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Artigo de periodico
The severity of neuropsychiatric symptoms is higher in early-onset than late-onset Alzheimer's disease (2022)
Falgas, Neus; Allen, Isabel E.; Spina, Salvatore; Grant, Harli; Escudero, Stefanie D. Pina; Merrilees, Jennifer; Gearhart, Rosalie; Rosen, Howard J.; Kramer, Joel H.; Seeley, William W.; Grinberg, Lea Tenenholz
Fonte: European journal of neurology. Unidade: FM
Assuntos: DOENÇA DE ALZHEIMER, DEMÊNCIA, SINAIS E SINTOMAS
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ABNT
FALGAS, Neus et al. The severity of neuropsychiatric symptoms is higher in early-onset than late-onset Alzheimer's disease. European journal of neurology, v. 29, n. 4, p. 957-967, 2022Tradução . . Disponível em: https://doi.org/10.1111/ene.15203. Acesso em: 17 ago. 2024.
APA
Falgas, N., Allen, I. E., Spina, S., Grant, H., Escudero, S. D. P., Merrilees, J., et al. (2022). The severity of neuropsychiatric symptoms is higher in early-onset than late-onset Alzheimer's disease. European journal of neurology, 29( 4), 957-967. doi:10.1111/ene.15203
NLM
Falgas N, Allen IE, Spina S, Grant H, Escudero SDP, Merrilees J, Gearhart R, Rosen HJ, Kramer JH, Seeley WW, Grinberg LT. The severity of neuropsychiatric symptoms is higher in early-onset than late-onset Alzheimer's disease [Internet]. European journal of neurology. 2022 ; 29( 4): 957-967.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1111/ene.15203
Vancouver
Falgas N, Allen IE, Spina S, Grant H, Escudero SDP, Merrilees J, Gearhart R, Rosen HJ, Kramer JH, Seeley WW, Grinberg LT. The severity of neuropsychiatric symptoms is higher in early-onset than late-onset Alzheimer's disease [Internet]. European journal of neurology. 2022 ; 29( 4): 957-967.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1111/ene.15203
Trabalho de evento-resumo periodico
Neurobiological basis of sleep disturbances in tauopathies: human wake-promoting neurons degenerate more in Alzheimer’s disease (2019)
Oh, Jun; Eser, Rana A.; Ehrenberg, Alexander J.; Morales, Dulce; Petersen, Cathrine; Theofilas, Panos; Resende, Elisa; Cosme, Celica; Seeley, William W.; Spina, Salvatore; Walsh, Christine M.; Neylan, Thomas C.; Miller, Bruce L.; Bittencourt, Jackson Cioni ; Grinberg, Lea Tenenholz
Fonte: Sleep. Nome do evento: Annual Meeting of the Associated Professional Sleep Societies. XIV Sleep and Neurodegeration. Unidades: ICB, FM
Assuntos: ANATOMIA, TRANSTORNOS DO SONO, DOENÇA DE ALZHEIMER, DOENÇAS NEURODEGENERATIVAS, IMUNOHISTOQUÍMICA, NEURÔNIOS, SONO
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OH, Jun et al. Neurobiological basis of sleep disturbances in tauopathies: human wake-promoting neurons degenerate more in Alzheimer’s disease. Sleep. North Carolina: Oxford University Press. Disponível em: https://doi.org/10.1093/sleep/zsz067.302. Acesso em: 17 ago. 2024. , 2019
APA
Oh, J., Eser, R. A., Ehrenberg, A. J., Morales, D., Petersen, C., Theofilas, P., et al. (2019). Neurobiological basis of sleep disturbances in tauopathies: human wake-promoting neurons degenerate more in Alzheimer’s disease. Sleep. North Carolina: Oxford University Press. doi:10.1093/sleep/zsz067.302
NLM
Oh J, Eser RA, Ehrenberg AJ, Morales D, Petersen C, Theofilas P, Resende E, Cosme C, Seeley WW, Spina S, Walsh CM, Neylan TC, Miller BL, Bittencourt JC, Grinberg LT. Neurobiological basis of sleep disturbances in tauopathies: human wake-promoting neurons degenerate more in Alzheimer’s disease [Internet]. Sleep. 2019 ; 42 A-123-124.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1093/sleep/zsz067.302
Vancouver
Oh J, Eser RA, Ehrenberg AJ, Morales D, Petersen C, Theofilas P, Resende E, Cosme C, Seeley WW, Spina S, Walsh CM, Neylan TC, Miller BL, Bittencourt JC, Grinberg LT. Neurobiological basis of sleep disturbances in tauopathies: human wake-promoting neurons degenerate more in Alzheimer’s disease [Internet]. Sleep. 2019 ; 42 A-123-124.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1093/sleep/zsz067.302
Artigo de periodico
Profound degeneration of wake-promoting neurons in Alzheimer’s disease (2019)
Oh, Jun; Eser, Rana A.; Ehrenberg, Alexander J.; Morales, Dulce; Petersen, Cathrine; Kudlacek, Jessica; Dunlop, Sara R.; Theofilas, Panos; Resende, Elisa D. P. F.; Cosme, Celica; Alho, Eduardo J. L.; Spina, Salvatore; Walsh, Christine M.; Miller, Bruce L.; Seeley, William W.; Bittencourt, Jackson Cioni ; Neylan, Thomas C.; Heinsen, Helmut; Grinberg, Lea T.
