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Artigo de periodico
Exercise improves orofacial pain and modifies neuropeptide expression in a rat model of Parkinson’s disease (2023)
Binda, Karina Henrique ; Daniel Oliveira Martins ; Chacur, Marucia
Source: Neurotoxicity Research. Unidade: ICB
Subjects: ANOTOMIA, DOR, RATOS, MODELOS ANIMAIS DE DOENÇAS, NEUROQUÍMICA DE ANIMAL, NEUROPEPTÍDEOS, CALCITONINA, HORMÔNIOS DO METABOLISMO OSTEOMINERAL, ANTAGONISTAS, RECEPTORES DE DROGA, CANABINOIDES, DOENÇA DE PARKINSON
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ABNT
BINDA, Karina Henrique e DANIEL OLIVEIRA MARTINS, e CHACUR, Marucia. Exercise improves orofacial pain and modifies neuropeptide expression in a rat model of Parkinson’s disease. Neurotoxicity Research, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1007/s12640-023-00651-6. Acesso em: 10 nov. 2024.
APA
Binda, K. H., Daniel Oliveira Martins,, & Chacur, M. (2023). Exercise improves orofacial pain and modifies neuropeptide expression in a rat model of Parkinson’s disease. Neurotoxicity Research, 1-12. doi:10.1007/s12640-023-00651-6
NLM
Binda KH, Daniel Oliveira Martins, Chacur M. Exercise improves orofacial pain and modifies neuropeptide expression in a rat model of Parkinson’s disease [Internet]. Neurotoxicity Research. 2023 ; 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-023-00651-6
Vancouver
Binda KH, Daniel Oliveira Martins, Chacur M. Exercise improves orofacial pain and modifies neuropeptide expression in a rat model of Parkinson’s disease [Internet]. Neurotoxicity Research. 2023 ; 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-023-00651-6
Artigo de periodico
Neurotoxicity assessment of 1-[(2,3-Dihydro-1-Benzofuran-2-yl)Methyl]Piperazine (LINS01 Series) derivatives and their protective effect on cocaine-induced neurotoxicity model in SH-SY5Y cell culture (2022)
Santos, Laísa Aliandro dos ; Santos, Gabriela Salles dos; Fernandes, Gustavo Ariel Borges ; Corrêa, Michelle Fidelis; Almeida, Carolina Aparecida de Faria; Fernandes, Liliam ; Marcourakis, Tania ; Fernandes, João Paulo S; Garcia, Raphael Caio Tamborelli
Source: Neurotoxicity Research. Unidade: FCF
Subjects: COCAÍNA, RECEPTORES DOPAMINÉRGICOS
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SANTOS, Laísa Aliandro dos et al. Neurotoxicity assessment of 1-[(2,3-Dihydro-1-Benzofuran-2-yl)Methyl]Piperazine (LINS01 Series) derivatives and their protective effect on cocaine-induced neurotoxicity model in SH-SY5Y cell culture. Neurotoxicity Research, v. 40, n. 6, p. 1653-1663, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12640-022-00601-8. Acesso em: 10 nov. 2024.
APA
Santos, L. A. dos, Santos, G. S. dos, Fernandes, G. A. B., Corrêa, M. F., Almeida, C. A. de F., Fernandes, L., et al. (2022). Neurotoxicity assessment of 1-[(2,3-Dihydro-1-Benzofuran-2-yl)Methyl]Piperazine (LINS01 Series) derivatives and their protective effect on cocaine-induced neurotoxicity model in SH-SY5Y cell culture. Neurotoxicity Research, 40( 6), 1653-1663. doi:10.1007/s12640-022-00601-8
NLM
Santos LA dos, Santos GS dos, Fernandes GAB, Corrêa MF, Almeida CA de F, Fernandes L, Marcourakis T, Fernandes JPS, Garcia RCT. Neurotoxicity assessment of 1-[(2,3-Dihydro-1-Benzofuran-2-yl)Methyl]Piperazine (LINS01 Series) derivatives and their protective effect on cocaine-induced neurotoxicity model in SH-SY5Y cell culture [Internet]. Neurotoxicity Research. 2022 ; 40( 6): 1653-1663.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-022-00601-8
Vancouver
Santos LA dos, Santos GS dos, Fernandes GAB, Corrêa MF, Almeida CA de F, Fernandes L, Marcourakis T, Fernandes JPS, Garcia RCT. Neurotoxicity assessment of 1-[(2,3-Dihydro-1-Benzofuran-2-yl)Methyl]Piperazine (LINS01 Series) derivatives and their protective effect on cocaine-induced neurotoxicity model in SH-SY5Y cell culture [Internet]. Neurotoxicity Research. 2022 ; 40( 6): 1653-1663.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-022-00601-8
Artigo de periodico
Global proteomic profile integrated to quantitative and morphometric assessment of enteric neurons: investigation of the mechanisms involved in the toxicity induced by acute fluoride exposure in the duodenum (2021)
Melo, Carina Guimaraes de Souza ; Zanoni, Jacqueline Nelisis; Souza, Sara Raquel Garcia de; Zignani, Isabela; Leite, Aline de Lima; Heubel, Alessandro Domingues; Perles, Juliana Vanessa Colombo Martins; Buzalaf, Marília Afonso Rabelo
Source: Neurotoxicity Research. Unidade: FOB
Subjects: PROTEÔMICA, MORFOMETRIA, NEURÔNIOS, TOXICIDADE DO FLÚOR, FLÚOR, DUODENO, SISTEMA NERVOSO ENTÉRICO
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MELO, Carina Guimaraes de Souza et al. Global proteomic profile integrated to quantitative and morphometric assessment of enteric neurons: investigation of the mechanisms involved in the toxicity induced by acute fluoride exposure in the duodenum. Neurotoxicity Research, v. 39, n. 3, p. 800-814, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12640-020-00296-9. Acesso em: 10 nov. 2024.
