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Artigo de periodico
Impact of hyperthyroidism on cardiac hypertrophy (2020)
Barreto-Chaves, Maria Luiza Morais ; Senger, Nathalia; Fevereiro, Marina Reingruber; Cesaro, Alice Cristina Parletta ; Takano, Ana Paula Cremasco
Source: Endocrine Connections. Unidade: ICB
Subjects: ANATOMIA, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, SISTEMA RENINA-ANGIOTENSINA, CÉLULAS MUSCULARES, INSUFICIÊNCIA CARDÍACA, SISTEMA NERVOSO SIMPÁTICO
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ABNT
BARRETO-CHAVES, Maria Luiza Morais et al. Impact of hyperthyroidism on cardiac hypertrophy. Endocrine Connections, v. 9, n. 3, p. R59–R69, 2020Tradução . . Disponível em: https://doi.org/10.1530/EC-19-0543. Acesso em: 05 jun. 2024.
APA
Barreto-Chaves, M. L. M., Senger, N., Fevereiro, M. R., Cesaro, A. C. P., & Takano, A. P. C. (2020). Impact of hyperthyroidism on cardiac hypertrophy. Endocrine Connections, 9( 3), R59–R69. doi:10.1530/EC-19-0543
NLM
Barreto-Chaves MLM, Senger N, Fevereiro MR, Cesaro ACP, Takano APC. Impact of hyperthyroidism on cardiac hypertrophy [Internet]. Endocrine Connections. 2020 ; 9( 3): R59–R69.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1530/EC-19-0543
Vancouver
Barreto-Chaves MLM, Senger N, Fevereiro MR, Cesaro ACP, Takano APC. Impact of hyperthyroidism on cardiac hypertrophy [Internet]. Endocrine Connections. 2020 ; 9( 3): R59–R69.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1530/EC-19-0543
Artigo de periodico
Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R (2020)
Lino, Caroline Antunes ; Teixeira, Larissa de Bortoli ; Simões, Sarah Capelupe; Silva, Tábatha de Oliveira ; Diniz, Gabriela Placoná ; Costa Neto, Claudio Miguel da ; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Cellular Physiology. Unidades: FMRP, ICB
Subjects: ANATOMIA, HIPERTROFIA VENTRICULAR ESQUERDA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, SISTEMA CARDIOVASCULAR, IMUNOHISTOQUÍMICA, PROTEÍNAS QUINASES
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LINO, Caroline Antunes et al. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R. Journal of Cellular Physiology, v. 236, n. 6, p. 4640-4654, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.30187. Acesso em: 05 jun. 2024.
APA
Lino, C. A., Teixeira, L. de B., Simões, S. C., Silva, T. de O., Diniz, G. P., Costa Neto, C. M. da, & Barreto-Chaves, M. L. M. (2020). Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R. Journal of Cellular Physiology, 236( 6), 4640-4654. doi:10.1002/jcp.30187
NLM
Lino CA, Teixeira L de B, Simões SC, Silva T de O, Diniz GP, Costa Neto CM da, Barreto-Chaves MLM. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R [Internet]. Journal of Cellular Physiology. 2020 ; 236( 6): 4640-4654.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1002/jcp.30187
Vancouver
Lino CA, Teixeira L de B, Simões SC, Silva T de O, Diniz GP, Costa Neto CM da, Barreto-Chaves MLM. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R [Internet]. Journal of Cellular Physiology. 2020 ; 236( 6): 4640-4654.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1002/jcp.30187
Artigo de periodico
The endocrinological component and signaling pathways associated to cardiac hypertrophy (2020)
Takano, Ana Paula Cremasco ; Senger, Nathalia ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, DOENÇAS CARDIOVASCULARES, HORMÔNIOS TIREOIDIANOS, INSULINA, SISTEMA RENINA-ANGIOTENSINA, CATECOLAMINAS, INSUFICIÊNCIA CARDÍACA
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TAKANO, Ana Paula Cremasco e SENGER, Nathalia e BARRETO-CHAVES, Maria Luiza Morais. The endocrinological component and signaling pathways associated to cardiac hypertrophy. Molecular and Cellular Endocrinology, v. 30, p. 13 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2020.110972. Acesso em: 05 jun. 2024.
