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Artigo de periodico
Influence of nitro ruthenium isomerization on photochemically induced nitric oxide release: Vasorelaxant activities (2023)
Silva, Alexia Marques; Negri, Laísa Bonafim; Biazzotto, Juliana Cristina ; Machado, Sergio de Paula; Santos, Jeimison Duarte ; Batista, Jorge Fernandes Nasser; Maia, Pedro Ivo S.; Deflon, Victor Marcelo ; Bendhack, Lusiane Maria ; Hamblin, Michael R.; Silva, Roberto Santana da
Source: Journal of Inorganic Biochemistry. Unidades: FCFRP, IQSC, FMRP, FFCLRP
Subjects: ÓXIDO NÍTRICO, VASODILATAÇÃO
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ABNT
SILVA, Alexia Marques et al. Influence of nitro ruthenium isomerization on photochemically induced nitric oxide release: Vasorelaxant activities. Journal of Inorganic Biochemistry, v. 243, p. 112166, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.jinorgbio.2023.112166. Acesso em: 18 nov. 2024.
APA
Silva, A. M., Negri, L. B., Biazzotto, J. C., Machado, S. de P., Santos, J. D., Batista, J. F. N., et al. (2023). Influence of nitro ruthenium isomerization on photochemically induced nitric oxide release: Vasorelaxant activities. Journal of Inorganic Biochemistry, 243, 112166. doi:10.1016/j.jinorgbio.2023.112166
NLM
Silva AM, Negri LB, Biazzotto JC, Machado S de P, Santos JD, Batista JFN, Maia PIS, Deflon VM, Bendhack LM, Hamblin MR, Silva RS da. Influence of nitro ruthenium isomerization on photochemically induced nitric oxide release: Vasorelaxant activities [Internet]. Journal of Inorganic Biochemistry. 2023 ;243 112166.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jinorgbio.2023.112166
Vancouver
Silva AM, Negri LB, Biazzotto JC, Machado S de P, Santos JD, Batista JFN, Maia PIS, Deflon VM, Bendhack LM, Hamblin MR, Silva RS da. Influence of nitro ruthenium isomerization on photochemically induced nitric oxide release: Vasorelaxant activities [Internet]. Journal of Inorganic Biochemistry. 2023 ;243 112166.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jinorgbio.2023.112166
Artigo de periodico
C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta (2021)
Pernomian, Laena; Prado, Alejandro Ferraz do; Silva, Bruno Rodrigues; Paula, Tiago Dal-Cin de; Grando, Marcella Daruge; Bendhack, Lusiane Maria
Source: Life Sciences. Unidades: FMRP, FCFRP
Subjects: PEPTÍDEO NATRIURÉTICO ATRIAL, PROTEÍNAS, HIPERTENSÃO, RATOS
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PERNOMIAN, Laena et al. C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta. Life Sciences, v. 272, p. 1-14, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119223. Acesso em: 18 nov. 2024.
APA
Pernomian, L., Prado, A. F. do, Silva, B. R., Paula, T. D. -C. de, Grando, M. D., & Bendhack, L. M. (2021). C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta. Life Sciences, 272, 1-14. doi:10.1016/j.lfs.2021.119223
NLM
Pernomian L, Prado AF do, Silva BR, Paula TD-C de, Grando MD, Bendhack LM. C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta [Internet]. Life Sciences. 2021 ; 272 1-14.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.lfs.2021.119223
Vancouver
Pernomian L, Prado AF do, Silva BR, Paula TD-C de, Grando MD, Bendhack LM. C-type natriuretic peptide-induced relaxation through cGMP-dependent protein kinase and SERCA activation is impaired in two kidney-one clip rat aorta [Internet]. Life Sciences. 2021 ; 272 1-14.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.lfs.2021.119223
Artigo de periodico
The role of glycocalyx and caveolae in vascular homeostasis and diseases (2021)
Potje, Simone Regina ; Paula, Tiago Dal-Cin de; Paulo, Michele; Bendhack, Lusiane Maria
Source: Frontiers in Physiology. Unidades: FCFRP, FMRP
Subjects: HOMEOSTASE, ARTERIOSCLEROSE, CÉLULAS ENDOTELIAIS, HIPERTENSÃO, MATRIZ EXTRACELULAR, ÓXIDO NÍTRICO, DOENÇAS CARDIOVASCULARES
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POTJE, Simone Regina et al. The role of glycocalyx and caveolae in vascular homeostasis and diseases. Frontiers in Physiology, v. 11, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphys.2020.620840. Acesso em: 18 nov. 2024.