APA
Melo, C. G. de S., Zanoni, J. N., Souza, S. R. G. de, Zignani, I., Leite, A. de L., Heubel, A. D., et al. (2021). Global proteomic profile integrated to quantitative and morphometric assessment of enteric neurons: investigation of the mechanisms involved in the toxicity induced by acute fluoride exposure in the duodenum. Neurotoxicity Research, 39( 3), 800-814. doi:10.1007/s12640-020-00296-9
NLM
Melo CG de S, Zanoni JN, Souza SRG de, Zignani I, Leite A de L, Heubel AD, Perles JVCM, Buzalaf MAR. Global proteomic profile integrated to quantitative and morphometric assessment of enteric neurons: investigation of the mechanisms involved in the toxicity induced by acute fluoride exposure in the duodenum [Internet]. Neurotoxicity Research. 2021 ; 39( 3): 800-814.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00296-9
Vancouver
Melo CG de S, Zanoni JN, Souza SRG de, Zignani I, Leite A de L, Heubel AD, Perles JVCM, Buzalaf MAR. Global proteomic profile integrated to quantitative and morphometric assessment of enteric neurons: investigation of the mechanisms involved in the toxicity induced by acute fluoride exposure in the duodenum [Internet]. Neurotoxicity Research. 2021 ; 39( 3): 800-814.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00296-9
Artigo de periodico
Interferon-γ involvement in the neuroinflammation associated with Parkinson’s disease and L-DOPA-induced dyskinesia (2021)
Ferrari, Daniele Pereira ; Bortolanza, Mariza ; Del Bel, Elaine Aparecida
Source: Neurotoxicity Research. Unidades: FMRP, FORP
Subjects: NEUROLOGIA, FÁRMACOS
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FERRARI, Daniele Pereira e BORTOLANZA, Mariza e DEL BEL, Elaine Aparecida. Interferon-γ involvement in the neuroinflammation associated with Parkinson’s disease and L-DOPA-induced dyskinesia. Neurotoxicity Research, v. 39, p. 705-719, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12640-021-00345-x. Acesso em: 10 nov. 2024.
APA
Ferrari, D. P., Bortolanza, M., & Del Bel, E. A. (2021). Interferon-γ involvement in the neuroinflammation associated with Parkinson’s disease and L-DOPA-induced dyskinesia. Neurotoxicity Research, 39, 705-719. doi:10.1007/s12640-021-00345-x
NLM
Ferrari DP, Bortolanza M, Del Bel EA. Interferon-γ involvement in the neuroinflammation associated with Parkinson’s disease and L-DOPA-induced dyskinesia [Internet]. Neurotoxicity Research. 2021 ; 39 705-719.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-021-00345-x
Vancouver
Ferrari DP, Bortolanza M, Del Bel EA. Interferon-γ involvement in the neuroinflammation associated with Parkinson’s disease and L-DOPA-induced dyskinesia [Internet]. Neurotoxicity Research. 2021 ; 39 705-719.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-021-00345-x
Artigo de periodico
Astrocyte intracellular Ca2+and TrkB signaling in the hippocampus could be involved in the beneficial behavioral effects of antidepressant treatment (2021)
Ferreira, Frederico Rogério; Cupido, Alexander; Catalin, Bogdan; Silva Junior, Wilson Araújo da ; Kirchhoff, Frank; Del Bel, Elaine Aparecida ; Guimarães, Francisco Silveira
Source: Neurotoxicity Research. Unidades: FMRP, FORP
Subjects: ASTRÓCITOS, CALMODULINA, ANTIDEPRESSIVOS
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ABNT
FERREIRA, Frederico Rogério et al. Astrocyte intracellular Ca2+and TrkB signaling in the hippocampus could be involved in the beneficial behavioral effects of antidepressant treatment. Neurotoxicity Research, v. 39, n. 3, p. 860-871, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12640-021-00334-0. Acesso em: 10 nov. 2024.
