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Artigo de periodico
Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients (2023)
Yurchenko, Andrey A. ; Rajabi, Fatemeh ; Braz-Petta, Tirzah ; Fassihi, Hiva ; Lehmann, Alan ; Nishigori, Chikako ; Wang, Jinxin; Padioleau, Ismael ; Gunbin, Konstantin ; Panunzi, Leonardo ; Morice-Picard, Fanny ; Laplante, Pierre; Robert, Caroline ; Kannouche, Patricia L. ; Sarasin, Alain ; Nikolaev, Sergey I. ; Menck, Carlos Frederico Martins
Fonte: Nature Communications. Unidade: ICB
Assuntos: MICROBIOLOPGIA, MUTAÇÃO GENÉTICA, REPARAÇÃO DE DNA, NEOPLASIAS CUTÂNEAS, DERMATOPATIAS, DOENÇAS GENÉTICAS, RADIAÇÃO ULTRAVIOLETA, GENÔMICA
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YURCHENKO, Andrey A. et al. Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients. Nature Communications, v. 14, p. 1-17, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41467-023-38311-0. Acesso em: 23 abr. 2024.
APA
Yurchenko, A. A., Rajabi, F., Braz-Petta, T., Fassihi, H., Lehmann, A., Nishigori, C., et al. (2023). Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients. Nature Communications, 14, 1-17. doi:10.1038/s41467-023-38311-0
NLM
Yurchenko AA, Rajabi F, Braz-Petta T, Fassihi H, Lehmann A, Nishigori C, Wang J, Padioleau I, Gunbin K, Panunzi L, Morice-Picard F, Laplante P, Robert C, Kannouche PL, Sarasin A, Nikolaev SI, Menck CFM. Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients [Internet]. Nature Communications. 2023 ; 14 1-17.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/s41467-023-38311-0
Vancouver
Yurchenko AA, Rajabi F, Braz-Petta T, Fassihi H, Lehmann A, Nishigori C, Wang J, Padioleau I, Gunbin K, Panunzi L, Morice-Picard F, Laplante P, Robert C, Kannouche PL, Sarasin A, Nikolaev SI, Menck CFM. Genomic mutation landscape of skin cancers from DNA repair-deficient xeroderma pigmentosum patients [Internet]. Nature Communications. 2023 ; 14 1-17.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/s41467-023-38311-0
Artigo de periodico
XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil (2022)
Castro, Ligia Pereira ; Vieira, Danilo Batista; Souza, Tiago Antonio de; Timoteo, Ana Rafaela de Souza ; Coutinho, Jessica Dayanna Landivar; Almeida, Isabel Cristina Pinheiro de ; Henriques, Sheila Ramos de Miranda; Azevedo, Fabio Medeiros de; Rosa, Reginaldo Cruz Alves ; Kannouche, Patricia L. ; Sarasin, Alain ; Menck, Carlos Frederico Martins ; Petta, Tirzah Braz
Fonte: Frontiers in Genetics. Unidades: ICB, FMRP
Assuntos: MICROBIOLOGIA, NEOPLASIAS CUTÂNEAS, DOENÇAS GENÉTICAS, RADIAÇÃO ULTRAVIOLETA, CÉLULAS CULTIVADAS DE TUMOR, SEQUENCIAMENTO GENÉTICO, GENES, MUTAÇÃO GENÉTICA, REPARAÇÃO DE DNA
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CASTRO, Ligia Pereira et al. XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil. Frontiers in Genetics, v. 12, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.3389/fgene.2021.784963. Acesso em: 23 abr. 2024.
