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Artigo de periodico
Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 (2023)
Simplicio, Janaina Aparecida ; Dourado, Thales de Mileto Henrique; Awata, Wanessa Mayumi Carvalho; Vale, Gabriel Tavares do ; Dias, Vinícius Ribeiro; Barros, Paula Rodrigues de ; Martinis, Bruno Spinosa de ; Tostes, Rita de Cássia Aleixo ; Tirapelli, Carlos Renato
Fonte: Life Sciences. Unidades: FFCLRP, FMRP, EERP
Assuntos: ETANOL, INTERLEUCINA 6, TECIDO ADIPOSO, ESTRESSE OXIDATIVO, HIPERTENSÃO
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ABNT
SIMPLICIO, Janaina Aparecida et al. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6. Life Sciences, v. 319, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2023.121526. Acesso em: 11 nov. 2024.
APA
Simplicio, J. A., Dourado, T. de M. H., Awata, W. M. C., Vale, G. T. do, Dias, V. R., Barros, P. R. de, et al. (2023). Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6. Life Sciences, 319, 1-10. doi:10.1016/j.lfs.2023.121526
NLM
Simplicio JA, Dourado T de MH, Awata WMC, Vale GT do, Dias VR, Barros PR de, Martinis BS de, Tostes R de CA, Tirapelli CR. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 [Internet]. Life Sciences. 2023 ; 319 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.121526
Vancouver
Simplicio JA, Dourado T de MH, Awata WMC, Vale GT do, Dias VR, Barros PR de, Martinis BS de, Tostes R de CA, Tirapelli CR. Ethanol consumption favors pro-contractile phenotype of perivascular adipose tissue: a role for interleukin-6 [Internet]. Life Sciences. 2023 ; 319 1-10.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.lfs.2023.121526
Artigo de periodico
Reactive oxygen species-dependent transient induction of genotoxicity by retene in human liver HepG2 cells (2023)
Scaramboni, Caroline ; Campos, Maria Lúcia Arruda de Moura ; Dorta, Daniel Junqueira ; Oliveira, Danielle Palma de ; Medeiros, Silvia Regina Batistuzzo de; Galvão, Marcos Felipe de Oliveira; Dreij, Kristian
Fonte: Toxicology in Vitro. Unidades: FFCLRP, FCFRP
Assuntos: BIOMASSA, HIDROCARBONETOS POLICÍCLICOS, ESTRESSE OXIDATIVO
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ABNT
SCARAMBONI, Caroline et al. Reactive oxygen species-dependent transient induction of genotoxicity by retene in human liver HepG2 cells. Toxicology in Vitro, v. 91, p. 1-9, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2023.105628. Acesso em: 11 nov. 2024.
APA
Scaramboni, C., Campos, M. L. A. de M., Dorta, D. J., Oliveira, D. P. de, Medeiros, S. R. B. de, Galvão, M. F. de O., & Dreij, K. (2023). Reactive oxygen species-dependent transient induction of genotoxicity by retene in human liver HepG2 cells. Toxicology in Vitro, 91, 1-9. doi:10.1016/j.tiv.2023.105628
NLM
Scaramboni C, Campos MLA de M, Dorta DJ, Oliveira DP de, Medeiros SRB de, Galvão MF de O, Dreij K. Reactive oxygen species-dependent transient induction of genotoxicity by retene in human liver HepG2 cells [Internet]. Toxicology in Vitro. 2023 ; 91 1-9.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.tiv.2023.105628
Vancouver
Scaramboni C, Campos MLA de M, Dorta DJ, Oliveira DP de, Medeiros SRB de, Galvão MF de O, Dreij K. Reactive oxygen species-dependent transient induction of genotoxicity by retene in human liver HepG2 cells [Internet]. Toxicology in Vitro. 2023 ; 91 1-9.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.tiv.2023.105628
Artigo de periodico
Targeting oxidative stress with polyphenols to fight liver diseases (2023)
Machado, Ivo F.; Miranda, Raul Ghiraldelli ; Dorta, Daniel Junqueira ; Rolo, Anabela Pinto; Palmeira, Carlos M.
Fonte: Antioxidants. Unidades: FFCLRP, FCFRP
Assuntos: FÍGADO, ESTRESSE OXIDATIVO, MITOCÔNDRIAS, ANTIOXIDANTES, POLIFENÓIS
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ABNT
MACHADO, Ivo F. et al. Targeting oxidative stress with polyphenols to fight liver diseases. Antioxidants, v. 12, n. 6, p. 1-25, 2023Tradução . . Disponível em: https://doi.org/10.3390/antiox12061212. Acesso em: 11 nov. 2024.
