Fonte: Journal of the American Society of Nephrology. Nome do evento: ASN Renal Week. Unidade: FM
Assuntos: PROLIFERAÇÃO CELULAR, GLOMERUNEFRITE, OSTEOBLASTO
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DIAS, Cristiane B. et al. Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture. Journal of the American Society of Nephrology. Washington: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 03 nov. 2024. , 2002APA
Dias, C. B., Gueiros, J. E. B., Reis, L. M. dos, Graciolli, F. J., Caparbo, V. F., Pereira, R. C., et al. (2002). Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture. Journal of the American Society of Nephrology. Washington: Faculdade de Medicina, Universidade de São Paulo.NLM
Dias CB, Gueiros JEB, Reis LM dos, Graciolli FJ, Caparbo VF, Pereira RC, Moyses RMA, Barros RT, Woronik V, Jorgetti V. Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture. Journal of the American Society of Nephrology. 2002 ; 13 576A.[citado 2024 nov. 03 ]Vancouver
Dias CB, Gueiros JEB, Reis LM dos, Graciolli FJ, Caparbo VF, Pereira RC, Moyses RMA, Barros RT, Woronik V, Jorgetti V. Non-azotemic glomerulonephritic proteinuiric patients may have low turnover bone disease and their sera inhibidor osteoblastic proliferation in culture. Journal of the American Society of Nephrology. 2002 ; 13 576A.[citado 2024 nov. 03 ]
