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  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: ESTRESSE OXIDATIVO, MITOCÔNDRIAS

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    • ABNT

      SIGOLO, Carlos Alexandre Oliveira et al. trans, trans-2,4-decadienal induces mitochondrial dysfunction and oxidative stress. Journal of Bioenergetics and Biomembranes, v. 40, n. 2, p. 103-109, 2008Tradução . . Disponível em: https://doi.org/10.1007/s10863-008-9137-y. Acesso em: 19 abr. 2024.
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      Sigolo, C. A. O., Di Mascio, P., Kowaltowski, A. J., Garcia, C. C. M., & Medeiros, M. H. G. de. (2008). trans, trans-2,4-decadienal induces mitochondrial dysfunction and oxidative stress. Journal of Bioenergetics and Biomembranes, 40( 2), 103-109. doi:10.1007/s10863-008-9137-y
    • NLM

      Sigolo CAO, Di Mascio P, Kowaltowski AJ, Garcia CCM, Medeiros MHG de. trans, trans-2,4-decadienal induces mitochondrial dysfunction and oxidative stress [Internet]. Journal of Bioenergetics and Biomembranes. 2008 ;40( 2): 103-109.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-008-9137-y
    • Vancouver

      Sigolo CAO, Di Mascio P, Kowaltowski AJ, Garcia CCM, Medeiros MHG de. trans, trans-2,4-decadienal induces mitochondrial dysfunction and oxidative stress [Internet]. Journal of Bioenergetics and Biomembranes. 2008 ;40( 2): 103-109.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-008-9137-y
  • Source: Journal of Bioenergetics and Biomembranes. Unidades: FMRP, FCFRP

    Subjects: CÉLULAS CULTIVADAS, MITOCÔNDRIAS, METABOLISMO DE GORDURA, FÍGADO GORDUROSO

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      DECHANDT, Carlos R. P. et al. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes. Journal of Bioenergetics and Biomembranes, v. 49, n. 5, p. 399-411, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10863-017-9725-9. Acesso em: 19 abr. 2024.
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      Dechandt, C. R. P., Zuccolotto-dos-Reis, F. H., Teodoro, B. G., Fernandes, A. M. A. P., Eberlin, M. N., Kettelhut, I. do C., et al. (2017). Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes. Journal of Bioenergetics and Biomembranes, 49( 5), 399-411. doi:10.1007/s10863-017-9725-9
    • NLM

      Dechandt CRP, Zuccolotto-dos-Reis FH, Teodoro BG, Fernandes AMAP, Eberlin MN, Kettelhut I do C, Curti C, Alberici LC. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 5): 399-411.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-017-9725-9
    • Vancouver

      Dechandt CRP, Zuccolotto-dos-Reis FH, Teodoro BG, Fernandes AMAP, Eberlin MN, Kettelhut I do C, Curti C, Alberici LC. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 5): 399-411.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-017-9725-9
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IFSC

    Subjects: MANGANÊS, MITOCÔNDRIAS, RATOS (EXPERIMENTOS), ENZIMAS, ANTIOXIDANTES (ATIVIDADE)

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      CHAVES, Juliana C. Araujo et al. Towards the mechanisms involved in the antioxidant action of 'Mn POT. III' [meso-tetrakis(4-N-methyl pyridinium) porphyrin] in mitochondria. Journal of Bioenergetics and Biomembranes, v. 43, n. 6, p. 663-671, 2011Tradução . . Disponível em: https://doi.org/10.1017/s10863-011-9382-3. Acesso em: 19 abr. 2024.
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      Chaves, J. C. A., Yokomizo, C. H., Kawai, C., Mugnol, K. C. U., Prieto, T., Nascimento, O. R., & Nantes, I. L. (2011). Towards the mechanisms involved in the antioxidant action of 'Mn POT. III' [meso-tetrakis(4-N-methyl pyridinium) porphyrin] in mitochondria. Journal of Bioenergetics and Biomembranes, 43( 6), 663-671. doi:10.1017/s10863-011-9382-3
    • NLM

