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Artigo de periodico
Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids (2022)
Costa, Tiago Januário da ; Barros, Paula Rodrigues de ; Duarte, Diego A. ; Silva-Neto, Júlio Alves da; Hott, Sara Cristina ; Santos-Silva, Thamyris ; Costa-Neto, Claudio M. ; McCarthy, Cameron G. ; Jiménez-Altayó, Francesc ; Dantas, Ana Paula Villela ; Passaglia, Rita de Cassia Aleixo Tostes ; Akamine, Eliana Hiromi ; Gomes, Felipe Villela
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FMRP, ICB
Assuntos: FARMACOLOGIA, PROSTAGLANDINAS, RECEPTORES ADRENÉRGICOS, MENOPAUSA, FÁRMACOS (SISTEMA CARDIOVASCULAR), ARTÉRIAS CARÓTIDAS, VASOS SANGUÍNEOS, CAMUNDONGOS, VASODILATAÇÃO, MODELOS ANIMAIS DE DOENÇAS, ENVELHECIMENTO
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ABNT
COSTA, Tiago Januário da et al. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, v. 324, n. 4, p. H417-H429, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00495.2022. Acesso em: 10 jun. 2024.
APA
Costa, T. J. da, Barros, P. R. de, Duarte, D. A., Silva-Neto, J. A. da, Hott, S. C., Santos-Silva, T., et al. (2022). Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, 324( 4), H417-H429. doi:https://doi.org/10.1152/ajpheart.00495.2022
NLM
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2022 ; 324( 4): H417-H429.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Vancouver
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2022 ; 324( 4): H417-H429.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Artigo de periodico
Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence (2021)
Barros, Paula Rodrigues de ; Costa, Tiago Januário da ; Akamine, Eliana Hiromi ; Passaglia, Rita de Cassia Aleixo Tostes
Fonte: Frontiers in Aging. Unidades: ICB, FMRP
Assuntos: FARMACOLOGIA, ENVELHECIMENTO, ENVELHECIMENTO CELULAR, DIFERENÇAS SEXUAIS (PSICOGÊNESE), MODELOS ANIMAIS DE DOENÇAS, ROEDORES, DOENÇAS CARDIOVASCULARES EM ANIMAL
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ABNT
BARROS, Paula Rodrigues de et al. Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence. Frontiers in Aging, v. 2, p. 1-22, 2021Tradução . . Disponível em: https://doi.org/10.3389/fragi.2021.727604. Acesso em: 10 jun. 2024.
APA
Barros, P. R. de, Costa, T. J. da, Akamine, E. H., & Passaglia, R. de C. A. T. (2021). Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence. Frontiers in Aging, 2, 1-22. doi:10.3389/fragi.2021.727604
NLM
Barros PR de, Costa TJ da, Akamine EH, Passaglia R de CAT. Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence [Internet]. Frontiers in Aging. 2021 ; 2 1-22.[citado 2024 jun. 10 ] Available from: https://doi.org/10.3389/fragi.2021.727604
Vancouver
Barros PR de, Costa TJ da, Akamine EH, Passaglia R de CAT. Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence [Internet]. Frontiers in Aging. 2021 ; 2 1-22.[citado 2024 jun. 10 ] Available from: https://doi.org/10.3389/fragi.2021.727604
Artigo de periodico
Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB (2020)
Senger, Nathalia ; Parletta, Aline C. ; Marques, Bruno Vinicius Duarte ; Campagnole-Santos, Maria Jose ; Santos, Robson A. S. ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná ; Akamine, Eliana Hiromi
Fonte: Journal of Cellular Physiology. Unidades: ICB, FMRP
Assuntos: FARMACOLOGIA, ANATOMIA, DOENÇAS CARDIOVASCULARES, GLÂNDULA TIREOIDE, ANGIOTENSINAS, TRIIODOTIRONINA, INSUFICIÊNCIA CARDÍACA, ANTIOXIDANTES, CATALASE, ATIVAÇÃO ENZIMÁTICA, HORMÔNIOS TIREOIDIANOS
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ABNT
SENGER, Nathalia et al. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB. Journal of Cellular Physiology, p. 14 , 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.30069. Acesso em: 10 jun. 2024.
