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Artigo de periodico
Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis (2021)
Saraiva, André Lopes; Peres, Raphael Sanches; Veras, Flávio Protasio; Talbot, Jhimmy; Lima, Kalil Alves de; Luiz, João Paulo Mesquita; Cunha, Thiago Mattar; Louzada-Junior, Paulo; Cunha, Fernando Queiróz; Alves-Filho, José Carlos
Source: International Immunopharmacology. Unidades: FCFRP, FMRP
Subjects: ARTRITE REUMATOIDE, PEPTÍDEOS, ARTICULAÇÕES, IMUNOLOGIA MÉDICA
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SARAIVA, André Lopes et al. Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis. International Immunopharmacology, v. 101, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2021.108363. Acesso em: 05 nov. 2024.
APA
Saraiva, A. L., Peres, R. S., Veras, F. P., Talbot, J., Lima, K. A. de, Luiz, J. P. M., et al. (2021). Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis. International Immunopharmacology, 101. doi:10.1016/j.intimp.2021.108363
NLM
Saraiva AL, Peres RS, Veras FP, Talbot J, Lima KA de, Luiz JPM, Cunha TM, Louzada-Junior P, Cunha FQ, Alves-Filho JC. Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis [Internet]. International Immunopharmacology. 2021 ; 101[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2021.108363
Vancouver
Saraiva AL, Peres RS, Veras FP, Talbot J, Lima KA de, Luiz JPM, Cunha TM, Louzada-Junior P, Cunha FQ, Alves-Filho JC. Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis [Internet]. International Immunopharmacology. 2021 ; 101[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2021.108363
Artigo de periodico
The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats (2021)
Abdalla, Henrique Ballassini; Napimoga, Marcelo Henrique; Maganin, Alexandre Gomes de Macedo; Lopes, Alexandre Hashimoto Pereira; Cunha, Thiago Mattar; Gill, Harvinder Singh; Napimoga, Juliana Trindade Clemente
Source: International Immunopharmacology. Unidade: FMRP
Subjects: ADENOSINA, ANALGÉSICOS, ANTI-INFLAMATÓRIOS, ARTRALGIA, MACRÓFAGOS, NOCICEPTIVIDADE
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ABDALLA, Henrique Ballassini et al. The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats. International Immunopharmacology, v. 97, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2021.107680. Acesso em: 05 nov. 2024.
APA
Abdalla, H. B., Napimoga, M. H., Maganin, A. G. de M., Lopes, A. H. P., Cunha, T. M., Gill, H. S., & Napimoga, J. T. C. (2021). The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats. International Immunopharmacology, 97. doi:10.1016/j.intimp.2021.107680
NLM
Abdalla HB, Napimoga MH, Maganin AG de M, Lopes AHP, Cunha TM, Gill HS, Napimoga JTC. The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats [Internet]. International Immunopharmacology. 2021 ; 97[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2021.107680
Vancouver
Abdalla HB, Napimoga MH, Maganin AG de M, Lopes AHP, Cunha TM, Gill HS, Napimoga JTC. The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats [Internet]. International Immunopharmacology. 2021 ; 97[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2021.107680
Artigo de periodico
Pitavastatin ameliorates autoimmune neuroinflammation by regulating the Treg/Th17 cell balance through inhibition of mevalonate metabolism (2021)
Prado, Douglas Silva ; Damasceno, Luis Eduardo Alves; Sonego, Andreza Buzolin; Rosa, Marcos Henrique; Martins, Timna Varela ; Fonseca, M. D. M.; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias
Source: International Immunopharmacology. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS, DIFERENCIAÇÃO CELULAR, CITOCINAS, GENÉTICA, MEDULA ESPINHAL, LINFÓCITOS T
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PRADO, Douglas Silva et al. Pitavastatin ameliorates autoimmune neuroinflammation by regulating the Treg/Th17 cell balance through inhibition of mevalonate metabolism. International Immunopharmacology, v. 91, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2020.107278. Acesso em: 05 nov. 2024.
