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Artigo de periodico
The role of inflammation in HPV carcinogenesis (2010)
Boccardo, Enrique ; Lepique, Ana Paula ; Villa, Luisa Lina
Source: Carcinogenesis. Unidade: ICB
Subjects: IMUNOLOGIA, MICROBIOLOGIA
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ABNT
BOCCARDO, Enrique e LEPIQUE, Ana Paula e VILLA, Luisa Lina. The role of inflammation in HPV carcinogenesis. Carcinogenesis, v. 31, n. 11, p. 1905-1912, 2010Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgq176. Acesso em: 19 abr. 2024.
APA
Boccardo, E., Lepique, A. P., & Villa, L. L. (2010). The role of inflammation in HPV carcinogenesis. Carcinogenesis, 31( 11), 1905-1912. doi:10.1093/carcin/bgq176
NLM
Boccardo E, Lepique AP, Villa LL. The role of inflammation in HPV carcinogenesis [Internet]. Carcinogenesis. 2010 ; 31( 11): 1905-1912.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgq176
Vancouver
Boccardo E, Lepique AP, Villa LL. The role of inflammation in HPV carcinogenesis [Internet]. Carcinogenesis. 2010 ; 31( 11): 1905-1912.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgq176
Artigo de periodico
The isatin-Schiff base copper(II) complex Cu(isaepy)(2) acts as delocalized lipophilic cation, yields widespread mitochondrial oxidative damage and induces AMP-activated protein kinase-dependent apoptosis (2009)
Filomeni, Giuseppe; Piccirillo, Sara; Graziani, Ilaria; Cardaci, Simone; Ferreira, Ana Maria da Costa ; Rotilio, Giuseppe; Ciriolo, Maria Rosa
Source: Carcinogenesis. Unidade: IQ
Subjects: BASES (QUÍMICA INORGÂNICA), COBRE, APOPTOSE
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ABNT
FILOMENI, Giuseppe et al. The isatin-Schiff base copper(II) complex Cu(isaepy)(2) acts as delocalized lipophilic cation, yields widespread mitochondrial oxidative damage and induces AMP-activated protein kinase-dependent apoptosis. Carcinogenesis, v. 30, n. 7, p. 1115-1124, 2009Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgp105. Acesso em: 19 abr. 2024.
APA
Filomeni, G., Piccirillo, S., Graziani, I., Cardaci, S., Ferreira, A. M. da C., Rotilio, G., & Ciriolo, M. R. (2009). The isatin-Schiff base copper(II) complex Cu(isaepy)(2) acts as delocalized lipophilic cation, yields widespread mitochondrial oxidative damage and induces AMP-activated protein kinase-dependent apoptosis. Carcinogenesis, 30( 7), 1115-1124. doi:10.1093/carcin/bgp105
NLM
Filomeni G, Piccirillo S, Graziani I, Cardaci S, Ferreira AM da C, Rotilio G, Ciriolo MR. The isatin-Schiff base copper(II) complex Cu(isaepy)(2) acts as delocalized lipophilic cation, yields widespread mitochondrial oxidative damage and induces AMP-activated protein kinase-dependent apoptosis [Internet]. Carcinogenesis. 2009 ; 30( 7): 1115-1124.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgp105
Vancouver
Filomeni G, Piccirillo S, Graziani I, Cardaci S, Ferreira AM da C, Rotilio G, Ciriolo MR. The isatin-Schiff base copper(II) complex Cu(isaepy)(2) acts as delocalized lipophilic cation, yields widespread mitochondrial oxidative damage and induces AMP-activated protein kinase-dependent apoptosis [Internet]. Carcinogenesis. 2009 ; 30( 7): 1115-1124.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgp105
Artigo de periodico
The chemopreventive activity of the butyric acid prodrug tributyrin in experimental rat hepatocarcinogenesis is associated with p53 acetylation and activation of the p53 apoptotic signaling pathway (2013)
De Conti, Aline; Tryndyak, Volodymyr; Koturbash, Igor; Heidor, Renato ; Kuroiwa-Trzmielina, Joice; Ong, Thomas Prates ; Beland, Frederick A; Moreno, Fernando Salvador ; Pogribny, Igor P
Source: Carcinogenesis. Unidade: FCF
Subjects: CARCINOGÊNESE ANIMAL, NEOPLASIAS (QUIMIOTERAPIA)
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DE CONTI, Aline et al. The chemopreventive activity of the butyric acid prodrug tributyrin in experimental rat hepatocarcinogenesis is associated with p53 acetylation and activation of the p53 apoptotic signaling pathway. Carcinogenesis, v. 34, n. 8, p. 1900-1906, 2013Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgt124. Acesso em: 19 abr. 2024.
