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Artigo de periodico
DUSP3 modulates IRES-dependent translation of mRNAs through dephosphorylation of the HNRNPC protein in cells under genotoxic stimulus (2024)
Ferruzo, Pault Yeison Minaya ; Boell, Viktor Kalbermatter ; Russo, Lilian Cristina ; Oliveira, Carla Columbano de ; Forti, Fabio Luis
Source: Biology Cell. Unidade: IQ
Subjects: RNA, PROTEÍNAS QUINASES
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FERRUZO, Pault Yeison Minaya et al. DUSP3 modulates IRES-dependent translation of mRNAs through dephosphorylation of the HNRNPC protein in cells under genotoxic stimulus. Biology Cell, 2024Tradução . . Disponível em: https://dx.doi.org/10.1111/boc.202300128. Acesso em: 03 jun. 2024.
APA
Ferruzo, P. Y. M., Boell, V. K., Russo, L. C., Oliveira, C. C. de, & Forti, F. L. (2024). DUSP3 modulates IRES-dependent translation of mRNAs through dephosphorylation of the HNRNPC protein in cells under genotoxic stimulus. Biology Cell. doi:10.1111/boc.202300128
NLM
Ferruzo PYM, Boell VK, Russo LC, Oliveira CC de, Forti FL. DUSP3 modulates IRES-dependent translation of mRNAs through dephosphorylation of the HNRNPC protein in cells under genotoxic stimulus [Internet]. Biology Cell. 2024 ;[citado 2024 jun. 03 ] Available from: https://dx.doi.org/10.1111/boc.202300128
Vancouver
Ferruzo PYM, Boell VK, Russo LC, Oliveira CC de, Forti FL. DUSP3 modulates IRES-dependent translation of mRNAs through dephosphorylation of the HNRNPC protein in cells under genotoxic stimulus [Internet]. Biology Cell. 2024 ;[citado 2024 jun. 03 ] Available from: https://dx.doi.org/10.1111/boc.202300128
Artigo de periodico
Unrepaired base excision repair intermediates in template DNA strands trigger replication fork collapse and PARP inhibitor sensitivity (2023)
Benitez, Almudena Serrano ; Wells, Sophie E ; Clarke, Lylah Drummond ; Russo, Lilian Cristina ; Thomas, John Christopher; Leal, Giovanna Araki; Farrow, Mark ; Edgerton, James Michael ; Balasubramanian, Shankar; Yang, Ming; Frezza, christian ; Gautam, Amit; Brazina, Jan; Burdova, Kamila ; Hoch, Nicolas Carlos ; Jackson, Stephen P; Caldecott, Keith W
Source: The Embo Journal. Unidade: IQ
Subjects: GENOMAS, MUTAÇÃO GENÉTICA
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BENITEZ, Almudena Serrano et al. Unrepaired base excision repair intermediates in template DNA strands trigger replication fork collapse and PARP inhibitor sensitivity. The Embo Journal, v. 42, n. 18, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.15252/embj.2022113190. Acesso em: 03 jun. 2024.
APA
Benitez, A. S., Wells, S. E., Clarke, L. D., Russo, L. C., Thomas, J. C., Leal, G. A., et al. (2023). Unrepaired base excision repair intermediates in template DNA strands trigger replication fork collapse and PARP inhibitor sensitivity. The Embo Journal, 42( 18), 1-19. doi:10.15252/embj.2022113190
NLM
Benitez AS, Wells SE, Clarke LD, Russo LC, Thomas JC, Leal GA, Farrow M, Edgerton JM, Balasubramanian S, Yang M, Frezza christian, Gautam A, Brazina J, Burdova K, Hoch NC, Jackson SP, Caldecott KW. Unrepaired base excision repair intermediates in template DNA strands trigger replication fork collapse and PARP inhibitor sensitivity [Internet]. The Embo Journal. 2023 ; 42( 18): 1-19.[citado 2024 jun. 03 ] Available from: https://doi.org/10.15252/embj.2022113190
Vancouver
Benitez AS, Wells SE, Clarke LD, Russo LC, Thomas JC, Leal GA, Farrow M, Edgerton JM, Balasubramanian S, Yang M, Frezza christian, Gautam A, Brazina J, Burdova K, Hoch NC, Jackson SP, Caldecott KW. Unrepaired base excision repair intermediates in template DNA strands trigger replication fork collapse and PARP inhibitor sensitivity [Internet]. The Embo Journal. 2023 ; 42( 18): 1-19.[citado 2024 jun. 03 ] Available from: https://doi.org/10.15252/embj.2022113190
Trabalho de evento-resumo
LINC00941 is an oncogenic long non-coding RNA regulated by KRAS in pancreatic cancer (2022)
Corrêa, Thalita Bueno; Heidrich, Vitor ; Gomes Filho, Sandro Mascena; Pellegrina, Diogo Vieira da Silva ; Hasenkamp, Lutero Augusto; Bertoldi, Ester Risério Matos ; Magalhães, Yuli Thamires ; Russo, Lilian Cristina ; Hoch, Nicolas Carlos ; Forti, Fabio Luis ; Reis, Eduardo Moraes ; Bassères, Daniela Sanchez
Source: Abstract Book. Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology/SBBq. Unidades: IQ, BIOINFORMÁTICA
Subjects: ADENOCARCINOMA, BIOMARCADORES
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CORRÊA, Thalita Bueno et al. LINC00941 is an oncogenic long non-coding RNA regulated by KRAS in pancreatic cancer. 2022, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular, 2022. Disponível em: https://www2.sbbq.org.br/reuniao/2022/images/livro_completo.pdf. Acesso em: 03 jun. 2024.
