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Artigo de periodico
Modified Atkins diet for drug-resistant epilepsy: A systematic review and meta-analysis of randomized controlled trials (2023)
Mutarelli, Antonio ; Nogueira, Alleh ; Felix, Nicole ; Godoi, Amanda ; Dagostin, Caroline Serafim ; Castro, Luiz Henrique Martins ; Telles, Joao Paulo Mota
Source: Seizure-european journal of epilepsy. Unidade: FM
Subjects: DIETA, EPILEPSIA, ANTICONVULSIVANTES, ENSAIO CLÍNICO CONTROLADO RANDOMIZADO
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ABNT
MUTARELLI, Antonio et al. Modified Atkins diet for drug-resistant epilepsy: A systematic review and meta-analysis of randomized controlled trials. Seizure-european journal of epilepsy, v. 112, p. 77-83, 2023Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/57458. Acesso em: 01 nov. 2024.
APA
Mutarelli, A., Nogueira, A., Felix, N., Godoi, A., Dagostin, C. S., Castro, L. H. M., & Telles, J. P. M. (2023). Modified Atkins diet for drug-resistant epilepsy: A systematic review and meta-analysis of randomized controlled trials. Seizure-european journal of epilepsy, 112, 77-83. doi:10.1016/j.seizure.2023.09.010
NLM
Mutarelli A, Nogueira A, Felix N, Godoi A, Dagostin CS, Castro LHM, Telles JPM. Modified Atkins diet for drug-resistant epilepsy: A systematic review and meta-analysis of randomized controlled trials [Internet]. Seizure-european journal of epilepsy. 2023 ; 112 77-83.[citado 2024 nov. 01 ] Available from: https://observatorio.fm.usp.br/handle/OPI/57458
Vancouver
Mutarelli A, Nogueira A, Felix N, Godoi A, Dagostin CS, Castro LHM, Telles JPM. Modified Atkins diet for drug-resistant epilepsy: A systematic review and meta-analysis of randomized controlled trials [Internet]. Seizure-european journal of epilepsy. 2023 ; 112 77-83.[citado 2024 nov. 01 ] Available from: https://observatorio.fm.usp.br/handle/OPI/57458
Artigo de periodico
Congenital fiber type disproportion caused by TPM3 mutation: a report of two atypical cases (2020)
Moreno, Cristiane Araujo Martins; Estephan, Eduardo de Paula; Fappi, Alan; Monges, Soledad; Lubieniecki, Fabiana; Abath Neto, Osorio Lopes; Reed, Umbertina Conti ; Donkervoort, Sandra; Harms, Matthew B; Bonnemann, Carsten; Zanoteli, Edmar
Source: Neuromuscular disorders. Unidade: FM
Subjects: BIOPOLÍMEROS, MIOPATIAS CONGÊNITAS ESTRUTURAIS, ESTUDO DE CASO
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MORENO, Cristiane Araujo Martins et al. Congenital fiber type disproportion caused by TPM3 mutation: a report of two atypical cases. Neuromuscular disorders, v. 30, n. 1, p. 54-58, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.nmd.2019.11.001. Acesso em: 01 nov. 2024.
APA
Moreno, C. A. M., Estephan, E. de P., Fappi, A., Monges, S., Lubieniecki, F., Abath Neto, O. L., et al. (2020). Congenital fiber type disproportion caused by TPM3 mutation: a report of two atypical cases. Neuromuscular disorders, 30( 1), 54-58. doi:10.1016/j.nmd.2019.11.001
NLM
Moreno CAM, Estephan E de P, Fappi A, Monges S, Lubieniecki F, Abath Neto OL, Reed UC, Donkervoort S, Harms MB, Bonnemann C, Zanoteli E. Congenital fiber type disproportion caused by TPM3 mutation: a report of two atypical cases [Internet]. Neuromuscular disorders. 2020 ; 30( 1): 54-58.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.nmd.2019.11.001
Vancouver
Moreno CAM, Estephan E de P, Fappi A, Monges S, Lubieniecki F, Abath Neto OL, Reed UC, Donkervoort S, Harms MB, Bonnemann C, Zanoteli E. Congenital fiber type disproportion caused by TPM3 mutation: a report of two atypical cases [Internet]. Neuromuscular disorders. 2020 ; 30( 1): 54-58.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.nmd.2019.11.001
Artigo de periodico
rAAV-related therapy fully rescues myonuclear and myofilament function in X-linked myotubular myopathy (2020)
Ross, Jacob A; Tasfaout, Hichem; Levy, Yotam; Morgan, Jennifer; Cowling, Belinda S; Laporte, Jocelyn; Zanoteli, Edmar ; Romero, Norma B; Lowe, Dawn A; Jungbluth, Heinz
Source: Acta neuropathologica communications. Unidade: FM
Subjects: MICROSCOPIA, CONTRAÇÃO MUSCULAR, FIBRAS MUSCULARES, MIOPATIAS CONGÊNITAS ESTRUTURAIS
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ABNT
ROSS, Jacob A et al. rAAV-related therapy fully rescues myonuclear and myofilament function in X-linked myotubular myopathy. Acta neuropathologica communications, v. 8, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1186/s40478-020-01048-8. Acesso em: 01 nov. 2024.