Fonte: Alzheimer's & Dementia. Unidade: ICB
Assuntos: ANATOMIA, DOENÇA DE ALZHEIMER, NEURÔNIOS, HISTAMINA, AUTÓPSIA
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ABNT
OH, Jun et al. Profound degeneration of wake-promoting neurons in Alzheimer’s disease. Alzheimer's & Dementia, v. 15, p. 1253-1263, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.jalz.2019.06.3916. Acesso em: 17 ago. 2024.
APA
Oh, J., Eser, R. A., Ehrenberg, A. J., Morales, D., Petersen, C., Kudlacek, J., et al. (2019). Profound degeneration of wake-promoting neurons in Alzheimer’s disease. Alzheimer's & Dementia, 15, 1253-1263. doi:10.1016/j.jalz.2019.06.3916
NLM
Oh J, Eser RA, Ehrenberg AJ, Morales D, Petersen C, Kudlacek J, Dunlop SR, Theofilas P, Resende EDPF, Cosme C, Alho EJL, Spina S, Walsh CM, Miller BL, Seeley WW, Bittencourt JC, Neylan TC, Heinsen H, Grinberg LT. Profound degeneration of wake-promoting neurons in Alzheimer’s disease [Internet]. Alzheimer's & Dementia. 2019 ; 15 1253-1263.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.jalz.2019.06.3916
Vancouver
Oh J, Eser RA, Ehrenberg AJ, Morales D, Petersen C, Kudlacek J, Dunlop SR, Theofilas P, Resende EDPF, Cosme C, Alho EJL, Spina S, Walsh CM, Miller BL, Seeley WW, Bittencourt JC, Neylan TC, Heinsen H, Grinberg LT. Profound degeneration of wake-promoting neurons in Alzheimer’s disease [Internet]. Alzheimer's & Dementia. 2019 ; 15 1253-1263.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.jalz.2019.06.3916
Artigo de periodico
Locus coeruleus volume and cell population changes during Alzheimer's disease progression: A stereological study in human postmortem brains with potential implication for early-stage biomarker discovery (2017)
Theofilas, Panos; Ehrenberg, Alexander J.; Dunlop, Sara; Alho, Ana T. Di Lorenzo; Nguy, Austin; Leite, Renata Elaine Paraizo; Rodriguez, Roberta Diehl; Mejia, Maria B.; Suemoto, Claudia K.; Ferretti-Rebustini, Renata Eloah de Lucena ; Polichiso, Livia; Nascimento, Camila F.; Seeley, William W.; Nitrini, Ricardo; Pasqualucci, Carlos Augusto; Jacob Filho, Wilson; Rueb, Udo; Neuhaus, John; Heinsen, Helmut; Grinberg, Lea T.
Fonte: Alzheimer's & Dementia. Unidade: FM
Assuntos: BIOMARCADORES, NEURÔNIOS, DOENÇA DE ALZHEIMER, CÉLULAS SANGUÍNEAS
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THEOFILAS, Panos et al. Locus coeruleus volume and cell population changes during Alzheimer's disease progression: A stereological study in human postmortem brains with potential implication for early-stage biomarker discovery. Alzheimer's & Dementia, v. 13, n. 3 , p. 236-246, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jalz.2016.06.2362. Acesso em: 17 ago. 2024.