      Chaves JCA, Yokomizo CH, Kawai C, Mugnol KCU, Prieto T, Nascimento OR, Nantes IL. Towards the mechanisms involved in the antioxidant action of 'Mn POT. III' [meso-tetrakis(4-N-methyl pyridinium) porphyrin] in mitochondria [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 6): 663-671.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1017/s10863-011-9382-3
    • Vancouver

      Chaves JCA, Yokomizo CH, Kawai C, Mugnol KCU, Prieto T, Nascimento OR, Nantes IL. Towards the mechanisms involved in the antioxidant action of 'Mn POT. III' [meso-tetrakis(4-N-methyl pyridinium) porphyrin] in mitochondria [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 6): 663-671.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1017/s10863-011-9382-3
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: DANO AO DNA, DNA MITOCONDRIAL

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      COSTA, R. A. P et al. The role of mitochondrial DNA damage in the citotoxicity of reactive oxygen species. Journal of Bioenergetics and Biomembranes, v. 43, n. 1, p. 25-29, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10863-011-9329-8. Acesso em: 19 abr. 2024.
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      Costa, R. A. P., Romagna, C. D., Pereira, J. L., & Souza-Pinto, N. C. de. (2011). The role of mitochondrial DNA damage in the citotoxicity of reactive oxygen species. Journal of Bioenergetics and Biomembranes, 43( 1), 25-29. doi:10.1007/s10863-011-9329-8
    • NLM

      Costa RAP, Romagna CD, Pereira JL, Souza-Pinto NC de. The role of mitochondrial DNA damage in the citotoxicity of reactive oxygen species [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 1): 25-29.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9329-8
    • Vancouver

      Costa RAP, Romagna CD, Pereira JL, Souza-Pinto NC de. The role of mitochondrial DNA damage in the citotoxicity of reactive oxygen species [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 1): 25-29.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9329-8
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: ICB

    Subjects: PARASITOLOGIA, TRYPANOSOMA CRUZI, DOENÇA DE CHAGAS, AMINOÁCIDOS

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      BARISÓN, M. J. et al. The active transport of histidine and its role in ATP production in Trypanosoma cruzi. Journal of Bioenergetics and Biomembranes, v. 48, n. 4, p. 437-449, 2016Tradução . . Disponível em: https://doi.org/10.1007/s10863-016-9665-9. Acesso em: 19 abr. 2024.
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      Barisón, M.  J., Damasceno, F.  S.  , Mantilla, B.  S.  , & Silber, A. M. (2016). The active transport of histidine and its role in ATP production in Trypanosoma cruzi. Journal of Bioenergetics and Biomembranes, 48( 4), 437-449. doi:10.1007/s10863-016-9665-9
    • NLM

      Barisón M J, Damasceno F S , Mantilla B S , Silber AM. The active transport of histidine and its role in ATP production in Trypanosoma cruzi [Internet]. Journal of Bioenergetics and Biomembranes. 2016 ; 48( 4): 437-449.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-016-9665-9
    • Vancouver

      Barisón M J, Damasceno F S , Mantilla B S , Silber AM. The active transport of histidine and its role in ATP production in Trypanosoma cruzi [Internet]. Journal of Bioenergetics and Biomembranes. 2016 ; 48( 4): 437-449.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-016-9665-9
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: FCFRP

    Subjects: ASPERGILLUS, MACRÓFAGOS, ESTRESSE OXIDATIVO

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      MAGNANI, Taisa et al. Silencing of mitochondrial alternative oxidase gene of Aspergillus fumigatus enhances reactive oxygen species production and killing of the fungus by macrophages. Journal of Bioenergetics and Biomembranes, v. 40, n. 6, p. 631-636, 2008Tradução . . Disponível em: https://doi.org/10.1007/s10863-008-9191-5. Acesso em: 19 abr. 2024.
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      Magnani, T., Soriani, F. M., Martins, V. de P., Policarpo, A. C. de F., Sorgi, C. A., Faccioli, L. H., et al. (2008). Silencing of mitochondrial alternative oxidase gene of Aspergillus fumigatus enhances reactive oxygen species production and killing of the fungus by macrophages. Journal of Bioenergetics and Biomembranes, 40( 6), 631-636. doi:10.1007/s10863-008-9191-5
    • NLM

