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Artigo de periodico
T lymphocyte exhaustion during human and experimental visceral leishmaniasis (2022)
Costa-Madeira, Juliana Cristina; Trindade, Gabrielly Bindo; Almeida, Paulo Henrique do Prado; Silva, João Santana da ; Carregaro, Vanessa
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: INFLAMAÇÃO, LINFÓCITOS T, SISTEMA IMUNE, IMUNOTERAPIA
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COSTA-MADEIRA, Juliana Cristina et al. T lymphocyte exhaustion during human and experimental visceral leishmaniasis. Frontiers in Immunology, v. 13, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.835711. Acesso em: 13 nov. 2024.
APA
Costa-Madeira, J. C., Trindade, G. B., Almeida, P. H. do P., Silva, J. S. da, & Carregaro, V. (2022). T lymphocyte exhaustion during human and experimental visceral leishmaniasis. Frontiers in Immunology, 13, 1-15. doi:10.3389/fimmu.2022.835711
NLM
Costa-Madeira JC, Trindade GB, Almeida PH do P, Silva JS da, Carregaro V. T lymphocyte exhaustion during human and experimental visceral leishmaniasis [Internet]. Frontiers in Immunology. 2022 ; 13 1-15.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2022.835711
Vancouver
Costa-Madeira JC, Trindade GB, Almeida PH do P, Silva JS da, Carregaro V. T lymphocyte exhaustion during human and experimental visceral leishmaniasis [Internet]. Frontiers in Immunology. 2022 ; 13 1-15.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2022.835711
Artigo de periodico
Efficacy of probiotics in children and adolescents with atopic dermatitis: a randomized, double-blind, placebo-controlled study (2022)
Andrade, Paula Danielle Santa Maria Albuquerque de; Silva, Jorgete Maria e; Carregaro, Vanessa; Sacramento, Laís Amorim ; Roberti, Luciana Rodrigues; Aragon, Davi Casale ; Carmona, Fabio ; Roxo Júnior, Pérsio
Source: Frontiers in Nutrition. Unidade: FMRP
Subjects: DERMATITE, TOLERÂNCIA IMUNOLÓGICA, PROBIÓTICOS
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ANDRADE, Paula Danielle Santa Maria Albuquerque de et al. Efficacy of probiotics in children and adolescents with atopic dermatitis: a randomized, double-blind, placebo-controlled study. Frontiers in Nutrition, v. 8, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3389/fnut.2021.833666. Acesso em: 13 nov. 2024.
APA
Andrade, P. D. S. M. A. de, Silva, J. M. e, Carregaro, V., Sacramento, L. A., Roberti, L. R., Aragon, D. C., et al. (2022). Efficacy of probiotics in children and adolescents with atopic dermatitis: a randomized, double-blind, placebo-controlled study. Frontiers in Nutrition, 8, 1-10. doi:10.3389/fnut.2021.833666
NLM
Andrade PDSMA de, Silva JM e, Carregaro V, Sacramento LA, Roberti LR, Aragon DC, Carmona F, Roxo Júnior P. Efficacy of probiotics in children and adolescents with atopic dermatitis: a randomized, double-blind, placebo-controlled study [Internet]. Frontiers in Nutrition. 2022 ; 8 1-10.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fnut.2021.833666
Vancouver
Andrade PDSMA de, Silva JM e, Carregaro V, Sacramento LA, Roberti LR, Aragon DC, Carmona F, Roxo Júnior P. Efficacy of probiotics in children and adolescents with atopic dermatitis: a randomized, double-blind, placebo-controlled study [Internet]. Frontiers in Nutrition. 2022 ; 8 1-10.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fnut.2021.833666
Artigo de periodico
Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection (2022)
Maruyama, Sandra Regina; Fuzo, Carlos Alessandro ; Oliveira, Antonio Edson Rocha; Rogerio, Luana Aparecida; Takamiya, Nayore Tamie; Pessenda, Gabriela; Melo, Enaldo Vieira de; Silva, Ângela Maria da; Jesus, Amelia Ribeiro de; Carregaro, Vanessa ; Nakaya, Helder Takashi Imoto ; Almeida, Roque Pacheco de; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, FCF, FCFRP
Subjects: LEISHMANIOSE VISCERAL, LEISHMANIA, RNA, SEQUENCIAMENTO GENÉTICO
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MARUYAMA, Sandra Regina et al. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection. Frontiers in Immunology, v. 13, p. 1-18, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.784463. Acesso em: 13 nov. 2024.