APA
Prado, D. S., Damasceno, L. E. A., Sonego, A. B., Rosa, M. H., Martins, T. V., Fonseca, M. D. M., et al. (2021). Pitavastatin ameliorates autoimmune neuroinflammation by regulating the Treg/Th17 cell balance through inhibition of mevalonate metabolism. International Immunopharmacology, 91. doi:10.1016/j.intimp.2020.107278
NLM
Prado DS, Damasceno LEA, Sonego AB, Rosa MH, Martins TV, Fonseca MDM, Cunha TM, Cunha F de Q, Alves Filho JCF. Pitavastatin ameliorates autoimmune neuroinflammation by regulating the Treg/Th17 cell balance through inhibition of mevalonate metabolism [Internet]. International Immunopharmacology. 2021 ; 91[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.107278
Vancouver
Prado DS, Damasceno LEA, Sonego AB, Rosa MH, Martins TV, Fonseca MDM, Cunha TM, Cunha F de Q, Alves Filho JCF. Pitavastatin ameliorates autoimmune neuroinflammation by regulating the Treg/Th17 cell balance through inhibition of mevalonate metabolism [Internet]. International Immunopharmacology. 2021 ; 91[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.107278
Artigo de periodico
Bothrops moojeni venom and BmooLAAO-I downmodulate CXCL8/IL-8 and CCL2/MCP-1 production and oxidative burst response, and upregulate CD11b expression in human neutrophils (2020)
Crott, Luciana Simon Pereira; Casare-Ogasawara, Tânia Mara; Ambrosio, Luciana ; Chaim, Luiz Fernando Princi; Morais, Fabiana Rossetto de; Cintra, Adélia Cristina Oliveira; Canicoba, Nathália Cristina; Tucci, Luiz Fernando Fortunato; Torqueti, Maria Regina ; Sampaio, Suely Vilela ; Marzocchi-Machado, Cleni Mara ; Castro, Fabíola Attié de
Source: International Immunopharmacology. Unidade: FCFRP
Subjects: VIPERIDAE, SERPENTES, ANTÍGENOS, SOBREVIVÊNCIA CELULAR, VENENOS, CITOCINAS, FARMACOLOGIA, AMINOÁCIDOS, NEUTRÓFILOS
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CROTT, Luciana Simon Pereira et al. Bothrops moojeni venom and BmooLAAO-I downmodulate CXCL8/IL-8 and CCL2/MCP-1 production and oxidative burst response, and upregulate CD11b expression in human neutrophils. International Immunopharmacology, v. 80, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.106154. Acesso em: 05 nov. 2024.
APA
Crott, L. S. P., Casare-Ogasawara, T. M., Ambrosio, L., Chaim, L. F. P., Morais, F. R. de, Cintra, A. C. O., et al. (2020). Bothrops moojeni venom and BmooLAAO-I downmodulate CXCL8/IL-8 and CCL2/MCP-1 production and oxidative burst response, and upregulate CD11b expression in human neutrophils. International Immunopharmacology, 80. doi:10.1016/j.intimp.2019.106154
NLM
Crott LSP, Casare-Ogasawara TM, Ambrosio L, Chaim LFP, Morais FR de, Cintra ACO, Canicoba NC, Tucci LFF, Torqueti MR, Sampaio SV, Marzocchi-Machado CM, Castro FA de. Bothrops moojeni venom and BmooLAAO-I downmodulate CXCL8/IL-8 and CCL2/MCP-1 production and oxidative burst response, and upregulate CD11b expression in human neutrophils [Internet]. International Immunopharmacology. 2020 ; 80[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.106154
Vancouver
Crott LSP, Casare-Ogasawara TM, Ambrosio L, Chaim LFP, Morais FR de, Cintra ACO, Canicoba NC, Tucci LFF, Torqueti MR, Sampaio SV, Marzocchi-Machado CM, Castro FA de. Bothrops moojeni venom and BmooLAAO-I downmodulate CXCL8/IL-8 and CCL2/MCP-1 production and oxidative burst response, and upregulate CD11b expression in human neutrophils [Internet]. International Immunopharmacology. 2020 ; 80[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.106154
Artigo de periodico
Interleukin-2 as immunotherapeutic in the autoimmune diseases (2020)
Valencia, Alexy Orozco; Knirsch, Marcos Camargo; Ferro, Emer Suavinho ; Stephano, Marco Antônio
Source: International Immunopharmacology. Unidades: ICB, FCF
Subjects: DOENÇAS AUTOIMUNES, INTERLEUCINA 2
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VALENCIA, Alexy Orozco et al. Interleukin-2 as immunotherapeutic in the autoimmune diseases. International Immunopharmacology, v. 81, p. 1-6 art. 106296, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2020.106296. Acesso em: 05 nov. 2024.
APA
Valencia, A. O., Knirsch, M. C., Ferro, E. S., & Stephano, M. A. (2020). Interleukin-2 as immunotherapeutic in the autoimmune diseases. International Immunopharmacology, 81, 1-6 art. 106296. doi:10.1016/j.intimp.2020.106296
NLM
Valencia AO, Knirsch MC, Ferro ES, Stephano MA. Interleukin-2 as immunotherapeutic in the autoimmune diseases [Internet]. International Immunopharmacology. 2020 ; 81 1-6 art. 106296.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.106296
Vancouver
Valencia AO, Knirsch MC, Ferro ES, Stephano MA. Interleukin-2 as immunotherapeutic in the autoimmune diseases [Internet]. International Immunopharmacology. 2020 ; 81 1-6 art. 106296.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.106296
Artigo de periodico
Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1 (2020)
Abdalla, Henrique Ballassini; Napimoga, Marcelo Henrique; Lopes, Alexandre Hashimoto Pereira; Maganin, Alexandre Gomes de Macedo; Cunha, Thiago Mattar; Van Dyke, Thomas E.; Napimoga, Juliana T. Clemente
Source: International Immunopharmacology. Unidade: FMRP
Subjects: CÁLCIO, CALMODULINA, IMUNOLOGIA, CARRAGENINA, CITOCINAS, LIPOPOLISSACARÍDEOS, NEUTRÓFILOS, ÓXIDO NÍTRICO, PROSTAGLANDINAS, ARTICULAÇÃO TEMPOROMANDIBULAR, MACRÓFAGOS
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ABDALLA, Henrique Ballassini et al. Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1. International Immunopharmacology, v. 84, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2020.106565. Acesso em: 05 nov. 2024.
