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Artigo de periodico
A novel diselenide attenuates the carrageenan-induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice (2024)
Lessa, Tássia Liz Araújo dos Santos ; Correia, Thiago Macêdo Lopes ; Santos, Talita Costa dos; Silva, Railmara Pereira da ; Silva, Beatriz Pereira da ; Cavallini, Maria Cláudia Magalhães ; Rocha, Leonardo Silva ; Peixoto, Albert Souza ; Cugnasca, Beatriz dos Santos ; Cervi, Gustavo ; Correra, Thiago Carita ; Gonçalves, Augusto Cesar; Cunha, Thiago Mattar ; Yatsuda, Regiane ; Magalhães, Amélia Cristina Mendes de ; Santos, Alcindo Aparecido dos ; Meotti, Flavia Carla ; Queiroz, Raphael Ferreira ; Festuccia, William Tadeu Lara
Source: Free Radical Research. Unidades: FMRP, IQ, ICB
Subjects: FISIOLOGIA, CARRAGENINA, ANTI-INFLAMATÓRIOS, SELÊNIO, INFLAMAÇÃO, NEUTRÓFILOS, PEROXIDASE, CAMUNDONGOS
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ABNT
LESSA, Tássia Liz Araújo dos Santos et al. A novel diselenide attenuates the carrageenan-induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice. Free Radical Research, v. 58, n. 4, p. 229-248, 2024Tradução . . Disponível em: https://doi.org/10.1080/10715762.2024.2336566. Acesso em: 11 jul. 2024.
APA
Lessa, T. L. A. dos S., Correia, T. M. L., Santos, T. C. dos, Silva, R. P. da, Silva, B. P. da, Cavallini, M. C. M., et al. (2024). A novel diselenide attenuates the carrageenan-induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice. Free Radical Research, 58( 4), 229-248. doi:https://doi.org/10.1080/10715762.2024.2336566
NLM
Lessa TLA dos S, Correia TML, Santos TC dos, Silva RP da, Silva BP da, Cavallini MCM, Rocha LS, Peixoto AS, Cugnasca B dos S, Cervi G, Correra TC, Gonçalves AC, Cunha TM, Yatsuda R, Magalhães ACM de, Santos AA dos, Meotti FC, Queiroz RF, Festuccia WTL. A novel diselenide attenuates the carrageenan-induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice [Internet]. Free Radical Research. 2024 ; 58( 4): 229-248.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1080/10715762.2024.2336566
Vancouver
Lessa TLA dos S, Correia TML, Santos TC dos, Silva RP da, Silva BP da, Cavallini MCM, Rocha LS, Peixoto AS, Cugnasca B dos S, Cervi G, Correra TC, Gonçalves AC, Cunha TM, Yatsuda R, Magalhães ACM de, Santos AA dos, Meotti FC, Queiroz RF, Festuccia WTL. A novel diselenide attenuates the carrageenan-induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice [Internet]. Free Radical Research. 2024 ; 58( 4): 229-248.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1080/10715762.2024.2336566
Artigo de periodico
Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction (2024)
Morelo, David Fernando Colon ; Wanderley, Carlos Wagner de Souza ; Turato, Walter Miguel; Borges, Vanessa F.; Franchin, Marcelo; Castanheira, Fernanda Vargas e Silva; Nascimento, Daniele Carvalho Bernardo; Prado, Douglas da Silva; Lima, Mikhael Haruo Fernandes de ; Volpon, Leila Costa; Kavaguti, Silvia K.; Carlotti, Ana Paula de Carvalho Panzeri ; Carmona, Fabio ; Franklin, Bernardo S.; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: British Journal of Pharmacology. Unidades: FCF, FMRP
Subjects: MACRÓFAGOS, SEPSE, CRIANÇAS, IMUNOSSUPRESSÃO, SISTEMA IMUNE
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ABNT
MORELO, David Fernando Colon et al. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction. British Journal of Pharmacology, v. 181, n. 8, p. 1308-1323, 2024Tradução . . Disponível em: https://doi.org/10.1111/bph.16286. Acesso em: 11 jul. 2024.
