ReP USP - Resultado da busca

Artigo de periodico
Expression of B lymphocyte-induced maturation protein 1 (Blimp-1) in keratinocyte and cytokine signalling drives human Th17 response in psoriasis (2023)
Costa, Lorena Carla Oliveira da; Gardinassi, Luiz Gustavo Araujo ; Veras, Flavio Protasio; Milanezi, Cristiane Maria; Ramalho, Leandra Naira Zambelli ; Benevides, Luciana; Alves Filho, José Carlos Farias ; Silva, João Santana da ; Souza, Cacilda da Silva
Source: Archives of Dermatological Research. Unidades: FCFRP, EERP, FMRP
Subjects: PSORÍASE, PROTEÍNAS, LINFÓCITOS, INTERLEUCINAS, CRESCIMENTO, CÉLULAS, CITOCINAS
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COSTA, Lorena Carla Oliveira da et al. Expression of B lymphocyte-induced maturation protein 1 (Blimp-1) in keratinocyte and cytokine signalling drives human Th17 response in psoriasis. Archives of Dermatological Research, v. 315, n. 3, p. 481-490, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00403-022-02379-3. Acesso em: 13 jun. 2024.
APA
Costa, L. C. O. da, Gardinassi, L. G. A., Veras, F. P., Milanezi, C. M., Ramalho, L. N. Z., Benevides, L., et al. (2023). Expression of B lymphocyte-induced maturation protein 1 (Blimp-1) in keratinocyte and cytokine signalling drives human Th17 response in psoriasis. Archives of Dermatological Research, 315( 3), 481-490. doi:10.1007/s00403-022-02379-3
NLM
Costa LCO da, Gardinassi LGA, Veras FP, Milanezi CM, Ramalho LNZ, Benevides L, Alves Filho JCF, Silva JS da, Souza C da S. Expression of B lymphocyte-induced maturation protein 1 (Blimp-1) in keratinocyte and cytokine signalling drives human Th17 response in psoriasis [Internet]. Archives of Dermatological Research. 2023 ; 315( 3): 481-490.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1007/s00403-022-02379-3
Vancouver
Costa LCO da, Gardinassi LGA, Veras FP, Milanezi CM, Ramalho LNZ, Benevides L, Alves Filho JCF, Silva JS da, Souza C da S. Expression of B lymphocyte-induced maturation protein 1 (Blimp-1) in keratinocyte and cytokine signalling drives human Th17 response in psoriasis [Internet]. Archives of Dermatological Research. 2023 ; 315( 3): 481-490.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1007/s00403-022-02379-3
Artigo de periodico
Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection (2022)
Maruyama, Sandra Regina; Fuzo, Carlos Alessandro ; Oliveira, Antonio Edson Rocha; Rogerio, Luana Aparecida; Takamiya, Nayore Tamie; Pessenda, Gabriela; Melo, Enaldo Vieira de; Silva, Ângela Maria da; Jesus, Amelia Ribeiro de; Carregaro, Vanessa ; Nakaya, Helder Takashi Imoto ; Almeida, Roque Pacheco de; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, FCF, FCFRP
Subjects: LEISHMANIOSE VISCERAL, LEISHMANIA, RNA, SEQUENCIAMENTO GENÉTICO
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MARUYAMA, Sandra Regina et al. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection. Frontiers in Immunology, v. 13, p. 1-18, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.784463. Acesso em: 13 jun. 2024.
APA
Maruyama, S. R., Fuzo, C. A., Oliveira, A. E. R., Rogerio, L. A., Takamiya, N. T., Pessenda, G., et al. (2022). Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection. Frontiers in Immunology, 13, 1-18. doi:10.3389/fimmu.2022.784463
NLM
Maruyama SR, Fuzo CA, Oliveira AER, Rogerio LA, Takamiya NT, Pessenda G, Melo EV de, Silva ÂM da, Jesus AR de, Carregaro V, Nakaya HTI, Almeida RP de, Silva JS da. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection [Internet]. Frontiers in Immunology. 2022 ; 13 1-18.[citado 2024 jun. 13 ] Available from: https://doi.org/10.3389/fimmu.2022.784463
Vancouver
Maruyama SR, Fuzo CA, Oliveira AER, Rogerio LA, Takamiya NT, Pessenda G, Melo EV de, Silva ÂM da, Jesus AR de, Carregaro V, Nakaya HTI, Almeida RP de, Silva JS da. Insight into the long noncoding RNA and mRNA coexpression profile in the human blood transcriptome upon Leishmania infantum infection [Internet]. Frontiers in Immunology. 2022 ; 13 1-18.[citado 2024 jun. 13 ] Available from: https://doi.org/10.3389/fimmu.2022.784463
Artigo de periodico
Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy (2022)
Silva, Maria Cláudia da; Fuzo, Carlos Alessandro ; Paiva, Isadora Marques ; Bibo, Naira Lopes; Oliveira, Maykon Tavares de; Soares, Hellen Anastácia da Silva; Chevillard, Christophe; Kalil Filho, Jorge Elias ; Cunha Neto, Edecio ; Cunha, Thiago Mattar ; Silva, João Santana da
Source: Immunobiology. Unidades: FM, FMRP, FCFRP
Subjects: MUTAÇÃO, DOENÇA DE CHAGAS, POLIMORFISMO
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SILVA, Maria Cláudia da et al. Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy. Immunobiology, v. 227, n. 5, p. 1-7, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2022.152242. Acesso em: 13 jun. 2024.
