Filtros : "Indexado na Base de Dados SCOPUS" "FISIOLOGIA" Limpar

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  • Source: Hormone Molecular Biology and Clinical Investigation. Unidade: ICB

    Assunto: FISIOLOGIA

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      ÉRIQUE CASTRO, e SILVA, Tiago E. Oliveira e FESTUCCIA, William Tadeu Lara. Critical review of beige adipocyte thermogenic activation and contributionto whole-body energy expenditure. Hormone Molecular Biology and Clinical Investigation, v. 31, n. 2, p. 1-9, 2017Tradução . . Disponível em: https://doi.org/10.1515/hmbci-2017-0042. Acesso em: 22 dez. 2024.
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      Érique Castro,, Silva, T. E. O., & Festuccia, W. T. L. (2017). Critical review of beige adipocyte thermogenic activation and contributionto whole-body energy expenditure. Hormone Molecular Biology and Clinical Investigation, 31( 2), 1-9. doi:10.1515/hmbci-2017-0042
    • NLM

      Érique Castro, Silva TEO, Festuccia WTL. Critical review of beige adipocyte thermogenic activation and contributionto whole-body energy expenditure [Internet]. Hormone Molecular Biology and Clinical Investigation. 2017 ; 31( 2): 1-9.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1515/hmbci-2017-0042
    • Vancouver

      Érique Castro, Silva TEO, Festuccia WTL. Critical review of beige adipocyte thermogenic activation and contributionto whole-body energy expenditure [Internet]. Hormone Molecular Biology and Clinical Investigation. 2017 ; 31( 2): 1-9.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1515/hmbci-2017-0042
  • Source: eLife. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      HAWKINS, Virginia E. et al. Purinergic regulation of vascular tone in the retrotrapezoid nucleus is specialized to support the drive to breathe. eLife, v. 6, p. 1-16, 2017Tradução . . Disponível em: https://doi.org/10.7554/eLife.25232. Acesso em: 22 dez. 2024.
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      Hawkins, V. E., Takakura, A. C., Trinh, A., Malheiros-Lima, M. R., Cleary, C. M., Wenker, I. C., et al. (2017). Purinergic regulation of vascular tone in the retrotrapezoid nucleus is specialized to support the drive to breathe. eLife, 6, 1-16. doi:10.7554/eLife.25232
    • NLM

      Hawkins VE, Takakura AC, Trinh A, Malheiros-Lima MR, Cleary CM, Wenker IC, Dubreuil T, Rodriguez EM, Nelson MT, Moreira T dos S, Mulkey DK. Purinergic regulation of vascular tone in the retrotrapezoid nucleus is specialized to support the drive to breathe [Internet]. eLife. 2017 ; 6 1-16.[citado 2024 dez. 22 ] Available from: https://doi.org/10.7554/eLife.25232
    • Vancouver

      Hawkins VE, Takakura AC, Trinh A, Malheiros-Lima MR, Cleary CM, Wenker IC, Dubreuil T, Rodriguez EM, Nelson MT, Moreira T dos S, Mulkey DK. Purinergic regulation of vascular tone in the retrotrapezoid nucleus is specialized to support the drive to breathe [Internet]. eLife. 2017 ; 6 1-16.[citado 2024 dez. 22 ] Available from: https://doi.org/10.7554/eLife.25232
  • Source: The Journal of Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      ANTUNES, Vagner Roberto. Hypertonic NaCl versus osmotic stimuli: distinct OVLT neurones can sense the difference to control sympathetic outflow and blood pressure. The Journal of Physiology, v. 595, n. 18, p. 6089-6090, 2017Tradução . . Disponível em: https://doi.org/10.1113/JP274868. Acesso em: 22 dez. 2024.
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      Antunes, V. R. (2017). Hypertonic NaCl versus osmotic stimuli: distinct OVLT neurones can sense the difference to control sympathetic outflow and blood pressure. The Journal of Physiology, 595( 18), 6089-6090. doi:10.1113/JP274868
    • NLM

      Antunes VR. Hypertonic NaCl versus osmotic stimuli: distinct OVLT neurones can sense the difference to control sympathetic outflow and blood pressure [Internet]. The Journal of Physiology. 2017 ; 595( 18): 6089-6090.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1113/JP274868
    • Vancouver