APA
De Conti, A., Tryndyak, V., Koturbash, I., Heidor, R., Kuroiwa-Trzmielina, J., Ong, T. P., et al. (2013). The chemopreventive activity of the butyric acid prodrug tributyrin in experimental rat hepatocarcinogenesis is associated with p53 acetylation and activation of the p53 apoptotic signaling pathway. Carcinogenesis, 34( 8), 1900-1906. doi:10.1093/carcin/bgt124
NLM
De Conti A, Tryndyak V, Koturbash I, Heidor R, Kuroiwa-Trzmielina J, Ong TP, Beland FA, Moreno FS, Pogribny IP. The chemopreventive activity of the butyric acid prodrug tributyrin in experimental rat hepatocarcinogenesis is associated with p53 acetylation and activation of the p53 apoptotic signaling pathway [Internet]. Carcinogenesis. 2013 ; 34( 8): 1900-1906.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgt124
Vancouver
De Conti A, Tryndyak V, Koturbash I, Heidor R, Kuroiwa-Trzmielina J, Ong TP, Beland FA, Moreno FS, Pogribny IP. The chemopreventive activity of the butyric acid prodrug tributyrin in experimental rat hepatocarcinogenesis is associated with p53 acetylation and activation of the p53 apoptotic signaling pathway [Internet]. Carcinogenesis. 2013 ; 34( 8): 1900-1906.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgt124
Artigo de periodico
TP53 and EGFR mutations in combination with lifestyle risk factors in tumours of the upper aerodigestive tract from South America (2010)
Szymanska, K.; Levi, J. E.; Menezes, A.; Wünsch Filho, Victor; Eluf-Neto, J.; Koifman, S.; Matos, E.; Daudt, A. W.; Curado, M. P.; Villar, S.; Pawlita, M.; Waterboer, T.; Boffetta, P.; Hainaut, P.; Brennan, P.
Source: Carcinogenesis. Unidades: FSP, FM
Subjects: MODO DE VIDA, FATORES DE RISCO, SISTEMA DIGESTÓRIO (PATOLOGIA), NEOPLASIAS DE CABEÇA E PESCOÇO, NEOPLASIAS GÁSTRICAS
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ABNT
SZYMANSKA, K. et al. TP53 and EGFR mutations in combination with lifestyle risk factors in tumours of the upper aerodigestive tract from South America. Carcinogenesis, v. 31, n. 6, p. 1054-1059, 2010Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgp212. Acesso em: 19 abr. 2024.
APA
Szymanska, K., Levi, J. E., Menezes, A., Wünsch Filho, V., Eluf-Neto, J., Koifman, S., et al. (2010). TP53 and EGFR mutations in combination with lifestyle risk factors in tumours of the upper aerodigestive tract from South America. Carcinogenesis, 31( 6), 1054-1059. doi:10.1093/carcin/bgp212
NLM
Szymanska K, Levi JE, Menezes A, Wünsch Filho V, Eluf-Neto J, Koifman S, Matos E, Daudt AW, Curado MP, Villar S, Pawlita M, Waterboer T, Boffetta P, Hainaut P, Brennan P. TP53 and EGFR mutations in combination with lifestyle risk factors in tumours of the upper aerodigestive tract from South America [Internet]. Carcinogenesis. 2010 ; 31( 6): 1054-1059.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgp212
Vancouver
Szymanska K, Levi JE, Menezes A, Wünsch Filho V, Eluf-Neto J, Koifman S, Matos E, Daudt AW, Curado MP, Villar S, Pawlita M, Waterboer T, Boffetta P, Hainaut P, Brennan P. TP53 and EGFR mutations in combination with lifestyle risk factors in tumours of the upper aerodigestive tract from South America [Internet]. Carcinogenesis. 2010 ; 31( 6): 1054-1059.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgp212
Artigo de periodico
Relationship between megacolon and carcinoma of the colon: an experimental approach (1996)
Garcia, Sérgio Britto; Oliveira, João Samuel Meira de; Pinto, L Z; Muccillo, Gerson; Zucoloto, S
Source: Carcinogenesis. Unidades: FMRP, FFCLRP
Subjects: NEOPLASIAS, CIRURGIA COLORRETAL
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GARCIA, Sérgio Britto et al. Relationship between megacolon and carcinoma of the colon: an experimental approach. Carcinogenesis, v. 17, n. 8 , p. 1777-9, 1996Tradução . . Acesso em: 19 abr. 2024.