APA
Ferreira, F. R., Cupido, A., Catalin, B., Silva Junior, W. A. da, Kirchhoff, F., Del Bel, E. A., & Guimarães, F. S. (2021). Astrocyte intracellular Ca2+and TrkB signaling in the hippocampus could be involved in the beneficial behavioral effects of antidepressant treatment. Neurotoxicity Research, 39( 3), 860-871. doi:10.1007/s12640-021-00334-0
NLM
Ferreira FR, Cupido A, Catalin B, Silva Junior WA da, Kirchhoff F, Del Bel EA, Guimarães FS. Astrocyte intracellular Ca2+and TrkB signaling in the hippocampus could be involved in the beneficial behavioral effects of antidepressant treatment [Internet]. Neurotoxicity Research. 2021 ; 39( 3): 860-871.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-021-00334-0
Vancouver
Ferreira FR, Cupido A, Catalin B, Silva Junior WA da, Kirchhoff F, Del Bel EA, Guimarães FS. Astrocyte intracellular Ca2+and TrkB signaling in the hippocampus could be involved in the beneficial behavioral effects of antidepressant treatment [Internet]. Neurotoxicity Research. 2021 ; 39( 3): 860-871.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-021-00334-0
Artigo de periodico
Endocannabinoid system attenuates oxaliplatin-induced peripheral sensory neuropathy through the activation of CB1 receptors (2021)
Pereira, Anamaria Falcão; Lisboa, Mario Roberto Pontes; Alves, Bruno Wesley de Freitas; Silva, Cristiane Maria Pereira da; Dias, Diego Bernarde Souza; Menezes, Karoline Luanne Santos; Cesário, Francisco Rafael Alves Santana; França, Jonas Costa de; Oliveira, Amanda Rocha de; Hallak, Jaime Eduardo Cecilio ; Zuardi, Antonio Waldo; Crippa, José Alexandre de Souza ; Alencar, Nylane Maria Nunes de; Lima Júnior, Roberto Cesar Pereira; Vale, Mariana Lima
Source: Neurotoxicity Research. Unidade: FMRP
Subjects: GÂNGLIOS ESPINHAIS, NOCICEPTIVIDADE, ANTINEOPLÁSICOS, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, CANABINOIDES
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PEREIRA, Anamaria Falcão et al. Endocannabinoid system attenuates oxaliplatin-induced peripheral sensory neuropathy through the activation of CB1 receptors. Neurotoxicity Research, v. 39, n. 6, p. 1782-1799, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12640-021-00442-x. Acesso em: 10 nov. 2024.
APA
Pereira, A. F., Lisboa, M. R. P., Alves, B. W. de F., Silva, C. M. P. da, Dias, D. B. S., Menezes, K. L. S., et al. (2021). Endocannabinoid system attenuates oxaliplatin-induced peripheral sensory neuropathy through the activation of CB1 receptors. Neurotoxicity Research, 39( 6), 1782-1799. doi:10.1007/s12640-021-00442-x
NLM
Pereira AF, Lisboa MRP, Alves BW de F, Silva CMP da, Dias DBS, Menezes KLS, Cesário FRAS, França JC de, Oliveira AR de, Hallak JEC, Zuardi AW, Crippa JA de S, Alencar NMN de, Lima Júnior RCP, Vale ML. Endocannabinoid system attenuates oxaliplatin-induced peripheral sensory neuropathy through the activation of CB1 receptors [Internet]. Neurotoxicity Research. 2021 ; 39( 6): 1782-1799.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-021-00442-x
Vancouver
Pereira AF, Lisboa MRP, Alves BW de F, Silva CMP da, Dias DBS, Menezes KLS, Cesário FRAS, França JC de, Oliveira AR de, Hallak JEC, Zuardi AW, Crippa JA de S, Alencar NMN de, Lima Júnior RCP, Vale ML. Endocannabinoid system attenuates oxaliplatin-induced peripheral sensory neuropathy through the activation of CB1 receptors [Internet]. Neurotoxicity Research. 2021 ; 39( 6): 1782-1799.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-021-00442-x
Artigo de periodico
The neurotrophic-like effect of carvacrol: perspective for axonal and synaptic regeneration (2021)
Sisti, Flávia Malvestio; Santos, Neife Aparecida Guinaim dos; Amaral, Lilian do ; Santos, Antônio Cardozo dos
Source: Neurotoxicity Research. Unidade: FCFRP
Subjects: AXÔNIOS, SISTEMA NERVOSO, NEURÔNIOS
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SISTI, Flávia Malvestio et al. The neurotrophic-like effect of carvacrol: perspective for axonal and synaptic regeneration. Neurotoxicity Research, v. 39, n. 3, p. 886-896, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12640-021-00341-1. Acesso em: 10 nov. 2024.
APA
Sisti, F. M., Santos, N. A. G. dos, Amaral, L. do, & Santos, A. C. dos. (2021). The neurotrophic-like effect of carvacrol: perspective for axonal and synaptic regeneration. Neurotoxicity Research, 39( 3), 886-896. doi:10.1007/s12640-021-00341-1
NLM
Sisti FM, Santos NAG dos, Amaral L do, Santos AC dos. The neurotrophic-like effect of carvacrol: perspective for axonal and synaptic regeneration [Internet]. Neurotoxicity Research. 2021 ; 39( 3): 886-896.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-021-00341-1
Vancouver
Sisti FM, Santos NAG dos, Amaral L do, Santos AC dos. The neurotrophic-like effect of carvacrol: perspective for axonal and synaptic regeneration [Internet]. Neurotoxicity Research. 2021 ; 39( 3): 886-896.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-021-00341-1
Artigo de periodico
A synthetic snake-venom-based tripeptide protects PC12 cells from the neurotoxicity of acrolein by improving axonal plasticity and bioenergetics (2020)
Bernardes, Carolina Petri ; Santos, Neife Aparecida Guinaim dos; Costa, Tassia R.; Sisti, Flávia Malvestio; Amaral, Lilian do ; Menaldo, Danilo Luccas ; Amstalden, Martin Krähenbühl; Ribeiro, Diego Luis ; Antunes, Lusânia Maria Greggi ; Sampaio, Suely Vilela ; Santos, Antônio Cardozo dos
Source: Neurotoxicity Research. Unidades: FCFRP, FMRP
Subjects: VENENOS, COBRAS, PROTEÍNAS QUINASES, ANTAGONISTAS, TOXICIDADE EM ANIMAL, APOLIPOPROTEÍNAS, BIOMARCADORES, DIFERENCIAÇÃO CELULAR, GLICOSE, PLASTICIDADE NEURONAL
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BERNARDES, Carolina Petri et al. A synthetic snake-venom-based tripeptide protects PC12 cells from the neurotoxicity of acrolein by improving axonal plasticity and bioenergetics. Neurotoxicity Research, v. 37, n. 1, p. 227-237, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-019-00111-0. Acesso em: 10 nov. 2024.