      Antunes VR. Hypertonic NaCl versus osmotic stimuli: distinct OVLT neurones can sense the difference to control sympathetic outflow and blood pressure [Internet]. The Journal of Physiology. 2017 ; 595( 18): 6089-6090.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1113/JP274868
  • Source: Cell. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      HAN, Wenfei et al. Integrated control of predatory hunting by the central nucleus of the amygdala. Cell, v. 168, n. 1-2, p. 311-324, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.cell.2016.12.027. Acesso em: 22 dez. 2024.
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      Han, W., Tellez, L. A., Rangel Jr., M. J., Motta, S. C., Zhang, X., Perez, I. O., et al. (2017). Integrated control of predatory hunting by the central nucleus of the amygdala. Cell, 168( 1-2), 311-324. doi:10.1016/j.cell.2016.12.027
    • NLM

      Han W, Tellez LA, Rangel Jr. MJ, Motta SC, Zhang X, Perez IO, Canteras NS, Shammah-Lagnado SJ, Pol AN van den, Araujo IE de. Integrated control of predatory hunting by the central nucleus of the amygdala [Internet]. Cell. 2017 ; 168( 1-2): 311-324.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.cell.2016.12.027
    • Vancouver

      Han W, Tellez LA, Rangel Jr. MJ, Motta SC, Zhang X, Perez IO, Canteras NS, Shammah-Lagnado SJ, Pol AN van den, Araujo IE de. Integrated control of predatory hunting by the central nucleus of the amygdala [Internet]. Cell. 2017 ; 168( 1-2): 311-324.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.cell.2016.12.027
  • Source: Journal of Diabetes Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      ESTEVES, João Victor e ENGUITA, Francisco Javier e MACHADO, Ubiratan Fabres. MicroRNAs-mediated regulation of skeletal muscle GLUT4 expressionand translocation in insulin resistance. Journal of Diabetes Research, v. 2017, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.1155/2017/7267910. Acesso em: 22 dez. 2024.
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      Esteves, J. V., Enguita, F. J., & Machado, U. F. (2017). MicroRNAs-mediated regulation of skeletal muscle GLUT4 expressionand translocation in insulin resistance. Journal of Diabetes Research, 2017, 1-11. doi:10.1155/2017/7267910
    • NLM

      Esteves JV, Enguita FJ, Machado UF. MicroRNAs-mediated regulation of skeletal muscle GLUT4 expressionand translocation in insulin resistance [Internet]. Journal of Diabetes Research. 2017 ; 2017 1-11.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1155/2017/7267910
    • Vancouver

      Esteves JV, Enguita FJ, Machado UF. MicroRNAs-mediated regulation of skeletal muscle GLUT4 expressionand translocation in insulin resistance [Internet]. Journal of Diabetes Research. 2017 ; 2017 1-11.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1155/2017/7267910
  • Source: Epilepsy & Behavior. Unidade: ICB

    Assunto: FISIOLOGIA

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      ROCHA, Anna Karynna Alves de Alencar et al. Altered MT1 and MT2 melatonin receptors expression in the hippocampus of pilocarpine-induced epileptic rats. Epilepsy & Behavior, v. 71, p. 23-24, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.yebeh.2017.01.020. Acesso em: 22 dez. 2024.
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      Rocha, A. K. A. de A., Lima, E. de, Amaral, F., Peres, R., Cipolla-Neto, J., & Amado, D. (2017). Altered MT1 and MT2 melatonin receptors expression in the hippocampus of pilocarpine-induced epileptic rats. Epilepsy & Behavior, 71, 23-24. doi:10.1016/j.yebeh.2017.01.020
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      Rocha AKA de A, Lima E de, Amaral F, Peres R, Cipolla-Neto J, Amado D. Altered MT1 and MT2 melatonin receptors expression in the hippocampus of pilocarpine-induced epileptic rats [Internet]. Epilepsy & Behavior. 2017 ; 71 23-24.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.yebeh.2017.01.020
    • Vancouver

      Rocha AKA de A, Lima E de, Amaral F, Peres R, Cipolla-Neto J, Amado D. Altered MT1 and MT2 melatonin receptors expression in the hippocampus of pilocarpine-induced epileptic rats [Internet]. Epilepsy & Behavior. 2017 ; 71 23-24.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.yebeh.2017.01.020
  • Source: Nutrition & Metabolism. Unidade: ICB

