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Artigo de periodico
The syk-coupled C-type lectin receptors dectin-2 and dectin-3 are involved in Paracoccidioides Brasiliensis recognition by human plasmacytoid dendritic cells (2018)
Preite, Nycolas Willian; Feriotti, Claudia; Silva, Bruno Borges da; Loures, Flávio Vieira; Condino-Neto, Antonio; Pontillo, Alessandra ; Calich, Vera Lucia Garcia
Fonte: Frontiers in Immunology. Unidade: ICB
Assuntos: CÉLULAS DENDRÍTICAS, PARACOCCIDIOIDES, PARACOCCIDIOIDES BRASILIENSIS, PARACOCCIDIOIDOMICOSE, LECTINAS, CITOCINAS, FUNGOS
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PREITE, Nycolas Willian et al. The syk-coupled C-type lectin receptors dectin-2 and dectin-3 are involved in Paracoccidioides Brasiliensis recognition by human plasmacytoid dendritic cells. Frontiers in Immunology, v. 9, p. 1-14, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00464. Acesso em: 10 jun. 2024.
APA
Preite, N. W., Feriotti, C., Silva, B. B. da, Loures, F. V., Condino-Neto, A., Pontillo, A., & Calich, V. L. G. (2018). The syk-coupled C-type lectin receptors dectin-2 and dectin-3 are involved in Paracoccidioides Brasiliensis recognition by human plasmacytoid dendritic cells. Frontiers in Immunology, 9, 1-14. doi:10.3389/fimmu.2018.00464
NLM
Preite NW, Feriotti C, Silva BB da, Loures FV, Condino-Neto A, Pontillo A, Calich VLG. The syk-coupled C-type lectin receptors dectin-2 and dectin-3 are involved in Paracoccidioides Brasiliensis recognition by human plasmacytoid dendritic cells [Internet]. Frontiers in Immunology. 2018 ; 9 1-14.[citado 2024 jun. 10 ] Available from: https://doi.org/10.3389/fimmu.2018.00464
Vancouver
Preite NW, Feriotti C, Silva BB da, Loures FV, Condino-Neto A, Pontillo A, Calich VLG. The syk-coupled C-type lectin receptors dectin-2 and dectin-3 are involved in Paracoccidioides Brasiliensis recognition by human plasmacytoid dendritic cells [Internet]. Frontiers in Immunology. 2018 ; 9 1-14.[citado 2024 jun. 10 ] Available from: https://doi.org/10.3389/fimmu.2018.00464
Artigo de periodico
FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes (2018)
Silva, Paula Maciel da; Caldeira, Izabela Daniel Sardinha; Santos, Icaro de Assis; Nishiyama, Ana Claudia Oliveira Carreira; Siqueira, Flávia Ramos de; Antonioli, Eliane; Goldberg, Anna Carla Renata Krepel; Belizário, José Ernesto; Garay-Malpartida, Humberto Miguel
Fonte: BMC Cancer. Unidades: ICB, EACH
Assuntos: APOPTOSE, CITOCINAS, NEOPLASIAS PROSTÁTICAS, CÉLULAS CULTIVADAS DE TUMOR
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SILVA, Paula Maciel da et al. FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes. BMC Cancer, v. 18, n. 1, p. 14, 2018Tradução . . Disponível em: https://doi.org/10.1186/s12885-017-3950-9. Acesso em: 10 jun. 2024.
APA
Silva, P. M. da, Caldeira, I. D. S., Santos, I. de A., Nishiyama, A. C. O. C., Siqueira, F. R. de, Antonioli, E., et al. (2018). FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes. BMC Cancer, 18( 1), 14. doi:10.1186/s12885-017-3950-9
NLM
Silva PM da, Caldeira IDS, Santos I de A, Nishiyama ACOC, Siqueira FR de, Antonioli E, Goldberg ACRK, Belizário JE, Garay-Malpartida HM. FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes [Internet]. BMC Cancer. 2018 ; 18( 1): 14.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1186/s12885-017-3950-9
Vancouver
Silva PM da, Caldeira IDS, Santos I de A, Nishiyama ACOC, Siqueira FR de, Antonioli E, Goldberg ACRK, Belizário JE, Garay-Malpartida HM. FAM3B/PANDER inhibits cell death and increases prostate tumor growth by modulating the expression of Bcl-2 and Bcl-XL cell survival genes [Internet]. BMC Cancer. 2018 ; 18( 1): 14.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1186/s12885-017-3950-9
Artigo de periodico
Gain-of-function SNPs in NLRP3 and ILIB genes confer protection against obesity T2D: undiscovered role of inflammasome genetics in metabolic homeostasis? (2018)
Rodrigues, Juliana de Moraes; Lima, Dhemerson Souza de; Leal, Vinicius N. C.; Bosco, Adriana A.; Sandrim, Valéria; Pontillo, Alessandra
Fonte: Endocrine. Unidade: ICB
Assuntos: CITOCINAS, HOMEOSTASE, INFLAMAÇÃO, LEUCÓCITOS, ESTRESSE OXIDATIVO, CITOPLASMA
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RODRIGUES, Juliana de Moraes et al. Gain-of-function SNPs in NLRP3 and ILIB genes confer protection against obesity T2D: undiscovered role of inflammasome genetics in metabolic homeostasis?. Endocrine, v. 60, p. 368-371, 2018Tradução . . Disponível em: https://doi.org/10.1007/s12020-017-1343-0. Acesso em: 10 jun. 2024.