APA
Maruyama, S. R., Fuzo, C. A., Oliveira, A. E. R., Rogerio, L. A., Takamiya, N. T., Pessenda, G., et al. (2022). Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection. Frontiers in Immunology, 13, 1-18. doi:10.3389/fimmu.2022.784463
NLM
Maruyama SR, Fuzo CA, Oliveira AER, Rogerio LA, Takamiya NT, Pessenda G, Melo EV de, Silva ÂM da, Jesus AR de, Carregaro V, Nakaya HTI, Almeida RP de, Silva JS da. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection [Internet]. Frontiers in Immunology. 2022 ; 13 1-18.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2022.784463
Vancouver
Maruyama SR, Fuzo CA, Oliveira AER, Rogerio LA, Takamiya NT, Pessenda G, Melo EV de, Silva ÂM da, Jesus AR de, Carregaro V, Nakaya HTI, Almeida RP de, Silva JS da. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection [Internet]. Frontiers in Immunology. 2022 ; 13 1-18.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2022.784463
Artigo de periodico
Neuronal cholinergic signaling constrains norepinephrine activity in the heart (2022)
Guimarães, Diogo A.; Aquino, Nayara Soares Sena ; Rocha-Resende, Cibele; Jesus, Itamar Couto Guedes ; Silva, Mário Morais; Scalzo, Sérgio A.; Fonseca, Roberta Cristelli; Durand, Marina T.; Pereira, Vanessa Carregaro; Tezini, Geisa C. S. V.; Oliveira, André; Prado, Vania F.; Stefanon, Ivanita; Salgado, Hélio César ; Prado, Marco Antonio Maximo; Szawka, Raphael E.; Guatimosim, Silvia
Source: American Journal of Physiology-Cell Physiology. Unidades: EACH, FMRP
Subjects: ADRENÉRGICOS, COLINÉRGICOS, NOREPINEFRINA, DOENÇAS CARDIOVASCULARES
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GUIMARÃES, Diogo A. et al. Neuronal cholinergic signaling constrains norepinephrine activity in the heart. American Journal of Physiology-Cell Physiology, v. 322, n. 4, p. C794-C801, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00031.2022. Acesso em: 13 nov. 2024.
APA
Guimarães, D. A., Aquino, N. S. S., Rocha-Resende, C., Jesus, I. C. G., Silva, M. M., Scalzo, S. A., et al. (2022). Neuronal cholinergic signaling constrains norepinephrine activity in the heart. American Journal of Physiology-Cell Physiology, 322( 4), C794-C801. doi:10.1152/ajpcell.00031.2022
NLM
Guimarães DA, Aquino NSS, Rocha-Resende C, Jesus ICG, Silva MM, Scalzo SA, Fonseca RC, Durand MT, Pereira VC, Tezini GCSV, Oliveira A, Prado VF, Stefanon I, Salgado HC, Prado MAM, Szawka RE, Guatimosim S. Neuronal cholinergic signaling constrains norepinephrine activity in the heart [Internet]. American Journal of Physiology-Cell Physiology. 2022 ; 322( 4): C794-C801.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpcell.00031.2022
Vancouver
Guimarães DA, Aquino NSS, Rocha-Resende C, Jesus ICG, Silva MM, Scalzo SA, Fonseca RC, Durand MT, Pereira VC, Tezini GCSV, Oliveira A, Prado VF, Stefanon I, Salgado HC, Prado MAM, Szawka RE, Guatimosim S. Neuronal cholinergic signaling constrains norepinephrine activity in the heart [Internet]. American Journal of Physiology-Cell Physiology. 2022 ; 322( 4): C794-C801.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpcell.00031.2022
Artigo de periodico
Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni (2020)
Mota, Ester Alves; Patrocínio, Andressa Barban do; Rodrigues, Vanderlei ; Silva, João Santana da ; Pereira, Vanessa Carregaro; Guerra-Sá, Renata
Source: PLOS Neglected Tropical Diseases. Unidade: FMRP
Subjects: SCHISTOSOMA MANSONI, METILAÇÃO DE DNA, ANIMAIS GENETICAMENTE MODIFICADOS, DOENÇAS PARASITÁRIAS, GRANULOMA, EXPRESSÃO GÊNICA, MICRORNAS
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MOTA, Ester Alves et al. Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni. PLOS Neglected Tropical Diseases, v. 14, n. 2, p. 1-24, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0008080. Acesso em: 13 nov. 2024.
