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Artigo de periodico
Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma (2018)
Goto, Renata Nishida; Sobral, Lays Martin ; Sousa, Lucas Oliveira ; Garcia, Cristiana Bernadelli; Lopes, Norberto Peporine ; Marín-Prida, Javier; Ochoa-Rodríguez, Estael; Verdecia-Reyes, Yamila; Pardo-Andreu, Gilberto Lázaro; Curti, Carlos ; Leopoldino, Andréia Machado
Fonte: European Journal of Pharmacology. Unidade: FCFRP
Assuntos: ANTINEOPLÁSICOS, APOPTOSE, NEOPLASIAS DE CABEÇA E PESCOÇO, MITOCÔNDRIAS, NEOVASCULARIZAÇÃO PATOLÓGICA
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GOTO, Renata Nishida et al. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma. European Journal of Pharmacology, v. 819, p. 198-206, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2017.12.009. Acesso em: 26 maio 2024.
APA
Goto, R. N., Sobral, L. M., Sousa, L. O., Garcia, C. B., Lopes, N. P., Marín-Prida, J., et al. (2018). Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma. European Journal of Pharmacology, 819, 198-206. doi:10.1016/j.ejphar.2017.12.009
NLM
Goto RN, Sobral LM, Sousa LO, Garcia CB, Lopes NP, Marín-Prida J, Ochoa-Rodríguez E, Verdecia-Reyes Y, Pardo-Andreu GL, Curti C, Leopoldino AM. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma [Internet]. European Journal of Pharmacology. 2018 ; 819 198-206.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.ejphar.2017.12.009
Vancouver
Goto RN, Sobral LM, Sousa LO, Garcia CB, Lopes NP, Marín-Prida J, Ochoa-Rodríguez E, Verdecia-Reyes Y, Pardo-Andreu GL, Curti C, Leopoldino AM. Anti-cancer activity of a new dihydropyridine derivative, VdiE-2N, in head and neck squamous cell carcinoma [Internet]. European Journal of Pharmacology. 2018 ; 819 198-206.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.ejphar.2017.12.009
Artigo de periodico
QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload (2017)
Durruthy, Michael González; Castro, Micheli; Nunes, Silvana Manske; Lima, Juliane Ventura; Alberici, Luciane Carla; Naal, Zeki; Sawazaki, David T. Atique; Curti, Carlos; Ruas, Caroline Pires; Gelesky, Marcos A.; Roy, Kunal; González-Díaz, Humberto; Monserrat, José M.
Fonte: Carbon. Unidade: FCFRP
Assuntos: NANOTUBOS DE CARBONO, MITOCÔNDRIAS, CURVAS DE DOSE-RESPOSTA, TEORIA DE PERTURBAÇÃO PARA OPERADORES LINEARES
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DURRUTHY, Michael González et al. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload. Carbon, v. 115, p. 312-330, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.carbon.2017.01.002. Acesso em: 26 maio 2024.
APA
Durruthy, M. G., Castro, M., Nunes, S. M., Lima, J. V., Alberici, L. C., Naal, Z., et al. (2017). QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload. Carbon, 115, 312-330. doi:10.1016/j.carbon.2017.01.002
NLM
Durruthy MG, Castro M, Nunes SM, Lima JV, Alberici LC, Naal Z, Sawazaki DTA, Curti C, Ruas CP, Gelesky MA, Roy K, González-Díaz H, Monserrat JM. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload [Internet]. Carbon. 2017 ; 115 312-330.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.carbon.2017.01.002
Vancouver
Durruthy MG, Castro M, Nunes SM, Lima JV, Alberici LC, Naal Z, Sawazaki DTA, Curti C, Ruas CP, Gelesky MA, Roy K, González-Díaz H, Monserrat JM. QSPR/QSAR-based Perturbation Theory approach and mechanistic electrochemical assays on carbon nanotubes with optimal properties against mitochondrial Fenton reaction experimentally induced by Fe2+-overload [Internet]. Carbon. 2017 ; 115 312-330.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.carbon.2017.01.002
Artigo de periodico
SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation (2017)
Almeida, Luciana O.; Cavalcanti Neto, Marinaldo P.; Sousa, Lucas Oliveira; Tannous, Maryna A.; Curti, Carlos; Leopoldino, Andréia Machado
Fonte: Oncotarget. Unidade: FCFRP
Assuntos: EPIGÊNESE GENÉTICA, NEOPLASIAS, METILAÇÃO DE DNA, PROTEÍNAS, CROMATINA
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ALMEIDA, Luciana O. et al. SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation. Oncotarget, v. 8, n. 16, p. 26802-26818, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.15818. Acesso em: 26 maio 2024.