APA
Rodrigues, J. de M., Lima, D. S. de, Leal, V. N. C., Bosco, A. A., Sandrim, V., & Pontillo, A. (2018). Gain-of-function SNPs in NLRP3 and ILIB genes confer protection against obesity T2D: undiscovered role of inflammasome genetics in metabolic homeostasis? Endocrine, 60, 368-371. doi:10.1007/s12020-017-1343-0
NLM
Rodrigues J de M, Lima DS de, Leal VNC, Bosco AA, Sandrim V, Pontillo A. Gain-of-function SNPs in NLRP3 and ILIB genes confer protection against obesity T2D: undiscovered role of inflammasome genetics in metabolic homeostasis? [Internet]. Endocrine. 2018 ; 60 368-371.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1007/s12020-017-1343-0
Vancouver
Rodrigues J de M, Lima DS de, Leal VNC, Bosco AA, Sandrim V, Pontillo A. Gain-of-function SNPs in NLRP3 and ILIB genes confer protection against obesity T2D: undiscovered role of inflammasome genetics in metabolic homeostasis? [Internet]. Endocrine. 2018 ; 60 368-371.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1007/s12020-017-1343-0
Artigo de periodico
Hyperglycemia induces inflammatory mediators in the human chorionic villous (2018)
Corrêa-Silva, Simone; Alencar, Aline Paixão; Moreli, Jusciele B. ; Borbely, Alexandre Urban ; Lima, Larissa de Sá ; Scavone, Cristoforo ; Damasceno, Débora Cristina ; Rudge, Marilza Vieira Cunha ; Calderon, Iracema Mattos Paranhos ; Bevilacqua, Estela Maris Andrade Forell
Fonte: Cytokine. Unidade: ICB
Assuntos: HIPERGLICEMIA, INFLAMAÇÃO, PLACENTA, PLASMA, CITOCINAS, GLICOSE
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CORRÊA-SILVA, Simone et al. Hyperglycemia induces inflammatory mediators in the human chorionic villous. Cytokine, v. 111, p. 41-48, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.cyto.2018.07.020. Acesso em: 10 jun. 2024.
APA
Corrêa-Silva, S., Alencar, A. P., Moreli, J. B., Borbely, A. U., Lima, L. de S., Scavone, C., et al. (2018). Hyperglycemia induces inflammatory mediators in the human chorionic villous. Cytokine, 111, 41-48. doi:10.1016/j.cyto.2018.07.020
NLM
Corrêa-Silva S, Alencar AP, Moreli JB, Borbely AU, Lima L de S, Scavone C, Damasceno DC, Rudge MVC, Calderon IMP, Bevilacqua EMAF. Hyperglycemia induces inflammatory mediators in the human chorionic villous [Internet]. Cytokine. 2018 ; 111 41-48.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.cyto.2018.07.020
Vancouver
Corrêa-Silva S, Alencar AP, Moreli JB, Borbely AU, Lima L de S, Scavone C, Damasceno DC, Rudge MVC, Calderon IMP, Bevilacqua EMAF. Hyperglycemia induces inflammatory mediators in the human chorionic villous [Internet]. Cytokine. 2018 ; 111 41-48.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.cyto.2018.07.020
Artigo de periodico
mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function (2017)
Paschoal, Vivian Almeida; Amano, Mariane Tami; Oliveira,Thiago Belchior de; Magdalon, Juliana; Perandini, Patricia Chimin; Andrade, Maynara Lucca; Ortiz-Silva, Milene; Castro, Érique; Yamashita, Alex Shimura; Rosa Neto, José Cesar ; Câmara, Niels Olsen Saraiva ; Festuccia, William Tadeu Lara
Fonte: Immunobiology. Unidade: ICB
Assuntos: FISIOLOGIA, HISTOLOGIA, IMUNOLOGIA, MACRÓFAGOS, TECIDO ADIPOSO, INFLAMAÇÃO, FENÓTIPOS, CITOCINAS, OBESIDADE, CAMUNDONGOS
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PASCHOAL, Vivian Almeida et al. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function. Immunobiology, v. 222, n. 2, p. 261-271, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2016.09.014. Acesso em: 10 jun. 2024.