APA
Mota, E. A., Patrocínio, A. B. do, Rodrigues, V., Silva, J. S. da, Pereira, V. C., & Guerra-Sá, R. (2020). Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni. PLOS Neglected Tropical Diseases, 14( 2), 1-24. doi:10.1371/journal.pntd.0008080
NLM
Mota EA, Patrocínio AB do, Rodrigues V, Silva JS da, Pereira VC, Guerra-Sá R. Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni [Internet]. PLOS Neglected Tropical Diseases. 2020 ; 14( 2): 1-24.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.pntd.0008080
Vancouver
Mota EA, Patrocínio AB do, Rodrigues V, Silva JS da, Pereira VC, Guerra-Sá R. Epigenetic and parasitological parameters are modulated in EBi3-/- mice infected with Schistosoma mansoni [Internet]. PLOS Neglected Tropical Diseases. 2020 ; 14( 2): 1-24.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.pntd.0008080
Artigo de periodico
TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection (2020)
Sacramento, Laís Amorim ; Benevides, Luciana; Maruyama, Sandra Regina Costa; Tavares, Lucas Alves ; Fukutani, Kiyoshi Ferreira; Françozo, Marcela; Sparwasser, Tim; Cunha, Fernando de Queiroz ; Almeida, Roque Pacheco de; Silva, João Santana da ; Carregaro, Vanessa
Source: PLOS Pathogens. Unidade: FMRP
Subjects: INFLAMAÇÃO, IMUNOLOGIA, INTERFERONS, LEISHMANIA INFANTUM, LEISHMANIOSE VISCERAL
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SACRAMENTO, Laís Amorim et al. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection. PLOS Pathogens, v. 16, n. 3, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1008435. Acesso em: 13 nov. 2024.
APA
Sacramento, L. A., Benevides, L., Maruyama, S. R. C., Tavares, L. A., Fukutani, K. F., Françozo, M., et al. (2020). TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection. PLOS Pathogens, 16( 3). doi:10.1371/journal.ppat.1008435
NLM
Sacramento LA, Benevides L, Maruyama SRC, Tavares LA, Fukutani KF, Françozo M, Sparwasser T, Cunha F de Q, Almeida RP de, Silva JS da, Carregaro V. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection [Internet]. PLOS Pathogens. 2020 ; 16( 3):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.ppat.1008435
Vancouver
Sacramento LA, Benevides L, Maruyama SRC, Tavares LA, Fukutani KF, Françozo M, Sparwasser T, Cunha F de Q, Almeida RP de, Silva JS da, Carregaro V. TLR4 abrogates the Th1 immune response through IRF1 and IFN-β to prevent immunopathology during L. infantum infection [Internet]. PLOS Pathogens. 2020 ; 16( 3):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.ppat.1008435
Artigo de periodico
Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis (2019)
Gupta, Gaurav; Santana, Alynne Karen Mendonça de; Gomes, Ciro Martins ; Turatti, Aline; Milanezi, Cristiane Maria; Bueno Filho, Roberto ; Fuzo, Carlos Alessandro ; Almeida, Roque Pacheco de ; Carregaro, Vanessa ; Roselino, Ana Maria Ferreira ; Silva, João Santana da
Source: Cellular Immunology. Unidades: FMRP, FCFRP
Subjects: LEISHMANIOSE CUTÂNEA, INFLAMAÇÃO
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GUPTA, Gaurav et al. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis. Cellular Immunology, v. 341, p. [8] , 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cellimm.2019.04.008. Acesso em: 13 nov. 2024.
APA
Gupta, G., Santana, A. K. M. de, Gomes, C. M., Turatti, A., Milanezi, C. M., Bueno Filho, R., et al. (2019). Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis. Cellular Immunology, 341, [8] . doi:10.1016/j.cellimm.2019.04.008
NLM
Gupta G, Santana AKM de, Gomes CM, Turatti A, Milanezi CM, Bueno Filho R, Fuzo CA, Almeida RP de, Carregaro V, Roselino AMF, Silva JS da. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis [Internet]. Cellular Immunology. 2019 ; 341 [8] .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.cellimm.2019.04.008
Vancouver
Gupta G, Santana AKM de, Gomes CM, Turatti A, Milanezi CM, Bueno Filho R, Fuzo CA, Almeida RP de, Carregaro V, Roselino AMF, Silva JS da. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis [Internet]. Cellular Immunology. 2019 ; 341 [8] .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.cellimm.2019.04.008
Artigo de periodico
NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection (2019)
Benevides, Luciana ; Saltarelli, Verônica Marcela; Pioto, Franciele ; Sacramento, Laís Amorim ; Dias, Murilo Solano; Rodríguez, Gretel Rodríguez ; Viola, João Paulo de Biaso ; Carregaro, Vanessa ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ESALQ
Subjects: INFLAMAÇÃO, LINFÓCITOS T, MONÓCITOS, TOXOPLASMA GONDII, TOXOPLASMOSE
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BENEVIDES, Luciana et al. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection. Frontiers in Immunology, v. 10, p. [10] , 2019Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2019.02105. Acesso em: 13 nov. 2024.
