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Artigo de periodico
Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae (2024)
Padovani, Barbara Nunes ; Fénero, Camila Ideli Morales ; Paredes, Lais Cavalieri ; Amaral, Mariana Abrantes do ; Amorocho, Omar Alberto Domínguez ; Cipelli, Marcella ; Gomes, Juliana Moreira Mendonça ; Silva, Eloisa Martins da ; Silva, Luisa Menezes ; Vieira, Raquel de Souza ; Pereira, Mariana Tominaga ; Cruz, Mario Costa ; Camara, Niels Olsen Saraiva
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: IMUNOLOGIA, ANTINEOPLÁSICOS, INSUFICIÊNCIA RENAL AGUDA, INFLAMAÇÃO, CITOCINAS, INTERLEUCINAS, TOXICIDADE EM ANIMAL, REAÇÃO EM CADEIA POR POLIMERASE, CITOMETRIA DE FLUXO, PEIXES DE ÁGUA DOCE, PEIXES TROPICAIS
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PADOVANI, Barbara Nunes et al. Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae. International Journal of Molecular Sciences, v. 25, n. 4, p. 16 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25042363. Acesso em: 02 jun. 2024.
APA
Padovani, B. N., Fénero, C. I. M., Paredes, L. C., Amaral, M. A. do, Amorocho, O. A. D., Cipelli, M., et al. (2024). Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae. International Journal of Molecular Sciences, 25( 4), 16 . doi:10.3390/ijms25042363
NLM
Padovani BN, Fénero CIM, Paredes LC, Amaral MA do, Amorocho OAD, Cipelli M, Gomes JMM, Silva EM da, Silva LM, Vieira R de S, Pereira MT, Cruz MC, Camara NOS. Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 4): 16 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3390/ijms25042363
Vancouver
Padovani BN, Fénero CIM, Paredes LC, Amaral MA do, Amorocho OAD, Cipelli M, Gomes JMM, Silva EM da, Silva LM, Vieira R de S, Pereira MT, Cruz MC, Camara NOS. Cisplatin toxicity causes neutrophil-mediated inflammation in Zebrafish larvae [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 4): 16 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3390/ijms25042363
Artigo de periodico
Pathogenic role of angiotensin II and the NF-κB system in a model of malignant hypertensive nephrosclerosis (2024)
Ávila, Victor Ferreira de; Foresto Neto, Orestes ; Arias, Simone Costa Alarcon; Malheiros, Denise Maria Avancini Costa ; Zatz, Roberto ; Fujihara, Clarice Kazue; Câmara, Niels Olsen Saraiva
Source: Hypertension Research. Unidades: FM, ICB
Subjects: IMUNOLOGIA, ANGIOTENSINA II, HIPERTENSÃO, NEFROPATIAS EM ANIMAL, NEFRITE ANIMAL, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, AMINAS (COMPOSTOS ORGÂNICOS), GLOMÉRULOS RENAIS, GLOMERUNEFRITE
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ÁVILA, Victor Ferreira de et al. Pathogenic role of angiotensin II and the NF-κB system in a model of malignant hypertensive nephrosclerosis. Hypertension Research, v. 42, n. 6, p. 778-789, 2024Tradução . . Disponível em: https://doi.org/10.1038/s41440-019-0226-6. Acesso em: 02 jun. 2024.
APA
Ávila, V. F. de, Foresto Neto, O., Arias, S. C. A., Malheiros, D. M. A. C., Zatz, R., Fujihara, C. K., & Câmara, N. O. S. (2024). Pathogenic role of angiotensin II and the NF-κB system in a model of malignant hypertensive nephrosclerosis. Hypertension Research, 42( 6), 778-789. doi:10.1038/s41440-019-0226-6
NLM
Ávila VF de, Foresto Neto O, Arias SCA, Malheiros DMAC, Zatz R, Fujihara CK, Câmara NOS. Pathogenic role of angiotensin II and the NF-κB system in a model of malignant hypertensive nephrosclerosis [Internet]. Hypertension Research. 2024 ; 42( 6): 778-789.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41440-019-0226-6
Vancouver
Ávila VF de, Foresto Neto O, Arias SCA, Malheiros DMAC, Zatz R, Fujihara CK, Câmara NOS. Pathogenic role of angiotensin II and the NF-κB system in a model of malignant hypertensive nephrosclerosis [Internet]. Hypertension Research. 2024 ; 42( 6): 778-789.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1038/s41440-019-0226-6
Artigo de periodico
Immunological signatures unveiled by integrative systems vaccinology characterization of dengue vaccination trials and natural infection (2024)
Plaça, Desireé Rodrigues ; Fonseca, Dennyson Leandro Mathias ; Marques, Alexandre Henrique Carvalho ; Pour, Shahab Zaki ; Usuda, Julia Nakanishi ; Baiocchi, Gabriela Crispim ; Prado, Caroline Aliane De Souza ; Salgado, Ranieri Coelho ; Filgueiras, Igor Salerno ; Freire, Paula Paccielli ; Miranda, Gustavo Cabral de ; Catar, Rusan ; Moll, Guido ; Jurisica, Igor ; Giil, Lasse Melvaer ; Rivino, Laura ; Ochs, Hans Dieter ; Miranda, Gustavo Cabral de ; Schimke, Lena Friederike ; Rocha, Vanderson Geraldo ; Camara, Niels Olsen Saraiva ; Calich, Vera Lúcia Garcia ; Marques, Otavio Cabral
Source: Frontiers in Immunology. Unidades: FM, ICB, FCF
Subjects: IMUNOLOGIA, DENGUE, INFECÇÕES POR ARBOVIRUS, VACINAS VIRAIS, VACINAÇÃO, RESPOSTAS
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PLAÇA, Desireé Rodrigues et al. Immunological signatures unveiled by integrative systems vaccinology characterization of dengue vaccination trials and natural infection. Frontiers in Immunology, v. 15, p. 17 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2024.1282754. Acesso em: 02 jun. 2024.