APA
Garcia, S. B., Oliveira, J. S. M. de, Pinto, L. Z., Muccillo, G., & Zucoloto, S. (1996). Relationship between megacolon and carcinoma of the colon: an experimental approach. Carcinogenesis, 17( 8 ), 1777-9.
NLM
Garcia SB, Oliveira JSM de, Pinto LZ, Muccillo G, Zucoloto S. Relationship between megacolon and carcinoma of the colon: an experimental approach. Carcinogenesis. 1996 ;17( 8 ): 1777-9.[citado 2024 abr. 19 ]
Vancouver
Garcia SB, Oliveira JSM de, Pinto LZ, Muccillo G, Zucoloto S. Relationship between megacolon and carcinoma of the colon: an experimental approach. Carcinogenesis. 1996 ;17( 8 ): 1777-9.[citado 2024 abr. 19 ]
Artigo de periodico
Reduction of malignant phenotype of HEPG2 cell is associated with the expression of connexin 26 but not connexin 32 (2001)
Yano, Tomohiro; Hernandez-Blazquez, Francisco Javier; Omori, Yasufumi; Yamasaki, Hiroshi
Source: Carcinogenesis. Unidade: FZEA
Subjects: CITOLOGIA, ONCOLOGIA, GENES SUPRESSORES DE TUMOR
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ABNT
YANO, Tomohiro et al. Reduction of malignant phenotype of HEPG2 cell is associated with the expression of connexin 26 but not connexin 32. Carcinogenesis, v. 22, n. 10, p. 1593-1600, 2001Tradução . . Disponível em: https://doi.org/10.1093/carcin/22.10.1593. Acesso em: 19 abr. 2024.
APA
Yano, T., Hernandez-Blazquez, F. J., Omori, Y., & Yamasaki, H. (2001). Reduction of malignant phenotype of HEPG2 cell is associated with the expression of connexin 26 but not connexin 32. Carcinogenesis, 22( 10), 1593-1600. doi:10.1093/carcin/22.10.1593
NLM
Yano T, Hernandez-Blazquez FJ, Omori Y, Yamasaki H. Reduction of malignant phenotype of HEPG2 cell is associated with the expression of connexin 26 but not connexin 32 [Internet]. Carcinogenesis. 2001 ; 22( 10): 1593-1600.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/22.10.1593
Vancouver
Yano T, Hernandez-Blazquez FJ, Omori Y, Yamasaki H. Reduction of malignant phenotype of HEPG2 cell is associated with the expression of connexin 26 but not connexin 32 [Internet]. Carcinogenesis. 2001 ; 22( 10): 1593-1600.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/22.10.1593
Artigo de periodico
Phenanthroline protects mammalian cells from hydrogen peroxide-induced gene mutation and morphological transformation (1989)
Nassi-Calo, L; Mello Filho, A. C; Meneghini, R.
Source: Carcinogenesis. Unidade: IQ
Subjects: MUTAÇÃO, ONCOGENES
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ABNT
NASSI-CALO, L e MELLO FILHO, A. C e MENEGHINI, R. Phenanthroline protects mammalian cells from hydrogen peroxide-induced gene mutation and morphological transformation. Carcinogenesis, v. 10, n. 6, p. 1055-7, 1989Tradução . . Disponível em: https://doi.org/10.1093/carcin/10.6.1055. Acesso em: 19 abr. 2024.
APA
Nassi-Calo, L., Mello Filho, A. C., & Meneghini, R. (1989). Phenanthroline protects mammalian cells from hydrogen peroxide-induced gene mutation and morphological transformation. Carcinogenesis, 10( 6), 1055-7. doi:10.1093/carcin/10.6.1055
NLM
Nassi-Calo L, Mello Filho AC, Meneghini R. Phenanthroline protects mammalian cells from hydrogen peroxide-induced gene mutation and morphological transformation [Internet]. Carcinogenesis. 1989 ; 10( 6): 1055-7.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/10.6.1055
Vancouver
Nassi-Calo L, Mello Filho AC, Meneghini R. Phenanthroline protects mammalian cells from hydrogen peroxide-induced gene mutation and morphological transformation [Internet]. Carcinogenesis. 1989 ; 10( 6): 1055-7.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/10.6.1055
Artigo de periodico
PD-1 blockage delays murine squamous cell carcinoma development (2014)
Belai, Eduardo Bertoli; Oliveira, Carine Ervolino; Gasparoto, Thais Helena; Ramos, Rodrigo Nalio; Torres, Sérgio Aparecido; Garlet, Gustavo Pompermaier; Cavassani, Karen Angélica; Torres, Sergio Aparecido; Silva, João Santana da; Campanelli, Ana Paula
Source: Carcinogenesis. Unidades: FOB, FMRP
Subjects: CARCINOMA DE CÉLULAS ESCAMOSAS, INFLAMAÇÃO
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ABNT
BELAI, Eduardo Bertoli et al. PD-1 blockage delays murine squamous cell carcinoma development. Carcinogenesis, v. 35, n. 2, p. 424-431, 2014Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgs305. Acesso em: 19 abr. 2024.