APA
Ross, J. A., Tasfaout, H., Levy, Y., Morgan, J., Cowling, B. S., Laporte, J., et al. (2020). rAAV-related therapy fully rescues myonuclear and myofilament function in X-linked myotubular myopathy. Acta neuropathologica communications, 8( 1). doi:10.1186/s40478-020-01048-8
NLM
Ross JA, Tasfaout H, Levy Y, Morgan J, Cowling BS, Laporte J, Zanoteli E, Romero NB, Lowe DA, Jungbluth H. rAAV-related therapy fully rescues myonuclear and myofilament function in X-linked myotubular myopathy [Internet]. Acta neuropathologica communications. 2020 ; 8( 1):[citado 2024 nov. 01 ] Available from: https://doi.org/10.1186/s40478-020-01048-8
Vancouver
Ross JA, Tasfaout H, Levy Y, Morgan J, Cowling BS, Laporte J, Zanoteli E, Romero NB, Lowe DA, Jungbluth H. rAAV-related therapy fully rescues myonuclear and myofilament function in X-linked myotubular myopathy [Internet]. Acta neuropathologica communications. 2020 ; 8( 1):[citado 2024 nov. 01 ] Available from: https://doi.org/10.1186/s40478-020-01048-8
Artigo de periodico
Bi-allelic GAD1 variants cause a neonatal onset syndromic developmental and epileptic encephalopathy (2020)
Chatron, Nicolas; Becker, Felicitas; Morsy, Heba; Schmidts, Miriam; Hardies, Katia; Tuysuz, Beyhan; Roselli, Sandra; Najafi, Maryam; Alkaya, Dilek Uludag; Kok, Fernando
Source: Brain. Unidade: FM
Subjects: FENÓTIPOS, FISSURA PALATINA, PACIENTES
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ABNT
CHATRON, Nicolas et al. Bi-allelic GAD1 variants cause a neonatal onset syndromic developmental and epileptic encephalopathy. Brain, v. 143, p. 1447-1461, 2020Tradução . . Disponível em: https://doi.org/10.1093/brain/awaa085. Acesso em: 01 nov. 2024.
APA
Chatron, N., Becker, F., Morsy, H., Schmidts, M., Hardies, K., Tuysuz, B., et al. (2020). Bi-allelic GAD1 variants cause a neonatal onset syndromic developmental and epileptic encephalopathy. Brain, 143, 1447-1461. doi:10.1093/brain/awaa085
NLM
Chatron N, Becker F, Morsy H, Schmidts M, Hardies K, Tuysuz B, Roselli S, Najafi M, Alkaya DU, Kok F. Bi-allelic GAD1 variants cause a neonatal onset syndromic developmental and epileptic encephalopathy [Internet]. Brain. 2020 ; 143 1447-1461.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1093/brain/awaa085
Vancouver
Chatron N, Becker F, Morsy H, Schmidts M, Hardies K, Tuysuz B, Roselli S, Najafi M, Alkaya DU, Kok F. Bi-allelic GAD1 variants cause a neonatal onset syndromic developmental and epileptic encephalopathy [Internet]. Brain. 2020 ; 143 1447-1461.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1093/brain/awaa085
Artigo de periodico
Molecular and histomorphological evaluation of female rats' urethral tissues after an innovative trauma model of prolonged vaginal distention: immediate, short-term and long-term effects (2019)
Bortolini, Maria A. T; Feitosa, Suellen M; Bilhar, Andreisa P. M; Salerno, Gisela G. R; Zanoteli, Edmar; Simoes, Manuel J; Castro, Rodrigo A
Source: International urogynecology journal. Unidade: FM
Subjects: INCONTINÊNCIA URINÁRIA, URETRA, MODELOS ANIMAIS, BIOLOGIA MOLECULAR, MIOSINA, COLÁGENO, FATORES DE CRESCIMENTO
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ABNT
BORTOLINI, Maria A. T et al. Molecular and histomorphological evaluation of female rats' urethral tissues after an innovative trauma model of prolonged vaginal distention: immediate, short-term and long-term effects. International urogynecology journal, v. 30, n. 3, p. 465-476, 2019Tradução . . Disponível em: https://doi.org/10.1007/s00192-018-3634-2. Acesso em: 01 nov. 2024.
APA
Bortolini, M. A. T., Feitosa, S. M., Bilhar, A. P. M., Salerno, G. G. R., Zanoteli, E., Simoes, M. J., & Castro, R. A. (2019). Molecular and histomorphological evaluation of female rats' urethral tissues after an innovative trauma model of prolonged vaginal distention: immediate, short-term and long-term effects. International urogynecology journal, 30( 3), 465-476. doi:10.1007/s00192-018-3634-2
NLM
Bortolini MAT, Feitosa SM, Bilhar APM, Salerno GGR, Zanoteli E, Simoes MJ, Castro RA. Molecular and histomorphological evaluation of female rats' urethral tissues after an innovative trauma model of prolonged vaginal distention: immediate, short-term and long-term effects [Internet]. International urogynecology journal. 2019 ; 30( 3): 465-476.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00192-018-3634-2
Vancouver
Bortolini MAT, Feitosa SM, Bilhar APM, Salerno GGR, Zanoteli E, Simoes MJ, Castro RA. Molecular and histomorphological evaluation of female rats' urethral tissues after an innovative trauma model of prolonged vaginal distention: immediate, short-term and long-term effects [Internet]. International urogynecology journal. 2019 ; 30( 3): 465-476.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00192-018-3634-2
Artigo de periodico
Altered cortical excitability in persistent idiopathic facial pain (2019)
Galhardoni, Ricardo; Andrade, Daniel Ciampi de; Puerta, Mariana Y. T; Brunoni, André Russowsky; Varotto, Bruna L. R; Siqueira, Jose T. T. de; Teixeira, Manoel Jacobsen; Siqueira, Silvia R. D. T
Source: Cephalalgia. Unidade: FM
Subjects: CÓRTEX CEREBRAL, DOR FACIAL, DOR CRÔNICA
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ABNT
GALHARDONI, Ricardo et al. Altered cortical excitability in persistent idiopathic facial pain. Cephalalgia, v. 39, n. 2, p. 219-228, 2019Tradução . . Disponível em: https://doi.org/10.1177/0333102418780426. Acesso em: 01 nov. 2024.
