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Artigo de periodico
Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 (2023)
Formiga, Rodrigo de Oliveira; Amaral, Flávia C.; Souza, Camila F.; Mendes, Daniel A. G. B.; Wanderley, Carlos Wagner de Souza ; Lorenzini, Cristina B.; Santos, Adara A.; Antônia, Juliana; Faria, Lucas F.; Natale, Caio C.; Paula, Nicholas M.; Cunha, Fernando de Queiroz
Fonte: British Journal of Pharmacology. Unidade: FMRP
Assuntos: COVID-19, LIPOPOLISSACARÍDEOS, METALOPROTEINASES, NEUTRÓFILOS, ESPÉCIES REATIVAS DE OXIGÊNIO, SEPSE
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ABNT
FORMIGA, Rodrigo de Oliveira et al. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, v. 180, n. 11, p. 1460-1481, 2023Tradução . . Disponível em: https://doi.org/10.1111/bph.16013. Acesso em: 02 nov. 2024.
APA
Formiga, R. de O., Amaral, F. C., Souza, C. F., Mendes, D. A. G. B., Wanderley, C. W. de S., Lorenzini, C. B., et al. (2023). Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19. British Journal of Pharmacology, 180( 11), 1460-1481. doi:10.1111/bph.16013
NLM
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1111/bph.16013
Vancouver
Formiga R de O, Amaral FC, Souza CF, Mendes DAGB, Wanderley CW de S, Lorenzini CB, Santos AA, Antônia J, Faria LF, Natale CC, Paula NM, Cunha F de Q. Neuraminidase is a host‐directed approach to regulate neutrophil responses in sepsis and COVID‐19 [Internet]. British Journal of Pharmacology. 2023 ; 180( 11): 1460-1481.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1111/bph.16013
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Fonte: Inflammation Research. Unidade: FMRP
Assuntos: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 02 nov. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Fonte: The Journal of Infectious Diseases. Unidade: FMRP
Assuntos: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 02 nov. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico-carta/editorial
Sepsis and COVID-19 [Editorial]: Cross-Talk in Signaling Pathways and in Therapeutic Perspectives. (2022)
Salomão, Reinaldo; Cunha, Fernando de Queiroz ; Dal-Pizzol, Felipe
Fonte: Frontiers in Medicine. Unidade: FMRP
Assuntos: SEPSE, COVID-19, EPIGÊNESE GENÉTICA, BIOMARCADORES
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ABNT
SALOMÃO, Reinaldo e CUNHA, Fernando de Queiroz e DAL-PIZZOL, Felipe. Sepsis and COVID-19 [Editorial]: Cross-Talk in Signaling Pathways and in Therapeutic Perspectives. Frontiers in Medicine. Lausanne: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.3389/fmed.2022.917792. Acesso em: 02 nov. 2024. , 2022
APA
Salomão, R., Cunha, F. de Q., & Dal-Pizzol, F. (2022). Sepsis and COVID-19 [Editorial]: Cross-Talk in Signaling Pathways and in Therapeutic Perspectives. Frontiers in Medicine. Lausanne: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.3389/fmed.2022.917792
NLM
Salomão R, Cunha F de Q, Dal-Pizzol F. Sepsis and COVID-19 [Editorial]: Cross-Talk in Signaling Pathways and in Therapeutic Perspectives. [Internet]. Frontiers in Medicine. 2022 ; 9 1-2.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3389/fmed.2022.917792
Vancouver
Salomão R, Cunha F de Q, Dal-Pizzol F. Sepsis and COVID-19 [Editorial]: Cross-Talk in Signaling Pathways and in Therapeutic Perspectives. [Internet]. Frontiers in Medicine. 2022 ; 9 1-2.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3389/fmed.2022.917792
Artigo de periodico
Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis (2021)
Valle, Gabriel Tavares do; Ricci, Sthefany Teodoro; Silva, Alessandra Oliveira; Tirapelli, Carlos Renato ; Ceron, Carla Speroni
Fonte: Canadian Journal of Physiology and Pharmacology. Unidade: EERP
Assuntos: ANIMAIS, PERÓXIDO DE HIDROGÊNIO, RATOS, ESTRESSE OXIDATIVO, SEPSE
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ABNT
VALLE, Gabriel Tavares do et al. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis. Canadian Journal of Physiology and Pharmacology, v. 99, n. 7, p. 699-707, 2021Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2020-0564. Acesso em: 02 nov. 2024.
