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Artigo de periodico
Deletion of growth hormone receptor in hypothalamic neurons affects the adaptation capacity to aerobic exercise (2021)
Pedroso, João Alfredo Bolivar ; Santos, Lucas Brecht Palos dos; Furigo, Isadora Clivatti ; Spagnol, Alexandre Roveratti ; Wasinski, Frederick ; List, Edward O. ; Kopchick, John J.; Donato Junior, José
Source: Peptides. Unidade: ICB
Subjects: FISIOLOGIA, GLICEMIA, LEPTINA, METABOLISMO, HORMÔNIO DO CRESCIMENTO, HIPOTÁLAMO, ESTRESSE, EXERCÍCIOS DE RESISTÊNCIA MUSCULAR, CAMUNDONGOS
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PEDROSO, João Alfredo Bolivar et al. Deletion of growth hormone receptor in hypothalamic neurons affects the adaptation capacity to aerobic exercise. Peptides, v. 135, p. 10 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.peptides.2020.170426. Acesso em: 02 jun. 2024.
APA
Pedroso, J. A. B., Santos, L. B. P. dos, Furigo, I. C., Spagnol, A. R., Wasinski, F., List, E. O., et al. (2021). Deletion of growth hormone receptor in hypothalamic neurons affects the adaptation capacity to aerobic exercise. Peptides, 135, 10 . doi:10.1016/j.peptides.2020.170426
NLM
Pedroso JAB, Santos LBP dos, Furigo IC, Spagnol AR, Wasinski F, List EO, Kopchick JJ, Donato Junior J. Deletion of growth hormone receptor in hypothalamic neurons affects the adaptation capacity to aerobic exercise [Internet]. Peptides. 2021 ; 135 10 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.peptides.2020.170426
Vancouver
Pedroso JAB, Santos LBP dos, Furigo IC, Spagnol AR, Wasinski F, List EO, Kopchick JJ, Donato Junior J. Deletion of growth hormone receptor in hypothalamic neurons affects the adaptation capacity to aerobic exercise [Internet]. Peptides. 2021 ; 135 10 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.peptides.2020.170426
Artigo de periodico
Plasmodium vivax infection compromises reticulocyte stability (2021)
Clark, Martha A. ; Kanjee, Usheer ; Rangel, Gabriel W. ; Chery, Laura ; Mascarenhas, Anjali ; Gomes, Edwin; Rathod, Pradipsinh K. ; Brugnara, Carlo ; Ferreira, Marcelo Urbano ; Duraisingh, Manoj T.
Source: Nature Communications. Unidade: ICB
Subjects: PARASITOLOGIA, PLASMODIUM, MALÁRIA, CITOMETRIA DE FLUXO, ERITRÓCITOS, INFECÇÕES POR PROTOZOÁRIOS
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CLARK, Martha A. et al. Plasmodium vivax infection compromises reticulocyte stability. Nature Communications, v. 12, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41467-021-21886-x. Acesso em: 02 jun. 2024.
APA
Clark, M. A., Kanjee, U., Rangel, G. W., Chery, L., Mascarenhas, A., Gomes, E., et al. (2021). Plasmodium vivax infection compromises reticulocyte stability. Nature Communications, 12, 1-11. doi:10.1038/s41467-021-21886-x
NLM
Clark MA, Kanjee U, Rangel GW, Chery L, Mascarenhas A, Gomes E, Rathod PK, Brugnara C, Ferreira MU, Duraisingh MT. Plasmodium vivax infection compromises reticulocyte stability [Internet]. Nature Communications. 2021 ; 12 1-11.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41467-021-21886-x
Vancouver
Clark MA, Kanjee U, Rangel GW, Chery L, Mascarenhas A, Gomes E, Rathod PK, Brugnara C, Ferreira MU, Duraisingh MT. Plasmodium vivax infection compromises reticulocyte stability [Internet]. Nature Communications. 2021 ; 12 1-11.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41467-021-21886-x
Artigo de periodico
Metalloprotease ADAMTS-1 decreases cell migration and invasion modulating the spatiotemporal dynamics of Cdc42 activity (2021)
Lima, Maíra de Assis; Silva, Suély Vieira da; Garrido, Orlando Serrano; Hülsemann, Maren; Neres, Luana dos Santos; Manzaneque, Juan Carlos Rodríguez ; Hodgson, Louis ; Freitas, Vanessa Morais
Source: Cellular Signalling. Unidade: ICB
Subjects: BIOLOGIA CELULAR, METALOPROTEINASES, PROTEINASES, MATRIZ EXTRACELULAR, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS OVARIANAS, FIBROBLASTOS, PROLIFERAÇÃO CELULAR
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LIMA, Maíra de Assis et al. Metalloprotease ADAMTS-1 decreases cell migration and invasion modulating the spatiotemporal dynamics of Cdc42 activity. Cellular Signalling, v. 77, p. 15 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.cellsig.2020.109827. Acesso em: 02 jun. 2024.