APA
Abdalla, H. B., Napimoga, M. H., Lopes, A. H. P., Maganin, A. G. de M., Cunha, T. M., Van Dyke, T. E., & Napimoga, J. T. C. (2020). Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1. International Immunopharmacology, 84. doi:10.1016/j.intimp.2020.106565
NLM
Abdalla HB, Napimoga MH, Lopes AHP, Maganin AG de M, Cunha TM, Van Dyke TE, Napimoga JTC. Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1 [Internet]. International Immunopharmacology. 2020 ; 84[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.106565
Vancouver
Abdalla HB, Napimoga MH, Lopes AHP, Maganin AG de M, Cunha TM, Van Dyke TE, Napimoga JTC. Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1 [Internet]. International Immunopharmacology. 2020 ; 84[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.106565
Artigo de periodico
Immunosenescence in chronic HIV infected patients impairs essential functions of their natural killer cells (2020)
Soares, Luana Silva; Espíndola, Milena Sobral; Zambuzi, Fabiana Albani; Lima, Leonardo Judson Galvão de; Cacemiro, Maira da Costa ; Soares, Murilo Racy ; Santana, Bárbara Amélia Aparecida; Calado, Rodrigo Tocantins ; Bollela, Valdes Roberto; Frantz, Fabiani Gai
Source: International Immunopharmacology. Unidades: FMRP, FCFRP
Subjects: DOENÇA AGUDA, DOENÇA CRÔNICA, CITOCINAS, METILAÇÃO DE DNA, INFECÇÕES POR HIV, IMUNOLOGIA, ESPÉCIES REATIVAS DE OXIGÊNIO
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SOARES, Luana Silva et al. Immunosenescence in chronic HIV infected patients impairs essential functions of their natural killer cells. International Immunopharmacology, v. 84, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2020.106568. Acesso em: 05 nov. 2024.
APA
Soares, L. S., Espíndola, M. S., Zambuzi, F. A., Lima, L. J. G. de, Cacemiro, M. da C., Soares, M. R., et al. (2020). Immunosenescence in chronic HIV infected patients impairs essential functions of their natural killer cells. International Immunopharmacology, 84. doi:10.1016/j.intimp.2020.106568
NLM
Soares LS, Espíndola MS, Zambuzi FA, Lima LJG de, Cacemiro M da C, Soares MR, Santana BAA, Calado RT, Bollela VR, Frantz FG. Immunosenescence in chronic HIV infected patients impairs essential functions of their natural killer cells [Internet]. International Immunopharmacology. 2020 ; 84[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.106568
Vancouver
Soares LS, Espíndola MS, Zambuzi FA, Lima LJG de, Cacemiro M da C, Soares MR, Santana BAA, Calado RT, Bollela VR, Frantz FG. Immunosenescence in chronic HIV infected patients impairs essential functions of their natural killer cells [Internet]. International Immunopharmacology. 2020 ; 84[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.106568
Artigo de periodico
Copaiba oil suppresses inflammation in asthmatic lungs of BALB/c mice induced with ovalbumin (2020)
Caputo, Ludmila S.; Campos, Maria Inês C.; Dias, Herbert Júnior; Crotti, Antonio Eduardo Miller ; Fajardo, Júlia B.; Vanelli, Chislene P.; Presto, Álvaro C. D.; Alves, Maria S.; Aarestrup, Fernando M.; Paula, Ana Claudia C.; Silva Filho, Ademar Alves da; Aarestrup, Beatriz J. V.; Pereira Júnior, Olavo dos Santos; Corrêa, José Otávio do A.
Source: International Immunopharmacology. Unidade: FFCLRP
Subjects: COPAÍBA, ASMA, PRODUTOS NATURAIS, ADJUVANTES IMUNOLÓGICOS
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CAPUTO, Ludmila S. et al. Copaiba oil suppresses inflammation in asthmatic lungs of BALB/c mice induced with ovalbumin. International Immunopharmacology, v. 80, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.106177. Acesso em: 05 nov. 2024.