    Assunto: FISIOLOGIA

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      CAMINHOTTO, Rennan de Oliveira et al. Oral β-hydroxybutyrate increases ketonemia, decreases visceral adipocyte volume and improves serum lipid profile in Wistar rats. Nutrition & Metabolism, v. 14, n. 31, p. 1-9, 2017Tradução . . Disponível em: https://doi.org/10.1186/s12986-017-0184-4. Acesso em: 22 dez. 2024.
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      Caminhotto, R. de O., Komino, A. C. M., Silva, F. de F., Andreotti, S., Sertié, R. A. L., Reis, G. B., & Lima, F. B. (2017). Oral β-hydroxybutyrate increases ketonemia, decreases visceral adipocyte volume and improves serum lipid profile in Wistar rats. Nutrition & Metabolism, 14( 31), 1-9. doi:10.1186/s12986-017-0184-4
    • NLM

      Caminhotto R de O, Komino ACM, Silva F de F, Andreotti S, Sertié RAL, Reis GB, Lima FB. Oral β-hydroxybutyrate increases ketonemia, decreases visceral adipocyte volume and improves serum lipid profile in Wistar rats [Internet]. Nutrition & Metabolism. 2017 ; 14( 31): 1-9.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1186/s12986-017-0184-4
    • Vancouver

      Caminhotto R de O, Komino ACM, Silva F de F, Andreotti S, Sertié RAL, Reis GB, Lima FB. Oral β-hydroxybutyrate increases ketonemia, decreases visceral adipocyte volume and improves serum lipid profile in Wistar rats [Internet]. Nutrition & Metabolism. 2017 ; 14( 31): 1-9.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1186/s12986-017-0184-4
  • Source: Molecular and Cellular Biochemistry. Unidade: ICB

    Assunto: FISIOLOGIA

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      GERLINGER-ROMERO, Frederico et al. Dysregulation between TRIM63/FBXO32 expression and soleus muscle wasting in diabetic rats: potential role of miR-1-3p, -29a/b-3p, and -133a/b-3p. Molecular and Cellular Biochemistry, v. 427, n. 1-2, p. 187-199, 2017Tradução . . Disponível em: https://doi.org/10.1007/s11010-016-2910-z. Acesso em: 22 dez. 2024.
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      Gerlinger-Romero, F., Yonamine, C. Y., Pinto Junior, D. C., Esteves, J. V. D. C., & Machado, U. F. (2017). Dysregulation between TRIM63/FBXO32 expression and soleus muscle wasting in diabetic rats: potential role of miR-1-3p, -29a/b-3p, and -133a/b-3p. Molecular and Cellular Biochemistry, 427( 1-2), 187-199. doi:10.1007/s11010-016-2910-z
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      Gerlinger-Romero F, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF. Dysregulation between TRIM63/FBXO32 expression and soleus muscle wasting in diabetic rats: potential role of miR-1-3p, -29a/b-3p, and -133a/b-3p [Internet]. Molecular and Cellular Biochemistry. 2017 ; 427( 1-2): 187-199.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1007/s11010-016-2910-z
    • Vancouver

      Gerlinger-Romero F, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF. Dysregulation between TRIM63/FBXO32 expression and soleus muscle wasting in diabetic rats: potential role of miR-1-3p, -29a/b-3p, and -133a/b-3p [Internet]. Molecular and Cellular Biochemistry. 2017 ; 427( 1-2): 187-199.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1007/s11010-016-2910-z
  • Source: The FASEB journal. Unidades: ICB, BIOINFORMÁTICA