APA
Galhardoni, R., Andrade, D. C. de, Puerta, M. Y. T., Brunoni, A. R., Varotto, B. L. R., Siqueira, J. T. T. de, et al. (2019). Altered cortical excitability in persistent idiopathic facial pain. Cephalalgia, 39( 2), 219-228. doi:10.1177/0333102418780426
NLM
Galhardoni R, Andrade DC de, Puerta MYT, Brunoni AR, Varotto BLR, Siqueira JTT de, Teixeira MJ, Siqueira SRDT. Altered cortical excitability in persistent idiopathic facial pain [Internet]. Cephalalgia. 2019 ; 39( 2): 219-228.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1177/0333102418780426
Vancouver
Galhardoni R, Andrade DC de, Puerta MYT, Brunoni AR, Varotto BLR, Siqueira JTT de, Teixeira MJ, Siqueira SRDT. Altered cortical excitability in persistent idiopathic facial pain [Internet]. Cephalalgia. 2019 ; 39( 2): 219-228.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1177/0333102418780426
Artigo de periodico
AMPA receptor GluA2 subunit defects are a cause of neurodevelopmental disorders (2019)
Salpietro, Vincenzo; Dixon, Christine L; Guo, Hui; Bello, Oscar D; Vandrovcova, Jana; Efthymiou, Stephanie; Maroofian, Reza; Heimer, Gali; Burglen, Lydie; Kok, Fernando
Source: Nature communications. Unidade: FM
Subjects: RECEPTORES DE GLUTAMATO, TRANSTORNOS MENTAIS DIAGNOSTICADOS NA INFÂNCIA, EXPRESSÃO GÊNICA, MUTAÇÃO GENÉTICA
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ABNT
SALPIETRO, Vincenzo et al. AMPA receptor GluA2 subunit defects are a cause of neurodevelopmental disorders. Nature communications, v. 10, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41467-019-10910-w. Acesso em: 01 nov. 2024.
APA
Salpietro, V., Dixon, C. L., Guo, H., Bello, O. D., Vandrovcova, J., Efthymiou, S., et al. (2019). AMPA receptor GluA2 subunit defects are a cause of neurodevelopmental disorders. Nature communications, 10. doi:10.1038/s41467-019-10910-w
NLM
Salpietro V, Dixon CL, Guo H, Bello OD, Vandrovcova J, Efthymiou S, Maroofian R, Heimer G, Burglen L, Kok F. AMPA receptor GluA2 subunit defects are a cause of neurodevelopmental disorders [Internet]. Nature communications. 2019 ; 10[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/s41467-019-10910-w
Vancouver
Salpietro V, Dixon CL, Guo H, Bello OD, Vandrovcova J, Efthymiou S, Maroofian R, Heimer G, Burglen L, Kok F. AMPA receptor GluA2 subunit defects are a cause of neurodevelopmental disorders [Internet]. Nature communications. 2019 ; 10[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/s41467-019-10910-w
Artigo de periodico
Glibenclamide in aneurysmatic subarachnoid hemorrhage (GASH): study protocol for a randomized controlled trial (2019)
Costa, Bruno Braga Sisnando da; Windlin, Isabela Costola; Koterba, Edwin; Yamaki, Vitor Nagai; Rabelo, Nicollas Nunes; Solla, Davi Jorge Fontoura; Teixeira, Manoel Jacobsen ; Figueiredo, Eberval Gadelha
Source: Trials. Unidade: FM
Subjects: HEMORRAGIA SUBARACNOIDE, ANEURISMA INTRACRANIANO, ANTIDIABÉTICOS (HIPOGLICEMIANTES), ENSAIO CLÍNICO CONTROLADO RANDOMIZADO
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ABNT
COSTA, Bruno Braga Sisnando da et al. Glibenclamide in aneurysmatic subarachnoid hemorrhage (GASH): study protocol for a randomized controlled trial. Trials, v. 20, 2019Tradução . . Disponível em: https://doi.org/10.1186/s13063-019-3517-y. Acesso em: 01 nov. 2024.
