Filtros : "ICB" "Indexado na Base de Dados Web of Science" "Journal of Hypertension" Removidos: "HU" "Alam, M F" "Atheneu" "Universidade de Santo Amaro (UNISA)" Limpar

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  • Source: Journal of Hypertension. Unidade: ICB

    Subjects: PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, RATOS WISTAR, NEURÔNIOS, GÂNGLIOS, TRONCO CEREBRAL, SISTEMA NERVOSO CENTRAL

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      MOREIRA, Thiago dos Santos et al. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat. Journal of Hypertension, v. 36, n. 12, p. 2444-2452, 2018Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000001871. Acesso em: 15 jun. 2024.
    • APA

      Moreira, T. dos S., Antunes, V. R., Falquetto, B., & Gonzalez, N. M. (2018). Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat. Journal of Hypertension, 36( 12), 2444-2452. doi:10.1097/HJH.0000000000001871
    • NLM

      Moreira T dos S, Antunes VR, Falquetto B, Gonzalez NM. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat [Internet]. Journal of Hypertension. 2018 ; 36( 12): 2444-2452.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1097/HJH.0000000000001871
    • Vancouver

      Moreira T dos S, Antunes VR, Falquetto B, Gonzalez NM. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat [Internet]. Journal of Hypertension. 2018 ; 36( 12): 2444-2452.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1097/HJH.0000000000001871
  • Source: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      WENCESLAU, Camilla F. e ROSSONI, Luciana Venturini. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, v. 32, n. 3, p. 542-555, 2014Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000000059. Acesso em: 15 jun. 2024.
    • APA

      Wenceslau, C. F., & Rossoni, L. V. (2014). Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, 32( 3), 542-555. doi:10.1097/HJH.0000000000000059
    • NLM

      Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
    • Vancouver

      Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1097/HJH.0000000000000059

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