APA
Silva, M. C. da, Fuzo, C. A., Paiva, I. M., Bibo, N. L., Oliveira, M. T. de, Soares, H. A. da S., et al. (2022). Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy. Immunobiology, 227( 5), 1-7. doi:10.1016/j.imbio.2022.152242
NLM
Silva MC da, Fuzo CA, Paiva IM, Bibo NL, Oliveira MT de, Soares HA da S, Chevillard C, Kalil Filho JE, Cunha Neto E, Cunha TM, Silva JS da. Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy [Internet]. Immunobiology. 2022 ; 227( 5): 1-7.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.imbio.2022.152242
Vancouver
Silva MC da, Fuzo CA, Paiva IM, Bibo NL, Oliveira MT de, Soares HA da S, Chevillard C, Kalil Filho JE, Cunha Neto E, Cunha TM, Silva JS da. Synonymous mutation rs1129293 is associated with PIK3CG expression and PI3Kγ activation in patients with chronic Chagas cardiomyopathy [Internet]. Immunobiology. 2022 ; 227( 5): 1-7.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.imbio.2022.152242
Artigo de periodico
NLRC4 inhibits NLRP3 inflammasome and abrogates effective antifungal CD8 + T cell responses (2021)
Souza, Camila Oliveira de; Ketelut-Carneiro, Natália; Milanezi, Cristiane Maria; Faccioli, Lucia Helena ; Gardinassi, Luiz G; Silva, João Santana da
Source: iScience. Unidades: EERP, FCFRP, FMRP
Subjects: IMUNOLOGIA, MICOLOGIA, ANTIFÚNGICOS
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SOUZA, Camila Oliveira de et al. NLRC4 inhibits NLRP3 inflammasome and abrogates effective antifungal CD8 + T cell responses. iScience, v. 24, n. 6, p. 1-19, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2021.102548. Acesso em: 13 jun. 2024.
APA
Souza, C. O. de, Ketelut-Carneiro, N., Milanezi, C. M., Faccioli, L. H., Gardinassi, L. G., & Silva, J. S. da. (2021). NLRC4 inhibits NLRP3 inflammasome and abrogates effective antifungal CD8 + T cell responses. iScience, 24( 6), 1-19. doi:10.1016/j.isci.2021.102548
NLM
Souza CO de, Ketelut-Carneiro N, Milanezi CM, Faccioli LH, Gardinassi LG, Silva JS da. NLRC4 inhibits NLRP3 inflammasome and abrogates effective antifungal CD8 + T cell responses [Internet]. iScience. 2021 ; 24( 6): 1-19.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.isci.2021.102548
Vancouver
Souza CO de, Ketelut-Carneiro N, Milanezi CM, Faccioli LH, Gardinassi LG, Silva JS da. NLRC4 inhibits NLRP3 inflammasome and abrogates effective antifungal CD8 + T cell responses [Internet]. iScience. 2021 ; 24( 6): 1-19.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.isci.2021.102548
Artigo de jornal-dep/entr
Substâncias estudadas pela USP de São Carlos se mostram 100% eficazes contra a Covid-19 (2021)
Albuquerque, Sérgio de ; Martins, Felipe Cardoso Prado ; Montanari, Carlos Alberto ; Silva, João Santana da
Source: Jornal da EPTV 2a. Edição. Unidades: FCFRP, IQSC, FMRP
Subjects: DOENÇA DE CHAGAS, MOLÉCULA, CORONAVIRUS
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ALBUQUERQUE, Sérgio de et al. Substâncias estudadas pela USP de São Carlos se mostram 100% eficazes contra a Covid-19. Jornal da EPTV 2a. Edição. São Carlos: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://g1.globo.com/sp/sao-carlos-regiao/noticia/2021/11/10/substancias-estudadas-pela-usp-de-sao-carlos-se-mostram-100percent-eficazes-contra-a-covid-19.ghtml. Acesso em: 13 jun. 2024. , 2021
APA
Albuquerque, S. de, Martins, F. C. P., Montanari, C. A., & Silva, J. S. da. (2021). Substâncias estudadas pela USP de São Carlos se mostram 100% eficazes contra a Covid-19. Jornal da EPTV 2a. Edição. São Carlos: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. Recuperado de https://g1.globo.com/sp/sao-carlos-regiao/noticia/2021/11/10/substancias-estudadas-pela-usp-de-sao-carlos-se-mostram-100percent-eficazes-contra-a-covid-19.ghtml
NLM
Albuquerque S de, Martins FCP, Montanari CA, Silva JS da. Substâncias estudadas pela USP de São Carlos se mostram 100% eficazes contra a Covid-19 [Internet]. Jornal da EPTV 2a. Edição. 2021 ;[citado 2024 jun. 13 ] Available from: https://g1.globo.com/sp/sao-carlos-regiao/noticia/2021/11/10/substancias-estudadas-pela-usp-de-sao-carlos-se-mostram-100percent-eficazes-contra-a-covid-19.ghtml
Vancouver
Albuquerque S de, Martins FCP, Montanari CA, Silva JS da. Substâncias estudadas pela USP de São Carlos se mostram 100% eficazes contra a Covid-19 [Internet]. Jornal da EPTV 2a. Edição. 2021 ;[citado 2024 jun. 13 ] Available from: https://g1.globo.com/sp/sao-carlos-regiao/noticia/2021/11/10/substancias-estudadas-pela-usp-de-sao-carlos-se-mostram-100percent-eficazes-contra-a-covid-19.ghtml