APA
Costa, B. B. S. da, Windlin, I. C., Koterba, E., Yamaki, V. N., Rabelo, N. N., Solla, D. J. F., et al. (2019). Glibenclamide in aneurysmatic subarachnoid hemorrhage (GASH): study protocol for a randomized controlled trial. Trials, 20. doi:10.1186/s13063-019-3517-y
NLM
Costa BBS da, Windlin IC, Koterba E, Yamaki VN, Rabelo NN, Solla DJF, Teixeira MJ, Figueiredo EG. Glibenclamide in aneurysmatic subarachnoid hemorrhage (GASH): study protocol for a randomized controlled trial [Internet]. Trials. 2019 ; 20[citado 2024 nov. 01 ] Available from: https://doi.org/10.1186/s13063-019-3517-y
Vancouver
Costa BBS da, Windlin IC, Koterba E, Yamaki VN, Rabelo NN, Solla DJF, Teixeira MJ, Figueiredo EG. Glibenclamide in aneurysmatic subarachnoid hemorrhage (GASH): study protocol for a randomized controlled trial [Internet]. Trials. 2019 ; 20[citado 2024 nov. 01 ] Available from: https://doi.org/10.1186/s13063-019-3517-y
Artigo de periodico
Clinical, ophthalmological, imaging and genetic features in Brazilian patients with ARSACS (2019)
Rezende Filho, Flavio Moura; Parkinson, Michael H; Pedroso, Jose Luiz; Poh, Roy; Faber, Ingrid; Lourenco, Charles Marques; Marques Junior, Wilson; Franca Junior, Marcondes Cavalcante; Kok, Fernando ; Sallum, Juliana M. Ferraz
Source: Parkinsonism & related disorders. Unidade: FM
Subjects: ATAXIA, NEUROIMAGEM, TOMOGRAFIA DE COERÊNCIA ÓPTICA, FENÓTIPOS, GENÓTIPOS, RETINA
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ABNT
REZENDE FILHO, Flavio Moura et al. Clinical, ophthalmological, imaging and genetic features in Brazilian patients with ARSACS. Parkinsonism & related disorders, v. 62, p. 148-155, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.parkreldis.2018.12.024. Acesso em: 01 nov. 2024.
APA
Rezende Filho, F. M., Parkinson, M. H., Pedroso, J. L., Poh, R., Faber, I., Lourenco, C. M., et al. (2019). Clinical, ophthalmological, imaging and genetic features in Brazilian patients with ARSACS. Parkinsonism & related disorders, 62, 148-155. doi:10.1016/j.parkreldis.2018.12.024
NLM
Rezende Filho FM, Parkinson MH, Pedroso JL, Poh R, Faber I, Lourenco CM, Marques Junior W, Franca Junior MC, Kok F, Sallum JMF. Clinical, ophthalmological, imaging and genetic features in Brazilian patients with ARSACS [Internet]. Parkinsonism & related disorders. 2019 ; 62 148-155.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.parkreldis.2018.12.024
Vancouver
Rezende Filho FM, Parkinson MH, Pedroso JL, Poh R, Faber I, Lourenco CM, Marques Junior W, Franca Junior MC, Kok F, Sallum JMF. Clinical, ophthalmological, imaging and genetic features in Brazilian patients with ARSACS [Internet]. Parkinsonism & related disorders. 2019 ; 62 148-155.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.parkreldis.2018.12.024
Artigo de periodico
Evolution of gamma knife capsulotomy for intractable obsessive-compulsive disorder (2019)
Miguel Filho, Eurípedes Constantino ; Lopes, Antonio C; Mclaughlin, Nicole C. R; Noren, Georg; Gentil, Andre F; Hamani, Clement; Shavitt, Roseli G; Batistuzzo, Marcelo C; Vattimo, Edoardo F. Q; Fonoff, Erich Talamoni
Source: Molecular psychiatry. Unidade: FM
Subjects: TERAPIA COGNITIVA, CÓRTEX PRÉ-FRONTAL, NEUROCIRURGIA, NEUROANATOMIA
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ABNT
MIGUEL FILHO, Eurípedes Constantino et al. Evolution of gamma knife capsulotomy for intractable obsessive-compulsive disorder. Molecular psychiatry, v. 24, n. 2, p. 218-240, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41380-018-0054-0. Acesso em: 01 nov. 2024.
APA
Miguel Filho, E. C., Lopes, A. C., Mclaughlin, N. C. R., Noren, G., Gentil, A. F., Hamani, C., et al. (2019). Evolution of gamma knife capsulotomy for intractable obsessive-compulsive disorder. Molecular psychiatry, 24( 2), 218-240. doi:10.1038/s41380-018-0054-0
NLM
Miguel Filho EC, Lopes AC, Mclaughlin NCR, Noren G, Gentil AF, Hamani C, Shavitt RG, Batistuzzo MC, Vattimo EFQ, Fonoff ET. Evolution of gamma knife capsulotomy for intractable obsessive-compulsive disorder [Internet]. Molecular psychiatry. 2019 ; 24( 2): 218-240.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/s41380-018-0054-0
Vancouver
Miguel Filho EC, Lopes AC, Mclaughlin NCR, Noren G, Gentil AF, Hamani C, Shavitt RG, Batistuzzo MC, Vattimo EFQ, Fonoff ET. Evolution of gamma knife capsulotomy for intractable obsessive-compulsive disorder [Internet]. Molecular psychiatry. 2019 ; 24( 2): 218-240.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/s41380-018-0054-0
Artigo de periodico-carta/editorial
Effects of dentate nucleus stimulation in spinocerebellar ataxia type 3 [Carta] (2019)
Cury, Rubens Gisbert; Franca, Carina; Silva, Valquiria; Barbosa, Egberto Reis; Capato, Tamine T. C; Lepski, Guilherme; Duarte, Kleber Paiva; Teixeira, Manoel Jacobsen ; Andrade, Daniel Ciampi de
Source: Parkinsonism & related disorders. Unidade: FM
Subjects: ATAXIA, DEGENERAÇÕES ESPINOCEREBELARES, ESTIMULAÇÃO CEREBRAL, CEREBELO, ESTIMULAÇÃO MAGNÉTICA TRANSCRANIANA, CUIDADOS PRÉ-OPERATÓRIOS
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ABNT
CURY, Rubens Gisbert et al. Effects of dentate nucleus stimulation in spinocerebellar ataxia type 3 [Carta]. Parkinsonism & related disorders. Oxford: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.parkreldis.2019.10.029. Acesso em: 01 nov. 2024. , 2019
