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Artigo de periodico
Vasoactive intestinal peptide promotes hypophagia and metabolic changes: role of paraventricular hypothalamic nucleus and nitric oxide (2022)
Zaia, Cassia Thaïs Bussamra Vieira; Uchôa, Ernane Torres; Santos, Angelo Alexander Torres dos; Ribeiro, Rachel Cezar de Andrade; Batista, Ana Carolina Seidel; Crespigio, Jefferson; Utida, Lawrence; Moura, Galiano Brazuna; Brownlow, Milene Lara; Garnica-Siqueira, Marcela Cristina; Reis, Wagner Luis; Antunes-Rodrigues, José ; Zaia, Dimas Augusto Morozin
Source: Brain Research Bulletin. Unidade: FMRP
Subjects: ARGININA, GLICEMIA, ÁCIDOS GRAXOS, INSULINA, ÓXIDO NÍTRICO, PEPTÍDEO INTESTINAL VASOATIVO
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ABNT
ZAIA, Cassia Thaïs Bussamra Vieira et al. Vasoactive intestinal peptide promotes hypophagia and metabolic changes: role of paraventricular hypothalamic nucleus and nitric oxide. Brain Research Bulletin, v. 189, p. 102-110, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.brainresbull.2022.08.021. Acesso em: 09 nov. 2024.
APA
Zaia, C. T. B. V., Uchôa, E. T., Santos, A. A. T. dos, Ribeiro, R. C. de A., Batista, A. C. S., Crespigio, J., et al. (2022). Vasoactive intestinal peptide promotes hypophagia and metabolic changes: role of paraventricular hypothalamic nucleus and nitric oxide. Brain Research Bulletin, 189, 102-110. doi:10.1016/j.brainresbull.2022.08.021
NLM
Zaia CTBV, Uchôa ET, Santos AAT dos, Ribeiro RC de A, Batista ACS, Crespigio J, Utida L, Moura GB, Brownlow ML, Garnica-Siqueira MC, Reis WL, Antunes-Rodrigues J, Zaia DAM. Vasoactive intestinal peptide promotes hypophagia and metabolic changes: role of paraventricular hypothalamic nucleus and nitric oxide [Internet]. Brain Research Bulletin. 2022 ; 189 102-110.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.brainresbull.2022.08.021
Vancouver
Zaia CTBV, Uchôa ET, Santos AAT dos, Ribeiro RC de A, Batista ACS, Crespigio J, Utida L, Moura GB, Brownlow ML, Garnica-Siqueira MC, Reis WL, Antunes-Rodrigues J, Zaia DAM. Vasoactive intestinal peptide promotes hypophagia and metabolic changes: role of paraventricular hypothalamic nucleus and nitric oxide [Internet]. Brain Research Bulletin. 2022 ; 189 102-110.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.brainresbull.2022.08.021
Artigo de periodico
GLP1R attenuates sympathetic response to high glucose via carotid body inhibition (2022)
Pauza, Audrys G.; Thakkar, Pratik; Tasic, Tatjana; Felippe, Igor; Bishop, Paul; Greenwood, Michael P.; Rysevaite-Kyguoliene, Kristina; Ast, Julia; Broichhagen, Johannes; Hodson, David J.; Salgado, Hélio César ; Pauza, Dainius H.; Japundzic-Zigon, Nina; Paton, Julian F. R.; Murphy, David
Source: Circulation Research. Unidade: FMRP
Subjects: HIPERTENSÃO, INSULINA, OBESIDADE, FATORES DE RISCO, GLICOSE
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ABNT
PAUZA, Audrys G. et al. GLP1R attenuates sympathetic response to high glucose via carotid body inhibition. Circulation Research, v. 130, n. 5, p. 694-707, 2022Tradução . . Disponível em: https://doi.org/10.1161/CIRCRESAHA.121.319874. Acesso em: 09 nov. 2024.