Artigo de periodico
Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment (2020)
Albuquerque, Sérgio de ; Cianni, Lorenzo ; Vita, Daniela de ; Lopes, Carla Duque ; Gomes, Ana S. M.; Gomes, Paula; Laughton, Charles; Leitão, Andrei ; Montanari, Carlos Alberto ; Montanari, Raphael ; Ribeiro, Jean Francisco Rosa ; Silva, João Santana da ; Teixeira, Cátia
Source: Chemical Biology & Drug Design. Unidades: FCFRP, IQSC, FMRP, EESC, ICMC
Subjects: DOENÇA DE CHAGAS, TRYPANOSOMA CRUZI
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ALBUQUERQUE, Sérgio de et al. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment. Chemical Biology & Drug Design, v. 96, p. 948–960, 2020Tradução . . Disponível em: https://doi.org/10.1111/cbdd.13663. Acesso em: 13 jun. 2024.
APA
Albuquerque, S. de, Cianni, L., Vita, D. de, Lopes, C. D., Gomes, A. S. M., Gomes, P., et al. (2020). Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment. Chemical Biology & Drug Design, 96, 948–960. doi:10.1111/cbdd.13663
NLM
Albuquerque S de, Cianni L, Vita D de, Lopes CD, Gomes ASM, Gomes P, Laughton C, Leitão A, Montanari CA, Montanari R, Ribeiro JFR, Silva JS da, Teixeira C. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment [Internet]. Chemical Biology & Drug Design. 2020 ; 96 948–960.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1111/cbdd.13663
Vancouver
Albuquerque S de, Cianni L, Vita D de, Lopes CD, Gomes ASM, Gomes P, Laughton C, Leitão A, Montanari CA, Montanari R, Ribeiro JFR, Silva JS da, Teixeira C. Molecular design aided by random forests and synthesis of potent trypanocidal agents as cruzain inhibitors for Chagas disease treatment [Internet]. Chemical Biology & Drug Design. 2020 ; 96 948–960.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1111/cbdd.13663
Artigo de periodico
Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole (2020)
Silva, Maria Cláudia da; Carbone, Débora Cardozo Bonfim; Diniz, Pedro Fernando; Fernandes, Fabrício Freitas; Fuzo, Carlos Alessandro; Trindade, Carolina Santos Pereira Cardoso; Chang, Marilene Rodrigues; Silva, João Santana da
Source: Mycopathologia. Unidades: FMRP, FCFRP
Subjects: CANDIDA, EXPRESSÃO GÊNICA, RESISTÊNCIA MICROBIANA ÀS DROGAS, CANDIDOSE, ANTIFÚNGICOS
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SILVA, Maria Cláudia da et al. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole. Mycopathologia, v. 185, n. 4, p. 675-684, 2020Tradução . . Disponível em: https://doi.org/10.1007/s11046-020-00465-6. Acesso em: 13 jun. 2024.
APA
Silva, M. C. da, Carbone, D. C. B., Diniz, P. F., Fernandes, F. F., Fuzo, C. A., Trindade, C. S. P. C., et al. (2020). Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole. Mycopathologia, 185( 4), 675-684. doi:10.1007/s11046-020-00465-6
NLM
Silva MC da, Carbone DCB, Diniz PF, Fernandes FF, Fuzo CA, Trindade CSPC, Chang MR, Silva JS da. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole [Internet]. Mycopathologia. 2020 ; 185( 4): 675-684.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1007/s11046-020-00465-6
Vancouver
Silva MC da, Carbone DCB, Diniz PF, Fernandes FF, Fuzo CA, Trindade CSPC, Chang MR, Silva JS da. Modulation of ERG genes expression in clinical isolates of Candida tropicalis susceptible and resistant to fluconazole and itraconazole [Internet]. Mycopathologia. 2020 ; 185( 4): 675-684.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1007/s11046-020-00465-6
Artigo de periodico
NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model (2020)
Sartori, Daniela Carlos ; Pérez, Malena Martínez ; Leite, Jefferson Antonio; Rocha, Fernanda Agostini; Martins, Larissa Marfori Sampaio; Pereira, Camila André; Silva, Thais Fernanda de Campos Fraga da ; Pucci, Taís Andrade; Ramos, Simone Gusmão ; Câmara, Niels Olsen Saraiva ; Bonato, Vânia Luiza Deperon ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB, FCFRP
Subjects: IMUNOLOGIA, LINFÓCITOS T, DIABETES MELLITUS, INFLAMAÇÃO, OBESIDADE, ADIPOSIDADE, CAMUNDONGOS, RESISTÊNCIA À INSULINA, HIPERGLICEMIA, LINFONODOS DE ANIMAL, EXPRESSÃO GÊNICA, BACTÉRIAS GRAM-NEGATIVAS, BACTEROIDES
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SARTORI, Daniela Carlos et al. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, v. 11, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01265. Acesso em: 13 jun. 2024.