APA
Benevides, L., Saltarelli, V. M., Pioto, F., Sacramento, L. A., Dias, M. S., Rodríguez, G. R., et al. (2019). NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection. Frontiers in Immunology, 10, [10] . doi:10.3389/fimmu.2019.02105
NLM
Benevides L, Saltarelli VM, Pioto F, Sacramento LA, Dias MS, Rodríguez GR, Viola JP de B, Carregaro V, Silva JS da. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection [Internet]. Frontiers in Immunology. 2019 ; 10 [10] .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2019.02105
Vancouver
Benevides L, Saltarelli VM, Pioto F, Sacramento LA, Dias MS, Rodríguez GR, Viola JP de B, Carregaro V, Silva JS da. NFAT1 regulates Ly6Chi monocyte recruitment to the CNS and plays an essential role in resistance to Toxoplasma gondii infection [Internet]. Frontiers in Immunology. 2019 ; 10 [10] .[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2019.02105
Artigo de periodico
B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation (2019)
Benevides, Luciana ; Costa, Renata Sesti ; Tavares, Lucas Alves; Russo, Momtchilo ; Martins, Gislâine A.; Silva, Luis Lamberti Pinto da ; Arruda, Luisa Karla de Paula ; Cunha, Fernando de Queiroz ; Carregaro, Vanessa ; Silva, João Santana da
Source: Journal of Allergy and Clinical Immunology. Unidades: ICB, FMRP
Subjects: ALERGIA E IMUNOLOGIA, DIFERENCIAÇÃO CELULAR, LINFÓCITOS B, INFLAMAÇÃO, CITOMETRIA DE FLUXO, ELISA, REAÇÃO EM CADEIA POR POLIMERASE, CAMUNDONGOS
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ABNT
BENEVIDES, Luciana et al. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation. Journal of Allergy and Clinical Immunology, v. 143, n. 3, p. 1119-1130, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.jaci.2018.06.046. Acesso em: 13 nov. 2024.
APA
Benevides, L., Costa, R. S., Tavares, L. A., Russo, M., Martins, G. A., Silva, L. L. P. da, et al. (2019). B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation. Journal of Allergy and Clinical Immunology, 143( 3), 1119-1130. doi:10.1016/j.jaci.2018.06.046
NLM
Benevides L, Costa RS, Tavares LA, Russo M, Martins GA, Silva LLP da, Arruda LK de P, Cunha F de Q, Carregaro V, Silva JS da. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation [Internet]. Journal of Allergy and Clinical Immunology. 2019 ; 143( 3): 1119-1130.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.jaci.2018.06.046
Vancouver
Benevides L, Costa RS, Tavares LA, Russo M, Martins GA, Silva LLP da, Arruda LK de P, Cunha F de Q, Carregaro V, Silva JS da. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation [Internet]. Journal of Allergy and Clinical Immunology. 2019 ; 143( 3): 1119-1130.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.jaci.2018.06.046
Artigo de periodico
T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses (2018)
Reynolds, C. J.; Suleyman, O. M.; Ortega-Prieto, A. M.; Skelton, J. K.; Bonnesoeur, P.; Blohm, A.; Carregaro, Vanessa ; Silva, João Santana da ; James, E. A.; Maillère, B.; Dorner, M.; Boyton, R. J.; Altmann, D. M.
Source: Scientific Reports. Unidade: FMRP
Subjects: ZIKA VÍRUS, GENES DE RESPOSTA IMUNE, LINFÓCITOS, REAÇÃO CRUZADA
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REYNOLDS, C. J. et al. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses. Scientific Reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-18781-1. Acesso em: 13 nov. 2024.
