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Artigo de periodico
Genotoxicity of ultraviolet light and sunlight in the bacterium Caulobacter crescentus: wavelength-dependence (2024)
León, Fabiana Fuentes ; Quintero-Ruiz, Nathalia ; Fernández-Silva, Frank Sidney ; Tamayo, Marioly Vernhes; Sánchez-Lamar, Angel ; Munford, Veridiana ; Menck, Carlos Frederico Martins ; Galhardo, Rodrigo da Silva
Source: Mutation Research - Genetic Toxicology and Environmental Mutagenesis. Unidade: ICB
Subjects: MICROBIOLOGIA, NUCLEOTÍDEOS, ANTIBIÓTICOS, GENÉTICA BACTERIANA, DANO AO DNA, RADIAÇÃO SOLAR E TERRESTRE, RADIAÇÃO ULTRAVIOLETA, OZÔNIO, MUTAÇÃO GENÉTICA, CITOTOXINAS, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS
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LEÓN, Fabiana Fuentes et al. Genotoxicity of ultraviolet light and sunlight in the bacterium Caulobacter crescentus: wavelength-dependence. Mutation Research - Genetic Toxicology and Environmental Mutagenesis, v. 894, p. 6 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.mrgentox.2024.503727. Acesso em: 03 jun. 2024.
APA
León, F. F., Quintero-Ruiz, N., Fernández-Silva, F. S., Tamayo, M. V., Sánchez-Lamar, A., Munford, V., et al. (2024). Genotoxicity of ultraviolet light and sunlight in the bacterium Caulobacter crescentus: wavelength-dependence. Mutation Research - Genetic Toxicology and Environmental Mutagenesis, 894, 6 . doi:10.1016/j.mrgentox.2024.503727
NLM
León FF, Quintero-Ruiz N, Fernández-Silva FS, Tamayo MV, Sánchez-Lamar A, Munford V, Menck CFM, Galhardo R da S. Genotoxicity of ultraviolet light and sunlight in the bacterium Caulobacter crescentus: wavelength-dependence [Internet]. Mutation Research - Genetic Toxicology and Environmental Mutagenesis. 2024 ; 894 6 .[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mrgentox.2024.503727
Vancouver
León FF, Quintero-Ruiz N, Fernández-Silva FS, Tamayo MV, Sánchez-Lamar A, Munford V, Menck CFM, Galhardo R da S. Genotoxicity of ultraviolet light and sunlight in the bacterium Caulobacter crescentus: wavelength-dependence [Internet]. Mutation Research - Genetic Toxicology and Environmental Mutagenesis. 2024 ; 894 6 .[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mrgentox.2024.503727
Artigo de periodico
Recombinant production of a highly efficient photolyase from Thermus thermophilus (2024)
Obreque, Karin Torres ; Gonçalves, Felipe Gobbi; Ferraro, Rafael Bertelli ; León, Fabiana Fuentes ; Silva, Tales Alexandre da Costa e ; Perego, Patrizia ; Rangel-Yagui, Carlota de Oliveira ; Monteiro, Gisele ; Menck, Carlos Frederico Martins
Source: Biotechnology Journal. Unidades: FCF, ICB
Subjects: MICROBIOLOGIA, DNA, REPARAÇÃO DE DNA, REPARAÇÃO DO DANO, BACTÉRIAS GRAM-NEGATIVAS, GENÉTICA BACTERIANA, RADIAÇÃO ULTRAVIOLETA, MUTAGÊNESE, ATIVAÇÃO ENZIMÁTICA
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OBREQUE, Karin Torres et al. Recombinant production of a highly efficient photolyase from Thermus thermophilus. Biotechnology Journal, v. 19, n. 2, p. 9 , 2024Tradução . . Disponível em: https://doi.org/10.1002/biot.202300325. Acesso em: 03 jun. 2024.
APA
Obreque, K. T., Gonçalves, F. G., Ferraro, R. B., León, F. F., Silva, T. A. da C. e, Perego, P., et al. (2024). Recombinant production of a highly efficient photolyase from Thermus thermophilus. Biotechnology Journal, 19( 2), 9 . doi:10.1002/biot.202300325
NLM
Obreque KT, Gonçalves FG, Ferraro RB, León FF, Silva TA da C e, Perego P, Rangel-Yagui C de O, Monteiro G, Menck CFM. Recombinant production of a highly efficient photolyase from Thermus thermophilus [Internet]. Biotechnology Journal. 2024 ; 19( 2): 9 .[citado 2024 jun. 03 ] Available from: https://doi.org/10.1002/biot.202300325
Vancouver
Obreque KT, Gonçalves FG, Ferraro RB, León FF, Silva TA da C e, Perego P, Rangel-Yagui C de O, Monteiro G, Menck CFM. Recombinant production of a highly efficient photolyase from Thermus thermophilus [Internet]. Biotechnology Journal. 2024 ; 19( 2): 9 .[citado 2024 jun. 03 ] Available from: https://doi.org/10.1002/biot.202300325
Artigo de periodico
Evaluation of Meibomian gland dysfunction using meibography in patients with xeroderma pigmentosum (2024)
Marcos, Allexya Affonso Antunes ; Freitas, Denise ; Hazarbassanov, Rossen Mihaylov ; Fernandes, Arthur Gustavo ; Fernandes, Arthur Gustavo ; Castro, Ligia Pereira ; Melo, Danilo Batista Vieira de ; Menck, Carlos Frederico Martins
Source: Arquivos Brasileiros de Oftalmologia. Unidade: ICB
Subjects: MICROBIOLOGIA, DOENÇAS PALPEBRAIS, REPARAÇÃO DE DNA, PÁLPEBRAS, DIAGNÓSTICO POR IMAGEM, OLHO, DOENÇAS DA PELE E DO TECIDO CONJUNTIVO, DERMATOPATIAS, NEOPLASIAS OCULARES, ESTUDOS TRANSVERSAIS
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MARCOS, Allexya Affonso Antunes et al. Evaluation of Meibomian gland dysfunction using meibography in patients with xeroderma pigmentosum. Arquivos Brasileiros de Oftalmologia, v. 87, n. 2, 2024Tradução . . Disponível em: https://doi.org/10.5935/0004-2749.2022-0319. Acesso em: 03 jun. 2024.