APA
Machado, I. F., Miranda, R. G., Dorta, D. J., Rolo, A. P., & Palmeira, C. M. (2023). Targeting oxidative stress with polyphenols to fight liver diseases. Antioxidants, 12( 6), 1-25. doi:10.3390/antiox12061212
NLM
Machado IF, Miranda RG, Dorta DJ, Rolo AP, Palmeira CM. Targeting oxidative stress with polyphenols to fight liver diseases [Internet]. Antioxidants. 2023 ; 12( 6): 1-25.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/antiox12061212
Vancouver
Machado IF, Miranda RG, Dorta DJ, Rolo AP, Palmeira CM. Targeting oxidative stress with polyphenols to fight liver diseases [Internet]. Antioxidants. 2023 ; 12( 6): 1-25.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/antiox12061212
Artigo de periodico-carta/editorial
Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II (2023)
Dorta, Daniel Junqueira ; Oliveira, Danielle Palma de ; Campos, Alline Cristina de ; Aschner, Michael
Fonte: Frontiers in Pharmacology. Unidades: FFCLRP, FCFRP, FMRP
Assuntos: CONTAMINAÇÃO AMBIENTAL, METAIS PESADOS, ESPÉCIES REATIVAS DE OXIGÊNIO, ESTRESSE OXIDATIVO, INFLAMAÇÃO, EPIGÊNESE GENÉTICA
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ABNT
DORTA, Daniel Junqueira et al. Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II. Frontiers in Pharmacology. Lausanne: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.3389/fphar.2023.1255471. Acesso em: 11 nov. 2024. , 2023
APA
Dorta, D. J., Oliveira, D. P. de, Campos, A. C. de, & Aschner, M. (2023). Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II. Frontiers in Pharmacology. Lausanne: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo. doi:10.3389/fphar.2023.1255471
NLM
Dorta DJ, Oliveira DP de, Campos AC de, Aschner M. Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II [Internet]. Frontiers in Pharmacology. 2023 ; 14 1-3.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fphar.2023.1255471
Vancouver
Dorta DJ, Oliveira DP de, Campos AC de, Aschner M. Neurobehavioral toxicity induced by environmental contaminants [Editorial]: mechanisms of neurodegeneration and neuroprotection, volume II [Internet]. Frontiers in Pharmacology. 2023 ; 14 1-3.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fphar.2023.1255471
Artigo de periodico
Improving Saccharomyces cerevisiae acid and oxidative stress resistance using a prokaryotic gene identified by functional metagenomics (2023)
Alves, Luana de Fátima ; Bortolucci, Jonatã ; Reginatto, Valeria ; Guazzaroni, Maria Eugenia ; Mussatto, Solange I.
Fonte: Heliyon. Unidade: FFCLRP
Assuntos: LEVEDURAS, SACCHAROMYCES, ESTRESSE OXIDATIVO, GENÔMICA
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ABNT
ALVES, Luana de Fátima et al. Improving Saccharomyces cerevisiae acid and oxidative stress resistance using a prokaryotic gene identified by functional metagenomics. Heliyon, v. 9, n. 4, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.heliyon.2023.e14838. Acesso em: 11 nov. 2024.
APA
Alves, L. de F., Bortolucci, J., Reginatto, V., Guazzaroni, M. E., & Mussatto, S. I. (2023). Improving Saccharomyces cerevisiae acid and oxidative stress resistance using a prokaryotic gene identified by functional metagenomics. Heliyon, 9( 4), 1-12. doi:10.1016/j.heliyon.2023.e14838
NLM
Alves L de F, Bortolucci J, Reginatto V, Guazzaroni ME, Mussatto SI. Improving Saccharomyces cerevisiae acid and oxidative stress resistance using a prokaryotic gene identified by functional metagenomics [Internet]. Heliyon. 2023 ; 9( 4): 1-12.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.heliyon.2023.e14838
Vancouver
Alves L de F, Bortolucci J, Reginatto V, Guazzaroni ME, Mussatto SI. Improving Saccharomyces cerevisiae acid and oxidative stress resistance using a prokaryotic gene identified by functional metagenomics [Internet]. Heliyon. 2023 ; 9( 4): 1-12.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.heliyon.2023.e14838
Artigo de periodico
Titanium dioxide nanoparticle (TiO2 NP) induces toxic effects on LA-9 mouse fibroblast cell line (2023)
Fattori, Ana Carolina Maragno ; Brassolatti, Patrícia ; Feitosa, Karina Alves; Gonçalves, Matheus Pedrino; Correia, Ricardo de Oliveira; Albuquerque, Yulli Roxenne; Rodolpho, Joice Margareth de Almeida ; Luna, Genoveva Lourdes Flores ; Bernardi, Juliana Cancino; Zucolotto, Valtencir ; Speglich, Carlos; Rossi, Karina Nogueira Zambone Pinto ; Aníbal, Fernanda de Freitas
Fonte: Cellular Physiology and Biochemistry. Unidades: IFSC, FFCLRP
Assuntos: NANOTECNOLOGIA, CITOTOXINAS, ESTRESSE OXIDATIVO, NANOPARTÍCULAS
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ABNT
FATTORI, Ana Carolina Maragno et al. Titanium dioxide nanoparticle (TiO2 NP) induces toxic effects on LA-9 mouse fibroblast cell line. Cellular Physiology and Biochemistry, v. 57, n. 2, p. 63-81, 2023Tradução . . Disponível em: https://doi.org/10.33594/000000616. Acesso em: 11 nov. 2024.
