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Artigo de periodico
Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy (2022)
Silva, Maria Cláudia da; Fuzo, Carlos Alessandro ; Paiva, Isadora Marques ; Bibo, Naira Lopes; Oliveira, Maykon Tavares de; Soares, Hellen Anastácia da Silva; Chevillard, Christophe; Kalil Filho, Jorge Elias ; Cunha Neto, Edecio ; Cunha, Thiago Mattar ; Silva, João Santana da
Source: Immunobiology. Unidades: FM, FMRP, FCFRP
Subjects: MUTAÇÃO, DOENÇA DE CHAGAS, POLIMORFISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Maria Cláudia da et al. Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy. Immunobiology, v. 227, n. 5, p. 1-7, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2022.152242. Acesso em: 16 nov. 2024.
APA
Silva, M. C. da, Fuzo, C. A., Paiva, I. M., Bibo, N. L., Oliveira, M. T. de, Soares, H. A. da S., et al. (2022). Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy. Immunobiology, 227( 5), 1-7. doi:10.1016/j.imbio.2022.152242
NLM
Silva MC da, Fuzo CA, Paiva IM, Bibo NL, Oliveira MT de, Soares HA da S, Chevillard C, Kalil Filho JE, Cunha Neto E, Cunha TM, Silva JS da. Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy [Internet]. Immunobiology. 2022 ; 227( 5): 1-7.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2022.152242
Vancouver
Silva MC da, Fuzo CA, Paiva IM, Bibo NL, Oliveira MT de, Soares HA da S, Chevillard C, Kalil Filho JE, Cunha Neto E, Cunha TM, Silva JS da. Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy [Internet]. Immunobiology. 2022 ; 227( 5): 1-7.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.imbio.2022.152242
Artigo de periodico
Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model (2019)
Pérez, Malena Martínez ; Martins, Larissa Marfori Sampaio; Dias, Murilo Solano; Pereira, Camila André; Leite, Jefferson Antonio; Gonçalves, Enrico Cerioni Spiropulos; Almeida, Paula Zaghetto de ; Freitas, Emanuelle Neivert de; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zoete, Marcel R. de; Ryffel, Bernhard; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Immunology. Unidade: FMRP
Subjects: DOENÇAS METABÓLICAS, INFLAMAÇÃO, OBESIDADE, IMUNOLOGIA, HOMEOSTASE, INTESTINOS
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ABNT
PÉREZ, Malena Martínez et al. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model. Immunology, v. 156, n. 4, p. 339-355, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13028. Acesso em: 16 nov. 2024.
APA
Pérez, M. M., Martins, L. M. S., Dias, M. S., Pereira, C. A., Leite, J. A., Gonçalves, E. C. S., et al. (2019). Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model. Immunology, 156( 4), 339-355. doi:10.1111/imm.13028
NLM
Pérez MM, Martins LMS, Dias MS, Pereira CA, Leite JA, Gonçalves ECS, Almeida PZ de, Freitas EN de, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Sartori DC. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model [Internet]. Immunology. 2019 ; 156( 4): 339-355.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/imm.13028
Vancouver
Pérez MM, Martins LMS, Dias MS, Pereira CA, Leite JA, Gonçalves ECS, Almeida PZ de, Freitas EN de, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Sartori DC. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model [Internet]. Immunology. 2019 ; 156( 4): 339-355.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/imm.13028
Artigo de periodico
The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression (2018)
Amano, Mariane Tami ; Castoldi, Angela ; Andrade-Oliveira, Vinicius ; Latância, Marcela Teatin ; Terra, Fernanda Fernandes ; Correa-Costa, Matheus ; Breda, Cristiane Naffah Souza ; Felizardo, Raphael José Ferreira ; Pereira, Welbert Oliveira de ; Silva, Marina Burgos da ; Miyagi, Mariana Yasue Saito ; Aguiar, Cristhiane Favero ; Hiyane, Meire Ioshie ; Silva, João Santana da ; Moura, Ivan Cruz ; Câmara, Niels Olsen Saraiva
Source: International Immunopharmacology. Unidades: ICB, FMRP, FCF, BIOTECNOLOGIA
Subjects: INSUFICIÊNCIA RENAL AGUDA, NEFROPATIAS, MACRÓFAGOS, IMUNOLOGIA
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ABNT
AMANO, Mariane Tami et al. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, v. 64, p. 151-161, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.08.020. Acesso em: 16 nov. 2024.
