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Artigo de periodico
Hyperactivation of sympathetic nerves drives depletion of melanocyte stem cells (2020)
Zhang, Bing; Ma, Sai; Rachmin, Inbal; He, Megan; Baral, Pankaj; Choi, Sekyu; Gonçalves, William A.; Shwartz, Yulia; Fast, Eva M.; Su, Yiqun; Zon, Leonard I.; Regev, Aviv; Buenrostro, Jason D.; Cunha, Thiago Mattar; Chiu, Isaac M.; Fisher, David E.; Hsu, Ya-Chieh
Source: Nature. Unidade: FMRP
Subjects: GLÂNDULAS ADRENAL ANIMAL, ADRENALECTOMIA ANIMAL, PROLIFERAÇÃO CELULAR, CÉLULAS CULTIVADAS, DENERVAÇÃO, COR, CABELO, NOREPINEFRINA, CÉLULAS-TRONCO, ESTRESSE PSICOLÓGICO, SISTEMA NERVOSO SIMPÁTICO
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ABNT
ZHANG, Bing et al. Hyperactivation of sympathetic nerves drives depletion of melanocyte stem cells. Nature, v. 577, n. 7792, p. 676-681, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41586-020-1935-3. Acesso em: 14 nov. 2024.
APA
Zhang, B., Ma, S., Rachmin, I., He, M., Baral, P., Choi, S., et al. (2020). Hyperactivation of sympathetic nerves drives depletion of melanocyte stem cells. Nature, 577( 7792), 676-681. doi:10.1038/s41586-020-1935-3
NLM
Zhang B, Ma S, Rachmin I, He M, Baral P, Choi S, Gonçalves WA, Shwartz Y, Fast EM, Su Y, Zon LI, Regev A, Buenrostro JD, Cunha TM, Chiu IM, Fisher DE, Hsu Y-C. Hyperactivation of sympathetic nerves drives depletion of melanocyte stem cells [Internet]. Nature. 2020 ; 577( 7792): 676-681.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1038/s41586-020-1935-3
Vancouver
Zhang B, Ma S, Rachmin I, He M, Baral P, Choi S, Gonçalves WA, Shwartz Y, Fast EM, Su Y, Zon LI, Regev A, Buenrostro JD, Cunha TM, Chiu IM, Fisher DE, Hsu Y-C. Hyperactivation of sympathetic nerves drives depletion of melanocyte stem cells [Internet]. Nature. 2020 ; 577( 7792): 676-681.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1038/s41586-020-1935-3
Artigo de periodico
Acute intermittent hypoxia in rats activates muscle proteolytic pathways through a glucocorticoid-dependent mechanism (2017)
Przygodda, Franciele; Manfredi, Leandro Henrique; Machado, Juliano; Gonçalves, Dawit A. P.; Zanon, Neusa Maria; Bonagamba, Leni Gomes Heck; Machado, Benedito Honório; Kettelhut, Isis do Carmo; Navegantes, Luiz Carlos Carvalho
Source: Journal of Applied Physiology. Unidade: FMRP
Subjects: ADRENALECTOMIA ANIMAL, ANÓXIA, INSULINA, ATROFIA MUSCULAR, METABOLISMO DE PROTEÍNA
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PRZYGODDA, Franciele et al. Acute intermittent hypoxia in rats activates muscle proteolytic pathways through a glucocorticoid-dependent mechanism. Journal of Applied Physiology, v. 122, n. 5, p. 1114-1124, 2017Tradução . . Disponível em: https://doi.org/10.1152/japplphysiol.00977.2015. Acesso em: 14 nov. 2024.
APA
Przygodda, F., Manfredi, L. H., Machado, J., Gonçalves, D. A. P., Zanon, N. M., Bonagamba, L. G. H., et al. (2017). Acute intermittent hypoxia in rats activates muscle proteolytic pathways through a glucocorticoid-dependent mechanism. Journal of Applied Physiology, 122( 5), 1114-1124. doi:10.1152/japplphysiol.00977.2015
NLM
Przygodda F, Manfredi LH, Machado J, Gonçalves DAP, Zanon NM, Bonagamba LGH, Machado BH, Kettelhut I do C, Navegantes LCC. Acute intermittent hypoxia in rats activates muscle proteolytic pathways through a glucocorticoid-dependent mechanism [Internet]. Journal of Applied Physiology. 2017 ; 122( 5): 1114-1124.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1152/japplphysiol.00977.2015
Vancouver
Przygodda F, Manfredi LH, Machado J, Gonçalves DAP, Zanon NM, Bonagamba LGH, Machado BH, Kettelhut I do C, Navegantes LCC. Acute intermittent hypoxia in rats activates muscle proteolytic pathways through a glucocorticoid-dependent mechanism [Internet]. Journal of Applied Physiology. 2017 ; 122( 5): 1114-1124.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1152/japplphysiol.00977.2015
Trabalho de evento-resumo
Adrenalectomy (ADX) reduces the orexigenic response to ghrelin (2015)
Porceban, Matheus; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: Abstracts. Conference titles: Annual Meeting and Expo of the Endocrine Society (ENDO). Unidade: FMRP
Subjects: GRELINA, ADRENALECTOMIA ANIMAL
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ABNT
PORCEBAN, Matheus e ANTUNES-RODRIGUES, José e ELIAS, Lucila Leico Kagohara. Adrenalectomy (ADX) reduces the orexigenic response to ghrelin. 2015, Anais.. San Diego: Endocrine Society, 2015. Disponível em: https://endo.confex.com/endo/2015endo/webprogram/Paper20704.html. Acesso em: 14 nov. 2024.