APA
Valle, G. T. do, Ricci, S. T., Silva, A. O., Tirapelli, C. R., & Ceron, C. S. (2021). Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis. Canadian Journal of Physiology and Pharmacology, 99( 7), 699-707. doi:10.1139/cjpp-2020-0564
NLM
Valle GT do, Ricci ST, Silva AO, Tirapelli CR, Ceron CS. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 7): 699-707.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1139/cjpp-2020-0564
Vancouver
Valle GT do, Ricci ST, Silva AO, Tirapelli CR, Ceron CS. Ethanol consumption increases renal dysfunction and mortality in a mice model of sub-lethal sepsis [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 7): 699-707.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1139/cjpp-2020-0564
Artigo de periodico
Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats (2019)
Awata, Wanessa Mayumi Carvalho; Gonzaga, Natália de Almeida; Borges, Vanessa de Fátima; Silva, Carla B. P; Tanus-Santos, José Eduardo ; Cunha, Fernando de Queiroz ; Tirapelli, Carlos Renato
Fonte: European Journal of Pharmacology. Unidades: FMRP, EERP
Assuntos: SEPSE, ÓXIDO NÍTRICO
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ABNT
AWATA, Wanessa Mayumi Carvalho et al. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, v. 863, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2019.172706. Acesso em: 02 nov. 2024.
APA
Awata, W. M. C., Gonzaga, N. de A., Borges, V. de F., Silva, C. B. P., Tanus-Santos, J. E., Cunha, F. de Q., & Tirapelli, C. R. (2019). Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats. European Journal of Pharmacology, 863. doi:10.1016/j.ejphar.2019.172706
NLM
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Vancouver
Awata WMC, Gonzaga N de A, Borges V de F, Silva CBP, Tanus-Santos JE, Cunha F de Q, Tirapelli CR. Perivascular adipose tissue contributes to lethal sepsis-induced vasoplegia in rats [Internet]. European Journal of Pharmacology. 2019 ; 863[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2019.172706
Artigo de periodico
Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms (2019)
Nakashima, Marcelo A.; Silva, Carla Beatriz Pereira da ; Gonzaga, Natália de Almeida; Simplicio, Janaina A.; Omoto, Ana Carolina Mieko ; Tirapelli, Luis Fernando; Tanus-Santos, José Eduardo; Tirapelli, Carlos Renato
Fonte: Cytokine. Unidades: FMRP, EERP
Assuntos: ESTRESSE OXIDATIVO, ETANOL, CITOTOXINAS, SEPSE, INTERLEUCINAS
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ABNT
NAKASHIMA, Marcelo A. et al. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, v. 121, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2019.154734. Acesso em: 02 nov. 2024.
APA
Nakashima, M. A., Silva, C. B. P. da, Gonzaga, N. de A., Simplicio, J. A., Omoto, A. C. M., Tirapelli, L. F., et al. (2019). Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms. Cytokine, 121. doi:10.1016/j.cyto.2019.154734
NLM
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Vancouver
Nakashima MA, Silva CBP da, Gonzaga N de A, Simplicio JA, Omoto ACM, Tirapelli LF, Tanus-Santos JE, Tirapelli CR. Chronic ethanol consumption increases reactive oxygen species generation and the synthesis of pro-inflammatory proteins in the heart through TNFR1-dependent mechanisms [Internet]. Cytokine. 2019 ; 121[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.cyto.2019.154734
Artigo de periodico
Factors influencing awareness of healthcare providers on maternal sepsis: a mixed-methods approach (2019)
Brizuela, Vanessa ; Bonet, Mercedes; Souza, João Paulo Dias de; Tunçalp, Özge; Viswanath, Kasisomayajula; Langer, Ana
Fonte: BMC Public Health. Unidade: FMRP
Assuntos: SEPSE, SERVIÇOS DE SAÚDE, MORTALIDADE MATERNA
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ABNT
BRIZUELA, Vanessa et al. Factors influencing awareness of healthcare providers on maternal sepsis: a mixed-methods approach. BMC Public Health, v. 19, p. [11] , 2019Tradução . . Disponível em: https://doi.org/10.1186/s12889-019-6920-0. Acesso em: 02 nov. 2024.
