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Artigo de periodico
The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells (2018)
Silveira, Elaine; Cavalcante, Isadora Pontes; Kremer, Jean Lucas; Mendonça, Pedro Omori Ribeiro de; Lotfi, Claudimara Ferini Pacicco
Fonte: Cancer Cell International. Unidade: ICB
Assuntos: CARCINOMA, QUIMIOTERÁPICOS, NEOPLASIAS DO CÓRTEX SUPRARRENAL, CÓRTEX ADRENAL, CITOTOXINAS, FÁRMACOS
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ABNT
SILVEIRA, Elaine et al. The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells. Cancer Cell International, v. 18, p. 16 , 2018Tradução . . Disponível em: https://doi.org/10.1186/s12935-018-0527-x. Acesso em: 10 nov. 2024.
APA
Silveira, E., Cavalcante, I. P., Kremer, J. L., Mendonça, P. O. R. de, & Lotfi, C. F. P. (2018). The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells. Cancer Cell International, 18, 16 . doi:10.1186/s12935-018-0527-x
NLM
Silveira E, Cavalcante IP, Kremer JL, Mendonça POR de, Lotfi CFP. The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells [Internet]. Cancer Cell International. 2018 ;18 16 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/s12935-018-0527-x
Vancouver
Silveira E, Cavalcante IP, Kremer JL, Mendonça POR de, Lotfi CFP. The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells [Internet]. Cancer Cell International. 2018 ;18 16 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/s12935-018-0527-x
Artigo de periodico
Independent of ErbB1 gene copy number, EGF stimulates migration but is not associated with cell proliferation in non-small cell lung cancer (2013)
Lauand, Camila; Rezende-Teixeira, Paula; Cortez, Beatriz Araújo; Niero, Evandro Luís de Oliveira; Machado-Santelli, Gláucia Maria
Fonte: Cancer Cell International. Unidade: ICB
Assuntos: NEOPLASIAS PULMONARES, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LAUAND, Camila et al. Independent of ErbB1 gene copy number, EGF stimulates migration but is not associated with cell proliferation in non-small cell lung cancer. Cancer Cell International, v. 13, n. 1, p. 1-15, 2013Tradução . . Disponível em: https://doi.org/10.1186/1475-2867-13-38. Acesso em: 10 nov. 2024.
APA
Lauand, C., Rezende-Teixeira, P., Cortez, B. A., Niero, E. L. de O., & Machado-Santelli, G. M. (2013). Independent of ErbB1 gene copy number, EGF stimulates migration but is not associated with cell proliferation in non-small cell lung cancer. Cancer Cell International, 13( 1), 1-15. doi:10.1186/1475-2867-13-38
NLM
Lauand C, Rezende-Teixeira P, Cortez BA, Niero EL de O, Machado-Santelli GM. Independent of ErbB1 gene copy number, EGF stimulates migration but is not associated with cell proliferation in non-small cell lung cancer [Internet]. Cancer Cell International. 2013 ; 13( 1): 1-15.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/1475-2867-13-38
Vancouver
Lauand C, Rezende-Teixeira P, Cortez BA, Niero EL de O, Machado-Santelli GM. Independent of ErbB1 gene copy number, EGF stimulates migration but is not associated with cell proliferation in non-small cell lung cancer [Internet]. Cancer Cell International. 2013 ; 13( 1): 1-15.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/1475-2867-13-38
Artigo de periodico
Effects of myenteric denervation on extracellular matrix fibers and mast cell distribution in normal stomach and gastric lesions (2010)
Estofolete, Cássia F.; Machado, Carla Botelho; Taboga, Sebastião R.; Polli-Lopes, Ana Cláudia; Gil, Cristiane D.
Fonte: Cancer Cell International. Unidade: FMRP
Assuntos: ESTÔMAGO, MASTÓCITOS, DENERVAÇÃO
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ABNT
ESTOFOLETE, Cássia F. et al. Effects of myenteric denervation on extracellular matrix fibers and mast cell distribution in normal stomach and gastric lesions. Cancer Cell International, v. 10, 2010Tradução . . Disponível em: https://doi.org/10.1186/1475-2867-10-18. Acesso em: 10 nov. 2024.