APA
Theofilas, P., Ehrenberg, A. J., Dunlop, S., Alho, A. T. D. L., Nguy, A., Leite, R. E. P., et al. (2017). Locus coeruleus volume and cell population changes during Alzheimer's disease progression: A stereological study in human postmortem brains with potential implication for early-stage biomarker discovery. Alzheimer's & Dementia, 13( 3 ), 236-246. doi:10.1016/j.jalz.2016.06.2362
NLM
Theofilas P, Ehrenberg AJ, Dunlop S, Alho ATDL, Nguy A, Leite REP, Rodriguez RD, Mejia MB, Suemoto CK, Ferretti-Rebustini RE de L, Polichiso L, Nascimento CF, Seeley WW, Nitrini R, Pasqualucci CA, Jacob Filho W, Rueb U, Neuhaus J, Heinsen H, Grinberg LT. Locus coeruleus volume and cell population changes during Alzheimer's disease progression: A stereological study in human postmortem brains with potential implication for early-stage biomarker discovery [Internet]. Alzheimer's & Dementia. 2017 ; 13( 3 ): 236-246.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.jalz.2016.06.2362
Vancouver
Theofilas P, Ehrenberg AJ, Dunlop S, Alho ATDL, Nguy A, Leite REP, Rodriguez RD, Mejia MB, Suemoto CK, Ferretti-Rebustini RE de L, Polichiso L, Nascimento CF, Seeley WW, Nitrini R, Pasqualucci CA, Jacob Filho W, Rueb U, Neuhaus J, Heinsen H, Grinberg LT. Locus coeruleus volume and cell population changes during Alzheimer's disease progression: A stereological study in human postmortem brains with potential implication for early-stage biomarker discovery [Internet]. Alzheimer's & Dementia. 2017 ; 13( 3 ): 236-246.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1016/j.jalz.2016.06.2362
Artigo de periodico
Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography (2015)
Sha, Sharon J.; Ghosh, Pia M.; Lee, Suzee E.; Rastelli, Chiara Corbetta; Jagust, William; Kormak, John; Rankin, Katherine P.; Grinberg, Lea T.; Vinters, Harry V.; Mendez, Mario F.; Dickson, Dennis W.; Seeley, William W.; Tempini, Marilu Gorno; Kramer, Joel; Boxer, Adam L.; Rabinovici, Gil D.
Fonte: Alzheimer's research & therapy. Unidade: FM
Assuntos: DOENÇA DE ALZHEIMER, IMAGEM POR RESSONÂNCIA MAGNÉTICA, TOMOGRAFIA COMPUTADORIZADA POR EMISSÃO DE PÓSITRON (TENDÊNCIAS), DOENÇAS NEURODEGENERATIVAS
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SHA, Sharon J. et al. Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography. Alzheimer's research & therapy, v. 7, p. 8, 2015Tradução . . Disponível em: https://doi.org/10.1186/s13195-014-0093-y. Acesso em: 17 ago. 2024.
APA
Sha, S. J., Ghosh, P. M., Lee, S. E., Rastelli, C. C., Jagust, W., Kormak, J., et al. (2015). Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography. Alzheimer's research & therapy, 7, 8. doi:10.1186/s13195-014-0093-y
NLM
Sha SJ, Ghosh PM, Lee SE, Rastelli CC, Jagust W, Kormak J, Rankin KP, Grinberg LT, Vinters HV, Mendez MF, Dickson DW, Seeley WW, Tempini MG, Kramer J, Boxer AL, Rabinovici GD. Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography [Internet]. Alzheimer's research & therapy. 2015 ; 7 8.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1186/s13195-014-0093-y
Vancouver
Sha SJ, Ghosh PM, Lee SE, Rastelli CC, Jagust W, Kormak J, Rankin KP, Grinberg LT, Vinters HV, Mendez MF, Dickson DW, Seeley WW, Tempini MG, Kramer J, Boxer AL, Rabinovici GD. Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography [Internet]. Alzheimer's research & therapy. 2015 ; 7 8.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1186/s13195-014-0093-y
Artigo de periodico
Criminal Behavior in Frontotemporal Dementia and Alzheimer Disease (2015)
Liljegren, Madeleine; Naasan, Georges; Temlett, Julia; Perry, David C.; Rankin, Katherine P.; Merrilees, Jennifer; Grinberg, Lea T.; Seeley, William W.; Englund, Elisabet; Miller,Bruce L.
