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Artigo de periodico
Targeting prion protein interactions in cancer (2015)
Santos, Tiago Góss dos; Lopes, Marilene Hohmuth ; Martins, Vilma Regina
Source: Prion. Unidade: ICB
Subjects: HISTOLOGIA, NEOPLASIAS, PROTEINAS (INTERAÇÃO)
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ABNT
SANTOS, Tiago Góss dos e LOPES, Marilene Hohmuth e MARTINS, Vilma Regina. Targeting prion protein interactions in cancer. Prion, v. 9, n. 3, p. 165-173, 2015Tradução . . Disponível em: https://doi.org/10.1080/19336896.2015.1027855. Acesso em: 06 set. 2024.
APA
Santos, T. G. dos, Lopes, M. H., & Martins, V. R. (2015). Targeting prion protein interactions in cancer. Prion, 9( 3), 165-173. doi:10.1080/19336896.2015.1027855
NLM
Santos TG dos, Lopes MH, Martins VR. Targeting prion protein interactions in cancer [Internet]. Prion. 2015 ; 9( 3): 165-173.[citado 2024 set. 06 ] Available from: https://doi.org/10.1080/19336896.2015.1027855
Vancouver
Santos TG dos, Lopes MH, Martins VR. Targeting prion protein interactions in cancer [Internet]. Prion. 2015 ; 9( 3): 165-173.[citado 2024 set. 06 ] Available from: https://doi.org/10.1080/19336896.2015.1027855
Artigo de periodico-carta/editorial
Inflammation in the disease: Mechanism and therapies 2014 (2015)
Seelaender, Marília Cerqueira Leite; Rosa Neto, José Cesar ; Pimentel, G. D.; Goldszmid, R. S.; Lira, F. S.
Source: Mediators of Inflammation. Unidade: ICB
Subjects: HISTOLOGIA, INFLAMAÇÃO
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ABNT
SEELAENDER, Marília Cerqueira Leite et al. Inflammation in the disease: Mechanism and therapies 2014. Mediators of Inflammation. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1155/2015/169852. Acesso em: 06 set. 2024. , 2015
APA
Seelaender, M. C. L., Rosa Neto, J. C., Pimentel, G. D., Goldszmid, R. S., & Lira, F. S. (2015). Inflammation in the disease: Mechanism and therapies 2014. Mediators of Inflammation. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1155/2015/169852
NLM
Seelaender MCL, Rosa Neto JC, Pimentel GD, Goldszmid RS, Lira FS. Inflammation in the disease: Mechanism and therapies 2014 [Internet]. Mediators of Inflammation. 2015 ; 2015 1-2.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2015/169852
Vancouver
Seelaender MCL, Rosa Neto JC, Pimentel GD, Goldszmid RS, Lira FS. Inflammation in the disease: Mechanism and therapies 2014 [Internet]. Mediators of Inflammation. 2015 ; 2015 1-2.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2015/169852
Artigo de periodico
Transforming growth factor β1 increases p27 levels via synthesis and degradation mechanisms in the hyperproliferative gastric epithelium in rats (2014)
Fiore, Ana Paula Zen Petisco; Osaki, Luciana H.; Gama, Patrícia
Source: PLOS ONE. Unidade: ICB
Assunto: HISTOLOGIA
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FIORE, Ana Paula Zen Petisco e OSAKI, Luciana H. e GAMA, Patrícia. Transforming growth factor β1 increases p27 levels via synthesis and degradation mechanisms in the hyperproliferative gastric epithelium in rats. PLOS ONE, v. 9, n. 7, p. 1-8, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0101965. Acesso em: 06 set. 2024.
APA
Fiore, A. P. Z. P., Osaki, L. H., & Gama, P. (2014). Transforming growth factor β1 increases p27 levels via synthesis and degradation mechanisms in the hyperproliferative gastric epithelium in rats. PLOS ONE, 9( 7), 1-8. doi:10.1371/journal.pone.0101965
NLM
Fiore APZP, Osaki LH, Gama P. Transforming growth factor β1 increases p27 levels via synthesis and degradation mechanisms in the hyperproliferative gastric epithelium in rats [Internet]. PLOS ONE. 2014 ; 9( 7): 1-8.[citado 2024 set. 06 ] Available from: https://doi.org/10.1371/journal.pone.0101965
Vancouver
Fiore APZP, Osaki LH, Gama P. Transforming growth factor β1 increases p27 levels via synthesis and degradation mechanisms in the hyperproliferative gastric epithelium in rats [Internet]. PLOS ONE. 2014 ; 9( 7): 1-8.[citado 2024 set. 06 ] Available from: https://doi.org/10.1371/journal.pone.0101965
Artigo de periodico
40LoVe and Samba are involved in Xenopus Neural Development and Functionally Distinct from hnRNP AB (2014)
Andreou, Maria; Yan, Chao Yun Irene ; Skourides, Paris A.