APA
Almeida, L. O., Cavalcanti Neto, M. P., Sousa, L. O., Tannous, M. A., Curti, C., & Leopoldino, A. M. (2017). SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation. Oncotarget, 8( 16), 26802-26818. doi:10.18632/oncotarget.15818
NLM
Almeida LO, Cavalcanti Neto MP, Sousa LO, Tannous MA, Curti C, Leopoldino AM. SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation [Internet]. Oncotarget. 2017 ; 8( 16): 26802-26818.[citado 2024 maio 26 ] Available from: https://doi.org/10.18632/oncotarget.15818
Vancouver
Almeida LO, Cavalcanti Neto MP, Sousa LO, Tannous MA, Curti C, Leopoldino AM. SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation [Internet]. Oncotarget. 2017 ; 8( 16): 26802-26818.[citado 2024 maio 26 ] Available from: https://doi.org/10.18632/oncotarget.15818
Artigo de periodico
Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes (2017)
Dechandt, Carlos R. P.; Zuccolotto-dos-Reis, Felippe H.; Teodoro, Bruno G.; Fernandes, Anna Maria A. P.; Eberlin, Marcos N.; Kettelhut, Isis do Carmo; Curti, Carlos; Alberici, Luciane Carla
Fonte: Journal of Bioenergetics and Biomembranes. Unidades: FMRP, FCFRP
Assuntos: CÉLULAS CULTIVADAS, MITOCÔNDRIAS, METABOLISMO DE GORDURA, FÍGADO GORDUROSO
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DECHANDT, Carlos R. P. et al. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes. Journal of Bioenergetics and Biomembranes, v. 49, n. 5, p. 399-411, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10863-017-9725-9. Acesso em: 26 maio 2024.
APA
Dechandt, C. R. P., Zuccolotto-dos-Reis, F. H., Teodoro, B. G., Fernandes, A. M. A. P., Eberlin, M. N., Kettelhut, I. do C., et al. (2017). Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes. Journal of Bioenergetics and Biomembranes, 49( 5), 399-411. doi:10.1007/s10863-017-9725-9
NLM
Dechandt CRP, Zuccolotto-dos-Reis FH, Teodoro BG, Fernandes AMAP, Eberlin MN, Kettelhut I do C, Curti C, Alberici LC. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 5): 399-411.[citado 2024 maio 26 ] Available from: https://doi.org/10.1007/s10863-017-9725-9
Vancouver
Dechandt CRP, Zuccolotto-dos-Reis FH, Teodoro BG, Fernandes AMAP, Eberlin MN, Kettelhut I do C, Curti C, Alberici LC. Triacsin C reduces lipid droplet formation and induces mitochondrial biogenesis in primary rat hepatocytes [Internet]. Journal of Bioenergetics and Biomembranes. 2017 ; 49( 5): 399-411.[citado 2024 maio 26 ] Available from: https://doi.org/10.1007/s10863-017-9725-9
Artigo de periodico
SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line (2017)
Sobral, Lays M.; Coletta, Ricardo D.; Alberici, Luciane Carla; Curti, Carlos; Leopoldino, Andréia Machado
Fonte: The FEBS Journal. Unidade: FCFRP
Assuntos: TOXICOLOGIA, CÉLULAS, METABOLISMO, MITOCÔNDRIAS, RESPIRAÇÃO
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SOBRAL, Lays M. et al. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, v. 284, n. 17, p. 2774-2785, 2017Tradução . . Disponível em: https://doi.org/10.1111/febs.14148. Acesso em: 26 maio 2024.
APA
Sobral, L. M., Coletta, R. D., Alberici, L. C., Curti, C., & Leopoldino, A. M. (2017). SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line. The FEBS Journal, 284( 17), 2774-2785. doi:10.1111/febs.14148
NLM
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 maio 26 ] Available from: https://doi.org/10.1111/febs.14148
Vancouver
Sobral LM, Coletta RD, Alberici LC, Curti C, Leopoldino AM. SET/I2PP2A overexpression induces phenotypic, molecular, and metabolic alterations in an oral keratinocyte cell line [Internet]. The FEBS Journal. 2017 ; 284( 17): 2774-2785.[citado 2024 maio 26 ] Available from: https://doi.org/10.1111/febs.14148
Artigo de periodico
Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants (2017)
González-Durruthy, Michael; Alberici, Luciane C.; Curti, Carlos; Naal, Zeki; Sawazaki, David Tatsuo A; Vázquez-Naya, José M.; González-Díaz, Humberto; Munteanu, Cristian R.