APA
Belai, E. B., Oliveira, C. E., Gasparoto, T. H., Ramos, R. N., Torres, S. A., Garlet, G. P., et al. (2014). PD-1 blockage delays murine squamous cell carcinoma development. Carcinogenesis, 35( 2), 424-431. doi:10.1093/carcin/bgs305
NLM
Belai EB, Oliveira CE, Gasparoto TH, Ramos RN, Torres SA, Garlet GP, Cavassani KA, Torres SA, Silva JS da, Campanelli AP. PD-1 blockage delays murine squamous cell carcinoma development [Internet]. Carcinogenesis. 2014 ; 35( 2): 424-431.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgs305
Vancouver
Belai EB, Oliveira CE, Gasparoto TH, Ramos RN, Torres SA, Garlet GP, Cavassani KA, Torres SA, Silva JS da, Campanelli AP. PD-1 blockage delays murine squamous cell carcinoma development [Internet]. Carcinogenesis. 2014 ; 35( 2): 424-431.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgs305
Artigo de periodico
Low amounts of the DNA repair XPA protein are sufficient to recover UV-resistance (2002)
Muotri, Alison Renato; Marchetto, M C.; Suzuki, M F.; Okazaki, K.; Lotfi, Claudimara Ferini Pacicco ; Brumatti, G.; Amarante-Mendes, João Gustavo Pessini ; Menck, Carlos Frederico Martins
Source: Carcinogenesis. Unidade: ICB
Subjects: ANATOMIA, MICROBIOLOGIA, IMUNOLOGIA
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ABNT
MUOTRI, Alison Renato et al. Low amounts of the DNA repair XPA protein are sufficient to recover UV-resistance. Carcinogenesis, v. 23, p. 1039-46, 2002Tradução . . Disponível em: https://doi.org/10.1093/carcin/23.6.1039. Acesso em: 19 abr. 2024.
APA
Muotri, A. R., Marchetto, M. C., Suzuki, M. F., Okazaki, K., Lotfi, C. F. P., Brumatti, G., et al. (2002). Low amounts of the DNA repair XPA protein are sufficient to recover UV-resistance. Carcinogenesis, 23, 1039-46. doi:10.1093/carcin/23.6.1039
NLM
Muotri AR, Marchetto MC, Suzuki MF, Okazaki K, Lotfi CFP, Brumatti G, Amarante-Mendes JGP, Menck CFM. Low amounts of the DNA repair XPA protein are sufficient to recover UV-resistance [Internet]. Carcinogenesis. 2002 ; 23 1039-46.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/23.6.1039
Vancouver
Muotri AR, Marchetto MC, Suzuki MF, Okazaki K, Lotfi CFP, Brumatti G, Amarante-Mendes JGP, Menck CFM. Low amounts of the DNA repair XPA protein are sufficient to recover UV-resistance [Internet]. Carcinogenesis. 2002 ; 23 1039-46.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/23.6.1039
Artigo de periodico
Involvement of escherichia coli exonuclease iii and endonuclease iv in the repair of singlet oxygen-induced dna demage (1996)
Agnez, L F; Oliveira, R L C; Di Mascio, Paolo ; Menck, Carlos Frederico Martins
Source: Carcinogenesis. Unidades: ICB, IQ
Subjects: BIOQUÍMICA, BIOLOGIA MOLECULAR
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ABNT
AGNEZ, L F et al. Involvement of escherichia coli exonuclease iii and endonuclease iv in the repair of singlet oxygen-induced dna demage. Carcinogenesis, v. 17, n. 5 , p. 1183-5, 1996Tradução . . Acesso em: 19 abr. 2024.
APA
Agnez, L. F., Oliveira, R. L. C., Di Mascio, P., & Menck, C. F. M. (1996). Involvement of escherichia coli exonuclease iii and endonuclease iv in the repair of singlet oxygen-induced dna demage. Carcinogenesis, 17( 5 ), 1183-5.