    Subjects: FISIOLOGIA, HISTOLOGIA

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      HENRIQUES, Felipe Santos et al. Early suppression of adipocyte lipid turnover induces immunometabolic modulation in cancer cachexia syndrome. The FASEB journal, v. 31, n. 5, p. 1976-1986, 2017Tradução . . Disponível em: https://doi.org/10.1096/fj.201601151R. Acesso em: 22 dez. 2024.
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      Henriques, F. S., Sertié, R. A. L., Franco, F. de O., Knobl, P., Neves, R. X., Andreotti, S., et al. (2017). Early suppression of adipocyte lipid turnover induces immunometabolic modulation in cancer cachexia syndrome. The FASEB journal, 31( 5), 1976-1986. doi:10.1096/fj.201601151R
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      Henriques FS, Sertié RAL, Franco F de O, Knobl P, Neves RX, Andreotti S, Lima FB, Guilherme A, Seelaender M, Batista Jr. ML. Early suppression of adipocyte lipid turnover induces immunometabolic modulation in cancer cachexia syndrome [Internet]. The FASEB journal. 2017 ; 31( 5): 1976-1986.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1096/fj.201601151R
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      Henriques FS, Sertié RAL, Franco F de O, Knobl P, Neves RX, Andreotti S, Lima FB, Guilherme A, Seelaender M, Batista Jr. ML. Early suppression of adipocyte lipid turnover induces immunometabolic modulation in cancer cachexia syndrome [Internet]. The FASEB journal. 2017 ; 31( 5): 1976-1986.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1096/fj.201601151R
  • Source: Biomed Research International. Unidade: ICB

    Subjects: ANATOMIA, BIOFÍSICA, FISIOLOGIA, SISTEMA MUSCULOSQUELÉTICO, INSULINA, CARBOIDRATOS, CAMUNDONGOS

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      CHRISTOFFOLETE, Marcelo Augusto et al. Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate. Biomed Research International, v. 2015, p. 1-8, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/282984. Acesso em: 22 dez. 2024.
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      Christoffolete, M. A., Silva, W. J., Ramos, G. V., Bento, M. R., Costa, M. O., Ribeiro, M. O., et al. (2015). Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate. Biomed Research International, 2015, 1-8. doi:10.1155/2015/282984
    • NLM

      Christoffolete MA, Silva WJ, Ramos GV, Bento MR, Costa MO, Ribeiro MO, Okamoto MM, Tania Helena Lohmann, Machado UF, Musarò A, Moriscot AS. Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate [Internet]. Biomed Research International. 2015 ; 2015 1-8.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1155/2015/282984
    • Vancouver

      Christoffolete MA, Silva WJ, Ramos GV, Bento MR, Costa MO, Ribeiro MO, Okamoto MM, Tania Helena Lohmann, Machado UF, Musarò A, Moriscot AS. Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate [Internet]. Biomed Research International. 2015 ; 2015 1-8.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1155/2015/282984
  • Source: Archives of Endocrinology and Metabolism. Unidade: ICB

    Subjects: FISIOLOGIA, OBESIDADE

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      LIMA, Fabio Bessa. How challenging is to establish parameters good enough to assess the repercussions of trials to fight obesity. Archives of Endocrinology and Metabolism. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1590/2359-3997000000036. Acesso em: 22 dez. 2024. , 2015
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      Lima, F. B. (2015). How challenging is to establish parameters good enough to assess the repercussions of trials to fight obesity. Archives of Endocrinology and Metabolism. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1590/2359-3997000000036
    • NLM

      Lima FB. How challenging is to establish parameters good enough to assess the repercussions of trials to fight obesity [Internet]. Archives of Endocrinology and Metabolism. 2015 ; 59( 3): 197-198.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1590/2359-3997000000036
    • Vancouver

      Lima FB. How challenging is to establish parameters good enough to assess the repercussions of trials to fight obesity [Internet]. Archives of Endocrinology and Metabolism. 2015 ; 59( 3): 197-198.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1590/2359-3997000000036
  • Source: Growth Hormone & IGF Research. Unidade: ICB

    Subjects: FISIOLOGIA, RATOS, LACTATOS

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      SALGUEIRO, Rafael Barrera et al. Lactate activates the somatotropic axis in rats. Growth Hormone & IGF Research, v. 6, p. 268-270, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ghir.2014.09.003. Acesso em: 22 dez. 2024.
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      Salgueiro, R. B., Garcia, R. A. P., Buonfiglio, D. do C., Peroni, C. N., & Nunes, M. T. (2015). Lactate activates the somatotropic axis in rats. Growth Hormone & IGF Research, 6, 268-270. doi:10.1016/j.ghir.2014.09.003
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      Salgueiro RB, Garcia RAP, Buonfiglio D do C, Peroni CN, Nunes MT. Lactate activates the somatotropic axis in rats [Internet]. Growth Hormone & IGF Research. 2015 ; 6 268-270.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.ghir.2014.09.003
    • Vancouver