APA
Senger, N., Parletta, A. C., Marques, B. V. D., Campagnole-Santos, M. J., Santos, R. A. S., Barreto-Chaves, M. L. M., et al. (2020). Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB. Journal of Cellular Physiology, 14 . doi:10.1002/jcp.30069
NLM
Senger N, Parletta AC, Marques BVD, Campagnole-Santos MJ, Santos RAS, Barreto-Chaves MLM, Diniz GP, Akamine EH. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB [Internet]. Journal of Cellular Physiology. 2020 ;14 .[citado 2024 jun. 10 ] Available from: https://doi.org/10.1002/jcp.30069
Vancouver
Senger N, Parletta AC, Marques BVD, Campagnole-Santos MJ, Santos RAS, Barreto-Chaves MLM, Diniz GP, Akamine EH. Angiotensin‐(1‐7) prevents T3‐induced cardiomyocyte hypertrophy by upregulating FOXO3/SOD1/catalase and downregulating NF‐ĸB [Internet]. Journal of Cellular Physiology. 2020 ;14 .[citado 2024 jun. 10 ] Available from: https://doi.org/10.1002/jcp.30069
Artigo de periodico
Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 (2019)
Costa, Tiago Januário ; Altayó, Francesc Jiménez ; Pereira, Cinthya Echem de Souza; Akamine, Eliana Hiromi ; Tostes, Rita de Cássia Aleixo ; Vila, Elisabet ; Dantas, Ana Paula ; Carvalho, Maria Helena Catelli de
Fonte: Cells. Unidades: ICB, FMRP
Assuntos: FAMACOLOGIA, ESTRÓGENOS, ARTÉRIAS CARÓTIDAS, EICOSANOIDES, ENSAIO CLÍNICO, ESTRADIOL, OVARIECTOMIA, CAMUNDONGOS
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ABNT
COSTA, Tiago Januário et al. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36. Cells, v. 8, n. 10, p. 17 , 2019Tradução . . Disponível em: https://doi.org/10.3390/cells8101217. Acesso em: 10 jun. 2024.
APA
Costa, T. J., Altayó, F. J., Pereira, C. E. de S., Akamine, E. H., Tostes, R. de C. A., Vila, E., et al. (2019). Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36. Cells, 8( 10), 17 . doi:10.3390/cells8101217
NLM
Costa TJ, Altayó FJ, Pereira CE de S, Akamine EH, Tostes R de CA, Vila E, Dantas AP, Carvalho MHC de. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 [Internet]. Cells. 2019 ; 8( 10): 17 .[citado 2024 jun. 10 ] Available from: https://doi.org/10.3390/cells8101217
Vancouver
Costa TJ, Altayó FJ, Pereira CE de S, Akamine EH, Tostes R de CA, Vila E, Dantas AP, Carvalho MHC de. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 [Internet]. Cells. 2019 ; 8( 10): 17 .[citado 2024 jun. 10 ] Available from: https://doi.org/10.3390/cells8101217
Artigo de periodico
Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension (2019)
Pereira, Cinthya Echem de Souza ; Costa, Tiago Januário da ; Oliveira, Vanessa; Colli, Lucas Giglio; Landgraf, Maristella de Almeida Vitta ; Franco, Maria do Carmo Pinho ; Landgraf, Richardt Gama ; Santos-Eichler, Rosângela Aparecida ; Bomfim, Gisele Facholi ; Rodrigues, Stephen Fernandes de Paula ; Akamine, Eliana Hiromi ; Carvalho, Maria Helena Catelli de
Fonte: Pharmacological Research. Unidades: ICB, FMRP
Assuntos: MITOCÔNDRIAS, DNA MITOCONDRIAL, DIFERENÇAS SEXUAIS ANIMAL, AORTA DE ANIMAL, RATOS WISTAR, ESTRESSE OXIDATIVO
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ABNT
PEREIRA, Cinthya Echem de Souza et al. Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension. Pharmacological Research, v. 144, p. 141-150, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2019.04.008. Acesso em: 10 jun. 2024.