APA
Lima, M. de A., Silva, S. V. da, Garrido, O. S., Hülsemann, M., Neres, L. dos S., Manzaneque, J. C. R., et al. (2021). Metalloprotease ADAMTS-1 decreases cell migration and invasion modulating the spatiotemporal dynamics of Cdc42 activity. Cellular Signalling, 77, 15 . doi:10.1016/j.cellsig.2020.109827
NLM
Lima M de A, Silva SV da, Garrido OS, Hülsemann M, Neres L dos S, Manzaneque JCR, Hodgson L, Freitas VM. Metalloprotease ADAMTS-1 decreases cell migration and invasion modulating the spatiotemporal dynamics of Cdc42 activity [Internet]. Cellular Signalling. 2021 ; 77 15 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.cellsig.2020.109827
Vancouver
Lima M de A, Silva SV da, Garrido OS, Hülsemann M, Neres L dos S, Manzaneque JCR, Hodgson L, Freitas VM. Metalloprotease ADAMTS-1 decreases cell migration and invasion modulating the spatiotemporal dynamics of Cdc42 activity [Internet]. Cellular Signalling. 2021 ; 77 15 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.cellsig.2020.109827
Artigo de periodico
miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation (2021)
Lima, Vanessa Morais; Liu, Jianming; Brandão, Bruna B. ; Lino, Caroline Antunes ; Silva, Camila Stephanie Balbino ; Ribeiro, Márcio Augusto Campos; Silva, Tiago Eugênio Oliveira da; Gregório, Caroline Cristiano Real ; Faria, Daniele de Paula ; Cederquist, Carly; Huang, Zhan-Peng; Hu, Xiaoyun ; Barreto-Chaves, Maria Luiza Morais ; Ferreira, Julio Cesar Batista ; Festuccia, William Tadeu Lara ; Mori, Marcelo A. ; Kahn, C. Ronald ; Wang, Da-Zhi ; Diniz, Gabriela Placoná
Source: Metabolism. Unidades: FM, ICB
Subjects: ANATOMIA, FISIOLOGIA, TECIDO ADIPOSO, DIETA, OBESIDADE, TERMOGÊNESE, METABOLISMO DE GORDURA, MICRORNAS, CAMUNDONGOS, ADIPOSIDADE
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LIMA, Vanessa Morais et al. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation. Metabolism, v. 117, p. 13 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2021.154723. Acesso em: 02 jun. 2024.
APA
Lima, V. M., Liu, J., Brandão, B. B., Lino, C. A., Silva, C. S. B., Ribeiro, M. A. C., et al. (2021). miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation. Metabolism, 117, 13 . doi:10.1016/j.metabol.2021.154723
NLM
Lima VM, Liu J, Brandão BB, Lino CA, Silva CSB, Ribeiro MAC, Silva TEO da, Gregório CCR, Faria D de P, Cederquist C, Huang Z-P, Hu X, Barreto-Chaves MLM, Ferreira JCB, Festuccia WTL, Mori MA, Kahn CR, Wang D-Z, Diniz GP. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation [Internet]. Metabolism. 2021 ; 117 13 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.metabol.2021.154723
Vancouver
Lima VM, Liu J, Brandão BB, Lino CA, Silva CSB, Ribeiro MAC, Silva TEO da, Gregório CCR, Faria D de P, Cederquist C, Huang Z-P, Hu X, Barreto-Chaves MLM, Ferreira JCB, Festuccia WTL, Mori MA, Kahn CR, Wang D-Z, Diniz GP. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation [Internet]. Metabolism. 2021 ; 117 13 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.metabol.2021.154723
Artigo de periodico
Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL (2020)
Wang, Jin-Lin ; Wang, Xiao‐Yu ; Wang, Deng‐Ke; Parker, Mark D. ; Musa-Aziz, Raif ; Popple, Jacob ; Guo, Yi‐Min; Min, Tian‐Xin; Xia, Tian; Tan, Min; Liu, Ying ; Boron, Walter F. ; Chen, Li‐Ming
Source: Journal of Physiology. Unidade: ICB
Subjects: FISIOLOGIA, AMÔNIA, HIPERTENSÃO, ACIDOSE, OÓCITOS, WESTERN BLOTTING, IMUNOFLUORESCÊNCIA EM ANIMAL, RATOS, ELETRÓLITOS
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WANG, Jin-Lin et al. Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL. Journal of Physiology, v. 598, n. 16, p. 3395-3415, 2020Tradução . . Disponível em: https://doi.org/10.1113/JP279009. Acesso em: 02 jun. 2024.
APA
Wang, J. -L., Wang, X. ‐Y., Wang, D. ‐K., Parker, M. D., Musa-Aziz, R., Popple, J., et al. (2020). Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL. Journal of Physiology, 598( 16), 3395-3415. doi:10.1113/JP279009
NLM
Wang J-L, Wang X‐Y, Wang D‐K, Parker MD, Musa-Aziz R, Popple J, Guo Y‐M, Min T‐X, Xia T, Tan M, Liu Y, Boron WF, Chen L‐M. Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL [Internet]. Journal of Physiology. 2020 ; 598( 16): 3395-3415.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1113/JP279009
Vancouver
Wang J-L, Wang X‐Y, Wang D‐K, Parker MD, Musa-Aziz R, Popple J, Guo Y‐M, Min T‐X, Xia T, Tan M, Liu Y, Boron WF, Chen L‐M. Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL [Internet]. Journal of Physiology. 2020 ; 598( 16): 3395-3415.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1113/JP279009
Artigo de periodico
Mast cells and basophils: from malevolent design to coevolutionary arms race (2020)
Nunes, Anderson de Sá ; Oliveira, Carlo José Freire de ; Ribeiro, José M.
Source: Trends in Parasitology. Unidade: ICB
Subjects: IMUNOLOGIA, ARTRÓPODES, BASÓFILOS, MASTÓCITOS, CÉLULAS SANGUÍNEAS
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NUNES, Anderson de Sá e OLIVEIRA, Carlo José Freire de e RIBEIRO, José M. Mast cells and basophils: from malevolent design to coevolutionary arms race. Trends in Parasitology, v. 36, n. 8, p. 655-659, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.pt.2020.05.003. Acesso em: 02 jun. 2024.