APA
Potje, S. R., Paula, T. D. -C. de, Paulo, M., & Bendhack, L. M. (2021). The role of glycocalyx and caveolae in vascular homeostasis and diseases. Frontiers in Physiology, 11, 1-13. doi:10.3389/fphys.2020.620840
NLM
Potje SR, Paula TD-C de, Paulo M, Bendhack LM. The role of glycocalyx and caveolae in vascular homeostasis and diseases [Internet]. Frontiers in Physiology. 2021 ; 11 1-13.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fphys.2020.620840
Vancouver
Potje SR, Paula TD-C de, Paulo M, Bendhack LM. The role of glycocalyx and caveolae in vascular homeostasis and diseases [Internet]. Frontiers in Physiology. 2021 ; 11 1-13.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fphys.2020.620840
Artigo de periodico
Thromboxane-prostanoid receptor activation blocks ATP-sensitive potassium channels in rat aortas (2021)
Santos, Jeimison Duarte; Paulo, Michele; Vercesi, Juliana Aparecida; Bendhack, Lusiane Maria
Source: Clinical and Experimental Pharmacology and Physiology. Unidades: FCFRP, FMRP
Subjects: TROMBOXANOS, RECEPTORES, CANAIS DE POTÁSSIO, RATOS, AORTA DE ANIMAL
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ABNT
SANTOS, Jeimison Duarte et al. Thromboxane-prostanoid receptor activation blocks ATP-sensitive potassium channels in rat aortas. Clinical and Experimental Pharmacology and Physiology, v. 48, n. 11, p. 1537-1546, 2021Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.13557. Acesso em: 18 nov. 2024.
APA
Santos, J. D., Paulo, M., Vercesi, J. A., & Bendhack, L. M. (2021). Thromboxane-prostanoid receptor activation blocks ATP-sensitive potassium channels in rat aortas. Clinical and Experimental Pharmacology and Physiology, 48( 11), 1537-1546. doi:10.1111/1440-1681.13557
NLM
Santos JD, Paulo M, Vercesi JA, Bendhack LM. Thromboxane-prostanoid receptor activation blocks ATP-sensitive potassium channels in rat aortas [Internet]. Clinical and Experimental Pharmacology and Physiology. 2021 ; 48( 11): 1537-1546.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/1440-1681.13557
Vancouver
Santos JD, Paulo M, Vercesi JA, Bendhack LM. Thromboxane-prostanoid receptor activation blocks ATP-sensitive potassium channels in rat aortas [Internet]. Clinical and Experimental Pharmacology and Physiology. 2021 ; 48( 11): 1537-1546.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/1440-1681.13557
Artigo de periodico
Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium (2021)
Potje, Simone Regina ; Isbatan, Ayman; Tostes, Rita de Cássia Aleixo ; Bendhack, Lusiane Maria ; Dull, Randal O.; Carvalho-de-Souza, João Luis; Chignalia, Andréia Zago
Source: Pharmacological Research. Unidades: FMRP, FCFRP
Subjects: AORTA TORÁCICA, AORTA TORÁCICA, PRESSÃO SANGUÍNEA, CÁLCIO, HIPERTENSÃO, NOREPINEFRINA
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POTJE, Simone Regina et al. Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium. Pharmacological Research, v. 172, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2021.105813. Acesso em: 18 nov. 2024.
APA
Potje, S. R., Isbatan, A., Tostes, R. de C. A., Bendhack, L. M., Dull, R. O., Carvalho-de-Souza, J. L., & Chignalia, A. Z. (2021). Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium. Pharmacological Research, 172, 1-11. doi:10.1016/j.phrs.2021.105813
NLM
Potje SR, Isbatan A, Tostes R de CA, Bendhack LM, Dull RO, Carvalho-de-Souza JL, Chignalia AZ. Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium [Internet]. Pharmacological Research. 2021 ; 172 1-11.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.phrs.2021.105813
Vancouver
Potje SR, Isbatan A, Tostes R de CA, Bendhack LM, Dull RO, Carvalho-de-Souza JL, Chignalia AZ. Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: focus on IP3R and calcium [Internet]. Pharmacological Research. 2021 ; 172 1-11.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.phrs.2021.105813
Artigo de periodico
Central administration of angiotensin-(1-7) improves vasopressin impairment and hypotensive response in experimental endotoxemia (2021)
Passaglia, Patrícia ; Faim, Felipe de Lima; Batalhão, Marcelo Eduardo; Stabile, Angelita Maria ; Bendhack, Lusiane Maria ; Antunes-Rodrigues, José ; Lacchini, Riccardo ; Cárnio, Evelin Capellari
Source: Cells. Unidades: EERP, FCFRP, FMRP
Subjects: ANGIOTENSINAS, INFLAMAÇÃO, VASOPRESSINAS, HIPOTENSÃO, ÓXIDO NÍTRICO, SISTEMA RENINA-ANGIOTENSINA, NEUROPEPTÍDEOS
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ABNT
PASSAGLIA, Patrícia et al. Central administration of angiotensin-(1-7) improves vasopressin impairment and hypotensive response in experimental endotoxemia. Cells, v. 10, n. 1, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10010105. Acesso em: 18 nov. 2024.