APA
Bernardes, C. P., Santos, N. A. G. dos, Costa, T. R., Sisti, F. M., Amaral, L. do, Menaldo, D. L., et al. (2020). A synthetic snake-venom-based tripeptide protects PC12 cells from the neurotoxicity of acrolein by improving axonal plasticity and bioenergetics. Neurotoxicity Research, 37( 1), 227-237. doi:10.1007/s12640-019-00111-0
NLM
Bernardes CP, Santos NAG dos, Costa TR, Sisti FM, Amaral L do, Menaldo DL, Amstalden MK, Ribeiro DL, Antunes LMG, Sampaio SV, Santos AC dos. A synthetic snake-venom-based tripeptide protects PC12 cells from the neurotoxicity of acrolein by improving axonal plasticity and bioenergetics [Internet]. Neurotoxicity Research. 2020 ; 37( 1): 227-237.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-019-00111-0
Vancouver
Bernardes CP, Santos NAG dos, Costa TR, Sisti FM, Amaral L do, Menaldo DL, Amstalden MK, Ribeiro DL, Antunes LMG, Sampaio SV, Santos AC dos. A synthetic snake-venom-based tripeptide protects PC12 cells from the neurotoxicity of acrolein by improving axonal plasticity and bioenergetics [Internet]. Neurotoxicity Research. 2020 ; 37( 1): 227-237.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-019-00111-0
Artigo de periodico
Effects of doxycycline in swiss mice predictive models of schizophrenia (2020)
Pereira, Ana Carolina de Castro Issy; Pedrazzi, João Francisco Cordeiro; Oosten, Anna Beatriz Saito van; Checheto, Thiago; Silva, Rafaela R.; Noël, François; Del Bel, Elaine Aparecida
Source: Neurotoxicity Research. Unidades: FCFRP, FORP, FMRP
Subjects: ESQUIZOFRENIA, MODELOS ANIMAIS, FARMACOLOGIA, ANTIPSICÓTICOS
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PEREIRA, Ana Carolina de Castro Issy et al. Effects of doxycycline in swiss mice predictive models of schizophrenia. Neurotoxicity Research, v. 38, n. 4, p. 1049-1060, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-020-00268-z. Acesso em: 10 nov. 2024.
APA
Pereira, A. C. de C. I., Pedrazzi, J. F. C., Oosten, A. B. S. van, Checheto, T., Silva, R. R., Noël, F., & Del Bel, E. A. (2020). Effects of doxycycline in swiss mice predictive models of schizophrenia. Neurotoxicity Research, 38( 4), 1049-1060. doi:10.1007/s12640-020-00268-z
NLM
Pereira AC de CI, Pedrazzi JFC, Oosten ABS van, Checheto T, Silva RR, Noël F, Del Bel EA. Effects of doxycycline in swiss mice predictive models of schizophrenia [Internet]. Neurotoxicity Research. 2020 ; 38( 4): 1049-1060.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00268-z
Vancouver
Pereira AC de CI, Pedrazzi JFC, Oosten ABS van, Checheto T, Silva RR, Noël F, Del Bel EA. Effects of doxycycline in swiss mice predictive models of schizophrenia [Internet]. Neurotoxicity Research. 2020 ; 38( 4): 1049-1060.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00268-z
Artigo de periodico
Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex (2020)
Kubrusly, Regina Célia Cussa; Martins, Robertta Silva; Souza, Lisiane de Santana; Carvalho, Mariana Pinheiro de; Borges-Martins, Vladimir Pedro Peralva; Sathler, Matheus Figueiredo; Ferreira, Danielle Dias Pinto; Pereira, Maurício dos Santos; Peccinalli, Ney Ronner; Pandolfo, Pablo; Reis, Ricardo Augusto de Melo; Ferreira, Gustavo Costa; Manhães, Alex Christian
Source: Neurotoxicity Research. Unidade: FORP
Subjects: COCAÍNA, MODELOS ANIMAIS, NEUROTRANSMISSORES, NEUROLOGIA, GABA, RECEPTORES, ATIVIDADE MOTORA
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ABNT
KUBRUSLY, Regina Célia Cussa et al. Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex. Neurotoxicity Research, v. 38, n. 3, p. 824-832, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-020-00259-0. Acesso em: 10 nov. 2024.
