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Artigo de periodico
The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells (2018)
Silveira, Elaine; Cavalcante, Isadora Pontes; Kremer, Jean Lucas; Mendonça, Pedro Omori Ribeiro de; Lotfi, Claudimara Ferini Pacicco
Source: Cancer Cell International. Unidade: ICB
Subjects: CARCINOMA, QUIMIOTERÁPICOS, NEOPLASIAS DO CÓRTEX SUPRARRENAL, CÓRTEX ADRENAL, CITOTOXINAS, FÁRMACOS
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ABNT
SILVEIRA, Elaine et al. The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells. Cancer Cell International, v. 18, p. 16 , 2018Tradução . . Disponível em: https://doi.org/10.1186/s12935-018-0527-x. Acesso em: 23 abr. 2024.
APA
Silveira, E., Cavalcante, I. P., Kremer, J. L., Mendonça, P. O. R. de, & Lotfi, C. F. P. (2018). The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells. Cancer Cell International, 18, 16 . doi:10.1186/s12935-018-0527-x
NLM
Silveira E, Cavalcante IP, Kremer JL, Mendonça POR de, Lotfi CFP. The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells [Internet]. Cancer Cell International. 2018 ;18 16 .[citado 2024 abr. 23 ] Available from: https://doi.org/10.1186/s12935-018-0527-x
Vancouver
Silveira E, Cavalcante IP, Kremer JL, Mendonça POR de, Lotfi CFP. The tyrosine kinase inhibitor nilotinib is more efficient than mitotane in decreasing cell viability in spheroids prepared from adrenocortical carcinoma cells [Internet]. Cancer Cell International. 2018 ;18 16 .[citado 2024 abr. 23 ] Available from: https://doi.org/10.1186/s12935-018-0527-x
Artigo de periodico
The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior (2018)
Teixeira, Lívia Clemente Motta ; Nils, Aline Vilar Machado; Battagello, Daniella Sabino ; Diniz, Giovanne Baroni ; Silva, Jéssica Andrade da ; Silva Junior, José Sinésio; Matos, Raphael Afonso ; Amaral, Fernanda Gaspar do; Xavier, Gilberto Fernando ; Bittencourt, Jackson Cioni ; Reiter, Russel J. ; Lucassen, Paul John ; Korosi, Aniko ; Cipolla Neto, José
Source: Hormones and Behavior. Unidades: IB, ICB
Subjects: MELATONINA, GLÂNDULA PINEAL, GRAVIDEZ, FOTOPERIODISMO, FETO, PARÂMETROS DE RESPOSTAS, LACTAÇÃO
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TEIXEIRA, Lívia Clemente Motta et al. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior. Hormones and Behavior, v. 105, p. 146-156, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2018.08.006. Acesso em: 23 abr. 2024.
APA
Teixeira, L. C. M., Nils, A. V. M., Battagello, D. S., Diniz, G. B., Silva, J. A. da, Silva Junior, J. S., et al. (2018). The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior. Hormones and Behavior, 105, 146-156. doi:10.1016/j.yhbeh.2018.08.006
NLM
Teixeira LCM, Nils AVM, Battagello DS, Diniz GB, Silva JA da, Silva Junior JS, Matos RA, Amaral FG do, Xavier GF, Bittencourt JC, Reiter RJ, Lucassen PJ, Korosi A, Cipolla Neto J. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior [Internet]. Hormones and Behavior. 2018 ; 105 146-156.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.yhbeh.2018.08.006
Vancouver
Teixeira LCM, Nils AVM, Battagello DS, Diniz GB, Silva JA da, Silva Junior JS, Matos RA, Amaral FG do, Xavier GF, Bittencourt JC, Reiter RJ, Lucassen PJ, Korosi A, Cipolla Neto J. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior [Internet]. Hormones and Behavior. 2018 ; 105 146-156.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.yhbeh.2018.08.006
Artigo de periodico
Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities (2018)
Kiyuna, Ligia Akemi; Albuquerque, Rudá Prestes e; Chen, Che-Hong; Rosen, Daria Mochly; Ferreira, Julio Cesar Batista
Source: Free Radical Biology and Medicine. Unidade: ICB
Subjects: BIOENERGÉTICA, MITOCÔNDRIAS, ALDEÍDOS, INSUFICIÊNCIA CARDÍACA, TREINAMENTO FÍSICO, ESTRESSE OXIDATIVO, BIOENERGÉTICA
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KIYUNA, Ligia Akemi et al. Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities. Free Radical Biology and Medicine, v. 129, p. 155-168, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2018.09.019. Acesso em: 23 abr. 2024.