APA
Nunes, A. de S., Oliveira, C. J. F. de, & Ribeiro, J. M. (2020). Mast cells and basophils: from malevolent design to coevolutionary arms race. Trends in Parasitology, 36( 8), 655-659. doi:10.1016/j.pt.2020.05.003
NLM
Nunes A de S, Oliveira CJF de, Ribeiro JM. Mast cells and basophils: from malevolent design to coevolutionary arms race [Internet]. Trends in Parasitology. 2020 ; 36( 8): 655-659.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.pt.2020.05.003
Vancouver
Nunes A de S, Oliveira CJF de, Ribeiro JM. Mast cells and basophils: from malevolent design to coevolutionary arms race [Internet]. Trends in Parasitology. 2020 ; 36( 8): 655-659.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.pt.2020.05.003
Artigo de periodico
AHR is a Zika virus host factor and a candidate target for antiviral therapy (2020)
Giovannoni, Federico ; Bosch, Irene ; Polonio, Carolina Manganeli ; Torti, María F.; Wheeler, Michael A.; Li, Zhaorong ; Romorini, Leonardo ; Varela, María S. Rodriguez; Rothhammer, Veit ; Barroso, Andreia ; Tjon, Emily C.; Sanmarco, Liliana M.; Takenaka, Maisa C.; Modaresi, Seyed Mohamad Sadegh ; Vázquez, Cristina Gutiérrez; Zanluqui, Nágela Ghabdan ; Santos, Nilton Barreto dos ; Munhoz, Carolina Demarchi ; Wang, Zhongyan; Damonte, Elsa B.; Sherr, David; Gehrke, Lee ; Peron, Jean Pierre Schatzmann ; Garcia, Cybele C. ; Quintana, Francisco J.
Source: Nature Neuroscience. Unidades: ICB, FM
Subjects: IMUNOLOGIA, FARMACOLOGIA, ZIKA VÍRUS, VÍRUS, FLAVIVIRUS, MICROCEFALIA, TRANSCRIÇÃO GÊNICA, GENOMAS, HIDROCARBONETOS, INTERFERON TIPO I, ANTIVIRAIS, REPLICAÇÃO VIRAL, NANOPARTÍCULAS
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GIOVANNONI, Federico et al. AHR is a Zika virus host factor and a candidate target for antiviral therapy. Nature Neuroscience, v. 23, p. 939–951, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41593-020-0664-0. Acesso em: 02 jun. 2024.
APA
Giovannoni, F., Bosch, I., Polonio, C. M., Torti, M. F., Wheeler, M. A., Li, Z., et al. (2020). AHR is a Zika virus host factor and a candidate target for antiviral therapy. Nature Neuroscience, 23, 939–951. doi:10.1038/s41593-020-0664-0
NLM
Giovannoni F, Bosch I, Polonio CM, Torti MF, Wheeler MA, Li Z, Romorini L, Varela MSR, Rothhammer V, Barroso A, Tjon EC, Sanmarco LM, Takenaka MC, Modaresi SMS, Vázquez CG, Zanluqui NG, Santos NB dos, Munhoz CD, Wang Z, Damonte EB, Sherr D, Gehrke L, Peron JPS, Garcia CC, Quintana FJ. AHR is a Zika virus host factor and a candidate target for antiviral therapy [Internet]. Nature Neuroscience. 2020 ; 23 939–951.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41593-020-0664-0
Vancouver
Giovannoni F, Bosch I, Polonio CM, Torti MF, Wheeler MA, Li Z, Romorini L, Varela MSR, Rothhammer V, Barroso A, Tjon EC, Sanmarco LM, Takenaka MC, Modaresi SMS, Vázquez CG, Zanluqui NG, Santos NB dos, Munhoz CD, Wang Z, Damonte EB, Sherr D, Gehrke L, Peron JPS, Garcia CC, Quintana FJ. AHR is a Zika virus host factor and a candidate target for antiviral therapy [Internet]. Nature Neuroscience. 2020 ; 23 939–951.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41593-020-0664-0
Artigo de periodico
Molecular evidence of sustained urban malaria transmission in Amazonian Brazil, 2014–2015 (2020)
Salla, Lais Camoese ; Rodrigues, Priscila Thihara ; Corder, Rodrigo Malavazi ; Johansen, Igor Cavallini ; Ferreira, Marcelo Urbano
Source: Epidemiology and Infection. Unidade: ICB
Subjects: PARASITOLOGIA, MALÁRIA, PLASMODIUM, ESTRUTURA MOLECULAR (QUÍMICA TEÓRICA), GENÓTIPOS, INFECÇÕES POR PROTOZOÁRIOS, HAPLOTIPOS, CLONAGEM ANIMAL, POPULAÇÃO URBANA
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SALLA, Lais Camoese et al. Molecular evidence of sustained urban malaria transmission in Amazonian Brazil, 2014–2015. Epidemiology and Infection, v. 148, p. 8 , 2020Tradução . . Disponível em: https://doi.org/10.1017/S0950268820000515. Acesso em: 02 jun. 2024.