APA
Fattori, A. C. M., Brassolatti, P., Feitosa, K. A., Gonçalves, M. P., Correia, R. de O., Albuquerque, Y. R., et al. (2023). Titanium dioxide nanoparticle (TiO2 NP) induces toxic effects on LA-9 mouse fibroblast cell line. Cellular Physiology and Biochemistry, 57( 2), 63-81. doi:10.33594/000000616
NLM
Fattori ACM, Brassolatti P, Feitosa KA, Gonçalves MP, Correia R de O, Albuquerque YR, Rodolpho JM de A, Luna GLF, Bernardi JC, Zucolotto V, Speglich C, Rossi KNZP, Aníbal F de F. Titanium dioxide nanoparticle (TiO2 NP) induces toxic effects on LA-9 mouse fibroblast cell line [Internet]. Cellular Physiology and Biochemistry. 2023 ; 57( 2): 63-81.[citado 2024 nov. 11 ] Available from: https://doi.org/10.33594/000000616
Vancouver
Fattori ACM, Brassolatti P, Feitosa KA, Gonçalves MP, Correia R de O, Albuquerque YR, Rodolpho JM de A, Luna GLF, Bernardi JC, Zucolotto V, Speglich C, Rossi KNZP, Aníbal F de F. Titanium dioxide nanoparticle (TiO2 NP) induces toxic effects on LA-9 mouse fibroblast cell line [Internet]. Cellular Physiology and Biochemistry. 2023 ; 57( 2): 63-81.[citado 2024 nov. 11 ] Available from: https://doi.org/10.33594/000000616
Artigo de periodico
The labile iron pool reacts rapidly and catalytically with Peroxynitrite (2021)
Condeles, André Luís; Toledo Junior, José Carlos
Fonte: Biomolecules. Unidade: FFCLRP
Assuntos: BIOQUÍMICA, FLUORESCÊNCIA, FERRO, ÓXIDO NÍTRICO, ESTRESSE OXIDATIVO
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ABNT
CONDELES, André Luís e TOLEDO JUNIOR, José Carlos. The labile iron pool reacts rapidly and catalytically with Peroxynitrite. Biomolecules, v. 11, n. 9, p. 1-19, 2021Tradução . . Disponível em: https://doi.org/10.3390/biom11091331. Acesso em: 11 nov. 2024.
APA
Condeles, A. L., & Toledo Junior, J. C. (2021). The labile iron pool reacts rapidly and catalytically with Peroxynitrite. Biomolecules, 11( 9), 1-19. doi:10.3390/biom11091331
NLM
Condeles AL, Toledo Junior JC. The labile iron pool reacts rapidly and catalytically with Peroxynitrite [Internet]. Biomolecules. 2021 ; 11( 9): 1-19.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/biom11091331
Vancouver
Condeles AL, Toledo Junior JC. The labile iron pool reacts rapidly and catalytically with Peroxynitrite [Internet]. Biomolecules. 2021 ; 11( 9): 1-19.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/biom11091331
Artigo de periodico
Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney (2021)
Silva, Carla Beatriz Pereira da ; Ceron, Carla S.; Mendes, Atlante Silva; Martinis, Bruno Spinosa de ; Castro, Michele Mazzaron de; Tirapelli, Carlos Renato
Fonte: Canadian Journal of Physiology and Pharmacology. Unidades: FFCLRP, FMRP, EERP
Assuntos: BEBIDAS ALCOÓLICAS, CREATINA, CITOCINAS, ETANOL, INFLAMAÇÃO, NEFROPATIAS, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
SILVA, Carla Beatriz Pereira da et al. Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney. Canadian Journal of Physiology and Pharmacology, v. 99, n. 10, p. 1016-1025, 2021Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2021-0108. Acesso em: 11 nov. 2024.