APA
Brizuela, V., Bonet, M., Souza, J. P. D. de, Tunçalp, Ö., Viswanath, K., & Langer, A. (2019). Factors influencing awareness of healthcare providers on maternal sepsis: a mixed-methods approach. BMC Public Health, 19, [11] . doi:10.1186/s12889-019-6920-0
NLM
Brizuela V, Bonet M, Souza JPD de, Tunçalp Ö, Viswanath K, Langer A. Factors influencing awareness of healthcare providers on maternal sepsis: a mixed-methods approach [Internet]. BMC Public Health. 2019 ; 19 [11] .[citado 2024 nov. 02 ] Available from: https://doi.org/10.1186/s12889-019-6920-0
Vancouver
Brizuela V, Bonet M, Souza JPD de, Tunçalp Ö, Viswanath K, Langer A. Factors influencing awareness of healthcare providers on maternal sepsis: a mixed-methods approach [Internet]. BMC Public Health. 2019 ; 19 [11] .[citado 2024 nov. 02 ] Available from: https://doi.org/10.1186/s12889-019-6920-0
Artigo de periodico
HSP-target of therapeutic agents in sepsis treatment (2019)
Vulczak, Anderson ; Catalão, Carlos Henrique Rocha ; Freitas, Luis Alexandre Pedro de; Rocha, Maria José Alves da
Fonte: International Journal of Molecular Sciences. Unidades: FCFRP, FORP, FMRP
Assuntos: SEPSE, INFLAMAÇÃO, PROTEÍNAS DO CHOQUE TÉRMICO, SISTEMA IMUNE, TERAPÊUTICA MÉDICA
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ABNT
VULCZAK, Anderson et al. HSP-target of therapeutic agents in sepsis treatment. International Journal of Molecular Sciences, v. 20, n. 17, p. [16] , 2019Tradução . . Disponível em: https://doi.org/10.3390/ijms20174255. Acesso em: 02 nov. 2024.
APA
Vulczak, A., Catalão, C. H. R., Freitas, L. A. P. de, & Rocha, M. J. A. da. (2019). HSP-target of therapeutic agents in sepsis treatment. International Journal of Molecular Sciences, 20( 17), [16] . doi:10.3390/ijms20174255
NLM
Vulczak A, Catalão CHR, Freitas LAP de, Rocha MJA da. HSP-target of therapeutic agents in sepsis treatment [Internet]. International Journal of Molecular Sciences. 2019 ; 20( 17): [16] .[citado 2024 nov. 02 ] Available from: https://doi.org/10.3390/ijms20174255
Vancouver
Vulczak A, Catalão CHR, Freitas LAP de, Rocha MJA da. HSP-target of therapeutic agents in sepsis treatment [Internet]. International Journal of Molecular Sciences. 2019 ; 20( 17): [16] .[citado 2024 nov. 02 ] Available from: https://doi.org/10.3390/ijms20174255
Artigo de periodico
Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis (2013)
Czaikoski, Paula Giselle; Nascimento, Daniele Carvalho; Sônego, Fabiane; Freitas, Andressa de; Turato, Walter Miguel; Carvalho, Michel A. de; Santos, Raquel Souza; Oliveira, Gisele Pena de; Samary, Cynthia dos Santos; Tefe-Silva, Cristiane; Alves Filho, José Carlos Farias; Ferreira, Sérgio Henrique; Rossi, Marcos Antonio; Rocco, Patricia Rieken Macedo; Spiller, Fernando; Cunha, Fernando de Queiroz
Fonte: Shock. Unidades: FCF, FMRP
Assuntos: OXIGENASE, INIBIDORES DE ENZIMAS, NEUTRÓFILOS, SEPSE, PNEUMONIA
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ABNT
CZAIKOSKI, Paula Giselle et al. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis. Shock, v. 39, n. 4, p. 389-396, 2013Tradução . . Disponível em: https://doi.org/10.1097/SHK.0b013e31828bbcf9. Acesso em: 02 nov. 2024.