APA
Sartori, D. C., Pérez, M. M., Leite, J. A., Rocha, F. A., Martins, L. M. S., Pereira, C. A., et al. (2020). NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, 11, 16 . doi:10.3389/fimmu.2020.01265
NLM
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 jun. 13 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Vancouver
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 jun. 13 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Artigo de periodico
Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease (2019)
Lopes, Carla Duque ; Possato, Bruna ; Gaspari, Ana Paula Segantin ; Oliveira, Ronaldo Junio ; Abram, Ulrich ; Almeida, José P. A; Rocho, Fernanda dos Reis ; Leitão, Andrei ; Montanari, Carlos Alberto ; Maia, Pedro Ivo da Silva ; Silva, João Santana da ; Albuquerque, Sérgio de ; Carneiro, Zumira Aparecida
Source: ACS Infectious Diseases. Unidades: IQSC, FMRP, FCFRP
Subjects: BIOQUÍMICA, DOENÇA DE CHAGAS
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ABNT
LOPES, Carla Duque et al. Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease. ACS Infectious Diseases, v. 5, n. 10, p. 1698-1707, 2019Tradução . . Disponível em: https://doi.org/10.1021/acsinfecdis.8b00284. Acesso em: 13 jun. 2024.
APA
Lopes, C. D., Possato, B., Gaspari, A. P. S., Oliveira, R. J., Abram, U., Almeida, J. P. A., et al. (2019). Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease. ACS Infectious Diseases, 5( 10), 1698-1707. doi:10.1021/acsinfecdis.8b00284
NLM
Lopes CD, Possato B, Gaspari APS, Oliveira RJ, Abram U, Almeida JPA, Rocho F dos R, Leitão A, Montanari CA, Maia PI da S, Silva JS da, Albuquerque S de, Carneiro ZA. Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease [Internet]. ACS Infectious Diseases. 2019 ; 5( 10): 1698-1707.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1021/acsinfecdis.8b00284
Vancouver
Lopes CD, Possato B, Gaspari APS, Oliveira RJ, Abram U, Almeida JPA, Rocho F dos R, Leitão A, Montanari CA, Maia PI da S, Silva JS da, Albuquerque S de, Carneiro ZA. Organometallic Gold(III) complex [Au(Hdamp)(L14)]+ (L1 = SNSDonating Thiosemicarbazone) as a candidate to new formulations against Chagas disease [Internet]. ACS Infectious Diseases. 2019 ; 5( 10): 1698-1707.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1021/acsinfecdis.8b00284
Artigo de periodico
Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis (2019)
Gupta, Gaurav; Santana, Alynne Karen Mendonça de; Gomes, Ciro Martins ; Turatti, Aline; Milanezi, Cristiane Maria; Bueno Filho, Roberto ; Fuzo, Carlos Alessandro ; Almeida, Roque Pacheco de ; Carregaro, Vanessa ; Roselino, Ana Maria Ferreira ; Silva, João Santana da
Source: Cellular Immunology. Unidades: FMRP, FCFRP
Subjects: LEISHMANIOSE CUTÂNEA, INFLAMAÇÃO
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GUPTA, Gaurav et al. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis. Cellular Immunology, v. 341, p. [8] , 2019Tradução . . Disponível em: https://doi.org/10.1016/j.cellimm.2019.04.008. Acesso em: 13 jun. 2024.
APA
Gupta, G., Santana, A. K. M. de, Gomes, C. M., Turatti, A., Milanezi, C. M., Bueno Filho, R., et al. (2019). Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis. Cellular Immunology, 341, [8] . doi:10.1016/j.cellimm.2019.04.008
NLM
Gupta G, Santana AKM de, Gomes CM, Turatti A, Milanezi CM, Bueno Filho R, Fuzo CA, Almeida RP de, Carregaro V, Roselino AMF, Silva JS da. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis [Internet]. Cellular Immunology. 2019 ; 341 [8] .[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.cellimm.2019.04.008
Vancouver
Gupta G, Santana AKM de, Gomes CM, Turatti A, Milanezi CM, Bueno Filho R, Fuzo CA, Almeida RP de, Carregaro V, Roselino AMF, Silva JS da. Inflammasome gene expression is associated with immunopathology in human localized cutaneous leishmaniasis [Internet]. Cellular Immunology. 2019 ; 341 [8] .[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.cellimm.2019.04.008
Artigo de periodico
The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy (2019)
Cardoso, Cristina Ribeiro de Barros ; Provinciatto, Pauline Rossetti; Godoi, Dannielle Fernandes; Fonseca, Monique Thaís Costa ; Ferreira, Beatriz Rossetti ; Teixeira, Gerlinde Agate Platais Brasil ; Cunha, Fernando de Queiroz ; Pinzan, Camila Figueiredo; Silva, João Santana da
Source: Allergologia et Immunopathologia. Unidades: FCFRP, EERP, FMRP
Subjects: HIPERSENSIBILIDADE ALIMENTAR, INFLAMAÇÃO, INTESTINOS, GASTROENTEROPATIAS
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CARDOSO, Cristina Ribeiro de Barros et al. The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy. Allergologia et Immunopathologia, v. 47, n. 6, p. 535-543, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.aller.2019.02.006. Acesso em: 13 jun. 2024.