APA
Plaça, D. R., Fonseca, D. L. M., Marques, A. H. C., Pour, S. Z., Usuda, J. N., Baiocchi, G. C., et al. (2024). Immunological signatures unveiled by integrative systems vaccinology characterization of dengue vaccination trials and natural infection. Frontiers in Immunology, 15, 17 . doi:10.3389/fimmu.2024.1282754
NLM
Plaça DR, Fonseca DLM, Marques AHC, Pour SZ, Usuda JN, Baiocchi GC, Prado CADS, Salgado RC, Filgueiras IS, Freire PP, Miranda GC de, Catar R, Moll G, Jurisica I, Giil LM, Rivino L, Ochs HD, Miranda GC de, Schimke LF, Rocha VG, Camara NOS, Calich VLG, Marques OC. Immunological signatures unveiled by integrative systems vaccinology characterization of dengue vaccination trials and natural infection [Internet]. Frontiers in Immunology. 2024 ; 15 17 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fimmu.2024.1282754.
Vancouver
Plaça DR, Fonseca DLM, Marques AHC, Pour SZ, Usuda JN, Baiocchi GC, Prado CADS, Salgado RC, Filgueiras IS, Freire PP, Miranda GC de, Catar R, Moll G, Jurisica I, Giil LM, Rivino L, Ochs HD, Miranda GC de, Schimke LF, Rocha VG, Camara NOS, Calich VLG, Marques OC. Immunological signatures unveiled by integrative systems vaccinology characterization of dengue vaccination trials and natural infection [Internet]. Frontiers in Immunology. 2024 ; 15 17 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fimmu.2024.1282754.
Artigo de periodico
Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity (2023)
Albornoz, Sandra Patrícia Palma ; Silva, Thais Fernanda de Campo Fraga da ; Carvalho, Renan Villanova Homem de ; Rodrigues, Tamara Silva; Gembre, Ana Flávia; Oliveira, Rômulo Silva de; Souza, Fernanda Mesquita de; Corrêa, Giseli Furlan ; Ramalho, Leandra Naira Zambelli ; Sartori, Daniela Carlos ; Câmara, Niels Olsen Saraiva ; Zamboni, Dario Simões ; Takahashi, Viviani Nardini ; Sorgi, Carlos Artério ; Faccioli, Lucia Helena ; Medeiros, Alexandra Ivo de ; Costa, Diego Luís ; Bonato, Vânia Luiza Deperon
Source: Journal of Pathology. Unidades: FMRP, ICB, FCFRP, FFCLRP, ESALQ
Assunto: IMUNOLOGIA
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ALBORNOZ, Sandra Patrícia Palma et al. Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity. Journal of Pathology, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.1002/path.6041. Acesso em: 02 jun. 2024.
APA
Albornoz, S. P. P., Silva, T. F. de C. F. da, Carvalho, R. V. H. de, Rodrigues, T. S., Gembre, A. F., Oliveira, R. S. de, et al. (2023). Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity. Journal of Pathology, 1-13. doi:10.1002/path.6041
NLM
Albornoz SPP, Silva TF de CF da, Carvalho RVH de, Rodrigues TS, Gembre AF, Oliveira RS de, Souza FM de, Corrêa GF, Ramalho LNZ, Sartori DC, Câmara NOS, Zamboni DS, Takahashi VN, Sorgi CA, Faccioli LH, Medeiros AI de, Costa DL, Bonato VLD. Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity [Internet]. Journal of Pathology. 2023 ; 1-13.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1002/path.6041
Vancouver
Albornoz SPP, Silva TF de CF da, Carvalho RVH de, Rodrigues TS, Gembre AF, Oliveira RS de, Souza FM de, Corrêa GF, Ramalho LNZ, Sartori DC, Câmara NOS, Zamboni DS, Takahashi VN, Sorgi CA, Faccioli LH, Medeiros AI de, Costa DL, Bonato VLD. Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity [Internet]. Journal of Pathology. 2023 ; 1-13.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1002/path.6041
Artigo de periodico
Infection-elicited microbiota promotes host adaptation to nutrient restriction (2023)
Siqueira, Mirian Krystel de ; Oliveira, Vinicius Andrade de ; Stacy, Apollo ; Guimarães, João Pedro Tôrres ; Custodio, Ricardo Wesley Alberca ; Castoldi, Angela ; Santos, Jaqueline Marques; Ferreira, Marcela Davoli ; Silva, Luisa Menezes ; Turato, Walter Miguel; Han, Seong-Ji ; Zaretsky, Arielle Glatman ; Hand, Timothy Wesley ; Belkaid, Yasmine ; Câmara, Niels Olsen Saraiva ; Russo, Momtchilo ; Negro, Sonia Jancar ; Fonseca, Denise Morais da
Source: National Academy of Sciences. Proceedings. Unidades: FCF, ICB
Subjects: IMUNOLOGIA, YERSINIA, CARBOIDRATOS, DESNUTRIÇÃO, TECIDO ADIPOSO, NUTRIENTES, CAMUNDONGOS, PATOGENIA ANIMAL, OBESIDADE, DIETA ANIMAL
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SIQUEIRA, Mirian Krystel de et al. Infection-elicited microbiota promotes host adaptation to nutrient restriction. National Academy of Sciences. Proceedings, v. 120, n. 4, p. 1-8, 2023Tradução . . Disponível em: https://doi.org/10.1073/pnas.2214484120. Acesso em: 02 jun. 2024.