APA
Silva, C. B. P. da, Ceron, C. S., Mendes, A. S., Martinis, B. S. de, Castro, M. M. de, & Tirapelli, C. R. (2021). Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney. Canadian Journal of Physiology and Pharmacology, 99( 10), 1016-1025. doi:10.1139/cjpp-2021-0108
NLM
Silva CBP da, Ceron CS, Mendes AS, Martinis BS de, Castro MM de, Tirapelli CR. Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 10): 1016-1025.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1139/cjpp-2021-0108
Vancouver
Silva CBP da, Ceron CS, Mendes AS, Martinis BS de, Castro MM de, Tirapelli CR. Inducible nitric oxide synthase (iNOS) mediates ethanol-induced redox imbalance and upregulation of inflammatory cytokines in the kidney [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 10): 1016-1025.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1139/cjpp-2021-0108
Artigo de periodico
Phospholipids modifications in human hepatoma cell lines (HepG2) exposed to silver and iron oxide nanoparticles (2020)
Adeyemi, Joseph A.; Sorgi, Carlos Arterio ; Machado, Ana Rita Thomazela ; Ogunjimi, Abayomi T.; Gardinassi, Luiz Gustavo Araujo ; Nardini, Viviani; Faccioli, Lucia Helena ; Antunes, Lusânia Maria Greggi ; Barbosa Júnior, Fernando
Fonte: Archives of Toxicology. Unidades: FFCLRP, FCFRP
Assuntos: MORTE, CÉLULAS, MUTAGÊNESE, ESTRESSE OXIDATIVO, TOXICOLOGIA, FENÓTIPOS, FOSFOLIPÍDEOS
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ABNT
ADEYEMI, Joseph A. et al. Phospholipids modifications in human hepatoma cell lines (HepG2) exposed to silver and iron oxide nanoparticles. Archives of Toxicology, v. 94, n. 8, p. 2625-2636, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00204-020-02789-0. Acesso em: 11 nov. 2024.
APA
Adeyemi, J. A., Sorgi, C. A., Machado, A. R. T., Ogunjimi, A. T., Gardinassi, L. G. A., Nardini, V., et al. (2020). Phospholipids modifications in human hepatoma cell lines (HepG2) exposed to silver and iron oxide nanoparticles. Archives of Toxicology, 94( 8), 2625-2636. doi:10.1007/s00204-020-02789-0
NLM
Adeyemi JA, Sorgi CA, Machado ART, Ogunjimi AT, Gardinassi LGA, Nardini V, Faccioli LH, Antunes LMG, Barbosa Júnior F. Phospholipids modifications in human hepatoma cell lines (HepG2) exposed to silver and iron oxide nanoparticles [Internet]. Archives of Toxicology. 2020 ; 94( 8): 2625-2636.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1007/s00204-020-02789-0
Vancouver
Adeyemi JA, Sorgi CA, Machado ART, Ogunjimi AT, Gardinassi LGA, Nardini V, Faccioli LH, Antunes LMG, Barbosa Júnior F. Phospholipids modifications in human hepatoma cell lines (HepG2) exposed to silver and iron oxide nanoparticles [Internet]. Archives of Toxicology. 2020 ; 94( 8): 2625-2636.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1007/s00204-020-02789-0
Artigo de periodico
A phytotoxicity assessment of the efficiency 2,4-D degradation by different oxidative processes (2020)
Forti, Juliane C.; Loretti, Gustavo H.; Tadayozzi, Yasmin S.; Andrade, Adalgisa Rodrigues de
Fonte: Journal of Environmental Management. Unidade: FFCLRP
Assuntos: HERBICIDAS, ELETRÓLISE, PERÓXIDO DE HIDROGÊNIO, OXIDAÇÃO, ESTRESSE OXIDATIVO, POLUIÇÃO DA ÁGUA
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ABNT
FORTI, Juliane C. et al. A phytotoxicity assessment of the efficiency 2,4-D degradation by different oxidative processes. Journal of Environmental Management, v. 266, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.jenvman.2020.110588. Acesso em: 11 nov. 2024.
APA
Forti, J. C., Loretti, G. H., Tadayozzi, Y. S., & Andrade, A. R. de. (2020). A phytotoxicity assessment of the efficiency 2,4-D degradation by different oxidative processes. Journal of Environmental Management, 266. doi:10.1016/j.jenvman.2020.110588
NLM
Forti JC, Loretti GH, Tadayozzi YS, Andrade AR de. A phytotoxicity assessment of the efficiency 2,4-D degradation by different oxidative processes [Internet]. Journal of Environmental Management. 2020 ; 266[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.jenvman.2020.110588
Vancouver
Forti JC, Loretti GH, Tadayozzi YS, Andrade AR de. A phytotoxicity assessment of the efficiency 2,4-D degradation by different oxidative processes [Internet]. Journal of Environmental Management. 2020 ; 266[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.jenvman.2020.110588
Artigo de periodico
Targeted cancer therapy using alpha-cyano-4-hydroxycinnamic acid as a novel vector molecule: a proof-of-concept study (2020)
Calori, Italo Rodrigo ; Piva, Henrique Luis ; Tedesco, Antônio Cláudio
Fonte: Journal of Drug Delivery Science and Technology. Unidade: FFCLRP
Assuntos: TERAPIA FOTODINÂMICA, CÉLULAS, NEOPLASIAS, ANTIOXIDANTES, LIPOSSOMOS, ESTRESSE OXIDATIVO
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ABNT
CALORI, Italo Rodrigo e PIVA, Henrique Luis e TEDESCO, Antônio Cláudio. Targeted cancer therapy using alpha-cyano-4-hydroxycinnamic acid as a novel vector molecule: a proof-of-concept study. Journal of Drug Delivery Science and Technology, v. 57, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.jddst.2020.101633. Acesso em: 11 nov. 2024.
