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Artigo de periodico
Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction (2020)
Paulo, Michele ; Costa, Daniela E. F. R.; Bonaventura, Daniella; Lunardi, Claure N.; Bendhack, Lusiane Maria
Source: Current Pharmaceutical Design. Unidades: FCFRP, ICB
Subjects: CÉLULAS ENDOTELIAIS, ENDOTÉLIO VASCULAR, ÓXIDO NÍTRICO, FARMACOLOGIA, DOENÇAS VASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAULO, Michele et al. Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction. Current Pharmaceutical Design, v. 26, n. 30, p. 3748-3759, 2020Tradução . . Disponível em: https://doi.org/10.2174/1381612826666200519114442. Acesso em: 15 jun. 2024.
APA
Paulo, M., Costa, D. E. F. R., Bonaventura, D., Lunardi, C. N., & Bendhack, L. M. (2020). Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction. Current Pharmaceutical Design, 26( 30), 3748-3759. doi:10.2174/1381612826666200519114442
NLM
Paulo M, Costa DEFR, Bonaventura D, Lunardi CN, Bendhack LM. Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction [Internet]. Current Pharmaceutical Design. 2020 ; 26( 30): 3748-3759.[citado 2024 jun. 15 ] Available from: https://doi.org/10.2174/1381612826666200519114442
Vancouver
Paulo M, Costa DEFR, Bonaventura D, Lunardi CN, Bendhack LM. Nitric oxide donors as ootential drugs for the treatment of vascular diseases due to endothelium dysfunction [Internet]. Current Pharmaceutical Design. 2020 ; 26( 30): 3748-3759.[citado 2024 jun. 15 ] Available from: https://doi.org/10.2174/1381612826666200519114442
Artigo de periodico
NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats (2019)
Araújo, Alice Valença ; Andrade, Fernanda Aparecida de; Paulo, Michele ; Paula, Tiago Dal Cin de; Potje, Simone Regina ; Pereira, Amanda de Carvalho; Bendhack, Lusiane Maria
Source: Nitric Oxide. Unidades: FCFRP, FMRP
Subjects: ÓXIDO NÍTRICO, HIPERTENSÃO, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAÚJO, Alice Valença et al. NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats. Nitric Oxide, v. 86, p. 12-20, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2019.02.004. Acesso em: 15 jun. 2024.
APA
Araújo, A. V., Andrade, F. A. de, Paulo, M., Paula, T. D. C. de, Potje, S. R., Pereira, A. de C., & Bendhack, L. M. (2019). NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats. Nitric Oxide, 86, 12-20. doi:10.1016/j.niox.2019.02.004
NLM
Araújo AV, Andrade FA de, Paulo M, Paula TDC de, Potje SR, Pereira A de C, Bendhack LM. NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats [Internet]. Nitric Oxide. 2019 ; 86 12-20.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1016/j.niox.2019.02.004
Vancouver
Araújo AV, Andrade FA de, Paulo M, Paula TDC de, Potje SR, Pereira A de C, Bendhack LM. NO donors induce vascular relaxation by different cellular mechanisms in hypertensive and normotensive rats [Internet]. Nitric Oxide. 2019 ; 86 12-20.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1016/j.niox.2019.02.004
Artigo de periodico
Nitric oxide signaling and the cross talk with prostanoids pathways in vascular system (2017)
Silva, Bruno R; Paula, Tiago D; Paulo, Michele; Bendhack, Lusiane Maria
Source: Medicinal Chemistry. Unidade: FCFRP
Subjects: ÓXIDO NÍTRICO, DOENÇAS CARDIOVASCULARES, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Bruno R et al. Nitric oxide signaling and the cross talk with prostanoids pathways in vascular system. Medicinal Chemistry, v. 13, n. 4, p. 319-333, 2017Tradução . . Disponível em: https://doi.org/10.2174/1573406412666161228115627. Acesso em: 15 jun. 2024.
