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Artigo de periodico
Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage (2022)
Pinto, Ana Paula ; Rocha, Alisson Luiz da ; Marafon, Bruno Brieda ; Nogueira, Jonatas Evandro ; Branco, Luiz Guilherme de Siqueira ; Pauli, José Rodrigo; Moura, Leandro Pereira de; Cintra, Dennys Esper; Ropelle, Eduardo Rochete; Silva, Adelino Sanchez Ramos da
Fonte: Clinical and Experimental Pharmacology and Physiology. Unidades: EEFERP, FORP, FMRP
Assuntos: DEGRANULAÇÃO CELULAR, INFLAMAÇÃO, INTERLEUCINA 6, EXERCÍCIO FÍSICO
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ABNT
PINTO, Ana Paula et al. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage. Clinical and Experimental Pharmacology and Physiology, v. 49, n. 8, p. 893-902, 2022Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.13677. Acesso em: 24 maio 2024.
APA
Pinto, A. P., Rocha, A. L. da, Marafon, B. B., Nogueira, J. E., Branco, L. G. de S., Pauli, J. R., et al. (2022). Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage. Clinical and Experimental Pharmacology and Physiology, 49( 8), 893-902. doi:10.1111/1440-1681.13677
NLM
Pinto AP, Rocha AL da, Marafon BB, Nogueira JE, Branco LG de S, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage [Internet]. Clinical and Experimental Pharmacology and Physiology. 2022 ; 49( 8): 893-902.[citado 2024 maio 24 ] Available from: https://doi.org/10.1111/1440-1681.13677
Vancouver
Pinto AP, Rocha AL da, Marafon BB, Nogueira JE, Branco LG de S, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Chronic rapamycin treatment decreases hepatic IL‐6 protein, but increases autophagy markers as a protective effect against the overtraining‐induced tissue damage [Internet]. Clinical and Experimental Pharmacology and Physiology. 2022 ; 49( 8): 893-902.[citado 2024 maio 24 ] Available from: https://doi.org/10.1111/1440-1681.13677
Artigo de periodico
Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats (2022)
Amorim, Mateus Ramos ; Jesus, Aline Alves de; Santos Junior, Nilton Nascimento dos ; Rocha, Maria José Alves da ; Nogueira, Jonatas Evandro ; Batalhão, Marcelo Eduardo; Cárnio, Evelin Capellari ; Branco, Luiz Guilherme de Siqueira
Fonte: Inflammation. Unidades: FORP, EEFERP, EERP, FMRP
Assuntos: CECO, CHOQUE SÉPTICO, ENDOTOXINAS, INFLAMAÇÃO, LIPOPOLISSACARÍDEOS, ESTRESSE OXIDATIVO
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ABNT
AMORIM, Mateus Ramos et al. Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats. Inflammation, v. 45, n. 3, p. 1239-1253, 2022Tradução . . Disponível em: https://doi.org/10.1007/s10753-021-01617-6. Acesso em: 24 maio 2024.
APA
Amorim, M. R., Jesus, A. A. de, Santos Junior, N. N. dos, Rocha, M. J. A. da, Nogueira, J. E., Batalhão, M. E., et al. (2022). Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats. Inflammation, 45( 3), 1239-1253. doi:10.1007/s10753-021-01617-6
NLM
Amorim MR, Jesus AA de, Santos Junior NN dos, Rocha MJA da, Nogueira JE, Batalhão ME, Cárnio EC, Branco LG de S. Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats [Internet]. Inflammation. 2022 ; 45( 3): 1239-1253.[citado 2024 maio 24 ] Available from: https://doi.org/10.1007/s10753-021-01617-6
Vancouver
Amorim MR, Jesus AA de, Santos Junior NN dos, Rocha MJA da, Nogueira JE, Batalhão ME, Cárnio EC, Branco LG de S. Autonomic disbalance during systemic inflammation is associated with oxidative stress changes in sepsis survivor rats [Internet]. Inflammation. 2022 ; 45( 3): 1239-1253.[citado 2024 maio 24 ] Available from: https://doi.org/10.1007/s10753-021-01617-6
Artigo de periodico
Molecular hydrogen potentiates hypothermia and prevents hypotension and fever in LPS-induced systemic inflammation (2019)
Saramago, Eduardo A.; Borges, Gabriela Silva ; Singolani Júnior, Carlitos G.; Nogueira, Jonatas Evandro; Soriano, Renato Nery; Cárnio, Evelin Capellari ; Branco, Luiz Guilherme de Siqueira
Fonte: Brain, Behavior, and Immunity. Unidades: EERP, FORP, EEFERP, EEFE
Assuntos: TEMPERATURA CORPORAL, ENDOTOXINAS, INFLAMAÇÃO, HIPOTENSÃO ANIMAL, HIPOTERMIA, INTERLEUCINA 6, SEPSE
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ABNT
SARAMAGO, Eduardo A. et al. Molecular hydrogen potentiates hypothermia and prevents hypotension and fever in LPS-induced systemic inflammation. Brain, Behavior, and Immunity, v. 75, p. 119-128, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2018.09.027. Acesso em: 24 maio 2024.