APA
Pauza, A. G., Thakkar, P., Tasic, T., Felippe, I., Bishop, P., Greenwood, M. P., et al. (2022). GLP1R attenuates sympathetic response to high glucose via carotid body inhibition. Circulation Research, 130( 5), 694-707. doi:10.1161/CIRCRESAHA.121.319874
NLM
Pauza AG, Thakkar P, Tasic T, Felippe I, Bishop P, Greenwood MP, Rysevaite-Kyguoliene K, Ast J, Broichhagen J, Hodson DJ, Salgado HC, Pauza DH, Japundzic-Zigon N, Paton JFR, Murphy D. GLP1R attenuates sympathetic response to high glucose via carotid body inhibition [Internet]. Circulation Research. 2022 ; 130( 5): 694-707.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1161/CIRCRESAHA.121.319874
Vancouver
Pauza AG, Thakkar P, Tasic T, Felippe I, Bishop P, Greenwood MP, Rysevaite-Kyguoliene K, Ast J, Broichhagen J, Hodson DJ, Salgado HC, Pauza DH, Japundzic-Zigon N, Paton JFR, Murphy D. GLP1R attenuates sympathetic response to high glucose via carotid body inhibition [Internet]. Circulation Research. 2022 ; 130( 5): 694-707.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1161/CIRCRESAHA.121.319874
Artigo de periodico
Insulin/IGF1 signalling mediates the effects of β2- adrenergic agonist on muscle proteostasis and growth (2019)
Gonçalves, Dawit Albieiro Pinheiro ; Silveira, Wilian de Assis ; Manfredi, Leandro Henrique ; Graça, Flávia A.; Armani, Andrea; Bertaggia, Enrico; O´Neill, Brian T.; Silva, Natalia Lautherbach Ennes da ; Machado, Juliano; Nogara, Leonardo; Pereira, Marcelo G.; Arcidiacono, Diletta; Realdon, Stefano; Kahn, C. Ronald; Sandri, Marco ; Kettelhut, Isis Carmo ; Navegantes, Luiz Carlos Carvalho
Source: Journal of Cachexia, Sarcopenia and Muscle. Unidade: FMRP
Subjects: INSULINA, METABOLISMO DE PROTEÍNA, MÚSCULO ESQUELÉTICO
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ABNT
GONÇALVES, Dawit Albieiro Pinheiro et al. Insulin/IGF1 signalling mediates the effects of β2- adrenergic agonist on muscle proteostasis and growth. Journal of Cachexia, Sarcopenia and Muscle, v. 10, n. 2, p. 455-475, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcsm.12395. Acesso em: 09 nov. 2024.
APA
Gonçalves, D. A. P., Silveira, W. de A., Manfredi, L. H., Graça, F. A., Armani, A., Bertaggia, E., et al. (2019). Insulin/IGF1 signalling mediates the effects of β2- adrenergic agonist on muscle proteostasis and growth. Journal of Cachexia, Sarcopenia and Muscle, 10( 2), 455-475. doi:10.1002/jcsm.12395
NLM
Gonçalves DAP, Silveira W de A, Manfredi LH, Graça FA, Armani A, Bertaggia E, O´Neill BT, Silva NLE da, Machado J, Nogara L, Pereira MG, Arcidiacono D, Realdon S, Kahn CR, Sandri M, Kettelhut IC, Navegantes LCC. Insulin/IGF1 signalling mediates the effects of β2- adrenergic agonist on muscle proteostasis and growth [Internet]. Journal of Cachexia, Sarcopenia and Muscle. 2019 ; 10( 2): 455-475.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/jcsm.12395
Vancouver
Gonçalves DAP, Silveira W de A, Manfredi LH, Graça FA, Armani A, Bertaggia E, O´Neill BT, Silva NLE da, Machado J, Nogara L, Pereira MG, Arcidiacono D, Realdon S, Kahn CR, Sandri M, Kettelhut IC, Navegantes LCC. Insulin/IGF1 signalling mediates the effects of β2- adrenergic agonist on muscle proteostasis and growth [Internet]. Journal of Cachexia, Sarcopenia and Muscle. 2019 ; 10( 2): 455-475.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1002/jcsm.12395
Artigo de periodico
The role of endocrine disruptors in ocular surface diseases (2019)
Pontelli, Regina Celia Nucci ; Souza, Marília Cristina Oliveira ; Fantucci, Marina Zílio ; Andrade, Mônica de; Rocha, Eduardo Melani
Source: Medical Hypotheses. Unidades: FMRP, FCFRP
Subjects: OFTALMOPATIAS, HOMEOSTASE, HORMÔNIOS SEXUAIS, HORMÔNIOS TIREOIDIANOS, INSULINA
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ABNT
PONTELLI, Regina Celia Nucci et al. The role of endocrine disruptors in ocular surface diseases. Medical Hypotheses, v. 122, p. 157-164, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mehy.2018.11.009. Acesso em: 09 nov. 2024.