APA
Castro, L. P., Vieira, D. B., Souza, T. A. de, Timoteo, A. R. de S., Coutinho, J. D. L., Almeida, I. C. P. de, et al. (2022). XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil. Frontiers in Genetics, 12, 1-8. doi:10.3389/fgene.2021.784963
NLM
Castro LP, Vieira DB, Souza TA de, Timoteo AR de S, Coutinho JDL, Almeida ICP de, Henriques SR de M, Azevedo FM de, Rosa RCA, Kannouche PL, Sarasin A, Menck CFM, Petta TB. XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil [Internet]. Frontiers in Genetics. 2022 ; 12 1-8.[citado 2024 abr. 23 ] Available from: https://doi.org/10.3389/fgene.2021.784963
Vancouver
Castro LP, Vieira DB, Souza TA de, Timoteo AR de S, Coutinho JDL, Almeida ICP de, Henriques SR de M, Azevedo FM de, Rosa RCA, Kannouche PL, Sarasin A, Menck CFM, Petta TB. XPC and POLH/XPV genes mutated in a genetic cluster of Xeroderma Pigmentosum patients in northeast Brazil [Internet]. Frontiers in Genetics. 2022 ; 12 1-8.[citado 2024 abr. 23 ] Available from: https://doi.org/10.3389/fgene.2021.784963
Artigo de periodico
Predominant role of DNA polymerase eta and p53-dependent translesion synthesis in the survival of ultraviolet-irradiated human cells (2017)
Lerner, Leticia Kock; Francisco, Guilherme; Soltys, Daniela T.; Rocha, Clarissa Ribeiro Reily; Quinet, Annabel; Vessoni, Alexandre Teixeira; Castro, Ligia Pereira; David, Taynah Ibrahim Picolo; Bustos, Silvina O.; Strauss, Bryan E.; Gottifredi, Vanesa; Stary, Anne; Sarasin, Alain; Chammas, Roger; Menck, Carlos Frederico Martins
Fonte: Nucleic Acids Research. Unidades: FM, ICB
Assuntos: DNA POLIMERASES, REPLICAÇÃO DO DNA, PROTEÍNAS PROTO-ONCOGÊNICAS, FIBROBLASTOS
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LERNER, Leticia Kock et al. Predominant role of DNA polymerase eta and p53-dependent translesion synthesis in the survival of ultraviolet-irradiated human cells. Nucleic Acids Research, v. 45, n. 3, p. 1270-1280, 2017Tradução . . Disponível em: https://doi.org/10.1093/nar/gkw1196. Acesso em: 23 abr. 2024.
APA
Lerner, L. K., Francisco, G., Soltys, D. T., Rocha, C. R. R., Quinet, A., Vessoni, A. T., et al. (2017). Predominant role of DNA polymerase eta and p53-dependent translesion synthesis in the survival of ultraviolet-irradiated human cells. Nucleic Acids Research, 45( 3), 1270-1280. doi:10.1093/nar/gkw1196
NLM
Lerner LK, Francisco G, Soltys DT, Rocha CRR, Quinet A, Vessoni AT, Castro LP, David TIP, Bustos SO, Strauss BE, Gottifredi V, Stary A, Sarasin A, Chammas R, Menck CFM. Predominant role of DNA polymerase eta and p53-dependent translesion synthesis in the survival of ultraviolet-irradiated human cells [Internet]. Nucleic Acids Research. 2017 ; 45( 3): 1270-1280.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1093/nar/gkw1196
Vancouver
Lerner LK, Francisco G, Soltys DT, Rocha CRR, Quinet A, Vessoni AT, Castro LP, David TIP, Bustos SO, Strauss BE, Gottifredi V, Stary A, Sarasin A, Chammas R, Menck CFM. Predominant role of DNA polymerase eta and p53-dependent translesion synthesis in the survival of ultraviolet-irradiated human cells [Internet]. Nucleic Acids Research. 2017 ; 45( 3): 1270-1280.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1093/nar/gkw1196
Artigo de periodico
Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome (2014)
Quinet, Annabel; Vessoni, Alexandre Teixeira; Rocha, Clarissa R. R.; Gottifredi, Vanessa; Biard, Denis; Sarasin, Alain; Menck, Carlos Frederico Martins ; Stary, Anne
Fonte: DNA Repair. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
QUINET, Annabel et al. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome. DNA Repair, v. 14, p. 27-38, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.dnarep.2013.12.005. Acesso em: 23 abr. 2024.