Fonte: Journal of Chemical Information and Modeling. Unidade: FCFRP
Assuntos: ENTROPIA, REGRESSÃO LINEAR, MITOCÔNDRIAS, NANOTUBOS DE CARBONO, BIOMEDICINA, OXIGÊNIO
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GONZÁLEZ-DURRUTHY, Michael et al. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants. Journal of Chemical Information and Modeling, v. 57, n. 5, p. 1029-1044, 2017Tradução . . Disponível em: https://doi.org/10.1021/acs.jcim.6b00458. Acesso em: 26 maio 2024.
APA
González-Durruthy, M., Alberici, L. C., Curti, C., Naal, Z., Sawazaki, D. T. A., Vázquez-Naya, J. M., et al. (2017). Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants. Journal of Chemical Information and Modeling, 57( 5), 1029-1044. doi:10.1021/acs.jcim.6b00458
NLM
González-Durruthy M, Alberici LC, Curti C, Naal Z, Sawazaki DTA, Vázquez-Naya JM, González-Díaz H, Munteanu CR. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants [Internet]. Journal of Chemical Information and Modeling. 2017 ; 57( 5): 1029-1044.[citado 2024 maio 26 ] Available from: https://doi.org/10.1021/acs.jcim.6b00458
Vancouver
González-Durruthy M, Alberici LC, Curti C, Naal Z, Sawazaki DTA, Vázquez-Naya JM, González-Díaz H, Munteanu CR. Experimental–computational study of carbon nanotube effects on mitochondrial respiration: in silico nano-QSPR machine learning models based on new raman spectra transform with markov–shannon entropy invariants [Internet]. Journal of Chemical Information and Modeling. 2017 ; 57( 5): 1029-1044.[citado 2024 maio 26 ] Available from: https://doi.org/10.1021/acs.jcim.6b00458
Artigo de periodico
The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice (2016)
Baraldi, Flávia G.; Vicentini, Tatiane M.; Teodoro, Bruno G.; Dalalio, Felipe M.; Dechandt, Carlos R. P.; Prado, Ieda M. R.; Curti, Carlos; Cardoso, Fernanda C.; Uyemura, Sérgio Akira; Alberici, Luciane Carla
Fonte: The Journal of Nutritional Biochemistry. Unidade: FCFRP
Assuntos: METABOLISMO, OBESIDADE, AZEITE, ÁCIDO LINOLEICO, ÁCIDOS GRAXOS OMEGA 6, MITOCÔNDRIAS
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BARALDI, Flávia G. et al. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice. The Journal of Nutritional Biochemistry, v. 28, p. 147-154, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2015.10.008. Acesso em: 26 maio 2024.
APA
Baraldi, F. G., Vicentini, T. M., Teodoro, B. G., Dalalio, F. M., Dechandt, C. R. P., Prado, I. M. R., et al. (2016). The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice. The Journal of Nutritional Biochemistry, 28, 147-154. doi:10.1016/j.jnutbio.2015.10.008
NLM
Baraldi FG, Vicentini TM, Teodoro BG, Dalalio FM, Dechandt CRP, Prado IMR, Curti C, Cardoso FC, Uyemura SA, Alberici LC. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice [Internet]. The Journal of Nutritional Biochemistry. 2016 ; 28 147-154.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.jnutbio.2015.10.008
Vancouver
Baraldi FG, Vicentini TM, Teodoro BG, Dalalio FM, Dechandt CRP, Prado IMR, Curti C, Cardoso FC, Uyemura SA, Alberici LC. The combination of conjugated linoleic acid (CLA) and extra virgin olive oil increases mitochondrial and body metabolism and prevents CLA-associated insulin resistance and liver hypertrophy in C57Bl/6 mice [Internet]. The Journal of Nutritional Biochemistry. 2016 ; 28 147-154.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.jnutbio.2015.10.008
Artigo de periodico
Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism (2016)
Rodrigues, Fernando P.; Carneiro, Zumira, A.; Mascharak, Pradip; Curti, Carlos; Silva, Roberto Santana da
Fonte: Coordination Chemistry Reviews. Unidade: FCFRP
Assuntos: RUTÊNIO, FÁRMACOS, CARCINOMA HEPATOCELULAR, NEOPLASIAS, CITOTOXINAS
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RODRIGUES, Fernando P. et al. Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism. Coordination Chemistry Reviews, v. 306, p. 701-707, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ccr.2015.03.028. Acesso em: 26 maio 2024.