APA
Czaikoski, P. G., Nascimento, D. C., Sônego, F., Freitas, A. de, Turato, W. M., Carvalho, M. A. de, et al. (2013). Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis. Shock, 39( 4), 389-396. doi:10.1097/SHK.0b013e31828bbcf9
NLM
Czaikoski PG, Nascimento DC, Sônego F, Freitas A de, Turato WM, Carvalho MA de, Santos RS, Oliveira GP de, Samary C dos S, Tefe-Silva C, Alves Filho JCF, Ferreira SH, Rossi MA, Rocco PRM, Spiller F, Cunha F de Q. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis [Internet]. Shock. 2013 ; 39( 4): 389-396.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/SHK.0b013e31828bbcf9
Vancouver
Czaikoski PG, Nascimento DC, Sônego F, Freitas A de, Turato WM, Carvalho MA de, Santos RS, Oliveira GP de, Samary C dos S, Tefe-Silva C, Alves Filho JCF, Ferreira SH, Rossi MA, Rocco PRM, Spiller F, Cunha F de Q. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis [Internet]. Shock. 2013 ; 39( 4): 389-396.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/SHK.0b013e31828bbcf9
Artigo de periodico
Cardiac involvement and treatment-related mortality after non-myeloablative haemopoietic stem-cell transplantation with unselected autologous peripheral blood for patients with systemic sclerosis: a retrospective analysi (2013)
Burt, Richard K; Oliveira, Maria Carolina; Shah, Sanjiv J; Moraes, Daniela A; Simoes, Belinda Pinto; Gheorghiade, Mihai; Schroeder, James; Ruderman, Eric; Farge, Dominique; Chai, Z. Jessie; Marjanovic, Zora; Jain, Sandeep; Morgan, Amy; Milanetti, Francesca; Han, Xiaoqiang; Jovanovic, Borko; Helenowski, Irene B.; Voltarelli, Julio Cesar
Fonte: The Lancet. Unidade: FMRP
Assuntos: CÉLULAS-TRONCO, TRANSPLANTES, SEPSE, PERICARDITE
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ABNT
BURT, Richard K et al. Cardiac involvement and treatment-related mortality after non-myeloablative haemopoietic stem-cell transplantation with unselected autologous peripheral blood for patients with systemic sclerosis: a retrospective analysi. The Lancet, v. 381, n. 9872, p. 1116-1124, 2013Tradução . . Disponível em: https://doi.org/10.1016/S0140-6736(12)62114-X. Acesso em: 02 nov. 2024.
APA
Burt, R. K., Oliveira, M. C., Shah, S. J., Moraes, D. A., Simoes, B. P., Gheorghiade, M., et al. (2013). Cardiac involvement and treatment-related mortality after non-myeloablative haemopoietic stem-cell transplantation with unselected autologous peripheral blood for patients with systemic sclerosis: a retrospective analysi. The Lancet, 381( 9872), 1116-1124. doi:10.1016/S0140-6736(12)62114-X
NLM
Burt RK, Oliveira MC, Shah SJ, Moraes DA, Simoes BP, Gheorghiade M, Schroeder J, Ruderman E, Farge D, Chai ZJ, Marjanovic Z, Jain S, Morgan A, Milanetti F, Han X, Jovanovic B, Helenowski IB, Voltarelli JC. Cardiac involvement and treatment-related mortality after non-myeloablative haemopoietic stem-cell transplantation with unselected autologous peripheral blood for patients with systemic sclerosis: a retrospective analysi [Internet]. The Lancet. 2013 ; 381( 9872): 1116-1124.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/S0140-6736(12)62114-X
Vancouver
Burt RK, Oliveira MC, Shah SJ, Moraes DA, Simoes BP, Gheorghiade M, Schroeder J, Ruderman E, Farge D, Chai ZJ, Marjanovic Z, Jain S, Morgan A, Milanetti F, Han X, Jovanovic B, Helenowski IB, Voltarelli JC. Cardiac involvement and treatment-related mortality after non-myeloablative haemopoietic stem-cell transplantation with unselected autologous peripheral blood for patients with systemic sclerosis: a retrospective analysi [Internet]. The Lancet. 2013 ; 381( 9872): 1116-1124.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/S0140-6736(12)62114-X
Artigo de periodico
Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation (2012)
Araújo, Alice V.; Ferezin, Camila Z.; Pereira, Amanda de C.; Rodrigues, Gerson J.; Grando, Marcella Daruge; Bonaventura, Daniella; Bendhack, Lusiane Maria
Fonte: Nitric Oxide. Unidade: FCFRP
Assuntos: ÓXIDO NÍTRICO, SEPSE, FARMACOLOGIA
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ABNT
ARAÚJO, Alice V. et al. Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation. Nitric Oxide, v. 27, n. 1, p. 59-66, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2012.04.005. Acesso em: 02 nov. 2024.