APA
Marcos, A. A. A., Freitas, D., Hazarbassanov, R. M., Fernandes, A. G., Fernandes, A. G., Castro, L. P., et al. (2024). Evaluation of Meibomian gland dysfunction using meibography in patients with xeroderma pigmentosum. Arquivos Brasileiros de Oftalmologia, 87( 2). doi:10.5935/0004-2749.2022-0319
NLM
Marcos AAA, Freitas D, Hazarbassanov RM, Fernandes AG, Fernandes AG, Castro LP, Melo DBV de, Menck CFM. Evaluation of Meibomian gland dysfunction using meibography in patients with xeroderma pigmentosum [Internet]. Arquivos Brasileiros de Oftalmologia. 2024 ; 87( 2):[citado 2024 jun. 03 ] Available from: https://doi.org/10.5935/0004-2749.2022-0319
Vancouver
Marcos AAA, Freitas D, Hazarbassanov RM, Fernandes AG, Fernandes AG, Castro LP, Melo DBV de, Menck CFM. Evaluation of Meibomian gland dysfunction using meibography in patients with xeroderma pigmentosum [Internet]. Arquivos Brasileiros de Oftalmologia. 2024 ; 87( 2):[citado 2024 jun. 03 ] Available from: https://doi.org/10.5935/0004-2749.2022-0319
Artigo de periodico
Exploring pradimicin-IRD antineoplastic mechanisms and related DNA repair pathways (2023)
Almeida, Larissa Costa de ; Calil, Felipe Antunes ; Moreno, Natália Cestari ; Teixeira, Paula Rezende ; Moraes, Luiz Alberto Beraldo de ; Jimenez, Paula Christine ; Menck, Carlos Frederico Martins ; Machado Neto, João Agostinho ; Lotufo, Leticia Veras Costa
Source: Chemico-Biological Interactions. Unidades: FFCLRP, ICB, IQ
Subjects: FARMACOLOGIA, MICROBIOLOGIA, ANTINEOPLÁSICOS, REPARAÇÃO DE DNA, DANO AO DNA, QUIMIOTERÁPICOS, NUCLEOTÍDEOS
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ALMEIDA, Larissa Costa de et al. Exploring pradimicin-IRD antineoplastic mechanisms and related DNA repair pathways. Chemico-Biological Interactions, v. 371, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2023.110342. Acesso em: 03 jun. 2024.
APA
Almeida, L. C. de, Calil, F. A., Moreno, N. C., Teixeira, P. R., Moraes, L. A. B. de, Jimenez, P. C., et al. (2023). Exploring pradimicin-IRD antineoplastic mechanisms and related DNA repair pathways. Chemico-Biological Interactions, 371, 1-10. doi:10.1016/j.cbi.2023.110342
NLM
Almeida LC de, Calil FA, Moreno NC, Teixeira PR, Moraes LAB de, Jimenez PC, Menck CFM, Machado Neto JA, Lotufo LVC. Exploring pradimicin-IRD antineoplastic mechanisms and related DNA repair pathways [Internet]. Chemico-Biological Interactions. 2023 ; 371 1-10.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.cbi.2023.110342
Vancouver
Almeida LC de, Calil FA, Moreno NC, Teixeira PR, Moraes LAB de, Jimenez PC, Menck CFM, Machado Neto JA, Lotufo LVC. Exploring pradimicin-IRD antineoplastic mechanisms and related DNA repair pathways [Internet]. Chemico-Biological Interactions. 2023 ; 371 1-10.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.cbi.2023.110342
Artigo de periodico
Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients (2023)
Yurchenko, Andrey A. ; Rajabi, Fatemeh ; Braz-Petta, Tirzah ; Fassihi, Hiva ; Lehmann, Alan ; Nishigori, Chikako ; Wang, Jinxin; Padioleau, Ismael ; Gunbin, Konstantin ; Panunzi, Leonardo ; Morice-Picard, Fanny ; Laplante, Pierre; Robert, Caroline ; Kannouche, Patricia L. ; Sarasin, Alain ; Nikolaev, Sergey I. ; Menck, Carlos Frederico Martins
Source: Nature Communications. Unidade: ICB
Subjects: MICROBIOLOPGIA, MUTAÇÃO GENÉTICA, REPARAÇÃO DE DNA, NEOPLASIAS CUTÂNEAS, DERMATOPATIAS, DOENÇAS GENÉTICAS, RADIAÇÃO ULTRAVIOLETA, GENÔMICA
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YURCHENKO, Andrey A. et al. Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients. Nature Communications, v. 14, p. 1-17, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41467-023-38311-0. Acesso em: 03 jun. 2024.