APA
Kiyuna, L. A., Albuquerque, R. P. e, Chen, C. -H., Rosen, D. M., & Ferreira, J. C. B. (2018). Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities. Free Radical Biology and Medicine, 129, 155-168. doi:10.1016/j.freeradbiomed.2018.09.019
NLM
Kiyuna LA, Albuquerque RP e, Chen C-H, Rosen DM, Ferreira JCB. Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities [Internet]. Free Radical Biology and Medicine. 2018 ; 129 155-168.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.09.019
Vancouver
Kiyuna LA, Albuquerque RP e, Chen C-H, Rosen DM, Ferreira JCB. Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities [Internet]. Free Radical Biology and Medicine. 2018 ; 129 155-168.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.09.019
Artigo de periodico
TCF21/POD-1, a transcritional regulator of SF-1/NR5A1, as a potential prognosis marker in adult and pediatric adrenocortical tumors (2018)
Passaia, Bárbara dos Santos; Dias, Matheus Henrique dos Santos; Kremer, Jean Lucas; Antonini, Sonir Roberto Rauber; Almeida, Madson Queiroz de; Fragoso, Maria Candida Barisson Villares; Lotfi, Claudimara Ferini Pacicco
Source: Frontiers in Endocrinology. Unidades: FMRP, ICB
Subjects: CÓRTEX ADRENAL, FATOR DE TRANSCRIÇÃO AP-1, CÉLULAS CULTIVADAS DE TUMOR, MARCADOR MOLECULAR, ADENOMA, CARCINOMA, EXPRESSÃO GÊNICA
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PASSAIA, Bárbara dos Santos et al. TCF21/POD-1, a transcritional regulator of SF-1/NR5A1, as a potential prognosis marker in adult and pediatric adrenocortical tumors. Frontiers in Endocrinology, v. 9, p. 14 , 2018Tradução . . Disponível em: https://doi.org/10.3389/fendo.2018.00038. Acesso em: 23 abr. 2024.
APA
Passaia, B. dos S., Dias, M. H. dos S., Kremer, J. L., Antonini, S. R. R., Almeida, M. Q. de, Fragoso, M. C. B. V., & Lotfi, C. F. P. (2018). TCF21/POD-1, a transcritional regulator of SF-1/NR5A1, as a potential prognosis marker in adult and pediatric adrenocortical tumors. Frontiers in Endocrinology, 9, 14 . doi:10.3389/fendo.2018.00038
NLM
Passaia B dos S, Dias MH dos S, Kremer JL, Antonini SRR, Almeida MQ de, Fragoso MCBV, Lotfi CFP. TCF21/POD-1, a transcritional regulator of SF-1/NR5A1, as a potential prognosis marker in adult and pediatric adrenocortical tumors [Internet]. Frontiers in Endocrinology. 2018 ; 9 14 .[citado 2024 abr. 23 ] Available from: https://doi.org/10.3389/fendo.2018.00038
Vancouver
Passaia B dos S, Dias MH dos S, Kremer JL, Antonini SRR, Almeida MQ de, Fragoso MCBV, Lotfi CFP. TCF21/POD-1, a transcritional regulator of SF-1/NR5A1, as a potential prognosis marker in adult and pediatric adrenocortical tumors [Internet]. Frontiers in Endocrinology. 2018 ; 9 14 .[citado 2024 abr. 23 ] Available from: https://doi.org/10.3389/fendo.2018.00038
Artigo de periodico
Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context (2018)
Bindi, Ricardo Passoni; Baldo, Marcus Vinícius Chrysóstomo ; Canteras, Newton Sabino
Source: Behavioural Brain Research. Unidade: ICB
Subjects: ANIMAIS PREDADORES, MEDO, MEMÓRIA, NEUROANATOMIA, AMÍGDALA DO CEREBELO, HIPOTÁLAMO
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ABNT
BINDI, Ricardo Passoni e BALDO, Marcus Vinícius Chrysóstomo e CANTERAS, Newton Sabino. Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context. Behavioural Brain Research, v. 342, p. 51-56, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2018.01.016. Acesso em: 23 abr. 2024.