APA
Siqueira, M. K. de, Oliveira, V. A. de, Stacy, A., Guimarães, J. P. T., Custodio, R. W. A., Castoldi, A., et al. (2023). Infection-elicited microbiota promotes host adaptation to nutrient restriction. National Academy of Sciences. Proceedings, 120( 4), 1-8. doi:10.1073/pnas.2214484120
NLM
Siqueira MK de, Oliveira VA de, Stacy A, Guimarães JPT, Custodio RWA, Castoldi A, Santos JM, Ferreira MD, Silva LM, Turato WM, Han S-J, Zaretsky AG, Hand TW, Belkaid Y, Câmara NOS, Russo M, Negro SJ, Fonseca DM da. Infection-elicited microbiota promotes host adaptation to nutrient restriction [Internet]. National Academy of Sciences. Proceedings. 2023 ; 120( 4): 1-8.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1073/pnas.2214484120
Vancouver
Siqueira MK de, Oliveira VA de, Stacy A, Guimarães JPT, Custodio RWA, Castoldi A, Santos JM, Ferreira MD, Silva LM, Turato WM, Han S-J, Zaretsky AG, Hand TW, Belkaid Y, Câmara NOS, Russo M, Negro SJ, Fonseca DM da. Infection-elicited microbiota promotes host adaptation to nutrient restriction [Internet]. National Academy of Sciences. Proceedings. 2023 ; 120( 4): 1-8.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1073/pnas.2214484120
Artigo de periodico
The role of dendritic cells in COVID-19 infection (2023)
Wang, Xuying; Miller, Heather; Byazrova, Maria G. ; Candotti, Fabio ; Benlagha, Kamel ; Lei, Jiahui; Filatov, Alexander ; Liu, Chaohong ; Câmara, Niels Olsen Saraiva
Source: Emerging Microbes & Infections. Unidade: ICB
Subjects: IMUNOLOGIA, CÉLULAS DENDRÍTICAS, CITOCINAS, COVID-19, INFECÇÕES POR VÍRUS DE RNA
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WANG, Xuying et al. The role of dendritic cells in COVID-19 infection. Emerging Microbes & Infections, v. 12, n. 1, p. 1-9, 2023Tradução . . Disponível em: https://doi.org/10.1080/22221751.2023.2195019. Acesso em: 02 jun. 2024.
APA
Wang, X., Miller, H., Byazrova, M. G., Candotti, F., Benlagha, K., Lei, J., et al. (2023). The role of dendritic cells in COVID-19 infection. Emerging Microbes & Infections, 12( 1), 1-9. doi:10.1080/22221751.2023.2195019
NLM
Wang X, Miller H, Byazrova MG, Candotti F, Benlagha K, Lei J, Filatov A, Liu C, Câmara NOS. The role of dendritic cells in COVID-19 infection [Internet]. Emerging Microbes & Infections. 2023 ; 12( 1): 1-9.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1080/22221751.2023.2195019
Vancouver
Wang X, Miller H, Byazrova MG, Candotti F, Benlagha K, Lei J, Filatov A, Liu C, Câmara NOS. The role of dendritic cells in COVID-19 infection [Internet]. Emerging Microbes & Infections. 2023 ; 12( 1): 1-9.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1080/22221751.2023.2195019
Artigo de periodico
Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression (2023)
Rubenich, Dominique S. ; Souza, Priscila O. de ; Omizollo, Natalia; Aubin, Mariana R. ; Basso, Paulo J. ; Silva, Luisa M. ; Silva, Eloisa M. ; Teixeira, Fernanda C. ; Gentil, Gabriela F. S.; Domagalski, Jordana ; Cunha, Maico T. ; Gadelha, Kerolainy A. ; Diel, Leonardo F. ; Gelsleichter, Nicolly E. ; Rubenich, Aline S.; Lenz, Gabriela S. ; Abreu, Aline M. ; Kroeff, Giselle M.; Ana H. Paz ; Visioli, Fernanda ; Lamers, Marcelo L. ; Wink, Marcia R. ; Worm, Paulo V. ; Araújo, Anelise B. ; Sévigny, Jean ; Ludwig, Nils ; Braganho, Elizandra ; Câmara, Niels Olsen Saraiva
Source: Frontiers in immunology. Unidade: ICB
Subjects: CAMUNDONGOS, CÉLULAS CULTIVADAS DE TUMOR, NEUTRÓFILOS, GLIOMA, NEOPLASIAS CEREBRAIS, DOENÇAS DO SISTEMA NERVOSO CENTRAL, METABOLISMO ENERGÉTICO, CITOCINAS, FÁRMACOS IMUNOSSUPRESSORES
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RUBENICH, Dominique S. et al. Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression. Frontiers in immunology, v. 14, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2023.1183465. Acesso em: 02 jun. 2024.
APA
Rubenich, D. S., Souza, P. O. de, Omizollo, N., Aubin, M. R., Basso, P. J., Silva, L. M., et al. (2023). Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression. Frontiers in immunology, 14. doi:10.3389/fimmu.2023.1183465
NLM
Rubenich DS, Souza PO de, Omizollo N, Aubin MR, Basso PJ, Silva LM, Silva EM, Teixeira FC, Gentil GFS, Domagalski J, Cunha MT, Gadelha KA, Diel LF, Gelsleichter NE, Rubenich AS, Lenz GS, Abreu AM, Kroeff GM, Ana H. Paz, Visioli F, Lamers ML, Wink MR, Worm PV, Araújo AB, Sévigny J, Ludwig N, Braganho E, Câmara NOS. Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression [Internet]. Frontiers in immunology. 2023 ; 14[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fimmu.2023.1183465
Vancouver
Rubenich DS, Souza PO de, Omizollo N, Aubin MR, Basso PJ, Silva LM, Silva EM, Teixeira FC, Gentil GFS, Domagalski J, Cunha MT, Gadelha KA, Diel LF, Gelsleichter NE, Rubenich AS, Lenz GS, Abreu AM, Kroeff GM, Ana H. Paz, Visioli F, Lamers ML, Wink MR, Worm PV, Araújo AB, Sévigny J, Ludwig N, Braganho E, Câmara NOS. Tumor-neutrophil crosstalk promotes in vitro and in vivo glioblastoma progression [Internet]. Frontiers in immunology. 2023 ; 14[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fimmu.2023.1183465
Artigo de periodico
The role of Raptor in lymphocytes differentiation and function (2023)
Tang, Jianing; Yang, Lu; Guan, Fei; Miller, Heather; James, Louisa K.; Benlagha, Kamel ; Kubo, Masato ; Heegaard, Steffen ; Lee, Pamela ; Lei, Jiahui; Zeng, Hu ; He, Chengwei; Zhai, Zhimin ; Liu, Chaohong ; Câmara, Niels Olsen Saraiva
Source: Frontiers in Immunology. Unidade: ICB
Subjects: LINFÓCITOS, METABOLISMO CELULAR, CITOCINAS, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
TANG, Jianing et al. The role of Raptor in lymphocytes differentiation and function. Frontiers in Immunology, v. 14, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2023.1146628. Acesso em: 02 jun. 2024.