APA
Cury, R. G., Franca, C., Silva, V., Barbosa, E. R., Capato, T. T. C., Lepski, G., et al. (2019). Effects of dentate nucleus stimulation in spinocerebellar ataxia type 3 [Carta]. Parkinsonism & related disorders. Oxford: Faculdade de Medicina, Universidade de São Paulo. doi:10.1016/j.parkreldis.2019.10.029
NLM
Cury RG, Franca C, Silva V, Barbosa ER, Capato TTC, Lepski G, Duarte KP, Teixeira MJ, Andrade DC de. Effects of dentate nucleus stimulation in spinocerebellar ataxia type 3 [Carta] [Internet]. Parkinsonism & related disorders. 2019 ; 69 91-93.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.parkreldis.2019.10.029
Vancouver
Cury RG, Franca C, Silva V, Barbosa ER, Capato TTC, Lepski G, Duarte KP, Teixeira MJ, Andrade DC de. Effects of dentate nucleus stimulation in spinocerebellar ataxia type 3 [Carta] [Internet]. Parkinsonism & related disorders. 2019 ; 69 91-93.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.parkreldis.2019.10.029
Artigo de periodico
The clinical spectrum of the congenital myasthenic syndrome resulting from COL13A1 mutations (2019)
Cruz, Pedro M. Rodriguez; Cossins, Judith; Estephan, Eduardo de Paula; Munell, Francina; Selby, Kathryn; Hirano, Michio; Maroofin, Reza; Mehrjardi, Mohammad Yahya Vahidi; Zanoteli, Edmar ; Reed, Umbertina Conti
Source: Brain. Unidade: FM
Subjects: DOENÇAS DA JUNÇÃO NEUROMUSCULAR, PROTEÍNAS DE MATRIZ EXTRACELULARES, SINAPSE, MUTAÇÃO GENÉTICA, RECEPTORES ADRENÉRGICOS, AMINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CRUZ, Pedro M. Rodriguez et al. The clinical spectrum of the congenital myasthenic syndrome resulting from COL13A1 mutations. Brain, v. 142, p. 1547-1560, 2019Tradução . . Disponível em: https://doi.org/10.1093/brain/awz107. Acesso em: 01 nov. 2024.
APA
Cruz, P. M. R., Cossins, J., Estephan, E. de P., Munell, F., Selby, K., Hirano, M., et al. (2019). The clinical spectrum of the congenital myasthenic syndrome resulting from COL13A1 mutations. Brain, 142, 1547-1560. doi:10.1093/brain/awz107
NLM
Cruz PMR, Cossins J, Estephan E de P, Munell F, Selby K, Hirano M, Maroofin R, Mehrjardi MYV, Zanoteli E, Reed UC. The clinical spectrum of the congenital myasthenic syndrome resulting from COL13A1 mutations [Internet]. Brain. 2019 ; 142 1547-1560.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1093/brain/awz107
Vancouver
Cruz PMR, Cossins J, Estephan E de P, Munell F, Selby K, Hirano M, Maroofin R, Mehrjardi MYV, Zanoteli E, Reed UC. The clinical spectrum of the congenital myasthenic syndrome resulting from COL13A1 mutations [Internet]. Brain. 2019 ; 142 1547-1560.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1093/brain/awz107
Artigo de periodico
Effects of mnemonic strategy training on brain activity and cognitive functioning of left-hemisphere ischemic stroke patients (2019)
Batista, Alana X; Bazan, Paulo R; Conforto, Adriana B; Ma, Maria da Graca M; Simon, Sharon S; Hampstead, Benjamin; Figueiredo, Eberval Gadelha; Miotto, Eliane C
Source: Neural plasticity. Unidade: FM
Subjects: ISQUEMIA CEREBRAL, TRANSTORNOS DA MEMÓRIA, TERAPIA COGNITIVA, MEMORIZAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BATISTA, Alana X et al. Effects of mnemonic strategy training on brain activity and cognitive functioning of left-hemisphere ischemic stroke patients. Neural plasticity, 2019Tradução . . Disponível em: https://doi.org/10.1155/2019/4172569. Acesso em: 01 nov. 2024.
APA
Batista, A. X., Bazan, P. R., Conforto, A. B., Ma, M. da G. M., Simon, S. S., Hampstead, B., et al. (2019). Effects of mnemonic strategy training on brain activity and cognitive functioning of left-hemisphere ischemic stroke patients. Neural plasticity. doi:10.1155/2019/4172569
NLM
Batista AX, Bazan PR, Conforto AB, Ma M da GM, Simon SS, Hampstead B, Figueiredo EG, Miotto EC. Effects of mnemonic strategy training on brain activity and cognitive functioning of left-hemisphere ischemic stroke patients [Internet]. Neural plasticity. 2019 ;[citado 2024 nov. 01 ] Available from: https://doi.org/10.1155/2019/4172569
Vancouver
Batista AX, Bazan PR, Conforto AB, Ma M da GM, Simon SS, Hampstead B, Figueiredo EG, Miotto EC. Effects of mnemonic strategy training on brain activity and cognitive functioning of left-hemisphere ischemic stroke patients [Internet]. Neural plasticity. 2019 ;[citado 2024 nov. 01 ] Available from: https://doi.org/10.1155/2019/4172569
Artigo de periodico
A review on shared clinical and molecular mechanisms between bipolar disorder and frontotemporal dementia (2019)
Nascimento, Camila; Nunes, Paula Villela; Rodriguez, Roberta Diehl; Takada, Leonel; Suemoto, Claudia Kimie; Grinberg, Lea Tenenholz; Nitrini, Ricardo; Lafer, Beny
Source: Progress in neuro-psychopharmacology & biological psychiatry. Unidade: FM
Subjects: TRANSTORNO BIPOLAR, DEMÊNCIA, BIOMARCADORES, MANIFESTAÇÕES NEUROLÓGICAS, DIAGNÓSTICO DIFERENCIAL
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ABNT
NASCIMENTO, Camila et al. A review on shared clinical and molecular mechanisms between bipolar disorder and frontotemporal dementia. Progress in neuro-psychopharmacology & biological psychiatry, v. 93, p. 269-283, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.pnpbp.2019.04.008. Acesso em: 01 nov. 2024.
