Source: Molecular Biology Reports. Unidade: FO
Subjects: ODONTOLOGIA, PATOLOGIA BUCAL, TABAGISMO, CARCINOMA, EXPRESSÃO GÊNICA, NEOVASCULARIZAÇÃO PATOLÓGICA, METÁSTASE NEOPLÁSICA
ABNT
SANTOS, Joaquim Gasparini dos et al. Nicotine exposure increases PIK3CA, AKT1, HIF-1α, GLUT1, CA9 and VEGF expression in oral potentially malignant lesions. Molecular Biology Reports, n. Ja 2024, 2024Tradução . . Acesso em: 12 nov. 2024.APA
Santos, J. G. dos, Oliveira, M. M., Otuyama, L. J., Mendes, S. O., Borçoi , A. R., Moreno, I. A. A., et al. (2024). Nicotine exposure increases PIK3CA, AKT1, HIF-1α, GLUT1, CA9 and VEGF expression in oral potentially malignant lesions. Molecular Biology Reports, ( Ja 2024). doi:10.21203/rs.3.rs-3832999/v1NLM
Santos JG dos, Oliveira MM, Otuyama LJ, Mendes SO, Borçoi AR, Moreno IAA, Peterle GT, Maia LL, Paris AFCS, Archanjo AB, Nunes FD, Silva AMA. Nicotine exposure increases PIK3CA, AKT1, HIF-1α, GLUT1, CA9 and VEGF expression in oral potentially malignant lesions. Molecular Biology Reports. 2024 ;( Ja 2024):[citado 2024 nov. 12 ]Vancouver
Santos JG dos, Oliveira MM, Otuyama LJ, Mendes SO, Borçoi AR, Moreno IAA, Peterle GT, Maia LL, Paris AFCS, Archanjo AB, Nunes FD, Silva AMA. Nicotine exposure increases PIK3CA, AKT1, HIF-1α, GLUT1, CA9 and VEGF expression in oral potentially malignant lesions. Molecular Biology Reports. 2024 ;( Ja 2024):[citado 2024 nov. 12 ]