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Artigo de periodico
Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome (2024)
Cesaro, Aline Cristina Parletta ; Cerri, Gabriela Cavazza ; Gasparini, Claudia Ribeiro Borba; Panico, Karine ; Vieira Júnior, Denival Nascimento ; Rodrigues, Larissa Maria Zacarias ; Senger, Nathalia ; Silva, Amanda de Almeida; Fevereiro, Marina Reingruber ; Diniz, Gabriela Placoná ; Irigoyen, Maria Cláudia Costa ; Chaves, Maria Luiza Morais Barreto de
Source: Pflügers Archiv - European Journal of Physiology. Unidades: ICB, FM
Subjects: ANATOMIA, HIPERTROFIA VENTRICULAR ESQUERDA, INSUFICIÊNCIA CARDÍACA ANIMAL, CARDIOMEGALIA ANIMAL, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, FREQUÊNCIA CARDÍACA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS
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ABNT
CESARO, Aline Cristina Parletta et al. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome. Pflügers Archiv - European Journal of Physiology, v. 476, p. 1065-1075, 2024Tradução . . Disponível em: https://doi.org/10.1007/s00424-024-02965-6. Acesso em: 10 nov. 2024.
APA
Cesaro, A. C. P., Cerri, G. C., Gasparini, C. R. B., Panico, K., Vieira Júnior, D. N., Rodrigues, L. M. Z., et al. (2024). Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome. Pflügers Archiv - European Journal of Physiology, 476, 1065-1075. doi:10.1007/s00424-024-02965-6
NLM
Cesaro ACP, Cerri GC, Gasparini CRB, Panico K, Vieira Júnior DN, Rodrigues LMZ, Senger N, Silva A de A, Fevereiro MR, Diniz GP, Irigoyen MCC, Chaves MLMB de. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome [Internet]. Pflügers Archiv - European Journal of Physiology. 2024 ; 476 1065-1075.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s00424-024-02965-6
Vancouver
Cesaro ACP, Cerri GC, Gasparini CRB, Panico K, Vieira Júnior DN, Rodrigues LMZ, Senger N, Silva A de A, Fevereiro MR, Diniz GP, Irigoyen MCC, Chaves MLMB de. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome [Internet]. Pflügers Archiv - European Journal of Physiology. 2024 ; 476 1065-1075.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s00424-024-02965-6
Artigo de periodico
Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice (2023)
Lino, Caroline Antunes ; Silva, Tábatha de Oliveira ; Lunardon, Guilherme ; Silva, Camila Stephanie Balbino ; Lima, Vanessa Morais ; Huang, Zhan-Peng ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná ; Takano, Ana Paula Cremasco
Source: Mechanisms of Ageing and Development. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, ADIPOSIDADE, TECIDO ADIPOSO, OBESIDADE, DIETA ANIMAL, GORDURAS NA DIETA, MICRORNAS, CAMUNDONGOS
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ABNT
LINO, Caroline Antunes et al. Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice. Mechanisms of Ageing and Development, v. 210, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.mad.2023.111775. Acesso em: 10 nov. 2024.
APA
Lino, C. A., Silva, T. de O., Lunardon, G., Silva, C. S. B., Lima, V. M., Huang, Z. -P., et al. (2023). Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice. Mechanisms of Ageing and Development, 210, 1-14. doi:10.1016/j.mad.2023.111775
NLM
Lino CA, Silva T de O, Lunardon G, Silva CSB, Lima VM, Huang Z-P, Donato Junior J, Barreto-Chaves MLM, Diniz GP, Takano APC. Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice [Internet]. Mechanisms of Ageing and Development. 2023 ; 210 1-14.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.mad.2023.111775
Vancouver
Lino CA, Silva T de O, Lunardon G, Silva CSB, Lima VM, Huang Z-P, Donato Junior J, Barreto-Chaves MLM, Diniz GP, Takano APC. Ablation of miRNA-22 protects against obesity-induced adipocyte senescence and ameliorates metabolic disorders in middle-aged mice [Internet]. Mechanisms of Ageing and Development. 2023 ; 210 1-14.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.mad.2023.111775
Artigo de periodico
miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction (2023)
Silva, Camila Stephanie Balbino ; Couto, Gisele Kruger ; Lino, Caroline Antunes ; Silva, Tábatha de Oliveira ; Lunardon, Guilherme ; Huang, Zhan-Peng ; Wang, Da Zhi ; Festuccia, William Tadeu Lara ; Barreto-Chaves, Maria Luiza Morais ; Rossoni, Luciana Venturini ; Diniz, Gabriela Placoná
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, FISIOLOGIA, AORTA DE ANIMAL, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, VASOS SANGUÍNEOS, TECIDO ADIPOSO, MICRORNAS, OBESIDADE, REAÇÃO EM CADEIA POR POLIMERASE, WESTERN BLOTTING
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ABNT
SILVA, Camila Stephanie Balbino et al. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, v. 316, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121416. Acesso em: 10 nov. 2024.