APA
Rodrigues, F. P., Carneiro, Z., Mascharak, P., Curti, C., & Silva, R. S. da. (2016). Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism. Coordination Chemistry Reviews, 306, 701-707. doi:10.1016/j.ccr.2015.03.028
NLM
Rodrigues FP, Carneiro Z, Mascharak P, Curti C, Silva RS da. Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism [Internet]. Coordination Chemistry Reviews. 2016 ; 306 701-707.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.ccr.2015.03.028
Vancouver
Rodrigues FP, Carneiro Z, Mascharak P, Curti C, Silva RS da. Incorporation of a ruthenium nitrosyl complex into liposomes, the nitric oxide released from these liposomes and HepG2 cell death mechanism [Internet]. Coordination Chemistry Reviews. 2016 ; 306 701-707.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.ccr.2015.03.028
Artigo de periodico
Lymph node or perineural invasion is associated with low mirR-15a, miR-34c and miR-199b levels in head and neck squamous cell carcinoma (2016)
Sousa, Lucas O.; Sobral, Lays M.; Matsumoto, Camila S.; Saggioro, Fabiano Pinto; López, Rossana Verónica Mendoza; Panepucci, Rodrigo Alexandre; Curti, Carlos; Silva Júnior, Wilson Araújo da; Greene, Lewis Joel; Leopoldino, Andréia Machado
Fonte: BBA- Biochimica et Biophysica Acta. Clinical. Unidades: SVOI, FCFRP, FMRP
Assuntos: LINFONODOS, CARCINOMA DE CÉLULAS ESCAMOSAS, DIAGNÓSTICO, TERAPÊUTICA MÉDICA, RNA
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SOUSA, Lucas O. et al. Lymph node or perineural invasion is associated with low mirR-15a, miR-34c and miR-199b levels in head and neck squamous cell carcinoma. BBA- Biochimica et Biophysica Acta. Clinical, v. 6, p. 159-164, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.bbacli.2016.11.001. Acesso em: 26 maio 2024.
APA
Sousa, L. O., Sobral, L. M., Matsumoto, C. S., Saggioro, F. P., López, R. V. M., Panepucci, R. A., et al. (2016). Lymph node or perineural invasion is associated with low mirR-15a, miR-34c and miR-199b levels in head and neck squamous cell carcinoma. BBA- Biochimica et Biophysica Acta. Clinical, 6, 159-164. doi:10.1016/j.bbacli.2016.11.001
NLM
Sousa LO, Sobral LM, Matsumoto CS, Saggioro FP, López RVM, Panepucci RA, Curti C, Silva Júnior WA da, Greene LJ, Leopoldino AM. Lymph node or perineural invasion is associated with low mirR-15a, miR-34c and miR-199b levels in head and neck squamous cell carcinoma [Internet]. BBA- Biochimica et Biophysica Acta. Clinical. 2016 ; 6 159-164.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.bbacli.2016.11.001
Vancouver
Sousa LO, Sobral LM, Matsumoto CS, Saggioro FP, López RVM, Panepucci RA, Curti C, Silva Júnior WA da, Greene LJ, Leopoldino AM. Lymph node or perineural invasion is associated with low mirR-15a, miR-34c and miR-199b levels in head and neck squamous cell carcinoma [Internet]. BBA- Biochimica et Biophysica Acta. Clinical. 2016 ; 6 159-164.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.bbacli.2016.11.001
Artigo de periodico
Mitoprotective activity of oxidized carbon nanotubes against mitochondrial swelling induced in multipe experimental conditions and predictions with new expected-value perturbation theory (2015)
González-Durruthy, Michael; Monserrat, Jose Maria; Alberici, Luciane Carla; Naal, Zeki; Curti, Carlos; González-Díaz, Humberto
Fonte: RSC Advances. Unidade: FCFRP
Assuntos: CITOTOXINAS, MITOCÔNDRIAS, FÁRMACOS IMUNOSSUPRESSORES, NANOTECNOLOGIA
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ABNT
GONZÁLEZ-DURRUTHY, Michael et al. Mitoprotective activity of oxidized carbon nanotubes against mitochondrial swelling induced in multipe experimental conditions and predictions with new expected-value perturbation theory. RSC Advances, v. 5, p. 103229-103245, 2015Tradução . . Disponível em: https://doi.org/10.1039/C5RA14435C. Acesso em: 26 maio 2024.