      Magnani T, Soriani FM, Martins V de P, Policarpo AC de F, Sorgi CA, Faccioli LH, Curti C, Uyemura SA. Silencing of mitochondrial alternative oxidase gene of Aspergillus fumigatus enhances reactive oxygen species production and killing of the fungus by macrophages [Internet]. Journal of Bioenergetics and Biomembranes. 2008 ; 40( 6): 631-636.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-008-9191-5
    • Vancouver

      Magnani T, Soriani FM, Martins V de P, Policarpo AC de F, Sorgi CA, Faccioli LH, Curti C, Uyemura SA. Silencing of mitochondrial alternative oxidase gene of Aspergillus fumigatus enhances reactive oxygen species production and killing of the fungus by macrophages [Internet]. Journal of Bioenergetics and Biomembranes. 2008 ; 40( 6): 631-636.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-008-9191-5
  • Source: Journal of Bioenergetics and Biomembranes. Unidades: ICB, IQ

    Subjects: ENVELHECIMENTO, MITOCÔNDRIAS

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      TAHARA, Erich Birelli et al. Respiratory and TCA cycle activities affect S. cerevisiae lifespan, response to caloric restriction and mtDNA stability. Journal of Bioenergetics and Biomembranes, v. 43, n. 5, p. 483-491, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10863-011-9377-0. Acesso em: 19 abr. 2024.
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      Tahara, E. B., Cezário, K., Souza-Pinto, N. C. de, Barros, M. H. de, & Kowaltowski, A. J. (2011). Respiratory and TCA cycle activities affect S. cerevisiae lifespan, response to caloric restriction and mtDNA stability. Journal of Bioenergetics and Biomembranes, 43( 5), 483-491. doi:10.1007/s10863-011-9377-0
    • NLM

      Tahara EB, Cezário K, Souza-Pinto NC de, Barros MH de, Kowaltowski AJ. Respiratory and TCA cycle activities affect S. cerevisiae lifespan, response to caloric restriction and mtDNA stability [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 5): 483-491.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9377-0
    • Vancouver

      Tahara EB, Cezário K, Souza-Pinto NC de, Barros MH de, Kowaltowski AJ. Respiratory and TCA cycle activities affect S. cerevisiae lifespan, response to caloric restriction and mtDNA stability [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 5): 483-491.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9377-0
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: BIOQUÍMICA, APOPTOSE, ÓXIDO NÍTRICO, CÁLCIO

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      FISKUM, Gary et al. Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress. Journal of Bioenergetics and Biomembranes, v. 36, n. 4, p. 347-352, 2004Tradução . . Acesso em: 19 abr. 2024.
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      Fiskum, G., Rosenthal, R. E., Vereczki, V., Martin, E., Hoffman, G. E., Chinopoulos, C., & Kowaltowski, A. J. (2004). Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress. Journal of Bioenergetics and Biomembranes, 36( 4), 347-352.
    • NLM

      Fiskum G, Rosenthal RE, Vereczki V, Martin E, Hoffman GE, Chinopoulos C, Kowaltowski AJ. Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress. Journal of Bioenergetics and Biomembranes. 2004 ; 36( 4): 347-352.[citado 2024 abr. 19 ]
    • Vancouver

      Fiskum G, Rosenthal RE, Vereczki V, Martin E, Hoffman GE, Chinopoulos C, Kowaltowski AJ. Protection against ischemic brain injury by inhibition of mitochondrial oxidative stress. Journal of Bioenergetics and Biomembranes. 2004 ; 36( 4): 347-352.[citado 2024 abr. 19 ]
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQSC

    Assunto: BIOFÍSICA

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      NEPOMUCENO, M. F. e TABAK, Marcel e VERCESI, A. E. Opposite effects of Mn(III) and Fe(III) forms of meso-Tetrakis(4-N-methyl pyridiniumyl) Porphyrins on isolated rat liver mitochondria. Journal of Bioenergetics and Biomembranes, v. 34, n. 1, p. 41-47, 2002Tradução . . Acesso em: 19 abr. 2024.
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      Nepomuceno, M. F., Tabak, M., & Vercesi, A. E. (2002). Opposite effects of Mn(III) and Fe(III) forms of meso-Tetrakis(4-N-methyl pyridiniumyl) Porphyrins on isolated rat liver mitochondria. Journal of Bioenergetics and Biomembranes, 34( 1), 41-47.
    • NLM