NLM
Agnez LF, Oliveira RLC, Di Mascio P, Menck CFM. Involvement of escherichia coli exonuclease iii and endonuclease iv in the repair of singlet oxygen-induced dna demage. Carcinogenesis. 1996 ;17( 5 ): 1183-5.[citado 2024 abr. 19 ]
Vancouver
Agnez LF, Oliveira RLC, Di Mascio P, Menck CFM. Involvement of escherichia coli exonuclease iii and endonuclease iv in the repair of singlet oxygen-induced dna demage. Carcinogenesis. 1996 ;17( 5 ): 1183-5.[citado 2024 abr. 19 ]
Artigo de periodico
Inhibitory effects of 'BETA'- caroteno on preneoplastic lesions induced in wistar rats by the resistant hepatocyte model (1991)
Moreno, Fernando Salvador ; Rizzi, M B S L; Dagli, Maria Lúcia Zaidan; Penteado, Marilene de Vuono Camargo
Source: Carcinogenesis. Unidades: FCF, FMVZ
Subjects: MEDICINA VETERINÁRIA, ONCOLOGIA
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ABNT
MORENO, Fernando Salvador et al. Inhibitory effects of 'BETA'- caroteno on preneoplastic lesions induced in wistar rats by the resistant hepatocyte model. Carcinogenesis, v. 12, n. 10, p. 1817-22, 1991Tradução . . Acesso em: 19 abr. 2024.
APA
Moreno, F. S., Rizzi, M. B. S. L., Dagli, M. L. Z., & Penteado, M. de V. C. (1991). Inhibitory effects of 'BETA'- caroteno on preneoplastic lesions induced in wistar rats by the resistant hepatocyte model. Carcinogenesis, 12( 10), 1817-22.
NLM
Moreno FS, Rizzi MBSL, Dagli MLZ, Penteado M de VC. Inhibitory effects of 'BETA'- caroteno on preneoplastic lesions induced in wistar rats by the resistant hepatocyte model. Carcinogenesis. 1991 ;12( 10): 1817-22.[citado 2024 abr. 19 ]
Vancouver
Moreno FS, Rizzi MBSL, Dagli MLZ, Penteado M de VC. Inhibitory effects of 'BETA'- caroteno on preneoplastic lesions induced in wistar rats by the resistant hepatocyte model. Carcinogenesis. 1991 ;12( 10): 1817-22.[citado 2024 abr. 19 ]
Artigo de periodico
Increased susceptibility to urethane-induced lung tumors in mice with decreased expression of connexin43 (2004)
Avanzo, José Luis; Mesnil, Marc; Hernandez-Blazquez, Francisco Javier; Mackowiak, Ivone Isabel; Mori, Cláudia Madalena Cabrera; Silva, Tereza Cristina da; Oloris, Sílvia Catarina Salgado; Gárate, Ana Paula; Massironi, Sílvia Maria Gomes; Yamasaki, Hiroshi; Dagli, Maria Lúcia Zaidan
Source: Carcinogenesis. Unidades: FMVZ, ICB
Subjects: ANTINEOPLÁSICOS (ADMINISTRAÇÃO E DOSAGEM), NEOPLASIAS PULMONARES
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ABNT
AVANZO, José Luis et al. Increased susceptibility to urethane-induced lung tumors in mice with decreased expression of connexin43. Carcinogenesis, v. 25, n. 10, p. 1973-1982, 2004Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgh193. Acesso em: 19 abr. 2024.
APA
Avanzo, J. L., Mesnil, M., Hernandez-Blazquez, F. J., Mackowiak, I. I., Mori, C. M. C., Silva, T. C. da, et al. (2004). Increased susceptibility to urethane-induced lung tumors in mice with decreased expression of connexin43. Carcinogenesis, 25( 10), 1973-1982. doi:10.1093/carcin/bgh193
NLM
Avanzo JL, Mesnil M, Hernandez-Blazquez FJ, Mackowiak II, Mori CMC, Silva TC da, Oloris SCS, Gárate AP, Massironi SMG, Yamasaki H, Dagli MLZ. Increased susceptibility to urethane-induced lung tumors in mice with decreased expression of connexin43 [Internet]. Carcinogenesis. 2004 ; 25( 10): 1973-1982.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgh193
Vancouver
Avanzo JL, Mesnil M, Hernandez-Blazquez FJ, Mackowiak II, Mori CMC, Silva TC da, Oloris SCS, Gárate AP, Massironi SMG, Yamasaki H, Dagli MLZ. Increased susceptibility to urethane-induced lung tumors in mice with decreased expression of connexin43 [Internet]. Carcinogenesis. 2004 ; 25( 10): 1973-1982.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgh193
Artigo de periodico
Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells (2021)
Freire, Thiago de Souza; Mori, M.P; Miranda, J. N. F. A; Muta, Lais Yoshie Morikawa; Machado, F. T; Moreno, N. C; Souza-Pinto, Nadja Cristhina de
Source: Carcinogenesis. Unidade: IQ
Subjects: MITOCÔNDRIAS, CÉLULAS, PERÓXIDO DE HIDROGÊNIO
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ABNT
FREIRE, Thiago de Souza et al. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells. Carcinogenesis, v. 42, n. 11, p. 1380-1389, 2021Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgab079. Acesso em: 19 abr. 2024.