APA
Kubrusly, R. C. C., Martins, R. S., Souza, L. de S., Carvalho, M. P. de, Borges-Martins, V. P. P., Sathler, M. F., et al. (2020). Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex. Neurotoxicity Research, 38( 3), 824-832. doi:10.1007/s12640-020-00259-0
NLM
Kubrusly RCC, Martins RS, Souza L de S, Carvalho MP de, Borges-Martins VPP, Sathler MF, Ferreira DDP, Pereira M dos S, Peccinalli NR, Pandolfo P, Reis RA de M, Ferreira GC, Manhães AC. Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex [Internet]. Neurotoxicity Research. 2020 ; 38( 3): 824-832.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00259-0
Vancouver
Kubrusly RCC, Martins RS, Souza L de S, Carvalho MP de, Borges-Martins VPP, Sathler MF, Ferreira DDP, Pereira M dos S, Peccinalli NR, Pandolfo P, Reis RA de M, Ferreira GC, Manhães AC. Single cocaine exposure inhibits GABA uptake via dopamine D1-like receptors in adolescent mice frontal cortex [Internet]. Neurotoxicity Research. 2020 ; 38( 3): 824-832.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00259-0
Artigo de periodico
Genistein and galantamine combinations decrease β-amyloid peptide (1-42)-induced genotoxicity and cell death in SH-SY5Y cell line: an in vitro and in silico approach for mimic of Alzheimer’s disease (2020)
Castillo, Willian Orlando; Palomino, Nilza Velasco; Takahashi, Catarina Satie; Giuliatti, Silvana
Source: Neurotoxicity Research. Unidades: FFCLRP, FMRP
Subjects: DOENÇA DE ALZHEIMER, ESTRÓGENOS, ESTRESSE OXIDATIVO, TESTES DE TOXICIDADE
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ABNT
CASTILLO, Willian Orlando et al. Genistein and galantamine combinations decrease β-amyloid peptide (1-42)-induced genotoxicity and cell death in SH-SY5Y cell line: an in vitro and in silico approach for mimic of Alzheimer’s disease. Neurotoxicity Research, v. 38, n. 3, p. 691-706, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-020-00243-8. Acesso em: 10 nov. 2024.
APA
Castillo, W. O., Palomino, N. V., Takahashi, C. S., & Giuliatti, S. (2020). Genistein and galantamine combinations decrease β-amyloid peptide (1-42)-induced genotoxicity and cell death in SH-SY5Y cell line: an in vitro and in silico approach for mimic of Alzheimer’s disease. Neurotoxicity Research, 38( 3), 691-706. doi:10.1007/s12640-020-00243-8
NLM
Castillo WO, Palomino NV, Takahashi CS, Giuliatti S. Genistein and galantamine combinations decrease β-amyloid peptide (1-42)-induced genotoxicity and cell death in SH-SY5Y cell line: an in vitro and in silico approach for mimic of Alzheimer’s disease [Internet]. Neurotoxicity Research. 2020 ; 38( 3): 691-706.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00243-8
Vancouver
Castillo WO, Palomino NV, Takahashi CS, Giuliatti S. Genistein and galantamine combinations decrease β-amyloid peptide (1-42)-induced genotoxicity and cell death in SH-SY5Y cell line: an in vitro and in silico approach for mimic of Alzheimer’s disease [Internet]. Neurotoxicity Research. 2020 ; 38( 3): 691-706.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00243-8
Artigo de periodico
Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia (2020)
Ferreira Junior, Nilson Carlos ; Pereira, Maurício dos Santos; Guimarães, Francisco Silveira ; Del Bel, Elaine Aparecida
Source: Neurotoxicity Research. Unidades: FMRP, FORP
Subjects: RECEPTORES, CANABINOIDES, INFLAMAÇÃO, DOENÇA DE PARKINSON
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ABNT
FERREIRA JUNIOR, Nilson Carlos et al. Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia. Neurotoxicity Research, v. 37, n. 1, p. 12-29, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-019-00109-8. Acesso em: 10 nov. 2024.
APA
Ferreira Junior, N. C., Pereira, M. dos S., Guimarães, F. S., & Del Bel, E. A. (2020). Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia. Neurotoxicity Research, 37( 1), 12-29. doi:10.1007/s12640-019-00109-8
NLM
Ferreira Junior NC, Pereira M dos S, Guimarães FS, Del Bel EA. Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia [Internet]. Neurotoxicity Research. 2020 ; 37( 1): 12-29.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-019-00109-8
Vancouver
Ferreira Junior NC, Pereira M dos S, Guimarães FS, Del Bel EA. Cannabidiol and cannabinoid compounds as potential strategies for treating Parkinson’s disease and l-DOPA-induced dyskinesia [Internet]. Neurotoxicity Research. 2020 ; 37( 1): 12-29.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-019-00109-8
Artigo de periodico
Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration (2020)
Catalão, Carlos Henrique Rocha ; Santos Junior, Nilton Nascimento dos ; Costa, Luís Henrique Angenendt da ; Souza, Anderson Oliveira; Cárnio, Evelin Capellari ; Sebollela, Adriano Silva ; Alberici, Luciane Carla ; Rocha, Maria José Alves da
Source: Neurotoxicity Research. Unidades: FMRP, EERP, FCFRP, FORP
Subjects: ENCEFALOPATIA ANIMAL, HIPOCAMPO, CÓRTEX PRÉ-FRONTAL, ASTRÓCITOS
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ABNT
CATALÃO, Carlos Henrique Rocha et al. Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration. Neurotoxicity Research, v. 38, n. 4, p. 871-886, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-020-00222-z. Acesso em: 10 nov. 2024.