APA
Corrêa, T. B., Heidrich, V., Gomes Filho, S. M., Pellegrina, D. V. da S., Hasenkamp, L. A., Bertoldi, E. R. M., et al. (2022). LINC00941 is an oncogenic long non-coding RNA regulated by KRAS in pancreatic cancer. In Abstract Book. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular. Recuperado de https://www2.sbbq.org.br/reuniao/2022/images/livro_completo.pdf
NLM
Corrêa TB, Heidrich V, Gomes Filho SM, Pellegrina DV da S, Hasenkamp LA, Bertoldi ERM, Magalhães YT, Russo LC, Hoch NC, Forti FL, Reis EM, Bassères DS. LINC00941 is an oncogenic long non-coding RNA regulated by KRAS in pancreatic cancer [Internet]. Abstract Book. 2022 ;[citado 2024 jun. 03 ] Available from: https://www2.sbbq.org.br/reuniao/2022/images/livro_completo.pdf
Vancouver
Corrêa TB, Heidrich V, Gomes Filho SM, Pellegrina DV da S, Hasenkamp LA, Bertoldi ERM, Magalhães YT, Russo LC, Hoch NC, Forti FL, Reis EM, Bassères DS. LINC00941 is an oncogenic long non-coding RNA regulated by KRAS in pancreatic cancer [Internet]. Abstract Book. 2022 ;[citado 2024 jun. 03 ] Available from: https://www2.sbbq.org.br/reuniao/2022/images/livro_completo.pdf
Artigo de periodico
Annotation and functional characterization of long noncoding RNAs deregulated in pancreatic adenocarcinoma (2022)
Paixão, Vinicius Ferreira da ; Sosa, Omar Júlio ; Pellegrina, Diogo Vieira da Silva; Dazzani, Bianca; Corrêa, Thalita Bueno; Bertoldi, Ester Risério Matos; Moraes, Luís Bruno da Cruz e Alves de; Pessoa, Diogo de Oliveira ; Oliveira, Victoria de Paiva; Zevallos, Ricardo Alberto Chiong; Russo, Lilian Cristina ; Forti, Fabio Luis ; Ferreira, João Eduardo ; Freitas, Helano Carioca; Jukemura, José ; Machado, Marcel Cerqueira Cesar ; Begnami, María Dirlei; Setubal, João Carlos ; Bassères, Daniela Sanchez ; Reis, Eduardo Moraes
Source: Cellular Oncology. Unidades: IQ, IME, FM, BIOINFORMÁTICA
Subjects: RNA, ADENOCARCINOMA
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PAIXÃO, Vinicius Ferreira da et al. Annotation and functional characterization of long noncoding RNAs deregulated in pancreatic adenocarcinoma. Cellular Oncology, v. 45, n. 3, p. 479-504, 2022Tradução . . Disponível em: https://doi.org/10.1007/s13402-022-00678-5. Acesso em: 03 jun. 2024.
APA
Paixão, V. F. da, Sosa, O. J., Pellegrina, D. V. da S., Dazzani, B., Corrêa, T. B., Bertoldi, E. R. M., et al. (2022). Annotation and functional characterization of long noncoding RNAs deregulated in pancreatic adenocarcinoma. Cellular Oncology, 45( 3), 479-504. doi:10.1007/s13402-022-00678-5
NLM
Paixão VF da, Sosa OJ, Pellegrina DV da S, Dazzani B, Corrêa TB, Bertoldi ERM, Moraes LB da C e A de, Pessoa D de O, Oliveira V de P, Zevallos RAC, Russo LC, Forti FL, Ferreira JE, Freitas HC, Jukemura J, Machado MCC, Begnami MD, Setubal JC, Bassères DS, Reis EM. Annotation and functional characterization of long noncoding RNAs deregulated in pancreatic adenocarcinoma [Internet]. Cellular Oncology. 2022 ; 45( 3): 479-504.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1007/s13402-022-00678-5
Vancouver
Paixão VF da, Sosa OJ, Pellegrina DV da S, Dazzani B, Corrêa TB, Bertoldi ERM, Moraes LB da C e A de, Pessoa D de O, Oliveira V de P, Zevallos RAC, Russo LC, Forti FL, Ferreira JE, Freitas HC, Jukemura J, Machado MCC, Begnami MD, Setubal JC, Bassères DS, Reis EM. Annotation and functional characterization of long noncoding RNAs deregulated in pancreatic adenocarcinoma [Internet]. Cellular Oncology. 2022 ; 45( 3): 479-504.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1007/s13402-022-00678-5
Artigo de periodico
Nucleophosmin protein dephosphorylation by DUSP3 is a fine-tuning regulator of p53 signaling to maintain genomic stability (2021)
Russo, Lilian Cristina ; Ferruzo, Pault Yeison Minaya ; Forti, Fabio Luis
Source: Frontiers in Cell and Developmental Biology. Unidade: IQ
Subjects: GENÔMICA, PROTEÍNAS TIROSINA FOSFATASES, DANO AO DNA
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RUSSO, Lilian Cristina e FERRUZO, Pault Yeison Minaya e FORTI, Fabio Luis. Nucleophosmin protein dephosphorylation by DUSP3 is a fine-tuning regulator of p53 signaling to maintain genomic stability. Frontiers in Cell and Developmental Biology, v. 9, p. 1-16 art. 624933, 2021Tradução . . Disponível em: https://doi.org/10.3389/fcell.2021.624933. Acesso em: 03 jun. 2024.