APA
Tang, J., Yang, L., Guan, F., Miller, H., James, L. K., Benlagha, K., et al. (2023). The role of Raptor in lymphocytes differentiation and function. Frontiers in Immunology, 14. doi:10.3389/fimmu.2023.1146628
NLM
Tang J, Yang L, Guan F, Miller H, James LK, Benlagha K, Kubo M, Heegaard S, Lee P, Lei J, Zeng H, He C, Zhai Z, Liu C, Câmara NOS. The role of Raptor in lymphocytes differentiation and function [Internet]. Frontiers in Immunology. 2023 ; 14[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fimmu.2023.1146628
Vancouver
Tang J, Yang L, Guan F, Miller H, James LK, Benlagha K, Kubo M, Heegaard S, Lee P, Lei J, Zeng H, He C, Zhai Z, Liu C, Câmara NOS. The role of Raptor in lymphocytes differentiation and function [Internet]. Frontiers in Immunology. 2023 ; 14[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fimmu.2023.1146628
Artigo de periodico
Integrative systems immunology uncovers molecular networks of the cell cycle that stratify COVID-19 severity (2023)
Prado, Caroline Aliane De Souza ; Fonseca, Dennyson Leandro Mathias da ; Singh, Youvika ; Filgueiras, Igor Salerno ; Baiocchi, Gabriela Crispim ; Plaça, Desirée Rodrigues ; Marques, Alexandre Henrique Carvalho ; Dantas-Komatsu, Raquel Costa Silva ; Usuda, Julia N. ; Freire, Paula Paccielli ; Salgado, Ranieri Coelho ; Napoleão, Sarah Maria da Silva ; Ramos, Rodrigo Nalio ; Rocha, Vanderson ; Guangyan Zhou; Catar, Rusan ; Guido Moll ; Giil, Lasse M. ; Mishra, Neha ; Tran, Florian ; Luchessi, Andre Ducati ; Ochs, Hans Dieter ; Igor Jurisica, Igor ; Schimke, Lena F. ; Câmara, Niels Olsen Saraiva ; Miranda, Gustavo Cabral de ; Calich, Vera Lúcia Garcia ; Nakaya, Helder Takashi Imoto ; Marques, Otavio Cabral
Source: Journal of Medical Virology. Unidades: ICB, BIOTECNOLOGIA, FCF, BIOINFORMÁTICA
Subjects: IMUNOLOGIA, IMUNOLOGIA, COVID-19, CICLO CELULAR, PROTEÔMICA, LEUCÓCITOS, NEUTRÓFILOS, IMUNOPROTEÍNAS, PROTEÍNAS QUINASES
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ABNT
PRADO, Caroline Aliane De Souza et al. Integrative systems immunology uncovers molecular networks of the cell cycle that stratify COVID-19 severity. Journal of Medical Virology, v. 95, n. 2, p. 1-18, 2023Tradução . . Disponível em: https://doi.org/10.1002/jmv.28450. Acesso em: 02 jun. 2024.
APA
Prado, C. A. D. S., Fonseca, D. L. M. da, Singh, Y., Filgueiras, I. S., Baiocchi, G. C., Plaça, D. R., et al. (2023). Integrative systems immunology uncovers molecular networks of the cell cycle that stratify COVID-19 severity. Journal of Medical Virology, 95( 2), 1-18. doi:10.1002/jmv.28450
NLM
Prado CADS, Fonseca DLM da, Singh Y, Filgueiras IS, Baiocchi GC, Plaça DR, Marques AHC, Dantas-Komatsu RCS, Usuda JN, Freire PP, Salgado RC, Napoleão SM da S, Ramos RN, Rocha V, Guangyan Zhou, Catar R, Guido Moll, Giil LM, Mishra N, Tran F, Luchessi AD, Ochs HD, Igor Jurisica I, Schimke LF, Câmara NOS, Miranda GC de, Calich VLG, Nakaya HTI, Marques OC. Integrative systems immunology uncovers molecular networks of the cell cycle that stratify COVID-19 severity [Internet]. Journal of Medical Virology. 2023 ; 95( 2): 1-18.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1002/jmv.28450
Vancouver
Prado CADS, Fonseca DLM da, Singh Y, Filgueiras IS, Baiocchi GC, Plaça DR, Marques AHC, Dantas-Komatsu RCS, Usuda JN, Freire PP, Salgado RC, Napoleão SM da S, Ramos RN, Rocha V, Guangyan Zhou, Catar R, Guido Moll, Giil LM, Mishra N, Tran F, Luchessi AD, Ochs HD, Igor Jurisica I, Schimke LF, Câmara NOS, Miranda GC de, Calich VLG, Nakaya HTI, Marques OC. Integrative systems immunology uncovers molecular networks of the cell cycle that stratify COVID-19 severity [Internet]. Journal of Medical Virology. 2023 ; 95( 2): 1-18.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1002/jmv.28450
Artigo de periodico
P2RX7 signaling drives the differentiation of Th1 cells through metabolic reprogramming for aerobic glycolysis (2023)
Salles, Érika Machado de ; Raeder, Paulo Lisboa ; Angeli, Claudia Blanes ; Santiago, Verônica Feijoli ; Souza, Cristiane Naffah de ; Ramalho, Theresa ; Câmara, Niels Olsen Saraiva ; Palmisano, Giuseppe ; Alvarez, José Maria ; Lima, Maria Regina D'Império
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, PARASITOLOGA, METABOLISMO CELULAR, LINFÓCITOS T, CITOCINAS, ADENOSINA TRIFOSFATO, DIFERENCIAÇÃO CELULAR, MALÁRIA, MITOCÔNDRIAS, PLASMODIUM, TRANSDUÇÃO DE SINAL CELULAR
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SALLES, Érika Machado de et al. P2RX7 signaling drives the differentiation of Th1 cells through metabolic reprogramming for aerobic glycolysis. Frontiers in Immunology, v. 14, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2023.1140426. Acesso em: 02 jun. 2024.