      Salgueiro RB, Garcia RAP, Buonfiglio D do C, Peroni CN, Nunes MT. Lactate activates the somatotropic axis in rats [Internet]. Growth Hormone & IGF Research. 2015 ; 6 268-270.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.ghir.2014.09.003
  • Source: Molecular Metabolism. Unidade: ICB

    Subjects: FISIOLOGIA, LEPTINA (DEFICIÊNCIA)

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      ZAMPIERI, Thais Tessari et al. SOCS3 deficiency in leptin receptor-expressing cells mitigates the development of pregnancy-induced metabolic changes. Molecular Metabolism, v. 4, n. 3, p. 237-245, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2014.12.005. Acesso em: 22 dez. 2024.
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      Zampieri, T. T., Lobo, A. M. R., Furigo, I. C., Pedroso, J. A. B., Buonfiglio, D. do C., & Donato Junior, J. (2015). SOCS3 deficiency in leptin receptor-expressing cells mitigates the development of pregnancy-induced metabolic changes. Molecular Metabolism, 4( 3), 237-245. doi:10.1016/j.molmet.2014.12.005
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      Zampieri TT, Lobo AMR, Furigo IC, Pedroso JAB, Buonfiglio D do C, Donato Junior J. SOCS3 deficiency in leptin receptor-expressing cells mitigates the development of pregnancy-induced metabolic changes [Internet]. Molecular Metabolism. 2015 ; 4( 3): 237-245.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.molmet.2014.12.005
    • Vancouver

      Zampieri TT, Lobo AMR, Furigo IC, Pedroso JAB, Buonfiglio D do C, Donato Junior J. SOCS3 deficiency in leptin receptor-expressing cells mitigates the development of pregnancy-induced metabolic changes [Internet]. Molecular Metabolism. 2015 ; 4( 3): 237-245.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.molmet.2014.12.005
  • Source: PLOS ONE. Unidade: ICB

    Assunto: FISIOLOGIA

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      KINJO, Erika et al. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus. PLOS ONE, v. 9, n. 10, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0109149. Acesso em: 22 dez. 2024.
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      Kinjo, E., Higa, G. S. V., Morya, E., Valle, A. C., Kihara, A. H., & Britto, L. R. G. de. (2014). Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus. PLOS ONE, 9( 10), 1-12. doi:10.1371/journal.pone.0109149
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      Kinjo E, Higa GSV, Morya E, Valle AC, Kihara AH, Britto LRG de. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus [Internet]. PLOS ONE. 2014 ; 9( 10): 1-12.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1371/journal.pone.0109149
    • Vancouver

      Kinjo E, Higa GSV, Morya E, Valle AC, Kihara AH, Britto LRG de. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus [Internet]. PLOS ONE. 2014 ; 9( 10): 1-12.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1371/journal.pone.0109149
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Subjects: FISIOLOGIA, HIPERTIREOIDISMO, EXPRESSÃO GÊNICA

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      SOUZA, K. de Picoli e NUNES, Maria Tereza. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood. Brazilian Journal of Medical and Biological Research, v. 47, n. 8, p. 670-678, 2014Tradução . . Disponível em: https://doi.org/10.1590/1414-431X20142875. Acesso em: 22 dez. 2024.
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      Souza, K. de P., & Nunes, M. T. (2014). Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood. Brazilian Journal of Medical and Biological Research, 47( 8), 670-678. doi:10.1590/1414-431X20142875
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      Souza K de P, Nunes MT. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood [Internet]. Brazilian Journal of Medical and Biological Research. 2014 ; 47( 8): 670-678.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1590/1414-431X20142875
    • Vancouver

      Souza K de P, Nunes MT. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood [Internet]. Brazilian Journal of Medical and Biological Research. 2014 ; 47( 8): 670-678.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1590/1414-431X20142875
  • Source: Journal of Physiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      ROBERTO SOBRINHO, Cleyton et al. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe. Journal of Physiology, v. 15, p. 1309-1323, 2014Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2013.268490. Acesso em: 22 dez. 2024.
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      Roberto Sobrinho, C., Wenker, I. C., Poss, E. M., Takakura, A. C., Moreira, T. dos S., & Mulkey, D. K. (2014). Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe. Journal of Physiology, 15, 1309-1323. doi:10.1113/jphysiol.2013.268490
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      Roberto Sobrinho C, Wenker IC, Poss EM, Takakura AC, Moreira T dos S, Mulkey DK. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe [Internet]. Journal of Physiology. 2014 ; 15 1309-1323.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1113/jphysiol.2013.268490
    • Vancouver