APA
Pontelli, R. C. N., Souza, M. C. O., Fantucci, M. Z., Andrade, M. de, & Rocha, E. M. (2019). The role of endocrine disruptors in ocular surface diseases. Medical Hypotheses, 122, 157-164. doi:10.1016/j.mehy.2018.11.009
NLM
Pontelli RCN, Souza MCO, Fantucci MZ, Andrade M de, Rocha EM. The role of endocrine disruptors in ocular surface diseases [Internet]. Medical Hypotheses. 2019 ; 122 157-164.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.mehy.2018.11.009
Vancouver
Pontelli RCN, Souza MCO, Fantucci MZ, Andrade M de, Rocha EM. The role of endocrine disruptors in ocular surface diseases [Internet]. Medical Hypotheses. 2019 ; 122 157-164.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.mehy.2018.11.009
Artigo de periodico
Abnormal T-Cell development in the thymus of non-obese diabetic mice: possible relationship with the pathogenesis of type 1 autoimmune diabetes (2018)
Mendes-da-Cruz, Daniella A.; Lemos, Julia P.; Passos, Geraldo Aleixo Silva; Savino, Wilson
Source: Frontiers in Endocrinology. Unidade: FORP
Subjects: DIABETES MELLITUS, DOENÇAS AUTOIMUNES, INSULINA, FATOR DE CRESCIMENTO INSULIN-LIKE I, MICRORNAS, TIMO (GLÂNDULA ENDÓCRINA)
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ABNT
MENDES-DA-CRUZ, Daniella A. et al. Abnormal T-Cell development in the thymus of non-obese diabetic mice: possible relationship with the pathogenesis of type 1 autoimmune diabetes. Frontiers in Endocrinology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fendo.2018.00381. Acesso em: 09 nov. 2024.
APA
Mendes-da-Cruz, D. A., Lemos, J. P., Passos, G. A. S., & Savino, W. (2018). Abnormal T-Cell development in the thymus of non-obese diabetic mice: possible relationship with the pathogenesis of type 1 autoimmune diabetes. Frontiers in Endocrinology, 9. doi:10.3389/fendo.2018.00381
NLM
Mendes-da-Cruz DA, Lemos JP, Passos GAS, Savino W. Abnormal T-Cell development in the thymus of non-obese diabetic mice: possible relationship with the pathogenesis of type 1 autoimmune diabetes [Internet]. Frontiers in Endocrinology. 2018 ; 9[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fendo.2018.00381
Vancouver
Mendes-da-Cruz DA, Lemos JP, Passos GAS, Savino W. Abnormal T-Cell development in the thymus of non-obese diabetic mice: possible relationship with the pathogenesis of type 1 autoimmune diabetes [Internet]. Frontiers in Endocrinology. 2018 ; 9[citado 2024 nov. 09 ] Available from: https://doi.org/10.3389/fendo.2018.00381
Artigo de periodico
Diabetes mellitus and sepsis: A challenging association (2017)
Trevelin, Silvia C.; Carlos, Daniela; Beretta, Matteo; Silva, João Santana da; Cunha, Fernando de Queiroz
Source: Shock. Unidade: FMRP
Subjects: DIABETES MELLITUS, SEPTICEMIA, INSULINA, SEPSE, GLICOSE
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ABNT
TREVELIN, Silvia C. et al. Diabetes mellitus and sepsis: A challenging association. Shock, v. 43, n. 7, p. 276-287, 2017Tradução . . Disponível em: https://doi.org/10.1097/SHK.0000000000000778. Acesso em: 09 nov. 2024.
