Filtros : "Indexado no Scopus" "Kowaltowski, Alicia Juliana" "IQ" Removidos: "FCFGRU999" "Tese (Doutorado)" "SBF" Limpar

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  • Source: Antioxidants & Redox Signaling. Unidade: IQ

    Subjects: MITOCÔNDRIAS, BIOQUÍMICA, ANGIOTENSINA II

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    • ABNT

      WOSNIAK JÚNIOR, João et al. Cross-talk between mitochondria and NADPH oxidase: effects of mild mitochondrial dysfunction on Angiotensin II-Mediated increase in nox isoform expression and activity in vascular smooth muscle cells. Antioxidants & Redox Signaling, v. 11, n. 6, p. 1265-1278, 2009Tradução . . Disponível em: https://doi.org/10.1089/ars.2008.2392. Acesso em: 13 jul. 2024.
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      Wosniak Júnior, J., Santos, C. X. da C. dos, Kowaltowski, A. J., & Laurindo, F. R. M. (2009). Cross-talk between mitochondria and NADPH oxidase: effects of mild mitochondrial dysfunction on Angiotensin II-Mediated increase in nox isoform expression and activity in vascular smooth muscle cells. Antioxidants & Redox Signaling, 11( 6), 1265-1278. doi:10.1089/ars.2008.2392
    • NLM

      Wosniak Júnior J, Santos CX da C dos, Kowaltowski AJ, Laurindo FRM. Cross-talk between mitochondria and NADPH oxidase: effects of mild mitochondrial dysfunction on Angiotensin II-Mediated increase in nox isoform expression and activity in vascular smooth muscle cells [Internet]. Antioxidants & Redox Signaling. 2009 ; 11( 6): 1265-1278.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1089/ars.2008.2392
    • Vancouver

      Wosniak Júnior J, Santos CX da C dos, Kowaltowski AJ, Laurindo FRM. Cross-talk between mitochondria and NADPH oxidase: effects of mild mitochondrial dysfunction on Angiotensin II-Mediated increase in nox isoform expression and activity in vascular smooth muscle cells [Internet]. Antioxidants & Redox Signaling. 2009 ; 11( 6): 1265-1278.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1089/ars.2008.2392
  • Source: Free Radical Biology and Medicine. Unidade: IQ

    Subjects: RADICAIS LIVRES, ESTRESSE OXIDATIVO, MITOCÔNDRIAS

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      TAHARA, Erich Birelli e NAVARETE, Felipe Donizeti Teston e KOWALTOWSKI, Alicia Juliana. Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation. Free Radical Biology and Medicine, v. 46, n. 9, p. 1283-1297, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2009.02.008. Acesso em: 13 jul. 2024.
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      Tahara, E. B., Navarete, F. D. T., & Kowaltowski, A. J. (2009). Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation. Free Radical Biology and Medicine, 46( 9), 1283-1297. doi:10.1016/j.freeradbiomed.2009.02.008
    • NLM

      Tahara EB, Navarete FDT, Kowaltowski AJ. Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation [Internet]. Free Radical Biology and Medicine. 2009 ; 46( 9): 1283-1297.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2009.02.008
    • Vancouver

      Tahara EB, Navarete FDT, Kowaltowski AJ. Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation [Internet]. Free Radical Biology and Medicine. 2009 ; 46( 9): 1283-1297.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2009.02.008
  • Source: Free Radical Biology and Medicine. Unidade: IQ

    Subjects: MITOCÔNDRIAS, BIOQUÍMICA, PEROXIDASE

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      FACUNDO, Heberty Di Tarso Fernandes e PAULA, Juliana Gabriela de e KOWALTOWSKI, Alicia Juliana. Mitochondrial ATP-sensitive 'K POT. +' channels are redox-sensitive pathways that control reactive oxygen species production. Free Radical Biology and Medicine, v. 42, n. 7, p. 1039-1048, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2007.01.001. Acesso em: 13 jul. 2024.
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      Facundo, H. D. T. F., Paula, J. G. de, & Kowaltowski, A. J. (2007). Mitochondrial ATP-sensitive 'K POT. +' channels are redox-sensitive pathways that control reactive oxygen species production. Free Radical Biology and Medicine, 42( 7), 1039-1048. doi:10.1016/j.freeradbiomed.2007.01.001
    • NLM