APA
Passaglia, P., Faim, F. de L., Batalhão, M. E., Stabile, A. M., Bendhack, L. M., Antunes-Rodrigues, J., et al. (2021). Central administration of angiotensin-(1-7) improves vasopressin impairment and hypotensive response in experimental endotoxemia. Cells, 10( 1), 1-16. doi:10.3390/cells10010105
NLM
Passaglia P, Faim F de L, Batalhão ME, Stabile AM, Bendhack LM, Antunes-Rodrigues J, Lacchini R, Cárnio EC. Central administration of angiotensin-(1-7) improves vasopressin impairment and hypotensive response in experimental endotoxemia [Internet]. Cells. 2021 ; 10( 1): 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells10010105
Vancouver
Passaglia P, Faim F de L, Batalhão ME, Stabile AM, Bendhack LM, Antunes-Rodrigues J, Lacchini R, Cárnio EC. Central administration of angiotensin-(1-7) improves vasopressin impairment and hypotensive response in experimental endotoxemia [Internet]. Cells. 2021 ; 10( 1): 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells10010105
Artigo de periodico
Effects of progesterone treatment on endothelium-dependent coronary relaxation in ovariectomized rats (2020)
Cunha, Tagana Rosa da; Giesen, Jéssyca Aparecida Soares; Rouver, Wender Nascimento; Costa, Eduardo Damasceno; Grando, Marcella Daruge; Lemos, Virgínia Soares; Bendhack, Lusiane Maria ; Santos, Roger Lyrio dos
Source: Life Sciences. Unidades: FMRP, FCFRP
Subjects: PROGESTERONA, ENDOTÉLIO, SISTEMA CARDIOVASCULAR, MODELOS ANIMAIS, OVARIECTOMIA
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ABNT
CUNHA, Tagana Rosa da et al. Effects of progesterone treatment on endothelium-dependent coronary relaxation in ovariectomized rats. Life Sciences, v. 247, p. 1-10, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2020.117391. Acesso em: 18 nov. 2024.
APA
Cunha, T. R. da, Giesen, J. A. S., Rouver, W. N., Costa, E. D., Grando, M. D., Lemos, V. S., et al. (2020). Effects of progesterone treatment on endothelium-dependent coronary relaxation in ovariectomized rats. Life Sciences, 247, 1-10. doi:10.1016/j.lfs.2020.117391
NLM
Cunha TR da, Giesen JAS, Rouver WN, Costa ED, Grando MD, Lemos VS, Bendhack LM, Santos RL dos. Effects of progesterone treatment on endothelium-dependent coronary relaxation in ovariectomized rats [Internet]. Life Sciences. 2020 ; 247 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.lfs.2020.117391
Vancouver
Cunha TR da, Giesen JAS, Rouver WN, Costa ED, Grando MD, Lemos VS, Bendhack LM, Santos RL dos. Effects of progesterone treatment on endothelium-dependent coronary relaxation in ovariectomized rats [Internet]. Life Sciences. 2020 ; 247 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.lfs.2020.117391
Artigo de periodico
Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors (2020)
Corrêa, José Wilson do Nascimento; Prado, Cibele Maria ; Riul, Maria Elena; Araújo, Alice Valença; Rossi, Marcos Antonio; Bendhack, Lusiane Maria
Source: Clinical and Experimental Pharmacology and Physiology. Unidades: FZEA, FCFRP
Subjects: HIPERTENSÃO, SISTEMA RENINA-ANGIOTENSINA, MODELOS ANIMAIS
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ABNT
CORRÊA, José Wilson do Nascimento et al. Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors. Clinical and Experimental Pharmacology and Physiology, v. 47, n. 12, p. 1965-1977, 2020Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.13384. Acesso em: 18 nov. 2024.