APA
Salles, É. M. de, Raeder, P. L., Angeli, C. B., Santiago, V. F., Souza, C. N. de, Ramalho, T., et al. (2023). P2RX7 signaling drives the differentiation of Th1 cells through metabolic reprogramming for aerobic glycolysis. Frontiers in Immunology, 14, 1-12. doi:10.3389/fimmu.2023.1140426
NLM
Salles ÉM de, Raeder PL, Angeli CB, Santiago VF, Souza CN de, Ramalho T, Câmara NOS, Palmisano G, Alvarez JM, Lima MRD'I. P2RX7 signaling drives the differentiation of Th1 cells through metabolic reprogramming for aerobic glycolysis [Internet]. Frontiers in Immunology. 2023 ; 14 1-12.[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fimmu.2023.1140426
Vancouver
Salles ÉM de, Raeder PL, Angeli CB, Santiago VF, Souza CN de, Ramalho T, Câmara NOS, Palmisano G, Alvarez JM, Lima MRD'I. P2RX7 signaling drives the differentiation of Th1 cells through metabolic reprogramming for aerobic glycolysis [Internet]. Frontiers in Immunology. 2023 ; 14 1-12.[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fimmu.2023.1140426
Artigo de periodico
Vitamin D and chronic kidney disease: insights on lipid metabolism of tubular epithelial cell and macrophages in tubulointerstitial fibrosis (2023)
Gonçalves, Luís Eduardo D.; Silva, Magaiver Andrade ; Basso, Paulo José ; Câmara, Niels Olsen Saraiva
Source: Frontiers in Physiology. Unidade: ICB
Subjects: IMUNOLOGIA, MATRIZ EXTRACELULAR, CÉLULAS EPITELIAIS, TÚBULOS RENAIS, NEFROPATIAS, LIPÍDEOS, MACRÓFAGOS, VITAMINA D
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONÇALVES, Luís Eduardo D. et al. Vitamin D and chronic kidney disease: insights on lipid metabolism of tubular epithelial cell and macrophages in tubulointerstitial fibrosis. Frontiers in Physiology, v. 14, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.3389/fphys.2023.1145233. Acesso em: 02 jun. 2024.
APA
Gonçalves, L. E. D., Silva, M. A., Basso, P. J., & Câmara, N. O. S. (2023). Vitamin D and chronic kidney disease: insights on lipid metabolism of tubular epithelial cell and macrophages in tubulointerstitial fibrosis. Frontiers in Physiology, 14, 1-11. doi:10.3389/fphys.2023.1145233
NLM
Gonçalves LED, Silva MA, Basso PJ, Câmara NOS. Vitamin D and chronic kidney disease: insights on lipid metabolism of tubular epithelial cell and macrophages in tubulointerstitial fibrosis [Internet]. Frontiers in Physiology. 2023 ; 14 1-11.[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fphys.2023.1145233
Vancouver
Gonçalves LED, Silva MA, Basso PJ, Câmara NOS. Vitamin D and chronic kidney disease: insights on lipid metabolism of tubular epithelial cell and macrophages in tubulointerstitial fibrosis [Internet]. Frontiers in Physiology. 2023 ; 14 1-11.[citado 2024 jun. 02 ] Available from: https://doi.org/10.3389/fphys.2023.1145233
Artigo de periodico
Sirtuins: key pieces in the host response to pathogens’ puzzle (2023)
Silva, Rafael de Freitas e ; Bassi, Gabriela ; Moretti, Nilmar Silvio ; Camara, Niels Olsen Saraiva
Source: Molecular Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, ENZIMAS, BACTÉRIAS, PROTEÍNAS, PROTOZOA, METABOLISMO CELULAR, REGULAÇÃO BACTERIANA DA EXPRESSÃO GÊNICA, SISTEMA IMUNE
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ABNT
SILVA, Rafael de Freitas e et al. Sirtuins: key pieces in the host response to pathogens’ puzzle. Molecular Immunology, v. 160, p. 150-160, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.molimm.2023.06.010. Acesso em: 02 jun. 2024.
APA
Silva, R. de F. e, Bassi, G., Moretti, N. S., & Camara, N. O. S. (2023). Sirtuins: key pieces in the host response to pathogens’ puzzle. Molecular Immunology, 160, 150-160. doi:10.1016/j.molimm.2023.06.010
NLM
Silva R de F e, Bassi G, Moretti NS, Camara NOS. Sirtuins: key pieces in the host response to pathogens’ puzzle [Internet]. Molecular Immunology. 2023 ; 160 150-160.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.molimm.2023.06.010
Vancouver
Silva R de F e, Bassi G, Moretti NS, Camara NOS. Sirtuins: key pieces in the host response to pathogens’ puzzle [Internet]. Molecular Immunology. 2023 ; 160 150-160.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.molimm.2023.06.010
Artigo de periodico
mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction (2023)
Souza, Gabriel Orefice de ; Teixeira, Pryscila Dryelle Sousa ; Camara, Niels Olsen Saraiva ; Donato Junior, José
Source: Cells. Unidade: ICB
Subjects: IMUNOLOGIA, FISIOLOGIA, HIPOTÁLAMO, PROTEÍNAS, CAMUNDONGOS, GLICOSE, ALIMENTAÇÃO ANIMAL, NEURÔNIOS, DIETA ANIMAL, PESOS E MEDIDAS CORPORAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Gabriel Orefice de et al. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction. Cells, v. 12, n. 20, p. 17 , 2023Tradução . . Disponível em: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf. Acesso em: 02 jun. 2024.