APA
Takano, A. P. C., Senger, N., & Barreto-Chaves, M. L. M. (2020). The endocrinological component and signaling pathways associated to cardiac hypertrophy. Molecular and Cellular Endocrinology, 30, 13 . doi:10.1016/j.mce.2020.110972
NLM
Takano APC, Senger N, Barreto-Chaves MLM. The endocrinological component and signaling pathways associated to cardiac hypertrophy [Internet]. Molecular and Cellular Endocrinology. 2020 ; 30 13 .[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.mce.2020.110972
Vancouver
Takano APC, Senger N, Barreto-Chaves MLM. The endocrinological component and signaling pathways associated to cardiac hypertrophy [Internet]. Molecular and Cellular Endocrinology. 2020 ; 30 13 .[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.mce.2020.110972
Artigo de periodico
The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity (2020)
Gewehr, Mayara Calegaro Ferrari ; Teixeira, Alexandre Abilio de Souza ; Santos, Bruna de Araújo Cardoso dos; Biondo, Luana Amorim ; Gozzo, Fábio Cesar ; Marques, Amanda Miquilini Cordibello ; Eichler, Rosangela Aparecida dos Santos ; Reckziegel, Patrícia ; Silva, Renée de Nazaré Oliveira da ; Santos, Nilton Barretos dos ; Câmara, Niels Olsen Saraiva ; Castoldi, Angela ; Barreto-Chaves, Maria Luiza Morais ; Dale, Camila Squarzoni ; Senger, Nathalia; Lima, joanna Darck Carola Correia ; Seelaender, Marilia Cerqueira Leite ; Inada, Aline Carla ; Akamine, Eliana Hiromi ; Castro, Leandro Mantovani de ; Rodrigues, Alice Cristina ; Rosa Neto, José Cesar ; Ferro, Emer Suavinho
Source: Biomolecules. Unidade: ICB
Subjects: IMUNOLOGIA, ANATOMIA, BIOLOGIA CELULAR, FARMACOLOGIA, METABOLISMO ENERGÉTICO, OBESIDADE, RESISTÊNCIA À INSULINA, DIETA ANIMAL, EXPRESSÃO GÊNICA, ENZIMAS HIDROLÍTICAS, ENZIMAS PROTEOLÍTICAS, RATOS, DIETA ANIMAL
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GEWEHR, Mayara Calegaro Ferrari et al. The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity. Biomolecules, v. 10, n. 2, p. 40 , 2020Tradução . . Disponível em: https://doi.org/10.3390/biom10020321. Acesso em: 05 jun. 2024.
APA
Gewehr, M. C. F., Teixeira, A. A. de S., Santos, B. de A. C. dos, Biondo, L. A., Gozzo, F. C., Marques, A. M. C., et al. (2020). The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity. Biomolecules, 10( 2), 40 . doi:10.3390/biom10020321
NLM
Gewehr MCF, Teixeira AA de S, Santos B de AC dos, Biondo LA, Gozzo FC, Marques AMC, Eichler RA dos S, Reckziegel P, Silva R de NO da, Santos NB dos, Câmara NOS, Castoldi A, Barreto-Chaves MLM, Dale CS, Senger N, Lima joanna DCC, Seelaender MCL, Inada AC, Akamine EH, Castro LM de, Rodrigues AC, Rosa Neto JC, Ferro ES. The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity [Internet]. Biomolecules. 2020 ; 10( 2): 40 .[citado 2024 jun. 05 ] Available from: https://doi.org/10.3390/biom10020321
Vancouver
Gewehr MCF, Teixeira AA de S, Santos B de AC dos, Biondo LA, Gozzo FC, Marques AMC, Eichler RA dos S, Reckziegel P, Silva R de NO da, Santos NB dos, Câmara NOS, Castoldi A, Barreto-Chaves MLM, Dale CS, Senger N, Lima joanna DCC, Seelaender MCL, Inada AC, Akamine EH, Castro LM de, Rodrigues AC, Rosa Neto JC, Ferro ES. The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity [Internet]. Biomolecules. 2020 ; 10( 2): 40 .[citado 2024 jun. 05 ] Available from: https://doi.org/10.3390/biom10020321
Artigo de periodico
Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism (2019)
Lino, Caroline Antunes; Demasi, Marilene; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, METABOLISMO DE PROTEÍNA, HORMÔNIOS TIREOIDIANOS, CORAÇÃO
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ABNT
LINO, Caroline Antunes e DEMASI, Marilene e BARRETO-CHAVES, Maria Luiza Morais. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism. Molecular and Cellular Endocrinology, v. 493, p. 1-9, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110451. Acesso em: 05 jun. 2024.