APA
Calori, I. R., Piva, H. L., & Tedesco, A. C. (2020). Targeted cancer therapy using alpha-cyano-4-hydroxycinnamic acid as a novel vector molecule: a proof-of-concept study. Journal of Drug Delivery Science and Technology, 57. doi:10.1016/j.jddst.2020.101633
NLM
Calori IR, Piva HL, Tedesco AC. Targeted cancer therapy using alpha-cyano-4-hydroxycinnamic acid as a novel vector molecule: a proof-of-concept study [Internet]. Journal of Drug Delivery Science and Technology. 2020 ; 57[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.jddst.2020.101633
Vancouver
Calori IR, Piva HL, Tedesco AC. Targeted cancer therapy using alpha-cyano-4-hydroxycinnamic acid as a novel vector molecule: a proof-of-concept study [Internet]. Journal of Drug Delivery Science and Technology. 2020 ; 57[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.jddst.2020.101633
Artigo de periodico
Implications of dichlorofluorescein photoinstability for detection of UVA-induced oxidative stress in fibroblasts and keratinocyte cells (2020)
Souza, Carla ; Mônico, Danielli Azevedo ; Tedesco, Antônio Cláudio
Fonte: Photochemical & Photobiological Sciences. Unidade: FFCLRP
Assuntos: SOBREVIVÊNCIA CELULAR, CÉLULAS CULTIVADAS, CRIANÇAS EM IDADE PRÉ-ESCOLAR, FIBROBLASTOS, ESTRESSE OXIDATIVO
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ABNT
SOUZA, Carla e MÔNICO, Danielli Azevedo e TEDESCO, Antônio Cláudio. Implications of dichlorofluorescein photoinstability for detection of UVA-induced oxidative stress in fibroblasts and keratinocyte cells. Photochemical & Photobiological Sciences, v. 19, n. 1, p. 40-48, 2020Tradução . . Disponível em: https://doi.org/10.1039/c9pp00415g. Acesso em: 11 nov. 2024.
APA
Souza, C., Mônico, D. A., & Tedesco, A. C. (2020). Implications of dichlorofluorescein photoinstability for detection of UVA-induced oxidative stress in fibroblasts and keratinocyte cells. Photochemical & Photobiological Sciences, 19( 1), 40-48. doi:10.1039/c9pp00415g
NLM
Souza C, Mônico DA, Tedesco AC. Implications of dichlorofluorescein photoinstability for detection of UVA-induced oxidative stress in fibroblasts and keratinocyte cells [Internet]. Photochemical & Photobiological Sciences. 2020 ; 19( 1): 40-48.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1039/c9pp00415g
Vancouver
Souza C, Mônico DA, Tedesco AC. Implications of dichlorofluorescein photoinstability for detection of UVA-induced oxidative stress in fibroblasts and keratinocyte cells [Internet]. Photochemical & Photobiological Sciences. 2020 ; 19( 1): 40-48.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1039/c9pp00415g
Artigo de periodico
Antioxidant activity of an aqueous leaf extract from Uncaria tomentosa and its major alkaloids mitraphylline and isomitraphylline in Caenorhabditis elegans (2019)
Azevedo, Bruna C.; Roxo, Mariana; Borges, Marcos de Carvalho; Peixoto, Herbenya; Crevelin, Eduardo José ; Bertoni, Bianca W.; Contini, Silvia H. T.; Lopes, Adriana A.; França, Suzelei C.; Pereira, Ana M. S.; Wink, Michael
Fonte: Molecules. Unidades: FMRP, FFCLRP
Assuntos: ANTIOXIDANTES, ESTRESSE OXIDATIVO, GENTIANALES, EXTRATOS (FORMAS FARMACÊUTICAS), ALCALOIDES
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ABNT
AZEVEDO, Bruna C. et al. Antioxidant activity of an aqueous leaf extract from Uncaria tomentosa and its major alkaloids mitraphylline and isomitraphylline in Caenorhabditis elegans. Molecules, v. 24, n. 18, p. [15] , 2019Tradução . . Disponível em: https://doi.org/10.3390/molecules24183299. Acesso em: 11 nov. 2024.