APA
Souza, G. O. de, Teixeira, P. D. S., Camara, N. O. S., & Donato Junior, J. (2023). mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction. Cells, 12( 20), 17 . doi:10.3390/cells12202442
NLM
Souza GO de, Teixeira PDS, Camara NOS, Donato Junior J. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction [Internet]. Cells. 2023 ; 12( 20): 17 .[citado 2024 jun. 02 ] Available from: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf
Vancouver
Souza GO de, Teixeira PDS, Camara NOS, Donato Junior J. mTORC1 signaling in AgRP neurons is not required to induce major neuroendocrine adaptations to food restriction [Internet]. Cells. 2023 ; 12( 20): 17 .[citado 2024 jun. 02 ] Available from: https://repositorio.usp.br/directbitstream/54a8af38-0118-46fd-a7ae-d9f06d3df018/mTORC1%20Signaling%20in%20AgRP%20Neurons%20Is%20Not%20Required%20to%20Induce%20Major%20Neuroendocrine%20Adaptations%20to%20Food%20Restriction.pdf
Artigo de periodico
Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis (2023)
Silva, Aline Ignacio Silvestre da; Cipelli, Marcella ; Takiishi, Tatiana ; Aguiar, Cristhiane Favero ; Terra, Fernanda Fernandes ; Nunes, Bruno Ghirotto ; Silva, Eloisa Martins da ; Castoldi, Angela ; Magalhães, Yuli Thamires ; Souza, Tiago Antonio de ; Hiyane, Meire Ioshie ; Oliveira, Vinicius Andrade de ; Forti, Fabio Luis ; Camara, Niels Olsen Saraiva
Source: bioRxiv. Unidades: ICB, IQ
Subjects: COLITE, CÉLULAS MIELOIDES
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ABNT
SILVA, Aline Ignacio Silvestre da et al. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis. bioRxiv, 2023Tradução . . Disponível em: https://doi.org/10.1101/2023.04.26.537895. Acesso em: 02 jun. 2024.
APA
Silva, A. I. S. da, Cipelli, M., Takiishi, T., Aguiar, C. F., Terra, F. F., Nunes, B. G., et al. (2023). Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis. bioRxiv. doi:10.1101/2023.04.26.537895
NLM
Silva AIS da, Cipelli M, Takiishi T, Aguiar CF, Terra FF, Nunes BG, Silva EM da, Castoldi A, Magalhães YT, Souza TA de, Hiyane MI, Oliveira VA de, Forti FL, Camara NOS. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis [Internet]. bioRxiv. 2023 ;[citado 2024 jun. 02 ] Available from: https://doi.org/10.1101/2023.04.26.537895
Vancouver
Silva AIS da, Cipelli M, Takiishi T, Aguiar CF, Terra FF, Nunes BG, Silva EM da, Castoldi A, Magalhães YT, Souza TA de, Hiyane MI, Oliveira VA de, Forti FL, Camara NOS. Lack of mTORC2 signaling in CD11c+ myeloid cells inhibits their migration and ameliorates experimental colitis [Internet]. bioRxiv. 2023 ;[citado 2024 jun. 02 ] Available from: https://doi.org/10.1101/2023.04.26.537895
Artigo de periodico
The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism (2023)
Foresto Neto, Orestes ; Silva, Ana Ruth Paolinetti Alves da ; Cipelli, Marcella ; Santana-Novelli, Fernanda Paula Roncon Santana ; Camara, Niels Olsen Saraiva
Source: Kidney Research and Clinical Practice. Unidade: ICB
Subjects: IMUNOLOGIA, SISTEMA IMUNE, METABOLISMO CELULAR, NEFROPATIAS, BIOTA INTESTINAL, ANÓXIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FORESTO NETO, Orestes et al. The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism. Kidney Research and Clinical Practice, v. 42, n. 5, p. 561-578, 2023Tradução . . Disponível em: https://doi.org/10.23876/j.krcp.23.012. Acesso em: 02 jun. 2024.
APA
Foresto Neto, O., Silva, A. R. P. A. da, Cipelli, M., Santana-Novelli, F. P. R. S., & Camara, N. O. S. (2023). The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism. Kidney Research and Clinical Practice, 42( 5), 561-578. doi:10.23876/j.krcp.23.012. Epub 2023 Jun 15.
NLM
Foresto Neto O, Silva ARPA da, Cipelli M, Santana-Novelli FPRS, Camara NOS. The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism [Internet]. Kidney Research and Clinical Practice. 2023 ; 42( 5): 561-578.[citado 2024 jun. 02 ] Available from: https://doi.org/10.23876/j.krcp.23.012
Vancouver
Foresto Neto O, Silva ARPA da, Cipelli M, Santana-Novelli FPRS, Camara NOS. The impact of hypoxia-inducible factors in the pathogenesis of kidney diseases: a link through cell metabolism [Internet]. Kidney Research and Clinical Practice. 2023 ; 42( 5): 561-578.[citado 2024 jun. 02 ] Available from: https://doi.org/10.23876/j.krcp.23.012
Artigo de periodico
AIM2 promotes TH17 cells differentiation by regulating RORgt transcription activity (2023)
Leite, Jefferson Antonio ; Menezes, Luísa ; Martins, Eloisa ; Rodrigues, Tamara Silva; Tavares, Lucas ; Ebering, Anna ; Schelmbauer, Carsten ; Cebinelli, Guilherme Cesar Martelossi ; Zinina, Valeriya ; Golden, Artemy ; Soshnikova, Natalia ; Zamboni, Dario S. ; Cunha, Fernando Q. ; Magdalena Huber ; Waisman, Ari ; Silva, João Santana da ; Carlos, Daniela ; Câmara, Niels Olsen Saraiva
Source: iScience. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, CAMUNDONGOS, PROTEÍNAS, DIFERENCIAÇÃO CELULAR, LINFÓCITOS T, CÉLULAS SANGUÍNEAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEITE, Jefferson Antonio et al. AIM2 promotes TH17 cells differentiation by regulating RORgt transcription activity. iScience, v. 26, n. 11, p. art. 108134. 19 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.108134. Acesso em: 02 jun. 2024.