APA
Quinet, A., Vessoni, A. T., Rocha, C. R. R., Gottifredi, V., Biard, D., Sarasin, A., et al. (2014). Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome. DNA Repair, 14, 27-38. doi:10.1016/j.dnarep.2013.12.005
NLM
Quinet A, Vessoni AT, Rocha CRR, Gottifredi V, Biard D, Sarasin A, Menck CFM, Stary A. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome [Internet]. DNA Repair. 2014 ; 14 27-38.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.dnarep.2013.12.005
Vancouver
Quinet A, Vessoni AT, Rocha CRR, Gottifredi V, Biard D, Sarasin A, Menck CFM, Stary A. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome [Internet]. DNA Repair. 2014 ; 14 27-38.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.dnarep.2013.12.005
Artigo de periodico
Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress (2013)
Soltys, Daniela Tathiana; Rocha, Clarissa R. R.; Lerner, Letícia K.; Souza, Tiago Antonio de; Munford, Veridiana; Cabral, Fernanda; Nardo, Tiziana; Stefanini, Miria; Sarasin, Alain; Cabral-Neto, Januário B.; Menck, Carlos Frederico Martins
Fonte: Human Mutation. Unidade: ICB
Assunto: MICROBIOLOGIA
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SOLTYS, Daniela Tathiana et al. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress. Human Mutation, v. 34, n. 3, p. 481-489, 2013Tradução . . Disponível em: https://doi.org/10.1002/humu.22259. Acesso em: 23 abr. 2024.
APA
Soltys, D. T., Rocha, C. R. R., Lerner, L. K., Souza, T. A. de, Munford, V., Cabral, F., et al. (2013). Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress. Human Mutation, 34( 3), 481-489. doi:10.1002/humu.22259
NLM
Soltys DT, Rocha CRR, Lerner LK, Souza TA de, Munford V, Cabral F, Nardo T, Stefanini M, Sarasin A, Cabral-Neto JB, Menck CFM. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress [Internet]. Human Mutation. 2013 ; 34( 3): 481-489.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1002/humu.22259
Vancouver
Soltys DT, Rocha CRR, Lerner LK, Souza TA de, Munford V, Cabral F, Nardo T, Stefanini M, Sarasin A, Cabral-Neto JB, Menck CFM. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress [Internet]. Human Mutation. 2013 ; 34( 3): 481-489.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1002/humu.22259
Artigo de periodico
Both XPA and DNA polymerase eta are necessary for the repair of doxorubicin-induced DNA lesions (2012)
Moraes, Maria Carolina S.; Andrade, Annabel Quinet de; Carvalho, Helotonio; Guecheva, Temenouga; Agnoletto, Mateus H.; Henriques, João A. P.; Sarasin, Alain; Stary, Anne; Saffi, Jenifer; Menck, Carlos Frederico Martins
Fonte: Cancer Letters. Unidade: ICB
Assunto: MICROBIOLOGIA
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MORAES, Maria Carolina S. et al. Both XPA and DNA polymerase eta are necessary for the repair of doxorubicin-induced DNA lesions. Cancer Letters, v. 314, n. 1, p. 108-118, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.canlet.2011.09.019. Acesso em: 23 abr. 2024.
APA
Moraes, M. C. S., Andrade, A. Q. de, Carvalho, H., Guecheva, T., Agnoletto, M. H., Henriques, J. A. P., et al. (2012). Both XPA and DNA polymerase eta are necessary for the repair of doxorubicin-induced DNA lesions. Cancer Letters, 314( 1), 108-118. doi:10.1016/j.canlet.2011.09.019
NLM
Moraes MCS, Andrade AQ de, Carvalho H, Guecheva T, Agnoletto MH, Henriques JAP, Sarasin A, Stary A, Saffi J, Menck CFM. Both XPA and DNA polymerase eta are necessary for the repair of doxorubicin-induced DNA lesions [Internet]. Cancer Letters. 2012 ; 314( 1): 108-118.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.canlet.2011.09.019
Vancouver
Moraes MCS, Andrade AQ de, Carvalho H, Guecheva T, Agnoletto MH, Henriques JAP, Sarasin A, Stary A, Saffi J, Menck CFM. Both XPA and DNA polymerase eta are necessary for the repair of doxorubicin-induced DNA lesions [Internet]. Cancer Letters. 2012 ; 314( 1): 108-118.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.canlet.2011.09.019
Artigo de periodico
Defective transcription/repair factor IIH recruitment to specific UV lesions in Trichothiodystrophy syndrome (2008)
Chiganças, Vanessa; Lima-Bessa, Keronninn M.; Stary, Anne; Menck, Carlos Frederico Martins ; Sarasin, Alain
Fonte: Cancer Research. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
CHIGANÇAS, Vanessa et al. Defective transcription/repair factor IIH recruitment to specific UV lesions in Trichothiodystrophy syndrome. Cancer Research, v. 68, n. 15, p. 6074-6083, 2008Tradução . . Acesso em: 23 abr. 2024.