APA
Caputo, L. S., Campos, M. I. C., Dias, H. J., Crotti, A. E. M., Fajardo, J. B., Vanelli, C. P., et al. (2020). Copaiba oil suppresses inflammation in asthmatic lungs of BALB/c mice induced with ovalbumin. International Immunopharmacology, 80. doi:10.1016/j.intimp.2019.106177
NLM
Caputo LS, Campos MIC, Dias HJ, Crotti AEM, Fajardo JB, Vanelli CP, Presto ÁCD, Alves MS, Aarestrup FM, Paula ACC, Silva Filho AA da, Aarestrup BJV, Pereira Júnior O dos S, Corrêa JO do A. Copaiba oil suppresses inflammation in asthmatic lungs of BALB/c mice induced with ovalbumin [Internet]. International Immunopharmacology. 2020 ; 80[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.106177
Vancouver
Caputo LS, Campos MIC, Dias HJ, Crotti AEM, Fajardo JB, Vanelli CP, Presto ÁCD, Alves MS, Aarestrup FM, Paula ACC, Silva Filho AA da, Aarestrup BJV, Pereira Júnior O dos S, Corrêa JO do A. Copaiba oil suppresses inflammation in asthmatic lungs of BALB/c mice induced with ovalbumin [Internet]. International Immunopharmacology. 2020 ; 80[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.106177
Artigo de periodico
Vestitol drives LPS-activated macrophages into M2 phenotype through modulation of NF-κB pathway (2020)
Bueno-Silva, Bruno ; Rosalen, Pedro Luiz ; Alencar, Severino Matias ; Mayer, Marcia Pinto Alves
Source: International Immunopharmacology. Unidades: ESALQ, ICB
Subjects: ANTI-INFLAMATÓRIOS, CITOCINAS, MICROBIOLOGIA, PRÓPOLIS, MACRÓFAGOS, FENÓTIPOS, INFLAMAÇÃO, PRODUTOS NATURAIS, NEUTRÓFILOS, EXPRESSÃO GÊNICA, ÓXIDO NÍTRICO
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ABNT
BUENO-SILVA, Bruno et al. Vestitol drives LPS-activated macrophages into M2 phenotype through modulation of NF-κB pathway. International Immunopharmacology, v. 82, p. 1-7, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2020.106329. Acesso em: 05 nov. 2024.
APA
Bueno-Silva, B., Rosalen, P. L., Alencar, S. M., & Mayer, M. P. A. (2020). Vestitol drives LPS-activated macrophages into M2 phenotype through modulation of NF-κB pathway. International Immunopharmacology, 82, 1-7. doi:10.1016/j.intimp.2020.106329
NLM
Bueno-Silva B, Rosalen PL, Alencar SM, Mayer MPA. Vestitol drives LPS-activated macrophages into M2 phenotype through modulation of NF-κB pathway [Internet]. International Immunopharmacology. 2020 ; 82 1-7.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.106329
Vancouver
Bueno-Silva B, Rosalen PL, Alencar SM, Mayer MPA. Vestitol drives LPS-activated macrophages into M2 phenotype through modulation of NF-κB pathway [Internet]. International Immunopharmacology. 2020 ; 82 1-7.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2020.106329
Artigo de periodico
BAY 41-2272 inhibits human neutrophil functions (2019)
Rosa, Paola Vendramini Ferreira; Carvalho, Marina Uchoa Wall Barbosa de; Pereira, Paulo Vitor Soeiro ; Harumi, Renata Cruz; Albuquerque, Rafael Sales de ; Antunes, Edson ; Condino Neto, Antonio
Source: International Immunopharmacology. Unidade: ICB
Subjects: NEUTRÓFILOS, IMUNOLOGIA, ADJUVANTES IMUNOLÓGICOS, MONÓCITOS, CITOCINAS
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ABNT
ROSA, Paola Vendramini Ferreira et al. BAY 41-2272 inhibits human neutrophil functions. International Immunopharmacology, v. 75, n. art. 105767, p. 7 , 2019Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.105767. Acesso em: 05 nov. 2024.