Fonte: JAMA Neurology. Unidade: FM
Assuntos: DEMÊNCIA, COMPORTAMENTO CRIMINAL, DOENÇA DE ALZHEIMER
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LILJEGREN, Madeleine et al. Criminal Behavior in Frontotemporal Dementia and Alzheimer Disease. JAMA Neurology, v. 72, n. 3, p. 295-300, 2015Tradução . . Disponível em: https://doi.org/10.1001/jamaneurol.2014.3781. Acesso em: 17 ago. 2024.
APA
Liljegren, M., Naasan, G., Temlett, J., Perry, D. C., Rankin, K. P., Merrilees, J., et al. (2015). Criminal Behavior in Frontotemporal Dementia and Alzheimer Disease. JAMA Neurology, 72( 3), 295-300. doi:10.1001/jamaneurol.2014.3781
NLM
Liljegren M, Naasan G, Temlett J, Perry DC, Rankin KP, Merrilees J, Grinberg LT, Seeley WW, Englund E, Miller BL. Criminal Behavior in Frontotemporal Dementia and Alzheimer Disease [Internet]. JAMA Neurology. 2015 ; 72( 3): 295-300.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1001/jamaneurol.2014.3781
Vancouver
Liljegren M, Naasan G, Temlett J, Perry DC, Rankin KP, Merrilees J, Grinberg LT, Seeley WW, Englund E, Miller BL. Criminal Behavior in Frontotemporal Dementia and Alzheimer Disease [Internet]. JAMA Neurology. 2015 ; 72( 3): 295-300.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1001/jamaneurol.2014.3781
Artigo de periodico
In vivo signatures of nonfluent/agrammatic primary progressive aphasia caused by FTLD pathology (2014)
Caso, Francesca; Mandelli, Maria Luisa; Henry, Maya; Gesierich, Benno; Bettcher, Brianne M.; Ogar, Jennifer; Filippi, Massimo; Comi, Giancarlo; Magnani, Giuseppe; Sidhu, Manu; Trojanowski, John Q.; Huang, Eric J.; Grinberg, Lea T.; Miller, Bruce L.; Dronkers, Nina; Seeley, William W.; Gorno-Tempini, Maria Luisa
Fonte: Neurology. Unidade: FM
Assuntos: COGNIÇÃO (ANORMALIDADES), AFASIA PROGRESSIVA, AGRAMATISMO
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CASO, Francesca et al. In vivo signatures of nonfluent/agrammatic primary progressive aphasia caused by FTLD pathology. Neurology, v. 82, n. 3, p. 239-247, 2014Tradução . . Disponível em: https://doi.org/10.1212/WNL.0000000000000031. Acesso em: 17 ago. 2024.
APA
Caso, F., Mandelli, M. L., Henry, M., Gesierich, B., Bettcher, B. M., Ogar, J., et al. (2014). In vivo signatures of nonfluent/agrammatic primary progressive aphasia caused by FTLD pathology. Neurology, 82( 3), 239-247. doi:10.1212/WNL.0000000000000031
NLM
Caso F, Mandelli ML, Henry M, Gesierich B, Bettcher BM, Ogar J, Filippi M, Comi G, Magnani G, Sidhu M, Trojanowski JQ, Huang EJ, Grinberg LT, Miller BL, Dronkers N, Seeley WW, Gorno-Tempini ML. In vivo signatures of nonfluent/agrammatic primary progressive aphasia caused by FTLD pathology [Internet]. Neurology. 2014 ; 82( 3): 239-247.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1212/WNL.0000000000000031
Vancouver
Caso F, Mandelli ML, Henry M, Gesierich B, Bettcher BM, Ogar J, Filippi M, Comi G, Magnani G, Sidhu M, Trojanowski JQ, Huang EJ, Grinberg LT, Miller BL, Dronkers N, Seeley WW, Gorno-Tempini ML. In vivo signatures of nonfluent/agrammatic primary progressive aphasia caused by FTLD pathology [Internet]. Neurology. 2014 ; 82( 3): 239-247.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1212/WNL.0000000000000031
Artigo de periodico
Distinct tau prion strains propagate in cells and mice and define different tauopathies (2014)
Sanders, David W.; Kaufman, Sarah K.; DeVos, Sarah L.; Sharma, Apurwa M.; Mirbaha, Hilda; Li, Aimin; Barker, Scarlett J.; Foley, Alex C.; Thorpe, Julian R.; Serpell, Louise C.; Miller, Timothy M.; Grinberg, Lea T.; Seeley, William W.; Diamond, Marc I.