      Nepomuceno MF, Tabak M, Vercesi AE. Opposite effects of Mn(III) and Fe(III) forms of meso-Tetrakis(4-N-methyl pyridiniumyl) Porphyrins on isolated rat liver mitochondria. Journal of Bioenergetics and Biomembranes. 2002 ; 34( 1): 41-47.[citado 2024 abr. 19 ]
    • Vancouver

      Nepomuceno MF, Tabak M, Vercesi AE. Opposite effects of Mn(III) and Fe(III) forms of meso-Tetrakis(4-N-methyl pyridiniumyl) Porphyrins on isolated rat liver mitochondria. Journal of Bioenergetics and Biomembranes. 2002 ; 34( 1): 41-47.[citado 2024 abr. 19 ]
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: BIOQUÍMICA, HIDRÓLISE, MITOCÔNDRIAS

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      BELISLE, Eduardo e KOWALTOWSKI, Alicia Juliana. Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis. Journal of Bioenergetics and Biomembranes, v. 34, n. 4, p. 285-298, 2002Tradução . . Acesso em: 19 abr. 2024.
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      Belisle, E., & Kowaltowski, A. J. (2002). Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis. Journal of Bioenergetics and Biomembranes, 34( 4), 285-298.
    • NLM

      Belisle E, Kowaltowski AJ. Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis. Journal of Bioenergetics and Biomembranes. 2002 ; 34( 4): 285-298.[citado 2024 abr. 19 ]
    • Vancouver

      Belisle E, Kowaltowski AJ. Opening of mitochondrial 'K POT. +' channels increases ischemic ATP levels by preventing hydrolysis. Journal of Bioenergetics and Biomembranes. 2002 ; 34( 4): 285-298.[citado 2024 abr. 19 ]
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: MITOCÔNDRIAS, ISQUEMIA, RADICAIS LIVRES

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      CARREIRA, Raquel de Souza et al. Nicorandil protects cardiac mitochondria against permeability transition induced by ischemia-reperfusion. Journal of Bioenergetics and Biomembranes, v. 40, n. 2, p. 95-102, 2008Tradução . . Disponível em: https://doi.org/10.1007/s10863-008-9133-2. Acesso em: 19 abr. 2024.
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      Carreira, R. de S., Monteiro, P., Kowaltowski, A. J., Gonçalves, L. M., & Providência, L. A. (2008). Nicorandil protects cardiac mitochondria against permeability transition induced by ischemia-reperfusion. Journal of Bioenergetics and Biomembranes, 40( 2), 95-102. doi:10.1007/s10863-008-9133-2
    • NLM

      Carreira R de S, Monteiro P, Kowaltowski AJ, Gonçalves LM, Providência LA. Nicorandil protects cardiac mitochondria against permeability transition induced by ischemia-reperfusion [Internet]. Journal of Bioenergetics and Biomembranes. 2008 ;40( 2): 95-102.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-008-9133-2
    • Vancouver

      Carreira R de S, Monteiro P, Kowaltowski AJ, Gonçalves LM, Providência LA. Nicorandil protects cardiac mitochondria against permeability transition induced by ischemia-reperfusion [Internet]. Journal of Bioenergetics and Biomembranes. 2008 ;40( 2): 95-102.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-008-9133-2
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: MITOCÔNDRIAS, CARCINOMA

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      FORNAZARI, Maynara et al. Neuronal differentiation involves a shift from glucose oxidation to fermentation. Journal of Bioenergetics and Biomembranes, v. 43, n. 5, p. 531-539, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10863-011-9374-3. Acesso em: 19 abr. 2024.
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      Fornazari, M., Nascimento, I. C., Nery, A. A., Silva, C. C. C. da, Kowaltowski, A. J., & Ulrich, H. (2011). Neuronal differentiation involves a shift from glucose oxidation to fermentation. Journal of Bioenergetics and Biomembranes, 43( 5), 531-539. doi:10.1007/s10863-011-9374-3
    • NLM

      Fornazari M, Nascimento IC, Nery AA, Silva CCC da, Kowaltowski AJ, Ulrich H. Neuronal differentiation involves a shift from glucose oxidation to fermentation [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 5): 531-539.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9374-3
    • Vancouver

