ReP USP - Resultado da busca

Filtros : "Sarasin, Alain" "Rocha, Clarissa R. R." Removido: "Moraes, Maria Carolina S." Limpar

Filtros

Collaboration
Normalized affiliation
- universidade de são paulo (usp) (~2)
Non normalized affiliation
- commissariat à l’énergie atomique et aux énergies alternatives, fontenay-aux-roses, france (1)
- federal university of rio de janeiro, instituto de biofísica carlos chagas filho, rio de janeiro, brasil (1)
- fundación instituto leloir, consejo nacional de investigaciones científicas y técnicas, buenos aires, argentina (1)
- istituto di genetica molecolare, consiglio nazionale delle ricerche, pavia, italy (1)
- university paris-sud, institut gustave roussy, centre national de la recherche, villejuif, france (1)

Artigo de periodico
Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress (2013)
Soltys, Daniela Tathiana; Rocha, Clarissa R. R.; Lerner, Letícia K.; Souza, Tiago Antonio de; Munford, Veridiana; Cabral, Fernanda; Nardo, Tiziana; Stefanini, Miria; Sarasin, Alain; Cabral-Neto, Januário B.; Menck, Carlos Frederico Martins
Source: Human Mutation. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOLTYS, Daniela Tathiana et al. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress. Human Mutation, v. 34, n. 3, p. 481-489, 2013Tradução . . Disponível em: https://doi.org/10.1002/humu.22259. Acesso em: 16 abr. 2024.
APA
Soltys, D. T., Rocha, C. R. R., Lerner, L. K., Souza, T. A. de, Munford, V., Cabral, F., et al. (2013). Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress. Human Mutation, 34( 3), 481-489. doi:10.1002/humu.22259
NLM
Soltys DT, Rocha CRR, Lerner LK, Souza TA de, Munford V, Cabral F, Nardo T, Stefanini M, Sarasin A, Cabral-Neto JB, Menck CFM. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress [Internet]. Human Mutation. 2013 ; 34( 3): 481-489.[citado 2024 abr. 16 ] Available from: https://doi.org/10.1002/humu.22259
Vancouver
Soltys DT, Rocha CRR, Lerner LK, Souza TA de, Munford V, Cabral F, Nardo T, Stefanini M, Sarasin A, Cabral-Neto JB, Menck CFM. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress [Internet]. Human Mutation. 2013 ; 34( 3): 481-489.[citado 2024 abr. 16 ] Available from: https://doi.org/10.1002/humu.22259
Artigo de periodico
Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome (2014)
Quinet, Annabel; Vessoni, Alexandre Teixeira; Rocha, Clarissa R. R.; Gottifredi, Vanessa; Biard, Denis; Sarasin, Alain; Menck, Carlos Frederico Martins ; Stary, Anne
Source: DNA Repair. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
QUINET, Annabel et al. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome. DNA Repair, v. 14, p. 27-38, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.dnarep.2013.12.005. Acesso em: 16 abr. 2024.
APA
Quinet, A., Vessoni, A. T., Rocha, C. R. R., Gottifredi, V., Biard, D., Sarasin, A., et al. (2014). Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome. DNA Repair, 14, 27-38. doi:10.1016/j.dnarep.2013.12.005
NLM
Quinet A, Vessoni AT, Rocha CRR, Gottifredi V, Biard D, Sarasin A, Menck CFM, Stary A. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome [Internet]. DNA Repair. 2014 ; 14 27-38.[citado 2024 abr. 16 ] Available from: https://doi.org/10.1016/j.dnarep.2013.12.005
Vancouver
Quinet A, Vessoni AT, Rocha CRR, Gottifredi V, Biard D, Sarasin A, Menck CFM, Stary A. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome [Internet]. DNA Repair. 2014 ; 14 27-38.[citado 2024 abr. 16 ] Available from: https://doi.org/10.1016/j.dnarep.2013.12.005

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