APA
Morelo, D. F. C., Wanderley, C. W. de S., Turato, W. M., Borges, V. F., Franchin, M., Castanheira, F. V. e S., et al. (2024). Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction. British Journal of Pharmacology, 181( 8), 1308-1323. doi:10.1111/bph.16286
NLM
Morelo DFC, Wanderley CW de S, Turato WM, Borges VF, Franchin M, Castanheira FV e S, Nascimento DCB, Prado D da S, Lima MHF de, Volpon LC, Kavaguti SK, Carlotti AP de CP, Carmona F, Franklin BS, Cunha TM, Alves Filho JCF, Cunha F de Q. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction [Internet]. British Journal of Pharmacology. 2024 ; 181( 8): 1308-1323.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1111/bph.16286
Vancouver
Morelo DFC, Wanderley CW de S, Turato WM, Borges VF, Franchin M, Castanheira FV e S, Nascimento DCB, Prado D da S, Lima MHF de, Volpon LC, Kavaguti SK, Carlotti AP de CP, Carmona F, Franklin BS, Cunha TM, Alves Filho JCF, Cunha F de Q. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction [Internet]. British Journal of Pharmacology. 2024 ; 181( 8): 1308-1323.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1111/bph.16286
Artigo de periodico
A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice (2024)
Lessa, Tássia Liz Araújo dos Santos; Correia, Thiago Macêdo Lopes ; Santos, Talita Costa dos; Silva, Railmara Pereira da ; Silva, Beatriz Pereira da ; Cavallini, Maria Cláudia Magalhães; Rocha, Leonardo Silva; Peixoto, Álbert Souza; Cugnasca, Beatriz dos Santos ; Cervi, Gustavo ; Correra, Thiago Carita ; Gonçalves, Augusto Cesar; Festuccia, William Tadeu Lara ; Cunha, Thiago Mattar ; Yatsuda, Regiane ; Magalhães, Amélia Cristina Mendes de ; Santos, Alcindo Aparecido dos ; Meotti, Flavia Carla ; Queiroz, Raphael Ferreira
Source: Free Radical Research. Unidades: IQ, ICB, FMRP
Subjects: NEUTRÓFILOS, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LESSA, Tássia Liz Araújo dos Santos et al. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice. Free Radical Research, 2024Tradução . . Disponível em: https://dx.doi.org/10.1080/10715762.2024.2336566. Acesso em: 11 jul. 2024.
APA
Lessa, T. L. A. dos S., Correia, T. M. L., Santos, T. C. dos, Silva, R. P. da, Silva, B. P. da, Cavallini, M. C. M., et al. (2024). A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice. Free Radical Research. doi:10.1080/10715762.2024.2336566
NLM
Lessa TLA dos S, Correia TML, Santos TC dos, Silva RP da, Silva BP da, Cavallini MCM, Rocha LS, Peixoto ÁS, Cugnasca B dos S, Cervi G, Correra TC, Gonçalves AC, Festuccia WTL, Cunha TM, Yatsuda R, Magalhães ACM de, Santos AA dos, Meotti FC, Queiroz RF. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice [Internet]. Free Radical Research. 2024 ;[citado 2024 jul. 11 ] Available from: https://dx.doi.org/10.1080/10715762.2024.2336566
Vancouver
Lessa TLA dos S, Correia TML, Santos TC dos, Silva RP da, Silva BP da, Cavallini MCM, Rocha LS, Peixoto ÁS, Cugnasca B dos S, Cervi G, Correra TC, Gonçalves AC, Festuccia WTL, Cunha TM, Yatsuda R, Magalhães ACM de, Santos AA dos, Meotti FC, Queiroz RF. A novel diselenide attenuates the carrageenan- induced inflammation by reducing neutrophil infiltration and the resulting tissue damage in mice [Internet]. Free Radical Research. 2024 ;[citado 2024 jul. 11 ] Available from: https://dx.doi.org/10.1080/10715762.2024.2336566
Artigo de periodico
Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients (2023)
Amaral, Natália Brasil do; Rodrigues, Tamara Silva; Giannini, Marcela Cavichioli; Lopes, Maria Isabel Fernandes; Bonjorno, Letícia Pastorelli; Menezes, Pamella Indira da Silva Oliveira; Dib, Saulo Musse; Gigante, Samara Libich Gusmão; Benatti, Maíra Nilson; Rezek, Uebe C.; Emrich-Filho, Laerte L.; Sousa, Betania A. A.; Almeida, Sérgio Couto Luna de; Luppino-Assad, Rodrigo; Veras, Flávio Protásio ; Schneider, Ayda Henriques; Leiria, Luiz Osório Silveira ; Cunha, Larissa Dias da; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Martins, Maria Auxiliadora ; Borges, Marcos de Carvalho ; Fonseca, Benedito Antonio Lopes da ; Bollela, Valdes Roberto ; Del-Ben, Cristina Marta ; Cunha, Fernando de Queiroz ; Santana, Rodrigo de Carvalho ; Vilar, Fernando Crivelenti; Zamboni, Dario Simões ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Source: Inflammation Research. Unidade: FMRP
Subjects: COVID-19, CITOCINAS, PROTEÍNAS, ALCALOIDES
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ABNT
AMARAL, Natália Brasil do et al. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, v. 72, n. 5, p. 895-899, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-023-01718-y. Acesso em: 11 jul. 2024.
APA
Amaral, N. B. do, Rodrigues, T. S., Giannini, M. C., Lopes, M. I. F., Bonjorno, L. P., Menezes, P. I. da S. O., et al. (2023). Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, 72( 5), 895-899. doi:10.1007/s00011-023-01718-y
NLM
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Vancouver
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 11 jul. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Parte de monografia/livro
CRISPR libraries and whole-genome screening to identify essential factors for viral infections (2023)
Paiva, Isadora Marques ; Damasceno, Samara; Cunha, Thiago Mattar
Source: Genome editing in biomedical sciences. Unidade: FMRP
Subjects: GENOMAS, INFECÇÕES POR VÍRUS DE RNA, GENÉTICA
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ABNT
PAIVA, Isadora Marques e DAMASCENO, Samara e CUNHA, Thiago Mattar. CRISPR libraries and whole-genome screening to identify essential factors for viral infections. Genome editing in biomedical sciences. Tradução . Cham: Springer, 2023. . Disponível em: https://doi.org/10.1007/978-3-031-33325-5_9. Acesso em: 11 jul. 2024.