APA
Nascimento, C., Nunes, P. V., Rodriguez, R. D., Takada, L., Suemoto, C. K., Grinberg, L. T., et al. (2019). A review on shared clinical and molecular mechanisms between bipolar disorder and frontotemporal dementia. Progress in neuro-psychopharmacology & biological psychiatry, 93, 269-283. doi:10.1016/j.pnpbp.2019.04.008
NLM
Nascimento C, Nunes PV, Rodriguez RD, Takada L, Suemoto CK, Grinberg LT, Nitrini R, Lafer B. A review on shared clinical and molecular mechanisms between bipolar disorder and frontotemporal dementia [Internet]. Progress in neuro-psychopharmacology & biological psychiatry. 2019 ; 93 269-283.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.pnpbp.2019.04.008
Vancouver
Nascimento C, Nunes PV, Rodriguez RD, Takada L, Suemoto CK, Grinberg LT, Nitrini R, Lafer B. A review on shared clinical and molecular mechanisms between bipolar disorder and frontotemporal dementia [Internet]. Progress in neuro-psychopharmacology & biological psychiatry. 2019 ; 93 269-283.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.pnpbp.2019.04.008
Artigo de periodico
214th ENMC International Workshop: Establishing an international consortium for gene discovery and clinical research for Congenital Muscle Disease, Heemskerk, the Netherlands, 6-18 October 2015 (2019)
Donkervoort, Sandra; Dowling, James J; Laporte, Jocelyn; Macarthur, Daniel; Bonnemann, Carsten G; Beggs, Alan; Bonne, Gisele; Bonnemann, Carsten; Donkervoort, Sandra; Zanoteli, Edmar
Source: Neuromuscular disorders. Unidade: FM
Subjects: DOENÇAS MUSCULARES, COOPERAÇÃO INTERNACIONAL, CONGRESSO INTERNACIONAL, GENÓTIPOS, FENÓTIPOS, TÉCNICAS GENÉTICAS, ESTUDOS DE COORTES
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ABNT
DONKERVOORT, Sandra et al. 214th ENMC International Workshop: Establishing an international consortium for gene discovery and clinical research for Congenital Muscle Disease, Heemskerk, the Netherlands, 6-18 October 2015. Neuromuscular disorders, v. 29, n. 8, p. 644-650, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.nmd.2019.07.002. Acesso em: 01 nov. 2024.
APA
Donkervoort, S., Dowling, J. J., Laporte, J., Macarthur, D., Bonnemann, C. G., Beggs, A., et al. (2019). 214th ENMC International Workshop: Establishing an international consortium for gene discovery and clinical research for Congenital Muscle Disease, Heemskerk, the Netherlands, 6-18 October 2015. Neuromuscular disorders, 29( 8), 644-650. doi:10.1016/j.nmd.2019.07.002
NLM
Donkervoort S, Dowling JJ, Laporte J, Macarthur D, Bonnemann CG, Beggs A, Bonne G, Bonnemann C, Donkervoort S, Zanoteli E. 214th ENMC International Workshop: Establishing an international consortium for gene discovery and clinical research for Congenital Muscle Disease, Heemskerk, the Netherlands, 6-18 October 2015 [Internet]. Neuromuscular disorders. 2019 ; 29( 8): 644-650.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.nmd.2019.07.002
Vancouver
Donkervoort S, Dowling JJ, Laporte J, Macarthur D, Bonnemann CG, Beggs A, Bonne G, Bonnemann C, Donkervoort S, Zanoteli E. 214th ENMC International Workshop: Establishing an international consortium for gene discovery and clinical research for Congenital Muscle Disease, Heemskerk, the Netherlands, 6-18 October 2015 [Internet]. Neuromuscular disorders. 2019 ; 29( 8): 644-650.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.nmd.2019.07.002
Artigo de periodico
Non-motor outcomes depend on location of neurostimulation in Parkinson's disease (2019)
Petry-schmelzer, Jan Niklas; Krause, Max; Dembek, Till A; Horn, Andreas; Evans, Julian; Ashkan, Keyoumars; Rizos, Alexandra; Silverdale, Monty; Schumacher, Wibke; Fonoff, Erich Talamoni
Source: Brain. Unidade: FM
Subjects: DOENÇA DE PARKINSON, ESTIMULAÇÃO CEREBRAL, NÚCLEOS TALÂMICOS, ESTUDOS PROSPECTIVOS, RESULTADO DE TRATAMENTO
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ABNT
PETRY-SCHMELZER, Jan Niklas et al. Non-motor outcomes depend on location of neurostimulation in Parkinson's disease. Brain, v. 142, p. 3592-3604, 2019Tradução . . Disponível em: https://doi.org/10.1093/brain/awz285. Acesso em: 01 nov. 2024.