APA
Russo, L. C., Ferruzo, P. Y. M., & Forti, F. L. (2021). Nucleophosmin protein dephosphorylation by DUSP3 is a fine-tuning regulator of p53 signaling to maintain genomic stability. Frontiers in Cell and Developmental Biology, 9, 1-16 art. 624933. doi:10.3389/fcell.2021.624933
NLM
Russo LC, Ferruzo PYM, Forti FL. Nucleophosmin protein dephosphorylation by DUSP3 is a fine-tuning regulator of p53 signaling to maintain genomic stability [Internet]. Frontiers in Cell and Developmental Biology. 2021 ; 9 1-16 art. 624933.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3389/fcell.2021.624933
Vancouver
Russo LC, Ferruzo PYM, Forti FL. Nucleophosmin protein dephosphorylation by DUSP3 is a fine-tuning regulator of p53 signaling to maintain genomic stability [Internet]. Frontiers in Cell and Developmental Biology. 2021 ; 9 1-16 art. 624933.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3389/fcell.2021.624933
Artigo de periodico
UV radiation-induced impairment of cellular morphology and motility is enhanced by DUSP3/VHR loss and FAK activation (2021)
Pereira, Nadine Ranieri; Russo, Lilian Cristina ; Forti, Fabio Luis
Source: Cell Biochemistry and Biophysics. Unidade: IQ
Subjects: RADIAÇÃO ULTRAVIOLETA, ENZIMAS, PROTEÍNAS, CÉLULAS, REPARAÇÃO DE DNA
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PEREIRA, Nadine Ranieri e RUSSO, Lilian Cristina e FORTI, Fabio Luis. UV radiation-induced impairment of cellular morphology and motility is enhanced by DUSP3/VHR loss and FAK activation. Cell Biochemistry and Biophysics, v. 79, p. 261–269, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12013-021-00966-1. Acesso em: 03 jun. 2024.
APA
Pereira, N. R., Russo, L. C., & Forti, F. L. (2021). UV radiation-induced impairment of cellular morphology and motility is enhanced by DUSP3/VHR loss and FAK activation. Cell Biochemistry and Biophysics, 79, 261–269. doi:10.1007/s12013-021-00966-1
NLM
Pereira NR, Russo LC, Forti FL. UV radiation-induced impairment of cellular morphology and motility is enhanced by DUSP3/VHR loss and FAK activation [Internet]. Cell Biochemistry and Biophysics. 2021 ; 79 261–269.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1007/s12013-021-00966-1
Vancouver
Pereira NR, Russo LC, Forti FL. UV radiation-induced impairment of cellular morphology and motility is enhanced by DUSP3/VHR loss and FAK activation [Internet]. Cell Biochemistry and Biophysics. 2021 ; 79 261–269.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1007/s12013-021-00966-1
Artigo de periodico
The SARS-CoV-2 Nsp3 macrodomain reverses PARP9/DTX3L-dependent ADP-ribosylation induced by interferon signalling (2021)
Russo, Lilian Cristina ; Tomasin, Rebeka; Matos, Isaac de Araújo ; Manucci, Antonio Carlos; Sowa, Sven T; Dale, Katie; Caldecott, Keith W; Lehtiö, Lari ; Schechtman, Deborah ; Meotti, Flavia Carla ; Bruni-Cardoso, Alexandre ; Hoch, Nicolas Carlos
Source: Journal of Biological Chemistry. Unidade: IQ
Subjects: CORONAVIRUS, COVID-19, ANTIVIRAIS
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RUSSO, Lilian Cristina et al. The SARS-CoV-2 Nsp3 macrodomain reverses PARP9/DTX3L-dependent ADP-ribosylation induced by interferon signalling. Journal of Biological Chemistry, v. 297, n. 3, p. 1-14 art. 101041, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.jbc.2021.101041. Acesso em: 03 jun. 2024.