APA
Trevelin, S. C., Carlos, D., Beretta, M., Silva, J. S. da, & Cunha, F. de Q. (2017). Diabetes mellitus and sepsis: A challenging association. Shock, 43( 7), 276-287. doi:10.1097/SHK.0000000000000778
NLM
Trevelin SC, Carlos D, Beretta M, Silva JS da, Cunha F de Q. Diabetes mellitus and sepsis: A challenging association [Internet]. Shock. 2017 ; 43( 7): 276-287.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1097/SHK.0000000000000778
Vancouver
Trevelin SC, Carlos D, Beretta M, Silva JS da, Cunha F de Q. Diabetes mellitus and sepsis: A challenging association [Internet]. Shock. 2017 ; 43( 7): 276-287.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1097/SHK.0000000000000778
Artigo de periodico
Overexpression of Mitogen-activated protein kinase phosphatase-3 (MKP-3) reduces FoxO1 phosphorylation in mice hypothalamus (2017)
Rodrigues, Bárbara de Almeida; Kuga, Gabriel Keine; Muñoz, Vitor Rosetto; Gaspar, Rafael Calais; Tavares, Mariana Rosolen; Botezelli, José Diego; Silva, Adelino Sanchez Ramos da ; Cintra, Dennys Esper; Moura, Leandro Pereira de; Simabuco, Fernando Moreira; Ropelle, Eduardo Rochete; Pauli, José Rodrigo
Source: Neuroscience Letters. Unidade: EEFERP
Subjects: HIPOTÁLAMO, INSULINA, REGULAÇÃO GÊNICA
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ABNT
RODRIGUES, Bárbara de Almeida et al. Overexpression of Mitogen-activated protein kinase phosphatase-3 (MKP-3) reduces FoxO1 phosphorylation in mice hypothalamus. Neuroscience Letters, v. 659, p. 14-17, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2017.08.067. Acesso em: 09 nov. 2024.
APA
Rodrigues, B. de A., Kuga, G. K., Muñoz, V. R., Gaspar, R. C., Tavares, M. R., Botezelli, J. D., et al. (2017). Overexpression of Mitogen-activated protein kinase phosphatase-3 (MKP-3) reduces FoxO1 phosphorylation in mice hypothalamus. Neuroscience Letters, 659, 14-17. doi:10.1016/j.neulet.2017.08.067
NLM
Rodrigues B de A, Kuga GK, Muñoz VR, Gaspar RC, Tavares MR, Botezelli JD, Silva ASR da, Cintra DE, Moura LP de, Simabuco FM, Ropelle ER, Pauli JR. Overexpression of Mitogen-activated protein kinase phosphatase-3 (MKP-3) reduces FoxO1 phosphorylation in mice hypothalamus [Internet]. Neuroscience Letters. 2017 ; 659 14-17.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.neulet.2017.08.067
Vancouver
Rodrigues B de A, Kuga GK, Muñoz VR, Gaspar RC, Tavares MR, Botezelli JD, Silva ASR da, Cintra DE, Moura LP de, Simabuco FM, Ropelle ER, Pauli JR. Overexpression of Mitogen-activated protein kinase phosphatase-3 (MKP-3) reduces FoxO1 phosphorylation in mice hypothalamus [Internet]. Neuroscience Letters. 2017 ; 659 14-17.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.neulet.2017.08.067
Texto na web