APA
Saramago, E. A., Borges, G. S., Singolani Júnior, C. G., Nogueira, J. E., Soriano, R. N., Cárnio, E. C., & Branco, L. G. de S. (2019). Molecular hydrogen potentiates hypothermia and prevents hypotension and fever in LPS-induced systemic inflammation. Brain, Behavior, and Immunity, 75, 119-128. doi:10.1016/j.bbi.2018.09.027
NLM
Saramago EA, Borges GS, Singolani Júnior CG, Nogueira JE, Soriano RN, Cárnio EC, Branco LG de S. Molecular hydrogen potentiates hypothermia and prevents hypotension and fever in LPS-induced systemic inflammation [Internet]. Brain, Behavior, and Immunity. 2019 ; 75 119-128.[citado 2024 maio 24 ] Available from: https://doi.org/10.1016/j.bbi.2018.09.027
Vancouver
Saramago EA, Borges GS, Singolani Júnior CG, Nogueira JE, Soriano RN, Cárnio EC, Branco LG de S. Molecular hydrogen potentiates hypothermia and prevents hypotension and fever in LPS-induced systemic inflammation [Internet]. Brain, Behavior, and Immunity. 2019 ; 75 119-128.[citado 2024 maio 24 ] Available from: https://doi.org/10.1016/j.bbi.2018.09.027
Artigo de periodico
Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production (2017)
Nogueira, Jonatas Evandro; Soriano, Renato Nery; Fernández, Rodrigo Alberto Restrepo; Francescato, Heloísa Della Coletta; Saia, Rafael Simone; Coimbra, Terezila Machado; Antunes-Rodrigues, José; Branco, Luiz Guilherme de Siqueira
Fonte: PLOS ONE. Unidades: EEFERP, FMRP, FORP
Assuntos: REGULAÇÃO DA TEMPERATURA CORPORAL, CITOCINAS, FEBRE, CONDICIONAMENTO FÍSICO, ÁREA PREÓPTICA DE ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NOGUEIRA, Jonatas Evandro et al. Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production. PLOS ONE, v. 12, n. 1, 2017Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0170468. Acesso em: 24 maio 2024.
APA
Nogueira, J. E., Soriano, R. N., Fernández, R. A. R., Francescato, H. D. C., Saia, R. S., Coimbra, T. M., et al. (2017). Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production. PLOS ONE, 12( 1). doi:10.1371/journal.pone.0170468
NLM
Nogueira JE, Soriano RN, Fernández RAR, Francescato HDC, Saia RS, Coimbra TM, Antunes-Rodrigues J, Branco LG de S. Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production [Internet]. PLOS ONE. 2017 ; 12( 1):[citado 2024 maio 24 ] Available from: https://doi.org/10.1371/journal.pone.0170468
Vancouver
Nogueira JE, Soriano RN, Fernández RAR, Francescato HDC, Saia RS, Coimbra TM, Antunes-Rodrigues J, Branco LG de S. Effect of physical exercise on the febrigenic signaling is modulated by preoptic hydrogen sulfide production [Internet]. PLOS ONE. 2017 ; 12( 1):[citado 2024 maio 24 ] Available from: https://doi.org/10.1371/journal.pone.0170468

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