Source: PLOS ONE. Unidade: ICB
Assunto: HISTOLOGIA
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ANDREOU, Maria e YAN, Chao Yun Irene e SKOURIDES, Paris A. 40LoVe and Samba are involved in Xenopus Neural Development and Functionally Distinct from hnRNP AB. PLOS ONE, v. 9, n. 1, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0085026. Acesso em: 06 set. 2024.
APA
Andreou, M., Yan, C. Y. I., & Skourides, P. A. (2014). 40LoVe and Samba are involved in Xenopus Neural Development and Functionally Distinct from hnRNP AB. PLOS ONE, 9( 1), 1-10. doi:10.1371/journal.pone.0085026
NLM
Andreou M, Yan CYI, Skourides PA. 40LoVe and Samba are involved in Xenopus Neural Development and Functionally Distinct from hnRNP AB [Internet]. PLOS ONE. 2014 ; 9( 1): 1-10.[citado 2024 set. 06 ] Available from: https://doi.org/10.1371/journal.pone.0085026
Vancouver
Andreou M, Yan CYI, Skourides PA. 40LoVe and Samba are involved in Xenopus Neural Development and Functionally Distinct from hnRNP AB [Internet]. PLOS ONE. 2014 ; 9( 1): 1-10.[citado 2024 set. 06 ] Available from: https://doi.org/10.1371/journal.pone.0085026
Artigo de periodico
EGFR signaling downstream of EGF regulates migration, invasion, and MMP secretion of immortalized cells derived from human ameloblastoma (2014)
Rosa, Marina Rolo Pinheiro da; Falcão, Aline Semblano Carreira; Fuzii, Hellen Thais; Kataoka, Maria Sueli da Silva; Ribeiro, André L. R.; Boccardo, Enrique ; Siqueira, Adriane Sousa de; Jaeger, Ruy Gastaldoni ; Pinheiro, João de Jesus Viana; Alves Júnior, Sérgio de Melo
Source: Tumor Biology. Unidade: ICB
Subjects: HISTOLOGIA, MICROBIOLOGIA
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ABNT
ROSA, Marina Rolo Pinheiro da et al. EGFR signaling downstream of EGF regulates migration, invasion, and MMP secretion of immortalized cells derived from human ameloblastoma. Tumor Biology, v. 35, n. 11, p. 11107-11120, 2014Tradução . . Disponível em: https://doi.org/10.1007/s13277-014-2401-3. Acesso em: 06 set. 2024.
APA
Rosa, M. R. P. da, Falcão, A. S. C., Fuzii, H. T., Kataoka, M. S. da S., Ribeiro, A. L. R., Boccardo, E., et al. (2014). EGFR signaling downstream of EGF regulates migration, invasion, and MMP secretion of immortalized cells derived from human ameloblastoma. Tumor Biology, 35( 11), 11107-11120. doi:10.1007/s13277-014-2401-3
NLM
Rosa MRP da, Falcão ASC, Fuzii HT, Kataoka MS da S, Ribeiro ALR, Boccardo E, Siqueira AS de, Jaeger RG, Pinheiro J de JV, Alves Júnior S de M. EGFR signaling downstream of EGF regulates migration, invasion, and MMP secretion of immortalized cells derived from human ameloblastoma [Internet]. Tumor Biology. 2014 ; 35( 11): 11107-11120.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s13277-014-2401-3
Vancouver
Rosa MRP da, Falcão ASC, Fuzii HT, Kataoka MS da S, Ribeiro ALR, Boccardo E, Siqueira AS de, Jaeger RG, Pinheiro J de JV, Alves Júnior S de M. EGFR signaling downstream of EGF regulates migration, invasion, and MMP secretion of immortalized cells derived from human ameloblastoma [Internet]. Tumor Biology. 2014 ; 35( 11): 11107-11120.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s13277-014-2401-3
Artigo de periodico
Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells (2014)
Martín-Rufián, Mercedes; Nascimento-Gomes, Renata; Higuero, Ana; Crisma, Amanda R.; Campos-Sandoval, José A.; Gómez-Garcia, María C.; Cardona, Carolina; Cheng, Tzuling; Lobo, Carolina; Segura, Juan A.; Alonso, Francisco J.; Szeliga, Monika; Albrecht, Jan; Curi, Rui; Márquez, Javier; Colquhoun, Alison ; DeBerardinis, Ralph J.; Matés, José M.