APA
Porceban, M., Antunes-Rodrigues, J., & Elias, L. L. K. (2015). Adrenalectomy (ADX) reduces the orexigenic response to ghrelin. In Abstracts. San Diego: Endocrine Society. Recuperado de https://endo.confex.com/endo/2015endo/webprogram/Paper20704.html
NLM
Porceban M, Antunes-Rodrigues J, Elias LLK. Adrenalectomy (ADX) reduces the orexigenic response to ghrelin [Internet]. Abstracts. 2015 ;[citado 2024 nov. 14 ] Available from: https://endo.confex.com/endo/2015endo/webprogram/Paper20704.html
Vancouver
Porceban M, Antunes-Rodrigues J, Elias LLK. Adrenalectomy (ADX) reduces the orexigenic response to ghrelin [Internet]. Abstracts. 2015 ;[citado 2024 nov. 14 ] Available from: https://endo.confex.com/endo/2015endo/webprogram/Paper20704.html
Artigo de periodico
Corticotrophin-releasing factor receptor 2 mediates the enhanced activation of satiety-related responses through oxytocin neurons in the paraventricular nucleus of the hypothalamus after adrenalectomy (2015)
Uchoa, Ernane Torres; Rorato, Rodrigo; Ruginsk, Silvia Graciela; Borges, Beatriz de Carvalho; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: Neuroscience Letters. Unidade: FMRP
Subjects: ADRENALECTOMIA ANIMAL, HORMÔNIO LIBERADOR DE CORTICOTROFINA, NEURÔNIOS, INGESTÃO, ALIMENTOS
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ABNT
UCHOA, Ernane Torres et al. Corticotrophin-releasing factor receptor 2 mediates the enhanced activation of satiety-related responses through oxytocin neurons in the paraventricular nucleus of the hypothalamus after adrenalectomy. Neuroscience Letters, v. 606, p. 123-128, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2015.08.045. Acesso em: 14 nov. 2024.
APA
Uchoa, E. T., Rorato, R., Ruginsk, S. G., Borges, B. de C., Antunes-Rodrigues, J., & Elias, L. L. K. (2015). Corticotrophin-releasing factor receptor 2 mediates the enhanced activation of satiety-related responses through oxytocin neurons in the paraventricular nucleus of the hypothalamus after adrenalectomy. Neuroscience Letters, 606, 123-128. doi:10.1016/j.neulet.2015.08.045
NLM
Uchoa ET, Rorato R, Ruginsk SG, Borges B de C, Antunes-Rodrigues J, Elias LLK. Corticotrophin-releasing factor receptor 2 mediates the enhanced activation of satiety-related responses through oxytocin neurons in the paraventricular nucleus of the hypothalamus after adrenalectomy [Internet]. Neuroscience Letters. 2015 ; 606 123-128.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.neulet.2015.08.045
Vancouver
Uchoa ET, Rorato R, Ruginsk SG, Borges B de C, Antunes-Rodrigues J, Elias LLK. Corticotrophin-releasing factor receptor 2 mediates the enhanced activation of satiety-related responses through oxytocin neurons in the paraventricular nucleus of the hypothalamus after adrenalectomy [Internet]. Neuroscience Letters. 2015 ; 606 123-128.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.neulet.2015.08.045
Artigo de periodico
Differential immunoreactivity of glucocorticoid receptor and GABA in GABAergic afferents to parvocellular neurons in the paraventricular nucleous (2013)
Souza, Leandro Marques de; Franci, Celso Rodrigues
Source: Neuroscience Letters. Unidade: FMRP
Subjects: ADRENALECTOMIA ANIMAL, NEURÔNIOS (EFEITOS DE DROGAS), RECEPTORES DE GLICOCORTICOIDES, GABA
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ABNT
SOUZA, Leandro Marques de e FRANCI, Celso Rodrigues. Differential immunoreactivity of glucocorticoid receptor and GABA in GABAergic afferents to parvocellular neurons in the paraventricular nucleous. Neuroscience Letters, v. 534, p. 199-204, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neulet.2012.12.018. Acesso em: 14 nov. 2024.