APA
Yurchenko, A. A., Rajabi, F., Braz-Petta, T., Fassihi, H., Lehmann, A., Nishigori, C., et al. (2023). Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients. Nature Communications, 14, 1-17. doi:10.1038/s41467-023-38311-0
NLM
Yurchenko AA, Rajabi F, Braz-Petta T, Fassihi H, Lehmann A, Nishigori C, Wang J, Padioleau I, Gunbin K, Panunzi L, Morice-Picard F, Laplante P, Robert C, Kannouche PL, Sarasin A, Nikolaev SI, Menck CFM. Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients [Internet]. Nature Communications. 2023 ; 14 1-17.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1038/s41467-023-38311-0
Vancouver
Yurchenko AA, Rajabi F, Braz-Petta T, Fassihi H, Lehmann A, Nishigori C, Wang J, Padioleau I, Gunbin K, Panunzi L, Morice-Picard F, Laplante P, Robert C, Kannouche PL, Sarasin A, Nikolaev SI, Menck CFM. Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients [Internet]. Nature Communications. 2023 ; 14 1-17.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1038/s41467-023-38311-0
Artigo de periodico
Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells (2023)
Florentino, Pilar Tavares Veras ; Vitorino, Francisca Nathália de Luna ; Mendes, D.; Cunha, Julia Pinheiro Chagas da ; Menck, Carlos Frederico Martins
Source: Journal of Proteomics. Unidade: ICB
Subjects: MICROBIOLOGIA, CROMATINA, DANO AO DNA, TRYPANOSOMA CRUZI, ANIMAIS PARASITOS, DOENÇA DE CHAGAS, APOPTOSE, PROTEÔMICA, DNA
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FLORENTINO, Pilar Tavares Veras et al. Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells. Journal of Proteomics, v. 272, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.jprot.2022.104773. Acesso em: 03 jun. 2024.
APA
Florentino, P. T. V., Vitorino, F. N. de L., Mendes, D., Cunha, J. P. C. da, & Menck, C. F. M. (2023). Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells. Journal of Proteomics, 272, 1-11. doi:10.1016/j.jprot.2022.104773
NLM
Florentino PTV, Vitorino FN de L, Mendes D, Cunha JPC da, Menck CFM. Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells [Internet]. Journal of Proteomics. 2023 ; 272 1-11.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.jprot.2022.104773
Vancouver
Florentino PTV, Vitorino FN de L, Mendes D, Cunha JPC da, Menck CFM. Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells [Internet]. Journal of Proteomics. 2023 ; 272 1-11.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.jprot.2022.104773
Artigo de periodico
The accurate bypass of pyrimidine dimers by DNA polymerase eta contributes to ultraviolet-induced mutagenesis (2023)
Menck, Carlos Frederico Martins ; Galhardo, Rodrigo da Silva ; Quinet, Annabel
Source: Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis. Unidade: ICB
Subjects: MICROBIOLOGIA, RADIAÇÃO ULTRAVIOLETA, DANO AO DNA, DNA POLIMERASES, REPLICAÇÃO DO DNA, MUTAGÊNESE, DOENÇAS GENÉTICAS, NEOPLASIAS CUTÂNEAS, MUTAÇÃO GENÉTICA
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MENCK, Carlos Frederico Martins e GALHARDO, Rodrigo da Silva e QUINET, Annabel. The accurate bypass of pyrimidine dimers by DNA polymerase eta contributes to ultraviolet-induced mutagenesis. Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, v. 828, p. 10 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.mrfmmm.2023.111840. Acesso em: 03 jun. 2024.
APA
Menck, C. F. M., Galhardo, R. da S., & Quinet, A. (2023). The accurate bypass of pyrimidine dimers by DNA polymerase eta contributes to ultraviolet-induced mutagenesis. Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 828, 10 . doi:10.1016/j.mrfmmm.2023.111840
NLM
Menck CFM, Galhardo R da S, Quinet A. The accurate bypass of pyrimidine dimers by DNA polymerase eta contributes to ultraviolet-induced mutagenesis [Internet]. Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis. 2023 ; 828 10 .[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mrfmmm.2023.111840
Vancouver
Menck CFM, Galhardo R da S, Quinet A. The accurate bypass of pyrimidine dimers by DNA polymerase eta contributes to ultraviolet-induced mutagenesis [Internet]. Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis. 2023 ; 828 10 .[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mrfmmm.2023.111840
Artigo de periodico
Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice (2022)
Kajitani, Gustavo Satoru; Quayle, Carolina de Barros Reis ; Garcia, Camila Carrião Machado ; Fotoran, Wesley Luzetti ; Santos, Juliana F. R. dos; Horst, Gijsbertus T. J. van der ; Hoeijmakers, Jan H. J. ; Menck, Carlos Frederico Martins
Source: Frontiers in Immunology. Unidades: ICB, IB, BIOTECNOLOGIA
Subjects: MICROBIOLOGIA, RADIAÇÃO ULTRAVIOLETA, REPARAÇÃO DE DNA, CÉLULAS MORTAS, NEOPLASIAS CUTÂNEAS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, PROLIFERAÇÃO CELULAR
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KAJITANI, Gustavo Satoru et al. Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice. Frontiers in Immunology, v. 13, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.800606. Acesso em: 03 jun. 2024.