Source: Journal of Molecular Medicine. Unidade: ICB
Subjects: FISIOLOGIA, HISTOLOGIA
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ABNT
MARTÍN-RUFIÁN, Mercedes et al. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, v. 92, n. 3, p. 277-290, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00109-013-1105-2. Acesso em: 06 set. 2024.
APA
Martín-Rufián, M., Nascimento-Gomes, R., Higuero, A., Crisma, A. R., Campos-Sandoval, J. A., Gómez-Garcia, M. C., et al. (2014). Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, 92( 3), 277-290. doi:10.1007/s00109-013-1105-2
NLM
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Vancouver
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Artigo de periodico
Preclinical evaluation of the combination of mTOR and proteasome inhibitors with radiotherapy in malignant peripheral nerve sheath tumors (2014)
Yamashita, Alex Shimura; Baia, Gilson Soares; Ho, J. S. Y.; Velarde, E.; Wong, J.; Gallia, Gary L.; Belzberg, A. J.; Kimura, Edna Teruko ; Riggins, Gregory J.
Source: Journal of Neuro-Oncology. Unidade: ICB
Subjects: HISTOLOGIA, RADIOTERAPIA, SISTEMA NERVOSO PERIFÉRICO, CÉLULAS CULTIVADAS DE TUMOR, PROLIFERAÇÃO CELULAR
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ABNT
YAMASHITA, Alex Shimura et al. Preclinical evaluation of the combination of mTOR and proteasome inhibitors with radiotherapy in malignant peripheral nerve sheath tumors. Journal of Neuro-Oncology, v. 118, n. 1, p. 83-92, 2014Tradução . . Disponível em: https://doi.org/10.1007/s11060-014-1422-5. Acesso em: 06 set. 2024.
APA
Yamashita, A. S., Baia, G. S., Ho, J. S. Y., Velarde, E., Wong, J., Gallia, G. L., et al. (2014). Preclinical evaluation of the combination of mTOR and proteasome inhibitors with radiotherapy in malignant peripheral nerve sheath tumors. Journal of Neuro-Oncology, 118( 1), 83-92. doi:10.1007/s11060-014-1422-5
NLM
Yamashita AS, Baia GS, Ho JSY, Velarde E, Wong J, Gallia GL, Belzberg AJ, Kimura ET, Riggins GJ. Preclinical evaluation of the combination of mTOR and proteasome inhibitors with radiotherapy in malignant peripheral nerve sheath tumors [Internet]. Journal of Neuro-Oncology. 2014 ; 118( 1): 83-92.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s11060-014-1422-5
Vancouver
Yamashita AS, Baia GS, Ho JSY, Velarde E, Wong J, Gallia GL, Belzberg AJ, Kimura ET, Riggins GJ. Preclinical evaluation of the combination of mTOR and proteasome inhibitors with radiotherapy in malignant peripheral nerve sheath tumors [Internet]. Journal of Neuro-Oncology. 2014 ; 118( 1): 83-92.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s11060-014-1422-5
Artigo de periodico
High iodine blocks a Notch/miR-19 loop activated by the BRAFV600E oncoprotein and restores the response to TGFβ in thyroid follicular cells (2014)
Fuziwara, Cesar Seigi; Kimura, Edna Teruko
Source: Thyroid. Unidade: ICB
Assunto: HISTOLOGIA
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ABNT
FUZIWARA, Cesar Seigi e KIMURA, Edna Teruko. High iodine blocks a Notch/miR-19 loop activated by the BRAFV600E oncoprotein and restores the response to TGFβ in thyroid follicular cells. Thyroid, v. 24, n. 3, p. 453-462, 2014Tradução . . Disponível em: https://doi.org/10.1089/thy.2013.0398. Acesso em: 06 set. 2024.