APA
Reynolds, C. J., Suleyman, O. M., Ortega-Prieto, A. M., Skelton, J. K., Bonnesoeur, P., Blohm, A., et al. (2018). T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses. Scientific Reports, 8. doi:10.1038/s41598-017-18781-1
NLM
Reynolds CJ, Suleyman OM, Ortega-Prieto AM, Skelton JK, Bonnesoeur P, Blohm A, Carregaro V, Silva JS da, James EA, Maillère B, Dorner M, Boyton RJ, Altmann DM. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/s41598-017-18781-1
Vancouver
Reynolds CJ, Suleyman OM, Ortega-Prieto AM, Skelton JK, Bonnesoeur P, Blohm A, Carregaro V, Silva JS da, James EA, Maillère B, Dorner M, Boyton RJ, Altmann DM. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/s41598-017-18781-1
Artigo de periodico
TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis (2018)
Peres, Raphael Sanches; Donate, Paula Barbim; Talbot, Jhimmy; Cecílio, Nerry Tatiana; Lobo, Patricia Rolim Mendonça; Machado, Caio Cavalcante; Lima, Kalil William Alves de; Oliveira, Renê Donizete Ribeiro de; Carregaro, Vanessa; Nakaya, Helder Takashi Imoto ; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Liew, Foo Yew; Louzada Júnior, Paulo; Cunha, Fernando Queiróz
Source: Journal of Autoimmunity. Unidades: FMRP, FCF
Subjects: ARTRITE REUMATOIDE, METOTREXATO
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PERES, Raphael Sanches et al. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, v. 90, p. 49-58, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jaut.2018.01.004. Acesso em: 13 nov. 2024.
APA
Peres, R. S., Donate, P. B., Talbot, J., Cecílio, N. T., Lobo, P. R. M., Machado, C. C., et al. (2018). TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, 90, 49-58. doi:10.1016/j.jaut.2018.01.004
NLM
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Vancouver
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Artigo de periodico
Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection (2017)
Lima, Mikhael Haruo Fernandes de; Sacramento, Laís Amorim; Quirino, Gustavo Fernando da Silva; Ferreira, Marcela Davoli; Benevides, Luciana; Santana, Alynne Karen M.; Cunha, Fernando de Queiroz; Almeida, Roque P.; Silva, João Santana da; Carregaro, Vanessa
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: LEISHMANIOSE VISCERAL, INFLAMAÇÃO, ADENOSINA
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00815. Acesso em: 13 nov. 2024.
APA
Lima, M. H. F. de, Sacramento, L. A., Quirino, G. F. da S., Ferreira, M. D., Benevides, L., Santana, A. K. M., et al. (2017). Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00815
NLM
Lima MHF de, Sacramento LA, Quirino GF da S, Ferreira MD, Benevides L, Santana AKM, Cunha F de Q, Almeida RP, Silva JS da, Carregaro V. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2017.00815
Vancouver
Lima MHF de, Sacramento LA, Quirino GF da S, Ferreira MD, Benevides L, Santana AKM, Cunha F de Q, Almeida RP, Silva JS da, Carregaro V. Leishmania infantum Parasites subvert the host inflammatory response through the adenosine A2A receptor to promote the establishment of infection [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2017.00815
Artigo de periodico
Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection (2017)
Sacramento, Laís A.; Costa, Jéssica L. da; Lima, Mikhael H. F. de; Sampaio, Pedro A.; Almeida, Roque P.; Cunha, Fernando Queiróz; Silva, João Santana da; Carregaro, Vanessa
Source: Frontiers in Microbiology. Unidade: FMRP
Subjects: LEISHMANIA INFANTUM, CÉLULAS DENDRÍTICAS, LEISHMANIOSE VISCERAL, NEUTRÓFILOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SACRAMENTO, Laís A. et al. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2017.00262. Acesso em: 13 nov. 2024.