APA
Chiganças, V., Lima-Bessa, K. M., Stary, A., Menck, C. F. M., & Sarasin, A. (2008). Defective transcription/repair factor IIH recruitment to specific UV lesions in Trichothiodystrophy syndrome. Cancer Research, 68( 15), 6074-6083.
NLM
Chiganças V, Lima-Bessa KM, Stary A, Menck CFM, Sarasin A. Defective transcription/repair factor IIH recruitment to specific UV lesions in Trichothiodystrophy syndrome. Cancer Research. 2008 ; 68( 15): 6074-6083.[citado 2024 abr. 23 ]
Vancouver
Chiganças V, Lima-Bessa KM, Stary A, Menck CFM, Sarasin A. Defective transcription/repair factor IIH recruitment to specific UV lesions in Trichothiodystrophy syndrome. Cancer Research. 2008 ; 68( 15): 6074-6083.[citado 2024 abr. 23 ]
Artigo de periodico
Resistance to ultraviolet-induced apoptosis in DNA repair deficient growth arrested human fibroblasts is not related to recovery from RNA transcription blockage (2008)
Costa, Renata M. A. da; Quayle, Carolina; Jacysyn, Jaqueline de Fátima; Amarante-Mendes, João Gustavo Pessini ; Sarasin, Alain; Menck, Carlos Frederico Martins
Fonte: Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis. Unidade: ICB
Assuntos: MICROBIOLOGIA, IMUNOLOGIA
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ABNT
COSTA, Renata M. A. da et al. Resistance to ultraviolet-induced apoptosis in DNA repair deficient growth arrested human fibroblasts is not related to recovery from RNA transcription blockage. Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis, v. 640, n. 1-2, p. 1-7, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.mrfmmm.2007.11.010. Acesso em: 23 abr. 2024.
APA
Costa, R. M. A. da, Quayle, C., Jacysyn, J. de F., Amarante-Mendes, J. G. P., Sarasin, A., & Menck, C. F. M. (2008). Resistance to ultraviolet-induced apoptosis in DNA repair deficient growth arrested human fibroblasts is not related to recovery from RNA transcription blockage. Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis, 640( 1-2), 1-7. doi:10.1016/j.mrfmmm.2007.11.010
NLM
Costa RMA da, Quayle C, Jacysyn J de F, Amarante-Mendes JGP, Sarasin A, Menck CFM. Resistance to ultraviolet-induced apoptosis in DNA repair deficient growth arrested human fibroblasts is not related to recovery from RNA transcription blockage [Internet]. Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis. 2008 ; 640( 1-2): 1-7.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.mrfmmm.2007.11.010
Vancouver
Costa RMA da, Quayle C, Jacysyn J de F, Amarante-Mendes JGP, Sarasin A, Menck CFM. Resistance to ultraviolet-induced apoptosis in DNA repair deficient growth arrested human fibroblasts is not related to recovery from RNA transcription blockage [Internet]. Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis. 2008 ; 640( 1-2): 1-7.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.mrfmmm.2007.11.010
Artigo de periodico
CPDs and 6-4PPs play different roles in UV-induced cell death in normal and NER-deficient human cells (2008)
Lima-Bessa, Keronninn Moreno de; Armelini, Melissa Gava; Chinganças, Vanessa; Jacysyn, Jacqueline F.; Amarante-Mendes, João Gustavo Pessini ; Sarasin, Alain; Menck, Carlos Frederico Martins
Fonte: DNA Repair. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
LIMA-BESSA, Keronninn Moreno de et al. CPDs and 6-4PPs play different roles in UV-induced cell death in normal and NER-deficient human cells. DNA Repair, v. 7, n. 2, p. 303-312, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.dnarep.2007.11.003. Acesso em: 23 abr. 2024.