APA
Paiva, I. M., Damasceno, S., & Cunha, T. M. (2023). CRISPR libraries and whole-genome screening to identify essential factors for viral infections. In Genome editing in biomedical sciences. Cham: Springer. doi:10.1007/978-3-031-33325-5_9
NLM
Paiva IM, Damasceno S, Cunha TM. CRISPR libraries and whole-genome screening to identify essential factors for viral infections [Internet]. In: Genome editing in biomedical sciences. Cham: Springer; 2023. [citado 2024 jul. 11 ] Available from: https://doi.org/10.1007/978-3-031-33325-5_9
Vancouver
Paiva IM, Damasceno S, Cunha TM. CRISPR libraries and whole-genome screening to identify essential factors for viral infections [Internet]. In: Genome editing in biomedical sciences. Cham: Springer; 2023. [citado 2024 jul. 11 ] Available from: https://doi.org/10.1007/978-3-031-33325-5_9
Artigo de periodico
Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection (2023)
Moreira, Thaiane Pinto; Sousa, Carla Daiane Ferreira de; Costa, Victor Rodrigues de Melo; Queiroz Junior, Celso Martins; Santos, Franciele Martins; Bonilha, Caio Santos ; Ésper, Lísia Maria; Nogueira, Mauricio Lacerda ; Cunha, Thiago Mattar ; Teixeira, Mauro Martins ; Costa, Vivian Vasconcelos; Souza, Daniele da Glória de
Source: Immunology. Unidade: FMRP
Subjects: ARTRALGIA, VÍRUS CHIKUNGUNYA, NECROSE, REPLICAÇÃO VIRAL
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ABNT
MOREIRA, Thaiane Pinto et al. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, v. 168, n. 3, p. 444-458, 2023Tradução . . Disponível em: https://doi.org/10.1111/imm.13583. Acesso em: 11 jul. 2024.
APA
Moreira, T. P., Sousa, C. D. F. de, Costa, V. R. de M., Queiroz Junior, C. M., Santos, F. M., Bonilha, C. S., et al. (2023). Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, 168( 3), 444-458. doi:10.1111/imm.13583
NLM
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1111/imm.13583
Vancouver
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1111/imm.13583
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: The Journal of Infectious Diseases. Unidade: FMRP
Subjects: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 11 jul. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico
Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons (2022)
Yang, Nicole J.; Isensee, Jörg; Neel, Dylan V; Quadros, Andreza Urba de; Zhang, Han-Xiong Bear; Lauzadis, Justas; Liu, Sai Man; Shiers, Stephanie; Belu, Andreea; Cunha, Thiago Mattar
Source: Nature Neuroscience. Unidade: FMRP
Subjects: TOXINAS, BACTÉRIAS, GÂNGLIOS ESPINHAIS, CÉLULAS-TRONCO, NOCICEPTORES, DOR, PEPTÍDEOS
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ABNT
YANG, Nicole J. et al. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons. Nature Neuroscience, v. 25, n. 2, p. 168-179, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41593-021-00973-8. Acesso em: 11 jul. 2024.
APA
Yang, N. J., Isensee, J., Neel, D. V., Quadros, A. U. de, Zhang, H. -X. B., Lauzadis, J., et al. (2022). Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons. Nature Neuroscience, 25( 2), 168-179. doi:10.1038/s41593-021-00973-8
NLM
Yang NJ, Isensee J, Neel DV, Quadros AU de, Zhang H-XB, Lauzadis J, Liu SM, Shiers S, Belu A, Cunha TM. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons [Internet]. Nature Neuroscience. 2022 ; 25( 2): 168-179.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1038/s41593-021-00973-8
Vancouver
Yang NJ, Isensee J, Neel DV, Quadros AU de, Zhang H-XB, Lauzadis J, Liu SM, Shiers S, Belu A, Cunha TM. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons [Internet]. Nature Neuroscience. 2022 ; 25( 2): 168-179.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1038/s41593-021-00973-8
Artigo de periodico
PD-1/PD-L1 inhibition enhances chemotherapy induced neuropathic pain by suppressing neuroimmune antinociceptive signaling (2022)
Wanderley, Carlos Wagner de Souza; Maganin, Alexandre Gomes de Macedo ; Mendes, Atlante Silva ; Silva, Conceição Elidianne Aníbal; Quadros, Andreza Urba de; Luiz, João Paulo Mesquita ; Silva, Camila Meirelles de Souza; Silva, Nicole Rodrigues; Oliveira, Francisco F; Gomes, Francisco Isaac Fernandes ; Restrepo, Jeffersson Leandro Jimenez ; Hernandez, Cesar Augusto Speck; Turaça, Fernanda ; Gabriel, Victor Lucena da; Pigatto, Glauce Regina; Nakaya, Helder Takashi Imoto ; Mota, Jose Mauricio; Sousa, Romualdo Barroso; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando Queiroz
Source: Cancer Immunology Research. Unidades: FCF, FMRP
Subjects: CITOTOXICIDADE IMUNOLÓGICA, IMUNOTERAPIA, MACRÓFAGOS, FARMACOTERAPIA
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ABNT
WANDERLEY, Carlos Wagner de Souza et al. PD-1/PD-L1 inhibition enhances chemotherapy induced neuropathic pain by suppressing neuroimmune antinociceptive signaling. Cancer Immunology Research, v. 10, n. 11, p. 1299–1308, 2022Tradução . . Disponível em: https://doi.org/10.1158/2326-6066.CIR-22-0003. Acesso em: 11 jul. 2024.