Usefulness of circulating E-selectin to early detection of the atherosclerotic process in the Brazilian Longitudinal Study of Adult Health (ELSA-Brasil) (2016)
Almeida-Pititto, Bianca de; Ribeiro-Filho, Fernando Flexa; Bittencourt, Márcio Sommer; Lotufo, Paulo Andrade; Benseñor, Isabela M; Ferreira, Sandra R G
Source: Diabetology & Metabolic Syndrome. Unidade: HU
Subjects: INSULINA, MOLÉCULAS DE ADESÃO CELULAR, FATORES DE RISCO, ARTERIOSCLEROSE, ESTUDOS LONGITUDINAIS, SAÚDE DO ADULTO
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ABNT
ALMEIDA-PITITTO, Bianca de et al. Usefulness of circulating E-selectin to early detection of the atherosclerotic process in the Brazilian Longitudinal Study of Adult Health (ELSA-Brasil). Diabetology & Metabolic Syndrome. London: BioMed Central. Disponível em: https://doi.org/10.1186/s13098-016-0133-9. Acesso em: 09 nov. 2024. , 2016
APA
Almeida-Pititto, B. de, Ribeiro-Filho, F. F., Bittencourt, M. S., Lotufo, P. A., Benseñor, I. M., & Ferreira, S. R. G. (2016). Usefulness of circulating E-selectin to early detection of the atherosclerotic process in the Brazilian Longitudinal Study of Adult Health (ELSA-Brasil). Diabetology & Metabolic Syndrome. London: BioMed Central. doi:10.1186/s13098-016-0133-9
NLM
Almeida-Pititto B de, Ribeiro-Filho FF, Bittencourt MS, Lotufo PA, Benseñor IM, Ferreira SRG. Usefulness of circulating E-selectin to early detection of the atherosclerotic process in the Brazilian Longitudinal Study of Adult Health (ELSA-Brasil) [Internet]. Diabetology & Metabolic Syndrome. 2016 ; 8 7 .[citado 2024 nov. 09 ] Available from: https://doi.org/10.1186/s13098-016-0133-9
Vancouver
Almeida-Pititto B de, Ribeiro-Filho FF, Bittencourt MS, Lotufo PA, Benseñor IM, Ferreira SRG. Usefulness of circulating E-selectin to early detection of the atherosclerotic process in the Brazilian Longitudinal Study of Adult Health (ELSA-Brasil) [Internet]. Diabetology & Metabolic Syndrome. 2016 ; 8 7 .[citado 2024 nov. 09 ] Available from: https://doi.org/10.1186/s13098-016-0133-9
Artigo de periodico
Excessive training impairs the insulin signal transduction in mice skeletal muscles (2016)
Pereira, Bruno C; Rocha, Alisson L. da; Pinto, Ana P.; Pauli, José R.; Moura, Leandro P. de; Mekary, Rania A.; Freitas, Ellen Cristini de ; Silva, Adelino Sanchez Ramos da
Source: Journal of Endocrinology. Unidade: EEFERP
Subjects: EXERCÍCIO FÍSICO, INSULINA, MEMBRANA PLASMÁTICA, RATOS
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ABNT
PEREIRA, Bruno C et al. Excessive training impairs the insulin signal transduction in mice skeletal muscles. Journal of Endocrinology, v. 230, n. 1, p. 93-104, 2016Tradução . . Disponível em: https://doi.org/10.1530/joe-16-0063. Acesso em: 09 nov. 2024.