APA
Sacramento, L. A., Costa, J. L. da, Lima, M. H. F. de, Sampaio, P. A., Almeida, R. P., Cunha, F. Q., et al. (2017). Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection. Frontiers in Microbiology, 8. doi:10.3389/fmicb.2017.00262
NLM
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Vancouver
Sacramento LA, Costa JL da, Lima MHF de, Sampaio PA, Almeida RP, Cunha FQ, Silva JS da, Carregaro V. Toll-like receptor 2 is required for inflammatory process development during leishmania infantum infection [Internet]. Frontiers in Microbiology. 2017 ; 8[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fmicb.2017.00262
Artigo de periodico
T1/ST2 deficient mice display protection against Leishmania infantum experimental infection (2017)
Khalid, Khalid Eltahir; Nascimento, Manuela Sales Lima; Sacramento, Laís Amorim; Costa, Diego Luís; Lima Júnior, Djalma Souza; Carregaro, Vanessa; Silva, João Santana da
Source: Acta Tropica. Unidade: FMRP
Subjects: LINFÓCITOS T, LEISHMANIA INFANTUM, INTERLEUCINAS, LEISHMANIOSE VISCERAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KHALID, Khalid Eltahir et al. T1/ST2 deficient mice display protection against Leishmania infantum experimental infection. Acta Tropica, v. 172, p. 1-6, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.actatropica.2017.04.011. Acesso em: 13 nov. 2024.
APA
Khalid, K. E., Nascimento, M. S. L., Sacramento, L. A., Costa, D. L., Lima Júnior, D. S., Carregaro, V., & Silva, J. S. da. (2017). T1/ST2 deficient mice display protection against Leishmania infantum experimental infection. Acta Tropica, 172, 1-6. doi:10.1016/j.actatropica.2017.04.011
NLM
Khalid KE, Nascimento MSL, Sacramento LA, Costa DL, Lima Júnior DS, Carregaro V, Silva JS da. T1/ST2 deficient mice display protection against Leishmania infantum experimental infection [Internet]. Acta Tropica. 2017 ; 172 1-6.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.actatropica.2017.04.011
Vancouver
Khalid KE, Nascimento MSL, Sacramento LA, Costa DL, Lima Júnior DS, Carregaro V, Silva JS da. T1/ST2 deficient mice display protection against Leishmania infantum experimental infection [Internet]. Acta Tropica. 2017 ; 172 1-6.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.actatropica.2017.04.011
Artigo de periodico
NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis (2016)
Nascimento, Manuela S.; Ferreira, Marcela D.; Quirino, Gustavo F. S.; Maruyama, Sandra R.; Krishnaswamy, Jayendra K.; Liu, Dong; Berlink, Jonilson; Fonseca, Denise Morais da ; Zamboni, Dario Simões; Carregaro, Vanessa; Almeida, Roque P.; Cunha, Thiago Mattar; Eisenbarth, Stephanie S.; Silva, João Santana da
Source: The Journal of Infectious Diseases. Unidade: FMRP
Subjects: LEISHMANIA INFANTUM, CÉLULAS DENDRÍTICAS, LEISHMANIOSE VISCERAL, MODELOS ANIMAIS DE DOENÇAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, Manuela S. et al. NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis. The Journal of Infectious Diseases, v. 214, n. 11, p. 1647-1657, 2016Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiw446. Acesso em: 13 nov. 2024.
APA
Nascimento, M. S., Ferreira, M. D., Quirino, G. F. S., Maruyama, S. R., Krishnaswamy, J. K., Liu, D., et al. (2016). NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis. The Journal of Infectious Diseases, 214( 11), 1647-1657. doi:10.1093/infdis/jiw446
NLM
Nascimento MS, Ferreira MD, Quirino GFS, Maruyama SR, Krishnaswamy JK, Liu D, Berlink J, Fonseca DM da, Zamboni DS, Carregaro V, Almeida RP, Cunha TM, Eisenbarth SS, Silva JS da. NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis [Internet]. The Journal of Infectious Diseases. 2016 ; 214( 11): 1647-1657.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/infdis/jiw446
Vancouver
Nascimento MS, Ferreira MD, Quirino GFS, Maruyama SR, Krishnaswamy JK, Liu D, Berlink J, Fonseca DM da, Zamboni DS, Carregaro V, Almeida RP, Cunha TM, Eisenbarth SS, Silva JS da. NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis [Internet]. The Journal of Infectious Diseases. 2016 ; 214( 11): 1647-1657.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/infdis/jiw446
Artigo de periodico
CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection (2016)
Costa, Diego Luís; Lima Júnior, Djalma de Souza; Nascimento, Manuela Sales Lima; Sacramento, Laís Amorim; Almeida, Roque Pacheco de; Carregaro, Vanessa; Silva, João Santana da
Source: Journal of Leukocyte Biology. Unidade: FMRP
Subjects: LEISHMANIOSE CUTÂNEA, LEISHMANIA BRASILIENSIS, QUIMIOCINAS, SISTEMA IMUNE, TERAPÊUTICA MÉDICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Diego Luís et al. CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection. Journal of Leukocyte Biology, v. 100, n. 2, p. 423-432, 2016Tradução . . Disponível em: https://doi.org/10.1189/jlb.4a0715-288r. Acesso em: 13 nov. 2024.