APA
Fuziwara, C. S., & Kimura, E. T. (2014). High iodine blocks a Notch/miR-19 loop activated by the BRAFV600E oncoprotein and restores the response to TGFβ in thyroid follicular cells. Thyroid, 24( 3), 453-462. doi:10.1089/thy.2013.0398
NLM
Fuziwara CS, Kimura ET. High iodine blocks a Notch/miR-19 loop activated by the BRAFV600E oncoprotein and restores the response to TGFβ in thyroid follicular cells [Internet]. Thyroid. 2014 ; 24( 3): 453-462.[citado 2024 set. 06 ] Available from: https://doi.org/10.1089/thy.2013.0398
Vancouver
Fuziwara CS, Kimura ET. High iodine blocks a Notch/miR-19 loop activated by the BRAFV600E oncoprotein and restores the response to TGFβ in thyroid follicular cells [Internet]. Thyroid. 2014 ; 24( 3): 453-462.[citado 2024 set. 06 ] Available from: https://doi.org/10.1089/thy.2013.0398
Artigo de periodico
Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling (2014)
Protzek, André O. P.; Costa-Júnior, José M.; Rezende, Luiz F.; Santos, Gustavo J.; Araújo, Tiago Gomes; Vettorazzi, Jean F.; Ortis, Fernanda ; Carneiro, Everardo M.; Rafacho, Alex; Boschero, Antonio C.
Source: International Journal of Endocrinology. Unidade: ICB
Subjects: HISTOLOGIA, INSULINA (RESISTÊNCIA), CAMUNDONGOS, RATOS WISTAR
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ABNT
PROTZEK, André O. P. et al. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling. International Journal of Endocrinology, v. 2014, p. 1-14, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/983453. Acesso em: 06 set. 2024.
APA
Protzek, A. O. P., Costa-Júnior, J. M., Rezende, L. F., Santos, G. J., Araújo, T. G., Vettorazzi, J. F., et al. (2014). Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling. International Journal of Endocrinology, 2014, 1-14. doi:10.1155/2014/983453
NLM
Protzek AOP, Costa-Júnior JM, Rezende LF, Santos GJ, Araújo TG, Vettorazzi JF, Ortis F, Carneiro EM, Rafacho A, Boschero AC. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling [Internet]. International Journal of Endocrinology. 2014 ; 2014 1-14.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/983453
Vancouver
Protzek AOP, Costa-Júnior JM, Rezende LF, Santos GJ, Araújo TG, Vettorazzi JF, Ortis F, Carneiro EM, Rafacho A, Boschero AC. Augmented β-Cell function and mass in glucocorticoid-treated rodents are associated with increased islet Ir-β/AKT/mTOR and decreased AMPK/ACC and AS160 signaling [Internet]. International Journal of Endocrinology. 2014 ; 2014 1-14.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/983453
Artigo de periodico
Decorin and biglycan immunolocalization in non-villous structures of healthy and pathological human placentas (2014)
Borbely, Alexandre Urban; Daher, Silvia; Mattar, Rosiane; Sun, Sue Y.; Knöfler, Martin; Bevilacqua, Estela Maris Andrade Forell ; Oliveira, Sergio Ferreira de
Source: Histopathology. Unidade: ICB
Assunto: HISTOLOGIA
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ABNT
BORBELY, Alexandre Urban et al. Decorin and biglycan immunolocalization in non-villous structures of healthy and pathological human placentas. Histopathology, v. 64, n. 5, p. 616-625, 2014Tradução . . Disponível em: https://doi.org/10.1111/his.12304. Acesso em: 06 set. 2024.