APA
González-Durruthy, M., Monserrat, J. M., Alberici, L. C., Naal, Z., Curti, C., & González-Díaz, H. (2015). Mitoprotective activity of oxidized carbon nanotubes against mitochondrial swelling induced in multipe experimental conditions and predictions with new expected-value perturbation theory. RSC Advances, 5, 103229-103245. doi:10.1039/C5RA14435C
NLM
González-Durruthy M, Monserrat JM, Alberici LC, Naal Z, Curti C, González-Díaz H. Mitoprotective activity of oxidized carbon nanotubes against mitochondrial swelling induced in multipe experimental conditions and predictions with new expected-value perturbation theory [Internet]. RSC Advances. 2015 ; 5 103229-103245.[citado 2024 maio 26 ] Available from: https://doi.org/10.1039/C5RA14435C
Vancouver
González-Durruthy M, Monserrat JM, Alberici LC, Naal Z, Curti C, González-Díaz H. Mitoprotective activity of oxidized carbon nanotubes against mitochondrial swelling induced in multipe experimental conditions and predictions with new expected-value perturbation theory [Internet]. RSC Advances. 2015 ; 5 103229-103245.[citado 2024 maio 26 ] Available from: https://doi.org/10.1039/C5RA14435C
Artigo de periodico
Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring (2015)
Alves, José L. de Brito; Nogueira, Viviane O.; Cavalcanti Neto, Marinaldo Pacífico; Leopoldino, Andréia Machado; Curti, Carlos; Colombari, Débora S. A.; Colombari, Eduardo; Wanderley, Almir G.; Leandro, Carol G.; Zoccal, Daniel B.; Costa-Silva, João H.
Fonte: The Journal of Nutrition. Unidade: FCFRP
Assuntos: DESNUTRIÇÃO PROTEICO-ENERGÉTICA, HIPERTENSÃO, SISTEMA NERVOSO SIMPÁTICO, RATOS
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ALVES, José L. de Brito et al. Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring. The Journal of Nutrition, v. 145, n. 5, p. 907-914, 2015Tradução . . Disponível em: https://doi.org/10.3945/jn.114.202804. Acesso em: 26 maio 2024.
APA
Alves, J. L. de B., Nogueira, V. O., Cavalcanti Neto, M. P., Leopoldino, A. M., Curti, C., Colombari, D. S. A., et al. (2015). Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring. The Journal of Nutrition, 145( 5), 907-914. doi:10.3945/jn.114.202804
NLM
Alves JL de B, Nogueira VO, Cavalcanti Neto MP, Leopoldino AM, Curti C, Colombari DSA, Colombari E, Wanderley AG, Leandro CG, Zoccal DB, Costa-Silva JH. Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring [Internet]. The Journal of Nutrition. 2015 ; 145( 5): 907-914.[citado 2024 maio 26 ] Available from: https://doi.org/10.3945/jn.114.202804
Vancouver
Alves JL de B, Nogueira VO, Cavalcanti Neto MP, Leopoldino AM, Curti C, Colombari DSA, Colombari E, Wanderley AG, Leandro CG, Zoccal DB, Costa-Silva JH. Maternal protein restriction increases respiratory and sympathetic activities and sensitizes peripheral chemoreflex in male rat offspring [Internet]. The Journal of Nutrition. 2015 ; 145( 5): 907-914.[citado 2024 maio 26 ] Available from: https://doi.org/10.3945/jn.114.202804
Artigo de periodico
SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario (2015)
Almeida, Luciana O; Goto, Renata N; Cavalcanti Neto, Marinaldo P.; Sousa, Lucas Oliveira; Curti, Carlos; Leopoldino, Andréia Machado
Fonte: Biochemical and Biophysical Research Communications. Unidade: FCFRP
Assuntos: MITOCÔNDRIAS, PROTEÍNAS, NEOPLASIAS, DOENÇA DE ALZHEIMER
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ABNT
ALMEIDA, Luciana O et al. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, v. 458, n. 2, p. 300-306, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2015.01.104. Acesso em: 26 maio 2024.
APA
Almeida, L. O., Goto, R. N., Cavalcanti Neto, M. P., Sousa, L. O., Curti, C., & Leopoldino, A. M. (2015). SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario. Biochemical and Biophysical Research Communications, 458( 2), 300-306. doi:10.1016/j.bbrc.2015.01.104
NLM
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104
Vancouver
Almeida LO, Goto RN, Cavalcanti Neto MP, Sousa LO, Curti C, Leopoldino AM. SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario [Internet]. Biochemical and Biophysical Research Communications. 2015 ; 458( 2): 300-306.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.bbrc.2015.01.104
Artigo de periodico
The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling (2015)
Andreu, Gilberto L. Pardo; Reis, Felippe H. Z; Dalalio, Felipe M; Figueredo, Yanier Nuñez; Rubio, Osmany Cuesta; Uyemura, Sérgio Akira; Curti, Carlos; Alberici, Luciane Carla
Fonte: Chemico-Biological Interactions. Unidade: FCFRP
Assuntos: PRÓPOLIS, MITOCÔNDRIAS, TESTES DE TOXICIDADE, TOXICOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREU, Gilberto L. Pardo et al. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, v. 228, p. 28-34, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2015.01.010. Acesso em: 26 maio 2024.