APA
Araújo, A. V., Ferezin, C. Z., Pereira, A. de C., Rodrigues, G. J., Grando, M. D., Bonaventura, D., & Bendhack, L. M. (2012). Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation. Nitric Oxide, 27( 1), 59-66. doi:10.1016/j.niox.2012.04.005
NLM
Araújo AV, Ferezin CZ, Pereira A de C, Rodrigues GJ, Grando MD, Bonaventura D, Bendhack LM. Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation [Internet]. Nitric Oxide. 2012 ; 27( 1): 59-66.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.niox.2012.04.005
Vancouver
Araújo AV, Ferezin CZ, Pereira A de C, Rodrigues GJ, Grando MD, Bonaventura D, Bendhack LM. Augmented nitric oxide production and up-regulation of endothelial nitric oxide synthase during cecal ligation and perforation [Internet]. Nitric Oxide. 2012 ; 27( 1): 59-66.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.niox.2012.04.005
Artigo de periodico
Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis (2011)
Paula Neto, Heitor Affonso de; Alves Filho, José Carlos Farias; Souto, Fabrício Oliveira; Spiller, Fernando; Amêndola, Rafael S.; Freitas, Andressa; Cunha, Fernando de Queiroz; Barja-Fidalgo, Christina
Fonte: Shock. Unidade: FMRP
Assuntos: UNIDADES DE TERAPIA INTENSIVA, SEPSE, INFECÇÃO HOSPITALAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAULA NETO, Heitor Affonso de et al. Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis. Shock, v. 35, n. 1, p. 17-27, 2011Tradução . . Acesso em: 02 nov. 2024.
APA
Paula Neto, H. A. de, Alves Filho, J. C. F., Souto, F. O., Spiller, F., Amêndola, R. S., Freitas, A., et al. (2011). Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis. Shock, 35( 1), 17-27.
NLM
Paula Neto HA de, Alves Filho JCF, Souto FO, Spiller F, Amêndola RS, Freitas A, Cunha F de Q, Barja-Fidalgo C. Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis. Shock. 2011 ; 35( 1): 17-27.[citado 2024 nov. 02 ]
Vancouver
Paula Neto HA de, Alves Filho JCF, Souto FO, Spiller F, Amêndola RS, Freitas A, Cunha F de Q, Barja-Fidalgo C. Inhibition of guanylyl cyclase restores neutrophil migration and maintains bactericidal activity increasing survival in sepsis. Shock. 2011 ; 35( 1): 17-27.[citado 2024 nov. 02 ]
Artigo de periodico
Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice (2011)
Spiller, Fernando; Costa, Carlotta; Souto, Fabrício Oliveira; Vinchi, Francesca; Mestriner, Fabiola Leslie Antunes Cardoso; Laure, Hélen Julie; Alves Filho, José C.; Freitas, Andressa; Rosa, José César; Ferreira, Sérgio Henrique; Altruda, Fiorella; Hirsch, Emilio; Greene, Lewis Joel; Tolosano, Emanuela; Cunha, Fernando de Queiroz
Fonte: American Journal of Respiratory and Critical Care Medicine. Unidade: FMRP
Assuntos: NEUTRÓFILOS, SEPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SPILLER, Fernando et al. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine, v. 183, n. 7, p. 922-931, 2011Tradução . . Acesso em: 02 nov. 2024.
APA
Spiller, F., Costa, C., Souto, F. O., Vinchi, F., Mestriner, F. L. A. C., Laure, H. J., et al. (2011). Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine, 183( 7), 922-931.
NLM
Spiller F, Costa C, Souto FO, Vinchi F, Mestriner FLAC, Laure HJ, Alves Filho JC, Freitas A, Rosa JC, Ferreira SH, Altruda F, Hirsch E, Greene LJ, Tolosano E, Cunha F de Q. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 7): 922-931.[citado 2024 nov. 02 ]
Vancouver
Spiller F, Costa C, Souto FO, Vinchi F, Mestriner FLAC, Laure HJ, Alves Filho JC, Freitas A, Rosa JC, Ferreira SH, Altruda F, Hirsch E, Greene LJ, Tolosano E, Cunha F de Q. Inhibition of neutrophil migration by hemopexin leads to increased mortality due to sepsis in mice. American Journal of Respiratory and Critical Care Medicine. 2011 ; 183( 7): 922-931.[citado 2024 nov. 02 ]
Artigo de periodico
Leukotriene synthesis inhibitor decreases vasopressin release in the early phase of sepsis (2011)
Martins, Thalita Freitas; Sorgi, Carlos Arterio; Faccioli, Lucia Helena; Rocha, Maria Jose Alves da
Fonte: Journal of Neuroimmunology. Unidades: FCFRP, FORP
Assuntos: LEUCOTRIENOS, VASOPRESSINAS, SEPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTINS, Thalita Freitas et al. Leukotriene synthesis inhibitor decreases vasopressin release in the early phase of sepsis. Journal of Neuroimmunology, v. 238, n. 1-2, p. 52-57, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.jneuroim.2011.08.001. Acesso em: 02 nov. 2024.
