, Magalhães YT, Souza TA de, Hiyane MI, Oliveira VA de, Forti FL, Camara NOS. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis [Internet]. bioRxiv. 2023 ;[citado 2025 nov. 27 ] Available from: https://doi.org/10.1101/2023.04.26.537895
Vancouver
Silva AIS da, Cipelli M, Takiishi T, Aguiar CF, Terra FF, Nunes BG, Silva EM da, Castoldi A, Magalhães YT, Souza TA de, Hiyane MI, Oliveira VA de, Forti FL, Camara NOS. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis [Internet]. bioRxiv. 2023 ;[citado 2025 nov. 27 ] Available from: https://doi.org/10.1101/2023.04.26.537895
Artigo de periodico
Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease (2023)
Cipelli, Marcella ; Silva, Eloisa Martins da ; Câmara, Niels Olsen Saraiva
Source: Current Clinical Microbiology Reports. Unidade: ICB
Subjects: IMUNOLOGIA, ENTEROPATIAS INFLAMATÓRIAS, BIOTA INTESTINAL, SISTEMA GASTROINTESTINAL, INTESTINOS
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CIPELLI, Marcella e SILVA, Eloisa Martins da e CÂMARA, Niels Olsen Saraiva. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease. Current Clinical Microbiology Reports, p. 11 , 2023Tradução . . Disponível em: https://doi.org/10.1007/s40588-023-00207-4. Acesso em: 27 nov. 2025.
APA
Cipelli, M., Silva, E. M. da, & Câmara, N. O. S. (2023). Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease. Current Clinical Microbiology Reports, 11 . doi:10.1007/s40588-023-00207-4
NLM
Cipelli M, Silva EM da, Câmara NOS. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease [Internet]. Current Clinical Microbiology Reports. 2023 ;11 .[citado 2025 nov. 27 ] Available from: https://doi.org/10.1007/s40588-023-00207-4
Vancouver
Cipelli M, Silva EM da, Câmara NOS. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease [Internet]. Current Clinical Microbiology Reports. 2023 ;11 .[citado 2025 nov. 27 ] Available from: https://doi.org/10.1007/s40588-023-00207-4
Artigo de periodico
MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes (2022)
Nunes, Bruno Ghirotto ; Oliveira, Danyllo Felipe de ; Cipelli, Marcella ; Basso, Paulo José ; Lima, Jean De ; Breda, Cristiane Naffah de Souza ; Ribeiro, Henrique Caldeira da ; Silva, Camille Cristine Caldeira da ; Sertié, Andréa L ; Oliveira, Antonio Edson Rocha ; Hiyane, Meire Ioshie ; Galdini, Elia G. ; Sussulini, Alessandra ; Oliveira, Enedina Maria Lobato de ; Zatz, Mayana ; Câmara, Niels Olsen Saraiva ; Nakaya, Helder Takashi Imoto ; Kowaltowski, Alicia Juliana
Source: Annals of Neurology. Unidades: ICB, IB, FCF, IQ
Subjects: IMUNOLOGIA, ASTRÓCITOS, CÉLULAS-TRONCO, ÁCIDO GLUTÂMICO, MITOCÔNDRIAS, ESCLEROSE MÚLTIPLA, MICROSCOPIA ELETRÔNICA, CITOMETRIA DE FLUXO, CITOCINAS, EXPRESSÃO GÊNICA
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NUNES, Bruno Ghirotto et al. MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes. Annals of Neurology, v. 91, n. 5, p. 652-659, 2022Tradução . . Disponível em: https://doi.org/10.1002/ana.26336. Acesso em: 27 nov. 2025.