      Roberto Sobrinho C, Wenker IC, Poss EM, Takakura AC, Moreira T dos S, Mulkey DK. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe [Internet]. Journal of Physiology. 2014 ; 15 1309-1323.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1113/jphysiol.2013.268490
  • Source: Journal of Molecular Medicine. Unidade: ICB

    Subjects: FISIOLOGIA, HISTOLOGIA

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      MARTÍN-RUFIÁN, Mercedes et al. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, v. 92, n. 3, p. 277-290, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00109-013-1105-2. Acesso em: 22 dez. 2024.
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      Martín-Rufián, M., Nascimento-Gomes, R., Higuero, A., Crisma, A. R., Campos-Sandoval, J. A., Gómez-Garcia, M. C., et al. (2014). Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, 92( 3), 277-290. doi:10.1007/s00109-013-1105-2
    • NLM

      Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
    • Vancouver

      Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
  • Source: Molecular Vision. Unidade: ICB

    Assunto: FISIOLOGIA

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      BUONFIGLIO, Daniella et al. Rat retina shows robust circadian expression of clock and clock output genes in explant culture. Molecular Vision, v. 20, p. 742-752, 2014Tradução . . Acesso em: 22 dez. 2024.
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      Buonfiglio, D., Malan, A., Sandu, C., Jaegger, C., Cipolla-Neto, J., Hicks, D., & Felder-Schmittbuhl, M. -P. (2014). Rat retina shows robust circadian expression of clock and clock output genes in explant culture. Molecular Vision, 20, 742-752.
    • NLM

      Buonfiglio D, Malan A, Sandu C, Jaegger C, Cipolla-Neto J, Hicks D, Felder-Schmittbuhl M-P. Rat retina shows robust circadian expression of clock and clock output genes in explant culture. Molecular Vision. 2014 ; 20 742-752.[citado 2024 dez. 22 ]
    • Vancouver

      Buonfiglio D, Malan A, Sandu C, Jaegger C, Cipolla-Neto J, Hicks D, Felder-Schmittbuhl M-P. Rat retina shows robust circadian expression of clock and clock output genes in explant culture. Molecular Vision. 2014 ; 20 742-752.[citado 2024 dez. 22 ]
  • Source: PLOS ONE. Unidade: ICB

    Subjects: FISIOLOGIA, MACRÓFAGOS, LIPOLISSACARIDEOS, INFLAMAÇÃO

    Acesso à fonteDOIHow to cite
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      FESTUCCIA, William Tadeu Lara et al. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide. PLOS ONE, v. 9, n. 4, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0095432. Acesso em: 22 dez. 2024.
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      Festuccia, W. T. L., Pouliot, P., Bakan, I., Sabatini, D. M., & Laplante, M. (2014). Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide. PLOS ONE, 9( 4), 1-12. doi:10.1371/journal.pone.0095432
    • NLM

      Festuccia WTL, Pouliot P, Bakan I, Sabatini DM, Laplante M. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide [Internet]. PLOS ONE. 2014 ; 9( 4): 1-12.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1371/journal.pone.0095432
    • Vancouver

      Festuccia WTL, Pouliot P, Bakan I, Sabatini DM, Laplante M. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide [Internet]. PLOS ONE. 2014 ; 9( 4): 1-12.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1371/journal.pone.0095432
  • Source: Mediators of Inflammation. Unidade: ICB

    Assunto: FISIOLOGIA

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      MOREIRA, Vanessa et al. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, v. 2014, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/105879. Acesso em: 22 dez. 2024.
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      Moreira, V., Lomonte, B., Vinolo, M. A. R., Curi, R., Gutiérrez, J. M., & Teixeira, C. (2014). An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, 2014, 1-10. doi:10.1155/2014/105879
    • NLM

      Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1155/2014/105879
    • Vancouver

      Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1155/2014/105879

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