APA
Silva, B. R., Paula, T. D., Paulo, M., & Bendhack, L. M. (2017). Nitric oxide signaling and the cross talk with prostanoids pathways in vascular system. Medicinal Chemistry, 13( 4), 319-333. doi:10.2174/1573406412666161228115627
NLM
Silva BR, Paula TD, Paulo M, Bendhack LM. Nitric oxide signaling and the cross talk with prostanoids pathways in vascular system [Internet]. Medicinal Chemistry. 2017 ; 13( 4): 319-333.[citado 2024 jun. 15 ] Available from: https://doi.org/10.2174/1573406412666161228115627
Vancouver
Silva BR, Paula TD, Paulo M, Bendhack LM. Nitric oxide signaling and the cross talk with prostanoids pathways in vascular system [Internet]. Medicinal Chemistry. 2017 ; 13( 4): 319-333.[citado 2024 jun. 15 ] Available from: https://doi.org/10.2174/1573406412666161228115627
Artigo de periodico
The nitric oxide donor RuBPY does not induce in vitro cross-tolerance with acetylcholine (2017)
Paulo, Michele; Grando, Marcella Daruge; Silva, Roberto Santana da; Minshall, Richard D.; Bendhack, Lusiane Maria
Source: Nitric Oxide. Unidade: FCFRP
Subjects: NITRATOS, ÓXIDO NÍTRICO, VEIA CAVA, ACETILCOLINA, VASODILATAÇÃO, CITOTOXICIDADE IMUNOLÓGICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAULO, Michele et al. The nitric oxide donor RuBPY does not induce in vitro cross-tolerance with acetylcholine. Nitric Oxide, v. 69, p. 69-77, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2017.05.004. Acesso em: 15 jun. 2024.
APA
Paulo, M., Grando, M. D., Silva, R. S. da, Minshall, R. D., & Bendhack, L. M. (2017). The nitric oxide donor RuBPY does not induce in vitro cross-tolerance with acetylcholine. Nitric Oxide, 69, 69-77. doi:10.1016/j.niox.2017.05.004
NLM
Paulo M, Grando MD, Silva RS da, Minshall RD, Bendhack LM. The nitric oxide donor RuBPY does not induce in vitro cross-tolerance with acetylcholine [Internet]. Nitric Oxide. 2017 ; 69 69-77.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1016/j.niox.2017.05.004
Vancouver
Paulo M, Grando MD, Silva RS da, Minshall RD, Bendhack LM. The nitric oxide donor RuBPY does not induce in vitro cross-tolerance with acetylcholine [Internet]. Nitric Oxide. 2017 ; 69 69-77.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1016/j.niox.2017.05.004
Artigo de periodico
Investigation of a novel trinuclear μ-oxo ruthenium complex as a potential nitric oxide releaser for biological purposes (2015)
Cacita, Natacha; Possato, Bruna; Silva, Camila F. N. da; Paulo, Michele; Formiga, André Luiz Barbosa; Bendhack, Lusiane Maria; Nikolaou, Sofia
Source: Inorganica Chimica Acta. Unidades: FCFRP, FFCLRP
Subjects: RUTÊNIO, ÓXIDO NÍTRICO, VASODILATAÇÃO, ESPECTROSCOPIA ELETRÔNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CACITA, Natacha et al. Investigation of a novel trinuclear μ-oxo ruthenium complex as a potential nitric oxide releaser for biological purposes. Inorganica Chimica Acta, v. 429, p. 114-121, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ica.2015.01.038. Acesso em: 15 jun. 2024.
APA
Cacita, N., Possato, B., Silva, C. F. N. da, Paulo, M., Formiga, A. L. B., Bendhack, L. M., & Nikolaou, S. (2015). Investigation of a novel trinuclear μ-oxo ruthenium complex as a potential nitric oxide releaser for biological purposes. Inorganica Chimica Acta, 429, 114-121. doi:10.1016/j.ica.2015.01.038
NLM
Cacita N, Possato B, Silva CFN da, Paulo M, Formiga ALB, Bendhack LM, Nikolaou S. Investigation of a novel trinuclear μ-oxo ruthenium complex as a potential nitric oxide releaser for biological purposes [Internet]. Inorganica Chimica Acta. 2015 ; 429 114-121.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1016/j.ica.2015.01.038
Vancouver
Cacita N, Possato B, Silva CFN da, Paulo M, Formiga ALB, Bendhack LM, Nikolaou S. Investigation of a novel trinuclear μ-oxo ruthenium complex as a potential nitric oxide releaser for biological purposes [Internet]. Inorganica Chimica Acta. 2015 ; 429 114-121.[citado 2024 jun. 15 ] Available from: https://doi.org/10.1016/j.ica.2015.01.038

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