APA
Lima-Bessa, K. M. de, Armelini, M. G., Chinganças, V., Jacysyn, J. F., Amarante-Mendes, J. G. P., Sarasin, A., & Menck, C. F. M. (2008). CPDs and 6-4PPs play different roles in UV-induced cell death in normal and NER-deficient human cells. DNA Repair, 7( 2), 303-312. doi:10.1016/j.dnarep.2007.11.003
NLM
Lima-Bessa KM de, Armelini MG, Chinganças V, Jacysyn JF, Amarante-Mendes JGP, Sarasin A, Menck CFM. CPDs and 6-4PPs play different roles in UV-induced cell death in normal and NER-deficient human cells [Internet]. DNA Repair. 2008 ; 7( 2): 303-312.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.dnarep.2007.11.003
Vancouver
Lima-Bessa KM de, Armelini MG, Chinganças V, Jacysyn JF, Amarante-Mendes JGP, Sarasin A, Menck CFM. CPDs and 6-4PPs play different roles in UV-induced cell death in normal and NER-deficient human cells [Internet]. DNA Repair. 2008 ; 7( 2): 303-312.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.dnarep.2007.11.003
Trabalho de evento-resumo
XPD mutations in the trichothiodystrophy syndrome affect the recruitment of the TFIIH factor only to CPD-containing sites (2007)
Chiganças, Vanessa; Lima-Bessa, Keronninn Moreno; Menck, Carlos Frederico Martins ; Sarasin, Alain
Fonte: Resumos. Nome do evento: Meeting in fundamental aspects of DNA repair and mutagenesis. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
CHIGANÇAS, Vanessa et al. XPD mutations in the trichothiodystrophy syndrome affect the recruitment of the TFIIH factor only to CPD-containing sites. 2007, Anais.. São Paulo: Universidade de São Paulo, 2007. . Acesso em: 23 abr. 2024.
APA
Chiganças, V., Lima-Bessa, K. M., Menck, C. F. M., & Sarasin, A. (2007). XPD mutations in the trichothiodystrophy syndrome affect the recruitment of the TFIIH factor only to CPD-containing sites. In Resumos. São Paulo: Universidade de São Paulo.
NLM
Chiganças V, Lima-Bessa KM, Menck CFM, Sarasin A. XPD mutations in the trichothiodystrophy syndrome affect the recruitment of the TFIIH factor only to CPD-containing sites. Resumos. 2007 ;[citado 2024 abr. 23 ]
Vancouver
Chiganças V, Lima-Bessa KM, Menck CFM, Sarasin A. XPD mutations in the trichothiodystrophy syndrome affect the recruitment of the TFIIH factor only to CPD-containing sites. Resumos. 2007 ;[citado 2024 abr. 23 ]
Artigo de periodico
Transcriptional profiles of unirradiated or UV-irradiated human cells expressing either the cancer-prone XPB/CS allele or the noncancer-prone XPB/TTD allele (2005)
Costa, Renata Maria Augusto da; Riou, Lydia; Paquola, Apuã; Menck, Carlos Frederico Martins ; Sarasin, Alain
Fonte: Oncogene. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
COSTA, Renata Maria Augusto da et al. Transcriptional profiles of unirradiated or UV-irradiated human cells expressing either the cancer-prone XPB/CS allele or the noncancer-prone XPB/TTD allele. Oncogene, v. 24, p. 1359-1374, 2005Tradução . . Disponível em: https://doi.org/10.1038/sj.onc.1208288. Acesso em: 23 abr. 2024.
APA
Costa, R. M. A. da, Riou, L., Paquola, A., Menck, C. F. M., & Sarasin, A. (2005). Transcriptional profiles of unirradiated or UV-irradiated human cells expressing either the cancer-prone XPB/CS allele or the noncancer-prone XPB/TTD allele. Oncogene, 24, 1359-1374. doi:10.1038/sj.onc.1208288
NLM
Costa RMA da, Riou L, Paquola A, Menck CFM, Sarasin A. Transcriptional profiles of unirradiated or UV-irradiated human cells expressing either the cancer-prone XPB/CS allele or the noncancer-prone XPB/TTD allele [Internet]. Oncogene. 2005 ; 24 1359-1374.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/sj.onc.1208288
Vancouver
Costa RMA da, Riou L, Paquola A, Menck CFM, Sarasin A. Transcriptional profiles of unirradiated or UV-irradiated human cells expressing either the cancer-prone XPB/CS allele or the noncancer-prone XPB/TTD allele [Internet]. Oncogene. 2005 ; 24 1359-1374.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/sj.onc.1208288
Artigo de periodico
Restoring DNA repair capacity of cells from three distinct diseases by XPD gene-recombinant adenovirus (2005)
Armelini, Melissa Gava; Muotri, Alysson Renato; Marchetto, Maria Carolina Nasser; Lima-Bessa, Keronninn Moreno; Sarasin, Alain; Menck, Carlos Frederico Martins
Fonte: Cancer Gene Therapy. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
ARMELINI, Melissa Gava et al. Restoring DNA repair capacity of cells from three distinct diseases by XPD gene-recombinant adenovirus. Cancer Gene Therapy, v. 12, p. 389-396, 2005Tradução . . Disponível em: https://doi.org/10.1038/sj.cgt.7700797. Acesso em: 23 abr. 2024.