APA
Silva, C. S. B., Couto, G. K., Lino, C. A., Silva, T. de O., Lunardon, G., Huang, Z. -P., et al. (2023). miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction. Life Sciences, 316, 1-10. doi:10.1016/j.lfs.2023.121416
NLM
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Vancouver
Silva CSB, Couto GK, Lino CA, Silva T de O, Lunardon G, Huang Z-P, Wang DZ, Festuccia WTL, Barreto-Chaves MLM, Rossoni LV, Diniz GP. miRNA-22 is involved in the aortic reactivity in physiological conditions and mediates obesity-induced perivascular adipose tissue dysfunction [Internet]. Life Sciences. 2023 ; 316 1-10.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.lfs.2023.121416
Artigo de periodico
The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy (2022)
Silva, Tábatha De Oliveira ; Lino, Caroline Antunes ; Miranda, Juliane Braga ; Silva, Camila Stephanie Balbino ; Lunardon, Guilherme ; Lima, Vanessa Morais ; Jensen, Leonardo ; Donato Junior, José ; Irigoyen, Maria Cláudia Costa ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Experimental Physiology. Unidades: ICB, FM
Subjects: ANATOMIA, FISIOLOGIA, CARDIOMEGALIA ANIMAL, OBESIDADE, DIETA ANIMAL, HIPERTROFIA VENTRICULAR ESQUERDA, MICRORNAS, FIBRAS MUSCULARES, MIOSINA, RNA MENSAGEIRO, CAMUNDONGOS
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ABNT
SILVA, Tábatha De Oliveira et al. The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy. Experimental Physiology, v. 107, n. 8, p. 892-905, 2022Tradução . . Disponível em: https://doi.org/10.1113/EP090315. Acesso em: 10 nov. 2024.
APA
Silva, T. D. O., Lino, C. A., Miranda, J. B., Silva, C. S. B., Lunardon, G., Lima, V. M., et al. (2022). The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy. Experimental Physiology, 107( 8), 892-905. doi:10.1113/EP090315
NLM
Silva TDO, Lino CA, Miranda JB, Silva CSB, Lunardon G, Lima VM, Jensen L, Donato Junior J, Irigoyen MCC, Barreto-Chaves MLM, Diniz GP. The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy [Internet]. Experimental Physiology. 2022 ; 107( 8): 892-905.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1113/EP090315
Vancouver
Silva TDO, Lino CA, Miranda JB, Silva CSB, Lunardon G, Lima VM, Jensen L, Donato Junior J, Irigoyen MCC, Barreto-Chaves MLM, Diniz GP. The miRNA-143-3p-Sox6-Myh7 pathway is altered in obesogenic diet-induced cardiac hypertrophy [Internet]. Experimental Physiology. 2022 ; 107( 8): 892-905.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1113/EP090315
Artigo de periodico
Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice (2022)
Miranda, Juliane Braga ; Lunardon, Guilherme ; Lima, Vanessa Morais ; Silva, Tábatha de Oliveira ; Lino, Caroline Antunes ; Jensen, Leonardo ; Irigoyen, Maria Cláudia Costa ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Cellular Physiology and Biochemistry. Unidades: FM, ICB
Subjects: FISIOLOGIA, ANATOMIA, DIETA ANIMAL, GLICOSE, ISQUEMIA, OBESIDADE, CAMUNDONGOS, DOENÇAS CARDIOVASCULARES EM ANIMAL, DOENÇAS METABÓLICAS
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ABNT
MIRANDA, Juliane Braga et al. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, v. 56, n. 3, p. 293-309, 2022Tradução . . Disponível em: https://doi.org/10.33594/000000535. Acesso em: 10 nov. 2024.