APA
Cardoso, C. R. de B., Provinciatto, P. R., Godoi, D. F., Fonseca, M. T. C., Ferreira, B. R., Teixeira, G. A. P. B., et al. (2019). The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy. Allergologia et Immunopathologia, 47( 6), 535-543. doi:10.1016/j.aller.2019.02.006
NLM
Cardoso CR de B, Provinciatto PR, Godoi DF, Fonseca MTC, Ferreira BR, Teixeira GAPB, Cunha F de Q, Pinzan CF, Silva JS da. The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy [Internet]. Allergologia et Immunopathologia. 2019 ; 47( 6): 535-543.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.aller.2019.02.006
Vancouver
Cardoso CR de B, Provinciatto PR, Godoi DF, Fonseca MTC, Ferreira BR, Teixeira GAPB, Cunha F de Q, Pinzan CF, Silva JS da. The signal transducer and activator of transcription 6 (STAT-6) mediates Th2 inflammation and tissue damage in a murine model of peanut-induced food allergy [Internet]. Allergologia et Immunopathologia. 2019 ; 47( 6): 535-543.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.aller.2019.02.006
Artigo de periodico
Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis (2019)
Ketelut-Carneiro, Natália ; Souza, Camila de Oliveira Silva e ; Benevides, Luciana ; Gardinassi, Luiz Gustavo Araujo ; Silva, Maria Claúdia; Tavares, Lucas Alves ; Zamboni, Dario Simões ; Silva, João Santana da
Source: PLoS Pathogens. Unidades: FMRP, FCFRP
Subjects: IMUNIDADE, INTERLEUCINA 1, MACRÓFAGOS, PARACOCCIDIOIDOMICOSE
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ABNT
KETELUT-CARNEIRO, Natália et al. Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis. PLoS Pathogens, v. 15, n. 8, p. e1007990, 2019Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1007990. Acesso em: 13 jun. 2024.
APA
Ketelut-Carneiro, N., Souza, C. de O. S. e, Benevides, L., Gardinassi, L. G. A., Silva, M. C., Tavares, L. A., et al. (2019). Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis. PLoS Pathogens, 15( 8), e1007990. doi:10.1371/journal.ppat.1007990
NLM
Ketelut-Carneiro N, Souza C de OS e, Benevides L, Gardinassi LGA, Silva MC, Tavares LA, Zamboni DS, Silva JS da. Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis [Internet]. PLoS Pathogens. 2019 ; 15( 8): e1007990.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1371/journal.ppat.1007990
Vancouver
Ketelut-Carneiro N, Souza C de OS e, Benevides L, Gardinassi LGA, Silva MC, Tavares LA, Zamboni DS, Silva JS da. Caspase-11-dependent IL-1α release boosts Th17 immunity against Paracoccidioides brasiliensis [Internet]. PLoS Pathogens. 2019 ; 15( 8): e1007990.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1371/journal.ppat.1007990
Artigo de periodico
A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis (2019)
Freitas Alves, V. B.; Sousa, B. Coutinho de; Fonseca, M. Thaís Costa; Ogata, H.; Caliari-Oliveira, Carolina; Yaochite, Juliana Navarro Ueda; Rodrigues-Junior, Valnês S. ; Chica, Javier Emilio Lazo; Silva, João Santana da ; Farias, Kelen Cristina Ribeiro Malmegrim de ; Pernomian, L.; Cardoso, C. Ribeiro
Source: Clinical and Experimental Immunology : The Journal of Translational Immunology. Unidades: FMRP, FCFRP
Subjects: ENTEROPATIAS INFLAMATÓRIAS, CÉLULAS ESTROMAIS, COLITE
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FREITAS ALVES, V. B. et al. A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis. Clinical and Experimental Immunology : The Journal of Translational Immunology, v. 196, n. 2, p. 139-154, 2019Tradução . . Disponível em: https://doi.org/10.1111/cei.13262. Acesso em: 13 jun. 2024.