APA
Borbely, A. U., Daher, S., Mattar, R., Sun, S. Y., Knöfler, M., Bevilacqua, E. M. A. F., & Oliveira, S. F. de. (2014). Decorin and biglycan immunolocalization in non-villous structures of healthy and pathological human placentas. Histopathology, 64( 5), 616-625. doi:10.1111/his.12304
NLM
Borbely AU, Daher S, Mattar R, Sun SY, Knöfler M, Bevilacqua EMAF, Oliveira SF de. Decorin and biglycan immunolocalization in non-villous structures of healthy and pathological human placentas [Internet]. Histopathology. 2014 ; 64( 5): 616-625.[citado 2024 set. 06 ] Available from: https://doi.org/10.1111/his.12304
Vancouver
Borbely AU, Daher S, Mattar R, Sun SY, Knöfler M, Bevilacqua EMAF, Oliveira SF de. Decorin and biglycan immunolocalization in non-villous structures of healthy and pathological human placentas [Internet]. Histopathology. 2014 ; 64( 5): 616-625.[citado 2024 set. 06 ] Available from: https://doi.org/10.1111/his.12304
Artigo de periodico
Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII (2014)
Santos, Gustavo Jorge dos; Ferreira, Sandra Mara; Ortis, Fernanda ; Rezende, Luiz Fernando; Li, Chengyang; Naji, Ali; Carneiro, Everardo Magalhães; Kaestner, Klaus H.; Boschero, Antonio Carlos
Source: Molecular Metabolism. Unidade: ICB
Subjects: HISTOLOGIA, METABOLISMO, SECREÇÃO, INSULINA
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ABNT
SANTOS, Gustavo Jorge dos et al. Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII. Molecular Metabolism, v. 3, n. 4, p. 484-489, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2014.03.011. Acesso em: 06 set. 2024.
APA
Santos, G. J. dos, Ferreira, S. M., Ortis, F., Rezende, L. F., Li, C., Naji, A., et al. (2014). Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII. Molecular Metabolism, 3( 4), 484-489. doi:10.1016/j.molmet.2014.03.011
NLM
Santos GJ dos, Ferreira SM, Ortis F, Rezende LF, Li C, Naji A, Carneiro EM, Kaestner KH, Boschero AC. Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII [Internet]. Molecular Metabolism. 2014 ; 3( 4): 484-489.[citado 2024 set. 06 ] Available from: https://doi.org/10.1016/j.molmet.2014.03.011
Vancouver
Santos GJ dos, Ferreira SM, Ortis F, Rezende LF, Li C, Naji A, Carneiro EM, Kaestner KH, Boschero AC. Metabolic memory of ß-cells controls insulin secretion and is mediated by CaMKII [Internet]. Molecular Metabolism. 2014 ; 3( 4): 484-489.[citado 2024 set. 06 ] Available from: https://doi.org/10.1016/j.molmet.2014.03.011
Artigo de periodico
Imaging of nonlinear microscopy of burned skin treated by ultra-high intensity laser pulses (2014)
Santos, Moises Oliveira dos; Benetti, Carolina; Pelegati, Vitor Bianchin; Cesar, Carlos Lenz; Rossi, Wagner de; Samad, Ricardo Elgul; Vieira Junior, Nilson Dias; Zezell, Denise Maria; Zorn, Telma Maria Tenório
Source: Biomedical Spectroscopy and Imaging. Unidade: ICB
Subjects: HISTOLOGIA, LASER, MICROSCOPIA, QUEIMADURAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Moises Oliveira dos et al. Imaging of nonlinear microscopy of burned skin treated by ultra-high intensity laser pulses. Biomedical Spectroscopy and Imaging, v. 3, n. 3, p. 293-300, 2014Tradução . . Disponível em: https://doi.org/10.3233/BSI-140085. Acesso em: 06 set. 2024.
APA
Santos, M. O. dos, Benetti, C., Pelegati, V. B., Cesar, C. L., Rossi, W. de, Samad, R. E., et al. (2014). Imaging of nonlinear microscopy of burned skin treated by ultra-high intensity laser pulses. Biomedical Spectroscopy and Imaging, 3( 3), 293-300. doi:10.3233/BSI-140085
NLM
Santos MO dos, Benetti C, Pelegati VB, Cesar CL, Rossi W de, Samad RE, Vieira Junior ND, Zezell DM, Zorn TMT. Imaging of nonlinear microscopy of burned skin treated by ultra-high intensity laser pulses [Internet]. Biomedical Spectroscopy and Imaging. 2014 ; 3( 3): 293-300.[citado 2024 set. 06 ] Available from: https://doi.org/10.3233/BSI-140085