APA
Miranda, J. B., Lunardon, G., Lima, V. M., Silva, T. de O., Lino, C. A., Jensen, L., et al. (2022). Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice. Cellular Physiology and Biochemistry, 56( 3), 293-309. doi:10.33594/000000535
NLM
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 nov. 10 ] Available from: https://doi.org/10.33594/000000535
Vancouver
Miranda JB, Lunardon G, Lima VM, Silva T de O, Lino CA, Jensen L, Irigoyen MCC, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Set7 deletion prevents glucose intolerance and improves the recovery of cardiac function after ischemia and reperfusion in obese female mice [Internet]. Cellular Physiology and Biochemistry. 2022 ; 56( 3): 293-309.[citado 2024 nov. 10 ] Available from: https://doi.org/10.33594/000000535
Artigo de periodico
SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism (2022)
Pedroso, João Alfredo Bolivar ; Silva, Ivson Bruno da ; Zampieri, Thais Tessari ; Totola, Leonardo Tedesco; Moreira, Thiago dos Santos ; Taniguti, Ana Paula Tiemi; Diniz, Gabriela Placoná ; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ANATOMIA, CARDIOPATIAS, HIPOTÁLAMO, LEPTINA, OBESIDADE, ENVELHECIMENTO, CAMUNDONGOS, METABOLISMO DE PROTEÍNA, METABOLISMO ENERGÉTICO, NEURÔNIOS, CITOCININAS, GLICOSE
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ABNT
PEDROSO, João Alfredo Bolivar et al. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism. International Journal of Molecular Sciences, v. 23, n. 12, p. 1-19, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23126484. Acesso em: 10 nov. 2024.
APA
Pedroso, J. A. B., Silva, I. B. da, Zampieri, T. T., Totola, L. T., Moreira, T. dos S., Taniguti, A. P. T., et al. (2022). SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism. International Journal of Molecular Sciences, 23( 12), 1-19. doi:10.3390/ijms23126484
NLM
Pedroso JAB, Silva IB da, Zampieri TT, Totola LT, Moreira T dos S, Taniguti APT, Diniz GP, Barreto-Chaves MLM, Donato Junior J. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 12): 1-19.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/ijms23126484
Vancouver
Pedroso JAB, Silva IB da, Zampieri TT, Totola LT, Moreira T dos S, Taniguti APT, Diniz GP, Barreto-Chaves MLM, Donato Junior J. SOCS3 ablation in Leptin Receptor-Expressing cells causes autonomic and cardiac dysfunctions in Middle-Aged mice despite improving energy and Glucose metabolism [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 12): 1-19.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/ijms23126484
Artigo de periodico
Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction (2022)
Lunardon, Guilherme ; Silva, Tábatha de Oliveira ; Lino, Caroline Antunes ; Miranda, Juliane Braga ; Silva, Amanda de Almeida; Irigoyen, Maria Cláudia Costa ; Takano, Ana Paula Cremasco ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Clinical Science (Online): an international journal of translational. Unidades: FM, ICB
Subjects: ANATOMIA, HIPERTROFIA VENTRICULAR ESQUERDA, CARDIOMEGALIA, CARDIOPATIAS, MIOCARDIOPATIAS, CÉLULAS MUSCULARES, CAMUNDONGOS, DNA MITOCONDRIAL
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ABNT
LUNARDON, Guilherme et al. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction. Clinical Science (Online): an international journal of translational, v. 136, p. 1537–1554, 2022Tradução . . Disponível em: https://doi.org/10.1042/CS20220466. Acesso em: 10 nov. 2024.