APA
Nunes, B. G., Oliveira, D. F. de, Cipelli, M., Basso, P. J., Lima, J. D., Breda, C. N. de S., et al. (2022). MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes. Annals of Neurology, 91( 5), 652-659. doi:10.1002/ana.26336
NLM
Nunes BG, Oliveira DF de, Cipelli M, Basso PJ, Lima JD, Breda CN de S, Ribeiro HC da, Silva CCC da, Sertié AL, Oliveira AER, Hiyane MI, Galdini EG, Sussulini A, Oliveira EML de, Zatz M, Câmara NOS, Nakaya HTI, Kowaltowski AJ. MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes [Internet]. Annals of Neurology. 2022 ; 91( 5): 652-659.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1002/ana.26336
Vancouver
Nunes BG, Oliveira DF de, Cipelli M, Basso PJ, Lima JD, Breda CN de S, Ribeiro HC da, Silva CCC da, Sertié AL, Oliveira AER, Hiyane MI, Galdini EG, Sussulini A, Oliveira EML de, Zatz M, Câmara NOS, Nakaya HTI, Kowaltowski AJ. MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes [Internet]. Annals of Neurology. 2022 ; 91( 5): 652-659.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1002/ana.26336
Artigo de periodico
Local and systemic STAT3 and p65 NF-KappaB expression as progression markers and functional targets for patients with cervical cancer (2020)
Rossetti, Renata Ariza Marques ; Silva Junior, Ildefonso Alves da ; Rodríguez, Gretel Rodríguez ; Alvarez, Karla Lucía Fernadez ; Stone, Simone Cardozo ; Cipelli, Marcella ; Silveira, Caio Raony Farina ; Beldi, Mariana Carmezim ; Mota, Giana Rabello ; Margarido, Paulo Francisco Ramos ; Baracat, Edmund Chada ; Uno, Miyuki; Villa, Luisa Lina ; Carvalho, Jesus de Paula ; Yokochi, Kaori; Rosa, Maria Beatriz S. F.; Lorenzi, Noely Paula Cristina ; Lepique, Ana Paula
Source: Frontiers in Oncology. Unidades: FM, ICB
Subjects: IMUNOLOGIA, NEOPLASIAS DO COLO UTERINO, INFECÇÕES POR PAPILLOMAVIRUS, HPV, VÍRUS DE DNA, ADJUVANTES IMUNOLÓGICOS, TRANSDUÇÃO DE SINAL CELULAR, CÉLULAS CULTIVADAS DE TUMOR, PROLIFERAÇÃO CELULAR
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ROSSETTI, Renata Ariza Marques et al. Local and systemic STAT3 and p65 NF-KappaB expression as progression markers and functional targets for patients with cervical cancer. Frontiers in Oncology, v. 10, p. 15 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fonc.2020.587132. Acesso em: 27 nov. 2025.
APA
Rossetti, R. A. M., Silva Junior, I. A. da, Rodríguez, G. R., Alvarez, K. L. F., Stone, S. C., Cipelli, M., et al. (2020). Local and systemic STAT3 and p65 NF-KappaB expression as progression markers and functional targets for patients with cervical cancer. Frontiers in Oncology, 10, 15 . doi:10.3389/fonc.2020.587132
NLM
Rossetti RAM, Silva Junior IA da, Rodríguez GR, Alvarez KLF, Stone SC, Cipelli M, Silveira CRF, Beldi MC, Mota GR, Margarido PFR, Baracat EC, Uno M, Villa LL, Carvalho J de P, Yokochi K, Rosa MBSF, Lorenzi NPC, Lepique AP. Local and systemic STAT3 and p65 NF-KappaB expression as progression markers and functional targets for patients with cervical cancer [Internet]. Frontiers in Oncology. 2020 ; 10 15 .[citado 2025 nov. 27 ] Available from: https://doi.org/10.3389/fonc.2020.587132
Vancouver
Rossetti RAM, Silva Junior IA da, Rodríguez GR, Alvarez KLF, Stone SC, Cipelli M, Silveira CRF, Beldi MC, Mota GR, Margarido PFR, Baracat EC, Uno M, Villa LL, Carvalho J de P, Yokochi K, Rosa MBSF, Lorenzi NPC, Lepique AP. Local and systemic STAT3 and p65 NF-KappaB expression as progression markers and functional targets for patients with cervical cancer [Internet]. Frontiers in Oncology. 2020 ; 10 15 .[citado 2025 nov. 27 ] Available from: https://doi.org/10.3389/fonc.2020.587132
Artigo de periodico
Host dysbiosis negatively impacts IL-9-producing T-cell differentiation and antitumour immunity (2020)
Almeida, Rafael Ribeiro ; Vieira, Raquel de Souza; Castoldi, Angela ; Terra, Fernanda Fernandes; Melo, Amanda Campelo Lima de; Canesso, Maria Cecília Campos ; Lemos, Luísa; Cipelli, Marcella ; Rana, Nisha; Hiyane, Meire Ioshie ; Pearce, Erika L. ; Martin, Flaviano dos Santos; Faria, Ana Maria Caetano de ; Câmara, Niels Olsen Saraiva
Source: BJC. Unidades: ICB, FM
Subjects: IMUNOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, LINFÓCITOS T, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, ANTIBIÓTICOS, MELANOMA ANIMAL
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ABNT
ALMEIDA, Rafael Ribeiro et al. Host dysbiosis negatively impacts IL-9-producing T-cell differentiation and antitumour immunity. BJC, v. 123, p. 534–541, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41416-020-0915-6. Acesso em: 27 nov. 2025.