APA
Wanderley, C. W. de S., Maganin, A. G. de M., Mendes, A. S., Silva, C. E. A., Quadros, A. U. de, Luiz, J. P. M., et al. (2022). PD-1/PD-L1 inhibition enhances chemotherapy induced neuropathic pain by suppressing neuroimmune antinociceptive signaling. Cancer Immunology Research, 10( 11), 1299–1308. doi:10.1158/2326-6066.CIR-22-0003
NLM
Wanderley CW de S, Maganin AG de M, Mendes AS, Silva CEA, Quadros AU de, Luiz JPM, Silva CM de S, Silva NR, Oliveira FF, Gomes FIF, Restrepo JLJ, Hernandez CAS, Turaça F, Gabriel VL da, Pigatto GR, Nakaya HTI, Mota JM, Sousa RB, Alves Filho JCF, Cunha TM, Cunha FQ. PD-1/PD-L1 inhibition enhances chemotherapy induced neuropathic pain by suppressing neuroimmune antinociceptive signaling [Internet]. Cancer Immunology Research. 2022 ; 10( 11): 1299–1308.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1158/2326-6066.CIR-22-0003
Vancouver
Wanderley CW de S, Maganin AG de M, Mendes AS, Silva CEA, Quadros AU de, Luiz JPM, Silva CM de S, Silva NR, Oliveira FF, Gomes FIF, Restrepo JLJ, Hernandez CAS, Turaça F, Gabriel VL da, Pigatto GR, Nakaya HTI, Mota JM, Sousa RB, Alves Filho JCF, Cunha TM, Cunha FQ. PD-1/PD-L1 inhibition enhances chemotherapy induced neuropathic pain by suppressing neuroimmune antinociceptive signaling [Internet]. Cancer Immunology Research. 2022 ; 10( 11): 1299–1308.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1158/2326-6066.CIR-22-0003
Artigo de periodico
Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation (2022)
Borghi, Sergio M.; Bussulo, Sylvia K. D.; Pinho-Ribeiro, Felipe A.; Fattori, Victor; Carvalho, Thacyana T.; Rasquel-Oliveira, Fernanda S.; Zaninelli, Tiago H.; Ferraz, Camila R.; Casella, Antônio M. B.; Cunha, Fernando de Queiroz ; Cunha, Thiago Mattar ; Casagrande, Rubia; Verri Júnior, Waldiceu A.
Source: Frontiers in Pharmacology. Unidade: FMRP
Subjects: EXERCÍCIOS DE RESISTÊNCIA MUSCULAR, MEDULA ESPINHAL, INFLAMAÇÃO, DOR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGHI, Sergio M. et al. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation. Frontiers in Pharmacology, v. 12, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphar.2021.734091. Acesso em: 11 jul. 2024.
APA
Borghi, S. M., Bussulo, S. K. D., Pinho-Ribeiro, F. A., Fattori, V., Carvalho, T. T., Rasquel-Oliveira, F. S., et al. (2022). Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation. Frontiers in Pharmacology, 12, 1-17. doi:10.3389/fphar.2021.734091
NLM
Borghi SM, Bussulo SKD, Pinho-Ribeiro FA, Fattori V, Carvalho TT, Rasquel-Oliveira FS, Zaninelli TH, Ferraz CR, Casella AMB, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation [Internet]. Frontiers in Pharmacology. 2022 ; 12 1-17.[citado 2024 jul. 11 ] Available from: https://doi.org/10.3389/fphar.2021.734091
Vancouver
Borghi SM, Bussulo SKD, Pinho-Ribeiro FA, Fattori V, Carvalho TT, Rasquel-Oliveira FS, Zaninelli TH, Ferraz CR, Casella AMB, Cunha F de Q, Cunha TM, Casagrande R, Verri Júnior WA. Intense acute swimming induces delayed-onset muscle soreness dependent on spinal cord neuroinflammation [Internet]. Frontiers in Pharmacology. 2022 ; 12 1-17.[citado 2024 jul. 11 ] Available from: https://doi.org/10.3389/fphar.2021.734091
Artigo de periodico
Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions (2022)
Brandolini, Laura; d’Angelo, Michele; Novelli, Rubina; Castelli, Vanessa; Giorgio, Cristina; Sirico, Anna; Cocchiaro, Pasquale; D’Egidio, Francesco; Benedetti, Elisabetta; Cristiano, Claudia; Bugatti, Antonella; Quadros, Andreza Urba de; Cunha, Thiago Mattar
Source: Cell Death & Disease. Unidade: FMRP
Subjects: ANTINEOPLÁSICOS, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, FARMACOTERAPIA, DOR CRÔNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRANDOLINI, Laura et al. Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions. Cell Death & Disease, v. 13, n. 5, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41419-022-04964-w. Acesso em: 11 jul. 2024.