Vancouver
Santos MO dos, Benetti C, Pelegati VB, Cesar CL, Rossi W de, Samad RE, Vieira Junior ND, Zezell DM, Zorn TMT. Imaging of nonlinear microscopy of burned skin treated by ultra-high intensity laser pulses [Internet]. Biomedical Spectroscopy and Imaging. 2014 ; 3( 3): 293-300.[citado 2024 set. 06 ] Available from: https://doi.org/10.3233/BSI-140085
Artigo de periodico-carta/editorial
Immunometabolism: molecular mechanisms, diseases, and therapies (2014)
Rosa Neto, José Cesar ; Lira, Fábio Santos; Festuccia, William Tadeu Lara
Source: Mediators of Inflammation. Unidade: ICB
Subjects: FISIOLOGIA, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSA NETO, José Cesar e LIRA, Fábio Santos e FESTUCCIA, William Tadeu Lara. Immunometabolism: molecular mechanisms, diseases, and therapies. Mediators of Inflammation. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1155/2014/585708. Acesso em: 06 set. 2024. , 2014
APA
Rosa Neto, J. C., Lira, F. S., & Festuccia, W. T. L. (2014). Immunometabolism: molecular mechanisms, diseases, and therapies. Mediators of Inflammation. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1155/2014/585708
NLM
Rosa Neto JC, Lira FS, Festuccia WTL. Immunometabolism: molecular mechanisms, diseases, and therapies [Internet]. Mediators of Inflammation. 2014 ; 2014 1-2.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/585708
Vancouver
Rosa Neto JC, Lira FS, Festuccia WTL. Immunometabolism: molecular mechanisms, diseases, and therapies [Internet]. Mediators of Inflammation. 2014 ; 2014 1-2.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/585708
Artigo de periodico
Role of mycosin Va in neuritogenesis of chick dorsal root ganglia nociceptive neurons (2014)
Kanno, Tatiane Yumi Nakamura; Espreafico, Enilza Maria; Yan, Chao Yun Irene
Source: Cell Biology Internacional. Unidades: ICB, FMRP
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KANNO, Tatiane Yumi Nakamura e ESPREAFICO, Enilza Maria e YAN, Chao Yun Irene. Role of mycosin Va in neuritogenesis of chick dorsal root ganglia nociceptive neurons. Cell Biology Internacional, v. 38, n. 3, p. 388-194, 2014Tradução . . Disponível em: https://doi.org/10.1002/cbin.10210. Acesso em: 06 set. 2024.
APA
Kanno, T. Y. N., Espreafico, E. M., & Yan, C. Y. I. (2014). Role of mycosin Va in neuritogenesis of chick dorsal root ganglia nociceptive neurons. Cell Biology Internacional, 38( 3), 388-194. doi:10.1002/cbin.10210
NLM
Kanno TYN, Espreafico EM, Yan CYI. Role of mycosin Va in neuritogenesis of chick dorsal root ganglia nociceptive neurons [Internet]. Cell Biology Internacional. 2014 ; 38( 3): 388-194.[citado 2024 set. 06 ] Available from: https://doi.org/10.1002/cbin.10210
Vancouver
Kanno TYN, Espreafico EM, Yan CYI. Role of mycosin Va in neuritogenesis of chick dorsal root ganglia nociceptive neurons [Internet]. Cell Biology Internacional. 2014 ; 38( 3): 388-194.[citado 2024 set. 06 ] Available from: https://doi.org/10.1002/cbin.10210
Artigo de periodico
Palmitoleic acid (N-7) attenuates the immunometabolic disturbances caused by a high-fat diet independently of PPARα (2014)
Souza, Camila Oliveira; Teixeira, Alexandre Abilio de Souza ; Lima, Edson A.; Batatinha, Helena Angelica Pereira ; Gomes, Lara M.; Carvalho-Silva, Milena; Mota, Isabela T.; Streck, Emilio Luiz; Hirabara, Sandro Massao ; Rosa Neto, José Cesar
Source: Mediators of Inflammation. Unidade: ICB
Subjects: HISTOLOGIA, ANTI-INFLAMATÓRIOS, METABOLISMO ANIMAL, IMUNOLOGIA VETERINÁRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Camila Oliveira et al. Palmitoleic acid (N-7) attenuates the immunometabolic disturbances caused by a high-fat diet independently of PPARα. Mediators of Inflammation, v. 2014, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/582197. Acesso em: 06 set. 2024.