APA
Bindi, R. P., Baldo, M. V. C., & Canteras, N. S. (2018). Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context. Behavioural Brain Research, 342, 51-56. doi:10.1016/j.bbr.2018.01.016
NLM
Bindi RP, Baldo MVC, Canteras NS. Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context [Internet]. Behavioural Brain Research. 2018 ; 342 51-56.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.bbr.2018.01.016
Vancouver
Bindi RP, Baldo MVC, Canteras NS. Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context [Internet]. Behavioural Brain Research. 2018 ; 342 51-56.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.bbr.2018.01.016
Artigo de periodico
Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model (2018)
Santos, Fabio Martinez dos; Silva, Joyce Teixeira; Rocha, Igor Rafael Correia; Martins, Daniel Oliveira; Chacur, Marucia
Source: Brain Research. Unidade: ICB
Subjects: NERVO CIÁTICO, SUBSTÂNCIA P, DOR, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, NEUROPEPTÍDEOS, FISIOTERAPIA, GÂNGLIOS, WESTERN BLOTTING
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ABNT
SANTOS, Fabio Martinez dos et al. Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model. Brain Research, v. 1687, p. 60-65, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2018.02.034. Acesso em: 23 abr. 2024.
APA
Santos, F. M. dos, Silva, J. T., Rocha, I. R. C., Martins, D. O., & Chacur, M. (2018). Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model. Brain Research, 1687, 60-65. doi:10.1016/j.brainres.2018.02.034
NLM
Santos FM dos, Silva JT, Rocha IRC, Martins DO, Chacur M. Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model [Internet]. Brain Research. 2018 ; 1687 60-65.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.brainres.2018.02.034
Vancouver
Santos FM dos, Silva JT, Rocha IRC, Martins DO, Chacur M. Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model [Internet]. Brain Research. 2018 ; 1687 60-65.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.brainres.2018.02.034
Artigo de periodico
Morphological characterization of the myenteric plexus of the ileum and distal colon of dogs affected by muscular dystrophy (2018)
Schäfer, Bárbara Tavares; Silveira, Mariana Póvoa; Palombit, Kelly; Mendes, Cristina Eusébio; Watanabe, Ii-Sei; Miglino, Maria Angélica; Castelucci, Patricia
Source: The Anatomical Record. Unidades: ICB, FMVZ
Subjects: CÃES, DISTROFIA MUSCULAR ANIMAL, INTESTINO DE ANIMAL, PLEXO MIENTÉRICO, CÃES, SISTEMA NERVOSO ENTÉRICO, IMUNOHISTOQUÍMICA, ÍLEO
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ABNT
SCHÄFER, Bárbara Tavares et al. Morphological characterization of the myenteric plexus of the ileum and distal colon of dogs affected by muscular dystrophy. The Anatomical Record, v. 301, n. 4, p. 673-685, 2018Tradução . . Disponível em: https://doi.org/10.1002/ar.23708. Acesso em: 23 abr. 2024.
APA
Schäfer, B. T., Silveira, M. P., Palombit, K., Mendes, C. E., Watanabe, I. -S., Miglino, M. A., & Castelucci, P. (2018). Morphological characterization of the myenteric plexus of the ileum and distal colon of dogs affected by muscular dystrophy. The Anatomical Record, 301( 4), 673-685. doi:10.1002/ar.23708
NLM
Schäfer BT, Silveira MP, Palombit K, Mendes CE, Watanabe I-S, Miglino MA, Castelucci P. Morphological characterization of the myenteric plexus of the ileum and distal colon of dogs affected by muscular dystrophy [Internet]. The Anatomical Record. 2018 ; 301( 4): 673-685.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1002/ar.23708
Vancouver
Schäfer BT, Silveira MP, Palombit K, Mendes CE, Watanabe I-S, Miglino MA, Castelucci P. Morphological characterization of the myenteric plexus of the ileum and distal colon of dogs affected by muscular dystrophy [Internet]. The Anatomical Record. 2018 ; 301( 4): 673-685.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1002/ar.23708
Artigo de periodico
Influence of the anteromedial thalamus on social defeat-associated contextual fear memory (2018)
Rangel Júnior, Miguel José; Baldo, Marcus Vinícius Chrysóstomo ; Canteras, Newton Sabino
Source: Behavioural Brain Research. Unidade: ICB
Subjects: MEDO, MEMÓRIA, NÚCLEOS TALÂMICOS, NEUROANATOMIA, TÁLAMO
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ABNT
RANGEL JÚNIOR, Miguel José e BALDO, Marcus Vinícius Chrysóstomo e CANTERAS, Newton Sabino. Influence of the anteromedial thalamus on social defeat-associated contextual fear memory. Behavioural Brain Research, v. 339, p. 269-277, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2017.10.038. Acesso em: 23 abr. 2024.