APA
Martins, T. F., Sorgi, C. A., Faccioli, L. H., & Rocha, M. J. A. da. (2011). Leukotriene synthesis inhibitor decreases vasopressin release in the early phase of sepsis. Journal of Neuroimmunology, 238( 1-2), 52-57. doi:10.1016/j.jneuroim.2011.08.001
NLM
Martins TF, Sorgi CA, Faccioli LH, Rocha MJA da. Leukotriene synthesis inhibitor decreases vasopressin release in the early phase of sepsis [Internet]. Journal of Neuroimmunology. 2011 ; 238( 1-2): 52-57.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.jneuroim.2011.08.001
Vancouver
Martins TF, Sorgi CA, Faccioli LH, Rocha MJA da. Leukotriene synthesis inhibitor decreases vasopressin release in the early phase of sepsis [Internet]. Journal of Neuroimmunology. 2011 ; 238( 1-2): 52-57.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.jneuroim.2011.08.001
Artigo de periodico
Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP) (2011)
Araújo, Alice Valença; Ferezin, Camila Zarinello; Rodrigues, Gerson Jhonatan; Lunardi, Claure Nain; Vercesi, Juliana Aparecida; Grando, Marcella Daruge; Bonaventura, Daniella; Bendhack, Lusiane Maria
Fonte: Vascualr Pharmacology. Unidade: FCFRP
Assuntos: ÓXIDO NÍTRICO, VASODILATAÇÃO, SEPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAÚJO, Alice Valença et al. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP). Vascualr Pharmacology, v. 54, n. 1-2, p. 44-51, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2010.12.002. Acesso em: 02 nov. 2024.
APA
Araújo, A. V., Ferezin, C. Z., Rodrigues, G. J., Lunardi, C. N., Vercesi, J. A., Grando, M. D., et al. (2011). Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP). Vascualr Pharmacology, 54( 1-2), 44-51. doi:10.1016/j.vph.2010.12.002
NLM
Araújo AV, Ferezin CZ, Rodrigues GJ, Lunardi CN, Vercesi JA, Grando MD, Bonaventura D, Bendhack LM. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP) [Internet]. Vascualr Pharmacology. 2011 ; 54( 1-2): 44-51.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.vph.2010.12.002
Vancouver
Araújo AV, Ferezin CZ, Rodrigues GJ, Lunardi CN, Vercesi JA, Grando MD, Bonaventura D, Bendhack LM. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP) [Internet]. Vascualr Pharmacology. 2011 ; 54( 1-2): 44-51.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.vph.2010.12.002
Artigo de periodico
Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death (2010)
Melo, Edielle S.; Goloubkova, Tatiana; Barbeiro, Denise F.; Gorjão, Renata; Vasconcelos, Dewton; Szabo, Csaba; Curi, Rui; Salgado, Thais Martins de Lima; Velasco, Irineu Tadeu; Soriano, Francisco Garcia
Fonte: Immunobiology. Unidades: FM, ICB, FMRP
Assuntos: ENDOTOXINAS, TOLERÂNCIA, EXPRESSÃO GÊNICA, LINFÓCITOS, APOPTOSE, CITOCINAS, SEPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MELO, Edielle S. et al. Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death. Immunobiology, v. 215, n. 6, p. 435-442, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2009.09.002. Acesso em: 02 nov. 2024.