APA
Petry-schmelzer, J. N., Krause, M., Dembek, T. A., Horn, A., Evans, J., Ashkan, K., et al. (2019). Non-motor outcomes depend on location of neurostimulation in Parkinson's disease. Brain, 142, 3592-3604. doi:10.1093/brain/awz285
NLM
Petry-schmelzer JN, Krause M, Dembek TA, Horn A, Evans J, Ashkan K, Rizos A, Silverdale M, Schumacher W, Fonoff ET. Non-motor outcomes depend on location of neurostimulation in Parkinson's disease [Internet]. Brain. 2019 ; 142 3592-3604.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1093/brain/awz285
Vancouver
Petry-schmelzer JN, Krause M, Dembek TA, Horn A, Evans J, Ashkan K, Rizos A, Silverdale M, Schumacher W, Fonoff ET. Non-motor outcomes depend on location of neurostimulation in Parkinson's disease [Internet]. Brain. 2019 ; 142 3592-3604.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1093/brain/awz285
Artigo de periodico
A novel ATP1A2 mutation in a patient with hypokalaemic periodic paralysis and CNS symptoms (2018)
Castaneda, Marisol Sampedro; Zanoteli, Edmar; Scalco, Renata S.; Scaramuzzi, Vinicius; Caldas, Vitor Marques; Reed, Umbertina Conti; Silva, Andre Macedo Serafim da; O'callaghan, Benjamin; Phadke, Rahul; Bugiardini, Enrico
Source: Brain. Unidade: FM
Subjects: DOENÇAS MUSCULARES, POTÁSSIO, PARESIA, PROTEÍNAS DE TRANSPORTE, MUTAÇÃO GENÉTICA
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ABNT
CASTANEDA, Marisol Sampedro et al. A novel ATP1A2 mutation in a patient with hypokalaemic periodic paralysis and CNS symptoms. Brain, v. 141, p. 3308-3318, 2018Tradução . . Disponível em: https://doi.org/10.1093/brain/awy283. Acesso em: 01 nov. 2024.
APA
Castaneda, M. S., Zanoteli, E., Scalco, R. S., Scaramuzzi, V., Caldas, V. M., Reed, U. C., et al. (2018). A novel ATP1A2 mutation in a patient with hypokalaemic periodic paralysis and CNS symptoms. Brain, 141, 3308-3318. doi:10.1093/brain/awy283
NLM
Castaneda MS, Zanoteli E, Scalco RS, Scaramuzzi V, Caldas VM, Reed UC, Silva AMS da, O'callaghan B, Phadke R, Bugiardini E. A novel ATP1A2 mutation in a patient with hypokalaemic periodic paralysis and CNS symptoms [Internet]. Brain. 2018 ; 141 3308-3318.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1093/brain/awy283
Vancouver
Castaneda MS, Zanoteli E, Scalco RS, Scaramuzzi V, Caldas VM, Reed UC, Silva AMS da, O'callaghan B, Phadke R, Bugiardini E. A novel ATP1A2 mutation in a patient with hypokalaemic periodic paralysis and CNS symptoms [Internet]. Brain. 2018 ; 141 3308-3318.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1093/brain/awy283
Artigo de periodico
Quantifying the accretion of hyperphosphorylated tau in the locus coeruleus and dorsal raphe nucleus: the pathological building blocks of early Alzheimer's disease (2017)
Ehrennberg, A. J; Nguy, A. K; Theofilas, P; Dunlop, S; Suemoto, Claudia Kimie; Alho, A. T Lorenzo; Leite, R. P; Rodriguez, R. Diehl; Mejia, M. B; Rüb, U; Farfel, José Marcelo; Ferretti-Rebustini, Renata Eloah de Lucena ; Nascimento, C. F.; Nitrini, Ricardo; Pasquallucci, Carlos Alberto; Jacob Filho, Wilson; Miller, B; Seeley, W. W; Heinsen, H; Grinberg, Lea Tenenholz
Source: Neuropathology and Applied Neurobiology. Unidades: EE, FM
Subjects: DOENÇA DE ALZHEIMER, CÉREBRO, TÉCNICAS CITOLÓGICAS, NEUROFISIOLOGIA
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ABNT
EHRENNBERG, A. J et al. Quantifying the accretion of hyperphosphorylated tau in the locus coeruleus and dorsal raphe nucleus: the pathological building blocks of early Alzheimer's disease. Neuropathology and Applied Neurobiology, 2017Tradução . . Disponível em: https://doi.org/10.1111/nan.12387. Acesso em: 01 nov. 2024.