APA
Freitas Alves, V. B., Sousa, B. C. de, Fonseca, M. T. C., Ogata, H., Caliari-Oliveira, C., Yaochite, J. N. U., et al. (2019). A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis. Clinical and Experimental Immunology : The Journal of Translational Immunology, 196( 2), 139-154. doi:10.1111/cei.13262
NLM
Freitas Alves VB, Sousa BC de, Fonseca MTC, Ogata H, Caliari-Oliveira C, Yaochite JNU, Rodrigues-Junior VS, Chica JEL, Silva JS da, Farias KCRM de, Pernomian L, Cardoso CR. A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis [Internet]. Clinical and Experimental Immunology : The Journal of Translational Immunology. 2019 ; 196( 2): 139-154.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1111/cei.13262
Vancouver
Freitas Alves VB, Sousa BC de, Fonseca MTC, Ogata H, Caliari-Oliveira C, Yaochite JNU, Rodrigues-Junior VS, Chica JEL, Silva JS da, Farias KCRM de, Pernomian L, Cardoso CR. A single administration of human adipose tissue-derived mesenchymal stromal cells (MSC) induces durable and sustained long-term regulation of inflammatory response in experimental colitis [Internet]. Clinical and Experimental Immunology : The Journal of Translational Immunology. 2019 ; 196( 2): 139-154.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1111/cei.13262
Artigo de periodico
Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis (2018)
Souza, Patrícia Reis de; Guimarães, Francielle Rodrigues; Sales-Campos, Helioswilton; Bonfá, Giuliano; Nardini, Viviani; Chica, Javier Emilio Lazo; Turato, Walter Miguel; Silva, João Santana da; Zamboni, Dario Simões; Cardoso, Cristina Ribeiro de Barros
Source: Immunobiology. Unidades: FCF, FMRP, FCFRP
Subjects: IMUNIDADE, ENTEROPATIAS INFLAMATÓRIAS, LINFÓCITOS T, CITOCININAS
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ABNT
SOUZA, Patrícia Reis de et al. Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis. Immunobiology, v. 223, n. 10, p. 577-585, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2018.07.003. Acesso em: 13 jun. 2024.
APA
Souza, P. R. de, Guimarães, F. R., Sales-Campos, H., Bonfá, G., Nardini, V., Chica, J. E. L., et al. (2018). Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis. Immunobiology, 223( 10), 577-585. doi:10.1016/j.imbio.2018.07.003
NLM
Souza PR de, Guimarães FR, Sales-Campos H, Bonfá G, Nardini V, Chica JEL, Turato WM, Silva JS da, Zamboni DS, Cardoso CR de B. Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis [Internet]. Immunobiology. 2018 ; 223( 10): 577-585.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.imbio.2018.07.003
Vancouver
Souza PR de, Guimarães FR, Sales-Campos H, Bonfá G, Nardini V, Chica JEL, Turato WM, Silva JS da, Zamboni DS, Cardoso CR de B. Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis [Internet]. Immunobiology. 2018 ; 223( 10): 577-585.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.imbio.2018.07.003
Artigo de periodico
Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence (2018)
Kannen, Vinicius; Sakita, Juliana Y.; Carneiro, Zumira A.; Bader, Michael; Alenina, Natalia; Teixeira, Regina R.; Oliveira, Enio C. de; Brunaldi, Mariângela O.; Gasparotto, Bianca; Sartori, Daniela Carlos; Fernandes, Cleverson R.; Silva, João Santana da; Andrade, Marcus V.; Silva Júnior, Wilson Araújo da; Uyemura, Sérgio Akira; Garcia, Sérgio Britto
Source: Digestive Diseases and Sciences. Unidades: FMRP, FCFRP
Subjects: DOENÇA DE CHAGAS, NEURÔNIOS, DOENÇAS PARASITÁRIAS
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ABNT
KANNEN, Vinicius et al. Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence. Digestive Diseases and Sciences, v. 63, n. 6, p. 1473-1484, 2018Tradução . . Disponível em: https://doi.org/10.1007/s10620-018-5015-6. Acesso em: 13 jun. 2024.
APA
Kannen, V., Sakita, J. Y., Carneiro, Z. A., Bader, M., Alenina, N., Teixeira, R. R., et al. (2018). Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence. Digestive Diseases and Sciences, 63( 6), 1473-1484. doi:10.1007/s10620-018-5015-6
NLM
Kannen V, Sakita JY, Carneiro ZA, Bader M, Alenina N, Teixeira RR, Oliveira EC de, Brunaldi MO, Gasparotto B, Sartori DC, Fernandes CR, Silva JS da, Andrade MV, Silva Júnior WA da, Uyemura SA, Garcia SB. Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence [Internet]. Digestive Diseases and Sciences. 2018 ; 63( 6): 1473-1484.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1007/s10620-018-5015-6
Vancouver
Kannen V, Sakita JY, Carneiro ZA, Bader M, Alenina N, Teixeira RR, Oliveira EC de, Brunaldi MO, Gasparotto B, Sartori DC, Fernandes CR, Silva JS da, Andrade MV, Silva Júnior WA da, Uyemura SA, Garcia SB. Mast cells and serotonin synthesis modulate chagas disease in the colon: clinical and experimental evidence [Internet]. Digestive Diseases and Sciences. 2018 ; 63( 6): 1473-1484.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1007/s10620-018-5015-6
Artigo de periodico
The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing (2018)
Guimarães, Francielle Rodrigues; Sales-Campos, Helioswilton; Nardini, Viviani; Costa, Thiago Alvares da; Fonseca, Monique Thaís Costa; Rodrigues Júnior, Virmondes; Sorgi, Carlos Arterio; Silva, João Santana da; Chica, Javier Emílio Lazo; Faccioli, Lucia Helena; Cardoso, Cristina Ribeiro de Barros
Source: Clinical Immunology. Unidades: FCFRP, FMRP
Subjects: LEUCOTRIENOS, INFLAMAÇÃO, PELE, BIOMARCADORES
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ABNT
GUIMARÃES, Francielle Rodrigues et al. The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing. Clinical Immunology, v. 190, p. 74-83, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.clim.2017.08.022. Acesso em: 13 jun. 2024.
