Source: Life Sciences. Unidades: FCFRP, EERP
Subjects: NITRATOS, AORTA, VASODILATAÇÃO, OXIGÊNIO
ABNT
DINIZ, Mariana C. et al. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, v. 176, p. 26-34, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2017.03.016. Acesso em: 18 ago. 2024.APA
Diniz, M. C., Olivon, V. C., Tavares, L. D., Simplicio, J. A., Gonzaga, N. A., Souza, D. G. de, et al. (2017). Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids. Life Sciences, 176, 26-34. doi:10.1016/j.lfs.2017.03.016NLM
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 ago. 18 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016Vancouver
Diniz MC, Olivon VC, Tavares LD, Simplicio JA, Gonzaga NA, Souza DG de, Bendhack LM, Tirapelli CR, Bonaventura D. Mechanisms underlying sodium nitroprusside-induced tolerance in the mouse aorta: role of ROS and cyclooxygenase-derived prostanoids [Internet]. Life Sciences. 2017 ; 176 26-34.[citado 2024 ago. 18 ] Available from: https://doi.org/10.1016/j.lfs.2017.03.016