APA
Freire, T. de S., Mori, M. P., Miranda, J. N. F. A., Muta, L. Y. M., Machado, F. T., Moreno, N. C., & Souza-Pinto, N. C. de. (2021). Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells. Carcinogenesis, 42( 11), 1380-1389. doi:10.1093/carcin/bgab079
NLM
Freire T de S, Mori MP, Miranda JNFA, Muta LYM, Machado FT, Moreno NC, Souza-Pinto NC de. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells [Internet]. Carcinogenesis. 2021 ; 42( 11): 1380-1389.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgab079
Vancouver
Freire T de S, Mori MP, Miranda JNFA, Muta LYM, Machado FT, Moreno NC, Souza-Pinto NC de. Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells [Internet]. Carcinogenesis. 2021 ; 42( 11): 1380-1389.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgab079
Artigo de periodico
Geranylgeraniol and beta-ionone inhibit hepatic preneoplastic lesions, cell proliferation, total plasma cholesterol and DNA damage during the initial phases of hepatocarcinogenesis, but only the former inhibits NF-'kappa'B activation (2005)
Espíndola, Roseli de Moura; Mazzantini, Rogério Pietro; Ong, Thomas Prates ; Conti, Aline de; Heidor, Renato ; Moreno, Fernando Salvador
Source: Carcinogenesis. Unidade: FCF
Subjects: MELANOMA ANIMAL, CARCINOGÊNESE ANIMAL
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ABNT
ESPÍNDOLA, Roseli de Moura et al. Geranylgeraniol and beta-ionone inhibit hepatic preneoplastic lesions, cell proliferation, total plasma cholesterol and DNA damage during the initial phases of hepatocarcinogenesis, but only the former inhibits NF-'kappa'B activation. Carcinogenesis, v. 26, n. 6, p. 1091-1099, 2005Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgi047. Acesso em: 19 abr. 2024.
APA
Espíndola, R. de M., Mazzantini, R. P., Ong, T. P., Conti, A. de, Heidor, R., & Moreno, F. S. (2005). Geranylgeraniol and beta-ionone inhibit hepatic preneoplastic lesions, cell proliferation, total plasma cholesterol and DNA damage during the initial phases of hepatocarcinogenesis, but only the former inhibits NF-'kappa'B activation. Carcinogenesis, 26( 6), 1091-1099. doi:10.1093/carcin/bgi047
NLM
Espíndola R de M, Mazzantini RP, Ong TP, Conti A de, Heidor R, Moreno FS. Geranylgeraniol and beta-ionone inhibit hepatic preneoplastic lesions, cell proliferation, total plasma cholesterol and DNA damage during the initial phases of hepatocarcinogenesis, but only the former inhibits NF-'kappa'B activation [Internet]. Carcinogenesis. 2005 ; 26( 6): 1091-1099.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgi047
Vancouver
Espíndola R de M, Mazzantini RP, Ong TP, Conti A de, Heidor R, Moreno FS. Geranylgeraniol and beta-ionone inhibit hepatic preneoplastic lesions, cell proliferation, total plasma cholesterol and DNA damage during the initial phases of hepatocarcinogenesis, but only the former inhibits NF-'kappa'B activation [Internet]. Carcinogenesis. 2005 ; 26( 6): 1091-1099.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgi047
Artigo de periodico
Formation of methyl radicals during the catalase mediated oxidation of formaldehyde hydrazone (1991)
Gomes, Suely Lopes ; Augusto, Ohara
Source: Carcinogenesis. Unidade: IQ
Subjects: BIOQUÍMICA, METABOLISMO, CITOLOGIA, BIOLOGIA CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOMES, Suely Lopes e AUGUSTO, Ohara. Formation of methyl radicals during the catalase mediated oxidation of formaldehyde hydrazone. Carcinogenesis, v. 12, n. 7 , p. 1351-3, 1991Tradução . . Disponível em: https://doi.org/10.1093/carcin/12.7.1351. Acesso em: 19 abr. 2024.