APA
Paschoal, V. A., Amano, M. T., Oliveira, T. B. de, Magdalon, J., Perandini, P. C., Andrade, M. L., et al. (2017). mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function. Immunobiology, 222( 2), 261-271. doi:10.1016/j.imbio.2016.09.014
NLM
Paschoal VA, Amano MT, Oliveira TB de, Magdalon J, Perandini PC, Andrade ML, Ortiz-Silva M, Castro É, Yamashita AS, Rosa Neto JC, Câmara NOS, Festuccia WTL. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function [Internet]. Immunobiology. 2017 ; 222( 2): 261-271.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.imbio.2016.09.014
Vancouver
Paschoal VA, Amano MT, Oliveira TB de, Magdalon J, Perandini PC, Andrade ML, Ortiz-Silva M, Castro É, Yamashita AS, Rosa Neto JC, Câmara NOS, Festuccia WTL. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function [Internet]. Immunobiology. 2017 ; 222( 2): 261-271.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.imbio.2016.09.014
Artigo de periodico
mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function (2017)
Paschoal, Vivian A.; Amano, Mariane T.; Belchior, Thiago; Magdalon, Juliana; Chimin, Patricia; Andrade, Maynara L.; Ortiz-Silva, Milene; Castro, Érique; Yamashita, Alex S.; Rosa Neto, José Cesar ; Câmara, Niels Olsen Saraiva ; Festuccia, William Tadeu Lara
Fonte: Immunobiology. Unidade: ICB
Assuntos: ADIPOSIDADE, CAMUNDONGOS, INFLAMAÇÃO, MACRÓFAGOS, OBESIDADE, CITOCINAS
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PASCHOAL, Vivian A. et al. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function. Immunobiology, v. 222, n. 2, p. 261-271, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2016.09.014. Acesso em: 10 jun. 2024.
APA
Paschoal, V. A., Amano, M. T., Belchior, T., Magdalon, J., Chimin, P., Andrade, M. L., et al. (2017). mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function. Immunobiology, 222( 2), 261-271. doi:10.1016/j.imbio.2016.09.014
NLM
Paschoal VA, Amano MT, Belchior T, Magdalon J, Chimin P, Andrade ML, Ortiz-Silva M, Castro É, Yamashita AS, Rosa Neto JC, Câmara NOS, Festuccia WTL. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function [Internet]. Immunobiology. 2017 ; 222( 2): 261-271.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.imbio.2016.09.014
Vancouver
Paschoal VA, Amano MT, Belchior T, Magdalon J, Chimin P, Andrade ML, Ortiz-Silva M, Castro É, Yamashita AS, Rosa Neto JC, Câmara NOS, Festuccia WTL. mTORC1 inhibition with rapamycin exacerbates adipose tissue inflammation in obese mice and dissociates macrophage phenotype from function [Internet]. Immunobiology. 2017 ; 222( 2): 261-271.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.imbio.2016.09.014
Artigo de periodico
Brazilian red propolis effects on peritoneal macrophage activity: nitric oxide, cell viability, pro-inflammatory cytokines and gene expression (2017)
Bueno-Silva, Bruno; Kawamoto, Dione; Suguimoto, Ellen Sayuri Ando; Casarin, Renato C. V.; Alencar, Severino Matias de; Rosalen, Pedro L.; Mayer, Marcia Pinto Alves
Fonte: Journal of Ethnopharmacology. Unidades: ESALQ, ICB
Assuntos: ANTI-INFLAMATÓRIOS, CITOCINAS, EXPRESSÃO GÊNICA, MACRÓFAGOS, ÓXIDO NÍTRICO, PRÓPOLIS
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BUENO-SILVA, Bruno et al. Brazilian red propolis effects on peritoneal macrophage activity: nitric oxide, cell viability, pro-inflammatory cytokines and gene expression. Journal of Ethnopharmacology, v. 207, p. 100-107, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jep.2017.06.015. Acesso em: 10 jun. 2024.
APA
Bueno-Silva, B., Kawamoto, D., Suguimoto, E. S. A., Casarin, R. C. V., Alencar, S. M. de, Rosalen, P. L., & Mayer, M. P. A. (2017). Brazilian red propolis effects on peritoneal macrophage activity: nitric oxide, cell viability, pro-inflammatory cytokines and gene expression. Journal of Ethnopharmacology, 207, 100-107. doi:10.1016/j.jep.2017.06.015
NLM
Bueno-Silva B, Kawamoto D, Suguimoto ESA, Casarin RCV, Alencar SM de, Rosalen PL, Mayer MPA. Brazilian red propolis effects on peritoneal macrophage activity: nitric oxide, cell viability, pro-inflammatory cytokines and gene expression [Internet]. Journal of Ethnopharmacology. 2017 ; 207 100-107.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.jep.2017.06.015
Vancouver
Bueno-Silva B, Kawamoto D, Suguimoto ESA, Casarin RCV, Alencar SM de, Rosalen PL, Mayer MPA. Brazilian red propolis effects on peritoneal macrophage activity: nitric oxide, cell viability, pro-inflammatory cytokines and gene expression [Internet]. Journal of Ethnopharmacology. 2017 ; 207 100-107.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.jep.2017.06.015
Artigo de periodico
Functional haplotypes in Interleukin 4 gene associated with periodontitis (2017)
Anovazzi, Giovana; Medeiros, Marcell Costa de; Pigossi, Suzane Cristina; Finoti, Livia Sertori; Mayer, Marcia Pinto Alves ; Rossa Junior, Carlos; Scarel-Caminaga, Raquel Mantuaneli
Fonte: PLOS ONE. Unidade: ICB
Assuntos: INTERLEUCINA 4, HAPLOTIPOS, PERIODONTITE CRÔNICA, FENÓTIPOS, CITOCINAS, ANTI-INFLAMATÓRIOS, VARIAÇÃO GENÉTICA, EXPRESSÃO GÊNICA
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ANOVAZZI, Giovana et al. Functional haplotypes in Interleukin 4 gene associated with periodontitis. PLOS ONE, v. 12, n. 1, p. 1-13, 2017Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0169870. Acesso em: 10 jun. 2024.