APA
Rosa, P. V. F., Carvalho, M. U. W. B. de, Pereira, P. V. S., Harumi, R. C., Albuquerque, R. S. de, Antunes, E., & Condino Neto, A. (2019). BAY 41-2272 inhibits human neutrophil functions. International Immunopharmacology, 75( art. 105767), 7 . doi:10.1016/j.intimp.2019.105767
NLM
Rosa PVF, Carvalho MUWB de, Pereira PVS, Harumi RC, Albuquerque RS de, Antunes E, Condino Neto A. BAY 41-2272 inhibits human neutrophil functions [Internet]. International Immunopharmacology. 2019 ; 75( art. 105767): 7 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.105767
Vancouver
Rosa PVF, Carvalho MUWB de, Pereira PVS, Harumi RC, Albuquerque RS de, Antunes E, Condino Neto A. BAY 41-2272 inhibits human neutrophil functions [Internet]. International Immunopharmacology. 2019 ; 75( art. 105767): 7 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.105767
Artigo de periodico
BAY 41-2272 inhibits human T lymphocyte functions (2019)
Carvalho, Marina Uchôa Wall Barbosa de; Rosa, Paola Vendramini Ferreira; Kubo, Christina Arslanian; Pereira, Paulo Vitor Soeiro ; Albuquerque, Rafael Sales de ; Antunes, Edson ; Condino Neto, Antonio
Source: International Immunopharmacology. Unidade: ICB
Subjects: IMUNOLOGIA, TESTE DE TUKEY, LINFÓCITOS T, DOENÇAS AUTOIMUNES, FÁRMACOS IMUNOSSUPRESSORES, INTERFERONS, IMUNOLOGIA
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ABNT
CARVALHO, Marina Uchôa Wall Barbosa de et al. BAY 41-2272 inhibits human T lymphocyte functions. International Immunopharmacology, v. 77, p. 7 , 2019Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.105976. Acesso em: 05 nov. 2024.
APA
Carvalho, M. U. W. B. de, Rosa, P. V. F., Kubo, C. A., Pereira, P. V. S., Albuquerque, R. S. de, Antunes, E., & Condino Neto, A. (2019). BAY 41-2272 inhibits human T lymphocyte functions. International Immunopharmacology, 77, 7 . doi:10.1016/j.intimp.2019.105976
NLM
Carvalho MUWB de, Rosa PVF, Kubo CA, Pereira PVS, Albuquerque RS de, Antunes E, Condino Neto A. BAY 41-2272 inhibits human T lymphocyte functions [Internet]. International Immunopharmacology. 2019 ; 77 7 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.105976
Vancouver
Carvalho MUWB de, Rosa PVF, Kubo CA, Pereira PVS, Albuquerque RS de, Antunes E, Condino Neto A. BAY 41-2272 inhibits human T lymphocyte functions [Internet]. International Immunopharmacology. 2019 ; 77 7 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.105976
Artigo de periodico
Hydrogen sulfide inhibits apoptosis and protects the bronchial epithelium in an allergic inflammation mice model (2019)
Mendes, Jackeline A. ; Ribeiro, Matheus C.; Reis, Filho Gustavo J. M. V.; Rocha, Thalita ; Muscará, Marcelo Nicolás (Orientador) ; Costa, Soraia Katia Pereira ; Ferreira, Heloisa H. A.
Source: International Immunopharmacology. Unidade: ICB
Subjects: FARMACOLOGIA, PNEUMOPATIAS, ASMA, EPITÉLIO, ALÉRGENOS, APOPTOSE, CITOCININAS, INFLAMAÇÃO, CAMUNDONGOS, EUTANÁSIA
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ABNT
MENDES, Jackeline A. et al. Hydrogen sulfide inhibits apoptosis and protects the bronchial epithelium in an allergic inflammation mice model. International Immunopharmacology, v. 73, p. 435-441, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.05.041. Acesso em: 05 nov. 2024.
APA
Mendes, J. A., Ribeiro, M. C., Reis, F. G. J. M. V., Rocha, T., Costa, S. K. P., & Ferreira, H. H. A. (2019). Hydrogen sulfide inhibits apoptosis and protects the bronchial epithelium in an allergic inflammation mice model. International Immunopharmacology, 73, 435-441. doi:10.1016/j.intimp.2019.05.041
NLM
Mendes JA, Ribeiro MC, Reis FGJMV, Rocha T, Costa SKP, Ferreira HHA. Hydrogen sulfide inhibits apoptosis and protects the bronchial epithelium in an allergic inflammation mice model [Internet]. International Immunopharmacology. 2019 ;73 435-441.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.05.041
Vancouver
Mendes JA, Ribeiro MC, Reis FGJMV, Rocha T, Costa SKP, Ferreira HHA. Hydrogen sulfide inhibits apoptosis and protects the bronchial epithelium in an allergic inflammation mice model [Internet]. International Immunopharmacology. 2019 ;73 435-441.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.05.041
Artigo de periodico
Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice (2019)
Brandão, Wesley Nogueira ; Andersen, Monica Levy ; Palermo-Neto, João ; Peron, Jean Pierre Schatzmann ; Zager, Adriano
Source: International Immunopharmacology. Unidades: FMVZ, ICB
Subjects: ENCEFALOMIELITE ANIMAL, PSICOFARMACOLOGIA, FÁRMACOS PSICOTRÓPICOS, INFLAMAÇÃO, RECEPTORES DE DOPAMINA, IMUNIDADE, ESCLEROSE MÚLTIPLA
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ABNT
BRANDÃO, Wesley Nogueira et al. Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice. International Immunopharmacology, v. 75, p. 1-7, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.105809. Acesso em: 05 nov. 2024.