APA
Gomes, S. L., & Augusto, O. (1991). Formation of methyl radicals during the catalase mediated oxidation of formaldehyde hydrazone. Carcinogenesis, 12( 7 ), 1351-3. doi:10.1093/carcin/12.7.1351
NLM
Gomes SL, Augusto O. Formation of methyl radicals during the catalase mediated oxidation of formaldehyde hydrazone [Internet]. Carcinogenesis. 1991 ;12( 7 ): 1351-3.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/12.7.1351
Vancouver
Gomes SL, Augusto O. Formation of methyl radicals during the catalase mediated oxidation of formaldehyde hydrazone [Internet]. Carcinogenesis. 1991 ;12( 7 ): 1351-3.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/12.7.1351
Texto na web
Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase (2005)
Ong, Thomas Prates ; Heidor, Renato ; Conti, Aline de; Dagli, Maria Lúcia Zaidan; Moreno, Fernando Salvador
Source: Carcinogenesis. Unidades: FMVZ, FCF
Subjects: CARCINOGÊNESE ANIMAL, HEPATOPATIAS, QUÍMICA ORGÂNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ONG, Thomas Prates et al. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. Oxford: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo. . Acesso em: 19 abr. 2024. , 2005
APA
Ong, T. P., Heidor, R., Conti, A. de, Dagli, M. L. Z., & Moreno, F. S. (2005). Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. Oxford: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo.
NLM
Ong TP, Heidor R, Conti A de, Dagli MLZ, Moreno FS. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. 2005 ; 1-43.[citado 2024 abr. 19 ]
Vancouver
Ong TP, Heidor R, Conti A de, Dagli MLZ, Moreno FS. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. 2005 ; 1-43.[citado 2024 abr. 19 ]
Artigo de periodico
Expression of human papillomavirus type 16 E7 oncoprotein alters keratinocytes expression profile in response to tumor necrosis factor-α (2010)
Boccardo, Enrique ; Baldi, Carina Victoria Manzini; Carvalho, Alex Fiorini; Rabachini, Tatiana; Torres, César; Barreta, Luiz André; Brentani, Helena; Villa, Luisa Lina
Source: Carcinogenesis. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOCCARDO, Enrique et al. Expression of human papillomavirus type 16 E7 oncoprotein alters keratinocytes expression profile in response to tumor necrosis factor-α. Carcinogenesis, v. 31, n. 3, p. 521-531, 2010Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgp333. Acesso em: 19 abr. 2024.
APA
Boccardo, E., Baldi, C. V. M., Carvalho, A. F., Rabachini, T., Torres, C., Barreta, L. A., et al. (2010). Expression of human papillomavirus type 16 E7 oncoprotein alters keratinocytes expression profile in response to tumor necrosis factor-α. Carcinogenesis, 31( 3), 521-531. doi:10.1093/carcin/bgp333
NLM
Boccardo E, Baldi CVM, Carvalho AF, Rabachini T, Torres C, Barreta LA, Brentani H, Villa LL. Expression of human papillomavirus type 16 E7 oncoprotein alters keratinocytes expression profile in response to tumor necrosis factor-α [Internet]. Carcinogenesis. 2010 ; 31( 3): 521-531.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgp333
Vancouver
Boccardo E, Baldi CVM, Carvalho AF, Rabachini T, Torres C, Barreta LA, Brentani H, Villa LL. Expression of human papillomavirus type 16 E7 oncoprotein alters keratinocytes expression profile in response to tumor necrosis factor-α [Internet]. Carcinogenesis. 2010 ; 31( 3): 521-531.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgp333
Artigo de periodico
Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver (2004)
Dagli, Maria Lúcia Zaidan; Yamasaki, Hiroshi; Krutovskikh, Vladimir; Omori, Yasufumi
Source: Carcinogenesis. Unidade: FMVZ
Subjects: FÍGADO (REGENERAÇÃO), CAMUNDONGOS, CARCINOGÊNESE ANIMAL, CONEXINAS, TRANSGENES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAGLI, Maria Lúcia Zaidan et al. Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver. Carcinogenesis, v. 25, n. 4, p. 483-492, 2004Tradução . . Disponível em: https://doi.org/10.1093/carcin/bgh050. Acesso em: 19 abr. 2024.