APA
Russo, L. C., Tomasin, R., Matos, I. de A., Manucci, A. C., Sowa, S. T., Dale, K., et al. (2021). The SARS-CoV-2 Nsp3 macrodomain reverses PARP9/DTX3L-dependent ADP-ribosylation induced by interferon signalling. Journal of Biological Chemistry, 297( 3), 1-14 art. 101041. doi:10.1016/j.jbc.2021.101041
NLM
Russo LC, Tomasin R, Matos I de A, Manucci AC, Sowa ST, Dale K, Caldecott KW, Lehtiö L, Schechtman D, Meotti FC, Bruni-Cardoso A, Hoch NC. The SARS-CoV-2 Nsp3 macrodomain reverses PARP9/DTX3L-dependent ADP-ribosylation induced by interferon signalling [Internet]. Journal of Biological Chemistry. 2021 ; 297( 3): 1-14 art. 101041.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.jbc.2021.101041
Vancouver
Russo LC, Tomasin R, Matos I de A, Manucci AC, Sowa ST, Dale K, Caldecott KW, Lehtiö L, Schechtman D, Meotti FC, Bruni-Cardoso A, Hoch NC. The SARS-CoV-2 Nsp3 macrodomain reverses PARP9/DTX3L-dependent ADP-ribosylation induced by interferon signalling [Internet]. Journal of Biological Chemistry. 2021 ; 297( 3): 1-14 art. 101041.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.jbc.2021.101041
Trabalho de evento-resumo periodico
DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress (2020)
Forti, Fabio Luis ; Russo, Lilian Cristina
Source: Cancer Research. Conference titles: AACR Annual Meeting. Unidade: IQ
Subjects: PROTEÍNAS NUCLEARES, GENÔMICA
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FORTI, Fabio Luis e RUSSO, Lilian Cristina. DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress. Cancer Research. Philadelphia: American Association for Cancer Research (AACR). Disponível em: https://doi.org/10.1158/1538-7445.AM2020-2372. Acesso em: 03 jun. 2024. , 2020
APA
Forti, F. L., & Russo, L. C. (2020). DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress. Cancer Research. Philadelphia: American Association for Cancer Research (AACR). doi:10.1158/1538-7445.AM2020-2372
NLM
Forti FL, Russo LC. DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress [Internet]. Cancer Research. 2020 ; 80( 16):[citado 2024 jun. 03 ] Available from: https://doi.org/10.1158/1538-7445.AM2020-2372
Vancouver
Forti FL, Russo LC. DUSP3-NPM-P53 axis: a new regulator ofgenomic stability of cells under genotoxic stress [Internet]. Cancer Research. 2020 ; 80( 16):[citado 2024 jun. 03 ] Available from: https://doi.org/10.1158/1538-7445.AM2020-2372
Artigo de periodico
DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins (2020)
Russo, Lilian Cristina ; Farias, Jéssica Oliveira; Forti, Fabio Luis
Source: Cell Cycle. Unidade: IQ
Subjects: DOENÇAS GENÉTICAS, PROLIFERAÇÃO CELULAR
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RUSSO, Lilian Cristina e FARIAS, Jéssica Oliveira e FORTI, Fabio Luis. DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins. Cell Cycle, v. 19, n. 12 p. 1545–1561, 2020Tradução . . Disponível em: https://doi.org/10.1080/15384101.2020.1762043. Acesso em: 03 jun. 2024.
APA
Russo, L. C., Farias, J. O., & Forti, F. L. (2020). DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins. Cell Cycle, 19( 12 p. 1545–1561). doi:10.1080/15384101.2020.1762043
NLM
Russo LC, Farias JO, Forti FL. DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins [Internet]. Cell Cycle. 2020 ; 19( 12 p. 1545–1561):[citado 2024 jun. 03 ] Available from: https://doi.org/10.1080/15384101.2020.1762043
Vancouver
Russo LC, Farias JO, Forti FL. DUSP3 maintains genomic stability and cell proliferation by modulating NER pathway and cell cycle regulatory proteins [Internet]. Cell Cycle. 2020 ; 19( 12 p. 1545–1561):[citado 2024 jun. 03 ] Available from: https://doi.org/10.1080/15384101.2020.1762043
Artigo de periodico
Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents (2020)
Silva, Luiz Eduardo ; Russo, Lilian Cristina ; Forti, Fabio Luis
Source: Experimental Cell Research. Unidade: IQ
Assunto: DNA
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SILVA, Luiz Eduardo e RUSSO, Lilian Cristina e FORTI, Fabio Luis. Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents. Experimental Cell Research, v. 395, p. 1-12 art. 112206, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.yexcr.2020.112206. Acesso em: 03 jun. 2024.