      Facundo HDTF, Paula JG de, Kowaltowski AJ. Mitochondrial ATP-sensitive 'K POT. +' channels are redox-sensitive pathways that control reactive oxygen species production [Internet]. Free Radical Biology and Medicine. 2007 ; 42( 7): 1039-1048.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2007.01.001
    • Vancouver

      Facundo HDTF, Paula JG de, Kowaltowski AJ. Mitochondrial ATP-sensitive 'K POT. +' channels are redox-sensitive pathways that control reactive oxygen species production [Internet]. Free Radical Biology and Medicine. 2007 ; 42( 7): 1039-1048.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2007.01.001
  • Source: Cardiovascular Research. Unidade: IQ

    Subjects: MITOCÔNDRIAS, ISQUEMIA, BIOQUÍMICA

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      CANCHERINI, Douglas Vasconcelos e QUELICONI, Bruno Barros e KOWALTOWSKI, Alicia Juliana. Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria. Cardiovascular Research, v. 73, n. 4, p. 720-728, 2007Tradução . . Acesso em: 13 jul. 2024.
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      Cancherini, D. V., Queliconi, B. B., & Kowaltowski, A. J. (2007). Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria. Cardiovascular Research, 73( 4), 720-728.
    • NLM

      Cancherini DV, Queliconi BB, Kowaltowski AJ. Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria. Cardiovascular Research. 2007 ; 73( 4): 720-728.[citado 2024 jul. 13 ]
    • Vancouver

      Cancherini DV, Queliconi BB, Kowaltowski AJ. Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria. Cardiovascular Research. 2007 ; 73( 4): 720-728.[citado 2024 jul. 13 ]
  • Source: Biochimica et Biophysica Acta - Molecular Basis of Disease. Unidade: IQ

    Subjects: ISQUEMIA, MITOCÔNDRIAS, CÁLCIO

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      FACUNDO, Hebert Di Tarso Fernandes e FORNAZARI, Maynara e KOWALTOWSKI, Alicia Juliana. Tissue protection mediated by mitochondrial 'K IND.+' channels. Biochimica et Biophysica Acta - Molecular Basis of Disease, v. 1762, n. 2, p. 202-212, 2006Tradução . . Acesso em: 13 jul. 2024.
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      Facundo, H. D. T. F., Fornazari, M., & Kowaltowski, A. J. (2006). Tissue protection mediated by mitochondrial 'K IND.+' channels. Biochimica et Biophysica Acta - Molecular Basis of Disease, 1762( 2), 202-212.
    • NLM

      Facundo HDTF, Fornazari M, Kowaltowski AJ. Tissue protection mediated by mitochondrial 'K IND.+' channels. Biochimica et Biophysica Acta - Molecular Basis of Disease. 2006 ; 1762( 2): 202-212.[citado 2024 jul. 13 ]
    • Vancouver

      Facundo HDTF, Fornazari M, Kowaltowski AJ. Tissue protection mediated by mitochondrial 'K IND.+' channels. Biochimica et Biophysica Acta - Molecular Basis of Disease. 2006 ; 1762( 2): 202-212.[citado 2024 jul. 13 ]
  • Source: Free Radical Biology and Medicine. Unidade: IQ

    Subjects: RADICAIS LIVRES, BIOQUÍMICA

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      FACUNDO, Heberty Di Tarso Fernandes et al. Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity. Free Radical Biology and Medicine, v. 40, n. 3, p. 469-479, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2005.08.041. Acesso em: 13 jul. 2024.
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      Facundo, H. D. T. F., Carreira, R. S., Paula, J. G. de, Santos, C. X. da C. dos, Ferranti, R., Laurindo, F. R. M., & Kowaltowski, A. J. (2006). Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity. Free Radical Biology and Medicine, 40( 3), 469-479. doi:10.1016/j.freeradbiomed.2005.08.041
    • NLM

      Facundo HDTF, Carreira RS, Paula JG de, Santos CX da C dos, Ferranti R, Laurindo FRM, Kowaltowski AJ. Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity [Internet]. Free Radical Biology and Medicine. 2006 ; 40( 3): 469-479.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2005.08.041
    • Vancouver