APA
Costa, D. L., Lima Júnior, D. de S., Nascimento, M. S. L., Sacramento, L. A., Almeida, R. P. de, Carregaro, V., & Silva, J. S. da. (2016). CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection. Journal of Leukocyte Biology, 100( 2), 423-432. doi:10.1189/jlb.4a0715-288r
NLM
Costa DL, Lima Júnior D de S, Nascimento MSL, Sacramento LA, Almeida RP de, Carregaro V, Silva JS da. CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection [Internet]. Journal of Leukocyte Biology. 2016 ; 100( 2): 423-432.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1189/jlb.4a0715-288r
Vancouver
Costa DL, Lima Júnior D de S, Nascimento MSL, Sacramento LA, Almeida RP de, Carregaro V, Silva JS da. CCR2 signaling contributes to the differentiation of protective inflammatory dendritic cells in Leishmania braziliensis infection [Internet]. Journal of Leukocyte Biology. 2016 ; 100( 2): 423-432.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1189/jlb.4a0715-288r
Artigo de periodico
Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response (2016)
Quirino, Gustavo F. S.; Nascimento, Manuela S. L.; Davoli-Ferreira, Marcela; Sacramento, Lais A.; Lima, Mikhael H. F.; Almeida, Roque P.; Carregaro, Vanessa; Silva, João Santana da
Source: Infection and Immunity. Unidade: FMRP
Subjects: INTERLEUCINAS, LEISHMANIA INFANTUM, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
QUIRINO, Gustavo F. S. et al. Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response. Infection and Immunity, v. 84, n. 8, p. 2289-2298, 2016Tradução . . Disponível em: https://doi.org/10.1128/IAI.00283-16. Acesso em: 13 nov. 2024.
APA
Quirino, G. F. S., Nascimento, M. S. L., Davoli-Ferreira, M., Sacramento, L. A., Lima, M. H. F., Almeida, R. P., et al. (2016). Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response. Infection and Immunity, 84( 8), 2289-2298. doi:10.1128/IAI.00283-16
NLM
Quirino GFS, Nascimento MSL, Davoli-Ferreira M, Sacramento LA, Lima MHF, Almeida RP, Carregaro V, Silva JS da. Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response [Internet]. Infection and Immunity. 2016 ; 84( 8): 2289-2298.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1128/IAI.00283-16
Vancouver
Quirino GFS, Nascimento MSL, Davoli-Ferreira M, Sacramento LA, Lima MHF, Almeida RP, Carregaro V, Silva JS da. Interleukin-27 (IL-27) mediates susceptibility to visceral Leishmaniasis by suppressing the IL-17-neutrophil response [Internet]. Infection and Immunity. 2016 ; 84( 8): 2289-2298.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1128/IAI.00283-16
Artigo de periodico
Expression of SOFAT by T- and B-lineage cells may contribute to bone loss (2016)
Jarry, Christian R; Martinez, Elizabeth F; Peruzzo, Daiane C; Carregaro, Vanessa; Sacramento, Laís A; Araújo, Vera C; Weitzmann, M. Neale; Napimoga, Marcelo H
Source: Molecular Medicine Reports. Unidade: FMRP
Subjects: CITOCINAS, OSTEOCLASTO, INFLAMAÇÃO, PERIODONTITE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JARRY, Christian R et al. Expression of SOFAT by T- and B-lineage cells may contribute to bone loss. Molecular Medicine Reports, v. 13, n. 5, p. 4252-4258, 2016Tradução . . Disponível em: https://doi.org/10.3892/mmr.2016.5045. Acesso em: 13 nov. 2024.