APA
Brandão, W. N., Andersen, M. L., Palermo-Neto, J., Peron, J. P. S., & Zager, A. (2019). Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice. International Immunopharmacology, 75, 1-7. doi:10.1016/j.intimp.2019.105809
NLM
Brandão WN, Andersen ML, Palermo-Neto J, Peron JPS, Zager A. Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice [Internet]. International Immunopharmacology. 2019 ; 75 1-7.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.105809
Vancouver
Brandão WN, Andersen ML, Palermo-Neto J, Peron JPS, Zager A. Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice [Internet]. International Immunopharmacology. 2019 ; 75 1-7.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.105809
Artigo de periodico
Estradiol replacement therapy regulates innate immune response in ovariectomized arthritic mice (2019)
Schneider, Ayda Henriques; Kanashiro, Alexandre; Dutra, Sabrina Graziani Veloso; Souza, Raquel do Nascimento de; Veras, Flávio Protásio; Cunha, Fernando de Queiroz ; Ulloa, Luis; Mecawi, Andre Souza; Reis, Luis Carlos; Malvar, David do Carmo
Source: International Immunopharmacology. Unidade: FMRP
Subjects: ESTRADIOL, ARTRITE, INFLAMAÇÃO, NEUTRÓFILOS, ENDOCRINOLOGIA, ESTRÓGENOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SCHNEIDER, Ayda Henriques et al. Estradiol replacement therapy regulates innate immune response in ovariectomized arthritic mice. International Immunopharmacology, v. 72, p. 504-510, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.04.048. Acesso em: 05 nov. 2024.
APA
Schneider, A. H., Kanashiro, A., Dutra, S. G. V., Souza, R. do N. de, Veras, F. P., Cunha, F. de Q., et al. (2019). Estradiol replacement therapy regulates innate immune response in ovariectomized arthritic mice. International Immunopharmacology, 72, 504-510. doi:10.1016/j.intimp.2019.04.048
NLM
Schneider AH, Kanashiro A, Dutra SGV, Souza R do N de, Veras FP, Cunha F de Q, Ulloa L, Mecawi AS, Reis LC, Malvar D do C. Estradiol replacement therapy regulates innate immune response in ovariectomized arthritic mice [Internet]. International Immunopharmacology. 2019 ; 72 504-510.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.04.048
Vancouver
Schneider AH, Kanashiro A, Dutra SGV, Souza R do N de, Veras FP, Cunha F de Q, Ulloa L, Mecawi AS, Reis LC, Malvar D do C. Estradiol replacement therapy regulates innate immune response in ovariectomized arthritic mice [Internet]. International Immunopharmacology. 2019 ; 72 504-510.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2019.04.048
Artigo de periodico
The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression (2018)
Amano, Mariane Tami ; Castoldi, Angela ; Andrade-Oliveira, Vinicius ; Latância, Marcela Teatin ; Terra, Fernanda Fernandes ; Correa-Costa, Matheus ; Breda, Cristiane Naffah Souza ; Felizardo, Raphael José Ferreira ; Pereira, Welbert Oliveira de ; Silva, Marina Burgos da ; Miyagi, Mariana Yasue Saito ; Aguiar, Cristhiane Favero ; Hiyane, Meire Ioshie ; Silva, João Santana da ; Moura, Ivan Cruz ; Câmara, Niels Olsen Saraiva
Source: International Immunopharmacology. Unidades: ICB, FMRP, FCF, BIOTECNOLOGIA
Subjects: INSUFICIÊNCIA RENAL AGUDA, NEFROPATIAS, MACRÓFAGOS, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMANO, Mariane Tami et al. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, v. 64, p. 151-161, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.08.020. Acesso em: 05 nov. 2024.
APA
Amano, M. T., Castoldi, A., Andrade-Oliveira, V., Latância, M. T., Terra, F. F., Correa-Costa, M., et al. (2018). The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, 64, 151-161. doi:10.1016/j.intimp.2018.08.020
NLM
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Vancouver
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Artigo de periodico
Repeated Domperidone treatment modulates pulmonary cytokines in LPS-induced acute lung injury in mice (2018)
Barreto, Talita Rossetto; Souza, Carolina Costola de; Margatho, Rafael Oliveira; Queiroz-Hazarbassanov, Nicolle; Rodrigues, Sandra Campos; Felicio, Luciano Freitas ; Palermo-Neto, João; Zager, Adriano
Source: International Immunopharmacology. Unidade: FMVZ
Subjects: FÁRMACOS GASTRINTESTINAIS, ADJUVANTES IMUNOLÓGICOS, CITOCINAS, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BARRETO, Talita Rossetto et al. Repeated Domperidone treatment modulates pulmonary cytokines in LPS-induced acute lung injury in mice. International Immunopharmacology, v. 56, p. 43-50, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.01.009. Acesso em: 05 nov. 2024.