APA
Silva, L. E., Russo, L. C., & Forti, F. L. (2020). Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents. Experimental Cell Research, 395, 1-12 art. 112206. doi:10.1016/j.yexcr.2020.112206
NLM
Silva LE, Russo LC, Forti FL. Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents [Internet]. Experimental Cell Research. 2020 ; 395 1-12 art. 112206.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.yexcr.2020.112206
Vancouver
Silva LE, Russo LC, Forti FL. Overactivated Cdc42 acts through Cdc42EP3/Borg2 and NCK to trigger DNA damage response signaling and sensitize cells to DNA-damaging agents [Internet]. Experimental Cell Research. 2020 ; 395 1-12 art. 112206.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.yexcr.2020.112206
Artigo de periodico
Functionalized nanoparticles as adjuvant to increase the cytotoxicity of metallodrugs toward tumor cells (2019)
Vieira, Eduardo Guimarães; Miguel, Rodrigo Bernardi; Silva, Daniel Rodrigues da; Fazzi, Rodrigo Boni; Couto, Ricardo Alexandre Alves de ; Marin, Jayr Henrique; Temperini, Márcia Laudelina Arruda ; Shinohara, Jorge da Silva; Toma, Henrique Eisi ; Russo, Lilian Cristina; Magalhães, Yuli Thamires; Dias Filho, Newton Luiz; Forti, Fabio Luis ; Ferreira, Ana Maria da Costa
Source: New Journal of Chemistry. Unidade: IQ
Subjects: NANOPARTÍCULAS, CITOTOXICIDADE IMUNOLÓGICA
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VIEIRA, Eduardo Guimarães et al. Functionalized nanoparticles as adjuvant to increase the cytotoxicity of metallodrugs toward tumor cells. New Journal of Chemistry, v. 43, n. 1, p. 386-398, 2019Tradução . . Disponível em: https://doi.org/10.1039/c8nj04654a. Acesso em: 03 jun. 2024.
APA
Vieira, E. G., Miguel, R. B., Silva, D. R. da, Fazzi, R. B., Couto, R. A. A. de, Marin, J. H., et al. (2019). Functionalized nanoparticles as adjuvant to increase the cytotoxicity of metallodrugs toward tumor cells. New Journal of Chemistry, 43( 1), 386-398. doi:10.1039/c8nj04654a
NLM
Vieira EG, Miguel RB, Silva DR da, Fazzi RB, Couto RAA de, Marin JH, Temperini MLA, Shinohara J da S, Toma HE, Russo LC, Magalhães YT, Dias Filho NL, Forti FL, Ferreira AM da C. Functionalized nanoparticles as adjuvant to increase the cytotoxicity of metallodrugs toward tumor cells [Internet]. New Journal of Chemistry. 2019 ; 43( 1): 386-398.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1039/c8nj04654a
Vancouver
Vieira EG, Miguel RB, Silva DR da, Fazzi RB, Couto RAA de, Marin JH, Temperini MLA, Shinohara J da S, Toma HE, Russo LC, Magalhães YT, Dias Filho NL, Forti FL, Ferreira AM da C. Functionalized nanoparticles as adjuvant to increase the cytotoxicity of metallodrugs toward tumor cells [Internet]. New Journal of Chemistry. 2019 ; 43( 1): 386-398.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1039/c8nj04654a
Artigo de periodico
Intracellular peptides in cell biology and pharmacology (2019)
Araujo, Christiane Bezerra de; Heimann, Andrea Sterman ; Remer, Ricardo A. ; Russo, Lilian Cristina ; Colquhoun, Alison ; Forti, Fabio Luis ; Ferro, Emer Suavinho
Source: Biomolecules. Unidades: ICB, IQ
Subjects: BIOLOGIA CELULAR, FARMACOLOGIA, PEPTÍDEOS, EPILEPSIA, NEOPLASIAS, OBESIDADE, RESISTÊNCIA À INSULINA, CANABINOIDES, PROTEÍNAS G
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ARAUJO, Christiane Bezerra de et al. Intracellular peptides in cell biology and pharmacology. Biomolecules, v. 9, n. 4, p. 1-28, 2019Tradução . . Disponível em: https://doi.org/10.3390/biom9040150. Acesso em: 03 jun. 2024.
APA
Araujo, C. B. de, Heimann, A. S., Remer, R. A., Russo, L. C., Colquhoun, A., Forti, F. L., & Ferro, E. S. (2019). Intracellular peptides in cell biology and pharmacology. Biomolecules, 9( 4), 1-28. doi:10.3390/biom9040150
NLM
Araujo CB de, Heimann AS, Remer RA, Russo LC, Colquhoun A, Forti FL, Ferro ES. Intracellular peptides in cell biology and pharmacology [Internet]. Biomolecules. 2019 ; 9( 4): 1-28.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3390/biom9040150
Vancouver
Araujo CB de, Heimann AS, Remer RA, Russo LC, Colquhoun A, Forti FL, Ferro ES. Intracellular peptides in cell biology and pharmacology [Internet]. Biomolecules. 2019 ; 9( 4): 1-28.[citado 2024 jun. 03 ] Available from: https://doi.org/10.3390/biom9040150
Parte de monografia/livro
Assessing the roles of Rho GTPases in cell DNA repair by the nucleotide excision repair pathway (2018)
Russo, Lilian Cristina; Minaya, Pault Yeison; Silva, Luiz Eduardo da; Forti, Fabio Luis
Source: Rho GTPases: Methods and Protocols. Unidade: IQ
Subjects: DNA, NUCLEOTÍDEOS
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ABNT
RUSSO, Lilian Cristina et al. Assessing the roles of Rho GTPases in cell DNA repair by the nucleotide excision repair pathway. Rho GTPases: Methods and Protocols. Tradução . New York: Humana Press, 2018. . Disponível em: https://doi.org/10.1007/978-1-4939-8612-5_22. Acesso em: 03 jun. 2024.