APA
Pereira, C. E. de S., Costa, T. J. da, Oliveira, V., Colli, L. G., Landgraf, M. de A. V., Franco, M. do C. P., et al. (2019). Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension. Pharmacological Research, 144, 141-150. doi:10.1016/j.phrs.2019.04.008
NLM
Pereira CE de S, Costa TJ da, Oliveira V, Colli LG, Landgraf M de AV, Franco M do CP, Landgraf RG, Santos-Eichler RA, Bomfim GF, Rodrigues SF de P, Akamine EH, Carvalho MHC de. Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension [Internet]. Pharmacological Research. 2019 ; 144 141-150.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.phrs.2019.04.008
Vancouver
Pereira CE de S, Costa TJ da, Oliveira V, Colli LG, Landgraf M de AV, Franco M do CP, Landgraf RG, Santos-Eichler RA, Bomfim GF, Rodrigues SF de P, Akamine EH, Carvalho MHC de. Mitochondrial DNA: a new driver for sex differences in spontaneous hypertension [Internet]. Pharmacological Research. 2019 ; 144 141-150.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.phrs.2019.04.008
Artigo de periodico
H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction (2016)
Costa, Rafael M.; Filgueira, Fernando P.; Passaglia, Rita de Cássia Aleixo Tostes; Carvalho, Maria Helena C.; Akamine, Eliana Hiromi ; Lobato, Nubia S.
Fonte: Vascular Pharmacology. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 10 jun. 2024.
APA
Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008
NLM
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Vancouver
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Artigo de periodico
Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats (2015)
Bomfim, G. F.; Echem, Cinthya; Martins, C. B.; Costa, T. J.; Sartoretto, Simone Marcieli; Santos, R. A. dos; Oliveira, M. A.; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo; Webb, R. C.; Carvalho, Maria Helena Catelli de
Fonte: Life Sciences. Unidades: ICB, FMRP
Assuntos: FARMACOLOGIA, INFLAMAÇÃO, HIPERTENSÃO, RATOS
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ABNT
BOMFIM, G. F. et al. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, v. 122, p. 1-7, 2015Tradução . . Disponível em: http://lattes.cnpq.br/5055165961832291. Acesso em: 10 jun. 2024.
APA
Bomfim, G. F., Echem, C., Martins, C. B., Costa, T. J., Sartoretto, S. M., Santos, R. A. dos, et al. (2015). Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, 122, 1-7. doi:10.1016/j.lfs.2014.12.001
NLM
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 jun. 10 ] Available from: http://lattes.cnpq.br/5055165961832291
Vancouver
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 jun. 10 ] Available from: http://lattes.cnpq.br/5055165961832291
Artigo de periodico
Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells (2015)
Costa, Tiago Januário da; Ceravolo, Graziela Scalianti; Santos, Rosangela Aparecida dos; Oliveira, Maria Aparecida de; Araújo, Priscila Xavier de; Giaquinto, Luciana Rigueiral; Tostes, Rita de Cássia Aleixo; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Dantas, Ana Paula; Carvalho, Maria Helena Catelli de
Fonte: American Journal of Physiology: Heart and Circulatory Physiology. Unidades: ICB, FMRP
Assuntos: FARMACOLOGIA, TESTOSTERONA, ESTROGENOS, AORTA DE ANIMAL, CÉLULAS ENDOTELIAIS
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ABNT
COSTA, Tiago Januário da et al. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells. American Journal of Physiology: Heart and Circulatory Physiology, v. 308, n. 7, p. H723-H732, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00681.2014. Acesso em: 10 jun. 2024.