APA
Andreu, G. L. P., Reis, F. H. Z., Dalalio, F. M., Figueredo, Y. N., Rubio, O. C., Uyemura, S. A., et al. (2015). The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling. Chemico-Biological Interactions, 228, 28-34. doi:10.1016/j.cbi.2015.01.010
NLM
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Vancouver
Andreu GLP, Reis FHZ, Dalalio FM, Figueredo YN, Rubio OC, Uyemura SA, Curti C, Alberici LC. The cytotoxic effects of brown Cuban propolis depend on the nemorosone content and may be mediated by mitochondrial uncoupling [Internet]. Chemico-Biological Interactions. 2015 ; 228 28-34.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.cbi.2015.01.010
Artigo de periodico
JM-20, a novel benzodiazepine-dihydropyridine hybrid molecule, protects mitochondria and prevents ischemic insult-mediated neural cell death in vitro (2014)
Nuñez-Figueredo, Yanier; Ramírez-Sánchez, Jeney; Delgado-Hernández, René; Porto-Verdecia, Marlene; Ochoa-Rodríguez, Estael; Verdecia-Reyes, Yamila; Marin-Prida, Javier; González-Durruthy, Michael; Uyemura, Sérgio Akira; Rodrigues, Fernando P.; Curti, Carlos; Souza, Diego O.; Pardo-Andreu, Gilberto L.
Fonte: European Journal of Pharmacology. Unidade: FCFRP
Assuntos: ACIDENTE VASCULAR CEREBRAL, ISQUEMIA CEREBRAL, FARMACOLOGIA, APOPTOSE, MITOCÔNDRIAS (EFEITOS DE DROGAS)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NUÑEZ-FIGUEREDO, Yanier et al. JM-20, a novel benzodiazepine-dihydropyridine hybrid molecule, protects mitochondria and prevents ischemic insult-mediated neural cell death in vitro. European Journal of Pharmacology, v. 726, p. 57-65, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2014.01.021. Acesso em: 26 maio 2024.
APA
Nuñez-Figueredo, Y., Ramírez-Sánchez, J., Delgado-Hernández, R., Porto-Verdecia, M., Ochoa-Rodríguez, E., Verdecia-Reyes, Y., et al. (2014). JM-20, a novel benzodiazepine-dihydropyridine hybrid molecule, protects mitochondria and prevents ischemic insult-mediated neural cell death in vitro. European Journal of Pharmacology, 726, 57-65. doi:10.1016/j.ejphar.2014.01.021
NLM
Nuñez-Figueredo Y, Ramírez-Sánchez J, Delgado-Hernández R, Porto-Verdecia M, Ochoa-Rodríguez E, Verdecia-Reyes Y, Marin-Prida J, González-Durruthy M, Uyemura SA, Rodrigues FP, Curti C, Souza DO, Pardo-Andreu GL. JM-20, a novel benzodiazepine-dihydropyridine hybrid molecule, protects mitochondria and prevents ischemic insult-mediated neural cell death in vitro [Internet]. European Journal of Pharmacology. 2014 ; 726 57-65.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.ejphar.2014.01.021
Vancouver
Nuñez-Figueredo Y, Ramírez-Sánchez J, Delgado-Hernández R, Porto-Verdecia M, Ochoa-Rodríguez E, Verdecia-Reyes Y, Marin-Prida J, González-Durruthy M, Uyemura SA, Rodrigues FP, Curti C, Souza DO, Pardo-Andreu GL. JM-20, a novel benzodiazepine-dihydropyridine hybrid molecule, protects mitochondria and prevents ischemic insult-mediated neural cell death in vitro [Internet]. European Journal of Pharmacology. 2014 ; 726 57-65.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.ejphar.2014.01.021
Trabalho de evento-resumo
Functional reconstitution of recombinant uncoupling protein from Aspergillus fumigatus (2014)
Cardoso, Fernanda Gomes; Suzuki, Silveli; Bálico, Laís de Lourdes de Lima; Santos, Emerson de Souza; Yoneda, Juliana Sakamoto; Ciancaglini, Pietro; Curti, Carlos; Uyemura, Sérgio Akira
Fonte: Abstracts. Nome do evento: Annual Meeting of Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). Unidades: FFCLRP, FCFRP
Assuntos: ASPERGILLUS, MITOCÔNDRIAS, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARDOSO, Fernanda Gomes et al. Functional reconstitution of recombinant uncoupling protein from Aspergillus fumigatus. 2014, Anais.. Foz do Iguaçu: SBBq, 2014. . Acesso em: 26 maio 2024.