APA
Guimarães, F. R., Sales-Campos, H., Nardini, V., Costa, T. A. da, Fonseca, M. T. C., Rodrigues Júnior, V., et al. (2018). The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing. Clinical Immunology, 190, 74-83. doi:10.1016/j.clim.2017.08.022
NLM
Guimarães FR, Sales-Campos H, Nardini V, Costa TA da, Fonseca MTC, Rodrigues Júnior V, Sorgi CA, Silva JS da, Chica JEL, Faccioli LH, Cardoso CR de B. The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing [Internet]. Clinical Immunology. 2018 ; 190 74-83.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.clim.2017.08.022
Vancouver
Guimarães FR, Sales-Campos H, Nardini V, Costa TA da, Fonseca MTC, Rodrigues Júnior V, Sorgi CA, Silva JS da, Chica JEL, Faccioli LH, Cardoso CR de B. The inhibition of 5-Lipoxygenase (5-LO) products leukotriene B4 (LTB 4 ) and cysteinyl leukotrienes (cysLTs) modulates the inflammatory response and improves cutaneous wound healing [Internet]. Clinical Immunology. 2018 ; 190 74-83.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.clim.2017.08.022
Artigo de periodico
Organometallic gold(III) complexes with hybrid SNS-donating thiosemicarbazone ligands: cytotoxicity and anti-Trypanosoma cruzi activity (2017)
Maia, P. I. S; Carneiro, Z. A; Lopes, C D; Oliveira, C. G; Silva, João Santana da; Albuquerque, Sérgio de; Hagenbach, A; Gust, R; Deflon, Victor Marcelo ; Abram, U
Source: Dalton Transactions. Unidades: IQSC, FCFRP, FMRP
Assunto: TOXICOLOGIA
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ABNT
MAIA, P. I. S et al. Organometallic gold(III) complexes with hybrid SNS-donating thiosemicarbazone ligands: cytotoxicity and anti-Trypanosoma cruzi activity. Dalton Transactions, v. 46, p. 2559-2571, 2017Tradução . . Disponível em: https://doi.org/10.1039/C6DT04307K. Acesso em: 13 jun. 2024.
APA
Maia, P. I. S., Carneiro, Z. A., Lopes, C. D., Oliveira, C. G., Silva, J. S. da, Albuquerque, S. de, et al. (2017). Organometallic gold(III) complexes with hybrid SNS-donating thiosemicarbazone ligands: cytotoxicity and anti-Trypanosoma cruzi activity. Dalton Transactions, 46, 2559-2571. doi:10.1039/C6DT04307K
NLM
Maia PIS, Carneiro ZA, Lopes CD, Oliveira CG, Silva JS da, Albuquerque S de, Hagenbach A, Gust R, Deflon VM, Abram U. Organometallic gold(III) complexes with hybrid SNS-donating thiosemicarbazone ligands: cytotoxicity and anti-Trypanosoma cruzi activity [Internet]. Dalton Transactions. 2017 ; 46 2559-2571.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1039/C6DT04307K
Vancouver
Maia PIS, Carneiro ZA, Lopes CD, Oliveira CG, Silva JS da, Albuquerque S de, Hagenbach A, Gust R, Deflon VM, Abram U. Organometallic gold(III) complexes with hybrid SNS-donating thiosemicarbazone ligands: cytotoxicity and anti-Trypanosoma cruzi activity [Internet]. Dalton Transactions. 2017 ; 46 2559-2571.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1039/C6DT04307K
Artigo de periodico
Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity (2017)
Hartmann, Ana Paula; Carvalho, Marcelo Rodrigues de; Bernardes, Lilian Sibelle Campos; Moraes, Milena Hoehr de; Melo, Eduardo Borges de; Lopes, Carla Duque; Steindel, Mario; Silva, João Santana da; Carvalho, Ivone
Source: European Journal of Medicinal Chemistry. Unidades: FCFRP, FMRP
Subjects: DOENÇA DE CHAGAS, TRYPANOSOMA CRUZI, INIBIDORES DE ENZIMAS
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ABNT
HARTMANN, Ana Paula et al. Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity. European Journal of Medicinal Chemistry, v. 140, p. 187-199, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejmech.2017.08.064. Acesso em: 13 jun. 2024.