APA
Jarry, C. R., Martinez, E. F., Peruzzo, D. C., Carregaro, V., Sacramento, L. A., Araújo, V. C., et al. (2016). Expression of SOFAT by T- and B-lineage cells may contribute to bone loss. Molecular Medicine Reports, 13( 5), 4252-4258. doi:10.3892/mmr.2016.5045
NLM
Jarry CR, Martinez EF, Peruzzo DC, Carregaro V, Sacramento LA, Araújo VC, Weitzmann MN, Napimoga MH. Expression of SOFAT by T- and B-lineage cells may contribute to bone loss [Internet]. Molecular Medicine Reports. 2016 ; 13( 5): 4252-4258.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3892/mmr.2016.5045
Vancouver
Jarry CR, Martinez EF, Peruzzo DC, Carregaro V, Sacramento LA, Araújo VC, Weitzmann MN, Napimoga MH. Expression of SOFAT by T- and B-lineage cells may contribute to bone loss [Internet]. Molecular Medicine Reports. 2016 ; 13( 5): 4252-4258.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3892/mmr.2016.5045
Artigo de periodico
Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection (2015)
Nascimento, Manuela Sales Lima; Carregaro, Vanessa; Lima Júnior, Djalma Souza; Costa, Diego Luís; Ryffel, Bernhard; Duthie, Malcolm S.; Jesus, Amélia de; Almeida, Roque Pacheco de; Silva, João Santana da
Source: Journal of Infectious Diseases. Unidade: FMRP
Subjects: IMUNOLOGIA CELULAR, INTERFERONS, INTERLEUCINAS, LEISHMANIOSE VISCERAL, LINFÓCITOS T
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, Manuela Sales Lima et al. Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection. Journal of Infectious Diseases, v. 211, n. 6, p. 1015-1025, 2015Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu531. Acesso em: 13 nov. 2024.
APA
Nascimento, M. S. L., Carregaro, V., Lima Júnior, D. S., Costa, D. L., Ryffel, B., Duthie, M. S., et al. (2015). Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection. Journal of Infectious Diseases, 211( 6), 1015-1025. doi:10.1093/infdis/jiu531
NLM
Nascimento MSL, Carregaro V, Lima Júnior DS, Costa DL, Ryffel B, Duthie MS, Jesus A de, Almeida RP de, Silva JS da. Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection [Internet]. Journal of Infectious Diseases. 2015 ; 211( 6): 1015-1025.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/infdis/jiu531
Vancouver
Nascimento MSL, Carregaro V, Lima Júnior DS, Costa DL, Ryffel B, Duthie MS, Jesus A de, Almeida RP de, Silva JS da. Interleukin 17A acts synergistically with interferon γ to promote protection against Leishmania infantum infection [Internet]. Journal of Infectious Diseases. 2015 ; 211( 6): 1015-1025.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1093/infdis/jiu531
Artigo de periodico
Nucleosides present on phlebotomine saliva induce immunossuppression and promote the infection establishment (2015)
Carregaro, Vanessa; Ribeiro, José M.; Valenzuela, Jesus G.; Souza Júnior, Djalma L.; Costa, Diego L.; Oliveira, Carlo J. F.; Sacramento, Laís A.; Nascimento, Manuela S. L.; Milanezi, Cristiane Maria; Cunha, Fernando de Queiroz; Silva, João Santana da
Source: PLOS Neglected Tropical Diseases. Unidade: FMRP
Subjects: INTERLEUCINA 10 (METABOLISMO), LEISHMANIA, NUCLEOSÍDEOS, SALIVA (QUÍMICA), IMUNOSSUPRESSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARREGARO, Vanessa et al. Nucleosides present on phlebotomine saliva induce immunossuppression and promote the infection establishment. PLOS Neglected Tropical Diseases, v. 9, n. 4, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0003600. Acesso em: 13 nov. 2024.
APA
Carregaro, V., Ribeiro, J. M., Valenzuela, J. G., Souza Júnior, D. L., Costa, D. L., Oliveira, C. J. F., et al. (2015). Nucleosides present on phlebotomine saliva induce immunossuppression and promote the infection establishment. PLOS Neglected Tropical Diseases, 9( 4). doi:10.1371/journal.pntd.0003600
NLM
Carregaro V, Ribeiro JM, Valenzuela JG, Souza Júnior DL, Costa DL, Oliveira CJF, Sacramento LA, Nascimento MSL, Milanezi CM, Cunha F de Q, Silva JS da. Nucleosides present on phlebotomine saliva induce immunossuppression and promote the infection establishment [Internet]. PLOS Neglected Tropical Diseases. 2015 ; 9( 4):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.pntd.0003600
Vancouver
Carregaro V, Ribeiro JM, Valenzuela JG, Souza Júnior DL, Costa DL, Oliveira CJF, Sacramento LA, Nascimento MSL, Milanezi CM, Cunha F de Q, Silva JS da. Nucleosides present on phlebotomine saliva induce immunossuppression and promote the infection establishment [Internet]. PLOS Neglected Tropical Diseases. 2015 ; 9( 4):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.pntd.0003600

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