APA
Salla, L. C., Rodrigues, P. T., Corder, R. M., Johansen, I. C., & Ferreira, M. U. (2020). Molecular evidence of sustained urban malaria transmission in Amazonian Brazil, 2014–2015. Epidemiology and Infection, 148, 8 . doi:10.1017/S0950268820000515
NLM
Salla LC, Rodrigues PT, Corder RM, Johansen IC, Ferreira MU. Molecular evidence of sustained urban malaria transmission in Amazonian Brazil, 2014–2015 [Internet]. Epidemiology and Infection. 2020 ; 148 8 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1017/S0950268820000515
Vancouver
Salla LC, Rodrigues PT, Corder RM, Johansen IC, Ferreira MU. Molecular evidence of sustained urban malaria transmission in Amazonian Brazil, 2014–2015 [Internet]. Epidemiology and Infection. 2020 ; 148 8 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1017/S0950268820000515
Artigo de periodico
Melanin as a virulence factor in different species of genus Paracoccidioides (2020)
Emidio, Elúzia Castro Peres; Jiménez, Martha Eugenia Urán ; Silva, Leandro Buffoni Roque da ; Dias, Lucas dos Santos ; Doprado, Mariana; Nosanchuk, Joshua D. ; Taborda, Carlos Pelleschi
Source: Journal of Fungi. Unidade: ICB
Subjects: MICROBIOLOGIA, MELANINAS, PARACOCCIDIOIDES, VIRULÊNCIA, PARACOCCIDIOIDOMICOSE, PREDISPOSIÇÃO GENÉTICA PARA DOENÇA, MICOSES, FUNGOS TERMÓFILOS, FAGOCITOSE, LEVEDURAS, MACRÓFAGOS, CAMUNDONGOS
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EMIDIO, Elúzia Castro Peres et al. Melanin as a virulence factor in different species of genus Paracoccidioides. Journal of Fungi, v. 6, n. 4, p. 11 , 2020Tradução . . Disponível em: https://doi.org/10.3390/jof6040291. Acesso em: 02 jun. 2024.
APA
Emidio, E. C. P., Jiménez, M. E. U., Silva, L. B. R. da, Dias, L. dos S., Doprado, M., Nosanchuk, J. D., & Taborda, C. P. (2020). Melanin as a virulence factor in different species of genus Paracoccidioides. Journal of Fungi, 6( 4), 11 . doi:10.3390/jof6040291
NLM
Emidio ECP, Jiménez MEU, Silva LBR da, Dias L dos S, Doprado M, Nosanchuk JD, Taborda CP. Melanin as a virulence factor in different species of genus Paracoccidioides [Internet]. Journal of Fungi. 2020 ; 6( 4): 11 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3390/jof6040291
Vancouver
Emidio ECP, Jiménez MEU, Silva LBR da, Dias L dos S, Doprado M, Nosanchuk JD, Taborda CP. Melanin as a virulence factor in different species of genus Paracoccidioides [Internet]. Journal of Fungi. 2020 ; 6( 4): 11 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3390/jof6040291
Artigo de periodico
Leukotriene B4 licenses inflammasome activation to enhance skin host defense (2020)
Salina, Ana Carolina Guerta ; Brandt, Stephanie L.; Klopfenstein, Nathan; Blackman, Amondrea ; Bazzano, Júlia Miranda Ribeiro ; Nunes, Anderson de Sá ; Glosson, Nicole Byers ; Brodskyn, Claudia ; Tavares, Natalia Machado ; Silva, Icaro Bonyek Santos da ; Medeiros, Alexandra Ivo ; Serezani, C. Henrique
Source: Proceedings of the National Academy of Sciences. Unidades: ICB, FMRP
Subjects: IMUNOLOGIA, LEUCOTRIENOS, INFLAMAÇÃO, FAGÓCITOS, CITOCINAS, QUIMIOTAXIA, QUIMIOCINAS
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SALINA, Ana Carolina Guerta et al. Leukotriene B4 licenses inflammasome activation to enhance skin host defense. Proceedings of the National Academy of Sciences, v. 48, p. 30619-30627, 2020Tradução . . Disponível em: https://doi.org/10.1073/pnas.2002732117. Acesso em: 02 jun. 2024.
APA
Salina, A. C. G., Brandt, S. L., Klopfenstein, N., Blackman, A., Bazzano, J. M. R., Nunes, A. de S., et al. (2020). Leukotriene B4 licenses inflammasome activation to enhance skin host defense. Proceedings of the National Academy of Sciences, 48, 30619-30627. doi:10.1073/pnas.2002732117
NLM
Salina ACG, Brandt SL, Klopfenstein N, Blackman A, Bazzano JMR, Nunes A de S, Glosson NB, Brodskyn C, Tavares NM, Silva IBS da, Medeiros AI, Serezani CH. Leukotriene B4 licenses inflammasome activation to enhance skin host defense [Internet]. Proceedings of the National Academy of Sciences. 2020 ; 48 30619-30627.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1073/pnas.2002732117
Vancouver
Salina ACG, Brandt SL, Klopfenstein N, Blackman A, Bazzano JMR, Nunes A de S, Glosson NB, Brodskyn C, Tavares NM, Silva IBS da, Medeiros AI, Serezani CH. Leukotriene B4 licenses inflammasome activation to enhance skin host defense [Internet]. Proceedings of the National Academy of Sciences. 2020 ; 48 30619-30627.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1073/pnas.2002732117
Artigo de periodico
Comparative analysis of infection by Rickettsia rickettsii Sheila Smith and Taiaçu strains in a murine model (2020)
Esteves, Eliane Virgínia da Silva ; Fongsaran, Chanida ; Langohr, Ingeborg M. ; Riley, Sean P. ; Labruna, Marcelo Bahia ; Daffre, Sirlei ; Fogaça, Andréa Cristina ; Macaluso, Kevin R.
Source: Pathogens. Unidades: ICB, FMVZ
Subjects: PARASITOLOGIA, RICKETTSIOSES EM ANIMAIS, FEBRE MACULOSA, VIRULÊNCIA, RICKETTSIA SPP, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS, CARRAPATOS, CAMUNDONGOS
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ESTEVES, Eliane Virgínia da Silva et al. Comparative analysis of infection by Rickettsia rickettsii Sheila Smith and Taiaçu strains in a murine model. Pathogens, v. 9, n. 9, p. 15 , 2020Tradução . . Disponível em: https://doi.org/10.3390/pathogens9090744. Acesso em: 02 jun. 2024.