APA
Costa, T. J. da, Ceravolo, G. S., Santos, R. A. dos, Oliveira, M. A. de, Araújo, P. X. de, Giaquinto, L. R., et al. (2015). Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells. American Journal of Physiology: Heart and Circulatory Physiology, 308( 7), H723-H732. doi:10.1152/ajpheart.00681.2014
NLM
Costa TJ da, Ceravolo GS, Santos RA dos, Oliveira MA de, Araújo PX de, Giaquinto LR, Tostes R de CA, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC de. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2015 ; 308( 7): H723-H732.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1152/ajpheart.00681.2014
Vancouver
Costa TJ da, Ceravolo GS, Santos RA dos, Oliveira MA de, Araújo PX de, Giaquinto LR, Tostes R de CA, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC de. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2015 ; 308( 7): H723-H732.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1152/ajpheart.00681.2014
Artigo de periodico
An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies (2014)
Ceravolo, Graziela S.; Montezano, Augusto C.; Jordão, Maria T.; Akamine, Eliana Hiromi ; Costa, Tiago Januário da; Takano, Ana Paula Cremasco ; Fernandes, Denise C.; Barreto-Chaves, Maria Luiza Morais ; Laurindo, Francisco R.; Tostes, Rita de Cássia; Fortes, Zuleica Bruno; Chopard, Renato Paulo; Touyz, Rhian M.; Carvalho, Maria Helena Catelli de
Fonte: PLOS ONE. Unidades: ICB, FMRP
Assuntos: ANATOMIA, FARMACOLOGIA
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ABNT
CERAVOLO, Graziela S. et al. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, v. 9, n. 11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0111117. Acesso em: 10 jun. 2024.
APA
Ceravolo, G. S., Montezano, A. C., Jordão, M. T., Akamine, E. H., Costa, T. J. da, Takano, A. P. C., et al. (2014). An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, 9( 11). doi:10.1371/journal.pone.0111117
NLM
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 jun. 10 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Vancouver
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 jun. 10 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Artigo de periodico
Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats (2013)
Sartoretto, Simone Marcieli; Santos-Eichler, Rosângela; Tostes, Rita de Cássia Aleixo; Carvalho, Maria Helena Catelli de; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno
Fonte: Journal of Diabetes. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
SARTORETTO, Simone Marcieli et al. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, v. 5, n. 2, p. 197-206, 2013Tradução . . Acesso em: 10 jun. 2024.
APA
Sartoretto, S. M., Santos-Eichler, R., Tostes, R. de C. A., Carvalho, M. H. C. de, Akamine, E. H., & Fortes, Z. B. (2013). Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, 5( 2), 197-206.
NLM
Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 jun. 10 ]
Vancouver
Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 jun. 10 ]
Artigo de periodico
Mechanisms of endothelial dysfunction in obesity-associated hypertension (2012)
Lobato, Nubia de Souza; Filgueira, Fernando Paranaiba; Akamine, Eliana Hiromi ; Passaglia, Rita de Cassia Aleixo Tostes; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno
Fonte: Brazilian Journal of Medical and Biological Research. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
LOBATO, Nubia de Souza et al. Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, v. 45, n. 5, p. 392-400, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2012007500058. Acesso em: 10 jun. 2024.
APA
Lobato, N. de S., Filgueira, F. P., Akamine, E. H., Passaglia, R. de C. A. T., Carvalho, M. H. C. de, & Fortes, Z. B. (2012). Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, 45( 5), 392-400. doi:10.1590/S0100-879X2012007500058
NLM
Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
Vancouver
Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
Artigo de periodico
Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats (2012)
Bomfim, Gisele Facholi; Santos, Rosangela A. dos; Oliveira, Maria Aparecida; Giachini, Fernanda R.; Akamine, Eliana Hiromi ; Passaglia, Rita de Cássia Aleixo Tostes; Fortes, Zuleica Bruno; Webb, R. Clinton; Carvalho, Maria Helena Catelli de
Fonte: Clinical Science. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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ABNT
BOMFIM, Gisele Facholi et al. Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats. Clinical Science, v. 122, n. 11, p. 535-543, 2012Tradução . . Disponível em: https://doi.org/10.1042/CS20110523. Acesso em: 10 jun. 2024.