APA
Hartmann, A. P., Carvalho, M. R. de, Bernardes, L. S. C., Moraes, M. H. de, Melo, E. B. de, Lopes, C. D., et al. (2017). Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity. European Journal of Medicinal Chemistry, 140, 187-199. doi:10.1016/j.ejmech.2017.08.064
NLM
Hartmann AP, Carvalho MR de, Bernardes LSC, Moraes MH de, Melo EB de, Lopes CD, Steindel M, Silva JS da, Carvalho I. Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity [Internet]. European Journal of Medicinal Chemistry. 2017 ; 140 187-199.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.ejmech.2017.08.064
Vancouver
Hartmann AP, Carvalho MR de, Bernardes LSC, Moraes MH de, Melo EB de, Lopes CD, Steindel M, Silva JS da, Carvalho I. Synthesis and 2D-QSAR studies of neolignan-based diaryl-tetrahydrofuran and -furan analogues with remarkable activity against Trypanosoma cruzi and assessment of the trypanothione reductase activity [Internet]. European Journal of Medicinal Chemistry. 2017 ; 140 187-199.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.ejmech.2017.08.064
Artigo de periodico
The impact of intestinal resection on the immune function of short bowel syndrome patients (2016)
Turato, Walter Miguel; Sales-Campos, Helioswilton; Braga, Camila Bitu Moreno; Cunha, Selma Freire de Carvalho da; Silvah, José Henrique; Silva, João Santana da; Marchini, Julio Sergio ; Cardoso, Cristina Ribeiro de Barros
Source: Human Immunology. Unidades: FCF, FMRP, FCFRP
Subjects: ENTEROPATIAS, LINFÓCITOS T, INTERLEUCINAS, SISTEMA IMUNE
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ABNT
TURATO, Walter Miguel et al. The impact of intestinal resection on the immune function of short bowel syndrome patients. Human Immunology, v. 77, p. 1202-1208, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.humimm.2016.07.237. Acesso em: 13 jun. 2024.
APA
Turato, W. M., Sales-Campos, H., Braga, C. B. M., Cunha, S. F. de C. da, Silvah, J. H., Silva, J. S. da, et al. (2016). The impact of intestinal resection on the immune function of short bowel syndrome patients. Human Immunology, 77, 1202-1208. doi:10.1016/j.humimm.2016.07.237
NLM
Turato WM, Sales-Campos H, Braga CBM, Cunha SF de C da, Silvah JH, Silva JS da, Marchini JS, Cardoso CR de B. The impact of intestinal resection on the immune function of short bowel syndrome patients [Internet]. Human Immunology. 2016 ; 77 1202-1208.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.humimm.2016.07.237
Vancouver
Turato WM, Sales-Campos H, Braga CBM, Cunha SF de C da, Silvah JH, Silva JS da, Marchini JS, Cardoso CR de B. The impact of intestinal resection on the immune function of short bowel syndrome patients [Internet]. Human Immunology. 2016 ; 77 1202-1208.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1016/j.humimm.2016.07.237
Artigo de periodico
Inflammation biomarkers of advanced disease in nongingival tissues of chronic periodontitis patients (2015)
Costa, Thiago Alvares da; Silva, Marcelo José Barbosa; Alves, Polyanna Miranda; Chica, Javier Emílio Lazo; Barcelos, Emilio Zorzo; Giani, Max Antonio Alves; Garlet, Gustavo Pompermaier; Silva, João Santana da; Rodrigues Júnior, Virmondes; Rodrigues, Denise Bertulucci Rocha; Cardoso, Cristina Ribeiro de Barros
Source: Mediators of Inflammation. Unidades: FOB, FMRP, FCFRP
Subjects: INFLAMAÇÃO, BIOMARCADORES, PERIODONTITE CRÔNICA
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ABNT
COSTA, Thiago Alvares da et al. Inflammation biomarkers of advanced disease in nongingival tissues of chronic periodontitis patients. Mediators of Inflammation, p. 1-10, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/983782. Acesso em: 13 jun. 2024.
APA
Costa, T. A. da, Silva, M. J. B., Alves, P. M., Chica, J. E. L., Barcelos, E. Z., Giani, M. A. A., et al. (2015). Inflammation biomarkers of advanced disease in nongingival tissues of chronic periodontitis patients. Mediators of Inflammation, 1-10. doi:10.1155/2015/983782
NLM
Costa TA da, Silva MJB, Alves PM, Chica JEL, Barcelos EZ, Giani MAA, Garlet GP, Silva JS da, Rodrigues Júnior V, Rodrigues DBR, Cardoso CR de B. Inflammation biomarkers of advanced disease in nongingival tissues of chronic periodontitis patients [Internet]. Mediators of Inflammation. 2015 ; 1-10.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1155/2015/983782
Vancouver
Costa TA da, Silva MJB, Alves PM, Chica JEL, Barcelos EZ, Giani MAA, Garlet GP, Silva JS da, Rodrigues Júnior V, Rodrigues DBR, Cardoso CR de B. Inflammation biomarkers of advanced disease in nongingival tissues of chronic periodontitis patients [Internet]. Mediators of Inflammation. 2015 ; 1-10.[citado 2024 jun. 13 ] Available from: https://doi.org/10.1155/2015/983782

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