APA
Anovazzi, G., Medeiros, M. C. de, Pigossi, S. C., Finoti, L. S., Mayer, M. P. A., Rossa Junior, C., & Scarel-Caminaga, R. M. (2017). Functional haplotypes in Interleukin 4 gene associated with periodontitis. PLOS ONE, 12( 1), 1-13. doi:10.1371/journal.pone.0169870
NLM
Anovazzi G, Medeiros MC de, Pigossi SC, Finoti LS, Mayer MPA, Rossa Junior C, Scarel-Caminaga RM. Functional haplotypes in Interleukin 4 gene associated with periodontitis [Internet]. PLOS ONE. 2017 ; 12( 1): 1-13.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1371/journal.pone.0169870
Vancouver
Anovazzi G, Medeiros MC de, Pigossi SC, Finoti LS, Mayer MPA, Rossa Junior C, Scarel-Caminaga RM. Functional haplotypes in Interleukin 4 gene associated with periodontitis [Internet]. PLOS ONE. 2017 ; 12( 1): 1-13.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1371/journal.pone.0169870
Artigo de periodico
Anti-inflammatory mechanisms of neovestitol from Brazilian red propolis in LPS-activated macrophages (2017)
Bueno-Silva, Bruno; Rosalen, Pedro Luis; Alencar, Severino Matias de; Mayer, Marcia Pinto Alves
Fonte: Journal of Functional Foods. Unidades: ESALQ, ICB
Assuntos: ANTI-INFLAMATÓRIOS, CITOCINAS, MACRÓFAGOS, PRÓPOLIS
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BUENO-SILVA, Bruno et al. Anti-inflammatory mechanisms of neovestitol from Brazilian red propolis in LPS-activated macrophages. Journal of Functional Foods, v. 36, p. 440–447, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jff.2017.07.029. Acesso em: 10 jun. 2024.
APA
Bueno-Silva, B., Rosalen, P. L., Alencar, S. M. de, & Mayer, M. P. A. (2017). Anti-inflammatory mechanisms of neovestitol from Brazilian red propolis in LPS-activated macrophages. Journal of Functional Foods, 36, 440–447. doi:10.1016/j.jff.2017.07.029
NLM
Bueno-Silva B, Rosalen PL, Alencar SM de, Mayer MPA. Anti-inflammatory mechanisms of neovestitol from Brazilian red propolis in LPS-activated macrophages [Internet]. Journal of Functional Foods. 2017 ; 36 440–447.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.jff.2017.07.029
Vancouver
Bueno-Silva B, Rosalen PL, Alencar SM de, Mayer MPA. Anti-inflammatory mechanisms of neovestitol from Brazilian red propolis in LPS-activated macrophages [Internet]. Journal of Functional Foods. 2017 ; 36 440–447.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.jff.2017.07.029
Artigo de periodico
Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ (2017)
Cabral-Marques, Otavio; Ramos, Rodrigo Nalio; Schimke, Lena Friederick; Khan, Taj Ali; Amaral, Eduardo Pinheiro; Bomfim, Caio César Barbosa; Reis Junior, Osvaldo; França, Tabata Takahashi; Kubo, Christina Arslanian; Lima, Joanna Darck Carola Correia; Weber, Cristina Worm; Ferreira, Janaíra Fernandes; Tavares, Fabiola Scancetti; Sun, Jing; Lima, Maria Regina D'Império ; Seelaender, Marília; Calich, Vera Lucia Garcia; Barbuto, José Alexandre Marzagão ; Costa-Carvalho, Beatriz Tavares; Riemekasten, Gabriela; Seminario, Gisela; Bezrodnik, Liliana; Notarangelo, Luigi; Torgerson, Troy R.; Ochs, Hans D.; Condino-Neto, Antonio
Fonte: Journal of Allergy and Clinical Immunology. Unidade: ICB
Assuntos: HISTOLOGIA, IMUNOLOGIA, MACRÓFAGOS, ADJUVANTES IMUNOLÓGICOS, CITOCINAS
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CABRAL-MARQUES, Otavio et al. Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ. Journal of Allergy and Clinical Immunology, v. 139, n. 3, p. 900-912, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.jaci.2016.07.018. Acesso em: 10 jun. 2024.