APA
Azevedo, B. C., Roxo, M., Borges, M. de C., Peixoto, H., Crevelin, E. J., Bertoni, B. W., et al. (2019). Antioxidant activity of an aqueous leaf extract from Uncaria tomentosa and its major alkaloids mitraphylline and isomitraphylline in Caenorhabditis elegans. Molecules, 24( 18), [15] . doi:10.3390/molecules24183299
NLM
Azevedo BC, Roxo M, Borges M de C, Peixoto H, Crevelin EJ, Bertoni BW, Contini SHT, Lopes AA, França SC, Pereira AMS, Wink M. Antioxidant activity of an aqueous leaf extract from Uncaria tomentosa and its major alkaloids mitraphylline and isomitraphylline in Caenorhabditis elegans [Internet]. Molecules. 2019 ; 24( 18): [15] .[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/molecules24183299
Vancouver
Azevedo BC, Roxo M, Borges M de C, Peixoto H, Crevelin EJ, Bertoni BW, Contini SHT, Lopes AA, França SC, Pereira AMS, Wink M. Antioxidant activity of an aqueous leaf extract from Uncaria tomentosa and its major alkaloids mitraphylline and isomitraphylline in Caenorhabditis elegans [Internet]. Molecules. 2019 ; 24( 18): [15] .[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/molecules24183299
Artigo de periodico
Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS (2018)
Ceron, Carla S.; Vale, Gabriel T. do; Simplicio, Janaina A.; Ricci, Sthefany T.; Martinis, Bruno Spinosa de; Freitas, Andressa de; Tirapelli, Carlos Renato
Fonte: European Journal of Pharmacology. Unidades: FFCLRP, EERP
Assuntos: ESTRESSE OXIDATIVO, ETANOL, CITOCINAS, SEPSE, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CERON, Carla S. et al. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS. European Journal of Pharmacology, v. 825, p. 39-47, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2018.02.008. Acesso em: 11 nov. 2024.
APA
Ceron, C. S., Vale, G. T. do, Simplicio, J. A., Ricci, S. T., Martinis, B. S. de, Freitas, A. de, & Tirapelli, C. R. (2018). Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS. European Journal of Pharmacology, 825, 39-47. doi:10.1016/j.ejphar.2018.02.008
NLM
Ceron CS, Vale GT do, Simplicio JA, Ricci ST, Martinis BS de, Freitas A de, Tirapelli CR. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS [Internet]. European Journal of Pharmacology. 2018 ; 825 39-47.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.ejphar.2018.02.008
Vancouver
Ceron CS, Vale GT do, Simplicio JA, Ricci ST, Martinis BS de, Freitas A de, Tirapelli CR. Chronic ethanol consumption increases vascular oxidative stress and the mortality induced by sub-lethal sepsis: potential role of iNOS [Internet]. European Journal of Pharmacology. 2018 ; 825 39-47.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.ejphar.2018.02.008
Artigo de periodico
The labile iron pool attenuates peroxynitrite-dependent damage and can no longer be considered solely a pro-oxidative cellular iron source (2018)
Damasceno, Fernando Cruvinel; Condeles, André Luís; Lopes, Angélica Kodama Bueno; Facci, Romulo Rodrigues; Linares, Edlaine ; Truzzi, Daniela Ramos ; Augusto, Ohara ; Toledo Junior, José Carlos
Fonte: Journal of Biological Chemistry. Unidades: IQ, FFCLRP
Assuntos: ÓXIDO NÍTRICO, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAMASCENO, Fernando Cruvinel et al. The labile iron pool attenuates peroxynitrite-dependent damage and can no longer be considered solely a pro-oxidative cellular iron source. Journal of Biological Chemistry, v. 293, p. 8530-8542 : + Supplementary materials ( S1-S13), 2018Tradução . . Disponível em: https://doi.org/10.1074/jbc.RA117.000883. Acesso em: 11 nov. 2024.