APA
Pereira, B. C., Rocha, A. L. da, Pinto, A. P., Pauli, J. R., Moura, L. P. de, Mekary, R. A., et al. (2016). Excessive training impairs the insulin signal transduction in mice skeletal muscles. Journal of Endocrinology, 230( 1), 93-104. doi:10.1530/joe-16-0063
NLM
Pereira BC, Rocha AL da, Pinto AP, Pauli JR, Moura LP de, Mekary RA, Freitas EC de, Silva ASR da. Excessive training impairs the insulin signal transduction in mice skeletal muscles [Internet]. Journal of Endocrinology. 2016 ; 230( 1): 93-104.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1530/joe-16-0063
Vancouver
Pereira BC, Rocha AL da, Pinto AP, Pauli JR, Moura LP de, Mekary RA, Freitas EC de, Silva ASR da. Excessive training impairs the insulin signal transduction in mice skeletal muscles [Internet]. Journal of Endocrinology. 2016 ; 230( 1): 93-104.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1530/joe-16-0063
Trabalho de evento-resumo periodico
Marker of endothelial dysfunction may improve the early detection of insulin resistance and subclinical atherosclerosis (2015)
Almeida-Pititto, Bianca de; Ribeiro Filho, Fernando Flexa; Bittencourt, Márcio Sommer; Benseñor, Isabela M; Lotufo, Paulo Andrade; Ferreira, Sandra Roberta Gouvea
Source: Diabetology & Metabolic Syndrome. Conference titles: Brazilian Diabetes Society Congress. Unidades: HU, FM, FSP
Subjects: ENDOTÉLIO VASCULAR, INSULINA, ARTERIOSCLEROSE, ESTUDOS LONGITUDINAIS, SAÚDE DO ADULTO
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ABNT
ALMEIDA-PITITTO, Bianca de et al. Marker of endothelial dysfunction may improve the early detection of insulin resistance and subclinical atherosclerosis. Diabetology & Metabolic Syndrome. London: BioMed Central. Disponível em: https://doi.org/10.1186/1758-5996-7-S1-A113. Acesso em: 09 nov. 2024. , 2015
APA
Almeida-Pititto, B. de, Ribeiro Filho, F. F., Bittencourt, M. S., Benseñor, I. M., Lotufo, P. A., & Ferreira, S. R. G. (2015). Marker of endothelial dysfunction may improve the early detection of insulin resistance and subclinical atherosclerosis. Diabetology & Metabolic Syndrome. London: BioMed Central. doi:10.1186/1758-5996-7-S1-A113
NLM
Almeida-Pititto B de, Ribeiro Filho FF, Bittencourt MS, Benseñor IM, Lotufo PA, Ferreira SRG. Marker of endothelial dysfunction may improve the early detection of insulin resistance and subclinical atherosclerosis [Internet]. Diabetology & Metabolic Syndrome. 2015 ; 7 A113.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1186/1758-5996-7-S1-A113
Vancouver
Almeida-Pititto B de, Ribeiro Filho FF, Bittencourt MS, Benseñor IM, Lotufo PA, Ferreira SRG. Marker of endothelial dysfunction may improve the early detection of insulin resistance and subclinical atherosclerosis [Internet]. Diabetology & Metabolic Syndrome. 2015 ; 7 A113.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1186/1758-5996-7-S1-A113
Artigo de periodico
Antidiabetic and antilipidemic effects of Manilkara zapota (2015)
Barbalho, Sandra Maria; Bueno, Patrícia Cincotto dos Santos; Delazari, Débora Souza; Guiguer, Elen Landgraf; Coqueiro, Daniel Pereira; Araújo, Adriano Cressoni; Souza, Maricelma da Silva Soares de; Farinazzi-Machado, Flávia M. V; Mendes, Claudemir Gregório; Groppo, Milton
Source: Journal of Medicinal Food. Unidade: FFCLRP
Subjects: ERICALES (COMPONENTES), GLICEMIA, INSULINA, LEPTINA
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ABNT
BARBALHO, Sandra Maria et al. Antidiabetic and antilipidemic effects of Manilkara zapota. Journal of Medicinal Food, v. 18, n. 3, p. 385-391, 2015Tradução . . Disponível em: https://doi.org/10.1089/jmf.2013.0170. Acesso em: 09 nov. 2024.