APA
Esteves, E. V. da S., Fongsaran, C., Langohr, I. M., Riley, S. P., Labruna, M. B., Daffre, S., et al. (2020). Comparative analysis of infection by Rickettsia rickettsii Sheila Smith and Taiaçu strains in a murine model. Pathogens, 9( 9), 15 . doi:10.3390/pathogens9090744
NLM
Esteves EV da S, Fongsaran C, Langohr IM, Riley SP, Labruna MB, Daffre S, Fogaça AC, Macaluso KR. Comparative analysis of infection by Rickettsia rickettsii Sheila Smith and Taiaçu strains in a murine model [Internet]. Pathogens. 2020 ; 9( 9): 15 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3390/pathogens9090744
Vancouver
Esteves EV da S, Fongsaran C, Langohr IM, Riley SP, Labruna MB, Daffre S, Fogaça AC, Macaluso KR. Comparative analysis of infection by Rickettsia rickettsii Sheila Smith and Taiaçu strains in a murine model [Internet]. Pathogens. 2020 ; 9( 9): 15 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3390/pathogens9090744
Artigo de periodico
Gut dysbiosis in Chagas disease. A possible link to the pathogenesis (2020)
Basqueira, Marcela de Souza ; Ribeiro, Roberto Marques; Oliveira, Léa Campos de; Moreira, Carlos Henrique Valente ; Martins, Roberta Cristina Ruedas; Franco, Diego Castillo ; Amado, Pâmela Pontes Penas ; Mayer, Marcia Pinto Alves ; Sabino, Ester Cerdeira
Source: Frontiers in Cellular and Infection Microbiology. Unidades: ICB, FM
Subjects: MICROBIOLOGIA, DOENÇA DE CHAGAS, TRYPANOSOMA CRUZI, FENÓTIPOS, DIVERSIDADE GENÉTICA, INFLAMAÇÃO
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BASQUEIRA, Marcela de Souza et al. Gut dysbiosis in Chagas disease. A possible link to the pathogenesis. Frontiers in Cellular and Infection Microbiology, v. 10, p. 8 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2020.00402. Acesso em: 02 jun. 2024.
APA
Basqueira, M. de S., Ribeiro, R. M., Oliveira, L. C. de, Moreira, C. H. V., Martins, R. C. R., Franco, D. C., et al. (2020). Gut dysbiosis in Chagas disease. A possible link to the pathogenesis. Frontiers in Cellular and Infection Microbiology, 10, 8 . doi:10.3389/fcimb.2020.00402
NLM
Basqueira M de S, Ribeiro RM, Oliveira LC de, Moreira CHV, Martins RCR, Franco DC, Amado PPP, Mayer MPA, Sabino EC. Gut dysbiosis in Chagas disease. A possible link to the pathogenesis [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10 8 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fcimb.2020.00402
Vancouver
Basqueira M de S, Ribeiro RM, Oliveira LC de, Moreira CHV, Martins RCR, Franco DC, Amado PPP, Mayer MPA, Sabino EC. Gut dysbiosis in Chagas disease. A possible link to the pathogenesis [Internet]. Frontiers in Cellular and Infection Microbiology. 2020 ; 10 8 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fcimb.2020.00402
Artigo de periodico
Growth hormone receptor deletion reduces the density of axonal projections from hypothalamic arcuate nucleus neurons (2020)
Wasinski, Frederick ; Furigo, Isadora Clivatti ; Teixeira, Pryscila Dryelle Sousa ; Lobo, Angela Maria Ramos ; Peroni, Cibele Nunes ; Bartolini, Paolo ; List, Edward O. ; Kopchick, John J. ; Donato Junior, José
Source: Neuroscience. Unidade: ICB
Subjects: FISIOLOGIA, HIPOTÁLAMO, INGESTÃO, HORMÔNIO DO CRESCIMENTO, AXÔNIOS, CAMUNDONGOS, LEPTINA, PEPTÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WASINSKI, Frederick et al. Growth hormone receptor deletion reduces the density of axonal projections from hypothalamic arcuate nucleus neurons. Neuroscience, v. 434, p. 136-147, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2020.03.037. Acesso em: 02 jun. 2024.
APA
Wasinski, F., Furigo, I. C., Teixeira, P. D. S., Lobo, A. M. R., Peroni, C. N., Bartolini, P., et al. (2020). Growth hormone receptor deletion reduces the density of axonal projections from hypothalamic arcuate nucleus neurons. Neuroscience, 434, 136-147. doi:10.1016/j.neuroscience.2020.03.037
NLM
Wasinski F, Furigo IC, Teixeira PDS, Lobo AMR, Peroni CN, Bartolini P, List EO, Kopchick JJ, Donato Junior J. Growth hormone receptor deletion reduces the density of axonal projections from hypothalamic arcuate nucleus neurons [Internet]. Neuroscience. 2020 ; 434 136-147.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2020.03.037
Vancouver
Wasinski F, Furigo IC, Teixeira PDS, Lobo AMR, Peroni CN, Bartolini P, List EO, Kopchick JJ, Donato Junior J. Growth hormone receptor deletion reduces the density of axonal projections from hypothalamic arcuate nucleus neurons [Internet]. Neuroscience. 2020 ; 434 136-147.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.neuroscience.2020.03.037
Artigo de periodico
Activation of oxytocin neurons improves cardiac function in a pressure-overload model of heart failure (2020)
Dyavanapalli, Jhansi ; Rodriguez, Jeannette; Santos, Carla Rocha dos; Escobar, Joan B.; Dwyer, Mary Kate ; Schloen, John ; Lee, Kyung-Min; Wolaver, Whitney; Wang, Xin; Dergacheva, Olga ; Michelini, Lisete Compagno ; Schunke, Kathryn Jaques ; Spurney, Christopher F. ; Kay, Matthew W. ; Mendelowitz, David
Source: JACC: Basic to Translational Science. Unidade: ICB
Subjects: FISIOLOGIA, OVÁRIO, HAMSTERS, INSUFICIÊNCIA CARDÍACA ANIMAL, INTERLEUCINAS, PRESSÃO SANGUÍNEA, OCITOCINA, HIPOTÁLAMO, SISTEMA NERVOSO PARASSIMPÁTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DYAVANAPALLI, Jhansi et al. Activation of oxytocin neurons improves cardiac function in a pressure-overload model of heart failure. JACC: Basic to Translational Science, v. 5, n. 5, p. 484-497, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.jacbts.2020.03.007. Acesso em: 02 jun. 2024.