Fonte: Neuron. Unidade: FM
Assuntos: DOENÇAS NEURODEGENERATIVAS, FENÓTIPOS, RATOS
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SANDERS, David W. et al. Distinct tau prion strains propagate in cells and mice and define different tauopathies. Neuron, v. 82, p. 1271-1288, 2014Tradução . . Disponível em: http://www.sciencedirect.com/science/article/pii/S0896627314003626. Acesso em: 17 ago. 2024.
APA
Sanders, D. W., Kaufman, S. K., DeVos, S. L., Sharma, A. M., Mirbaha, H., Li, A., et al. (2014). Distinct tau prion strains propagate in cells and mice and define different tauopathies. Neuron, 82, 1271-1288. Recuperado de http://www.sciencedirect.com/science/article/pii/S0896627314003626
NLM
Sanders DW, Kaufman SK, DeVos SL, Sharma AM, Mirbaha H, Li A, Barker SJ, Foley AC, Thorpe JR, Serpell LC, Miller TM, Grinberg LT, Seeley WW, Diamond MI. Distinct tau prion strains propagate in cells and mice and define different tauopathies [Internet]. Neuron. 2014 ; 82 1271-1288.[citado 2024 ago. 17 ] Available from: http://www.sciencedirect.com/science/article/pii/S0896627314003626
Vancouver
Sanders DW, Kaufman SK, DeVos SL, Sharma AM, Mirbaha H, Li A, Barker SJ, Foley AC, Thorpe JR, Serpell LC, Miller TM, Grinberg LT, Seeley WW, Diamond MI. Distinct tau prion strains propagate in cells and mice and define different tauopathies [Internet]. Neuron. 2014 ; 82 1271-1288.[citado 2024 ago. 17 ] Available from: http://www.sciencedirect.com/science/article/pii/S0896627314003626
Artigo de periodico
Primary age-related tauopathy (PART): a common pathology associated with human aging (2014)
Crary, John F.; Trojanowski, John Q.; Schneider, Julie A.; Abisambra, Jose F.; Abner, Erin L.; Alafuzoff, Irina; Arnold, Steven E.; Attems, Johannes; Beach, Thomas G.; Bigio, Eileen H.; Cairns, Nigel J.; Dickson, Dennis W.; Gearing, Marla; Grinberg, Lea T.; Hof, Patrick R.; Hyman, Bradley T.; Jellinger, Kurt; Jicha, Gregory A.; Kovacs, Gabor G.; Knopman, David S.; Kofler, Julia; Kukull, Walter A.; Mackenzie, Ian R.; Masliah, Eliezer; McKee, Ann; Montine, Thomas J.; Murray, Melissa E.; Neltner, Janna H.; Santa-Maria, Ismael; Seeley, William W.; Serrano-Pozo, Alberto; Shelanski, Michael L.; Stein, Thor; Takao, Masaki; Thal, Dietmar R.; Toledo, Jonathan B.; Troncoso, Juan C.; Vonsattel, Jean Paul; White, Charles L.; Wisniewski, Thomas; Woltjer, Randall L.; Yamada, Masahito; Nelson, Peter T.
Fonte: Acta Neuropathologica. Unidade: FM
Assuntos: LOBO TEMPORAL, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, BIOMARCADORES, NEURÔNIOS (PATOLOGIA)
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CRARY, John F. et al. Primary age-related tauopathy (PART): a common pathology associated with human aging. Acta Neuropathologica, v. 128, n. 6, p. 755-766, 2014Tradução . . Disponível em: http://link.springer.com/article/10.1007/s00401-014-1349-0. Acesso em: 17 ago. 2024.