APA
Barreto, T. R., Souza, C. C. de, Margatho, R. O., Queiroz-Hazarbassanov, N., Rodrigues, S. C., Felicio, L. F., et al. (2018). Repeated Domperidone treatment modulates pulmonary cytokines in LPS-induced acute lung injury in mice. International Immunopharmacology, 56, 43-50. doi:10.1016/j.intimp.2018.01.009
NLM
Barreto TR, Souza CC de, Margatho RO, Queiroz-Hazarbassanov N, Rodrigues SC, Felicio LF, Palermo-Neto J, Zager A. Repeated Domperidone treatment modulates pulmonary cytokines in LPS-induced acute lung injury in mice [Internet]. International Immunopharmacology. 2018 ; 56 43-50.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2018.01.009
Vancouver
Barreto TR, Souza CC de, Margatho RO, Queiroz-Hazarbassanov N, Rodrigues SC, Felicio LF, Palermo-Neto J, Zager A. Repeated Domperidone treatment modulates pulmonary cytokines in LPS-induced acute lung injury in mice [Internet]. International Immunopharmacology. 2018 ; 56 43-50.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2018.01.009
Artigo de periodico
Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide (2018)
Dornelas-Filho, Amílcar Figueiredo; Pereira, Venúcia Bruna Magalhães; Wong, Deysi Viviana T.; Nobre, Lívia Maria Soares; Melo, Anielle Torres; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner Souza; Nour, Mariana Lima; Araújo, Lis Caetano Nobrega Costa; Silva, Renan Oliveira; Pinto, Francisco Maxwell Martins; Bingana, Rudy Diavila; Souza, Marcellus Henrique Loiola Ponte de; Alencar, Nylane Maria Nunes; Silva, Paulo Goberlânio B.; Alves, Ana Paula Negreiros Nunes; Almeida, Paulo Roberto Carvalho; Cunha, Fernando de Queiroz; Lima Júnior, Roberto César P.
Source: International Immunopharmacology. Unidade: FMRP
Subjects: ANTINEOPLÁSICOS, CISTITE, HEMORRAGIA, NEUTRÓFILOS, POLISSACARÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DORNELAS-FILHO, Amílcar Figueiredo et al. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide. International Immunopharmacology, v. 62, p. 96-108, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.06.031. Acesso em: 05 nov. 2024.
APA
Dornelas-Filho, A. F., Pereira, V. B. M., Wong, D. V. T., Nobre, L. M. S., Melo, A. T., Silva, C. M. de S., et al. (2018). Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide. International Immunopharmacology, 62, 96-108. doi:10.1016/j.intimp.2018.06.031
NLM
Dornelas-Filho AF, Pereira VBM, Wong DVT, Nobre LMS, Melo AT, Silva CM de S, Wanderley CWS, Nour ML, Araújo LCNC, Silva RO, Pinto FMM, Bingana RD, Souza MHLP de, Alencar NMN, Silva PGB, Alves APNN, Almeida PRC, Cunha F de Q, Lima Júnior RCP. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide [Internet]. International Immunopharmacology. 2018 ; 62 96-108.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2018.06.031
Vancouver
Dornelas-Filho AF, Pereira VBM, Wong DVT, Nobre LMS, Melo AT, Silva CM de S, Wanderley CWS, Nour ML, Araújo LCNC, Silva RO, Pinto FMM, Bingana RD, Souza MHLP de, Alencar NMN, Silva PGB, Alves APNN, Almeida PRC, Cunha F de Q, Lima Júnior RCP. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide [Internet]. International Immunopharmacology. 2018 ; 62 96-108.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2018.06.031
Artigo de periodico
Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms (2016)
Mendes, Saulo J. F.; Sousa, Fernanda I. A. F.; Pereira, Domingos M. S.; Ferro, Thiago A. F.; Pereira, Ione C. P.; Silva, Bruna L. R.; Pinheiro, Aruanã J. M. C. R.; Mouchrek, Adriana Q. S; Monteiro-Neto, Valério; Costa, Soraia Katia Pereira ; Nascimento, José L. M.; Grisottoa, Marcos A. G.; Costa, Robson da; Fernandes, Elizabeth S.
Source: International Immunopharmacology. Unidade: ICB
Subjects: FARMACOLOGIA, ÓLEOS ESSENCIAIS, CANELA
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ABNT
MENDES, Saulo J. F. et al. Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms. International Immunopharmacology, v. 34, p. 60-70, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2016.02.012. Acesso em: 05 nov. 2024.