APA
Souza, L. M. de, & Franci, C. R. (2013). Differential immunoreactivity of glucocorticoid receptor and GABA in GABAergic afferents to parvocellular neurons in the paraventricular nucleous. Neuroscience Letters, 534, 199-204. doi:10.1016/j.neulet.2012.12.018
NLM
Souza LM de, Franci CR. Differential immunoreactivity of glucocorticoid receptor and GABA in GABAergic afferents to parvocellular neurons in the paraventricular nucleous [Internet]. Neuroscience Letters. 2013 ; 534 199-204.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.neulet.2012.12.018
Vancouver
Souza LM de, Franci CR. Differential immunoreactivity of glucocorticoid receptor and GABA in GABAergic afferents to parvocellular neurons in the paraventricular nucleous [Internet]. Neuroscience Letters. 2013 ; 534 199-204.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.neulet.2012.12.018
Artigo de periodico
Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency (2013)
Uchoa, Ernane Torres; Zahm, Daniel S.; Borges, Beatriz de Carvalho; Rorato, Rodrigo; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: Experimental Physiology. Unidade: FMRP
Subjects: OCITOCINA, HIPOTÁLAMO, ADRENALECTOMIA ANIMAL
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ABNT
UCHOA, Ernane Torres et al. Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency. Experimental Physiology, v. 98, n. 10, p. 1495-1504, 2013Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2013.073726. Acesso em: 14 nov. 2024.
APA
Uchoa, E. T., Zahm, D. S., Borges, B. de C., Rorato, R., Antunes-Rodrigues, J., & Elias, L. L. K. (2013). Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency. Experimental Physiology, 98( 10), 1495-1504. doi:10.1113/expphysiol.2013.073726
NLM
Uchoa ET, Zahm DS, Borges B de C, Rorato R, Antunes-Rodrigues J, Elias LLK. Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency [Internet]. Experimental Physiology. 2013 ; 98( 10): 1495-1504.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1113/expphysiol.2013.073726
Vancouver
Uchoa ET, Zahm DS, Borges B de C, Rorato R, Antunes-Rodrigues J, Elias LLK. Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency [Internet]. Experimental Physiology. 2013 ; 98( 10): 1495-1504.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1113/expphysiol.2013.073726
Artigo de periodico
Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses (2009)
Uchôa, Ernane Torres; Sabino, Henrique Augusto C.; Ruginsk, Silvia Graciela; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: Journal of Applied Physiology. Unidade: FMRP
Subjects: HIPOTÁLAMO, ADRENALECTOMIA ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
UCHÔA, Ernane Torres et al. Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses. Journal of Applied Physiology, v. 106, n. 2, p. 596-604, 2009Tradução . . Disponível em: https://doi.org/10.1152/japplphysiol.90865.2008. Acesso em: 14 nov. 2024.
APA
Uchôa, E. T., Sabino, H. A. C., Ruginsk, S. G., Antunes-Rodrigues, J., & Elias, L. L. K. (2009). Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses. Journal of Applied Physiology, 106( 2), 596-604. doi:10.1152/japplphysiol.90865.2008
NLM
Uchôa ET, Sabino HAC, Ruginsk SG, Antunes-Rodrigues J, Elias LLK. Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses [Internet]. Journal of Applied Physiology. 2009 ; 106( 2): 596-604.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1152/japplphysiol.90865.2008
Vancouver
Uchôa ET, Sabino HAC, Ruginsk SG, Antunes-Rodrigues J, Elias LLK. Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses [Internet]. Journal of Applied Physiology. 2009 ; 106( 2): 596-604.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1152/japplphysiol.90865.2008
Artigo de periodico
Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats (2008)
Germano, C. M. R.; Castro, Margaret de; Rorato, R.; Costa, D. B.; Antunes-Rodrigues, José; Elias, Carol Fuzeti; Elias, Lucila Leico Kagohara
Source: Hormone and Metabolic Research. Unidades: ICB, FMRP
Subjects: ADRENALECTOMIA ANIMAL, FISIOLOGIA
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ABNT
GERMANO, C. M. R. et al. Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats. Hormone and Metabolic Research, v. 40, p. 842-847, 2008Tradução . . Acesso em: 14 nov. 2024.
APA
Germano, C. M. R., Castro, M. de, Rorato, R., Costa, D. B., Antunes-Rodrigues, J., Elias, C. F., & Elias, L. L. K. (2008). Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats. Hormone and Metabolic Research, 40, 842-847.