      Fornazari M, Nascimento IC, Nery AA, Silva CCC da, Kowaltowski AJ, Ulrich H. Neuronal differentiation involves a shift from glucose oxidation to fermentation [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 5): 531-539.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9374-3
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: BIOQUÍMICA, MITOCÔNDRIAS

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      COSSO, Ricardo G. et al. Mitochondrial permeability transition induced by chemically generated singlet oxygen. Journal of Bioenergetics and Biomembranes, v. 34, n. 3, p. 157-163, 2002Tradução . . Acesso em: 19 abr. 2024.
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      Cosso, R. G., Turim, J., Nantes, I. L., Almeida, A. M., Di Mascio, P., & Vercesi, A. E. (2002). Mitochondrial permeability transition induced by chemically generated singlet oxygen. Journal of Bioenergetics and Biomembranes, 34( 3), 157-163.
    • NLM

      Cosso RG, Turim J, Nantes IL, Almeida AM, Di Mascio P, Vercesi AE. Mitochondrial permeability transition induced by chemically generated singlet oxygen. Journal of Bioenergetics and Biomembranes. 2002 ; 34( 3): 157-163.[citado 2024 abr. 19 ]
    • Vancouver

      Cosso RG, Turim J, Nantes IL, Almeida AM, Di Mascio P, Vercesi AE. Mitochondrial permeability transition induced by chemically generated singlet oxygen. Journal of Bioenergetics and Biomembranes. 2002 ; 34( 3): 157-163.[citado 2024 abr. 19 ]
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: FCFRP

    Subjects: PARACOCCIDIOIDES BRASILIENSIS, MITOCÔNDRIAS, FARMACOLOGIA

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      MARTINS, Vicente de Paulo et al. Mitochondrial function in the yeast form of the pathogenic fungus Paracoccidioides brasiliensis. Journal of Bioenergetics and Biomembranes, v. 40, n. 4, p. 297-305, 2008Tradução . . Disponível em: https://doi.org/10.1007/s10863-008-9163-9. Acesso em: 19 abr. 2024.
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      Martins, V. de P., Soriani, F. M., Magnani, T., Tudella, V. G., Goldman, G. H., Curti, C., & Uyemura, S. A. (2008). Mitochondrial function in the yeast form of the pathogenic fungus Paracoccidioides brasiliensis. Journal of Bioenergetics and Biomembranes, 40( 4), 297-305. doi:10.1007/s10863-008-9163-9
    • NLM

      Martins V de P, Soriani FM, Magnani T, Tudella VG, Goldman GH, Curti C, Uyemura SA. Mitochondrial function in the yeast form of the pathogenic fungus Paracoccidioides brasiliensis [Internet]. Journal of Bioenergetics and Biomembranes. 2008 ; 40( 4): 297-305.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-008-9163-9
    • Vancouver

      Martins V de P, Soriani FM, Magnani T, Tudella VG, Goldman GH, Curti C, Uyemura SA. Mitochondrial function in the yeast form of the pathogenic fungus Paracoccidioides brasiliensis [Internet]. Journal of Bioenergetics and Biomembranes. 2008 ; 40( 4): 297-305.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-008-9163-9
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: SISTEMA NERVOSO CENTRAL, MITOCÔNDRIAS, BIOQUÍMICA

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      MELO, Daniela R et al. Mitochondrial energy metabolism in neurodegeneration associated with methylmalonic acidemia. Journal of Bioenergetics and Biomembranes, v. 43, n. 1, p. 39-46, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10863-011-9330-2. Acesso em: 19 abr. 2024.
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      Melo, D. R., Kowaltowski, A. J., Wajner, M., & Castilho, R. F. (2011). Mitochondrial energy metabolism in neurodegeneration associated with methylmalonic acidemia. Journal of Bioenergetics and Biomembranes, 43( 1), 39-46. doi:10.1007/s10863-011-9330-2
    • NLM

      Melo DR, Kowaltowski AJ, Wajner M, Castilho RF. Mitochondrial energy metabolism in neurodegeneration associated with methylmalonic acidemia [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 1): 39-46.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9330-2
    • Vancouver