APA
Brandolini, L., d’Angelo, M., Novelli, R., Castelli, V., Giorgio, C., Sirico, A., et al. (2022). Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions. Cell Death & Disease, 13( 5), 1-15. doi:10.1038/s41419-022-04964-w
NLM
Brandolini L, d’Angelo M, Novelli R, Castelli V, Giorgio C, Sirico A, Cocchiaro P, D’Egidio F, Benedetti E, Cristiano C, Bugatti A, Quadros AU de, Cunha TM. Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions [Internet]. Cell Death & Disease. 2022 ; 13( 5): 1-15.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1038/s41419-022-04964-w
Vancouver
Brandolini L, d’Angelo M, Novelli R, Castelli V, Giorgio C, Sirico A, Cocchiaro P, D’Egidio F, Benedetti E, Cristiano C, Bugatti A, Quadros AU de, Cunha TM. Paclitaxel binds and activates C5aR1: a new potential therapeutic target for the prevention of chemotherapy-induced peripheral neuropathy and hypersensitivity reactions [Internet]. Cell Death & Disease. 2022 ; 13( 5): 1-15.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1038/s41419-022-04964-w
Artigo de periodico
Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice (2022)
Castro, Paula A. T. S.; Barbosa, Germanna M.; Machanocker, Dafiner H.; Peres, Raphael S.; Cunha, Thiago Mattar ; Cunha, Jonathan E.; Oliveira, Francisco F. B.; Ramalho, Fernando Silva ; Russo, Thiago L.; Cunha, Fernando de Queiroz ; Salvini, Tania F.
Source: PLOS ONE. Unidade: FMRP
Subjects: CRIOTERAPIA, INFLAMAÇÃO, ARTRITE, JOELHO
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ABNT
CASTRO, Paula A. T. S. et al. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice. PLOS ONE, v. 17, n. 1, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0261667. Acesso em: 11 jul. 2024.
APA
Castro, P. A. T. S., Barbosa, G. M., Machanocker, D. H., Peres, R. S., Cunha, T. M., Cunha, J. E., et al. (2022). Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice. PLOS ONE, 17( 1), 1-14. doi:10.1371/journal.pone.0261667
NLM
Castro PATS, Barbosa GM, Machanocker DH, Peres RS, Cunha TM, Cunha JE, Oliveira FFB, Ramalho FS, Russo TL, Cunha F de Q, Salvini TF. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice [Internet]. PLOS ONE. 2022 ; 17( 1): 1-14.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1371/journal.pone.0261667
Vancouver
Castro PATS, Barbosa GM, Machanocker DH, Peres RS, Cunha TM, Cunha JE, Oliveira FFB, Ramalho FS, Russo TL, Cunha F de Q, Salvini TF. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice [Internet]. PLOS ONE. 2022 ; 17( 1): 1-14.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1371/journal.pone.0261667
Artigo de periodico
Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection (2022)
Almeida, Leticia de; Silva, Alexandre Luiz Neves; Rodrigues, Tamara Silva; Oliveira, Samuel; Ishimoto, Adriene Yumi; Seribelli, Amanda Aparecida; Becerra, Amanda; Andrade, Warrison A; Ataíde, Marco Antônio; Caetano, Camila C. S.; Sá, Keyla Santos Guedes de; Pelisson, Natália; Martins, Ronaldo Bragança; Souza, Juliano de Paula; Arruda, Eurico; Batah, Sabrina Setembre; Castro, Ricardo; Frantz, Fabiani Gai; Cunha, Fernando Queiróz; Cunha, Thiago Mattar; Fabro, Alexandre Todorovic; Cunha, Larissa Dias; Louzada-Junior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões
Source: Science Advances. Unidades: FMRP, FCFRP
Subjects: INFECÇÕES RESPIRATÓRIAS, COVID-19, ADJUVANTES IMUNOLÓGICOS, INFLAMAÇÃO
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ABNT
ALMEIDA, Leticia de et al. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection. Science Advances, v. 8, n. 37, 2022Tradução . . Disponível em: https://doi.org/10.1126/sciadv.abo5400. Acesso em: 11 jul. 2024.