APA
Leite, J. A., Menezes, L., Martins, E., Rodrigues, T. S., Tavares, L., Ebering, A., et al. (2023). AIM2 promotes TH17 cells differentiation by regulating RORgt transcription activity. iScience, 26( 11), art. 108134. 19 . doi:10.1016/j.isci.2023.108134
NLM
Leite JA, Menezes L, Martins E, Rodrigues TS, Tavares L, Ebering A, Schelmbauer C, Cebinelli GCM, Zinina V, Golden A, Soshnikova N, Zamboni DS, Cunha FQ, Magdalena Huber, Waisman A, Silva JS da, Carlos D, Câmara NOS. AIM2 promotes TH17 cells differentiation by regulating RORgt transcription activity [Internet]. iScience. 2023 ; 26( 11): art. 108134. 19 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.isci.2023.108134
Vancouver
Leite JA, Menezes L, Martins E, Rodrigues TS, Tavares L, Ebering A, Schelmbauer C, Cebinelli GCM, Zinina V, Golden A, Soshnikova N, Zamboni DS, Cunha FQ, Magdalena Huber, Waisman A, Silva JS da, Carlos D, Câmara NOS. AIM2 promotes TH17 cells differentiation by regulating RORgt transcription activity [Internet]. iScience. 2023 ; 26( 11): art. 108134. 19 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1016/j.isci.2023.108134
Artigo de periodico
Cross-sectional analysis reveals autoantibody signatures associated with COVID-19 severity (2023)
Baiocchi, Gabriela Crispim ; Vojdani, Aristo ; Rosenberg, Avi Z. ; Vojdani, Elroy ; Halpert, Gilad ; Yuri Ostrinski ; Israel Zyskind ; Filgueiras, Igor Salerno ; Schimke, Lena Friederike ; Marques, Alexandre Henrique Carvalho ; Giil, Lasse M. ; Lavi, Yael Bublil ; Silverberg, Jonathan I. ; Zimmerman, Jason; Hill, Dana Ashley ; Thornton, Amanda ; Kim, Myungjin; Vito, Roberta De ; Fonseca, Dennyson Leandro Mathias da ; Plaça, Desireé Rodrigues ; Freire, Paula Paccielli ; Carmen Scheibenbogen ; Heidecke, Harald; Lattin, Miriam T.; Ochs, Hans D. ; Gabriela Riemekasten ; Howard Amital ; Yehuda Shoenfeld ; Câmara, Niels Olsen Saraiva ; Calich, Vera Lúcia Garcia ; Marques, Otavio Cabral
Source: Journal of Medical Virology. Unidades: ICB, FCF
Subjects: IMUNOLOGIA, COVID-19, INFECÇÕES POR CORONAVIRUS, INFECÇÕES POR VÍRUS DE RNA, DOENÇAS AUTOIMUNES, AUTOANTICORPOS, IMUNOPROTEÍNAS, CITOCINAS, QUIMIOCINAS, IMUNOGLOBULINAS, ESTUDOS TRANSVERSAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BAIOCCHI, Gabriela Crispim et al. Cross-sectional analysis reveals autoantibody signatures associated with COVID-19 severity. Journal of Medical Virology, v. 95, n. 1, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.1002/jmv.28538. Acesso em: 02 jun. 2024.
APA
Baiocchi, G. C., Vojdani, A., Rosenberg, A. Z., Vojdani, E., Halpert, G., Yuri Ostrinski,, et al. (2023). Cross-sectional analysis reveals autoantibody signatures associated with COVID-19 severity. Journal of Medical Virology, 95( 1), 1-13. doi:10.1002/jmv.28538
NLM
Baiocchi GC, Vojdani A, Rosenberg AZ, Vojdani E, Halpert G, Yuri Ostrinski, Israel Zyskind, Filgueiras IS, Schimke LF, Marques AHC, Giil LM, Lavi YB, Silverberg JI, Zimmerman J, Hill DA, Thornton A, Kim M, Vito RD, Fonseca DLM da, Plaça DR, Freire PP, Carmen Scheibenbogen, Heidecke H, Lattin MT, Ochs HD, Gabriela Riemekasten, Howard Amital, Yehuda Shoenfeld, Câmara NOS, Calich VLG, Marques OC. Cross-sectional analysis reveals autoantibody signatures associated with COVID-19 severity [Internet]. Journal of Medical Virology. 2023 ; 95( 1): 1-13.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1002/jmv.28538
Vancouver
Baiocchi GC, Vojdani A, Rosenberg AZ, Vojdani E, Halpert G, Yuri Ostrinski, Israel Zyskind, Filgueiras IS, Schimke LF, Marques AHC, Giil LM, Lavi YB, Silverberg JI, Zimmerman J, Hill DA, Thornton A, Kim M, Vito RD, Fonseca DLM da, Plaça DR, Freire PP, Carmen Scheibenbogen, Heidecke H, Lattin MT, Ochs HD, Gabriela Riemekasten, Howard Amital, Yehuda Shoenfeld, Câmara NOS, Calich VLG, Marques OC. Cross-sectional analysis reveals autoantibody signatures associated with COVID-19 severity [Internet]. Journal of Medical Virology. 2023 ; 95( 1): 1-13.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1002/jmv.28538
Artigo de periodico
Tear flm immunological profile in patients with ocular graft versus Host Disease (2023)
Serapicos, Patricia ; Kim, Cinthia ; Barros, Sabrina Leite; Jordão, Isa Maria Bastos Mendes Silva ; Hiyane, Meire Ioshie ; Sousa, Luciene Barbosa de ; Zecchin, Victor Gottardello ; Câmara, Niels Olsen Saraiva ; Oliveira, Lauro Augusto de
Source: Ocular Immunology and Inflammation. Unidade: ICB
Subjects: IMUNOLOGIA, DOENÇA ENXERTO-HOSPEDEIRO, OFTALMOPATIAS, QUIMIOCINAS, INTERLEUCINA 8, BIOMARCADORES, MEDULA ÓSSEA, OLHO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SERAPICOS, Patricia et al. Tear flm immunological profile in patients with ocular graft versus Host Disease. Ocular Immunology and Inflammation, v. 31, n. 4, p. 701–709, 2023Tradução . . Disponível em: https://doi.org/10.1080/09273948.2022.2046794. Acesso em: 02 jun. 2024.