APA
Russo, L. C., Minaya, P. Y., Silva, L. E. da, & Forti, F. L. (2018). Assessing the roles of Rho GTPases in cell DNA repair by the nucleotide excision repair pathway. In Rho GTPases: Methods and Protocols. New York: Humana Press. doi:10.1007/978-1-4939-8612-5_22
NLM
Russo LC, Minaya PY, Silva LE da, Forti FL. Assessing the roles of Rho GTPases in cell DNA repair by the nucleotide excision repair pathway [Internet]. In: Rho GTPases: Methods and Protocols. New York: Humana Press; 2018. [citado 2024 jun. 03 ] Available from: https://doi.org/10.1007/978-1-4939-8612-5_22
Vancouver
Russo LC, Minaya PY, Silva LE da, Forti FL. Assessing the roles of Rho GTPases in cell DNA repair by the nucleotide excision repair pathway [Internet]. In: Rho GTPases: Methods and Protocols. New York: Humana Press; 2018. [citado 2024 jun. 03 ] Available from: https://doi.org/10.1007/978-1-4939-8612-5_22
Artigo de periodico
Revisiting the roles of VHR/DUSP3 phosphatase in human diseases (2018)
Russo, Lilian Cristina; Farias, Jéssica Oliveira; Ferruzo, Pault Yeison Minaya; Monteiro, Lucas Falção; Forti, Fabio Luis
Source: Clinics. Unidade: IQ
Assunto: PROTEÍNAS TIROSINA FOSFATASES
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ABNT
RUSSO, Lilian Cristina et al. Revisiting the roles of VHR/DUSP3 phosphatase in human diseases. Clinics, v. 73, p. 1-7, 2018Tradução . . Disponível em: https://doi.org/10.6061/clinics/2018/e466s. Acesso em: 03 jun. 2024.
APA
Russo, L. C., Farias, J. O., Ferruzo, P. Y. M., Monteiro, L. F., & Forti, F. L. (2018). Revisiting the roles of VHR/DUSP3 phosphatase in human diseases. Clinics, 73, 1-7. doi:10.6061/clinics/2018/e466s
NLM
Russo LC, Farias JO, Ferruzo PYM, Monteiro LF, Forti FL. Revisiting the roles of VHR/DUSP3 phosphatase in human diseases [Internet]. Clinics. 2018 ; 73 1-7.[citado 2024 jun. 03 ] Available from: https://doi.org/10.6061/clinics/2018/e466s
Vancouver
Russo LC, Farias JO, Ferruzo PYM, Monteiro LF, Forti FL. Revisiting the roles of VHR/DUSP3 phosphatase in human diseases [Internet]. Clinics. 2018 ; 73 1-7.[citado 2024 jun. 03 ] Available from: https://doi.org/10.6061/clinics/2018/e466s
Artigo de periodico
DUSP3/VHR: a druggable dual phosphatase for human diseases (2018)
Monteiro, Lucas Falção; Ferruzo, Pault Yeison Minaya; Russo, Lilian Cristina; Farias, Jéssica Oliveira; Forti, Fabio Luis
Source: Reviews of Physiology Biochemistry and Pharmacology. Unidade: IQ
Subjects: PROTEÍNAS QUINASES, PROTEÍNAS TIROSINA FOSFATASES
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ABNT
MONTEIRO, Lucas Falção et al. DUSP3/VHR: a druggable dual phosphatase for human diseases. Reviews of Physiology Biochemistry and Pharmacology, v. 176, p. 1-35, 2018Tradução . . Disponível em: https://doi.org/10.1007/112_2018_12. Acesso em: 03 jun. 2024.
APA
Monteiro, L. F., Ferruzo, P. Y. M., Russo, L. C., Farias, J. O., & Forti, F. L. (2018). DUSP3/VHR: a druggable dual phosphatase for human diseases. Reviews of Physiology Biochemistry and Pharmacology, 176, 1-35. doi:10.1007/112_2018_12
NLM
Monteiro LF, Ferruzo PYM, Russo LC, Farias JO, Forti FL. DUSP3/VHR: a druggable dual phosphatase for human diseases [Internet]. Reviews of Physiology Biochemistry and Pharmacology. 2018 ; 176 1-35.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1007/112_2018_12
Vancouver
Monteiro LF, Ferruzo PYM, Russo LC, Farias JO, Forti FL. DUSP3/VHR: a druggable dual phosphatase for human diseases [Internet]. Reviews of Physiology Biochemistry and Pharmacology. 2018 ; 176 1-35.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1007/112_2018_12
Artigo de periodico
Loss of DUSP3 activity radiosensitizes human tumor cell lines via attenuation of DNA repair pathways (2017)
Torres, Thompson Eusébio Pavan; Russo, Lilian Cristina; Santos, Alexsandro dos; Marques, Gabriela Rodrigues; Magalhães, Yuli Thamires; Tabassum, Sartaj; Forti, Fabio Luis
Source: Biochimica et Biophysica Acta - General Subjects. Unidade: IQ
Subjects: RADIAÇÃO GAMA, DANO AO DNA
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ABNT
TORRES, Thompson Eusébio Pavan et al. Loss of DUSP3 activity radiosensitizes human tumor cell lines via attenuation of DNA repair pathways. Biochimica et Biophysica Acta - General Subjects, v. 1861, n. 7, p. 1879-1897, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbagen.2017.04.004. Acesso em: 03 jun. 2024.