APA
Cabral-Marques, O., Ramos, R. N., Schimke, L. F., Khan, T. A., Amaral, E. P., Bomfim, C. C. B., et al. (2017). Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ. Journal of Allergy and Clinical Immunology, 139( 3), 900-912. doi:10.1016/j.jaci.2016.07.018
NLM
Cabral-Marques O, Ramos RN, Schimke LF, Khan TA, Amaral EP, Bomfim CCB, Reis Junior O, França TT, Kubo CA, Lima JDCC, Weber CW, Ferreira JF, Tavares FS, Sun J, Lima MRD'I, Seelaender M, Calich VLG, Barbuto JAM, Costa-Carvalho BT, Riemekasten G, Seminario G, Bezrodnik L, Notarangelo L, Torgerson TR, Ochs HD, Condino-Neto A. Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ [Internet]. Journal of Allergy and Clinical Immunology. 2017 ; 139( 3): 900-912.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.jaci.2016.07.018
Vancouver
Cabral-Marques O, Ramos RN, Schimke LF, Khan TA, Amaral EP, Bomfim CCB, Reis Junior O, França TT, Kubo CA, Lima JDCC, Weber CW, Ferreira JF, Tavares FS, Sun J, Lima MRD'I, Seelaender M, Calich VLG, Barbuto JAM, Costa-Carvalho BT, Riemekasten G, Seminario G, Bezrodnik L, Notarangelo L, Torgerson TR, Ochs HD, Condino-Neto A. Human CD40 ligand deficiency dysregulates the macrophage transcriptome causing functional defects that are improved by exogenous IFN-γ [Internet]. Journal of Allergy and Clinical Immunology. 2017 ; 139( 3): 900-912.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.jaci.2016.07.018
Artigo de periodico
Functionality and opposite roles of two interleukin 4 haplotypes in immune cells (2017)
Anovazzi, G.; Medeiros, M. C.; Pigossi, S. C.; Finoti, L. S.; Souza, T. M. Moreira; Mayer, Marcia Pinto Alves ; Zanelli, C. F.; Valentini, S. R.; Rossa-Junior, C.; Scarel-Caminaga, R. M.
Fonte: Genes and Immunity. Unidade: ICB
Assuntos: MICROBIOLOGIA, HAPLOTIPOS, CITOCINAS, PERIODONTITE CRÔNICA, EXPRESSÃO GÊNICA, POLIMORFISMO, CITOMETRIA DE FLUXO, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
ANOVAZZI, G. et al. Functionality and opposite roles of two interleukin 4 haplotypes in immune cells. Genes and Immunity, v. 18, n. 1, p. 33-41, 2017Tradução . . Disponível em: https://doi.org/10.1038/gene.2016.47. Acesso em: 10 jun. 2024.
APA
Anovazzi, G., Medeiros, M. C., Pigossi, S. C., Finoti, L. S., Souza, T. M. M., Mayer, M. P. A., et al. (2017). Functionality and opposite roles of two interleukin 4 haplotypes in immune cells. Genes and Immunity, 18( 1), 33-41. doi:10.1038/gene.2016.47
NLM
Anovazzi G, Medeiros MC, Pigossi SC, Finoti LS, Souza TMM, Mayer MPA, Zanelli CF, Valentini SR, Rossa-Junior C, Scarel-Caminaga RM. Functionality and opposite roles of two interleukin 4 haplotypes in immune cells [Internet]. Genes and Immunity. 2017 ; 18( 1): 33-41.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1038/gene.2016.47
Vancouver
Anovazzi G, Medeiros MC, Pigossi SC, Finoti LS, Souza TMM, Mayer MPA, Zanelli CF, Valentini SR, Rossa-Junior C, Scarel-Caminaga RM. Functionality and opposite roles of two interleukin 4 haplotypes in immune cells [Internet]. Genes and Immunity. 2017 ; 18( 1): 33-41.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1038/gene.2016.47
Artigo de periodico
Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients (2016)
Bastos, Alliny de Souza; Graves, Dana T; Loureiro, Ana Paula de Melo ; Rossa Junior, Carlos; Corbi, Sâmia Cruz Tfaile; Frizzera, Fausto; Caminaga, Raquel Mantuaneli Scarel; Câmara, Niels Olsen Saraiva ; Andriankaja, Oelisoa M; Hiyane, Meire Ioshie; Orrico, Silvana Regina Perez
Fonte: Journal of Diabetes and Its Complications. Unidades: FCF, ICB
Assuntos: PEROXIDASE, DIABETES MELLITUS NÃO INSULINO-DEPENDENTE, CITOCINAS, DISLIPIDEMIAS, INFLAMAÇÃO
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ABNT
BASTOS, Alliny de Souza et al. Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients. Journal of Diabetes and Its Complications, v. 30, n. 8, p. 1593–1599, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jdiacomp.2016.07.011. Acesso em: 10 jun. 2024.