APA
Lunardon, G., Silva, T. de O., Lino, C. A., Miranda, J. B., Silva, A. de A., Irigoyen, M. C. C., et al. (2022). Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction. Clinical Science (Online): an international journal of translational, 136, 1537–1554. doi:10.1042/CS20220466
NLM
Lunardon G, Silva T de O, Lino CA, Miranda JB, Silva A de A, Irigoyen MCC, Takano APC, Barreto-Chaves MLM, Diniz GP. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction [Internet]. Clinical Science (Online): an international journal of translational. 2022 ; 136 1537–1554.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1042/CS20220466
Vancouver
Lunardon G, Silva T de O, Lino CA, Miranda JB, Silva A de A, Irigoyen MCC, Takano APC, Barreto-Chaves MLM, Diniz GP. Set7 deletion attenuates isoproterenol-induced cardiac fibrosis and delays cardiac dysfunction [Internet]. Clinical Science (Online): an international journal of translational. 2022 ; 136 1537–1554.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1042/CS20220466
Artigo de periodico
Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R) (2022)
Lino, Caroline Antunes ; Barreto-Chaves, Maria Luiza Morais
Source: Cellular Signalling. Unidade: ICB
Subjects: ANATOMIA, DOENÇAS CARDIOVASCULARES, SISTEMA RENINA-ANGIOTENSINA, HIPERTROFIA VENTRICULAR ESQUERDA, INFARTO DO MIOCÁRDIO, ALDOSTERONA, INFECÇÕES POR CORONAVIRUS, ANGIOTENSINA II, OLIGOPEPTÍDEOS
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ABNT
LINO, Caroline Antunes e BARRETO-CHAVES, Maria Luiza Morais. Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R). Cellular Signalling, v. 92, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.cellsig.2022.110253. Acesso em: 10 nov. 2024.
APA
Lino, C. A., & Barreto-Chaves, M. L. M. (2022). Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R). Cellular Signalling, 92, 1-9. doi:10.1016/j.cellsig.2022.110253
NLM
Lino CA, Barreto-Chaves MLM. Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R) [Internet]. Cellular Signalling. 2022 ; 92 1-9.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.cellsig.2022.110253
Vancouver
Lino CA, Barreto-Chaves MLM. Beta-arrestins in the context of cardiovascular diseases: focusing on angiotensin II type 1 receptor (AT1R) [Internet]. Cellular Signalling. 2022 ; 92 1-9.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.cellsig.2022.110253
Artigo de periodico
Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB (2020)
Senger, Nathalia ; Parletta, Aline C. ; Marques, Bruno Vinicius Duarte ; Campagnole-Santos, Maria Jose ; Santos, Robson A. S. ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná ; Akamine, Eliana Hiromi
Source: Journal of Cellular Physiology. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, ANATOMIA, DOENÇAS CARDIOVASCULARES, GLÂNDULA TIREOIDE, ANGIOTENSINAS, TRIIODOTIRONINA, INSUFICIÊNCIA CARDÍACA, ANTIOXIDANTES, CATALASE, ATIVAÇÃO ENZIMÁTICA, HORMÔNIOS TIREOIDIANOS
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ABNT
SENGER, Nathalia et al. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB. Journal of Cellular Physiology, p. 14 , 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.30069. Acesso em: 10 nov. 2024.
APA
Senger, N., Parletta, A. C., Marques, B. V. D., Campagnole-Santos, M. J., Santos, R. A. S., Barreto-Chaves, M. L. M., et al. (2020). Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB. Journal of Cellular Physiology, 14 . doi:10.1002/jcp.30069
NLM
Senger N, Parletta AC, Marques BVD, Campagnole-Santos MJ, Santos RAS, Barreto-Chaves MLM, Diniz GP, Akamine EH. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB [Internet]. Journal of Cellular Physiology. 2020 ;14 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1002/jcp.30069
Vancouver
Senger N, Parletta AC, Marques BVD, Campagnole-Santos MJ, Santos RAS, Barreto-Chaves MLM, Diniz GP, Akamine EH. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB [Internet]. Journal of Cellular Physiology. 2020 ;14 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1002/jcp.30069

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