APA
Dagli, M. L. Z., Yamasaki, H., Krutovskikh, V., & Omori, Y. (2004). Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver. Carcinogenesis, 25( 4), 483-492. doi:10.1093/carcin/bgh050
NLM
Dagli MLZ, Yamasaki H, Krutovskikh V, Omori Y. Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver [Internet]. Carcinogenesis. 2004 ; 25( 4): 483-492.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgh050
Vancouver
Dagli MLZ, Yamasaki H, Krutovskikh V, Omori Y. Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver [Internet]. Carcinogenesis. 2004 ; 25( 4): 483-492.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/bgh050
Artigo de periodico
Contribution of hydrazines-derived alkyl radicals to cytotoxicity and transformation induced in normal C-myc-overexpressing mouse fibroblasts (1998)
Gamberini, M; Cidade, M R; Valotta, L A; Armelin, Mari Cleide Sogayar; Leite, L C C
Source: Carcinogenesis. Unidade: IQ
Assunto: BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GAMBERINI, M et al. Contribution of hydrazines-derived alkyl radicals to cytotoxicity and transformation induced in normal C-myc-overexpressing mouse fibroblasts. Carcinogenesis, v. 19, n. 1, p. 147-155, 1998Tradução . . Disponível em: https://doi.org/10.1093/carcin/19.1.147. Acesso em: 19 abr. 2024.
APA
Gamberini, M., Cidade, M. R., Valotta, L. A., Armelin, M. C. S., & Leite, L. C. C. (1998). Contribution of hydrazines-derived alkyl radicals to cytotoxicity and transformation induced in normal C-myc-overexpressing mouse fibroblasts. Carcinogenesis, 19( 1), 147-155. doi:10.1093/carcin/19.1.147
NLM
Gamberini M, Cidade MR, Valotta LA, Armelin MCS, Leite LCC. Contribution of hydrazines-derived alkyl radicals to cytotoxicity and transformation induced in normal C-myc-overexpressing mouse fibroblasts [Internet]. Carcinogenesis. 1998 ; 19( 1): 147-155.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/19.1.147
Vancouver
Gamberini M, Cidade MR, Valotta LA, Armelin MCS, Leite LCC. Contribution of hydrazines-derived alkyl radicals to cytotoxicity and transformation induced in normal C-myc-overexpressing mouse fibroblasts [Internet]. Carcinogenesis. 1998 ; 19( 1): 147-155.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/19.1.147
Artigo de periodico
Chronic trypanosoma cruzi infection associated with low incidence of 1,2-dimethylhydrazine-induced colon cancer in rats (2001)
Oliveira, E. C.; Leite, M. S. B.; Miranda, J. A. R.; Andrade, A. L. S. S.; Garcia, Sérgio Britto; Luquetti, A. O.; Moreira, H.
Source: Carcinogenesis. Unidade: FMRP
Subjects: TRYPANOSOMA CRUZI, NEOPLASIAS DO COLON, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, E. C. et al. Chronic trypanosoma cruzi infection associated with low incidence of 1,2-dimethylhydrazine-induced colon cancer in rats. Carcinogenesis, v. 22, n. 5, p. 737-740, 2001Tradução . . Disponível em: https://doi.org/10.1093/carcin/22.5.737. Acesso em: 19 abr. 2024.
APA
Oliveira, E. C., Leite, M. S. B., Miranda, J. A. R., Andrade, A. L. S. S., Garcia, S. B., Luquetti, A. O., & Moreira, H. (2001). Chronic trypanosoma cruzi infection associated with low incidence of 1,2-dimethylhydrazine-induced colon cancer in rats. Carcinogenesis, 22( 5), 737-740. doi:10.1093/carcin/22.5.737
NLM
Oliveira EC, Leite MSB, Miranda JAR, Andrade ALSS, Garcia SB, Luquetti AO, Moreira H. Chronic trypanosoma cruzi infection associated with low incidence of 1,2-dimethylhydrazine-induced colon cancer in rats [Internet]. Carcinogenesis. 2001 ; 22( 5): 737-740.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/22.5.737
Vancouver
Oliveira EC, Leite MSB, Miranda JAR, Andrade ALSS, Garcia SB, Luquetti AO, Moreira H. Chronic trypanosoma cruzi infection associated with low incidence of 1,2-dimethylhydrazine-induced colon cancer in rats [Internet]. Carcinogenesis. 2001 ; 22( 5): 737-740.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1093/carcin/22.5.737

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