APA
Damasceno, F. C., Condeles, A. L., Lopes, A. K. B., Facci, R. R., Linares, E., Truzzi, D. R., et al. (2018). The labile iron pool attenuates peroxynitrite-dependent damage and can no longer be considered solely a pro-oxidative cellular iron source. Journal of Biological Chemistry, 293, 8530-8542 : + Supplementary materials ( S1-S13). doi:10.1074/jbc.RA117.000883
NLM
Damasceno FC, Condeles AL, Lopes AKB, Facci RR, Linares E, Truzzi DR, Augusto O, Toledo Junior JC. The labile iron pool attenuates peroxynitrite-dependent damage and can no longer be considered solely a pro-oxidative cellular iron source [Internet]. Journal of Biological Chemistry. 2018 ; 293 8530-8542 : + Supplementary materials ( S1-S13).[citado 2024 nov. 11 ] Available from: https://doi.org/10.1074/jbc.RA117.000883
Vancouver
Damasceno FC, Condeles AL, Lopes AKB, Facci RR, Linares E, Truzzi DR, Augusto O, Toledo Junior JC. The labile iron pool attenuates peroxynitrite-dependent damage and can no longer be considered solely a pro-oxidative cellular iron source [Internet]. Journal of Biological Chemistry. 2018 ; 293 8530-8542 : + Supplementary materials ( S1-S13).[citado 2024 nov. 11 ] Available from: https://doi.org/10.1074/jbc.RA117.000883
Artigo de periodico
Nebivolol prevents vascular oxidative stress and hypertension in rats chronically treated with ethanol (2018)
Vale, Gabriel T. do; Simplicio, Janaina A.; Gonzaga, Natália A.; Yokota, Rodrigo; Ribeiro, Amanda A.; Casarini, Dulce Elena; De Martinis, Bruno Spinosa; Tirapelli, Carlos Renato
Fonte: Atherosclerosis. Unidades: FFCLRP, EERP
Assuntos: ETANOL, HIPERTENSÃO, ESTRESSE OXIDATIVO, RECEPTORES ADRENÉRGICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VALE, Gabriel T. do et al. Nebivolol prevents vascular oxidative stress and hypertension in rats chronically treated with ethanol. Atherosclerosis, v. 274, p. 67-76, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.atherosclerosis.2018.04.041. Acesso em: 11 nov. 2024.
APA
Vale, G. T. do, Simplicio, J. A., Gonzaga, N. A., Yokota, R., Ribeiro, A. A., Casarini, D. E., et al. (2018). Nebivolol prevents vascular oxidative stress and hypertension in rats chronically treated with ethanol. Atherosclerosis, 274, 67-76. doi:10.1016/j.atherosclerosis.2018.04.041
NLM
Vale GT do, Simplicio JA, Gonzaga NA, Yokota R, Ribeiro AA, Casarini DE, De Martinis BS, Tirapelli CR. Nebivolol prevents vascular oxidative stress and hypertension in rats chronically treated with ethanol [Internet]. Atherosclerosis. 2018 ; 274 67-76.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.atherosclerosis.2018.04.041
Vancouver
Vale GT do, Simplicio JA, Gonzaga NA, Yokota R, Ribeiro AA, Casarini DE, De Martinis BS, Tirapelli CR. Nebivolol prevents vascular oxidative stress and hypertension in rats chronically treated with ethanol [Internet]. Atherosclerosis. 2018 ; 274 67-76.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.atherosclerosis.2018.04.041
Artigo de periodico
Ethanol withdrawal increases blood pressure and vascular oxidative stress: a role for angiotensin type 1 receptors (2018)
Gonzaga, Natália A.; Vale, Gabriel T. do; Parente, Juliana M.; Yokota, Rodrigo; De Martinis, Bruno Spinosa; Casarini, Dulce E.; Castro, Michele Mazzaron de; Tirapelli, Carlos Renato
Fonte: Journal of the American Society of Hypertension. Unidades: FFCLRP, FMRP, EERP
Assuntos: ETANOL, HIPERTENSÃO, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO, RECEPTORES DE ANGIOTENSINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONZAGA, Natália A. et al. Ethanol withdrawal increases blood pressure and vascular oxidative stress: a role for angiotensin type 1 receptors. Journal of the American Society of Hypertension, v. 12, n. 7, p. 561-573, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jash.2018.03.012. Acesso em: 11 nov. 2024.
APA
Gonzaga, N. A., Vale, G. T. do, Parente, J. M., Yokota, R., De Martinis, B. S., Casarini, D. E., et al. (2018). Ethanol withdrawal increases blood pressure and vascular oxidative stress: a role for angiotensin type 1 receptors. Journal of the American Society of Hypertension, 12( 7), 561-573. doi:10.1016/j.jash.2018.03.012
NLM
Gonzaga NA, Vale GT do, Parente JM, Yokota R, De Martinis BS, Casarini DE, Castro MM de, Tirapelli CR. Ethanol withdrawal increases blood pressure and vascular oxidative stress: a role for angiotensin type 1 receptors [Internet]. Journal of the American Society of Hypertension. 2018 ; 12( 7): 561-573.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.jash.2018.03.012
Vancouver
Gonzaga NA, Vale GT do, Parente JM, Yokota R, De Martinis BS, Casarini DE, Castro MM de, Tirapelli CR. Ethanol withdrawal increases blood pressure and vascular oxidative stress: a role for angiotensin type 1 receptors [Internet]. Journal of the American Society of Hypertension. 2018 ; 12( 7): 561-573.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.jash.2018.03.012
Artigo de periodico
Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species (2017)
Leite, Letícia N.; Vale, Gabriel T. do; Simplicio, Janaína A.; De Martinis, Bruno Spinosa; Carneiro, Fernando Silva; Tirapelli, Carlos Renato
Fonte: European Journal of Pharmacology. Unidades: EERP, FFCLRP, FMRP
Assuntos: ETANOL, ESTRESSE OXIDATIVO, DISFUNÇÃO ERÉTIL, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEITE, Letícia N. et al. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species. European Journal of Pharmacology, v. 804, p. 82-93, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2017.03.024. Acesso em: 11 nov. 2024.