APA
Rangel Júnior, M. J., Baldo, M. V. C., & Canteras, N. S. (2018). Influence of the anteromedial thalamus on social defeat-associated contextual fear memory. Behavioural Brain Research, 339, 269-277. doi:10.1016/j.bbr.2017.10.038
NLM
Rangel Júnior MJ, Baldo MVC, Canteras NS. Influence of the anteromedial thalamus on social defeat-associated contextual fear memory [Internet]. Behavioural Brain Research. 2018 ; 339 269-277.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.bbr.2017.10.038
Vancouver
Rangel Júnior MJ, Baldo MVC, Canteras NS. Influence of the anteromedial thalamus on social defeat-associated contextual fear memory [Internet]. Behavioural Brain Research. 2018 ; 339 269-277.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.bbr.2017.10.038
Artigo de periodico
Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure (2017)
Campos, Juliane Cruz; Queliconi, Bruno Barros; Bozi, Luiz Henrique Marchesi; Bechara, Luiz Roberto Grassmann; Dourado, Paulo Magno Martins; Andres, Allen M; Jannig, Paulo Roberto; Gomes, Katia Maria Sampaio; Zambelli, Vanessa Olzon; Resende, Cibele Rocha; Fonseca, Silvia Carolina Guatimosim; Brum, Patricia Chakur; Mochly-Rosen, Daria; Gottlieb, Roberta A; Kowaltowski, Alicia Juliana ; Ferreira, Julio Cesar Batista
Source: Autophagy. Unidades: EEFE, IQ, ICB
Subjects: DOENÇAS CARDIOVASCULARES, EXERCÍCIO FÍSICO, MITOCÔNDRIAS, BIOENERGÉTICA
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CAMPOS, Juliane Cruz et al. Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure. Autophagy, v. 13, n. 8, p. 1304-1317, 2017Tradução . . Disponível em: https://doi.org/10.1080/15548627.2017.1325062. Acesso em: 23 abr. 2024.
APA
Campos, J. C., Queliconi, B. B., Bozi, L. H. M., Bechara, L. R. G., Dourado, P. M. M., Andres, A. M., et al. (2017). Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure. Autophagy, 13( 8), 1304-1317. doi:10.1080/15548627.2017.1325062
NLM
Campos JC, Queliconi BB, Bozi LHM, Bechara LRG, Dourado PMM, Andres AM, Jannig PR, Gomes KMS, Zambelli VO, Resende CR, Fonseca SCG, Brum PC, Mochly-Rosen D, Gottlieb RA, Kowaltowski AJ, Ferreira JCB. Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure [Internet]. Autophagy. 2017 ; 13( 8): 1304-1317.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1080/15548627.2017.1325062
Vancouver
Campos JC, Queliconi BB, Bozi LHM, Bechara LRG, Dourado PMM, Andres AM, Jannig PR, Gomes KMS, Zambelli VO, Resende CR, Fonseca SCG, Brum PC, Mochly-Rosen D, Gottlieb RA, Kowaltowski AJ, Ferreira JCB. Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure [Internet]. Autophagy. 2017 ; 13( 8): 1304-1317.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1080/15548627.2017.1325062
Artigo de periodico
Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy (2018)
Campos, Juliane Cruz; Baehr, Leslie M; Gomes, Kátia Maria Sampaio; Bechara, Luiz Roberto Grassmann; Voltarelli, Vanessa Azevedo; Bozi, Luiz Henrique Marchesi; Ribeiro, Márcio Augusto Campos; Ferreira, Nikolas Dresch; Moreira, José Bianco Nascimento; Brum, Patrícia Chakur ; Bodine, Sue C; Ferreira, Julio Cesar Batista
Source: Scientific Reports. Unidades: EEFE, ICB
Subjects: DOENÇAS NEUROMUSCULARES, EXERCÍCIO FÍSICO, TREINAMENTO AERÓBIO, MÚSCULO ESQUELÉTICO, ATROFIA MUSCULAR, NEUROBIOLOGIA, RATOS
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ABNT
CAMPOS, Juliane Cruz et al. Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy. Scientific Reports, v. 8, n. 1, p. 14 , 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-30365-1. Acesso em: 23 abr. 2024.