APA
Barbalho, S. M., Bueno, P. C. dos S., Delazari, D. S., Guiguer, E. L., Coqueiro, D. P., Araújo, A. C., et al. (2015). Antidiabetic and antilipidemic effects of Manilkara zapota. Journal of Medicinal Food, 18( 3), 385-391. doi:10.1089/jmf.2013.0170
NLM
Barbalho SM, Bueno PC dos S, Delazari DS, Guiguer EL, Coqueiro DP, Araújo AC, Souza M da SS de, Farinazzi-Machado FMV, Mendes CG, Groppo M. Antidiabetic and antilipidemic effects of Manilkara zapota [Internet]. Journal of Medicinal Food. 2015 ; 18( 3): 385-391.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1089/jmf.2013.0170
Vancouver
Barbalho SM, Bueno PC dos S, Delazari DS, Guiguer EL, Coqueiro DP, Araújo AC, Souza M da SS de, Farinazzi-Machado FMV, Mendes CG, Groppo M. Antidiabetic and antilipidemic effects of Manilkara zapota [Internet]. Journal of Medicinal Food. 2015 ; 18( 3): 385-391.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1089/jmf.2013.0170
Artigo de periodico
The role of gut-liver axis in the restriction of intrauterine growth in a model of experimental gastroschisis (2013)
Bueno, Márcia Pereira; Gonçalves, Frances Lilian Lanhellas; Guadagnini, Dioze; Barini, Ricardo; Saad, Mario José Abdalla; Gallindo, Rodrigo Melo; Schmidt, Augusto Frederico; Sbragia, Lourenço
Source: Acta Cirúrgica Brasileira. Unidade: FMRP
Subjects: FETO (CRESCIMENTO E DESENVOLVIMENTO), RECEPTORES, INSULINA, RATOS (EXPERIMENTOS)
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ABNT
BUENO, Márcia Pereira et al. The role of gut-liver axis in the restriction of intrauterine growth in a model of experimental gastroschisis. Acta Cirúrgica Brasileira, v. 28, p. 3-7, 2013Tradução . . Disponível em: https://doi.org/10.1590/S0102-86502013001300002. Acesso em: 09 nov. 2024.
APA
Bueno, M. P., Gonçalves, F. L. L., Guadagnini, D., Barini, R., Saad, M. J. A., Gallindo, R. M., et al. (2013). The role of gut-liver axis in the restriction of intrauterine growth in a model of experimental gastroschisis. Acta Cirúrgica Brasileira, 28, 3-7. doi:10.1590/S0102-86502013001300002
NLM
Bueno MP, Gonçalves FLL, Guadagnini D, Barini R, Saad MJA, Gallindo RM, Schmidt AF, Sbragia L. The role of gut-liver axis in the restriction of intrauterine growth in a model of experimental gastroschisis [Internet]. Acta Cirúrgica Brasileira. 2013 ; 28 3-7.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1590/S0102-86502013001300002
Vancouver
Bueno MP, Gonçalves FLL, Guadagnini D, Barini R, Saad MJA, Gallindo RM, Schmidt AF, Sbragia L. The role of gut-liver axis in the restriction of intrauterine growth in a model of experimental gastroschisis [Internet]. Acta Cirúrgica Brasileira. 2013 ; 28 3-7.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1590/S0102-86502013001300002
Artigo de periodico
Obesity due to Melanocortin 4 Receptor (MC4R) Deficiency Is Associated with Increased Linear Growth and Final Height, Fasting Hyperinsulinemia, and Incompletely Suppressed Growth Hormone Secretion (2011)
Martinelli Junior, Carlos Eduardo; Keogh, Julia M.; Greenfield, Jerry R.; Henning, Elana; Klaauw, Agatha A. van der; Blackwood, Anthea; O’Rahilly, Stephen; Roelfsema, Ferdinand; Camacho-Hübner, Cecilia; Pijl, Hanno; Farooqi, I. Sadaf
Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FMRP
Subjects: OBESIDADE (GENÉTICA), ANTROPOMETRIA, PROTEÍNAS DE TRANSPORTE, GLICOPROTEÍNAS, INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTINELLI JUNIOR, Carlos Eduardo et al. Obesity due to Melanocortin 4 Receptor (MC4R) Deficiency Is Associated with Increased Linear Growth and Final Height, Fasting Hyperinsulinemia, and Incompletely Suppressed Growth Hormone Secretion. Journal of Clinical Endocrinology and Metabolism, v. 96, n. 1, p. E181-E188, 2011Tradução . . Disponível em: https://doi.org/10.1210/jc.2010-1369. Acesso em: 09 nov. 2024.
