Source: PLOS ONE. Unidade: FMRP
Subjects: MIOCARDIOPATIAS, MODELOS ANIMAIS DE DOENÇAS, EXPRESSÃO GÊNICA, SEPSE, MICROSCOPIA DE FLUORESCÊNCIA
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FREITAS, Ana Carolina Silva et al. Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition. PLOS ONE, v. 11, n. 11, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0166839. Acesso em: 14 jun. 2024.APA
Freitas, A. C. S., Figueiredo, M. J., Campos, E. C., Soave, D. F., Ramos, S. G., Tanowitz, H. B., & Celes, M. R. N. (2016). Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition. PLOS ONE, 11( 11). doi:10.1371/journal.pone.0166839NLM
Freitas ACS, Figueiredo MJ, Campos EC, Soave DF, Ramos SG, Tanowitz HB, Celes MRN. Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition [Internet]. PLOS ONE. 2016 ; 11( 11):[citado 2024 jun. 14 ] Available from: https://doi.org/10.1371/journal.pone.0166839Vancouver
Freitas ACS, Figueiredo MJ, Campos EC, Soave DF, Ramos SG, Tanowitz HB, Celes MRN. Activation of both the calpain and ubiquitin-proteasome systems contributes to septic cardiomyopathy through dystrophin loss/disruption and mTOR inhibition [Internet]. PLOS ONE. 2016 ; 11( 11):[citado 2024 jun. 14 ] Available from: https://doi.org/10.1371/journal.pone.0166839