APA
Bastos, A. de S., Graves, D. T., Loureiro, A. P. de M., Rossa Junior, C., Corbi, S. C. T., Frizzera, F., et al. (2016). Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients. Journal of Diabetes and Its Complications, 30( 8), 1593–1599. doi:10.1016/j.jdiacomp.2016.07.011
NLM
Bastos A de S, Graves DT, Loureiro AP de M, Rossa Junior C, Corbi SCT, Frizzera F, Caminaga RMS, Câmara NOS, Andriankaja OM, Hiyane MI, Orrico SRP. Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients [Internet]. Journal of Diabetes and Its Complications. 2016 ; 30( 8): 1593–1599.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.07.011
Vancouver
Bastos A de S, Graves DT, Loureiro AP de M, Rossa Junior C, Corbi SCT, Frizzera F, Caminaga RMS, Câmara NOS, Andriankaja OM, Hiyane MI, Orrico SRP. Diabetes and increased lipid peroxidation are associated with systemic inflammation even in well-controlled patients [Internet]. Journal of Diabetes and Its Complications. 2016 ; 30( 8): 1593–1599.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.jdiacomp.2016.07.011
Artigo de periodico
Monocyte-derived dendritic cells reflect the immune functional status of a chromophobe renal cell carcinoma patient: could it be a general phenomenon? (2015)
Salomon, Maria A. Clavijo; Ramos, Rodrigo Nalio; Crippa, Alexandre; Pizzo, Célia Regina Pinto; Santos, Patricia Cruz Bergami; Barbuto, José Alexandre Marzagão
Fonte: Cancer Immunology and Immunotherapy. Unidade: ICB
Assuntos: IMUNOLOGIA, CÉLULAS DENDRITICAS, CÉLULAS CULTIVADAS DE TUMOR, CITOMETRIA DE FLUXO, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SALOMON, Maria A. Clavijo et al. Monocyte-derived dendritic cells reflect the immune functional status of a chromophobe renal cell carcinoma patient: could it be a general phenomenon?. Cancer Immunology and Immunotherapy, v. 64, n. 2, p. 161-171, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00262-014-1625-9. Acesso em: 10 jun. 2024.
APA
Salomon, M. A. C., Ramos, R. N., Crippa, A., Pizzo, C. R. P., Santos, P. C. B., & Barbuto, J. A. M. (2015). Monocyte-derived dendritic cells reflect the immune functional status of a chromophobe renal cell carcinoma patient: could it be a general phenomenon? Cancer Immunology and Immunotherapy, 64( 2), 161-171. doi:10.1007/s00262-014-1625-9
NLM
Salomon MAC, Ramos RN, Crippa A, Pizzo CRP, Santos PCB, Barbuto JAM. Monocyte-derived dendritic cells reflect the immune functional status of a chromophobe renal cell carcinoma patient: could it be a general phenomenon? [Internet]. Cancer Immunology and Immunotherapy. 2015 ; 64( 2): 161-171.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1007/s00262-014-1625-9
Vancouver
Salomon MAC, Ramos RN, Crippa A, Pizzo CRP, Santos PCB, Barbuto JAM. Monocyte-derived dendritic cells reflect the immune functional status of a chromophobe renal cell carcinoma patient: could it be a general phenomenon? [Internet]. Cancer Immunology and Immunotherapy. 2015 ; 64( 2): 161-171.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1007/s00262-014-1625-9
Artigo de periodico
Adipokines as drug targets in diabetes and underlying disturbances (2015)
Andrade-Oliveira, Vinicius; Câmara, Niels Olsen Saraiva ; Moraes-Vieira, Pedro Manoel Mendes de
Fonte: Journal of Diabetes Research. Unidade: ICB
Assuntos: IMUNOLOGIA, INFLAMAÇÃO, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDRADE-OLIVEIRA, Vinicius e CÂMARA, Niels Olsen Saraiva e MORAES-VIEIRA, Pedro Manoel Mendes de. Adipokines as drug targets in diabetes and underlying disturbances. Journal of Diabetes Research, v. 2015, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/681612. Acesso em: 10 jun. 2024.
APA
Andrade-Oliveira, V., Câmara, N. O. S., & Moraes-Vieira, P. M. M. de. (2015). Adipokines as drug targets in diabetes and underlying disturbances. Journal of Diabetes Research, 2015, 1-11. doi:10.1155/2015/681612
NLM
Andrade-Oliveira V, Câmara NOS, Moraes-Vieira PMM de. Adipokines as drug targets in diabetes and underlying disturbances [Internet]. Journal of Diabetes Research. 2015 ; 2015 1-11.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1155/2015/681612
Vancouver
Andrade-Oliveira V, Câmara NOS, Moraes-Vieira PMM de. Adipokines as drug targets in diabetes and underlying disturbances [Internet]. Journal of Diabetes Research. 2015 ; 2015 1-11.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1155/2015/681612
Artigo de periodico
Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis (2015)
Harbort, C. J.; Soeiro-Pereira, Paulo Vitor; Bernuth, Horst von; Kaindl, Angela M.; Costa-Carvalho, Beatriz Tavares; Condino-Neto, Antonio; Reichenbach, Janine; Roesler, Joachim; Zychlinsky, Arturo; Amulic, Borko
Fonte: Blood. Unidade: ICB
Assuntos: IMUNOLOGIA, APOPTOSE, NEUTRÓFILOS, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HARBORT, C. J. et al. Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis. Blood, v. 126, n. 26, p. 2842-2851, 2015Tradução . . Disponível em: https://doi.org/10.1182/blood-2015-05-645424. Acesso em: 10 jun. 2024.