      Melo DR, Kowaltowski AJ, Wajner M, Castilho RF. Mitochondrial energy metabolism in neurodegeneration associated with methylmalonic acidemia [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43( 1): 39-46.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9330-2
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: FCFRP

    Subjects: HIPERTRIGLICERIDEMIA, MITOCÔNDRIAS, POTÁSSIO

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      ALBERICI, Luciane Carla e VERCESI, Anibal E. e OLIVEIRA, Helena C. F. Mitochondrial energy metabolism and redox responses to hypertriglyceridemia. Journal of Bioenergetics and Biomembranes, v. 43, p. 19-23, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10863-011-9326-y. Acesso em: 19 abr. 2024.
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      Alberici, L. C., Vercesi, A. E., & Oliveira, H. C. F. (2011). Mitochondrial energy metabolism and redox responses to hypertriglyceridemia. Journal of Bioenergetics and Biomembranes, 43, 19-23. doi:10.1007/s10863-011-9326-y
    • NLM

      Alberici LC, Vercesi AE, Oliveira HCF. Mitochondrial energy metabolism and redox responses to hypertriglyceridemia [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43 19-23.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9326-y
    • Vancouver

      Alberici LC, Vercesi AE, Oliveira HCF. Mitochondrial energy metabolism and redox responses to hypertriglyceridemia [Internet]. Journal of Bioenergetics and Biomembranes. 2011 ; 43 19-23.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-011-9326-y
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: TRYPANOSOMA CRUZI, MITOCÔNDRIAS

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    • ABNT

      CARRANZA, Julio César et al. Mitochondrial bioenergetics and redox state are unaltered in Trypanosoma cruzi isolates with compromised mitochondrial complex I subunit genes. Journal of Bioenergetics and Biomembranes, v. 41, n. 3, p. 299-308, 2009Tradução . . Disponível em: https://doi.org/10.1007/s10863-009-9228-4. Acesso em: 19 abr. 2024.
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      Carranza, J. C., Kowaltowski, A. J., Mendonça, M. A. G., Oliveira, T. C. de, Gadelha, F. R., & Zingales, B. (2009). Mitochondrial bioenergetics and redox state are unaltered in Trypanosoma cruzi isolates with compromised mitochondrial complex I subunit genes. Journal of Bioenergetics and Biomembranes, 41( 3), 299-308. doi:10.1007/s10863-009-9228-4
    • NLM

      Carranza JC, Kowaltowski AJ, Mendonça MAG, Oliveira TC de, Gadelha FR, Zingales B. Mitochondrial bioenergetics and redox state are unaltered in Trypanosoma cruzi isolates with compromised mitochondrial complex I subunit genes [Internet]. Journal of Bioenergetics and Biomembranes. 2009 ; 41( 3): 299-308.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-009-9228-4
    • Vancouver

      Carranza JC, Kowaltowski AJ, Mendonça MAG, Oliveira TC de, Gadelha FR, Zingales B. Mitochondrial bioenergetics and redox state are unaltered in Trypanosoma cruzi isolates with compromised mitochondrial complex I subunit genes [Internet]. Journal of Bioenergetics and Biomembranes. 2009 ; 41( 3): 299-308.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-009-9228-4
  • Source: Journal of Bioenergetics and Biomembranes. Unidade: IQ

    Subjects: RADICAIS LIVRES, POTÁSSIO, CÁLCIO

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      FACUNDO, Hebert Di Tarso Fernandes e PAULA, Juliane Gomes de e KOWALTOWSKI, Alicia Juliana. Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death. Journal of Bioenergetics and Biomembranes, v. 37, n. 2, p. 75-82, 2005Tradução . . Acesso em: 19 abr. 2024.
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      Facundo, H. D. T. F., Paula, J. G. de, & Kowaltowski, A. J. (2005). Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death. Journal of Bioenergetics and Biomembranes, 37( 2), 75-82.
    • NLM

      Facundo HDTF, Paula JG de, Kowaltowski AJ. Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death. Journal of Bioenergetics and Biomembranes. 2005 ; 37( 2): 75-82.[citado 2024 abr. 19 ]
    • Vancouver