APA
Catalão, C. H. R., Santos Junior, N. N. dos, Costa, L. H. A. da, Souza, A. O., Cárnio, E. C., Sebollela, A. S., et al. (2020). Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration. Neurotoxicity Research, 38( 4), 871-886. doi:10.1007/s12640-020-00222-z
NLM
Catalão CHR, Santos Junior NN dos, Costa LHA da, Souza AO, Cárnio EC, Sebollela AS, Alberici LC, Rocha MJA da. Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration [Internet]. Neurotoxicity Research. 2020 ; 38( 4): 871-886.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00222-z
Vancouver
Catalão CHR, Santos Junior NN dos, Costa LHA da, Souza AO, Cárnio EC, Sebollela AS, Alberici LC, Rocha MJA da. Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration [Internet]. Neurotoxicity Research. 2020 ; 38( 4): 871-886.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-020-00222-z
Artigo de periodico
Effects of cannabidiol on diabetes outcomes and chronic cerebral hypoperfusion comorbidities in middle-aged rats (2019)
Santiago, Amanda Nunes; Mori, Marco Aurélio; Guimarães, Francisco Silveira; Milani, Humberto; Oliveira, Rúbia Maria Weffort de
Source: Neurotoxicity Research. Unidade: FMRP
Subjects: ISQUEMIA CEREBRAL, CANABINOIDES, DIABETES MELLITUS, NEURÔNIOS, RATOS
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ABNT
SANTIAGO, Amanda Nunes et al. Effects of cannabidiol on diabetes outcomes and chronic cerebral hypoperfusion comorbidities in middle-aged rats. Neurotoxicity Research, v. 35, n. 2, p. 463-474, 2019Tradução . . Disponível em: https://doi.org/10.1007/s12640-018-9972-5. Acesso em: 10 nov. 2024.
APA
Santiago, A. N., Mori, M. A., Guimarães, F. S., Milani, H., & Oliveira, R. M. W. de. (2019). Effects of cannabidiol on diabetes outcomes and chronic cerebral hypoperfusion comorbidities in middle-aged rats. Neurotoxicity Research, 35( 2), 463-474. doi:10.1007/s12640-018-9972-5
NLM
Santiago AN, Mori MA, Guimarães FS, Milani H, Oliveira RMW de. Effects of cannabidiol on diabetes outcomes and chronic cerebral hypoperfusion comorbidities in middle-aged rats [Internet]. Neurotoxicity Research. 2019 ; 35( 2): 463-474.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-018-9972-5
Vancouver
Santiago AN, Mori MA, Guimarães FS, Milani H, Oliveira RMW de. Effects of cannabidiol on diabetes outcomes and chronic cerebral hypoperfusion comorbidities in middle-aged rats [Internet]. Neurotoxicity Research. 2019 ; 35( 2): 463-474.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-018-9972-5
Artigo de periodico
Suppressing nNOS enzyme by small-interfering RNAs protects SH-SY5Y cells and nigral dopaminergic neurons from 6-OHDA injury (2019)
Titze-de-Almeida, Ricardo; Titze-de-Almeida, Simoneide S; Ferreira, Nadia Rubia; Fontanari, Caroline; Faccioli, Lucia Helena ; Del Bel, Elaine Aparecida
Source: Neurotoxicity Research. Unidades: FCFRP, FORP
Subjects: ÓXIDO NÍTRICO, DOENÇA DE PARKINSON, ENZIMAS HIDROLÍTICAS
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ABNT
TITZE-DE-ALMEIDA, Ricardo et al. Suppressing nNOS enzyme by small-interfering RNAs protects SH-SY5Y cells and nigral dopaminergic neurons from 6-OHDA injury. Neurotoxicity Research, v. 36, n. 1, p. 117-131, 2019Tradução . . Disponível em: https://doi.org/10.1007/s12640-019-00043-9. Acesso em: 10 nov. 2024.