APA
Torres, T. E. P., Russo, L. C., Santos, A. dos, Marques, G. R., Magalhães, Y. T., Tabassum, S., & Forti, F. L. (2017). Loss of DUSP3 activity radiosensitizes human tumor cell lines via attenuation of DNA repair pathways. Biochimica et Biophysica Acta - General Subjects, 1861( 7), 1879-1897. doi:10.1016/j.bbagen.2017.04.004
NLM
Torres TEP, Russo LC, Santos A dos, Marques GR, Magalhães YT, Tabassum S, Forti FL. Loss of DUSP3 activity radiosensitizes human tumor cell lines via attenuation of DNA repair pathways [Internet]. Biochimica et Biophysica Acta - General Subjects. 2017 ; 1861( 7): 1879-1897.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.bbagen.2017.04.004
Vancouver
Torres TEP, Russo LC, Santos A dos, Marques GR, Magalhães YT, Tabassum S, Forti FL. Loss of DUSP3 activity radiosensitizes human tumor cell lines via attenuation of DNA repair pathways [Internet]. Biochimica et Biophysica Acta - General Subjects. 2017 ; 1861( 7): 1879-1897.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.bbagen.2017.04.004
Artigo de periodico
Interferon-gamma activity is potentiated by an intracellular peptide derived from the human 19S ATPase regulatory subunit 4 of the proteasome (2017)
Monte, Elisabete Rodrigues C.; Rossato, Cristiano; Llanos, Ricardo Pariona; Russo, Lilian Cristina; Castro, Leandro Mantovani de; Gozzo, Fábio C.; Araujo, Christiane B. de; Peron, Jean Pierre Schatzmann ; Sant'Anna, Osvaldo Augusto; Ferro, Emer Suavinho ; Rioli, Vanessa
Source: Journal of Proteomics. Unidade: ICB
Subjects: FARMACOLOGIA, IMUNOLOGIA, INTERFERONS, METABOLISMO DE PROTEÍNA, ENZIMAS PROTEOLÍTICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MONTE, Elisabete Rodrigues C. et al. Interferon-gamma activity is potentiated by an intracellular peptide derived from the human 19S ATPase regulatory subunit 4 of the proteasome. Journal of Proteomics, v. 151, p. 74-82, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jprot.2016.08.003. Acesso em: 03 jun. 2024.
APA
Monte, E. R. C., Rossato, C., Llanos, R. P., Russo, L. C., Castro, L. M. de, Gozzo, F. C., et al. (2017). Interferon-gamma activity is potentiated by an intracellular peptide derived from the human 19S ATPase regulatory subunit 4 of the proteasome. Journal of Proteomics, 151, 74-82. doi:10.1016/j.jprot.2016.08.003
NLM
Monte ERC, Rossato C, Llanos RP, Russo LC, Castro LM de, Gozzo FC, Araujo CB de, Peron JPS, Sant'Anna OA, Ferro ES, Rioli V. Interferon-gamma activity is potentiated by an intracellular peptide derived from the human 19S ATPase regulatory subunit 4 of the proteasome [Internet]. Journal of Proteomics. 2017 ; 151 74-82.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.jprot.2016.08.003
Vancouver
Monte ERC, Rossato C, Llanos RP, Russo LC, Castro LM de, Gozzo FC, Araujo CB de, Peron JPS, Sant'Anna OA, Ferro ES, Rioli V. Interferon-gamma activity is potentiated by an intracellular peptide derived from the human 19S ATPase regulatory subunit 4 of the proteasome [Internet]. Journal of Proteomics. 2017 ; 151 74-82.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.jprot.2016.08.003
Artigo de periodico
A novel intracellular peptide derived from `G IND. 1´/S cyclin D2 induces cell death (2014)
Araujo, Christiane Bezerra de; Russo, Lilian Cristina; Castro, Leandro Mantovani de; Forti, Fabio Luis ; Monte, Elisabete R. do; Rioli, Vanessa; Gozzo, Fabio Cesar; Colquhoun, Alison ; Ferro, Emer Suavinho
Source: Journal of Biological Chemistry. Unidades: ICB, IQ
Subjects: PEPTÍDEOS, METABOLISMO DE PROTEÍNA
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ABNT
ARAUJO, Christiane Bezerra de et al. A novel intracellular peptide derived from `G IND. 1´/S cyclin D2 induces cell death. Journal of Biological Chemistry, v. 289, n. 24, p. 16711-16726, 2014Tradução . . Disponível em: https://doi.org/10.1074/jbc.M113.537118. Acesso em: 03 jun. 2024.