APA
Almeida, R. R., Vieira, R. de S., Castoldi, A., Terra, F. F., Melo, A. C. L. de, Canesso, M. C. C., et al. (2020). Host dysbiosis negatively impacts IL-9-producing T-cell differentiation and antitumour immunity. BJC, 123, 534–541. doi:10.1038/s41416-020-0915-6
NLM
Almeida RR, Vieira R de S, Castoldi A, Terra FF, Melo ACL de, Canesso MCC, Lemos L, Cipelli M, Rana N, Hiyane MI, Pearce EL, Martin F dos S, Faria AMC de, Câmara NOS. Host dysbiosis negatively impacts IL-9-producing T-cell differentiation and antitumour immunity [Internet]. BJC. 2020 ; 123 534–541.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1038/s41416-020-0915-6
Vancouver
Almeida RR, Vieira R de S, Castoldi A, Terra FF, Melo ACL de, Canesso MCC, Lemos L, Cipelli M, Rana N, Hiyane MI, Pearce EL, Martin F dos S, Faria AMC de, Câmara NOS. Host dysbiosis negatively impacts IL-9-producing T-cell differentiation and antitumour immunity [Internet]. BJC. 2020 ; 123 534–541.[citado 2025 nov. 27 ] Available from: https://doi.org/10.1038/s41416-020-0915-6
Artigo de periodico
Swainsonine, an alpha-mannosidase inhibitor, may worsen cervical cancer progression through the increase in myeloid derived suppressor cells population (2019)
Silveira, Caio Raony Farina ; Cipelli, Marcella ; Manzine, Carolina; Santos, Silvia Helena Rabelo dos ; Zeferino, Luiz Carlos ; Rodríguez, Gretel Rodríguez ; Assis, Josiane Betim de ; Herbster, Suellen da Silva Gomes ; Berardinelli, Isabel Cristina; Laginha, Fabio Martins ; Boccardo, Enrique ; Villa, Luisa Lina ; Termini, Lara ; Lepique, Ana Paula
Source: Plos one. Unidades: ICB, FM
Subjects: NEOPLASIAS DO COLO UTERINO, ENZIMAS HIDROLÍTICAS, ALCALOIDES, CÉLULAS MIELOIDES
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ABNT
SILVEIRA, Caio Raony Farina et al. Swainsonine, an alpha-mannosidase inhibitor, may worsen cervical cancer progression through the increase in myeloid derived suppressor cells population. Plos one, v. 14, n. 3, 2019Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0213184. Acesso em: 27 nov. 2025.
APA
Silveira, C. R. F., Cipelli, M., Manzine, C., Santos, S. H. R. dos, Zeferino, L. C., Rodríguez, G. R., et al. (2019). Swainsonine, an alpha-mannosidase inhibitor, may worsen cervical cancer progression through the increase in myeloid derived suppressor cells population. Plos one, 14( 3). doi:10.1371/journal.pone.0213184
NLM
Silveira CRF, Cipelli M, Manzine C, Santos SHR dos, Zeferino LC, Rodríguez GR, Assis JB de, Herbster S da SG, Berardinelli IC, Laginha FM, Boccardo E, Villa LL, Termini L, Lepique AP. Swainsonine, an alpha-mannosidase inhibitor, may worsen cervical cancer progression through the increase in myeloid derived suppressor cells population [Internet]. Plos one. 2019 ; 14( 3):[citado 2025 nov. 27 ] Available from: https://doi.org/10.1371/journal.pone.0213184
Vancouver
Silveira CRF, Cipelli M, Manzine C, Santos SHR dos, Zeferino LC, Rodríguez GR, Assis JB de, Herbster S da SG, Berardinelli IC, Laginha FM, Boccardo E, Villa LL, Termini L, Lepique AP. Swainsonine, an alpha-mannosidase inhibitor, may worsen cervical cancer progression through the increase in myeloid derived suppressor cells population [Internet]. Plos one. 2019 ; 14( 3):[citado 2025 nov. 27 ] Available from: https://doi.org/10.1371/journal.pone.0213184

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