APA
Souza, C. O., Teixeira, A. A. de S., Lima, E. A., Batatinha, H. A. P., Gomes, L. M., Carvalho-Silva, M., et al. (2014). Palmitoleic acid (N-7) attenuates the immunometabolic disturbances caused by a high-fat diet independently of PPARα. Mediators of Inflammation, 2014, 1-12. doi:10.1155/2014/582197
NLM
Souza CO, Teixeira AA de S, Lima EA, Batatinha HAP, Gomes LM, Carvalho-Silva M, Mota IT, Streck EL, Hirabara SM, Rosa Neto JC. Palmitoleic acid (N-7) attenuates the immunometabolic disturbances caused by a high-fat diet independently of PPARα [Internet]. Mediators of Inflammation. 2014 ; 2014 1-12.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/582197
Vancouver
Souza CO, Teixeira AA de S, Lima EA, Batatinha HAP, Gomes LM, Carvalho-Silva M, Mota IT, Streck EL, Hirabara SM, Rosa Neto JC. Palmitoleic acid (N-7) attenuates the immunometabolic disturbances caused by a high-fat diet independently of PPARα [Internet]. Mediators of Inflammation. 2014 ; 2014 1-12.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/582197
Artigo de periodico
Morphologic study of the liver of lambari (Astyanax altiparanae) with emphasis on the distribution of cytokeratin (2014)
Chehade, Chayrra; Cassel, Mônica; Borella, Maria Inês; Costa, Fabiano Gonçalves
Source: Fish Physiology and. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHEHADE, Chayrra et al. Morphologic study of the liver of lambari (Astyanax altiparanae) with emphasis on the distribution of cytokeratin. Fish Physiology and, v. 40, n. 2, p. 571-576, 2014Tradução . . Disponível em: https://doi.org/10.1007/s10695-013-9867-6. Acesso em: 06 set. 2024.
APA
Chehade, C., Cassel, M., Borella, M. I., & Costa, F. G. (2014). Morphologic study of the liver of lambari (Astyanax altiparanae) with emphasis on the distribution of cytokeratin. Fish Physiology and, 40( 2), 571-576. doi:10.1007/s10695-013-9867-6
NLM
Chehade C, Cassel M, Borella MI, Costa FG. Morphologic study of the liver of lambari (Astyanax altiparanae) with emphasis on the distribution of cytokeratin [Internet]. Fish Physiology and. 2014 ; 40( 2): 571-576.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s10695-013-9867-6
Vancouver
Chehade C, Cassel M, Borella MI, Costa FG. Morphologic study of the liver of lambari (Astyanax altiparanae) with emphasis on the distribution of cytokeratin [Internet]. Fish Physiology and. 2014 ; 40( 2): 571-576.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s10695-013-9867-6
Artigo de periodico
Copper removal using a heavy-metal resistant microbial consortium in a fixed-bed reactor (2014)
Carpio, Isis E. Mejias; Machado-Santelli, Glaucia Maria; Sakata, Solange Kazumi; Ferreira Filho, Sidney Seckler ; Rodrigues, Debora Frigi
Source: Water Research. Unidades: ICB, EP
Subjects: HISTOLOGIA, AGUA CONTAMINADA, RIOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARPIO, Isis E. Mejias et al. Copper removal using a heavy-metal resistant microbial consortium in a fixed-bed reactor. Water Research, v. 62, p. 156-166, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.watres.2014.05.043. Acesso em: 06 set. 2024.
APA
Carpio, I. E. M., Machado-Santelli, G. M., Sakata, S. K., Ferreira Filho, S. S., & Rodrigues, D. F. (2014). Copper removal using a heavy-metal resistant microbial consortium in a fixed-bed reactor. Water Research, 62, 156-166. doi:10.1016/j.watres.2014.05.043
NLM
Carpio IEM, Machado-Santelli GM, Sakata SK, Ferreira Filho SS, Rodrigues DF. Copper removal using a heavy-metal resistant microbial consortium in a fixed-bed reactor [Internet]. Water Research. 2014 ; 62 156-166.[citado 2024 set. 06 ] Available from: https://doi.org/10.1016/j.watres.2014.05.043
Vancouver
Carpio IEM, Machado-Santelli GM, Sakata SK, Ferreira Filho SS, Rodrigues DF. Copper removal using a heavy-metal resistant microbial consortium in a fixed-bed reactor [Internet]. Water Research. 2014 ; 62 156-166.[citado 2024 set. 06 ] Available from: https://doi.org/10.1016/j.watres.2014.05.043
Artigo de periodico
DNA damage and its cellular response in mother and fetus exposed to hyperglycemic environment (2014)
Moreli, Jusciele Brogin; Santos, Janine Hertzog; Rocha, Clarissa Ribeiro; Damasceno, Débora Cristina; Morceli, Glilciane; Rudge, Marilza Vieira; Bevilacqua, Estela Maris Andrade Forell ; Calderon, Iracema Mattos Paranhos
Source: BioMed Research International. Unidade: ICB
Subjects: HISTOLOGIA, FETO, DANO AO DNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORELI, Jusciele Brogin et al. DNA damage and its cellular response in mother and fetus exposed to hyperglycemic environment. BioMed Research International, v. 2014, p. 1-9, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/676758. Acesso em: 06 set. 2024.