APA
Araujo, C. B. de, Russo, L. C., Castro, L. M. de, Forti, F. L., Monte, E. R. do, Rioli, V., et al. (2014). A novel intracellular peptide derived from `G IND. 1´/S cyclin D2 induces cell death. Journal of Biological Chemistry, 289( 24), 16711-16726. doi:10.1074/jbc.M113.537118
NLM
Araujo CB de, Russo LC, Castro LM de, Forti FL, Monte ER do, Rioli V, Gozzo FC, Colquhoun A, Ferro ES. A novel intracellular peptide derived from `G IND. 1´/S cyclin D2 induces cell death [Internet]. Journal of Biological Chemistry. 2014 ; 289( 24): 16711-16726.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1074/jbc.M113.537118
Vancouver
Araujo CB de, Russo LC, Castro LM de, Forti FL, Monte ER do, Rioli V, Gozzo FC, Colquhoun A, Ferro ES. A novel intracellular peptide derived from `G IND. 1´/S cyclin D2 induces cell death [Internet]. Journal of Biological Chemistry. 2014 ; 289( 24): 16711-16726.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1074/jbc.M113.537118
Artigo de periodico
Natural intracellular peptides can modulate the interactions of mouse brain proteins and thimet oligopeptidase with 14-3-3ε and calmodulin (2012)
Russo, Lilian Cristina; Asega, Amanda Francine; Castro, Leandro Mantovani de; Negraes, Priscilla D; Cruz, Lilian; Gozzo, Fabio Cesar; Ulrich, Henning ; Camargo, Antonio Carlos Martins de; Rioli, Vanessa; Ferro, Emer Suavinho
Source: Proteomics. Unidades: ICB, IQ
Subjects: CÁLCIO, PEPTÍDEOS, BIOLOGIA CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RUSSO, Lilian Cristina et al. Natural intracellular peptides can modulate the interactions of mouse brain proteins and thimet oligopeptidase with 14-3-3ε and calmodulin. Proteomics, v. 12, n. 17, p. 2641-2655, 2012Tradução . . Disponível em: https://doi.org/10.1002/pmic.201200032. Acesso em: 03 jun. 2024.
APA
Russo, L. C., Asega, A. F., Castro, L. M. de, Negraes, P. D., Cruz, L., Gozzo, F. C., et al. (2012). Natural intracellular peptides can modulate the interactions of mouse brain proteins and thimet oligopeptidase with 14-3-3ε and calmodulin. Proteomics, 12( 17), 2641-2655. doi:10.1002/pmic.201200032
NLM
Russo LC, Asega AF, Castro LM de, Negraes PD, Cruz L, Gozzo FC, Ulrich H, Camargo ACM de, Rioli V, Ferro ES. Natural intracellular peptides can modulate the interactions of mouse brain proteins and thimet oligopeptidase with 14-3-3ε and calmodulin [Internet]. Proteomics. 2012 ; 12( 17): 2641-2655.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1002/pmic.201200032
Vancouver
Russo LC, Asega AF, Castro LM de, Negraes PD, Cruz L, Gozzo FC, Ulrich H, Camargo ACM de, Rioli V, Ferro ES. Natural intracellular peptides can modulate the interactions of mouse brain proteins and thimet oligopeptidase with 14-3-3ε and calmodulin [Internet]. Proteomics. 2012 ; 12( 17): 2641-2655.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1002/pmic.201200032
Trabalho de evento
Analysis of intracellular peptidic content in beta 2 microglobulin/TAP1 knockout mice by mass spectrometry (2010)
Castro, Leandro Mantovani de; Berti, Denise Aparecida; Russo, Lilian Cristina; Coelho, Verônica; Oliveira, Vitor Marcelo Silveira Bueno Brandão de; Ferro, Emer Suavinho
Source: Program & Abstracts. Conference titles: Meeting of the Brazilian Society for Cell Biology - SBBC. Unidade: ICB
Assunto: HISTOLOGIA
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ABNT
CASTRO, Leandro Mantovani de et al. Analysis of intracellular peptidic content in beta 2 microglobulin/TAP1 knockout mice by mass spectrometry. 2010, Anais.. São Paulo: Sociedade Brasileira de Biologia Celular, 2010. . Acesso em: 03 jun. 2024.
APA
Castro, L. M. de, Berti, D. A., Russo, L. C., Coelho, V., Oliveira, V. M. S. B. B. de, & Ferro, E. S. (2010). Analysis of intracellular peptidic content in beta 2 microglobulin/TAP1 knockout mice by mass spectrometry. In Program & Abstracts. São Paulo: Sociedade Brasileira de Biologia Celular.
NLM
Castro LM de, Berti DA, Russo LC, Coelho V, Oliveira VMSBB de, Ferro ES. Analysis of intracellular peptidic content in beta 2 microglobulin/TAP1 knockout mice by mass spectrometry. Program & Abstracts. 2010 ;[citado 2024 jun. 03 ]
Vancouver
Castro LM de, Berti DA, Russo LC, Coelho V, Oliveira VMSBB de, Ferro ES. Analysis of intracellular peptidic content in beta 2 microglobulin/TAP1 knockout mice by mass spectrometry. Program & Abstracts. 2010 ;[citado 2024 jun. 03 ]

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