APA
Ehrennberg, A. J., Nguy, A. K., Theofilas, P., Dunlop, S., Suemoto, C. K., Alho, A. T. L., et al. (2017). Quantifying the accretion of hyperphosphorylated tau in the locus coeruleus and dorsal raphe nucleus: the pathological building blocks of early Alzheimer's disease. Neuropathology and Applied Neurobiology. doi:10.1111/nan.12387
NLM
Ehrennberg AJ, Nguy AK, Theofilas P, Dunlop S, Suemoto CK, Alho ATL, Leite RP, Rodriguez RD, Mejia MB, Rüb U, Farfel JM, Ferretti-Rebustini RE de L, Nascimento CF, Nitrini R, Pasquallucci CA, Jacob Filho W, Miller B, Seeley WW, Heinsen H, Grinberg LT. Quantifying the accretion of hyperphosphorylated tau in the locus coeruleus and dorsal raphe nucleus: the pathological building blocks of early Alzheimer's disease [Internet]. Neuropathology and Applied Neurobiology. 2017 ;[citado 2024 nov. 01 ] Available from: https://doi.org/10.1111/nan.12387
Vancouver
Ehrennberg AJ, Nguy AK, Theofilas P, Dunlop S, Suemoto CK, Alho ATL, Leite RP, Rodriguez RD, Mejia MB, Rüb U, Farfel JM, Ferretti-Rebustini RE de L, Nascimento CF, Nitrini R, Pasquallucci CA, Jacob Filho W, Miller B, Seeley WW, Heinsen H, Grinberg LT. Quantifying the accretion of hyperphosphorylated tau in the locus coeruleus and dorsal raphe nucleus: the pathological building blocks of early Alzheimer's disease [Internet]. Neuropathology and Applied Neurobiology. 2017 ;[citado 2024 nov. 01 ] Available from: https://doi.org/10.1111/nan.12387
Artigo de periodico
Collybistin binds and inhibits mTORC1 signaling: a potential novel mechanism contributing to intellectual disability and autism (2016)
Machado, Camila Oliveira Freitas; Griesi-Oliveira, Karina; Rosenberg, Carla ; Kok, Fernando; Martins, Stephanie; Passos-Bueno, Maria Rita ; Sertie, Andrea Laurato
Source: European Journal of Human Genetics. Unidades: FM, IB
Subjects: SÍNDROME X METABÓLICA, FIBROBLASTOS, TÉCNICAS CITOLÓGICAS
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ABNT
MACHADO, Camila Oliveira Freitas et al. Collybistin binds and inhibits mTORC1 signaling: a potential novel mechanism contributing to intellectual disability and autism. European Journal of Human Genetics, v. 24, n. 1, p. 59-65, 2016Tradução . . Disponível em: https://doi.org/10.1038/ejhg.2015.69. Acesso em: 01 nov. 2024.
APA
Machado, C. O. F., Griesi-Oliveira, K., Rosenberg, C., Kok, F., Martins, S., Passos-Bueno, M. R., & Sertie, A. L. (2016). Collybistin binds and inhibits mTORC1 signaling: a potential novel mechanism contributing to intellectual disability and autism. European Journal of Human Genetics, 24( 1), 59-65. doi:10.1038/ejhg.2015.69
NLM
Machado COF, Griesi-Oliveira K, Rosenberg C, Kok F, Martins S, Passos-Bueno MR, Sertie AL. Collybistin binds and inhibits mTORC1 signaling: a potential novel mechanism contributing to intellectual disability and autism [Internet]. European Journal of Human Genetics. 2016 ; 24( 1): 59-65.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/ejhg.2015.69
Vancouver
Machado COF, Griesi-Oliveira K, Rosenberg C, Kok F, Martins S, Passos-Bueno MR, Sertie AL. Collybistin binds and inhibits mTORC1 signaling: a potential novel mechanism contributing to intellectual disability and autism [Internet]. European Journal of Human Genetics. 2016 ; 24( 1): 59-65.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/ejhg.2015.69
Artigo de periodico
Quantitative proteomic analysis shows differentially expressed HSPB1 in glioblastoma as a discriminating short from long survival factor and NOVA1 as a differentiation factor between low-grade astrocytoma and oligodendroglioma (2015)
Gimenez, Marcela; Marie, Suely Kazue; Oba-Shinjo, Sueli; Uno, Miyuki; Izumi, Clarice; Oliveira, João Bosco; Rosa, Jose Cesar
Source: BMC Cancer. Unidades: FM, FMRP
Subjects: GLIOMA, NEOPLASIAS CEREBRAIS, SISTEMA NERVOSO CENTRAL, PROTEÍNAS (ESTUDO)
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ABNT
GIMENEZ, Marcela et al. Quantitative proteomic analysis shows differentially expressed HSPB1 in glioblastoma as a discriminating short from long survival factor and NOVA1 as a differentiation factor between low-grade astrocytoma and oligodendroglioma. BMC Cancer, v. 15, n. 481, p. (online), 2015Tradução . . Disponível em: https://doi.org/10.1186/s12885-015-1473-9. Acesso em: 01 nov. 2024.
APA
Gimenez, M., Marie, S. K., Oba-Shinjo, S., Uno, M., Izumi, C., Oliveira, J. B., & Rosa, J. C. (2015). Quantitative proteomic analysis shows differentially expressed HSPB1 in glioblastoma as a discriminating short from long survival factor and NOVA1 as a differentiation factor between low-grade astrocytoma and oligodendroglioma. BMC Cancer, 15( 481), (online). doi:10.1186/s12885-015-1473-9
NLM
Gimenez M, Marie SK, Oba-Shinjo S, Uno M, Izumi C, Oliveira JB, Rosa JC. Quantitative proteomic analysis shows differentially expressed HSPB1 in glioblastoma as a discriminating short from long survival factor and NOVA1 as a differentiation factor between low-grade astrocytoma and oligodendroglioma [Internet]. BMC Cancer. 2015 ; 15( 481): (online).[citado 2024 nov. 01 ] Available from: https://doi.org/10.1186/s12885-015-1473-9
Vancouver
Gimenez M, Marie SK, Oba-Shinjo S, Uno M, Izumi C, Oliveira JB, Rosa JC. Quantitative proteomic analysis shows differentially expressed HSPB1 in glioblastoma as a discriminating short from long survival factor and NOVA1 as a differentiation factor between low-grade astrocytoma and oligodendroglioma [Internet]. BMC Cancer. 2015 ; 15( 481): (online).[citado 2024 nov. 01 ] Available from: https://doi.org/10.1186/s12885-015-1473-9

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