APA
Lino, C. A., Demasi, M., & Barreto-Chaves, M. L. M. (2019). Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism. Molecular and Cellular Endocrinology, 493, 1-9. doi:10.1016/j.mce.2019.110451
NLM
Lino CA, Demasi M, Barreto-Chaves MLM. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism [Internet]. Molecular and Cellular Endocrinology. 2019 ; 493 1-9.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.mce.2019.110451
Vancouver
Lino CA, Demasi M, Barreto-Chaves MLM. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism [Internet]. Molecular and Cellular Endocrinology. 2019 ; 493 1-9.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.mce.2019.110451
Artigo de periodico
Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan (2019)
Leme, Janaina M. M.; Ohar, Erina; Santiago, Veronica Feijoli ; Barros, Mário Henrique de ; Netto, Luis Eduardo Soares ; Pimenta, Daniel Carvalho ; Mariano, Douglas Oscar Ceolin ; Oliveira, Cristiano Luis Pinto de ; Bicev, Renata Naporano ; Barreto-Chaves, Maria Luiza Morais ; Lino, Caroline Antunes ; Demasi, Marilene
Source: Archives of Biochemistry and Biophysics. Unidades: ICB, IF
Subjects: MUTAÇÃO, ANATOMIA, MICROBIOLOGIA, CRONOLOGIA
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LEME, Janaina M. M. et al. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan. Archives of Biochemistry and Biophysics, v. 666, p. 63-72, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.abb.2019.03.012. Acesso em: 05 jun. 2024.
APA
Leme, J. M. M., Ohar, E., Santiago, V. F., Barros, M. H. de, Netto, L. E. S., Pimenta, D. C., et al. (2019). Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan. Archives of Biochemistry and Biophysics, 666, 63-72. doi:10.1016/j.abb.2019.03.012
NLM
Leme JMM, Ohar E, Santiago VF, Barros MH de, Netto LES, Pimenta DC, Mariano DOC, Oliveira CLP de, Bicev RN, Barreto-Chaves MLM, Lino CA, Demasi M. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan [Internet]. Archives of Biochemistry and Biophysics. 2019 ; 666 63-72.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.abb.2019.03.012
Vancouver
Leme JMM, Ohar E, Santiago VF, Barros MH de, Netto LES, Pimenta DC, Mariano DOC, Oliveira CLP de, Bicev RN, Barreto-Chaves MLM, Lino CA, Demasi M. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan [Internet]. Archives of Biochemistry and Biophysics. 2019 ; 666 63-72.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.abb.2019.03.012
Artigo de periodico
Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus (2019)
Palombit, Kelly ; Mendes, Cristina Eusébio ; Lima, Wothan Tavares de ; Barreto-Chaves, Maria Luiza Morais ; Castelucci, Patricia
Source: Digestive Diseases and Sciences. Unidade: ICB
Subjects: ANATOMIA, FARMACOLOGIA, PLEXO MIENTÉRICO, ISQUEMIA, REPERFUSÃO, ÍLEO
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ABNT
PALOMBIT, Kelly et al. Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus. Digestive Diseases and Sciences, v. 64, p. 1815-1829, 2019Tradução . . Disponível em: https://doi.org/10.1007/s10620-019-05496-8. Acesso em: 05 jun. 2024.