APA
Armelini, M. G., Muotri, A. R., Marchetto, M. C. N., Lima-Bessa, K. M., Sarasin, A., & Menck, C. F. M. (2005). Restoring DNA repair capacity of cells from three distinct diseases by XPD gene-recombinant adenovirus. Cancer Gene Therapy, 12, 389-396. doi:10.1038/sj.cgt.7700797
NLM
Armelini MG, Muotri AR, Marchetto MCN, Lima-Bessa KM, Sarasin A, Menck CFM. Restoring DNA repair capacity of cells from three distinct diseases by XPD gene-recombinant adenovirus [Internet]. Cancer Gene Therapy. 2005 ; 12 389-396.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/sj.cgt.7700797
Vancouver
Armelini MG, Muotri AR, Marchetto MCN, Lima-Bessa KM, Sarasin A, Menck CFM. Restoring DNA repair capacity of cells from three distinct diseases by XPD gene-recombinant adenovirus [Internet]. Cancer Gene Therapy. 2005 ; 12 389-396.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/sj.cgt.7700797
Artigo de periodico
CPD - photolyase adenovirus - mediated gene transfer in normal and DNA - repair - deficient human cells (2004)
Chiganças, Vanessa; Sarasin, Alain; Menck, Carlos Frederico Martins
Fonte: Journal of Cell Science. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
CHIGANÇAS, Vanessa e SARASIN, Alain e MENCK, Carlos Frederico Martins. CPD - photolyase adenovirus - mediated gene transfer in normal and DNA - repair - deficient human cells. Journal of Cell Science, v. 117, p. 3579-3592, 2004Tradução . . Acesso em: 23 abr. 2024.
APA
Chiganças, V., Sarasin, A., & Menck, C. F. M. (2004). CPD - photolyase adenovirus - mediated gene transfer in normal and DNA - repair - deficient human cells. Journal of Cell Science, 117, 3579-3592.
NLM
Chiganças V, Sarasin A, Menck CFM. CPD - photolyase adenovirus - mediated gene transfer in normal and DNA - repair - deficient human cells. Journal of Cell Science. 2004 ; 117 3579-3592.[citado 2024 abr. 23 ]
Vancouver
Chiganças V, Sarasin A, Menck CFM. CPD - photolyase adenovirus - mediated gene transfer in normal and DNA - repair - deficient human cells. Journal of Cell Science. 2004 ; 117 3579-3592.[citado 2024 abr. 23 ]
Artigo de periodico
Effect of cell confluence on ultraviolet light apoptotic responses in DNA repair deficient cells (2003)
Carvalho, Helotonio; Costa, Renata Maria Augusto da; Chiganças, Vanessa; Weinlich, Ricardo; Brumatti, Gabriela; Amarante-Mendes, João Gustavo Pessini ; Sarasin, Alain; Menck, Carlos Frederico Martins
Fonte: Mutation Research. Unidade: ICB
Assunto: PARASITOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, Helotonio et al. Effect of cell confluence on ultraviolet light apoptotic responses in DNA repair deficient cells. Mutation Research, v. 544, p. 159-166, 2003Tradução . . Disponível em: https://doi.org/10.1016/j.mrrev.2003.06.004. Acesso em: 23 abr. 2024.