APA
Corrêa, J. W. do N., Prado, C. M., Riul, M. E., Araújo, A. V., Rossi, M. A., & Bendhack, L. M. (2020). Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors. Clinical and Experimental Pharmacology and Physiology, 47( 12), 1965-1977. doi:10.1111/1440-1681.13384
NLM
Corrêa JW do N, Prado CM, Riul ME, Araújo AV, Rossi MA, Bendhack LM. Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors [Internet]. Clinical and Experimental Pharmacology and Physiology. 2020 ; 47( 12): 1965-1977.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/1440-1681.13384
Vancouver
Corrêa JW do N, Prado CM, Riul ME, Araújo AV, Rossi MA, Bendhack LM. Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors [Internet]. Clinical and Experimental Pharmacology and Physiology. 2020 ; 47( 12): 1965-1977.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/1440-1681.13384
Artigo de periodico
Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats (2020)
Passaglia, Patrícia; Faim, Felipe de Lima; Batalhão, Marcelo Eduardo; Bendhack, Lusiane Maria ; Antunes-Rodrigues, José; Ulloa, Luis; Kanashiro, Alexandre; Cárnio, Evelin Capellari
Source: Brain, Behavior, and Immunity. Unidades: EERP, FCFRP, FMRP
Subjects: ANGIOTENSINAS, LIPOPOLISSACARÍDEOS, SISTEMA NERVOSO SIMPÁTICO, INFLAMAÇÃO, NEUROIMUNOMODULAÇÃO, HIPOTENSÃO
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ABNT
PASSAGLIA, Patrícia et al. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats. Brain, Behavior, and Immunity, v. 88, p. 606-618, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2020.04.059. Acesso em: 18 nov. 2024.
APA
Passaglia, P., Faim, F. de L., Batalhão, M. E., Bendhack, L. M., Antunes-Rodrigues, J., Ulloa, L., et al. (2020). Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats. Brain, Behavior, and Immunity, 88, 606-618. doi:10.1016/j.bbi.2020.04.059
NLM
Passaglia P, Faim F de L, Batalhão ME, Bendhack LM, Antunes-Rodrigues J, Ulloa L, Kanashiro A, Cárnio EC. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats [Internet]. Brain, Behavior, and Immunity. 2020 ; 88 606-618.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbi.2020.04.059
Vancouver
Passaglia P, Faim F de L, Batalhão ME, Bendhack LM, Antunes-Rodrigues J, Ulloa L, Kanashiro A, Cárnio EC. Central angiotensin-(1–7) attenuates systemic inflammation via activation of sympathetic signaling in endotoxemic rats [Internet]. Brain, Behavior, and Immunity. 2020 ; 88 606-618.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.bbi.2020.04.059
Artigo de periodico
Testosterone increases bradykinin-induced relaxation in the coronary bed of hypertensive rats (2020)
Arapa-Diaz, Juan Carlos; Rouver, Wender do Nascimento; Giesen, Jéssyca Aparecida Soares; Grando, Marcela Daruge; Bendhack, Lusiane Maria ; Santos, Roger Lyrio dos
Source: Journal of Molecular Endocrinology. Unidades: FMRP, FCFRP
Subjects: ANIMAIS, BIOMARCADORES, PRESSÃO SANGUÍNEA, BRADICININA, VASOS CORONÁRIOS, ENDOTÉLIO, HIPERTENSÃO
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ABNT
ARAPA-DIAZ, Juan Carlos et al. Testosterone increases bradykinin-induced relaxation in the coronary bed of hypertensive rats. Journal of Molecular Endocrinology, v. 65, n. 4, p. 125-134, 2020Tradução . . Disponível em: https://doi.org/10.1530/JME-20-0153. Acesso em: 18 nov. 2024.
APA
Arapa-Diaz, J. C., Rouver, W. do N., Giesen, J. A. S., Grando, M. D., Bendhack, L. M., & Santos, R. L. dos. (2020). Testosterone increases bradykinin-induced relaxation in the coronary bed of hypertensive rats. Journal of Molecular Endocrinology, 65( 4), 125-134. doi:10.1530/JME-20-0153
NLM
Arapa-Diaz JC, Rouver W do N, Giesen JAS, Grando MD, Bendhack LM, Santos RL dos. Testosterone increases bradykinin-induced relaxation in the coronary bed of hypertensive rats [Internet]. Journal of Molecular Endocrinology. 2020 ; 65( 4): 125-134.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1530/JME-20-0153
Vancouver
Arapa-Diaz JC, Rouver W do N, Giesen JAS, Grando MD, Bendhack LM, Santos RL dos. Testosterone increases bradykinin-induced relaxation in the coronary bed of hypertensive rats [Internet]. Journal of Molecular Endocrinology. 2020 ; 65( 4): 125-134.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1530/JME-20-0153
Artigo de periodico
Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction (2020)
Paulo, Michele ; Costa, Daniela E. F. R.; Bonaventura, Daniella; Lunardi, Claure N.; Bendhack, Lusiane Maria
Source: Current Pharmaceutical Design. Unidades: FCFRP, ICB
Subjects: CÉLULAS ENDOTELIAIS, ENDOTÉLIO VASCULAR, ÓXIDO NÍTRICO, FARMACOLOGIA, DOENÇAS VASCULARES
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ABNT
PAULO, Michele et al. Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction. Current Pharmaceutical Design, v. 26, n. 30, p. 3748-3759, 2020Tradução . . Disponível em: https://doi.org/10.2174/1381612826666200519114442. Acesso em: 18 nov. 2024.