APA
Cardoso, F. G., Suzuki, S., Bálico, L. de L. de L., Santos, E. de S., Yoneda, J. S., Ciancaglini, P., et al. (2014). Functional reconstitution of recombinant uncoupling protein from Aspergillus fumigatus. In Abstracts. Foz do Iguaçu: SBBq.
NLM
Cardoso FG, Suzuki S, Bálico L de L de L, Santos E de S, Yoneda JS, Ciancaglini P, Curti C, Uyemura SA. Functional reconstitution of recombinant uncoupling protein from Aspergillus fumigatus. Abstracts. 2014 ;[citado 2024 maio 26 ]
Vancouver
Cardoso FG, Suzuki S, Bálico L de L de L, Santos E de S, Yoneda JS, Ciancaglini P, Curti C, Uyemura SA. Functional reconstitution of recombinant uncoupling protein from Aspergillus fumigatus. Abstracts. 2014 ;[citado 2024 maio 26 ]
Artigo de periodico
Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation (2014)
Almeida, Luciana O.; Garcia, Cristiana Bernadelli; Silva, Flávia Amoroso Matos e; Curti, Carlos; Leopoldino, Andréia Machado
Fonte: Biochemical and Biophysical Research Communications. Unidade: FCFRP
Assuntos: CARCINOMA DE CÉLULAS ESCAMOSAS, LINHAGEM CELULAR, PROLIFERAÇÃO CELULAR, REGULAÇÃO GÊNICA, NEOPLASIAS DE CABEÇA E PESCOÇO, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Luciana O. et al. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation. Biochemical and Biophysical Research Communications, v. 445, n. 1, p. 196\2013202, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2014.01.175. Acesso em: 26 maio 2024.
APA
Almeida, L. O., Garcia, C. B., Silva, F. A. M. e, Curti, C., & Leopoldino, A. M. (2014). Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation. Biochemical and Biophysical Research Communications, 445( 1), 196\2013202. doi:10.1016/j.bbrc.2014.01.175
NLM
Almeida LO, Garcia CB, Silva FAM e, Curti C, Leopoldino AM. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation [Internet]. Biochemical and Biophysical Research Communications. 2014 ; 445( 1): 196\2013202.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.bbrc.2014.01.175
Vancouver
Almeida LO, Garcia CB, Silva FAM e, Curti C, Leopoldino AM. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation [Internet]. Biochemical and Biophysical Research Communications. 2014 ; 445( 1): 196\2013202.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.bbrc.2014.01.175
Artigo de periodico
Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models (2014)
Sobral, Lays M.; Sousa, Lucas Oliveira; Coletta, Ricardo Della; Cabral, Hamilton; Greene, Lewis Joel; Tajara, Eloiza Helena; Gutkind, J. Silvio; Curti, Carlos; Leopoldino, Andréia Machado
Fonte: Molecular Cancer. Unidades: FCFRP, FMRP
Assuntos: NEOPLASIAS DE CABEÇA E PESCOÇO, CARCINOMA DE CÉLULAS ESCAMOSAS, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOBRAL, Lays M. et al. Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models. Molecular Cancer, v. 13, 2014Tradução . . Disponível em: https://doi.org/10.1186/1476-4598-13-32. Acesso em: 26 maio 2024.
APA
Sobral, L. M., Sousa, L. O., Coletta, R. D., Cabral, H., Greene, L. J., Tajara, E. H., et al. (2014). Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models. Molecular Cancer, 13. doi:10.1186/1476-4598-13-32
NLM
Sobral LM, Sousa LO, Coletta RD, Cabral H, Greene LJ, Tajara EH, Gutkind JS, Curti C, Leopoldino AM. Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models [Internet]. Molecular Cancer. 2014 ; 13[citado 2024 maio 26 ] Available from: https://doi.org/10.1186/1476-4598-13-32
Vancouver
Sobral LM, Sousa LO, Coletta RD, Cabral H, Greene LJ, Tajara EH, Gutkind JS, Curti C, Leopoldino AM. Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models [Internet]. Molecular Cancer. 2014 ; 13[citado 2024 maio 26 ] Available from: https://doi.org/10.1186/1476-4598-13-32
Artigo de periodico
Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death (2014)
Reis, Felippe H. Z.; Pardo-Andreu, Gilberto L.; Nuñez-Figueredo, Yanier; Cuesta-Rubio, Osmany; Marín-Prida, Javier; Uyemura, Sérgio Akira; Curti, Carlos; Alberici, Luciane Carla
Fonte: Chemico-Biological Interactions. Unidade: FCFRP
Assuntos: ADENOSINA (METABOLISMO), ANTINEOPLÁSICOS (FARMACOLOGIA), TRANSPORTE BIOLÓGICO (EFEITOS DE DROGAS), MITOCÔNDRIAS, PRODUTOS NATURAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REIS, Felippe H. Z. et al. Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death. Chemico-Biological Interactions, v. 212, p. 20\201329, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2014.01.015. Acesso em: 26 maio 2024.