APA
Carvalho, H., Costa, R. M. A. da, Chiganças, V., Weinlich, R., Brumatti, G., Amarante-Mendes, J. G. P., et al. (2003). Effect of cell confluence on ultraviolet light apoptotic responses in DNA repair deficient cells. Mutation Research, 544, 159-166. doi:10.1016/j.mrrev.2003.06.004
NLM
Carvalho H, Costa RMA da, Chiganças V, Weinlich R, Brumatti G, Amarante-Mendes JGP, Sarasin A, Menck CFM. Effect of cell confluence on ultraviolet light apoptotic responses in DNA repair deficient cells [Internet]. Mutation Research. 2003 ; 544 159-166.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.mrrev.2003.06.004
Vancouver
Carvalho H, Costa RMA da, Chiganças V, Weinlich R, Brumatti G, Amarante-Mendes JGP, Sarasin A, Menck CFM. Effect of cell confluence on ultraviolet light apoptotic responses in DNA repair deficient cells [Internet]. Mutation Research. 2003 ; 544 159-166.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/j.mrrev.2003.06.004
Artigo de periodico
Complementation of the DNA repair deficiency in human Xeroderma Pigmentosum group A and C cells by recombinant adenovirus-mediated gene transfer (2002)
Muotri, Alysson Renato; Marchetto, Maria Carolina Nasser; Zerbini, Luiz Fernando Celidônio; Libermann, Towia A.; Ventura, Armando Morais ; Sarasin, Alain; Menck, Carlos Frederico Martins
Fonte: Human Gene Therapy. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MUOTRI, Alysson Renato et al. Complementation of the DNA repair deficiency in human Xeroderma Pigmentosum group A and C cells by recombinant adenovirus-mediated gene transfer. Human Gene Therapy, v. 13, p. 1833-1844, 2002Tradução . . Acesso em: 23 abr. 2024.
APA
Muotri, A. R., Marchetto, M. C. N., Zerbini, L. F. C., Libermann, T. A., Ventura, A. M., Sarasin, A., & Menck, C. F. M. (2002). Complementation of the DNA repair deficiency in human Xeroderma Pigmentosum group A and C cells by recombinant adenovirus-mediated gene transfer. Human Gene Therapy, 13, 1833-1844.
NLM
Muotri AR, Marchetto MCN, Zerbini LFC, Libermann TA, Ventura AM, Sarasin A, Menck CFM. Complementation of the DNA repair deficiency in human Xeroderma Pigmentosum group A and C cells by recombinant adenovirus-mediated gene transfer. Human Gene Therapy. 2002 ;13 1833-1844.[citado 2024 abr. 23 ]
Vancouver
Muotri AR, Marchetto MCN, Zerbini LFC, Libermann TA, Ventura AM, Sarasin A, Menck CFM. Complementation of the DNA repair deficiency in human Xeroderma Pigmentosum group A and C cells by recombinant adenovirus-mediated gene transfer. Human Gene Therapy. 2002 ;13 1833-1844.[citado 2024 abr. 23 ]
Artigo de periodico
Mutagenic fingerprint of ozone in human cells (2002)
Jorge, Soraia Attié Calil; Menck, Carlos Frederico Martins ; Sies, Helmut; Osborne, Martin R; Phillips, David H; Sarasin, Alain; Stary, Anne
Fonte: DNA Repair. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JORGE, Soraia Attié Calil et al. Mutagenic fingerprint of ozone in human cells. DNA Repair, p. 369-378, 2002Tradução . . Disponível em: https://doi.org/10.1016/s1568-7864(02)00011-3. Acesso em: 23 abr. 2024.
APA
Jorge, S. A. C., Menck, C. F. M., Sies, H., Osborne, M. R., Phillips, D. H., Sarasin, A., & Stary, A. (2002). Mutagenic fingerprint of ozone in human cells. DNA Repair, 369-378. doi:10.1016/s1568-7864(02)00011-3
NLM
Jorge SAC, Menck CFM, Sies H, Osborne MR, Phillips DH, Sarasin A, Stary A. Mutagenic fingerprint of ozone in human cells [Internet]. DNA Repair. 2002 ; 369-378.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/s1568-7864(02)00011-3
Vancouver
Jorge SAC, Menck CFM, Sies H, Osborne MR, Phillips DH, Sarasin A, Stary A. Mutagenic fingerprint of ozone in human cells [Internet]. DNA Repair. 2002 ; 369-378.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1016/s1568-7864(02)00011-3

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