APA
Paulo, M., Costa, D. E. F. R., Bonaventura, D., Lunardi, C. N., & Bendhack, L. M. (2020). Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction. Current Pharmaceutical Design, 26( 30), 3748-3759. doi:10.2174/1381612826666200519114442
NLM
Paulo M, Costa DEFR, Bonaventura D, Lunardi CN, Bendhack LM. Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction [Internet]. Current Pharmaceutical Design. 2020 ; 26( 30): 3748-3759.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2174/1381612826666200519114442
Vancouver
Paulo M, Costa DEFR, Bonaventura D, Lunardi CN, Bendhack LM. Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction [Internet]. Current Pharmaceutical Design. 2020 ; 26( 30): 3748-3759.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2174/1381612826666200519114442
Artigo de periodico
Exposure to acetaminophen impairs vasodilation, increases oxidative stress and changes arterial morphology of rats (2019)
Porto, Hellen Karine Paes; Grando, Marcella Daruge; Ramalho, Leandra Naira Zambelli ; Valadares, Marize Campos ; Bendhack, Lusiane Maria ; Batista, Aline Carvalho ; Rocha, Matheus Lavorenti
Source: Archives of Toxicology. Unidades: FCFRP, FMRP
Subjects: SISTEMA CARDIOVASCULAR, PARACETAMOL, ESTRESSE OXIDATIVO, VASODILATAÇÃO
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ABNT
PORTO, Hellen Karine Paes et al. Exposure to acetaminophen impairs vasodilation, increases oxidative stress and changes arterial morphology of rats. Archives of Toxicology, v. 93, n. 7, p. 1955-1964, 2019Tradução . . Disponível em: https://doi.org/10.1007/s00204-019-02463-0. Acesso em: 18 nov. 2024.
APA
Porto, H. K. P., Grando, M. D., Ramalho, L. N. Z., Valadares, M. C., Bendhack, L. M., Batista, A. C., & Rocha, M. L. (2019). Exposure to acetaminophen impairs vasodilation, increases oxidative stress and changes arterial morphology of rats. Archives of Toxicology, 93( 7), 1955-1964. doi:10.1007/s00204-019-02463-0
NLM
Porto HKP, Grando MD, Ramalho LNZ, Valadares MC, Bendhack LM, Batista AC, Rocha ML. Exposure to acetaminophen impairs vasodilation, increases oxidative stress and changes arterial morphology of rats [Internet]. Archives of Toxicology. 2019 ; 93( 7): 1955-1964.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00204-019-02463-0
Vancouver
Porto HKP, Grando MD, Ramalho LNZ, Valadares MC, Bendhack LM, Batista AC, Rocha ML. Exposure to acetaminophen impairs vasodilation, increases oxidative stress and changes arterial morphology of rats [Internet]. Archives of Toxicology. 2019 ; 93( 7): 1955-1964.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s00204-019-02463-0
Trabalho de evento-resumo
Espécies reativas de oxigênio podem modular negativamente o relaxamento induzido pelo nitroprussiato de sódio em aortas de ratos hipertensos (2019)
Silva, Matheus Henrique da; Grando, Marcella Daruge ; Vercesi, Juliana Aparecida; Bendhack, Lusiane Maria
Source: Resumos. Conference titles: Simpósio Internacional de Iniciação Científica e Tecnológica da USP (SIICUSP). Unidades: FMRP, FCFRP
Subjects: HIPERTENSÃO, RATOS WISTAR, FÁRMACOS (SISTEMA CARDIOVASCULAR), VASODILATAÇÃO
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ABNT
SILVA, Matheus Henrique da et al. Espécies reativas de oxigênio podem modular negativamente o relaxamento induzido pelo nitroprussiato de sódio em aortas de ratos hipertensos. 2019, Anais.. Ribeirão Preto: FCFRP-USP, 2019. Disponível em: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210. Acesso em: 18 nov. 2024.