      Facundo HDTF, Paula JG de, Kowaltowski AJ. Mitochondrial ATP-sensitive 'K POT.+' channels prevent oxidative stress, permeability transition and cell death. Journal of Bioenergetics and Biomembranes. 2005 ; 37( 2): 75-82.[citado 2024 abr. 19 ]
  • Source: Journal of Bioenergetics and Biomembranes. Unidades: IQ, IB, ICB

    Subjects: ANATOMIA, METABOLISMO ANIMAL, HIPOCAMPU DE ANIMAL, MEMÓRIA ANIMAL, MITOCÔNDRIAS, DIETA ANIMAL, CÓRTEX CEREBRAL DE ANIMAL, BIOENERGÉTICA, RATOS WISTAR

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      VILELA, Wembley Rodrigues et al. Metabolic dysfunction induced by HFD + L-NAME preferentially affects hippocampal mitochondria, impacting spatial memory in rats. Journal of Bioenergetics and Biomembranes, p. 13 , 2024Tradução . . Disponível em: https://doi.org/10.1007/s10863-024-10005-2. Acesso em: 19 abr. 2024.
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      Vilela, W. R., Ramalho, L. S., Bechara, L. R. G., Costa, J. V. C., Serna, J. D. C., Bem, A. F. de, et al. (2024). Metabolic dysfunction induced by HFD + L-NAME preferentially affects hippocampal mitochondria, impacting spatial memory in rats. Journal of Bioenergetics and Biomembranes, 13 . doi:10.1007/s10863-024-10005-2
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      Vilela WR, Ramalho LS, Bechara LRG, Costa JVC, Serna JDC, Bem AF de, Kowaltowski AJ, Xavier GF, Ferreira JCB. Metabolic dysfunction induced by HFD + L-NAME preferentially affects hippocampal mitochondria, impacting spatial memory in rats [Internet]. Journal of Bioenergetics and Biomembranes. 2024 ;13 .[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-024-10005-2
    • Vancouver

      Vilela WR, Ramalho LS, Bechara LRG, Costa JVC, Serna JDC, Bem AF de, Kowaltowski AJ, Xavier GF, Ferreira JCB. Metabolic dysfunction induced by HFD + L-NAME preferentially affects hippocampal mitochondria, impacting spatial memory in rats [Internet]. Journal of Bioenergetics and Biomembranes. 2024 ;13 .[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-024-10005-2
  • Source: Journal of Bioenergetics and Biomembranes. Unidades: FZEA, IQ

    Subjects: TRYPANOSOMA CRUZI, MITOCÔNDRIAS, LECTINAS DE PLANTAS, CÉLULAS MORTAS

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    • ABNT

      FERNANDES, M. P. et al. Mechanism of Trypanosoma cruzi death induced by Cratylia mollis seed lectin. Journal of Bioenergetics and Biomembranes, v. 42, n. 1, p. 69-78, 2010Tradução . . Disponível em: https://doi.org/10.1007/s10863-010-9268-9. Acesso em: 19 abr. 2024.
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      Fernandes, M. P., Inada, N. M., Chiaratti, M. R., Araújo, F. F. B., Meirelles, F. V., Correia, M. T. S., et al. (2010). Mechanism of Trypanosoma cruzi death induced by Cratylia mollis seed lectin. Journal of Bioenergetics and Biomembranes, 42( 1), 69-78. doi:10.1007/s10863-010-9268-9
    • NLM

      Fernandes MP, Inada NM, Chiaratti MR, Araújo FFB, Meirelles FV, Correia MTS, Coelho LCBB, Alves MJM, Gadelha FR, Vercesi AE. Mechanism of Trypanosoma cruzi death induced by Cratylia mollis seed lectin [Internet]. Journal of Bioenergetics and Biomembranes. 2010 ; 42( 1): 69-78.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-010-9268-9
    • Vancouver

      Fernandes MP, Inada NM, Chiaratti MR, Araújo FFB, Meirelles FV, Correia MTS, Coelho LCBB, Alves MJM, Gadelha FR, Vercesi AE. Mechanism of Trypanosoma cruzi death induced by Cratylia mollis seed lectin [Internet]. Journal of Bioenergetics and Biomembranes. 2010 ; 42( 1): 69-78.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1007/s10863-010-9268-9

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