APA
Dyavanapalli, J., Rodriguez, J., Santos, C. R. dos, Escobar, J. B., Dwyer, M. K., Schloen, J., et al. (2020). Activation of oxytocin neurons improves cardiac function in a pressure-overload model of heart failure. JACC: Basic to Translational Science, 5( 5), 484-497. doi:10.1016/j.jacbts.2020.03.007
NLM
Dyavanapalli J, Rodriguez J, Santos CR dos, Escobar JB, Dwyer MK, Schloen J, Lee K-M, Wolaver W, Wang X, Dergacheva O, Michelini LC, Schunke KJ, Spurney CF, Kay MW, Mendelowitz D. Activation of oxytocin neurons improves cardiac function in a pressure-overload model of heart failure [Internet]. JACC: Basic to Translational Science. 2020 ; 5( 5): 484-497.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.jacbts.2020.03.007
Vancouver
Dyavanapalli J, Rodriguez J, Santos CR dos, Escobar JB, Dwyer MK, Schloen J, Lee K-M, Wolaver W, Wang X, Dergacheva O, Michelini LC, Schunke KJ, Spurney CF, Kay MW, Mendelowitz D. Activation of oxytocin neurons improves cardiac function in a pressure-overload model of heart failure [Internet]. JACC: Basic to Translational Science. 2020 ; 5( 5): 484-497.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.jacbts.2020.03.007
Artigo de periodico
Short-term exposure to air pollution (PM2.5) induces hypothalamic inflammation, and long-term leads to leptin resistance and obesity via Tlr4/Ikbke in mice (2020)
Campolim, Clara Machado; Weissmann, Lais; Ferreira, Clílton Kraüss de Oliveira; Zordão, Olivia Pizetta ; Dornellas, Ana Paula Segantine; Castro, Gisele de ; Zanotto, Tamires Marques; Boico, Vitor Ferreira; Quaresma, Paula Gabriele Fernandes ; Lima, Raquel Patrícia Ataíde ; Donato Junior, José ; Veras, Mariana Matera ; Saldiva, Paulo Hilário Nascimento ; Kim, Young-Bum; Prada, Patricia Oliveira
Source: Scientific Reports. Unidades: ICB, FM
Subjects: FISIOLOGIA, CAMUNDONGOS, DIETA ANIMAL, OBESIDADE, HIPOTÁLAMO, ADIPOSIDADE, TECIDO ADIPOSO, LEPTINA, METABÓLITOS, INSULINA, INFLAMAÇÃO
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ABNT
CAMPOLIM, Clara Machado et al. Short-term exposure to air pollution (PM2.5) induces hypothalamic inflammation, and long-term leads to leptin resistance and obesity via Tlr4/Ikbke in mice. Scientific Reports, v. 10, p. 14 , 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-67040-3. Acesso em: 02 jun. 2024.
APA
Campolim, C. M., Weissmann, L., Ferreira, C. K. de O., Zordão, O. P., Dornellas, A. P. S., Castro, G. de, et al. (2020). Short-term exposure to air pollution (PM2.5) induces hypothalamic inflammation, and long-term leads to leptin resistance and obesity via Tlr4/Ikbke in mice. Scientific Reports, 10, 14 . doi:10.1038/s41598-020-67040-3
NLM
Campolim CM, Weissmann L, Ferreira CK de O, Zordão OP, Dornellas APS, Castro G de, Zanotto TM, Boico VF, Quaresma PGF, Lima RPA, Donato Junior J, Veras MM, Saldiva PHN, Kim Y-B, Prada PO. Short-term exposure to air pollution (PM2.5) induces hypothalamic inflammation, and long-term leads to leptin resistance and obesity via Tlr4/Ikbke in mice [Internet]. Scientific Reports. 2020 ; 10 14 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41598-020-67040-3
Vancouver
Campolim CM, Weissmann L, Ferreira CK de O, Zordão OP, Dornellas APS, Castro G de, Zanotto TM, Boico VF, Quaresma PGF, Lima RPA, Donato Junior J, Veras MM, Saldiva PHN, Kim Y-B, Prada PO. Short-term exposure to air pollution (PM2.5) induces hypothalamic inflammation, and long-term leads to leptin resistance and obesity via Tlr4/Ikbke in mice [Internet]. Scientific Reports. 2020 ; 10 14 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41598-020-67040-3
Artigo de periodico
Environmental control of mammary carcinoma cell expansion by acidification and spheroid formation in vitro (2020)
Ralph, Ana Carolina Lima ; Valadão, Iuri Cordeiro ; Cardoso, Elaine Cristina; Martins, Vilma Regina ; Oliveira, Luanda Mara da Silva; Bevilacqua, Estela Maris Andrade Forell ; Geraldo, Murilo Vieira ; Jaeger, Ruy Gastaldoni ; Goldberg, Gary S.; Freitas, Vanessa Morais
Source: Scientific Reports. Unidades: ICB, FM
Subjects: BIOLOGIA CELULAR, NEOPLASIAS MAMÁRIAS, CARCINOMA, CÉLULAS CULTIVADAS DE TUMOR, QUIMIOTERÁPICOS, FIBROBLASTOS, CÉLULAS MORTAS, FENÓTIPOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RALPH, Ana Carolina Lima et al. Environmental control of mammary carcinoma cell expansion by acidification and spheroid formation in vitro. Scientific Reports, v. 10, p. 12 , 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-78989-6. Acesso em: 02 jun. 2024.