APA
Palombit, K., Mendes, C. E., Lima, W. T. de, Barreto-Chaves, M. L. M., & Castelucci, P. (2019). Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus. Digestive Diseases and Sciences, 64, 1815-1829. doi:10.1007/s10620-019-05496-8
NLM
Palombit K, Mendes CE, Lima WT de, Barreto-Chaves MLM, Castelucci P. Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus [Internet]. Digestive Diseases and Sciences. 2019 ; 64 1815-1829.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1007/s10620-019-05496-8
Vancouver
Palombit K, Mendes CE, Lima WT de, Barreto-Chaves MLM, Castelucci P. Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus [Internet]. Digestive Diseases and Sciences. 2019 ; 64 1815-1829.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1007/s10620-019-05496-8
Artigo de periodico
Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia (2019)
Lima, Vanessa Morais ; Lino, Caroline Antunes ; Senger, Nathalia ; Silva, Tábatha de Oliveira ; Fonseca, Renata Inzinna Bernardo; Bader, Michael ; Santos, Robson Augusto Sousa ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, ANGIOTENSINA II, METABOLISMO, OBESIDADE, GLICOSE
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ABNT
LIMA, Vanessa Morais et al. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia. Molecular and Cellular Endocrinology, v. 498, p. 1-10, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110576. Acesso em: 05 jun. 2024.
APA
Lima, V. M., Lino, C. A., Senger, N., Silva, T. de O., Fonseca, R. I. B., Bader, M., et al. (2019). Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia. Molecular and Cellular Endocrinology, 498, 1-10. doi:10.1016/j.mce.2019.110576
NLM
Lima VM, Lino CA, Senger N, Silva T de O, Fonseca RIB, Bader M, Santos RAS, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-10.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.mce.2019.110576
Vancouver
Lima VM, Lino CA, Senger N, Silva T de O, Fonseca RIB, Bader M, Santos RAS, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-10.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.mce.2019.110576
Artigo de periodico
MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control (2018)
Santana, Danni Yohani; Salgado, Rafael Moysés; Fevereiro, Marina Reingruber; Nascimento, Rogério Silva do; Fonseca, Raissa; Câmara, Niels Olsen Saraiva ; Epiphanio, Sabrina ; Marinho, Claudio Romero Farias ; Barreto-Chaves, Maria Luiza Morais ; Lima, Maria Regina D'Império ; Mosig, José Maria Álvarez
Source: PloS Neglected Tropical Diseases. Unidades: ICB, FCF
Subjects: TRYPANOSOMA CRUZI, CORAÇÃO, DOENÇA DE CHAGAS, PARASITISMO
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ABNT
SANTANA, Danni Yohani et al. MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control. PloS Neglected Tropical Diseases, v. 12, n. 8, p. 1-19, 2018Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0006617. Acesso em: 05 jun. 2024.
APA
Santana, D. Y., Salgado, R. M., Fevereiro, M. R., Nascimento, R. S. do, Fonseca, R., Câmara, N. O. S., et al. (2018). MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control. PloS Neglected Tropical Diseases, 12( 8), 1-19. doi:10.1371/journal.pntd.0006617
NLM
Santana DY, Salgado RM, Fevereiro MR, Nascimento RS do, Fonseca R, Câmara NOS, Epiphanio S, Marinho CRF, Barreto-Chaves MLM, Lima MRD'I, Mosig JMÁ. MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control [Internet]. PloS Neglected Tropical Diseases. 2018 ; 12( 8): 1-19.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1371/journal.pntd.0006617
Vancouver
Santana DY, Salgado RM, Fevereiro MR, Nascimento RS do, Fonseca R, Câmara NOS, Epiphanio S, Marinho CRF, Barreto-Chaves MLM, Lima MRD'I, Mosig JMÁ. MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control [Internet]. PloS Neglected Tropical Diseases. 2018 ; 12( 8): 1-19.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1371/journal.pntd.0006617
Artigo de periodico
Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway (2018)
Senger, Nathalia ; Melo, Marcos Barrouin; Diniz, Gabriela Placoná ; Santos, Maria José Campagnole dos ; Santos, Robson Augusto Souza dos; Barreto-Chaves, Maria Luiza Morais
Source: Clinical Science. Unidade: ICB
Subjects: ANGIOTENSINAS, CORAÇÃO, HIPERTIREOIDISMO, SISTEMA RENINA-ANGIOTENSINA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, ANATOMIA
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ABNT
SENGER, Nathalia et al. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, v. 132, n. 11, p. 1117-1133, 2018Tradução . . Disponível em: https://doi.org/10.1042/CS20171606. Acesso em: 05 jun. 2024.