APA
Melo, E. S., Goloubkova, T., Barbeiro, D. F., Gorjão, R., Vasconcelos, D., Szabo, C., et al. (2010). Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death. Immunobiology, 215( 6), 435-442. doi:10.1016/j.imbio.2009.09.002
NLM
Melo ES, Goloubkova T, Barbeiro DF, Gorjão R, Vasconcelos D, Szabo C, Curi R, Salgado TM de L, Velasco IT, Soriano FG. Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death [Internet]. Immunobiology. 2010 ; 215( 6): 435-442.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.imbio.2009.09.002
Vancouver
Melo ES, Goloubkova T, Barbeiro DF, Gorjão R, Vasconcelos D, Szabo C, Curi R, Salgado TM de L, Velasco IT, Soriano FG. Endotoxin tolerance: Selective alterations in gene expression and protection against lymphocyte death [Internet]. Immunobiology. 2010 ; 215( 6): 435-442.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.imbio.2009.09.002
Artigo de periodico
Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins (2010)
Celes, Mara Rúbia Nunes; Dueñas, Diego Torres; Prado, Cibele Maria; Campos, Érica Carolina; Moreira, Jorge Eduardo; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Fonte: Shock. Unidade: FMRP
Assuntos: SARCOLEMA, SEPSE, MIOCARDIOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CELES, Mara Rúbia Nunes et al. Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins. Shock, v. 33, n. 3, p. 322-331, 2010Tradução . . Acesso em: 02 nov. 2024.
APA
Celes, M. R. N., Dueñas, D. T., Prado, C. M., Campos, É. C., Moreira, J. E., Cunha, F. de Q., & Rossi, M. A. (2010). Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins. Shock, 33( 3), 322-331.
NLM
Celes MRN, Dueñas DT, Prado CM, Campos ÉC, Moreira JE, Cunha F de Q, Rossi MA. Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins. Shock. 2010 ; 33( 3): 322-331.[citado 2024 nov. 02 ]
Vancouver
Celes MRN, Dueñas DT, Prado CM, Campos ÉC, Moreira JE, Cunha F de Q, Rossi MA. Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins. Shock. 2010 ; 33( 3): 322-331.[citado 2024 nov. 02 ]
Artigo de periodico
Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis (2010)
Celes, Mara Rúbia Nunes; Malvestio, Lygia M.; Torres-Dueñas, Diego; Blefari, Valdecir; Campos, Érica Carolina; Ramos, Simone Gusmão; Prado, Cibele Maria; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Fonte: Laboratory Investigation. Unidade: FMRP
Assuntos: SARCOLEMA, SEPSE, MIOCARDIOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CELES, Mara Rúbia Nunes et al. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, v. 90, n. 4, p. 531-542, 2010Tradução . . Disponível em: https://doi.org/10.1186/cc9157. Acesso em: 02 nov. 2024.
APA
Celes, M. R. N., Malvestio, L. M., Torres-Dueñas, D., Blefari, V., Campos, É. C., Ramos, S. G., et al. (2010). Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, 90( 4), 531-542. doi:10.1186/cc9157
NLM
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1186/cc9157
Vancouver
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1186/cc9157
Artigo de periodico
Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis (2010)
Nascimento, Daniele C.; Alves-Filho, José Carlos; Sônego, Fabiane; Fukada, Sandra Yasuyo; Pereira, Marcelo S.; Benjamim, Cláudia; Zamboni, Dario Simões; Silva, João Santana da; Cunha, Fernando de Queiroz
Fonte: Critical Care Medicine. Unidade: FMRP
Assuntos: SEPSE, FÁRMACOS IMUNOSSUPRESSORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, Daniele C. et al. Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis. Critical Care Medicine, v. 38, n. 8, p. 1718-1725, 2010Tradução . . Disponível em: https://doi.org/10.1097/ccm.0b013e3181e78ad0. Acesso em: 02 nov. 2024.
APA
Nascimento, D. C., Alves-Filho, J. C., Sônego, F., Fukada, S. Y., Pereira, M. S., Benjamim, C., et al. (2010). Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis. Critical Care Medicine, 38( 8), 1718-1725. doi:10.1097/ccm.0b013e3181e78ad0
NLM
Nascimento DC, Alves-Filho JC, Sônego F, Fukada SY, Pereira MS, Benjamim C, Zamboni DS, Silva JS da, Cunha F de Q. Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis [Internet]. Critical Care Medicine. 2010 ; 38( 8): 1718-1725.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/ccm.0b013e3181e78ad0
Vancouver
Nascimento DC, Alves-Filho JC, Sônego F, Fukada SY, Pereira MS, Benjamim C, Zamboni DS, Silva JS da, Cunha F de Q. Role of regulatory T cells in long-term immune dysfunction associated with severe sepsis [Internet]. Critical Care Medicine. 2010 ; 38( 8): 1718-1725.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1097/ccm.0b013e3181e78ad0

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