APA
Serapicos, P., Kim, C., Barros, S. L., Jordão, I. M. B. M. S., Hiyane, M. I., Sousa, L. B. de, et al. (2023). Tear flm immunological profile in patients with ocular graft versus Host Disease. Ocular Immunology and Inflammation, 31( 4), 701–709. doi:10.1080/09273948.2022.2046794
NLM
Serapicos P, Kim C, Barros SL, Jordão IMBMS, Hiyane MI, Sousa LB de, Zecchin VG, Câmara NOS, Oliveira LA de. Tear flm immunological profile in patients with ocular graft versus Host Disease [Internet]. Ocular Immunology and Inflammation. 2023 ; 31( 4): 701–709.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1080/09273948.2022.2046794
Vancouver
Serapicos P, Kim C, Barros SL, Jordão IMBMS, Hiyane MI, Sousa LB de, Zecchin VG, Câmara NOS, Oliveira LA de. Tear flm immunological profile in patients with ocular graft versus Host Disease [Internet]. Ocular Immunology and Inflammation. 2023 ; 31( 4): 701–709.[citado 2024 jun. 02 ] Available from: https://doi.org/10.1080/09273948.2022.2046794
Artigo de periodico
Transcriptome analysis of six tissues obtained post-mortem from sepsis patients (2023)
Silva, Fabiano Pinheiro da ; Gonçalves, André Nicolau Aquime ; Duarte Neto, Amaro Nunes ; Dias, Thomaz Lüscher ; Barbeiro, Hermes Vieira ; Nakaya, Helder Takashi Imoto ; Camara, Niels Olsen Saraiva
Source: Journal of Cellular and Molecular Medicine. Unidades: FCF, ICB
Subjects: IMUNOLOGIA, SEPSE, SEPTICEMIA, INFECÇÕES BACTERIANAS, CHOQUE SÉPTICO, RNA, SEQUENCIAMENTO GENÉTICO, TRANSCRIÇÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Fabiano Pinheiro da et al. Transcriptome analysis of six tissues obtained post-mortem from sepsis patients. Journal of Cellular and Molecular Medicine, p. 11 , 2023Tradução . . Disponível em: https://doi.org/10.1111/jcmm.17938. Acesso em: 02 jun. 2024.
APA
Silva, F. P. da, Gonçalves, A. N. A., Duarte Neto, A. N., Dias, T. L., Barbeiro, H. V., Nakaya, H. T. I., & Camara, N. O. S. (2023). Transcriptome analysis of six tissues obtained post-mortem from sepsis patients. Journal of Cellular and Molecular Medicine, 11 . doi:10.1111/jcmm.17938
NLM
Silva FP da, Gonçalves ANA, Duarte Neto AN, Dias TL, Barbeiro HV, Nakaya HTI, Camara NOS. Transcriptome analysis of six tissues obtained post-mortem from sepsis patients [Internet]. Journal of Cellular and Molecular Medicine. 2023 ;11 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1111/jcmm.17938
Vancouver
Silva FP da, Gonçalves ANA, Duarte Neto AN, Dias TL, Barbeiro HV, Nakaya HTI, Camara NOS. Transcriptome analysis of six tissues obtained post-mortem from sepsis patients [Internet]. Journal of Cellular and Molecular Medicine. 2023 ;11 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1111/jcmm.17938
Artigo de periodico
Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease (2023)
Cipelli, Marcella ; Silva, Eloisa Martins da ; Câmara, Niels Olsen Saraiva
Source: Current Clinical Microbiology Reports. Unidade: ICB
Subjects: IMUNOLOGIA, ENTEROPATIAS INFLAMATÓRIAS, BIOTA INTESTINAL, SISTEMA GASTROINTESTINAL, INTESTINOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CIPELLI, Marcella e SILVA, Eloisa Martins da e CÂMARA, Niels Olsen Saraiva. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease. Current Clinical Microbiology Reports, p. 11 , 2023Tradução . . Disponível em: https://doi.org/10.1007/s40588-023-00207-4. Acesso em: 02 jun. 2024.
APA
Cipelli, M., Silva, E. M. da, & Câmara, N. O. S. (2023). Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease. Current Clinical Microbiology Reports, 11 . doi:10.1007/s40588-023-00207-4
NLM
Cipelli M, Silva EM da, Câmara NOS. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease [Internet]. Current Clinical Microbiology Reports. 2023 ;11 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1007/s40588-023-00207-4
Vancouver
Cipelli M, Silva EM da, Câmara NOS. Gut microbiota resilience mechanisms against pthogen infection and its role in infammatory Bowel Disease [Internet]. Current Clinical Microbiology Reports. 2023 ;11 .[citado 2024 jun. 02 ] Available from: https://doi.org/10.1007/s40588-023-00207-4

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