APA
Martinelli Junior, C. E., Keogh, J. M., Greenfield, J. R., Henning, E., Klaauw, A. A. van der, Blackwood, A., et al. (2011). Obesity due to Melanocortin 4 Receptor (MC4R) Deficiency Is Associated with Increased Linear Growth and Final Height, Fasting Hyperinsulinemia, and Incompletely Suppressed Growth Hormone Secretion. Journal of Clinical Endocrinology and Metabolism, 96( 1), E181-E188. doi:10.1210/jc.2010-1369
NLM
Martinelli Junior CE, Keogh JM, Greenfield JR, Henning E, Klaauw AA van der, Blackwood A, O’Rahilly S, Roelfsema F, Camacho-Hübner C, Pijl H, Farooqi IS. Obesity due to Melanocortin 4 Receptor (MC4R) Deficiency Is Associated with Increased Linear Growth and Final Height, Fasting Hyperinsulinemia, and Incompletely Suppressed Growth Hormone Secretion [Internet]. Journal of Clinical Endocrinology and Metabolism. 2011 ; 96( 1): E181-E188.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1210/jc.2010-1369
Vancouver
Martinelli Junior CE, Keogh JM, Greenfield JR, Henning E, Klaauw AA van der, Blackwood A, O’Rahilly S, Roelfsema F, Camacho-Hübner C, Pijl H, Farooqi IS. Obesity due to Melanocortin 4 Receptor (MC4R) Deficiency Is Associated with Increased Linear Growth and Final Height, Fasting Hyperinsulinemia, and Incompletely Suppressed Growth Hormone Secretion [Internet]. Journal of Clinical Endocrinology and Metabolism. 2011 ; 96( 1): E181-E188.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1210/jc.2010-1369
Artigo de periodico
Generation and characterization of human insulin-releasing cell lines (2009)
Labriola, Leticia ; Peters, María Giselle; Krogh, Karin; Stigliano, Ivan; Terra, Letícia Ferreira; Buchanan, Leticia; Machado, Marcel Cerqueira César; Joffé, Elisa Bal de Kier; Puricelli, Lydia; Sogayar, Mari Cleide
Source: BMC Cell Biology. Unidades: FM, IQ
Subjects: INSULINA, LINHAGEM CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LABRIOLA, Leticia et al. Generation and characterization of human insulin-releasing cell lines. BMC Cell Biology, v. 10, n. 49, p. 1-16, 2009Tradução . . Disponível em: https://doi.org/10.1186/1471-2121-10-49. Acesso em: 09 nov. 2024.
APA
Labriola, L., Peters, M. G., Krogh, K., Stigliano, I., Terra, L. F., Buchanan, L., et al. (2009). Generation and characterization of human insulin-releasing cell lines. BMC Cell Biology, 10( 49), 1-16. doi:10.1186/1471-2121-10-49
NLM
Labriola L, Peters MG, Krogh K, Stigliano I, Terra LF, Buchanan L, Machado MCC, Joffé EB de K, Puricelli L, Sogayar MC. Generation and characterization of human insulin-releasing cell lines [Internet]. BMC Cell Biology. 2009 ; 10( 49): 1-16.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1186/1471-2121-10-49
Vancouver
Labriola L, Peters MG, Krogh K, Stigliano I, Terra LF, Buchanan L, Machado MCC, Joffé EB de K, Puricelli L, Sogayar MC. Generation and characterization of human insulin-releasing cell lines [Internet]. BMC Cell Biology. 2009 ; 10( 49): 1-16.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1186/1471-2121-10-49

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