APA
Mendes, S. J. F., Sousa, F. I. A. F., Pereira, D. M. S., Ferro, T. A. F., Pereira, I. C. P., Silva, B. L. R., et al. (2016). Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms. International Immunopharmacology, 34, 60-70. doi:10.1016/j.intimp.2016.02.012
NLM
Mendes SJF, Sousa FIAF, Pereira DMS, Ferro TAF, Pereira ICP, Silva BLR, Pinheiro AJMCR, Mouchrek AQS, Monteiro-Neto V, Costa SKP, Nascimento JLM, Grisottoa MAG, Costa R da, Fernandes ES. Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms [Internet]. International Immunopharmacology. 2016 ;34 60-70.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2016.02.012
Vancouver
Mendes SJF, Sousa FIAF, Pereira DMS, Ferro TAF, Pereira ICP, Silva BLR, Pinheiro AJMCR, Mouchrek AQS, Monteiro-Neto V, Costa SKP, Nascimento JLM, Grisottoa MAG, Costa R da, Fernandes ES. Cinnamaldehyde modulates LPS-induced systemic inflammatory response syndrome through TRPA1-dependent and independent mechanisms [Internet]. International Immunopharmacology. 2016 ;34 60-70.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2016.02.012
Artigo de periodico
Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation (2016)
Campos, Daiana; Ravagnani, Felipe G.; Gurgueira, Sonia A.; Vercesi, Anibal E.; Teixeira, Simone A.; Costa, Soraia Katia Pereira ; Muscará, Marcelo Nicolás ; Ferreira, Heloisa H. A.
Source: International Immunopharmacology. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAMPOS, Daiana et al. Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation. International Immunopharmacology, v. 39, p. 57-62, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2016.07.009. Acesso em: 05 nov. 2024.
APA
Campos, D., Ravagnani, F. G., Gurgueira, S. A., Vercesi, A. E., Teixeira, S. A., Costa, S. K. P., et al. (2016). Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation. International Immunopharmacology, 39, 57-62. doi:10.1016/j.intimp.2016.07.009
NLM
Campos D, Ravagnani FG, Gurgueira SA, Vercesi AE, Teixeira SA, Costa SKP, Muscará MN, Ferreira HHA. Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation [Internet]. International Immunopharmacology. 2016 ; 39 57-62.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2016.07.009
Vancouver
Campos D, Ravagnani FG, Gurgueira SA, Vercesi AE, Teixeira SA, Costa SKP, Muscará MN, Ferreira HHA. Increased glutathione levels contribute to the beneficial effects of hydrogen sulfide and inducible nitric oxide inhibition in allergic lung inflammation [Internet]. International Immunopharmacology. 2016 ; 39 57-62.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2016.07.009
Artigo de periodico
Inflammatory mediators involved in the paw edema and hyperalgesia induced by Batroxase, a metalloproteinase isolated from Bothrops atrox snake venom (2015)
Toni, Lanuze G. B. de; Menaldo, Danilo L; Cintra, Adélia Cristina Oliveira; Figueiredo, Maria J; Souza, Anderson Roberto de; Maximiano, William M. A; Jamur, Maria Celia; Souza, Glória Emília Petto de; Sampaio, Suely Vilela
Source: International Immunopharmacology. Unidades: FCFRP, FMRP
Subjects: VENENOS DE ORIGEM ANIMAL, LEUCOTRIENOS, ÓXIDO NÍTRICO
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ABNT
TONI, Lanuze G. B. de et al. Inflammatory mediators involved in the paw edema and hyperalgesia induced by Batroxase, a metalloproteinase isolated from Bothrops atrox snake venom. International Immunopharmacology, v. 28, n. 1, p. 199-207, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2015.06.001. Acesso em: 05 nov. 2024.
APA
Toni, L. G. B. de, Menaldo, D. L., Cintra, A. C. O., Figueiredo, M. J., Souza, A. R. de, Maximiano, W. M. A., et al. (2015). Inflammatory mediators involved in the paw edema and hyperalgesia induced by Batroxase, a metalloproteinase isolated from Bothrops atrox snake venom. International Immunopharmacology, 28( 1), 199-207. doi:10.1016/j.intimp.2015.06.001
NLM
Toni LGB de, Menaldo DL, Cintra ACO, Figueiredo MJ, Souza AR de, Maximiano WMA, Jamur MC, Souza GEP de, Sampaio SV. Inflammatory mediators involved in the paw edema and hyperalgesia induced by Batroxase, a metalloproteinase isolated from Bothrops atrox snake venom [Internet]. International Immunopharmacology. 2015 ; 28( 1): 199-207.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2015.06.001
Vancouver
Toni LGB de, Menaldo DL, Cintra ACO, Figueiredo MJ, Souza AR de, Maximiano WMA, Jamur MC, Souza GEP de, Sampaio SV. Inflammatory mediators involved in the paw edema and hyperalgesia induced by Batroxase, a metalloproteinase isolated from Bothrops atrox snake venom [Internet]. International Immunopharmacology. 2015 ; 28( 1): 199-207.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.intimp.2015.06.001

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