APA
Titze-de-Almeida, R., Titze-de-Almeida, S. S., Ferreira, N. R., Fontanari, C., Faccioli, L. H., & Del Bel, E. A. (2019). Suppressing nNOS enzyme by small-interfering RNAs protects SH-SY5Y cells and nigral dopaminergic neurons from 6-OHDA injury. Neurotoxicity Research, 36( 1), 117-131. doi:10.1007/s12640-019-00043-9
NLM
Titze-de-Almeida R, Titze-de-Almeida SS, Ferreira NR, Fontanari C, Faccioli LH, Del Bel EA. Suppressing nNOS enzyme by small-interfering RNAs protects SH-SY5Y cells and nigral dopaminergic neurons from 6-OHDA injury [Internet]. Neurotoxicity Research. 2019 ; 36( 1): 117-131.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-019-00043-9
Vancouver
Titze-de-Almeida R, Titze-de-Almeida SS, Ferreira NR, Fontanari C, Faccioli LH, Del Bel EA. Suppressing nNOS enzyme by small-interfering RNAs protects SH-SY5Y cells and nigral dopaminergic neurons from 6-OHDA injury [Internet]. Neurotoxicity Research. 2019 ; 36( 1): 117-131.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-019-00043-9
Artigo de periodico
The antidiabetic drug liraglutide minimizes the non-cholinergic neurotoxicity of the pesticide mipafox in SH-SY5Y cells (2019)
Fernandes, Lais Silva; Santos, Neife Aparecida Guinaim dos; Emerick, Guilherme Luz ; Santos, Antônio Cardozo dos
Source: Neurotoxicity Research. Unidade: FCFRP
Subjects: LINHAGEM CELULAR, GLICOSE, FÁRMACOS (METABOLISMO E NUTRIÇÃO), INTERLEUCINA 1, PESTICIDAS
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ABNT
FERNANDES, Lais Silva et al. The antidiabetic drug liraglutide minimizes the non-cholinergic neurotoxicity of the pesticide mipafox in SH-SY5Y cells. Neurotoxicity Research, v. 35, n. 1, p. 150-159, 2019Tradução . . Disponível em: https://doi.org/10.1007/s12640-018-9941-z. Acesso em: 10 nov. 2024.
APA
Fernandes, L. S., Santos, N. A. G. dos, Emerick, G. L., & Santos, A. C. dos. (2019). The antidiabetic drug liraglutide minimizes the non-cholinergic neurotoxicity of the pesticide mipafox in SH-SY5Y cells. Neurotoxicity Research, 35( 1), 150-159. doi:10.1007/s12640-018-9941-z
NLM
Fernandes LS, Santos NAG dos, Emerick GL, Santos AC dos. The antidiabetic drug liraglutide minimizes the non-cholinergic neurotoxicity of the pesticide mipafox in SH-SY5Y cells [Internet]. Neurotoxicity Research. 2019 ; 35( 1): 150-159.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-018-9941-z
Vancouver
Fernandes LS, Santos NAG dos, Emerick GL, Santos AC dos. The antidiabetic drug liraglutide minimizes the non-cholinergic neurotoxicity of the pesticide mipafox in SH-SY5Y cells [Internet]. Neurotoxicity Research. 2019 ; 35( 1): 150-159.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-018-9941-z
Artigo de periodico
Caffeic Acid Phenethyl Ester (CAPE) protects PC12 cells against cisplatin-induced neurotoxicity by activating the AMPK/SIRT1, MAPK/Erk, and PI3k/Akt signaling pathways (2019)
Ferreira, Rafaela Scalco; Santos, Neife Aparecida Guinaim dos; Bernardes, Carolina Petri ; Sisti, Flávia Malvestio; Amaral, Lilian; Fontana, Andréia Cristina Karklin; Santos, Antônio Cardozo dos
Source: Neurotoxicity Research. Unidade: FCFRP
Subjects: DIFERENCIAÇÃO CELULAR, SOBREVIVÊNCIA CELULAR, INIBIDORES DE ENZIMAS, NEUROBLASTOMA
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ABNT
FERREIRA, Rafaela Scalco et al. Caffeic Acid Phenethyl Ester (CAPE) protects PC12 cells against cisplatin-induced neurotoxicity by activating the AMPK/SIRT1, MAPK/Erk, and PI3k/Akt signaling pathways. Neurotoxicity Research, v. 36, n. 1, p. 175-192, 2019Tradução . . Disponível em: https://doi.org/10.1007/s12640-019-00042-w. Acesso em: 10 nov. 2024.
APA
Ferreira, R. S., Santos, N. A. G. dos, Bernardes, C. P., Sisti, F. M., Amaral, L., Fontana, A. C. K., & Santos, A. C. dos. (2019). Caffeic Acid Phenethyl Ester (CAPE) protects PC12 cells against cisplatin-induced neurotoxicity by activating the AMPK/SIRT1, MAPK/Erk, and PI3k/Akt signaling pathways. Neurotoxicity Research, 36( 1), 175-192. doi:10.1007/s12640-019-00042-w
NLM
Ferreira RS, Santos NAG dos, Bernardes CP, Sisti FM, Amaral L, Fontana ACK, Santos AC dos. Caffeic Acid Phenethyl Ester (CAPE) protects PC12 cells against cisplatin-induced neurotoxicity by activating the AMPK/SIRT1, MAPK/Erk, and PI3k/Akt signaling pathways [Internet]. Neurotoxicity Research. 2019 ; 36( 1): 175-192.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-019-00042-w
Vancouver
Ferreira RS, Santos NAG dos, Bernardes CP, Sisti FM, Amaral L, Fontana ACK, Santos AC dos. Caffeic Acid Phenethyl Ester (CAPE) protects PC12 cells against cisplatin-induced neurotoxicity by activating the AMPK/SIRT1, MAPK/Erk, and PI3k/Akt signaling pathways [Internet]. Neurotoxicity Research. 2019 ; 36( 1): 175-192.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-019-00042-w
Artigo de periodico
Mild exercise differently affects proteostasis and oxidative stress on motor areas during neurodegeneration: a comparative study of three treadmill running protocols (2019)
Melo, Karla P; Silva, Carolliny Moura da; Almeida, Michael Fernandes de; Chaves, Rodrigo dos Santos; Marcourakis, Tania ; Cardoso, Sandra M; Demasi, Marilene; Netto, Luis Eduardo Soares ; Ferrari, Merari de Fátima Ramires
Source: Neurotoxicity Research. Unidades: FCF, IB
Subjects: EXERCÍCIO FÍSICO, PROTEINASES, ESTRESSE OXIDATIVO
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ABNT
MELO, Karla P et al. Mild exercise differently affects proteostasis and oxidative stress on motor areas during neurodegeneration: a comparative study of three treadmill running protocols. Neurotoxicity Research, v. 35, p. 410-420, 2019Tradução . . Disponível em: https://doi.org/10.1007/s12640-018-9966-3. Acesso em: 10 nov. 2024.