APA
Silva, M. H. da, Grando, M. D., Vercesi, J. A., & Bendhack, L. M. (2019). Espécies reativas de oxigênio podem modular negativamente o relaxamento induzido pelo nitroprussiato de sódio em aortas de ratos hipertensos. In Resumos. Ribeirão Preto: FCFRP-USP. Recuperado de https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
NLM
Silva MH da, Grando MD, Vercesi JA, Bendhack LM. Espécies reativas de oxigênio podem modular negativamente o relaxamento induzido pelo nitroprussiato de sódio em aortas de ratos hipertensos [Internet]. Resumos. 2019 ;[citado 2024 nov. 18 ] Available from: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
Vancouver
Silva MH da, Grando MD, Vercesi JA, Bendhack LM. Espécies reativas de oxigênio podem modular negativamente o relaxamento induzido pelo nitroprussiato de sódio em aortas de ratos hipertensos [Internet]. Resumos. 2019 ;[citado 2024 nov. 18 ] Available from: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
Artigo de periodico
Apocynin alters redox signaling in conductance and resistance vessels of spontaneously hypertensive rats (2019)
Graton, Murilo Eduardo ; Potje, Simone Regina ; Troiano, Jéssica Antonini ; Vale, Gabriel Tavares do ; Perassa, Ligia Arnedo; Nakamune, Ana Claudia de Melo Stevanato ; Tirapelli, Carlos Renato ; Bendhack, Lusiane Maria ; Antoniali, Cristina
Source: Free Radical Biology and Medicine. Unidades: EERP, FCFRP
Subjects: ANGIOTENSINA II, ÓXIDO NÍTRICO, RADICAIS LIVRES
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ABNT
GRATON, Murilo Eduardo et al. Apocynin alters redox signaling in conductance and resistance vessels of spontaneously hypertensive rats. Free Radical Biology and Medicine, v. 134, p. 53-63, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2018.12.026. Acesso em: 18 nov. 2024.
APA
Graton, M. E., Potje, S. R., Troiano, J. A., Vale, G. T. do, Perassa, L. A., Nakamune, A. C. de M. S., et al. (2019). Apocynin alters redox signaling in conductance and resistance vessels of spontaneously hypertensive rats. Free Radical Biology and Medicine, 134, 53-63. doi:10.1016/j.freeradbiomed.2018.12.026
NLM
Graton ME, Potje SR, Troiano JA, Vale GT do, Perassa LA, Nakamune AC de MS, Tirapelli CR, Bendhack LM, Antoniali C. Apocynin alters redox signaling in conductance and resistance vessels of spontaneously hypertensive rats [Internet]. Free Radical Biology and Medicine. 2019 ; 134 53-63.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.12.026
Vancouver
Graton ME, Potje SR, Troiano JA, Vale GT do, Perassa LA, Nakamune AC de MS, Tirapelli CR, Bendhack LM, Antoniali C. Apocynin alters redox signaling in conductance and resistance vessels of spontaneously hypertensive rats [Internet]. Free Radical Biology and Medicine. 2019 ; 134 53-63.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.12.026
Artigo de periodico
Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production (2019)
Potje, Simone Regina ; Grando, Marcella Daruge ; Chignalia, Andreia Z.; Antoniali, Cristina; Bendhack, Lusiane Maria
Source: Scientific Reports. Unidade: FCFRP
Subjects: ARTÉRIAS, MEMBRANA PLASMÁTICA, COLESTEROL, HIPERTENSÃO, ÓXIDO NÍTRICO
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ABNT
POTJE, Simone Regina et al. Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production. Scientific Reports, v. 9, p. [16] , 2019Tradução . . Disponível em: https://doi.org/10.1038/s41598-019-43193-8. Acesso em: 18 nov. 2024.
APA
Potje, S. R., Grando, M. D., Chignalia, A. Z., Antoniali, C., & Bendhack, L. M. (2019). Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production. Scientific Reports, 9, [16] . doi:10.1038/s41598-019-43193-8
NLM
Potje SR, Grando MD, Chignalia AZ, Antoniali C, Bendhack LM. Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production [Internet]. Scientific Reports. 2019 ; 9 [16] .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-019-43193-8
Vancouver
Potje SR, Grando MD, Chignalia AZ, Antoniali C, Bendhack LM. Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production [Internet]. Scientific Reports. 2019 ; 9 [16] .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-019-43193-8
Trabalho de evento-resumo
Óxido nítrico produzido pelas células endoteliais contribui para a vasodilatação induzida pelo iloprost (2019)
Kikugava, Tayna Frigel; Bendhack, Lusiane Maria
Source: Resumos. Conference titles: Simpósio Internacional de Iniciação Científica e Tecnológica da USP (SIICUSP). Unidade: FCFRP
Subjects: ÓXIDO NÍTRICO, CÉLULAS ENDOTELIAIS, VASODILATAÇÃO, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KIKUGAVA, Tayna Frigel e BENDHACK, Lusiane Maria. Óxido nítrico produzido pelas células endoteliais contribui para a vasodilatação induzida pelo iloprost. 2019, Anais.. Ribeirão Preto: FCFRP-USP, 2019. Disponível em: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210. Acesso em: 18 nov. 2024.