APA
Harbort, C. J., Soeiro-Pereira, P. V., Bernuth, H. von, Kaindl, A. M., Costa-Carvalho, B. T., Condino-Neto, A., et al. (2015). Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis. Blood, 126( 26), 2842-2851. doi:10.1182/blood-2015-05-645424
NLM
Harbort CJ, Soeiro-Pereira PV, Bernuth H von, Kaindl AM, Costa-Carvalho BT, Condino-Neto A, Reichenbach J, Roesler J, Zychlinsky A, Amulic B. Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis [Internet]. Blood. 2015 ; 126( 26): 2842-2851.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1182/blood-2015-05-645424
Vancouver
Harbort CJ, Soeiro-Pereira PV, Bernuth H von, Kaindl AM, Costa-Carvalho BT, Condino-Neto A, Reichenbach J, Roesler J, Zychlinsky A, Amulic B. Neutrophil oxidative burst activates ATM to regulate cytokine production and apoptosis [Internet]. Blood. 2015 ; 126( 26): 2842-2851.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1182/blood-2015-05-645424
Artigo de periodico
Inflammatory markers in gingival crevicular fluid of periodontitis patients with type 2 diabetes mellitus according to glycemic control: A pilot study (2015)
Longo, Priscila Larcher; Artese, Hilana Paula Carillo; Horliana, Anna Carolina Ratto Tempestini; Gomes, Giovane Hisse; Romito, Giuseppe Alexandre; Dib, Sérgio Atala; Mayer, Marcia Pinto Alves
Fonte: Dental Research Journal. Unidades: FO, ICB
Assuntos: CITOCINAS, DIABETES MELLITUS, FLUÍDO CREVICULAR GENGIVAL, INFLAMAÇÃO, PERIODONTIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LONGO, Priscila Larcher et al. Inflammatory markers in gingival crevicular fluid of periodontitis patients with type 2 diabetes mellitus according to glycemic control: A pilot study. Dental Research Journal, v. 12, n. 5, p. 449–455, 2015Tradução . . Disponível em: https://doi.org/10.4103/1735-3327.166193. Acesso em: 10 jun. 2024.
APA
Longo, P. L., Artese, H. P. C., Horliana, A. C. R. T., Gomes, G. H., Romito, G. A., Dib, S. A., & Mayer, M. P. A. (2015). Inflammatory markers in gingival crevicular fluid of periodontitis patients with type 2 diabetes mellitus according to glycemic control: A pilot study. Dental Research Journal, 12( 5), 449–455. doi:10.4103/1735-3327.166193
NLM
Longo PL, Artese HPC, Horliana ACRT, Gomes GH, Romito GA, Dib SA, Mayer MPA. Inflammatory markers in gingival crevicular fluid of periodontitis patients with type 2 diabetes mellitus according to glycemic control: A pilot study [Internet]. Dental Research Journal. 2015 ; 12( 5): 449–455.[citado 2024 jun. 10 ] Available from: https://doi.org/10.4103/1735-3327.166193
Vancouver
Longo PL, Artese HPC, Horliana ACRT, Gomes GH, Romito GA, Dib SA, Mayer MPA. Inflammatory markers in gingival crevicular fluid of periodontitis patients with type 2 diabetes mellitus according to glycemic control: A pilot study [Internet]. Dental Research Journal. 2015 ; 12( 5): 449–455.[citado 2024 jun. 10 ] Available from: https://doi.org/10.4103/1735-3327.166193
Artigo de periodico
Behçet's disease heterogeneity: cytokine production and oxidative burst of phagocytes are altered in patients with severe manifestations (2015)
Perazzio, S. F.; Soeiro-Pereira, Paulo Vitor; Souza, A. W. S. de; Condino-Neto, Antonio; Andrade, L. E. C.
Fonte: Clinical and Experimental Rheumatology. Unidade: ICB
Assuntos: IMUNOLOGIA, DOENÇA AUTOIMUNES, SINDROME DE BEHCET, CITOCINAS, FAGÓCITOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PERAZZIO, S. F. et al. Behçet's disease heterogeneity: cytokine production and oxidative burst of phagocytes are altered in patients with severe manifestations. Clinical and Experimental Rheumatology, v. 33, n. 6 supl 94, p. S85-S95, 2015Tradução . . Disponível em: https://www.researchgate.net/publication/282125997. Acesso em: 10 jun. 2024.