APA
Melo, K. P., Silva, C. M. da, Almeida, M. F. de, Chaves, R. dos S., Marcourakis, T., Cardoso, S. M., et al. (2019). Mild exercise differently affects proteostasis and oxidative stress on motor areas during neurodegeneration: a comparative study of three treadmill running protocols. Neurotoxicity Research, 35, 410-420. doi:10.1007/s12640-018-9966-3
NLM
Melo KP, Silva CM da, Almeida MF de, Chaves R dos S, Marcourakis T, Cardoso SM, Demasi M, Netto LES, Ferrari M de FR. Mild exercise differently affects proteostasis and oxidative stress on motor areas during neurodegeneration: a comparative study of three treadmill running protocols [Internet]. Neurotoxicity Research. 2019 ; 35 410-420.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-018-9966-3
Vancouver
Melo KP, Silva CM da, Almeida MF de, Chaves R dos S, Marcourakis T, Cardoso SM, Demasi M, Netto LES, Ferrari M de FR. Mild exercise differently affects proteostasis and oxidative stress on motor areas during neurodegeneration: a comparative study of three treadmill running protocols [Internet]. Neurotoxicity Research. 2019 ; 35 410-420.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-018-9966-3
Artigo de periodico
Nociceptive response to L-DOPA-induced dyskinesia in hemiparkinsonian rats (2018)
Nascimento, Glauce Crivelaro do ; Santos, Keila Dayane Bariotto dos; Leite-Panissi, Christie Ramos Andrade ; Del Bel, Elaine Aparecida ; Bortolanza, Mariza
Source: Neurotoxicity Research. Unidades: FFCLRP, FORP, FMRP
Subjects: DISCINESIAS, RATOS, DOENÇA DE PARKINSON, TOXINAS
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ABNT
NASCIMENTO, Glauce Crivelaro do et al. Nociceptive response to L-DOPA-induced dyskinesia in hemiparkinsonian rats. Neurotoxicity Research, v. 34, n. 4, p. 799-807, 2018Tradução . . Disponível em: https://doi.org/10.1007/s12640-018-9896-0. Acesso em: 10 nov. 2024.
APA
Nascimento, G. C. do, Santos, K. D. B. dos, Leite-Panissi, C. R. A., Del Bel, E. A., & Bortolanza, M. (2018). Nociceptive response to L-DOPA-induced dyskinesia in hemiparkinsonian rats. Neurotoxicity Research, 34( 4), 799-807. doi:10.1007/s12640-018-9896-0
NLM
Nascimento GC do, Santos KDB dos, Leite-Panissi CRA, Del Bel EA, Bortolanza M. Nociceptive response to L-DOPA-induced dyskinesia in hemiparkinsonian rats [Internet]. Neurotoxicity Research. 2018 ; 34( 4): 799-807.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-018-9896-0
Vancouver
Nascimento GC do, Santos KDB dos, Leite-Panissi CRA, Del Bel EA, Bortolanza M. Nociceptive response to L-DOPA-induced dyskinesia in hemiparkinsonian rats [Internet]. Neurotoxicity Research. 2018 ; 34( 4): 799-807.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-018-9896-0
Artigo de periodico
Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling (2018)
Vrechi, Talita Aparecida de Moraes; Crunfli, Fernanda; Costa, Andressa Pereira; Torrão, Andréa da Silva
Source: Neurotoxicity Research. Unidade: ICB
Subjects: CANNABIS, RETÍCULO ENDOPLASMÁTICO, IMMUNOBLOTTING, DEGENERAÇÃO NEURAL, INFLAMAÇÃO, MORTE CEREBRAL, NEURÔNIOS, SISTEMA NERVOSO CENTRAL
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ABNT
VRECHI, Talita Aparecida de Moraes et al. Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling. Neurotoxicity Research, v. 33, n. 4, p. 846-855, 2018Tradução . . Disponível em: https://doi.org/10.1007/s12640-017-9839-1. Acesso em: 10 nov. 2024.
APA
Vrechi, T. A. de M., Crunfli, F., Costa, A. P., & Torrão, A. da S. (2018). Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling. Neurotoxicity Research, 33( 4), 846-855. doi:10.1007/s12640-017-9839-1
NLM
Vrechi TA de M, Crunfli F, Costa AP, Torrão A da S. Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling [Internet]. Neurotoxicity Research. 2018 ; 33( 4): 846-855.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-017-9839-1
Vancouver
Vrechi TA de M, Crunfli F, Costa AP, Torrão A da S. Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling [Internet]. Neurotoxicity Research. 2018 ; 33( 4): 846-855.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12640-017-9839-1

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