APA
Kikugava, T. F., & Bendhack, L. M. (2019). Óxido nítrico produzido pelas células endoteliais contribui para a vasodilatação induzida pelo iloprost. In Resumos. Ribeirão Preto: FCFRP-USP. Recuperado de https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
NLM
Kikugava TF, Bendhack LM. Óxido nítrico produzido pelas células endoteliais contribui para a vasodilatação induzida pelo iloprost [Internet]. Resumos. 2019 ;[citado 2024 nov. 18 ] Available from: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
Vancouver
Kikugava TF, Bendhack LM. Óxido nítrico produzido pelas células endoteliais contribui para a vasodilatação induzida pelo iloprost [Internet]. Resumos. 2019 ;[citado 2024 nov. 18 ] Available from: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
Artigo de periodico
NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats (2019)
Araújo, Alice Valença ; Andrade, Fernanda Aparecida de; Paulo, Michele ; Paula, Tiago Dal Cin de; Potje, Simone Regina ; Pereira, Amanda de Carvalho; Bendhack, Lusiane Maria
Source: Nitric Oxide. Unidades: FCFRP, FMRP
Subjects: ÓXIDO NÍTRICO, HIPERTENSÃO, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAÚJO, Alice Valença et al. NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats. Nitric Oxide, v. 86, p. 12-20, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2019.02.004. Acesso em: 18 nov. 2024.
APA
Araújo, A. V., Andrade, F. A. de, Paulo, M., Paula, T. D. C. de, Potje, S. R., Pereira, A. de C., & Bendhack, L. M. (2019). NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats. Nitric Oxide, 86, 12-20. doi:10.1016/j.niox.2019.02.004
NLM
Araújo AV, Andrade FA de, Paulo M, Paula TDC de, Potje SR, Pereira A de C, Bendhack LM. NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats [Internet]. Nitric Oxide. 2019 ; 86 12-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.niox.2019.02.004
Vancouver
Araújo AV, Andrade FA de, Paulo M, Paula TDC de, Potje SR, Pereira A de C, Bendhack LM. NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats [Internet]. Nitric Oxide. 2019 ; 86 12-20.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.niox.2019.02.004
Artigo de periodico
Matrix metalloproteinase-2-induced epidermal growth factor receptor transactivation impairs redox balance in vascular smooth muscle cells and facilitates vascular contraction (2018)
Prado, Alejandro Ferraz do; Pernomian, Laena; Azevedo, Aline; Costa, Rute Alves Pereira e; Rizzi, Elen; Ramos, Junia; Leme, Adriana Franco Paes; Bendhack, Lusiane Maria; Tanus-Santos, José Eduardo; Gerlach, Raquel Fernanda
Source: Redox Biology. Unidades: FORP, FCFRP, FMRP
Subjects: ANTIOXIDANTES, METALOPROTEINASES, CÉLULAS MUSCULARES
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ABNT
PRADO, Alejandro Ferraz do et al. Matrix metalloproteinase-2-induced epidermal growth factor receptor transactivation impairs redox balance in vascular smooth muscle cells and facilitates vascular contraction. Redox Biology, v. 18, p. 181-190, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.redox.2018.07.005. Acesso em: 18 nov. 2024.