APA
Moreli, J. B., Santos, J. H., Rocha, C. R., Damasceno, D. C., Morceli, G., Rudge, M. V., et al. (2014). DNA damage and its cellular response in mother and fetus exposed to hyperglycemic environment. BioMed Research International, 2014, 1-9. doi:10.1155/2014/676758
NLM
Moreli JB, Santos JH, Rocha CR, Damasceno DC, Morceli G, Rudge MV, Bevilacqua EMAF, Calderon IMP. DNA damage and its cellular response in mother and fetus exposed to hyperglycemic environment [Internet]. BioMed Research International. 2014 ; 2014 1-9.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/676758
Vancouver
Moreli JB, Santos JH, Rocha CR, Damasceno DC, Morceli G, Rudge MV, Bevilacqua EMAF, Calderon IMP. DNA damage and its cellular response in mother and fetus exposed to hyperglycemic environment [Internet]. BioMed Research International. 2014 ; 2014 1-9.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/676758
Artigo de periodico
Topical verapamil as a scar modulator (2014)
Boggio, Ricardo Frota; Boggio, Leonardo Frota; Galvão, Bruno Luiz; Santelli, Glaucia Maria Machado
Source: Aesthetic Plastic Surgery. Unidade: ICB
Subjects: HISTOLOGIA, CANAIS DE CÁLCIO, VASODILATAÇÃO, FARMACOS (SISTEMA CARDIOVASCULAR)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOGGIO, Ricardo Frota et al. Topical verapamil as a scar modulator. Aesthetic Plastic Surgery, v. 38, n. 5, p. 968-975, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00266-014-0400-9. Acesso em: 06 set. 2024.
APA
Boggio, R. F., Boggio, L. F., Galvão, B. L., & Santelli, G. M. M. (2014). Topical verapamil as a scar modulator. Aesthetic Plastic Surgery, 38( 5), 968-975. doi:10.1007/s00266-014-0400-9
NLM
Boggio RF, Boggio LF, Galvão BL, Santelli GMM. Topical verapamil as a scar modulator [Internet]. Aesthetic Plastic Surgery. 2014 ; 38( 5): 968-975.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s00266-014-0400-9
Vancouver
Boggio RF, Boggio LF, Galvão BL, Santelli GMM. Topical verapamil as a scar modulator [Internet]. Aesthetic Plastic Surgery. 2014 ; 38( 5): 968-975.[citado 2024 set. 06 ] Available from: https://doi.org/10.1007/s00266-014-0400-9
Artigo de periodico
MicroRNA deregulation in anaplastic thyroid cancer biology (2014)
Fuziwara, Cesar Seigi; Kimura, Edna Teruko
Source: International Journal of Endocrinology. Unidade: ICB
Subjects: HISTOLOGIA, NEOPLASIAS DA TIREÓIDE, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FUZIWARA, Cesar Seigi e KIMURA, Edna Teruko. MicroRNA deregulation in anaplastic thyroid cancer biology. International Journal of Endocrinology, v. 2014, p. 1-8, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/743450. Acesso em: 06 set. 2024.
APA
Fuziwara, C. S., & Kimura, E. T. (2014). MicroRNA deregulation in anaplastic thyroid cancer biology. International Journal of Endocrinology, 2014, 1-8. doi:10.1155/2014/743450
NLM
Fuziwara CS, Kimura ET. MicroRNA deregulation in anaplastic thyroid cancer biology [Internet]. International Journal of Endocrinology. 2014 ; 2014 1-8.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/743450
Vancouver
Fuziwara CS, Kimura ET. MicroRNA deregulation in anaplastic thyroid cancer biology [Internet]. International Journal of Endocrinology. 2014 ; 2014 1-8.[citado 2024 set. 06 ] Available from: https://doi.org/10.1155/2014/743450

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