      Facundo HDTF, Carreira RS, Paula JG de, Santos CX da C dos, Ferranti R, Laurindo FRM, Kowaltowski AJ. Ischemic preconditioning requires increases in reactive oxygen release independent of mitochondrial 'K POT.+' channel activity [Internet]. Free Radical Biology and Medicine. 2006 ; 40( 3): 469-479.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2005.08.041
  • Source: Gastroenterology. Unidade: IQ

    Subjects: MITOCÔNDRIAS, BIOQUÍMICA

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      ALBERICI, Luciane Carla et al. Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity. Gastroenterology, v. 131, n. 4, p. 1228-1234, 2006Tradução . . Acesso em: 13 jul. 2024.
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      Alberici, L. C., Oliveira, H. C. F. de, Patrício, P. R., Kowaltowski, A. J., & Vercesi, A. E. (2006). Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity. Gastroenterology, 131( 4), 1228-1234.
    • NLM

      Alberici LC, Oliveira HCF de, Patrício PR, Kowaltowski AJ, Vercesi AE. Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity. Gastroenterology. 2006 ; 131( 4): 1228-1234.[citado 2024 jul. 13 ]
    • Vancouver

      Alberici LC, Oliveira HCF de, Patrício PR, Kowaltowski AJ, Vercesi AE. Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity. Gastroenterology. 2006 ; 131( 4): 1228-1234.[citado 2024 jul. 13 ]
  • Source: Experimental Neurology. Unidade: IQ

    Subjects: DOENÇAS METABÓLICAS, BIOQUÍMICA

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      KOWALTOWSKI, Alicia Juliana et al. Diazoxide protects against methylmalonate-induced neuronal toxicity. Experimental Neurology, v. 201, n. 1, p. 165-171, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2006.04.004. Acesso em: 13 jul. 2024.
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      Kowaltowski, A. J., Maciel, E. N., Fornazari, M., & Castilho, R. F. (2006). Diazoxide protects against methylmalonate-induced neuronal toxicity. Experimental Neurology, 201( 1), 165-171. doi:10.1016/j.expneurol.2006.04.004
    • NLM

      Kowaltowski AJ, Maciel EN, Fornazari M, Castilho RF. Diazoxide protects against methylmalonate-induced neuronal toxicity [Internet]. Experimental Neurology. 2006 ; 201( 1): 165-171.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.expneurol.2006.04.004
    • Vancouver

      Kowaltowski AJ, Maciel EN, Fornazari M, Castilho RF. Diazoxide protects against methylmalonate-induced neuronal toxicity [Internet]. Experimental Neurology. 2006 ; 201( 1): 165-171.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.expneurol.2006.04.004
  • Source: Free Radical Biology and Medicine. Unidade: IQ

    Subjects: RADICAIS LIVRES, APOPTOSE, ANTIOXIDANTES, NECROSE, BIOQUÍMICA

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      KOWALTOWSKI, Alicia Juliana e FENTON, Robert G. e FISKUM, Gary. Bcl-2 family proteins regulate mitochondrial reactive oxygen production and protect against oxidative stress. Free Radical Biology and Medicine, v. 37, n. 11, p. 1845-1853, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2004.09.005. Acesso em: 13 jul. 2024.
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      Kowaltowski, A. J., Fenton, R. G., & Fiskum, G. (2004). Bcl-2 family proteins regulate mitochondrial reactive oxygen production and protect against oxidative stress. Free Radical Biology and Medicine, 37( 11), 1845-1853. doi:10.1016/j.freeradbiomed.2004.09.005
    • NLM

      Kowaltowski AJ, Fenton RG, Fiskum G. Bcl-2 family proteins regulate mitochondrial reactive oxygen production and protect against oxidative stress [Internet]. Free Radical Biology and Medicine. 2004 ; 37( 11): 1845-1853.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2004.09.005
    • Vancouver

      Kowaltowski AJ, Fenton RG, Fiskum G. Bcl-2 family proteins regulate mitochondrial reactive oxygen production and protect against oxidative stress [Internet]. Free Radical Biology and Medicine. 2004 ; 37( 11): 1845-1853.[citado 2024 jul. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2004.09.005

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