APA
Almeida, L. de, Silva, A. L. N., Rodrigues, T. S., Oliveira, S., Ishimoto, A. Y., Seribelli, A. A., et al. (2022). Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection. Science Advances, 8( 37). doi:10.1126/sciadv.abo5400
NLM
Almeida L de, Silva ALN, Rodrigues TS, Oliveira S, Ishimoto AY, Seribelli AA, Becerra A, Andrade WA, Ataíde MA, Caetano CCS, Sá KSG de, Pelisson N, Martins RB, Souza J de P, Arruda E, Batah SS, Castro R, Frantz FG, Cunha FQ, Cunha TM, Fabro AT, Cunha LD, Louzada-Junior P, Oliveira RDR de, Zamboni DS. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection [Internet]. Science Advances. 2022 ; 8( 37):[citado 2024 jul. 11 ] Available from: https://doi.org/10.1126/sciadv.abo5400
Vancouver
Almeida L de, Silva ALN, Rodrigues TS, Oliveira S, Ishimoto AY, Seribelli AA, Becerra A, Andrade WA, Ataíde MA, Caetano CCS, Sá KSG de, Pelisson N, Martins RB, Souza J de P, Arruda E, Batah SS, Castro R, Frantz FG, Cunha FQ, Cunha TM, Fabro AT, Cunha LD, Louzada-Junior P, Oliveira RDR de, Zamboni DS. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection [Internet]. Science Advances. 2022 ; 8( 37):[citado 2024 jul. 11 ] Available from: https://doi.org/10.1126/sciadv.abo5400
Artigo de periodico
SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients (2022)
Pontelli, Marjorie C.; Castro, Ítalo A.; Martins, Ronaldo B; La Serra, Leonardo; Veras, Flávio P.; Nascimento, Daniele C; Silva, Camila M.; Cardoso, Ricardo S; Rosales, Roberta Ribeiro Costa; Gomes, Rogério; Lima, Thais M.; Souza, Juliano P.; Vitti, Brenda C.; Caetité, Diego B.; Lima, Mikhael Haruo Fernandes de; Stumpf, Spencer D; Thompson, Cassandra E; Bloyet, Louis-Marie; Toller-Kawahisa, Juliana E.; Giannini, Marcela C.; Bonjorno, Letícia P; Lopes, Maria Isabel Fernandes; Batah, Sabrina Setembre; Siyuan, Li; Luppino-Assad, Rodrigo; Almeida, Sergio C L; Oliveira, Fabiola R; Benatti, Maíra N; Pontes, Lorena Lobo de Figueiredo; Santana, Rodrigo Carvalho; Vilar, Fernando C; Auxiliadora-Martins, Maria; Shi, Pei-Yong; Cunha, Thiago Mattar; Calado, Rodrigo Tocantins; Alves-Filho, José Carlos; Zamboni, Dario Simões; Fabro, Alexandre Todorovic; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro; Whelan, Sean P J; Cunha, Fernando Queiróz; Arruda, Eurico
Source: Journal of Molecular Cell Biology. Unidades: FCFRP, FMRP
Subjects: COVID-19, LINFÓCITOS, MONÓCITOS, APOPTOSE
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ABNT
PONTELLI, Marjorie C. et al. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients. Journal of Molecular Cell Biology, v. 14, n. 4, 2022Tradução . . Disponível em: https://doi.org/10.1093/jmcb/mjac021. Acesso em: 11 jul. 2024.
APA
Pontelli, M. C., Castro, Í. A., Martins, R. B., La Serra, L., Veras, F. P., Nascimento, D. C., et al. (2022). SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients. Journal of Molecular Cell Biology, 14( 4). doi:10.1093/jmcb/mjac021
NLM
Pontelli MC, Castro ÍA, Martins RB, La Serra L, Veras FP, Nascimento DC, Silva CM, Cardoso RS, Rosales RRC, Gomes R, Lima TM, Souza JP, Vitti BC, Caetité DB, Lima MHF de, Stumpf SD, Thompson CE, Bloyet L-M, Toller-Kawahisa JE, Giannini MC, Bonjorno LP, Lopes MIF, Batah SS, Siyuan L, Luppino-Assad R, Almeida SCL, Oliveira FR, Benatti MN, Pontes LL de F, Santana RC, Vilar FC, Auxiliadora-Martins M, Shi P-Y, Cunha TM, Calado RT, Alves-Filho JC, Zamboni DS, Fabro AT, Louzada Júnior P, Oliveira RDR, Whelan SPJ, Cunha FQ, Arruda E. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients [Internet]. Journal of Molecular Cell Biology. 2022 ; 14( 4):[citado 2024 jul. 11 ] Available from: https://doi.org/10.1093/jmcb/mjac021
Vancouver
Pontelli MC, Castro ÍA, Martins RB, La Serra L, Veras FP, Nascimento DC, Silva CM, Cardoso RS, Rosales RRC, Gomes R, Lima TM, Souza JP, Vitti BC, Caetité DB, Lima MHF de, Stumpf SD, Thompson CE, Bloyet L-M, Toller-Kawahisa JE, Giannini MC, Bonjorno LP, Lopes MIF, Batah SS, Siyuan L, Luppino-Assad R, Almeida SCL, Oliveira FR, Benatti MN, Pontes LL de F, Santana RC, Vilar FC, Auxiliadora-Martins M, Shi P-Y, Cunha TM, Calado RT, Alves-Filho JC, Zamboni DS, Fabro AT, Louzada Júnior P, Oliveira RDR, Whelan SPJ, Cunha FQ, Arruda E. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients [Internet]. Journal of Molecular Cell Biology. 2022 ; 14( 4):[citado 2024 jul. 11 ] Available from: https://doi.org/10.1093/jmcb/mjac021
Artigo de periodico
STING is an intrinsic checkpoint inhibitor that restrains the TH17 cell pathogenic program (2022)
Damasceno, Luis Eduardo Alves ; Cebinelli, Guilherme Cesar Martelossi ; Fernandes, Mariane Font ; Nascimento, Daniele Carvalho ; Públio, Gabriel Azevedo ; Vinolo, Marco Aurelio Ramirez ; Oliveira, Sergio Costa ; Sparwasser, Tim; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias
Source: Cell Reports. Unidades: FMRP, ICB
Subjects: CÉLULAS CULTIVADAS, INTERLEUCINA 10, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
DAMASCENO, Luis Eduardo Alves et al. STING is an intrinsic checkpoint inhibitor that restrains the TH17 cell pathogenic program. Cell Reports, v. 39, n. 8, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2022.110838. Acesso em: 11 jul. 2024.