APA
Campos, J. C., Baehr, L. M., Gomes, K. M. S., Bechara, L. R. G., Voltarelli, V. A., Bozi, L. H. M., et al. (2018). Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy. Scientific Reports, 8( 1), 14 . doi:10.1038/s41598-018-30365-1
NLM
Campos JC, Baehr LM, Gomes KMS, Bechara LRG, Voltarelli VA, Bozi LHM, Ribeiro MAC, Ferreira ND, Moreira JBN, Brum PC, Bodine SC, Ferreira JCB. Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy [Internet]. Scientific Reports. 2018 ; 8( 1): 14 .[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/s41598-018-30365-1
Vancouver
Campos JC, Baehr LM, Gomes KMS, Bechara LRG, Voltarelli VA, Bozi LHM, Ribeiro MAC, Ferreira ND, Moreira JBN, Brum PC, Bodine SC, Ferreira JCB. Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy [Internet]. Scientific Reports. 2018 ; 8( 1): 14 .[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/s41598-018-30365-1
Artigo de periodico
Cardioprotection induced by a brief exposure to acetaldehyde: role of aldehyde dehydrogenase 2 (2018)
Ueta, Cintia Bagne; Campos, Juliane Cruz; Albuquerque, Rudá Prestes e; Lima, Vanessa Morais; Disatnik, Marie-Hélène; Sanchez, Angelica Bianchini; Chen, Che-Hong; Medeiros, Marisa Helena Gennari de ; Yang, Wenjin; Rosen, Daria Mochly; Ferreira, Julio Cesar Batista
Source: Cardiovascular Research. Unidades: IQ, ICB
Subjects: ISQUEMIA, ÁLCOOL, MITOCÔNDRIAS, DOENÇAS CARDIOVASCULARES
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ABNT
UETA, Cintia Bagne et al. Cardioprotection induced by a brief exposure to acetaldehyde: role of aldehyde dehydrogenase 2. Cardiovascular Research, v. 114, n. 7, p. 1006-1015, 2018Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvy070. Acesso em: 23 abr. 2024.
APA
Ueta, C. B., Campos, J. C., Albuquerque, R. P. e, Lima, V. M., Disatnik, M. -H., Sanchez, A. B., et al. (2018). Cardioprotection induced by a brief exposure to acetaldehyde: role of aldehyde dehydrogenase 2. Cardiovascular Research, 114( 7), 1006-1015. doi:10.1093/cvr/cvy070
NLM
Ueta CB, Campos JC, Albuquerque RP e, Lima VM, Disatnik M-H, Sanchez AB, Chen C-H, Medeiros MHG de, Yang W, Rosen DM, Ferreira JCB. Cardioprotection induced by a brief exposure to acetaldehyde: role of aldehyde dehydrogenase 2 [Internet]. Cardiovascular Research. 2018 ; 114( 7): 1006-1015.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1093/cvr/cvy070
Vancouver
Ueta CB, Campos JC, Albuquerque RP e, Lima VM, Disatnik M-H, Sanchez AB, Chen C-H, Medeiros MHG de, Yang W, Rosen DM, Ferreira JCB. Cardioprotection induced by a brief exposure to acetaldehyde: role of aldehyde dehydrogenase 2 [Internet]. Cardiovascular Research. 2018 ; 114( 7): 1006-1015.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1093/cvr/cvy070

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