APA
Senger, N., Melo, M. B., Diniz, G. P., Santos, M. J. C. dos, Santos, R. A. S. dos, & Barreto-Chaves, M. L. M. (2018). Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, 132( 11), 1117-1133. doi:10.1042/CS20171606
NLM
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1042/CS20171606
Vancouver
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1042/CS20171606
Artigo de periodico
MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone (2017)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Moreno, Camila Rodrigues; Senger, Nathalia; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Cellular Physiology. Unidade: ICB
Subjects: ANATOMIA, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, RNA DE TRANSFERÊNCIA, SISTEMA RENINA-ANGIOTENSINA, RATOS WISTAR, BLOQUEIO CARDÍACO
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ABNT
DINIZ, Gabriela Placoná et al. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, v. 232, n. 12, p. 3360-3368, 2017Tradução . . Disponível em: https://doi.org/10.1002/jcp.25781. Acesso em: 05 jun. 2024.
APA
Diniz, G. P., Lino, C. A., Moreno, C. R., Senger, N., & Barreto-Chaves, M. L. M. (2017). MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, 232( 12), 3360-3368. doi:10.1002/jcp.25781
NLM
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1002/jcp.25781
Vancouver
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1002/jcp.25781
Artigo de periodico
S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone (2017)
Takano, Ana Paula Cremasco ; Munhoz, Carolina Demarchi ; Moriscot, Anselmo Sigari ; Gupta, Sudhiranjan; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Molecular Medicine. Unidade: ICB
Assunto: ANATOMIA
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ABNT
TAKANO, Ana Paula Cremasco et al. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, v. 95, n. 6, p. 671-682, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00109-017-1511-y. Acesso em: 05 jun. 2024.
APA
Takano, A. P. C., Munhoz, C. D., Moriscot, A. S., Gupta, S., & Barreto-Chaves, M. L. M. (2017). S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, 95( 6), 671-682. doi:10.1007/s00109-017-1511-y
NLM
Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
Vancouver
Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
Artigo de periodico
Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy (2017)
Diniz, Gabriela Placoná ; Huang, Zhan-Peng; Liu, Jianming; Chen, Jinghai; Ding, Jian; Fonseca, Renata Inzinna; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José ; Hu, Xiaoyun; Wang, Da-Zhi
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, v. 131, n. 24, p. 2885-2900, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20171368. Acesso em: 05 jun. 2024.
APA
Diniz, G. P., Huang, Z. -P., Liu, J., Chen, J., Ding, J., Fonseca, R. I., et al. (2017). Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, 131( 24), 2885-2900. doi:10.1042/CS20171368
NLM
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1042/CS20171368
Vancouver
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1042/CS20171368
Artigo de periodico
MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets (2016)
Guedes, Elaine Castilho ; França, Gustavo Starvaggi ; Lino, Caroline Antunes ; Koyama, Fernanda Christtanini ; Moreira, Luana do Nascimento; Alexandre, Juliana Gomes ; Galante, Pedro Alexandre Favoretto ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Source: Journal of Cellular Physiology. Unidades: ICB, IQ
Subjects: ANATOMIA, MICRORNAS, EXPRESSÃO GÊNICA, HIPERTROFIA VENTRICULAR ESQUERDA, MIOCÁRDIO, DIETA ANIMAL, CAMUNDONGOS, GLICEMIA, JEJUNO DE ANIMAL, INSULINA, RNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUEDES, Elaine Castilho et al. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets. Journal of Cellular Physiology, v. 231, n. 8, p. 1771-1783, 2016Tradução . . Disponível em: https://doi.org/10.1002/jcp.25280. Acesso em: 05 jun. 2024.