APA
Amano, M. T., Castoldi, A., Andrade-Oliveira, V., Latância, M. T., Terra, F. F., Correa-Costa, M., et al. (2018). The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, 64, 151-161. doi:10.1016/j.intimp.2018.08.020
NLM
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Vancouver
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Artigo de periodico
T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses (2018)
Reynolds, C. J.; Suleyman, O. M.; Ortega-Prieto, A. M.; Skelton, J. K.; Bonnesoeur, P.; Blohm, A.; Carregaro, Vanessa ; Silva, João Santana da ; James, E. A.; Maillère, B.; Dorner, M.; Boyton, R. J.; Altmann, D. M.
Source: Scientific Reports. Unidade: FMRP
Subjects: ZIKA VÍRUS, GENES DE RESPOSTA IMUNE, LINFÓCITOS, REAÇÃO CRUZADA
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ABNT
REYNOLDS, C. J. et al. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses. Scientific Reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-18781-1. Acesso em: 16 nov. 2024.
APA
Reynolds, C. J., Suleyman, O. M., Ortega-Prieto, A. M., Skelton, J. K., Bonnesoeur, P., Blohm, A., et al. (2018). T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses. Scientific Reports, 8. doi:10.1038/s41598-017-18781-1
NLM
Reynolds CJ, Suleyman OM, Ortega-Prieto AM, Skelton JK, Bonnesoeur P, Blohm A, Carregaro V, Silva JS da, James EA, Maillère B, Dorner M, Boyton RJ, Altmann DM. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41598-017-18781-1
Vancouver
Reynolds CJ, Suleyman OM, Ortega-Prieto AM, Skelton JK, Bonnesoeur P, Blohm A, Carregaro V, Silva JS da, James EA, Maillère B, Dorner M, Boyton RJ, Altmann DM. T cell immunity to Zika virus targets immunodominant epitopes that show cross-reactivity with other Flaviviruses [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41598-017-18781-1
Artigo de periodico
Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis (2018)
Silva, Maria C; Ferreira, Marcela Davoli; Medina, Tiago S; Costa, Renata Sesti; Silva, Grace K; Lopes, Carla D; Cardozo, Lucas Esteves; Gava, Fábio N; Lyroni, Konstantina; Dias, Fabricio C; Frade, Amanda F; Baron, Monique; Nakaya, Helder Takashi Imoto ; Figueiredo, Florêncio; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Tsatsanis, Christos; Chevillard, Christophe; Cunha-Neto, Edécio; Hirsch, Emilio; Silva, João Santana da; Cunha, Thiago Mattar
Source: Nature Communications. Unidades: FCF, FMRP, FM
Subjects: TRYPANOSOMA CRUZI, MIOCARDIOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Maria C et al. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. Nature Communications, v. 9, p. 1-14 art. 1513, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41467-018-03986-3. Acesso em: 16 nov. 2024.
APA
Silva, M. C., Ferreira, M. D., Medina, T. S., Costa, R. S., Silva, G. K., Lopes, C. D., et al. (2018). Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. Nature Communications, 9, 1-14 art. 1513. doi:10.1038/s41467-018-03986-3
NLM
Silva MC, Ferreira MD, Medina TS, Costa RS, Silva GK, Lopes CD, Cardozo LE, Gava FN, Lyroni K, Dias FC, Frade AF, Baron M, Nakaya HTI, Figueiredo F, Alves Filho JCF, Cunha F de Q, Tsatsanis C, Chevillard C, Cunha-Neto E, Hirsch E, Silva JS da, Cunha TM. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis [Internet]. Nature Communications. 2018 ; 9 1-14 art. 1513.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41467-018-03986-3
Vancouver
Silva MC, Ferreira MD, Medina TS, Costa RS, Silva GK, Lopes CD, Cardozo LE, Gava FN, Lyroni K, Dias FC, Frade AF, Baron M, Nakaya HTI, Figueiredo F, Alves Filho JCF, Cunha F de Q, Tsatsanis C, Chevillard C, Cunha-Neto E, Hirsch E, Silva JS da, Cunha TM. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis [Internet]. Nature Communications. 2018 ; 9 1-14 art. 1513.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41467-018-03986-3
Artigo de periodico
Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model (2017)
Carlos, Daniela ; Costa, Frederico Ribeiro Campos ; Pereira, Camila André; Rocha, Fernanda Agostini; Yaochite, Juliana Navarro Ueda; Oliveira, Gabriela G.; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Câmara, Niels Olsen Saraiva ; Ryffel, Bernhard; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB
Subjects: CITOCINAS, DIABETES MELLITUS, DNA MITOCONDRIAL
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ABNT
CARLOS, Daniela et al. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00164. Acesso em: 16 nov. 2024.
APA
Carlos, D., Costa, F. R. C., Pereira, C. A., Rocha, F. A., Yaochite, J. N. U., Oliveira, G. G., et al. (2017). Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00164
NLM
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Vancouver
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 16 ] Available from: https://doi.org/10.3389/fimmu.2017.00164

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