APA
Prado, A. F. do, Pernomian, L., Azevedo, A., Costa, R. A. P. e, Rizzi, E., Ramos, J., et al. (2018). Matrix metalloproteinase-2-induced epidermal growth factor receptor transactivation impairs redox balance in vascular smooth muscle cells and facilitates vascular contraction. Redox Biology, 18, 181-190. doi:10.1016/j.redox.2018.07.005
NLM
Prado AF do, Pernomian L, Azevedo A, Costa RAP e, Rizzi E, Ramos J, Leme AFP, Bendhack LM, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinase-2-induced epidermal growth factor receptor transactivation impairs redox balance in vascular smooth muscle cells and facilitates vascular contraction [Internet]. Redox Biology. 2018 ; 18 181-190.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.redox.2018.07.005
Vancouver
Prado AF do, Pernomian L, Azevedo A, Costa RAP e, Rizzi E, Ramos J, Leme AFP, Bendhack LM, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinase-2-induced epidermal growth factor receptor transactivation impairs redox balance in vascular smooth muscle cells and facilitates vascular contraction [Internet]. Redox Biology. 2018 ; 18 181-190.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.redox.2018.07.005
Artigo de periodico
Activation of TP receptors induces high release of PGI2 in coronary arteries of renal hypertensive rats (2018)
Paula, Tiago Dal-Cin de; Silva, Bruno Rodrigues da; Grando, Marcella Daruge; Souza, Hugo Celso Dutra de; Bendhack, Lusiane Maria
Source: Journal of Molecular and Cellular Cardiology. Unidades: FCFRP, FMRP
Subjects: HIPERTENSÃO, VASOS CORONÁRIOS, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAULA, Tiago Dal-Cin de et al. Activation of TP receptors induces high release of PGI2 in coronary arteries of renal hypertensive rats. Journal of Molecular and Cellular Cardiology, v. 122, p. 125-133, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.yjmcc.2018.08.007. Acesso em: 18 nov. 2024.
APA
Paula, T. D. -C. de, Silva, B. R. da, Grando, M. D., Souza, H. C. D. de, & Bendhack, L. M. (2018). Activation of TP receptors induces high release of PGI2 in coronary arteries of renal hypertensive rats. Journal of Molecular and Cellular Cardiology, 122, 125-133. doi:10.1016/j.yjmcc.2018.08.007
NLM
Paula TD-C de, Silva BR da, Grando MD, Souza HCD de, Bendhack LM. Activation of TP receptors induces high release of PGI2 in coronary arteries of renal hypertensive rats [Internet]. Journal of Molecular and Cellular Cardiology. 2018 ; 122 125-133.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.yjmcc.2018.08.007
Vancouver
Paula TD-C de, Silva BR da, Grando MD, Souza HCD de, Bendhack LM. Activation of TP receptors induces high release of PGI2 in coronary arteries of renal hypertensive rats [Internet]. Journal of Molecular and Cellular Cardiology. 2018 ; 122 125-133.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.yjmcc.2018.08.007
Artigo de periodico
LmrBPP9: A synthetic bradykinin-potentiating peptide from Lachesis muta rhombeata venom that inhibits the angiotensin-converting enzyme activity in vitro and reduces the blood pressure of hypertensive rats (2018)
Pinheiro-Júnior, Ernesto Lopes ; Boldrini-França, Johara; Araújo, Luciana Mattoso Pires de Campos; Santos-Filho, Norival Alves; Bendhack, Lusiane Maria ; Cilli, Eduardo Maffud; Arantes, Eliane Candiani
Source: Peptides. Unidade: FCFRP
Subjects: PEPTÍDEOS, BRADICININA, VENENOS, COBRAS, TOXINAS, ANGIOTENSINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINHEIRO-JÚNIOR, Ernesto Lopes et al. LmrBPP9: A synthetic bradykinin-potentiating peptide from Lachesis muta rhombeata venom that inhibits the angiotensin-converting enzyme activity in vitro and reduces the blood pressure of hypertensive rats. Peptides, v. 102, p. 1-7, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.peptides.2018.01.015. Acesso em: 18 nov. 2024.
APA
Pinheiro-Júnior, E. L., Boldrini-França, J., Araújo, L. M. P. de C., Santos-Filho, N. A., Bendhack, L. M., Cilli, E. M., & Arantes, E. C. (2018). LmrBPP9: A synthetic bradykinin-potentiating peptide from Lachesis muta rhombeata venom that inhibits the angiotensin-converting enzyme activity in vitro and reduces the blood pressure of hypertensive rats. Peptides, 102, 1-7. doi:10.1016/j.peptides.2018.01.015
NLM
Pinheiro-Júnior EL, Boldrini-França J, Araújo LMP de C, Santos-Filho NA, Bendhack LM, Cilli EM, Arantes EC. LmrBPP9: A synthetic bradykinin-potentiating peptide from Lachesis muta rhombeata venom that inhibits the angiotensin-converting enzyme activity in vitro and reduces the blood pressure of hypertensive rats [Internet]. Peptides. 2018 ; 102 1-7.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.peptides.2018.01.015
Vancouver
Pinheiro-Júnior EL, Boldrini-França J, Araújo LMP de C, Santos-Filho NA, Bendhack LM, Cilli EM, Arantes EC. LmrBPP9: A synthetic bradykinin-potentiating peptide from Lachesis muta rhombeata venom that inhibits the angiotensin-converting enzyme activity in vitro and reduces the blood pressure of hypertensive rats [Internet]. Peptides. 2018 ; 102 1-7.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.peptides.2018.01.015

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