APA
Estofolete, C. F., Machado, C. B., Taboga, S. R., Polli-Lopes, A. C., & Gil, C. D. (2010). Effects of myenteric denervation on extracellular matrix fibers and mast cell distribution in normal stomach and gastric lesions. Cancer Cell International, 10. doi:10.1186/1475-2867-10-18
NLM
Estofolete CF, Machado CB, Taboga SR, Polli-Lopes AC, Gil CD. Effects of myenteric denervation on extracellular matrix fibers and mast cell distribution in normal stomach and gastric lesions [Internet]. Cancer Cell International. 2010 ; 10[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/1475-2867-10-18
Vancouver
Estofolete CF, Machado CB, Taboga SR, Polli-Lopes AC, Gil CD. Effects of myenteric denervation on extracellular matrix fibers and mast cell distribution in normal stomach and gastric lesions [Internet]. Cancer Cell International. 2010 ; 10[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/1475-2867-10-18
Artigo de periodico
Common molecular pathways involved in human CD133+/CD34+ progenitor cell expansion and cancer (2007)
Okamoto, Oswaldo Keith ; Carvalho, Ana Carolina S. R.; Marti, Luciana C; Vêncio, Ricardo Z.; Moreira-Filho, Carlos Alberto
Fonte: Cancer Cell International. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OKAMOTO, Oswaldo Keith et al. Common molecular pathways involved in human CD133+/CD34+ progenitor cell expansion and cancer. Cancer Cell International, v. 8, p. 1-12, 2007Tradução . . Disponível em: https://doi.org/10.1186/1475-2867-7-11. Acesso em: 10 nov. 2024.
APA
Okamoto, O. K., Carvalho, A. C. S. R., Marti, L. C., Vêncio, R. Z., & Moreira-Filho, C. A. (2007). Common molecular pathways involved in human CD133+/CD34+ progenitor cell expansion and cancer. Cancer Cell International, 8, 1-12. doi:10.1186/1475-2867-7-11
NLM
Okamoto OK, Carvalho ACSR, Marti LC, Vêncio RZ, Moreira-Filho CA. Common molecular pathways involved in human CD133+/CD34+ progenitor cell expansion and cancer [Internet]. Cancer Cell International. 2007 ; 8 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/1475-2867-7-11
Vancouver
Okamoto OK, Carvalho ACSR, Marti LC, Vêncio RZ, Moreira-Filho CA. Common molecular pathways involved in human CD133+/CD34+ progenitor cell expansion and cancer [Internet]. Cancer Cell International. 2007 ; 8 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/1475-2867-7-11
Artigo de periodico
Retroviral transfer of the p161 NK4a cDNA inhibits C6 glioma formation in wistar rats (2002)
Strauss, Bryan E.; Fontes, Ricardo B V.; Lotfi, Claudimara Ferini Pacicco ; Skorupa, Ana Lucia; Bartol, Ione; Cipolla-Neto, José; Costanzi-Strauss, Eugenia
Fonte: Cancer Cell International. Unidade: ICB
Assuntos: FISIOLOGIA, ANATOMIA, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
STRAUSS, Bryan E. et al. Retroviral transfer of the p161 NK4a cDNA inhibits C6 glioma formation in wistar rats. Cancer Cell International, v. 2, p. 1-9, 2002Tradução . . Acesso em: 10 nov. 2024.
APA
Strauss, B. E., Fontes, R. B. V., Lotfi, C. F. P., Skorupa, A. L., Bartol, I., Cipolla-Neto, J., & Costanzi-Strauss, E. (2002). Retroviral transfer of the p161 NK4a cDNA inhibits C6 glioma formation in wistar rats. Cancer Cell International, 2, 1-9.
NLM
Strauss BE, Fontes RBV, Lotfi CFP, Skorupa AL, Bartol I, Cipolla-Neto J, Costanzi-Strauss E. Retroviral transfer of the p161 NK4a cDNA inhibits C6 glioma formation in wistar rats. Cancer Cell International. 2002 ; 2 1-9.[citado 2024 nov. 10 ]
Vancouver
Strauss BE, Fontes RBV, Lotfi CFP, Skorupa AL, Bartol I, Cipolla-Neto J, Costanzi-Strauss E. Retroviral transfer of the p161 NK4a cDNA inhibits C6 glioma formation in wistar rats. Cancer Cell International. 2002 ; 2 1-9.[citado 2024 nov. 10 ]

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