APA
Ralph, A. C. L., Valadão, I. C., Cardoso, E. C., Martins, V. R., Oliveira, L. M. da S., Bevilacqua, E. M. A. F., et al. (2020). Environmental control of mammary carcinoma cell expansion by acidification and spheroid formation in vitro. Scientific Reports, 10, 12 . doi:10.1038/s41598-020-78989-6
NLM
Ralph ACL, Valadão IC, Cardoso EC, Martins VR, Oliveira LM da S, Bevilacqua EMAF, Geraldo MV, Jaeger RG, Goldberg GS, Freitas VM. Environmental control of mammary carcinoma cell expansion by acidification and spheroid formation in vitro [Internet]. Scientific Reports. 2020 ; 10 12 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41598-020-78989-6
Vancouver
Ralph ACL, Valadão IC, Cardoso EC, Martins VR, Oliveira LM da S, Bevilacqua EMAF, Geraldo MV, Jaeger RG, Goldberg GS, Freitas VM. Environmental control of mammary carcinoma cell expansion by acidification and spheroid formation in vitro [Internet]. Scientific Reports. 2020 ; 10 12 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41598-020-78989-6
Artigo de periodico
Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins (2020)
Kumar, Namrata ; Moreno, Natália Cestari ; Feltes, Bruno C. ; Menck, Carlos Frederico Martins ; Houten, Bennett Van
Source: Genetics and Molecular Biology. Unidades: ICB, IQ
Subjects: MICROBIOLOGIA, DANO AO DNA, MUTAGÊNESE, MUTAÇÃO GENÉTICA, NUCLEOTÍDEOS, CARCINOGÊNESE, ESTRESSE OXIDATIVO, APOPTOSE, GENOMAS
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ABNT
KUMAR, Namrata et al. Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins. Genetics and Molecular Biology, v. 43, n. 1, p. 14 , 2020Tradução . . Disponível em: https://doi.org/10.1590/1678-4685-gmb-2019-0104. Acesso em: 02 jun. 2024.
APA
Kumar, N., Moreno, N. C., Feltes, B. C., Menck, C. F. M., & Houten, B. V. (2020). Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins. Genetics and Molecular Biology, 43( 1), 14 . doi:10.1590/1678-4685-gmb-2019-0104
NLM
Kumar N, Moreno NC, Feltes BC, Menck CFM, Houten BV. Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins [Internet]. Genetics and Molecular Biology. 2020 ; 43( 1): 14 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1590/1678-4685-gmb-2019-0104
Vancouver
Kumar N, Moreno NC, Feltes BC, Menck CFM, Houten BV. Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins [Internet]. Genetics and Molecular Biology. 2020 ; 43( 1): 14 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1590/1678-4685-gmb-2019-0104
Artigo de periodico
Eicosapentaenoic acid regulates inflammatory pathways through modulation of transcripts and miRNA in adipose tissue of obese mice (2020)
Ramalho, Theresa Raquel de Oliveira ; Pahlavani, Mandana ; Kalupahana, Nishan ; Wijayatunga, Nadeeja ; Ramalingam, Latha ; Negro, Sonia Jancar ; Moussa, Naima Moustaid
Source: Biomolecules. Unidade: ICB
Subjects: IMUNOLOGIA, EICOSANOIDES, ÁCIDOS EICOSANOICOS, INFLAMAÇÃO, MICRORNAS, TECIDO ADIPOSO, CAMUNDONGOS, OBESIDADE, LEUCOTRIENOS, EXPRESSÃO GÊNICA, RNA, RESISTÊNCIA À INSULINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RAMALHO, Theresa Raquel de Oliveira et al. Eicosapentaenoic acid regulates inflammatory pathways through modulation of transcripts and miRNA in adipose tissue of obese mice. Biomolecules, v. 10, n. 9, p. 14 , 2020Tradução . . Disponível em: https://doi.org/10.3390/biom10091292. Acesso em: 02 jun. 2024.
APA
Ramalho, T. R. de O., Pahlavani, M., Kalupahana, N., Wijayatunga, N., Ramalingam, L., Negro, S. J., & Moussa, N. M. (2020). Eicosapentaenoic acid regulates inflammatory pathways through modulation of transcripts and miRNA in adipose tissue of obese mice. Biomolecules, 10( 9), 14 . doi:10.3390/biom10091292
NLM
Ramalho TR de O, Pahlavani M, Kalupahana N, Wijayatunga N, Ramalingam L, Negro SJ, Moussa NM. Eicosapentaenoic acid regulates inflammatory pathways through modulation of transcripts and miRNA in adipose tissue of obese mice [Internet]. Biomolecules. 2020 ; 10( 9): 14 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3390/biom10091292
Vancouver
Ramalho TR de O, Pahlavani M, Kalupahana N, Wijayatunga N, Ramalingam L, Negro SJ, Moussa NM. Eicosapentaenoic acid regulates inflammatory pathways through modulation of transcripts and miRNA in adipose tissue of obese mice [Internet]. Biomolecules. 2020 ; 10( 9): 14 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3390/biom10091292
Artigo de periodico
Strains used in whole organism Plasmodium falciparum vaccine trials differ in genome structure, sequence, and immunogenic potential (2020)
Moser, Kara A.; Drábek, Elliott F.; Dwivedi, Ankit; Stucke, Emily M.; Crabtree, Jonathan ; Dara, Antoine ; Shah, Zalak; Adams, Matthew; Li, Tao; Rodrigues, Priscila Thihara ; Koren, Sergey ; Phillipy, Adam M.; Munro, James B. ; Ouattara, Amed; Sparklin, Benjamin C.; Hotopp, Julie C. Dunning ; Lyke, Kirsten E.; Sadzewicz, Lisa ; Tallon, Luke J.; Spring, Michele D.; Jongsakul, Krisada ; Lon, Chanthap ; Saunders, David L.; Ferreira, Marcelo Urbano ; Nyunt, Myaing M. ; Laufer, Miriam K. ; Travassos, Mark A. ; Sauerwein, Robert W. ; Harrison, Shannon Takala ; Fraser, Claire M. ; Sim, B. Kim Lee ; Hoffman, Stephen L. ; Plowe, Christopher ; Silva, Joana C.