NLM
Germano CMR, Castro M de, Rorato R, Costa DB, Antunes-Rodrigues J, Elias CF, Elias LLK. Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats. Hormone and Metabolic Research. 2008 ; 40 842-847.[citado 2024 nov. 14 ]
Vancouver
Germano CMR, Castro M de, Rorato R, Costa DB, Antunes-Rodrigues J, Elias CF, Elias LLK. Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats. Hormone and Metabolic Research. 2008 ; 40 842-847.[citado 2024 nov. 14 ]
Artigo de periodico
Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons (2008)
Rorato, R.; Borges, B. C.; Benedetti, M.; Germano, C. M. R.; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: Hormones and Behavior. Unidade: FMRP
Subjects: NEUROENDOCRINOLOGIA, HIPOTÁLAMO, ADRENALECTOMIA ANIMAL
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ABNT
RORATO, R. et al. Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons. Hormones and Behavior, v. 54, n. 1, p. 134-142, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2008.02.011. Acesso em: 14 nov. 2024.
APA
Rorato, R., Borges, B. C., Benedetti, M., Germano, C. M. R., Antunes-Rodrigues, J., & Elias, L. L. K. (2008). Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons. Hormones and Behavior, 54( 1), 134-142. doi:10.1016/j.yhbeh.2008.02.011
NLM
Rorato R, Borges BC, Benedetti M, Germano CMR, Antunes-Rodrigues J, Elias LLK. Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons [Internet]. Hormones and Behavior. 2008 ; 54( 1): 134-142.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.yhbeh.2008.02.011
Vancouver
Rorato R, Borges BC, Benedetti M, Germano CMR, Antunes-Rodrigues J, Elias LLK. Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons [Internet]. Hormones and Behavior. 2008 ; 54( 1): 134-142.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.yhbeh.2008.02.011
Artigo de periodico
The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat (2007)
Laguna-Abreu, M. T. C.; Margatho, L.; Germano, C. M. R.; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara; Castro, Margaret de
Source: Stress. Unidade: FMRP
Subjects: VASOPRESSINAS, ADRENALECTOMIA ANIMAL
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ABNT
LAGUNA-ABREU, M. T. C. et al. The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat. Stress, v. 10, n. 4, p. 332-341, 2007Tradução . . Disponível em: https://doi.org/10.1080/10253890701287614. Acesso em: 14 nov. 2024.
APA
Laguna-Abreu, M. T. C., Margatho, L., Germano, C. M. R., Antunes-Rodrigues, J., Elias, L. L. K., & Castro, M. de. (2007). The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat. Stress, 10( 4), 332-341. doi:10.1080/10253890701287614
NLM
Laguna-Abreu MTC, Margatho L, Germano CMR, Antunes-Rodrigues J, Elias LLK, Castro M de. The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat [Internet]. Stress. 2007 ; 10( 4): 332-341.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1080/10253890701287614
Vancouver
Laguna-Abreu MTC, Margatho L, Germano CMR, Antunes-Rodrigues J, Elias LLK, Castro M de. The effect of adrenalectomy on Fos expression in vasopressinergic and oxytocinergic neurons in response to stress in the rat [Internet]. Stress. 2007 ; 10( 4): 332-341.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1080/10253890701287614
Artigo de periodico
Efeito da adrenalectomia sobre a sensibilidade à epilepsia em ratos wistar (2003)
Rodrigues, Marcelo; Roncy-Pereira, R. N.; Garcia-Cairasco, Norberto
Source: Medicina. Conference titles: Prêmio Rocha Lima da FMRP-USP. Unidade: FMRP
Subjects: ADRENALECTOMIA ANIMAL, RATOS, EPILEPSIA
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ABNT
RODRIGUES, Marcelo e RONCY-PEREIRA, R. N. e GARCIA-CAIRASCO, Norberto. Efeito da adrenalectomia sobre a sensibilidade à epilepsia em ratos wistar. Medicina, v. 36, p. 18, 2003Tradução . . Acesso em: 14 nov. 2024.
APA
Rodrigues, M., Roncy-Pereira, R. N., & Garcia-Cairasco, N. (2003). Efeito da adrenalectomia sobre a sensibilidade à epilepsia em ratos wistar. Medicina, 36, 18.
NLM
Rodrigues M, Roncy-Pereira RN, Garcia-Cairasco N. Efeito da adrenalectomia sobre a sensibilidade à epilepsia em ratos wistar. Medicina. 2003 ; 36 18.[citado 2024 nov. 14 ]
Vancouver
Rodrigues M, Roncy-Pereira RN, Garcia-Cairasco N. Efeito da adrenalectomia sobre a sensibilidade à epilepsia em ratos wistar. Medicina. 2003 ; 36 18.[citado 2024 nov. 14 ]

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