APA
Reis, F. H. Z., Pardo-Andreu, G. L., Nuñez-Figueredo, Y., Cuesta-Rubio, O., Marín-Prida, J., Uyemura, S. A., et al. (2014). Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death. Chemico-Biological Interactions, 212, 20\201329. doi:10.1016/j.cbi.2014.01.015
NLM
Reis FHZ, Pardo-Andreu GL, Nuñez-Figueredo Y, Cuesta-Rubio O, Marín-Prida J, Uyemura SA, Curti C, Alberici LC. Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death [Internet]. Chemico-Biological Interactions. 2014 ; 212 20\201329.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.cbi.2014.01.015
Vancouver
Reis FHZ, Pardo-Andreu GL, Nuñez-Figueredo Y, Cuesta-Rubio O, Marín-Prida J, Uyemura SA, Curti C, Alberici LC. Clusianone, a naturally occurring nemorosone regioisomer, uncouples rat liver mitochondria and induces HepG2 cell death [Internet]. Chemico-Biological Interactions. 2014 ; 212 20\201329.[citado 2024 maio 26 ] Available from: https://doi.org/10.1016/j.cbi.2014.01.015
Artigo de periodico
Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement (2014)
Lopes, Rheure Alves Moreira; Neves, Karla Bianca; Pestana, Cezar Rangel; Queiroz, André Lima; Zanotto, Camila Ziliotto; Chignalia, Andréia Zago; Valim, Yara Maria Lucisano; Silveira, Leonardo dos Reis; Curti, Carlos; Passaglia, Rita de Cassia Aleixo Tostes
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FCFRP, EEFERP, FMRP
Assuntos: TESTOSTERONA, APOPTOSE, CÉLULAS MUSCULARES, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Rheure Alves Moreira et al. Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement. American Journal of Physiology. Heart and Circulatory Physiology, v. 306, n. 11, p. H1485-H1494, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00809.2013. Acesso em: 26 maio 2024.
APA
Lopes, R. A. M., Neves, K. B., Pestana, C. R., Queiroz, A. L., Zanotto, C. Z., Chignalia, A. Z., et al. (2014). Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement. American Journal of Physiology. Heart and Circulatory Physiology, 306( 11), H1485-H1494. doi:10.1152/ajpheart.00809.2013
NLM
Lopes RAM, Neves KB, Pestana CR, Queiroz AL, Zanotto CZ, Chignalia AZ, Valim YML, Silveira L dos R, Curti C, Passaglia R de CAT. Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2014 ; 306( 11): H1485-H1494.[citado 2024 maio 26 ] Available from: https://doi.org/10.1152/ajpheart.00809.2013
Vancouver
Lopes RAM, Neves KB, Pestana CR, Queiroz AL, Zanotto CZ, Chignalia AZ, Valim YML, Silveira L dos R, Curti C, Passaglia R de CAT. Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2014 ; 306( 11): H1485-H1494.[citado 2024 maio 26 ] Available from: https://doi.org/10.1152/ajpheart.00809.2013
Trabalho de evento-resumo
Effect of the acyl-coa synthetase inhibitor triacsin C on mitochondrial energetics (2013)
Reis, F. H. Z.; Prado, Ieda Maria Razaboni; Curti, Carlos; Alberici, Luciane Carla
Fonte: Abstracts. Nome do evento: Annual Meeting of SBBq. Unidade: FCFRP
Assuntos: OBESIDADE, MITOCÔNDRIAS, METABOLISMO ENERGÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REIS, F. H. Z. et al. Effect of the acyl-coa synthetase inhibitor triacsin C on mitochondrial energetics. 2013, Anais.. Foz do Iguaçu: SBBq, 2013. . Acesso em: 26 maio 2024.
APA
Reis, F. H. Z., Prado, I. M. R., Curti, C., & Alberici, L. C. (2013). Effect of the acyl-coa synthetase inhibitor triacsin C on mitochondrial energetics. In Abstracts. Foz do Iguaçu: SBBq.
NLM
Reis FHZ, Prado IMR, Curti C, Alberici LC. Effect of the acyl-coa synthetase inhibitor triacsin C on mitochondrial energetics. Abstracts. 2013 ;[citado 2024 maio 26 ]
Vancouver
Reis FHZ, Prado IMR, Curti C, Alberici LC. Effect of the acyl-coa synthetase inhibitor triacsin C on mitochondrial energetics. Abstracts. 2013 ;[citado 2024 maio 26 ]

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