APA
Guedes, E. C., França, G. S., Lino, C. A., Koyama, F. C., Moreira, L. do N., Alexandre, J. G., et al. (2016). MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets. Journal of Cellular Physiology, 231( 8), 1771-1783. doi:10.1002/jcp.25280
NLM
Guedes EC, França GS, Lino CA, Koyama FC, Moreira L do N, Alexandre JG, Galante PAF, Barreto-Chaves MLM, Diniz GP. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets [Internet]. Journal of Cellular Physiology. 2016 ; 231( 8): 1771-1783.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1002/jcp.25280
Vancouver
Guedes EC, França GS, Lino CA, Koyama FC, Moreira L do N, Alexandre JG, Galante PAF, Barreto-Chaves MLM, Diniz GP. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets [Internet]. Journal of Cellular Physiology. 2016 ; 231( 8): 1771-1783.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1002/jcp.25280
Artigo de periodico
Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy (2016)
Diniz, Gabriela Placoná ; Senger, Nathalia; Carneiro-Ramos, Marcela S.; Santos, Robson A. S.; Barreto-Chaves, Maria Luiza Morais
Source: Therapeutic Advances in Cardiovascular Disease. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy. Therapeutic Advances in Cardiovascular Disease, v. 10, n. 4, p. 192-202, 2016Tradução . . Disponível em: https://doi.org/10.1177/1753944715623228. Acesso em: 05 jun. 2024.
APA
Diniz, G. P., Senger, N., Carneiro-Ramos, M. S., Santos, R. A. S., & Barreto-Chaves, M. L. M. (2016). Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy. Therapeutic Advances in Cardiovascular Disease, 10( 4), 192-202. doi:10.1177/1753944715623228
NLM
Diniz GP, Senger N, Carneiro-Ramos MS, Santos RAS, Barreto-Chaves MLM. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy [Internet]. Therapeutic Advances in Cardiovascular Disease. 2016 ; 10( 4): 192-202.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1177/1753944715623228
Vancouver
Diniz GP, Senger N, Carneiro-Ramos MS, Santos RAS, Barreto-Chaves MLM. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy [Internet]. Therapeutic Advances in Cardiovascular Disease. 2016 ; 10( 4): 192-202.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1177/1753944715623228
Artigo de periodico
Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats (2015)
Gabriel-Costa, Daniele; Cunha, Telma Fatima da; Bechara, Luiz Roberto Grassmann; Fortunato, Rodrigo Soares; Bozi, Luiz Henrique Marchesi; Coelho, Marcele de Almeida; Barreto-Chaves, Maria Luiza Morais ; Brum, Patricia Chakur
Source: PLOS ONE. Unidades: EEFE, ICB
Subjects: CORAÇÃO, OXIDAÇÃO, SUPERÓXIDO DISMUTASE, MITOCÔNDRIAS, ANTIOXIDANTES, VENTRÍCULO CARDÍACO, FOSFATOS, MÚSCULO ESQUELÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GABRIEL-COSTA, Daniele et al. Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats. PLOS ONE, v. 10, n. 5, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0127843. Acesso em: 05 jun. 2024.
APA
Gabriel-Costa, D., Cunha, T. F. da, Bechara, L. R. G., Fortunato, R. S., Bozi, L. H. M., Coelho, M. de A., et al. (2015). Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats. PLOS ONE, 10( 5). doi:10.1371/journal.pone.0127843
NLM
Gabriel-Costa D, Cunha TF da, Bechara LRG, Fortunato RS, Bozi LHM, Coelho M de A, Barreto-Chaves MLM, Brum PC. Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats [Internet]. PLOS ONE. 2015 ; 10( 5):[citado 2024 jun. 05 ] Available from: https://doi.org/10.1371/journal.pone.0127843
Vancouver
Gabriel-Costa D, Cunha TF da, Bechara LRG, Fortunato RS, Bozi LHM, Coelho M de A, Barreto-Chaves MLM, Brum PC. Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats [Internet]. PLOS ONE. 2015 ; 10( 5):[citado 2024 jun. 05 ] Available from: https://doi.org/10.1371/journal.pone.0127843
Artigo de periodico
Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders (2015)
Lino, Caroline Antunes; Silva, Ivson Bezerra da; Shibata, Caroline Emy Rodrigues; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, HIPERTIREOIDISMO, RATOS WISTAR, DOENÇAS CARDIOVASCULARES EM ANIMAL, SISTEMA RENINA-ANGIOTENSINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LINO, Caroline Antunes et al. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders. Molecular and Cellular Endocrinology, v. 416, p. 1-8, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.08.015. Acesso em: 05 jun. 2024.