APA
Kajitani, G. S., Quayle, C. de B. R., Garcia, C. C. M., Fotoran, W. L., Santos, J. F. R. dos, Horst, G. T. J. van der, et al. (2022). Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice. Frontiers in Immunology, 13, 1-10. doi:10.3389/fimmu.2022.800606
NLM
Kajitani GS, Quayle C de BR, Garcia CCM, Fotoran WL, Santos JFR dos, Horst GTJ van der, Hoeijmakers JHJ, Menck CFM. Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice [Internet]. Frontiers in Immunology. 2022 ; 13 1-10.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3389/fimmu.2022.800606
Vancouver
Kajitani GS, Quayle C de BR, Garcia CCM, Fotoran WL, Santos JFR dos, Horst GTJ van der, Hoeijmakers JHJ, Menck CFM. Photorepair of either CPD or 6-4PP DNA lesions in basal Keratinocytes attenuates ultraviolet-induced skin effects in nucleotide excision repair deficient mice [Internet]. Frontiers in Immunology. 2022 ; 13 1-10.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3389/fimmu.2022.800606
Artigo de periodico
ATM pathway is essential for HPV–positive human cervical cancer-derived cell lines viability and proliferation (2022)
Abjaude, Walason da Silva; Prati, Bruna; Munford, Veridiana ; Montenegro, Aline; Lino, Vanesca de Souza ; Herbster, Suellen da Silva Gomes ; Rabachini, Tatiana ; Termini, Lara ; Menck, Carlos Frederico Martins ; Boccardo, Enrique
Source: Pathogens. Unidades: ICB, FM
Subjects: MICROBIOLOGIA, NEOPLASIAS DO COLO UTERINO, PROLIFERAÇÃO CELULAR, HPV, CÉLULAS CULTIVADAS DE TUMOR, REPLICAÇÃO VIRAL, REPLICAÇÃO DO DNA
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ABJAUDE, Walason da Silva et al. ATM pathway is essential for HPV–positive human cervical cancer-derived cell lines viability and proliferation. Pathogens, v. 11, n. 6, p. 1-19, 2022Tradução . . Disponível em: https://doi.org/10.3390/pathogens11060637. Acesso em: 03 jun. 2024.
APA
Abjaude, W. da S., Prati, B., Munford, V., Montenegro, A., Lino, V. de S., Herbster, S. da S. G., et al. (2022). ATM pathway is essential for HPV–positive human cervical cancer-derived cell lines viability and proliferation. Pathogens, 11( 6), 1-19. doi:10.3390/pathogens11060637
NLM
Abjaude W da S, Prati B, Munford V, Montenegro A, Lino V de S, Herbster S da SG, Rabachini T, Termini L, Menck CFM, Boccardo E. ATM pathway is essential for HPV–positive human cervical cancer-derived cell lines viability and proliferation [Internet]. Pathogens. 2022 ; 11( 6): 1-19.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3390/pathogens11060637
Vancouver
Abjaude W da S, Prati B, Munford V, Montenegro A, Lino V de S, Herbster S da SG, Rabachini T, Termini L, Menck CFM, Boccardo E. ATM pathway is essential for HPV–positive human cervical cancer-derived cell lines viability and proliferation [Internet]. Pathogens. 2022 ; 11( 6): 1-19.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3390/pathogens11060637
Artigo de periodico
Detection of post-replicative gaps accumulation and repair in human cells using the DNA fiber assay (2022)
Martins, Davi Jardim ; Tirman, Stephanie; Quinet, Annabel ; Menck, Carlos Frederico Martins
Source: Journal of Visualized Experiments. Unidade: ICB
Subjects: MICROBIOLOGIA, REPLICAÇÃO DO DNA, REPARAÇÃO DE DNA, DNA, NUCLEOTÍDEOS, GENOMAS
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MARTINS, Davi Jardim et al. Detection of post-replicative gaps accumulation and repair in human cells using the DNA fiber assay. Journal of Visualized Experiments, v. 180, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3791/63448. Acesso em: 03 jun. 2024.
APA
Martins, D. J., Tirman, S., Quinet, A., & Menck, C. F. M. (2022). Detection of post-replicative gaps accumulation and repair in human cells using the DNA fiber assay. Journal of Visualized Experiments, 180, 1-14. doi:10.3791/63448
NLM
Martins DJ, Tirman S, Quinet A, Menck CFM. Detection of post-replicative gaps accumulation and repair in human cells using the DNA fiber assay [Internet]. Journal of Visualized Experiments. 2022 ; 180 1-14.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3791/63448
Vancouver
Martins DJ, Tirman S, Quinet A, Menck CFM. Detection of post-replicative gaps accumulation and repair in human cells using the DNA fiber assay [Internet]. Journal of Visualized Experiments. 2022 ; 180 1-14.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3791/63448
Artigo de periodico
Ru(II)/amino acid complexes inhibit the progression of breast cancer cells through multiple mechanism-induced apoptosis (2022)
Andrade, Francyelli Mello ; Guedes, Adriana P. M.; Pires, Wanessa C.; Sá, Vivianne de Souza Velozo ; Delmond, Kezia A.; Mendes, Davi ; Silva, Matheus Molina ; Matuda, Larissa ; Sousa, Maria Alice Montes de ; Reis, Paulo Melo ; Gomes, Clever C. ; Castro, Carlos Henrique; Almeida, Márcio Aurélio Pinheiro; Menck, Carlos Frederico Martins ; Batista, Alzir Azevedo ; Burikhanov, Ravshan ; Rangnekar, Vivek M. ; Lacerda, Elisângela Silveira
Source: Journal of Inorganic Biochemistry. Unidade: ICB
Subjects: MICROBIOLOGIA, APOPTOSE, CÉLULAS MORTAS, AMINOÁCIDOS, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS MAMÁRIAS, RUTÊNIO, METÁSTASE NEOPLÁSICA
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ANDRADE, Francyelli Mello et al. Ru(II)/amino acid complexes inhibit the progression of breast cancer cells through multiple mechanism-induced apoptosis. Journal of Inorganic Biochemistry, v. 226, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.jinorgbio.2021.111625. Acesso em: 03 jun. 2024.