APA
Crary, J. F., Trojanowski, J. Q., Schneider, J. A., Abisambra, J. F., Abner, E. L., Alafuzoff, I., et al. (2014). Primary age-related tauopathy (PART): a common pathology associated with human aging. Acta Neuropathologica, 128( 6), 755-766. doi:10.1007/s00401-014-1349-0
NLM
Crary JF, Trojanowski JQ, Schneider JA, Abisambra JF, Abner EL, Alafuzoff I, Arnold SE, Attems J, Beach TG, Bigio EH, Cairns NJ, Dickson DW, Gearing M, Grinberg LT, Hof PR, Hyman BT, Jellinger K, Jicha GA, Kovacs GG, Knopman DS, Kofler J, Kukull WA, Mackenzie IR, Masliah E, McKee A, Montine TJ, Murray ME, Neltner JH, Santa-Maria I, Seeley WW, Serrano-Pozo A, Shelanski ML, Stein T, Takao M, Thal DR, Toledo JB, Troncoso JC, Vonsattel JP, White CL, Wisniewski T, Woltjer RL, Yamada M, Nelson PT. Primary age-related tauopathy (PART): a common pathology associated with human aging [Internet]. Acta Neuropathologica. 2014 ; 128( 6): 755-766.[citado 2024 ago. 17 ] Available from: http://link.springer.com/article/10.1007/s00401-014-1349-0
Vancouver
Crary JF, Trojanowski JQ, Schneider JA, Abisambra JF, Abner EL, Alafuzoff I, Arnold SE, Attems J, Beach TG, Bigio EH, Cairns NJ, Dickson DW, Gearing M, Grinberg LT, Hof PR, Hyman BT, Jellinger K, Jicha GA, Kovacs GG, Knopman DS, Kofler J, Kukull WA, Mackenzie IR, Masliah E, McKee A, Montine TJ, Murray ME, Neltner JH, Santa-Maria I, Seeley WW, Serrano-Pozo A, Shelanski ML, Stein T, Takao M, Thal DR, Toledo JB, Troncoso JC, Vonsattel JP, White CL, Wisniewski T, Woltjer RL, Yamada M, Nelson PT. Primary age-related tauopathy (PART): a common pathology associated with human aging [Internet]. Acta Neuropathologica. 2014 ; 128( 6): 755-766.[citado 2024 ago. 17 ] Available from: http://link.springer.com/article/10.1007/s00401-014-1349-0
Artigo de periodico
Practical utility of amyloid and FDG-PET in an academic dementia center (2014)
Sánchez-Juan, Pascual; Ghosh, Pia M.; Hagen, Jayne; Gesierich, Benno; Henry, Maya; Grinberg, Lea T.; O'Neil, James P.; Janabi, Mustafa; Huang, Eric J.; Trojanowski, John Q.; Vinters, Harry V.; Gorno-Tempini, Marilu; Seeley, William W.; Boxer, Adam L.; Rosen, Howard J.; Kramer, Joel H.; Miller, Bruce L.; Jagust, William J.; Rabinovici, Gil D.
Fonte: Neurology. Unidade: FM
Assuntos: DEMÊNCIA, DIAGNÓSTICO POR IMAGEM, DOENÇAS DEGENERATIVAS
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ABNT
SÁNCHEZ-JUAN, Pascual et al. Practical utility of amyloid and FDG-PET in an academic dementia center. Neurology, v. 82, n. 3, p. 230-238, 2014Tradução . . Disponível em: https://doi.org/10.1212/WNL.0000000000000032. Acesso em: 17 ago. 2024.
APA
Sánchez-Juan, P., Ghosh, P. M., Hagen, J., Gesierich, B., Henry, M., Grinberg, L. T., et al. (2014). Practical utility of amyloid and FDG-PET in an academic dementia center. Neurology, 82( 3), 230-238. doi:10.1212/WNL.0000000000000032
NLM
Sánchez-Juan P, Ghosh PM, Hagen J, Gesierich B, Henry M, Grinberg LT, O'Neil JP, Janabi M, Huang EJ, Trojanowski JQ, Vinters HV, Gorno-Tempini M, Seeley WW, Boxer AL, Rosen HJ, Kramer JH, Miller BL, Jagust WJ, Rabinovici GD. Practical utility of amyloid and FDG-PET in an academic dementia center [Internet]. Neurology. 2014 ; 82( 3): 230-238.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1212/WNL.0000000000000032
Vancouver
Sánchez-Juan P, Ghosh PM, Hagen J, Gesierich B, Henry M, Grinberg LT, O'Neil JP, Janabi M, Huang EJ, Trojanowski JQ, Vinters HV, Gorno-Tempini M, Seeley WW, Boxer AL, Rosen HJ, Kramer JH, Miller BL, Jagust WJ, Rabinovici GD. Practical utility of amyloid and FDG-PET in an academic dementia center [Internet]. Neurology. 2014 ; 82( 3): 230-238.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1212/WNL.0000000000000032
Artigo de periodico
Progranulin Mutations as Risk Factors for Alzheimer Disease (2013)
Perry, David C.; Lehmann, Manja; Yokoyama, Jennifer S.; Karydas, Anna; Lee, Jason JiYong; Coppola, Giovanni; Grinberg, Lea T.; Geschwind, Dan; Seeley, William W.; Miller, Bruce L.; Rosen, Howard; Rabinovici, Gil
Fonte: JAMA Neurology. Unidade: FM
Assuntos: FATORES DE RISCO, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE ALZHEIMER, MUTAÇÃO GENÉTICA
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ABNT
PERRY, David C. et al. Progranulin Mutations as Risk Factors for Alzheimer Disease. JAMA Neurology, v. 70, n. 6, p. 774-778, 2013Tradução . . Disponível em: https://doi.org/10.1001/2013.jamaneurol.393. Acesso em: 17 ago. 2024.