APA
Lino, C. A., Silva, I. B. da, Shibata, C. E. R., & Barreto-Chaves, M. L. M. (2015). Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders. Molecular and Cellular Endocrinology, 416, 1-8. doi:10.1016/j.mce.2015.08.015
NLM
Lino CA, Silva IB da, Shibata CER, Barreto-Chaves MLM. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders [Internet]. Molecular and Cellular Endocrinology. 2015 ; 416 1-8.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.mce.2015.08.015
Vancouver
Lino CA, Silva IB da, Shibata CER, Barreto-Chaves MLM. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders [Internet]. Molecular and Cellular Endocrinology. 2015 ; 416 1-8.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1016/j.mce.2015.08.015
Artigo de periodico
Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor (2015)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Guedes, Elaine Castilho; Moreira, Luana do Nascimento; Barreto-Chaves, Maria Luiza Morais
Source: Basic Research in Cardiology. Unidade: ICB
Subjects: ANATOMIA, HORMÔNIOS TIREOIDIANOS, SISTEMA RENINA-ANGIOTENSINA, ANGIOTENSINA II
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, v. 110, n. 5, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00395-015-0504-7. Acesso em: 05 jun. 2024.
APA
Diniz, G. P., Lino, C. A., Guedes, E. C., Moreira, L. do N., & Barreto-Chaves, M. L. M. (2015). Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, 110( 5), 1-11. doi:10.1007/s00395-015-0504-7
NLM
Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
Vancouver
Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
Artigo de periodico
An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies (2014)
Ceravolo, Graziela S.; Montezano, Augusto C.; Jordão, Maria T.; Akamine, Eliana Hiromi ; Costa, Tiago Januário da; Takano, Ana Paula Cremasco ; Fernandes, Denise C.; Barreto-Chaves, Maria Luiza Morais ; Laurindo, Francisco R.; Tostes, Rita de Cássia; Fortes, Zuleica Bruno; Chopard, Renato Paulo; Touyz, Rhian M.; Carvalho, Maria Helena Catelli de
Source: PLOS ONE. Unidades: ICB, FMRP
Subjects: ANATOMIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CERAVOLO, Graziela S. et al. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, v. 9, n. 11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0111117. Acesso em: 05 jun. 2024.
APA
Ceravolo, G. S., Montezano, A. C., Jordão, M. T., Akamine, E. H., Costa, T. J. da, Takano, A. P. C., et al. (2014). An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, 9( 11). doi:10.1371/journal.pone.0111117
NLM
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 jun. 05 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Vancouver
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 jun. 05 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Artigo de periodico
Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion (2013)
Tavares, Felix Meira; Silva, Ivson Bezerra da; Gomes, Dayane Aparecida; Barreto-Chaves, Maria Luiza Morais
Source: Cardiovascular Drugs and Therapy. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TAVARES, Felix Meira et al. Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion. Cardiovascular Drugs and Therapy, v. 27, n. 5, p. 393-402, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10557-013-6473-x. Acesso em: 05 jun. 2024.
APA
Tavares, F. M., Silva, I. B. da, Gomes, D. A., & Barreto-Chaves, M. L. M. (2013). Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion. Cardiovascular Drugs and Therapy, 27( 5), 393-402. doi:10.1007/s10557-013-6473-x
NLM
Tavares FM, Silva IB da, Gomes DA, Barreto-Chaves MLM. Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 5): 393-402.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1007/s10557-013-6473-x
Vancouver
Tavares FM, Silva IB da, Gomes DA, Barreto-Chaves MLM. Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 5): 393-402.[citado 2024 jun. 05 ] Available from: https://doi.org/10.1007/s10557-013-6473-x

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