APA
Leite, L. N., Vale, G. T. do, Simplicio, J. A., De Martinis, B. S., Carneiro, F. S., & Tirapelli, C. R. (2017). Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species. European Journal of Pharmacology, 804, 82-93. doi:10.1016/j.ejphar.2017.03.024
NLM
Leite LN, Vale GT do, Simplicio JA, De Martinis BS, Carneiro FS, Tirapelli CR. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species [Internet]. European Journal of Pharmacology. 2017 ; 804 82-93.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.ejphar.2017.03.024
Vancouver
Leite LN, Vale GT do, Simplicio JA, De Martinis BS, Carneiro FS, Tirapelli CR. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species [Internet]. European Journal of Pharmacology. 2017 ; 804 82-93.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.ejphar.2017.03.024
Artigo de periodico
NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature (2016)
Marchi, Kátia Colombo; Ceron, Carla Speroni; Muniz, Jaqueline J.; De Martinis, Bruno Spinosa; Tanus-Santos, José Eduardo; Tirapelli, Carlos Renato
Fonte: Alcohol and Alcoholism. Unidades: FFCLRP, FMRP, EERP
Assuntos: HIPERTENSÃO, ESTRESSE OXIDATIVO, ETANOL, ENDOTÉLIO VASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARCHI, Kátia Colombo et al. NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature. Alcohol and Alcoholism, v. 51, n. 5, p. 522-534, 2016Tradução . . Disponível em: https://doi.org/10.1093/alcalc/agw043. Acesso em: 11 nov. 2024.
APA
Marchi, K. C., Ceron, C. S., Muniz, J. J., De Martinis, B. S., Tanus-Santos, J. E., & Tirapelli, C. R. (2016). NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature. Alcohol and Alcoholism, 51( 5), 522-534. doi:10.1093/alcalc/agw043
NLM
Marchi KC, Ceron CS, Muniz JJ, De Martinis BS, Tanus-Santos JE, Tirapelli CR. NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature [Internet]. Alcohol and Alcoholism. 2016 ; 51( 5): 522-534.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1093/alcalc/agw043
Vancouver
Marchi KC, Ceron CS, Muniz JJ, De Martinis BS, Tanus-Santos JE, Tirapelli CR. NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature [Internet]. Alcohol and Alcoholism. 2016 ; 51( 5): 522-534.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1093/alcalc/agw043
Artigo de periodico
Ethanol withdrawal increases oxidative stress and reduces nitric oxide bioavailability in the vasculature of rats (2015)
Gonzaga, Natália de Almeida; Mecawi, André S.; Antunes-Rodrigues, José; De Martinis, Bruno Spinosa; Padovan, Cláudia Maria; Tirapelli, Carlos Renato
Fonte: Alcohol. Unidades: FMRP, FFCLRP, EERP
Assuntos: HIPERTENSÃO, ESTRESSE OXIDATIVO, ETANOL, ÓXIDO NÍTRICO, SISTEMA CARDIOVASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONZAGA, Natália de Almeida et al. Ethanol withdrawal increases oxidative stress and reduces nitric oxide bioavailability in the vasculature of rats. Alcohol, v. 49, n. 1, p. 47–56, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.alcohol.2014.12.001. Acesso em: 11 nov. 2024.
APA
Gonzaga, N. de A., Mecawi, A. S., Antunes-Rodrigues, J., De Martinis, B. S., Padovan, C. M., & Tirapelli, C. R. (2015). Ethanol withdrawal increases oxidative stress and reduces nitric oxide bioavailability in the vasculature of rats. Alcohol, 49( 1), 47–56. doi:10.1016/j.alcohol.2014.12.001
NLM
Gonzaga N de A, Mecawi AS, Antunes-Rodrigues J, De Martinis BS, Padovan CM, Tirapelli CR. Ethanol withdrawal increases oxidative stress and reduces nitric oxide bioavailability in the vasculature of rats [Internet]. Alcohol. 2015 ; 49( 1): 47–56.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.alcohol.2014.12.001
Vancouver
Gonzaga N de A, Mecawi AS, Antunes-Rodrigues J, De Martinis BS, Padovan CM, Tirapelli CR. Ethanol withdrawal increases oxidative stress and reduces nitric oxide bioavailability in the vasculature of rats [Internet]. Alcohol. 2015 ; 49( 1): 47–56.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.alcohol.2014.12.001

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