APA
Andrade, F. M., Guedes, A. P. M., Pires, W. C., Sá, V. de S. V., Delmond, K. A., Mendes, D., et al. (2022). Ru(II)/amino acid complexes inhibit the progression of breast cancer cells through multiple mechanism-induced apoptosis. Journal of Inorganic Biochemistry, 226, 1-12. doi:10.1016/j.jinorgbio.2021.111625
NLM
Andrade FM, Guedes APM, Pires WC, Sá V de SV, Delmond KA, Mendes D, Silva MM, Matuda L, Sousa MAM de, Reis PM, Gomes CC, Castro CH, Almeida MAP, Menck CFM, Batista AA, Burikhanov R, Rangnekar VM, Lacerda ES. Ru(II)/amino acid complexes inhibit the progression of breast cancer cells through multiple mechanism-induced apoptosis [Internet]. Journal of Inorganic Biochemistry. 2022 ; 226 1-12.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.jinorgbio.2021.111625
Vancouver
Andrade FM, Guedes APM, Pires WC, Sá V de SV, Delmond KA, Mendes D, Silva MM, Matuda L, Sousa MAM de, Reis PM, Gomes CC, Castro CH, Almeida MAP, Menck CFM, Batista AA, Burikhanov R, Rangnekar VM, Lacerda ES. Ru(II)/amino acid complexes inhibit the progression of breast cancer cells through multiple mechanism-induced apoptosis [Internet]. Journal of Inorganic Biochemistry. 2022 ; 226 1-12.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.jinorgbio.2021.111625
Artigo de periodico
Current state of knowledge of human DNA polymerase eta protein structure and disease-causing mutations (2022)
Feltes, Bruno César ; Menck, Carlos Frederico Martins
Source: Mutation Research - Reviews. Unidade: ICB
Subjects: MICROBIOLOGIA, REPLICAÇÃO DO DNA, DNA POLIMERASES, DANO AO DNA, MUTAÇÃO GENÉTICA, DOENÇAS GENÉTICAS, NEOPLASIAS CUTÂNEAS, CÉLULAS CULTIVADAS DE TUMOR
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FELTES, Bruno César e MENCK, Carlos Frederico Martins. Current state of knowledge of human DNA polymerase eta protein structure and disease-causing mutations. Mutation Research - Reviews, v. 790, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mrrev.2022.108436. Acesso em: 03 jun. 2024.
APA
Feltes, B. C., & Menck, C. F. M. (2022). Current state of knowledge of human DNA polymerase eta protein structure and disease-causing mutations. Mutation Research - Reviews, 790, 1-15. doi:10.1016/j.mrrev.2022.108436
NLM
Feltes BC, Menck CFM. Current state of knowledge of human DNA polymerase eta protein structure and disease-causing mutations [Internet]. Mutation Research - Reviews. 2022 ; 790 1-15.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mrrev.2022.108436
Vancouver
Feltes BC, Menck CFM. Current state of knowledge of human DNA polymerase eta protein structure and disease-causing mutations [Internet]. Mutation Research - Reviews. 2022 ; 790 1-15.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mrrev.2022.108436
Artigo de periodico
XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil (2022)
Castro, Ligia Pereira ; Vieira, Danilo Batista; Souza, Tiago Antonio de; Timoteo, Ana Rafaela de Souza ; Coutinho, Jessica Dayanna Landivar; Almeida, Isabel Cristina Pinheiro de ; Henriques, Sheila Ramos de Miranda; Azevedo, Fabio Medeiros de; Rosa, Reginaldo Cruz Alves ; Kannouche, Patricia L. ; Sarasin, Alain ; Menck, Carlos Frederico Martins ; Petta, Tirzah Braz
Source: Frontiers in Genetics. Unidades: ICB, FMRP
Subjects: MICROBIOLOGIA, NEOPLASIAS CUTÂNEAS, DOENÇAS GENÉTICAS, RADIAÇÃO ULTRAVIOLETA, CÉLULAS CULTIVADAS DE TUMOR, SEQUENCIAMENTO GENÉTICO, GENES, MUTAÇÃO GENÉTICA, REPARAÇÃO DE DNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CASTRO, Ligia Pereira et al. XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil. Frontiers in Genetics, v. 12, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.3389/fgene.2021.784963. Acesso em: 03 jun. 2024.
APA
Castro, L. P., Vieira, D. B., Souza, T. A. de, Timoteo, A. R. de S., Coutinho, J. D. L., Almeida, I. C. P. de, et al. (2022). XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil. Frontiers in Genetics, 12, 1-8. doi:10.3389/fgene.2021.784963
NLM
Castro LP, Vieira DB, Souza TA de, Timoteo AR de S, Coutinho JDL, Almeida ICP de, Henriques SR de M, Azevedo FM de, Rosa RCA, Kannouche PL, Sarasin A, Menck CFM, Petta TB. XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil [Internet]. Frontiers in Genetics. 2022 ; 12 1-8.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3389/fgene.2021.784963
Vancouver
Castro LP, Vieira DB, Souza TA de, Timoteo AR de S, Coutinho JDL, Almeida ICP de, Henriques SR de M, Azevedo FM de, Rosa RCA, Kannouche PL, Sarasin A, Menck CFM, Petta TB. XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil [Internet]. Frontiers in Genetics. 2022 ; 12 1-8.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3389/fgene.2021.784963
Artigo de periodico
A therapeutic DNA vaccine and gemcitabine act synergistically to eradicate HPV-associated tumors in a preclinical model (2021)
Silva, Jamile Ramos da ; Moreno, Ana Carolina Ramos ; Sales, Natiely Silva ; Silva, Mariângela de Oliveira; Aps, Luana Raposo de Melo Morais ; Ribeiro, Bruna Felicio Milazzotto Maldonado Porchia ; Rodrigues, Karine Bitencourt ; Moreno, Natália Cestari ; Carvalho, Aléxia Adrianne Venceslau ; Menck, Carlos Frederico Martins ; Diniz, Mariana de Oliveira; Ferreira, Luís Carlos de Souza
Source: OncoImmunology. Unidades: ICB, BIOTECNOLOGIA, IQ
Subjects: MICROBIOLOGIA, NEOPLASIAS, HPV, QUIMIOTERAPIA, VACINAS VIRAIS, CÉLULAS CULTIVADAS DE TUMOR, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Jamile Ramos da et al. A therapeutic DNA vaccine and gemcitabine act synergistically to eradicate HPV-associated tumors in a preclinical model. OncoImmunology, v. 10, n. 1, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.1080/2162402X.2021.1949896. Acesso em: 03 jun. 2024.