APA
Perry, D. C., Lehmann, M., Yokoyama, J. S., Karydas, A., Lee, J. J. Y., Coppola, G., et al. (2013). Progranulin Mutations as Risk Factors for Alzheimer Disease. JAMA Neurology, 70( 6), 774-778. doi:10.1001/2013.jamaneurol.393
NLM
Perry DC, Lehmann M, Yokoyama JS, Karydas A, Lee JJY, Coppola G, Grinberg LT, Geschwind D, Seeley WW, Miller BL, Rosen H, Rabinovici G. Progranulin Mutations as Risk Factors for Alzheimer Disease [Internet]. JAMA Neurology. 2013 ; 70( 6): 774-778.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1001/2013.jamaneurol.393
Vancouver
Perry DC, Lehmann M, Yokoyama JS, Karydas A, Lee JJY, Coppola G, Grinberg LT, Geschwind D, Seeley WW, Miller BL, Rosen H, Rabinovici G. Progranulin Mutations as Risk Factors for Alzheimer Disease [Internet]. JAMA Neurology. 2013 ; 70( 6): 774-778.[citado 2024 ago. 17 ] Available from: https://doi.org/10.1001/2013.jamaneurol.393
Artigo de periodico
Argyrophilic grain disease differs from other tauopathies by lacking tau acetylation (2013)
Grinberg, Lea Tenenholz; Wang, Xuehua; Wang, Chao; Sohn, Peter Dongmin; Theofilas, Panos; Sidhu, Manu; Arevalo, John Benjamin; Heinsen, Helmut; Huang, Eric J.; Rosen, Howard; Miller, Bruce L.; Gan, Li; Seeley, William W.
Fonte: Acta Neuropathologica. Unidade: FM
Assuntos: AUTÓPSIA, IMUNOHISTOQUÍMICA, ANTICORPOS MONOCLONAIS, SISTEMA NERVOSO CENTRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GRINBERG, Lea Tenenholz et al. Argyrophilic grain disease differs from other tauopathies by lacking tau acetylation. Acta Neuropathologica, v. 125, n. 4, p. 581-593, 2013Tradução . . Disponível em: http://link.springer.com/content/pdf/10.1007%2Fs00401-013-1080-2.pdf. Acesso em: 17 ago. 2024.
APA
Grinberg, L. T., Wang, X., Wang, C., Sohn, P. D., Theofilas, P., Sidhu, M., et al. (2013). Argyrophilic grain disease differs from other tauopathies by lacking tau acetylation. Acta Neuropathologica, 125( 4), 581-593. doi:10.1007/s00401-013-1080-2
NLM
Grinberg LT, Wang X, Wang C, Sohn PD, Theofilas P, Sidhu M, Arevalo JB, Heinsen H, Huang EJ, Rosen H, Miller BL, Gan L, Seeley WW. Argyrophilic grain disease differs from other tauopathies by lacking tau acetylation [Internet]. Acta Neuropathologica. 2013 ; 125( 4): 581-593.[citado 2024 ago. 17 ] Available from: http://link.springer.com/content/pdf/10.1007%2Fs00401-013-1080-2.pdf
Vancouver
Grinberg LT, Wang X, Wang C, Sohn PD, Theofilas P, Sidhu M, Arevalo JB, Heinsen H, Huang EJ, Rosen H, Miller BL, Gan L, Seeley WW. Argyrophilic grain disease differs from other tauopathies by lacking tau acetylation [Internet]. Acta Neuropathologica. 2013 ; 125( 4): 581-593.[citado 2024 ago. 17 ] Available from: http://link.springer.com/content/pdf/10.1007%2Fs00401-013-1080-2.pdf

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