APA
Damasceno, L. E. A., Cebinelli, G. C. M., Fernandes, M. F., Nascimento, D. C., Públio, G. A., Vinolo, M. A. R., et al. (2022). STING is an intrinsic checkpoint inhibitor that restrains the TH17 cell pathogenic program. Cell Reports, 39( 8), 1-16. doi:10.1016/j.celrep.2022.110838
NLM
Damasceno LEA, Cebinelli GCM, Fernandes MF, Nascimento DC, Públio GA, Vinolo MAR, Oliveira SC, Sparwasser T, Cunha TM, Cunha F de Q, Alves Filho JCF. STING is an intrinsic checkpoint inhibitor that restrains the TH17 cell pathogenic program [Internet]. Cell Reports. 2022 ; 39( 8): 1-16.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1016/j.celrep.2022.110838
Vancouver
Damasceno LEA, Cebinelli GCM, Fernandes MF, Nascimento DC, Públio GA, Vinolo MAR, Oliveira SC, Sparwasser T, Cunha TM, Cunha F de Q, Alves Filho JCF. STING is an intrinsic checkpoint inhibitor that restrains the TH17 cell pathogenic program [Internet]. Cell Reports. 2022 ; 39( 8): 1-16.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1016/j.celrep.2022.110838
Artigo de periodico
Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts (2022)
Charlie-Silva, Ives ; Feitosa, Natália M.; Pontes, Leticia Gomes de ; Fernandes, Bianca Helena Ventura ; Nóbrega, Rafael Henrique ; Gomes, Juliana Moreira Mendonça; Prata, Mariana N. L.; Ferraris, Fausto k; Freitas, Anderson Zanardi de ; Ribeiro, Martha Simões ; Cunha, Thiago Mattar
Source: Frontiers in Immunology. Unidades: FM, ICB, IPEN, FMRP
Subjects: PROTEÍNAS, CARRAGENINA, GLICOSAMINOGLICANAS, INFLAMAÇÃO, LEUCÓCITOS, NEUTRÓFILOS, PROTEÔMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHARLIE-SILVA, Ives et al. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts. Frontiers in Immunology, v. 13, p. 1-21, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.1019201. Acesso em: 11 jul. 2024.
APA
Charlie-Silva, I., Feitosa, N. M., Pontes, L. G. de, Fernandes, B. H. V., Nóbrega, R. H., Gomes, J. M. M., et al. (2022). Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts. Frontiers in Immunology, 13, 1-21. doi:10.3389/fimmu.2022.1019201
NLM
Charlie-Silva I, Feitosa NM, Pontes LG de, Fernandes BHV, Nóbrega RH, Gomes JMM, Prata MNL, Ferraris F k, Freitas AZ de, Ribeiro MS, Cunha TM. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts [Internet]. Frontiers in Immunology. 2022 ; 13 1-21.[citado 2024 jul. 11 ] Available from: https://doi.org/10.3389/fimmu.2022.1019201
Vancouver
Charlie-Silva I, Feitosa NM, Pontes LG de, Fernandes BHV, Nóbrega RH, Gomes JMM, Prata MNL, Ferraris F k, Freitas AZ de, Ribeiro MS, Cunha TM. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts [Internet]. Frontiers in Immunology. 2022 ; 13 1-21.[citado 2024 jul. 11 ] Available from: https://doi.org/10.3389/fimmu.2022.1019201
Artigo de periodico
Angiotensin type 2 receptor antagonism as a new target to manage gout (2022)
Vieira, Thiago Neves; Saraiva, André L. Lopes; Guimarães, Rafaela Mano ; Luiz, João Paulo Mesquita ; Pinto, Larissa Garcia; Ávila, Veridiana de Melo Rodrigues; Goulart, Luiz Ricardo ; Cunha Júnior, Jair Pereira; McNaughton, Peter Anthony; Cunha, Thiago Mattar ; Ferreira, Juliano; Silva, Cássia Regina
Source: Inflammopharmacology. Unidade: FMRP
Subjects: DOR, FARMACOTERAPIA, ANGIOTENSINA II, ANTI-INFLAMATÓRIOS, ANTIOXIDANTES, ARTRITE, ÁCIDO ÚRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VIEIRA, Thiago Neves et al. Angiotensin type 2 receptor antagonism as a new target to manage gout. Inflammopharmacology, v. 30, n. 6, p. 2399-2410, 2022Tradução . . Disponível em: https://doi.org/10.1007/s10787-022-01076-x. Acesso em: 11 jul. 2024.