APA
Silva, J. R. da, Moreno, A. C. R., Sales, N. S., Silva, M. de O., Aps, L. R. de M. M., Ribeiro, B. F. M. M. P., et al. (2021). A therapeutic DNA vaccine and gemcitabine act synergistically to eradicate HPV-associated tumors in a preclinical model. OncoImmunology, 10( 1), 1-13. doi:10.1080/2162402X.2021.1949896
NLM
Silva JR da, Moreno ACR, Sales NS, Silva M de O, Aps LR de MM, Ribeiro BFMMP, Rodrigues KB, Moreno NC, Carvalho AAV, Menck CFM, Diniz M de O, Ferreira LC de S. A therapeutic DNA vaccine and gemcitabine act synergistically to eradicate HPV-associated tumors in a preclinical model [Internet]. OncoImmunology. 2021 ; 10( 1): 1-13.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1080/2162402X.2021.1949896
Vancouver
Silva JR da, Moreno ACR, Sales NS, Silva M de O, Aps LR de MM, Ribeiro BFMMP, Rodrigues KB, Moreno NC, Carvalho AAV, Menck CFM, Diniz M de O, Ferreira LC de S. A therapeutic DNA vaccine and gemcitabine act synergistically to eradicate HPV-associated tumors in a preclinical model [Internet]. OncoImmunology. 2021 ; 10( 1): 1-13.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1080/2162402X.2021.1949896
Artigo de periodico
Neurovascular dysfunction and neuroinflammation in a Cockayne syndrome mouse model (2021)
Kajitan, Gustavo Satoru; Brace, Lear ; Villarreal, José Humberto Trevino ; Trocha, Kaspar; MacArthur, Michael Robert ; Vose, Sarah; Vargas, Dorathy; Bronson, Roderick ; Mitchell, Sarah Jayne; Menck, Carlos Frederico Martins ; Mitchell, James Robert
Source: Aging. Unidade: ICB
Subjects: MICROBIOLOGIA, DOENÇAS GENÉTICAS, ENVELHECIMENTO, DOENÇAS NEURODEGENERATIVAS, DANO AO DNA, REPARAÇÃO DE DNA, NUCLEOTÍDEOS, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, RADIAÇÃO ULTRAVIOLETA, NEURÓGLIA, CÉLULAS ENDOTELIAIS, BARREIRA HEMATO-ENCEFÁLICA, COCAÍNA
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ABNT
KAJITAN, Gustavo Satoru et al. Neurovascular dysfunction and neuroinflammation in a Cockayne syndrome mouse model. Aging, v. 13, n. 19 p. 22710—22731, 2021Tradução . . Disponível em: https://doi.org/10.18632/aging.203617. Acesso em: 03 jun. 2024.
APA
Kajitan, G. S., Brace, L., Villarreal, J. H. T., Trocha, K., MacArthur, M. R., Vose, S., et al. (2021). Neurovascular dysfunction and neuroinflammation in a Cockayne syndrome mouse model. Aging, 13( 19 p. 22710—22731). doi:10.18632/aging.203617
NLM
Kajitan GS, Brace L, Villarreal JHT, Trocha K, MacArthur MR, Vose S, Vargas D, Bronson R, Mitchell SJ, Menck CFM, Mitchell JR. Neurovascular dysfunction and neuroinflammation in a Cockayne syndrome mouse model [Internet]. Aging. 2021 ; 13( 19 p. 22710—22731):[citado 2024 jun. 03 ] Available from: https://doi.org/10.18632/aging.203617
Vancouver
Kajitan GS, Brace L, Villarreal JHT, Trocha K, MacArthur MR, Vose S, Vargas D, Bronson R, Mitchell SJ, Menck CFM, Mitchell JR. Neurovascular dysfunction and neuroinflammation in a Cockayne syndrome mouse model [Internet]. Aging. 2021 ; 13( 19 p. 22710—22731):[citado 2024 jun. 03 ] Available from: https://doi.org/10.18632/aging.203617
Artigo de periodico
DNA damage and oxidative stress in human cells infected by Trypanosoma cruzi (2021)
Florentino, Pilar Tavares Veras ; Mendes, Davi ; Vitorino, Francisca Nathalia de Luna ; Martins, Davi Jardim ; Cunha, Julia P. C. ; Mortara, Renato A. ; Menck, Carlos Frederico Martins
Source: PL o S Pathogens. Unidades: ICB, BIOTECNOLOGIA
Subjects: MICROBIOLOGIA, DANOS AO DNA, ESTRESSE OXIDATIVO, TRYPANOSOMA CRUZI, INFECÇÕES POR PROTOZOÁRIOS, REPARAÇÃO DE DNA, CROMATINA, ANTIOXIDANTES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FLORENTINO, Pilar Tavares Veras et al. DNA damage and oxidative stress in human cells infected by Trypanosoma cruzi. PL o S Pathogens, v. 17, n. 4, p. 1-20, 2021Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1009502. Acesso em: 03 jun. 2024.