Source: Genome Medicine: medicine in the post-genomic era. Unidade: ICB
Subjects: PARASITOLOGIA, PLASMODIUM FALCIPARUM, INFECÇÕES POR PROTOZOÁRIOS, VACINAS, MALÁRIA, GENOMAS, EPITOPOS, FATORES DE TRANSCRIÇÃO
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ABNT
MOSER, Kara A. et al. Strains used in whole organism Plasmodium falciparum vaccine trials differ in genome structure, sequence, and immunogenic potential. Genome Medicine: medicine in the post-genomic era, v. 12, n. 1, p. 17 , 2020Tradução . . Disponível em: https://doi.org/10.1186/s13073-019-0708-9. Acesso em: 02 jun. 2024.
APA
Moser, K. A., Drábek, E. F., Dwivedi, A., Stucke, E. M., Crabtree, J., Dara, A., et al. (2020). Strains used in whole organism Plasmodium falciparum vaccine trials differ in genome structure, sequence, and immunogenic potential. Genome Medicine: medicine in the post-genomic era, 12( 1), 17 . doi:10.1186/s13073-019-0708-9
NLM
Moser KA, Drábek EF, Dwivedi A, Stucke EM, Crabtree J, Dara A, Shah Z, Adams M, Li T, Rodrigues PT, Koren S, Phillipy AM, Munro JB, Ouattara A, Sparklin BC, Hotopp JCD, Lyke KE, Sadzewicz L, Tallon LJ, Spring MD, Jongsakul K, Lon C, Saunders DL, Ferreira MU, Nyunt MM, Laufer MK, Travassos MA, Sauerwein RW, Harrison ST, Fraser CM, Sim BKL, Hoffman SL, Plowe C, Silva JC. Strains used in whole organism Plasmodium falciparum vaccine trials differ in genome structure, sequence, and immunogenic potential [Internet]. Genome Medicine: medicine in the post-genomic era. 2020 ; 12( 1): 17 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1186/s13073-019-0708-9
Vancouver
Moser KA, Drábek EF, Dwivedi A, Stucke EM, Crabtree J, Dara A, Shah Z, Adams M, Li T, Rodrigues PT, Koren S, Phillipy AM, Munro JB, Ouattara A, Sparklin BC, Hotopp JCD, Lyke KE, Sadzewicz L, Tallon LJ, Spring MD, Jongsakul K, Lon C, Saunders DL, Ferreira MU, Nyunt MM, Laufer MK, Travassos MA, Sauerwein RW, Harrison ST, Fraser CM, Sim BKL, Hoffman SL, Plowe C, Silva JC. Strains used in whole organism Plasmodium falciparum vaccine trials differ in genome structure, sequence, and immunogenic potential [Internet]. Genome Medicine: medicine in the post-genomic era. 2020 ; 12( 1): 17 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1186/s13073-019-0708-9
Artigo de periodico
Modelling the epidemiology of residual Plasmodium vivax malaria in a heterogeneous host population: a case study in the Amazon Basin (2020)
Corder, Rodrigo Malavazi ; Ferreira, Marcelo Urbano ; Gomes, Maria Gabriela Miranda
Source: PL o S Computational Biology. Unidade: ICB
Subjects: PARASITOLOGIA, PLASMODIUM, MALÁRIA, POPULAÇÃO, MODELOS EPIDEMIOLOGICOS, MODELOS MATEMÁTICOS, ESTUDO DE CASO
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ABNT
CORDER, Rodrigo Malavazi e FERREIRA, Marcelo Urbano e GOMES, Maria Gabriela Miranda. Modelling the epidemiology of residual Plasmodium vivax malaria in a heterogeneous host population: a case study in the Amazon Basin. PL o S Computational Biology, v. 16, n. 3, p. 18 , 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.pcbi.1007377. Acesso em: 02 jun. 2024.
APA
Corder, R. M., Ferreira, M. U., & Gomes, M. G. M. (2020). Modelling the epidemiology of residual Plasmodium vivax malaria in a heterogeneous host population: a case study in the Amazon Basin. PL o S Computational Biology, 16( 3), 18 . doi:10.1371/journal.pcbi.1007377
NLM
Corder RM, Ferreira MU, Gomes MGM. Modelling the epidemiology of residual Plasmodium vivax malaria in a heterogeneous host population: a case study in the Amazon Basin [Internet]. PL o S Computational Biology. 2020 ; 16( 3): 18 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1371/journal.pcbi.1007377
Vancouver
Corder RM, Ferreira MU, Gomes MGM. Modelling the epidemiology of residual Plasmodium vivax malaria in a heterogeneous host population: a case study in the Amazon Basin [Internet]. PL o S Computational Biology. 2020 ; 16( 3): 18 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1371/journal.pcbi.1007377

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