APA
Vieira, T. N., Saraiva, A. L. L., Guimarães, R. M., Luiz, J. P. M., Pinto, L. G., Ávila, V. de M. R., et al. (2022). Angiotensin type 2 receptor antagonism as a new target to manage gout. Inflammopharmacology, 30( 6), 2399-2410. doi:10.1007/s10787-022-01076-x
NLM
Vieira TN, Saraiva ALL, Guimarães RM, Luiz JPM, Pinto LG, Ávila V de MR, Goulart LR, Cunha Júnior JP, McNaughton PA, Cunha TM, Ferreira J, Silva CR. Angiotensin type 2 receptor antagonism as a new target to manage gout [Internet]. Inflammopharmacology. 2022 ; 30( 6): 2399-2410.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1007/s10787-022-01076-x
Vancouver
Vieira TN, Saraiva ALL, Guimarães RM, Luiz JPM, Pinto LG, Ávila V de MR, Goulart LR, Cunha Júnior JP, McNaughton PA, Cunha TM, Ferreira J, Silva CR. Angiotensin type 2 receptor antagonism as a new target to manage gout [Internet]. Inflammopharmacology. 2022 ; 30( 6): 2399-2410.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1007/s10787-022-01076-x
Artigo de periodico
Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice (2022)
Castro, Paula A. T. S.; Machanocker, Dafiner H.; Luna, Genoveva F.; Barbosa, Germanna M.; Cunha, Jonathan E.; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Russo, Thiago L.; Salvini, Tania F.
Source: BioMed Research International. Unidade: FMRP
Subjects: CRIOTERAPIA, INFLAMAÇÃO, ARTRITE, JOELHO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CASTRO, Paula A. T. S. et al. Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice. BioMed Research International, p. 1-9, 2022Tradução . . Disponível em: https://doi.org/10.1155/2022/7442289. Acesso em: 11 jul. 2024.
APA
Castro, P. A. T. S., Machanocker, D. H., Luna, G. F., Barbosa, G. M., Cunha, J. E., Cunha, T. M., et al. (2022). Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice. BioMed Research International, 1-9. doi:10.1155/2022/7442289
NLM
Castro PATS, Machanocker DH, Luna GF, Barbosa GM, Cunha JE, Cunha TM, Cunha F de Q, Russo TL, Salvini TF. Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice [Internet]. BioMed Research International. 2022 ; 1-9.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1155/2022/7442289
Vancouver
Castro PATS, Machanocker DH, Luna GF, Barbosa GM, Cunha JE, Cunha TM, Cunha F de Q, Russo TL, Salvini TF. Clinical-like cryotherapy in acute knee arthritis protects neuromuscular junctions of quadriceps and reduces joint inflammation in mice [Internet]. BioMed Research International. 2022 ; 1-9.[citado 2024 jul. 11 ] Available from: https://doi.org/10.1155/2022/7442289
Artigo de periodico
Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice (2022)
Rodrigues, Patrícia Brito; Gomes, Giovanni Freitas; Angelim, Monara K. S. C.; Souza, Gabriela F.; Muraro, Stéfanie Primon ; Toledo-Teixeira, Daniel A.; Rattis, Bruna Amanda da Cruz; Passos, Amanda Stephane Cruz dos; Pral, Laís Passariello ; Rodovalho, Vinícius de Rezende ; Gomes, Arilson Bernardo dos Santos P.; Matheus, Valquíria Aparecida ; Antunes, André Saraiva Leão Marcelo ; Crunfli, Fernanda ; Antunes, Krist Helen; Souza, Ana Paula Duarte de; Consonni, Sílvio Roberto ; Leiria, Luiz Osório Silveira ; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Moraes-Vieira, Pedro Manoel Mendes de ; Modena, José Luiz Proenca ; Vinolo, Marco Aurelio Ramirez
Source: Cells. Unidade: FMRP
Subjects: COVID-19, MODELOS ANIMAIS DE DOENÇAS, QUIMIOTERÁPICOS, INFECÇÕES POR CORONAVIRUS, INFECÇÕES RESPIRATÓRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Patrícia Brito et al. Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice. Cells, v. 11, n. 16, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.3390/cells11162572. Acesso em: 11 jul. 2024.
APA
Rodrigues, P. B., Gomes, G. F., Angelim, M. K. S. C., Souza, G. F., Muraro, S. P., Toledo-Teixeira, D. A., et al. (2022). Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice. Cells, 11( 16), 1-16. doi:10.3390/cells11162572
NLM
Rodrigues PB, Gomes GF, Angelim MKSC, Souza GF, Muraro SP, Toledo-Teixeira DA, Rattis BA da C, Passos ASC dos, Pral LP, Rodovalho V de R, Gomes AB dos SP, Matheus VA, Antunes ASLM, Crunfli F, Antunes KH, Souza APD de, Consonni SR, Leiria LOS, Alves Filho JCF, Cunha TM, Moraes-Vieira PMM de, Modena JLP, Vinolo MAR. Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice [Internet]. Cells. 2022 ; 11( 16): 1-16.[citado 2024 jul. 11 ] Available from: https://doi.org/10.3390/cells11162572
Vancouver
Rodrigues PB, Gomes GF, Angelim MKSC, Souza GF, Muraro SP, Toledo-Teixeira DA, Rattis BA da C, Passos ASC dos, Pral LP, Rodovalho V de R, Gomes AB dos SP, Matheus VA, Antunes ASLM, Crunfli F, Antunes KH, Souza APD de, Consonni SR, Leiria LOS, Alves Filho JCF, Cunha TM, Moraes-Vieira PMM de, Modena JLP, Vinolo MAR. Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice [Internet]. Cells. 2022 ; 11( 16): 1-16.[citado 2024 jul. 11 ] Available from: https://doi.org/10.3390/cells11162572

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