APA
Florentino, P. T. V., Mendes, D., Vitorino, F. N. de L., Martins, D. J., Cunha, J. P. C., Mortara, R. A., & Menck, C. F. M. (2021). DNA damage and oxidative stress in human cells infected by Trypanosoma cruzi. PL o S Pathogens, 17( 4), 1-20. doi:10.1371/journal.ppat.1009502
NLM
Florentino PTV, Mendes D, Vitorino FN de L, Martins DJ, Cunha JPC, Mortara RA, Menck CFM. DNA damage and oxidative stress in human cells infected by Trypanosoma cruzi [Internet]. PL o S Pathogens. 2021 ; 17( 4): 1-20.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1371/journal.ppat.1009502
Vancouver
Florentino PTV, Mendes D, Vitorino FN de L, Martins DJ, Cunha JPC, Mortara RA, Menck CFM. DNA damage and oxidative stress in human cells infected by Trypanosoma cruzi [Internet]. PL o S Pathogens. 2021 ; 17( 4): 1-20.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1371/journal.ppat.1009502
Artigo de periodico
Mutagenicity profile induced by UVB Light in human Xeroderma Pigmentosum group C cells (2021)
Ruiz, Nathalia Quintero ; Corradi, Camila ; Moreno, Natália Cestari ; Souza, Tiago Antonio de ; Castro, Ligia Pereira ; Rocha, Clarissa Ribeiro Reily ; Menck, Carlos Frederico Martins
Source: Photochemistry and Photobiology. Unidades: ICB, BIOINFORMÁTICA, IQ
Subjects: MICROBIOLOGIA, DANO AO DNA, NUCLEOTÍDEOS, GENOMAS, RADIAÇÃO ULTRAVIOLETA, NEOPLASIAS CUTÂNEAS, MUTAÇÃO, FIBROBLASTOS, MUTAGÊNESE, SEQUENCIAMENTO GENÉTICO, REPARAÇÃO DE DNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RUIZ, Nathalia Quintero et al. Mutagenicity profile induced by UVB Light in human Xeroderma Pigmentosum group C cells. Photochemistry and Photobiology, p. 1-19, 2021Tradução . . Disponível em: https://doi.org/10.1111/php.13516. Acesso em: 03 jun. 2024.
APA
Ruiz, N. Q., Corradi, C., Moreno, N. C., Souza, T. A. de, Castro, L. P., Rocha, C. R. R., & Menck, C. F. M. (2021). Mutagenicity profile induced by UVB Light in human Xeroderma Pigmentosum group C cells. Photochemistry and Photobiology, 1-19. doi:10.1111/php.13516
NLM
Ruiz NQ, Corradi C, Moreno NC, Souza TA de, Castro LP, Rocha CRR, Menck CFM. Mutagenicity profile induced by UVB Light in human Xeroderma Pigmentosum group C cells [Internet]. Photochemistry and Photobiology. 2021 ; 1-19.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1111/php.13516
Vancouver
Ruiz NQ, Corradi C, Moreno NC, Souza TA de, Castro LP, Rocha CRR, Menck CFM. Mutagenicity profile induced by UVB Light in human Xeroderma Pigmentosum group C cells [Internet]. Photochemistry and Photobiology. 2021 ; 1-19.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1111/php.13516
Artigo de periodico
Xeroderma pigmentosum variant: squamous cell carcinoma of the lower lip harboring exon 11 mutation of POLH (2021)
Pereira, Thaís dos Santos Fontes ; Castro, Ligia Pereira ; Menck, Carlos Frederico Martins ; Maia, Maria Helena Thomaz ; Souza, Lucas Lacerda de; Fonseca, Felipe Paiva; Pontes, Helder Antônio Rebelo; Pontes, Flavia Sirotheau Correa; Gomez, Ricardo Santiago
Source: Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. Unidade: ICB
Subjects: MICROBIOLOGIA, DANO AO DNA, DOENÇAS GENÉTICAS, NUCLEOTÍDEOS, CARCINOMA DE CÉLULAS ESCAMOSAS, SEQUENCIAMENTO GENÉTICO, MUTAÇÃO GENÉTICA, FENÓTIPOS, GENÓTIPOS, REAÇÃO EM CADEIA POR POLIMERASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Thaís dos Santos Fontes et al. Xeroderma pigmentosum variant: squamous cell carcinoma of the lower lip harboring exon 11 mutation of POLH. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology, v. 132, n. 3, p. e97-e105, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.oooo.2021.03.013. Acesso em: 03 jun. 2024.