APA
Bomfim, G. F., Santos, R. A. dos, Oliveira, M. A., Giachini, F. R., Akamine, E. H., Passaglia, R. de C. A. T., et al. (2012). Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats. Clinical Science, 122( 11), 535-543. doi:10.1042/CS20110523
NLM
Bomfim GF, Santos RA dos, Oliveira MA, Giachini FR, Akamine EH, Passaglia R de CAT, Fortes ZB, Webb RC, Carvalho MHC de. Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats [Internet]. Clinical Science. 2012 ; 122( 11): 535-543.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1042/CS20110523
Vancouver
Bomfim GF, Santos RA dos, Oliveira MA, Giachini FR, Akamine EH, Passaglia R de CAT, Fortes ZB, Webb RC, Carvalho MHC de. Toll-like receptor 4 contributes to blood pressure regulation and vascular contraction in spontaneously hypertensive rats [Internet]. Clinical Science. 2012 ; 122( 11): 535-543.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1042/CS20110523
Artigo de periodico
Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats (2012)
Lobato, Núbia de Souza; Filgueira, Fernando Paranaiba; Hagihara, Graziela Neves; Akamine, Eliana Hiromi ; Pariz, J. R.; Passaglia, Rita de Cássia Aleixo Tostes; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno
Fonte: Life Sciences. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOBATO, Núbia de Souza et al. Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats. Life Sciences, v. 90, n. 5-6, p. 228-235, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2011.11.005. Acesso em: 10 jun. 2024.
APA
Lobato, N. de S., Filgueira, F. P., Hagihara, G. N., Akamine, E. H., Pariz, J. R., Passaglia, R. de C. A. T., et al. (2012). Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats. Life Sciences, 90( 5-6), 228-235. doi:10.1016/j.lfs.2011.11.005
NLM
Lobato N de S, Filgueira FP, Hagihara GN, Akamine EH, Pariz JR, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats [Internet]. Life Sciences. 2012 ; 90( 5-6): 228-235.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.lfs.2011.11.005
Vancouver
Lobato N de S, Filgueira FP, Hagihara GN, Akamine EH, Pariz JR, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Improvement of metabolic parameters and vascular function by metformin in obese non-diabetic rats [Internet]. Life Sciences. 2012 ; 90( 5-6): 228-235.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.lfs.2011.11.005
Artigo de periodico
Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats (2012)
Filgueira, Fernando Paranaíba; Lobato, Núbia de Souza; Dos Santos, R. A.; Oliveira, M. A.; Akamine, Eliana Hiromi ; Tostes-Passaglia, Rita de Cássia Aleixo; Fortes, Zuleica Bruno; Carvalho, Maria Helena Catelli de
Fonte: Life Sciences. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FILGUEIRA, Fernando Paranaíba et al. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats. Life Sciences, v. 90, n. 17-18, p. 689-694, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2012.01.017. Acesso em: 10 jun. 2024.
APA
Filgueira, F. P., Lobato, N. de S., Dos Santos, R. A., Oliveira, M. A., Akamine, E. H., Tostes-Passaglia, R. de C. A., et al. (2012). Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats. Life Sciences, 90( 17-18), 689-694. doi:10.1016/j.lfs.2012.01.017
NLM
Filgueira FP, Lobato N de S, Dos Santos RA, Oliveira MA, Akamine EH, Tostes-Passaglia R de CA, Fortes ZB, Carvalho MHC de. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats [Internet]. Life Sciences. 2012 ; 90( 17-18): 689-694.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
Vancouver
Filgueira FP, Lobato N de S, Dos Santos RA, Oliveira MA, Akamine EH, Tostes-Passaglia R de CA, Fortes ZB, Carvalho MHC de. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats [Internet]. Life Sciences. 2012 ; 90( 17-18): 689-694.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
Artigo de periodico
Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats (2011)
Camporez, João Paulo Gabriel; Akamine, Eliana Hiromi ; Davel, Ana Paula Couto; Franci, Celso Rodrigues; Rossoni, Luciana Venturini ; Carvalho, Carla Roberta de Oliveira
Fonte: Journal of Physiology. Unidades: ICB, FMRP
Assuntos: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAMPOREZ, João Paulo Gabriel et al. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, v. 589, n. 10, p. 2585-2596, 2011Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2011.206078. Acesso em: 10 jun. 2024.
APA
Camporez, J. P. G., Akamine, E. H., Davel, A. P. C., Franci, C. R., Rossoni, L. V., & Carvalho, C. R. de O. (2011). Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, 589( 10), 2585-2596. doi:10.1113/jphysiol.2011.206078
NLM
Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
Vancouver
Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078

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