APA
Pereira, T. dos S. F., Castro, L. P., Menck, C. F. M., Maia, M. H. T., Souza, L. L. de, Fonseca, F. P., et al. (2021). Xeroderma pigmentosum variant: squamous cell carcinoma of the lower lip harboring exon 11 mutation of POLH. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology, 132( 3), e97-e105. doi:10.1016/j.oooo.2021.03.013
NLM
Pereira T dos SF, Castro LP, Menck CFM, Maia MHT, Souza LL de, Fonseca FP, Pontes HAR, Pontes FSC, Gomez RS. Xeroderma pigmentosum variant: squamous cell carcinoma of the lower lip harboring exon 11 mutation of POLH [Internet]. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. 2021 ; 132( 3): e97-e105.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.oooo.2021.03.013
Vancouver
Pereira T dos SF, Castro LP, Menck CFM, Maia MHT, Souza LL de, Fonseca FP, Pontes HAR, Pontes FSC, Gomez RS. Xeroderma pigmentosum variant: squamous cell carcinoma of the lower lip harboring exon 11 mutation of POLH [Internet]. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. 2021 ; 132( 3): e97-e105.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.oooo.2021.03.013
Artigo de periodico
Biallelic UBE4A loss-of-function variants cause intellectual disability and global developmental delay (2021)
Melo, Uirá Souto ; Bonner, Devon ; Lloyd, Kevin C. Kent ; Moshiri, Ala ; Willis, Brandon ; Lanoue, Louise ; Bower, Lynette; Leonard, Brian C. ; Martins, Davi Jardim ; Gomes, Fernando ; Leite, Felipe de Souza ; Oliveira, Danyllo ; Kitajima, João Paulo; Monteiro, Fabiola P. ; Zatz, Mayana ; Menck, Carlos Frederico Martins ; Wheeler, Matthew T. ; Bernstein, Jonathan A. ; Dumas, Kevin; Spiteri, Elizabeth ; Di Donato, Nataliya ; Jahn, Arne ; Hashem, Mais; Alsaif, Hessa S. ; Chedrawi, Aziza; Alkuraya, Fowzan S. ; Kok, Fernando ; Byers, Heather M.
Source: Genetics in Medicine. Unidades: IB, ICB, FM
Subjects: MICROBIOLOGIA, EXONS, GENOMAS, PESSOAS COM DEFICIÊNCIA INTELECTUAL, DOENÇAS DO DESENVOLVIMENTO ÓSSEO, VARIAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MELO, Uirá Souto et al. Biallelic UBE4A loss-of-function variants cause intellectual disability and global developmental delay. Genetics in Medicine, v. 23, n. 4, p. 6611-668, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41436-020-01047-z. Acesso em: 03 jun. 2024.
APA
Melo, U. S., Bonner, D., Lloyd, K. C. K., Moshiri, A., Willis, B., Lanoue, L., et al. (2021). Biallelic UBE4A loss-of-function variants cause intellectual disability and global developmental delay. Genetics in Medicine, 23( 4), 6611-668. doi:10.1038/s41436-020-01047-z
NLM
Melo US, Bonner D, Lloyd KCK, Moshiri A, Willis B, Lanoue L, Bower L, Leonard BC, Martins DJ, Gomes F, Leite F de S, Oliveira D, Kitajima JP, Monteiro FP, Zatz M, Menck CFM, Wheeler MT, Bernstein JA, Dumas K, Spiteri E, Di Donato N, Jahn A, Hashem M, Alsaif HS, Chedrawi A, Alkuraya FS, Kok F, Byers HM. Biallelic UBE4A loss-of-function variants cause intellectual disability and global developmental delay [Internet]. Genetics in Medicine. 2021 ; 23( 4): 6611-668.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1038/s41436-020-01047-z
Vancouver
Melo US, Bonner D, Lloyd KCK, Moshiri A, Willis B, Lanoue L, Bower L, Leonard BC, Martins DJ, Gomes F, Leite F de S, Oliveira D, Kitajima JP, Monteiro FP, Zatz M, Menck CFM, Wheeler MT, Bernstein JA, Dumas K, Spiteri E, Di Donato N, Jahn A, Hashem M, Alsaif HS, Chedrawi A, Alkuraya FS, Kok F, Byers HM. Biallelic UBE4A loss-of-function variants cause intellectual disability and global developmental delay [Internet]. Genetics in Medicine. 2021 ; 23( 4): 6611-668.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1038/s41436-020-01047-z
Artigo de periodico
Loss of Melanopsin (OPN4) leads to a faster cell cycle progression and growth in Murine Melanocytes (2021)
Assis, Leonardo Vinícius Monteiro de ; Moraes, Maria Nathália ; Mendes, Davi ; Silva, Matheus Molina; Menck, Carlos Frederico Martins ; Castrucci, Ana Maria de Lauro
Source: Current Issues in Molecular Biology. Unidades: ICB, IB
Subjects: MICROBIOLOGIA, MELANOMA, PROTEÍNAS DA MEMBRANA, CICLO CELULAR, EXPRESSÃO GÊNICA, FATORES DE TRANSCRIÇÃO, PELE, NEOPLASIAS CUTÂNEAS, PIGMENTOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ASSIS, Leonardo Vinícius Monteiro de et al. Loss of Melanopsin (OPN4) leads to a faster cell cycle progression and growth in Murine Melanocytes. Current Issues in Molecular Biology, v. 43, n. 3, p. 1436-1450, 2021Tradução . . Disponível em: https://doi.org/10.3390/cimb43030101. Acesso em: 03 jun. 2024.
APA
Assis, L. V. M. de, Moraes, M. N., Mendes, D., Silva, M. M., Menck, C. F. M., & Castrucci, A. M. de L. (2021). Loss of Melanopsin (OPN4) leads to a faster cell cycle progression and growth in Murine Melanocytes. Current Issues in Molecular Biology, 43( 3), 1436-1450. doi:10.3390/cimb43030101
NLM
Assis LVM de, Moraes MN, Mendes D, Silva MM, Menck CFM, Castrucci AM de L. Loss of Melanopsin (OPN4) leads to a faster cell cycle progression and growth in Murine Melanocytes [Internet]. Current Issues in Molecular Biology. 2021 ; 43( 3): 1436-1450.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3390/cimb43030101
Vancouver
Assis LVM de, Moraes MN, Mendes D, Silva MM, Menck CFM, Castrucci AM de L. Loss of Melanopsin (OPN4) leads to a faster cell cycle progression and growth in Murine Melanocytes [Internet]. Current Issues in Molecular Biology. 2021 ; 43( 3): 1436-1450.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3390/cimb43030101

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