APA
Perazzio, S. F., Soeiro-Pereira, P. V., Souza, A. W. S. de, Condino-Neto, A., & Andrade, L. E. C. (2015). Behçet's disease heterogeneity: cytokine production and oxidative burst of phagocytes are altered in patients with severe manifestations. Clinical and Experimental Rheumatology, 33( 6 supl 94), S85-S95. Recuperado de https://www.researchgate.net/publication/282125997
NLM
Perazzio SF, Soeiro-Pereira PV, Souza AWS de, Condino-Neto A, Andrade LEC. Behçet's disease heterogeneity: cytokine production and oxidative burst of phagocytes are altered in patients with severe manifestations [Internet]. Clinical and Experimental Rheumatology. 2015 ; 33( 6 supl 94): S85-S95.[citado 2024 jun. 10 ] Available from: https://www.researchgate.net/publication/282125997
Vancouver
Perazzio SF, Soeiro-Pereira PV, Souza AWS de, Condino-Neto A, Andrade LEC. Behçet's disease heterogeneity: cytokine production and oxidative burst of phagocytes are altered in patients with severe manifestations [Internet]. Clinical and Experimental Rheumatology. 2015 ; 33( 6 supl 94): S85-S95.[citado 2024 jun. 10 ] Available from: https://www.researchgate.net/publication/282125997
Artigo de periodico
CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis (2015)
Luque, Maria Carolina A.; Gutierrez, Paulo S.; Debbas, Victor; Kalil, Jorge; Stolf, Beatriz Simonsen
Fonte: Molecular Immunology. Unidades: FM, ICB
Assuntos: ADERÊNCIA CELULAR, CITOCINAS, INFLAMAÇÃO, ARTERIOSCLEROSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LUQUE, Maria Carolina A. et al. CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis. Molecular Immunology, v. 67, n. 2, p. 559-567, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.molimm.2015.07.028. Acesso em: 10 jun. 2024.
APA
Luque, M. C. A., Gutierrez, P. S., Debbas, V., Kalil, J., & Stolf, B. S. (2015). CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis. Molecular Immunology, 67( 2), 559-567. doi:10.1016/j.molimm.2015.07.028
NLM
Luque MCA, Gutierrez PS, Debbas V, Kalil J, Stolf BS. CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis [Internet]. Molecular Immunology. 2015 ; 67( 2): 559-567.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.molimm.2015.07.028
Vancouver
Luque MCA, Gutierrez PS, Debbas V, Kalil J, Stolf BS. CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis [Internet]. Molecular Immunology. 2015 ; 67( 2): 559-567.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.molimm.2015.07.028
Artigo de periodico
Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency (2015)
Walter, Jolan E.; Rosen, Lindsey B.; Csomos, Krisztian; Rosenberg, Jacob M.; Mathew, Divij; Keszei, Marton; Ujhazi, Boglarka; Chen, Karin; Lee, Yu Nee; Tirosh, Irit; Dobbs, Kerry; Al-Herz, Waleed; Cowan, Morton J.; Puck, Jennifer; Blessing, Jack J.; Grimley, Michael S.; Malech, Harry; Ravin, Suk See De; Gennery, Andrew R.; Abraham, Roshini S.; Joshi, Avni Y.; Boyce, Thomas G.; Butte, Manish J.; Nadeau, Kari C.; Balboni, Imelda; Sullivan, Kathleen E.; Akhter, Javeed; Adeli, Mehdi; El-Feky, Reem A.; El-Ghoneimy, Dalia H.; Dbaibo, Ghassan; Wakim, Rima; Azzari, Chiara; Palma, Paolo; Cancrini, Caterina; Capuder, Kelly; Condino-Neto, Antonio; Costa-Carvalho, Beatriz Tavares; Oliveira, João Bosco; Roifman, Chaim; Buchbinder, David; Kumanovics, Attila; Franco, Jose Luis; Niehues, Tim; Schuetz, Catharina; Kuijpers, Taco; Yee, Christina; Chou, Janet; Masaad, Michel J.; Geha, Raif; Uzel, Gulbu; Gelman, Rebecca; Holland, Steven M.; Recher, Mike; Utz, Paul J.; Browne, Sarah K.; Notarangelo, Luigi D.
Fonte: Journal of Clinical Investigation. Unidade: ICB
Assuntos: IMUNOLOGIA, ANTICORPOS, CITOCINAS, MUTAÇÃO GENÉTICA, DOENÇAS AUTOIMUNE, DOENÇAS GENÉTICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WALTER, Jolan E. et al. Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency. Journal of Clinical Investigation, v. 125, n. 11, p. 4135-4148, 2015Tradução . . Disponível em: https://doi.org/10.1172/JCI80477. Acesso em: 10 jun. 2024.
APA
Walter, J. E., Rosen, L. B., Csomos, K., Rosenberg, J. M., Mathew, D., Keszei, M., et al. (2015). Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency. Journal of Clinical Investigation, 125( 11), 4135-4148. doi:10.1172/JCI80477
NLM
Walter JE, Rosen LB, Csomos K, Rosenberg JM, Mathew D, Keszei M, Ujhazi B, Chen K, Lee YN, Tirosh I, Dobbs K, Al-Herz W, Cowan MJ, Puck J, Blessing JJ, Grimley MS, Malech H, Ravin SSD, Gennery AR, Abraham RS, Joshi AY, Boyce TG, Butte MJ, Nadeau KC, Balboni I, Sullivan KE, Akhter J, Adeli M, El-Feky RA, El-Ghoneimy DH, Dbaibo G, Wakim R, Azzari C, Palma P, Cancrini C, Capuder K, Condino-Neto A, Costa-Carvalho BT, Oliveira JB, Roifman C, Buchbinder D, Kumanovics A, Franco JL, Niehues T, Schuetz C, Kuijpers T, Yee C, Chou J, Masaad MJ, Geha R, Uzel G, Gelman R, Holland SM, Recher M, Utz PJ, Browne SK, Notarangelo LD. Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency [Internet]. Journal of Clinical Investigation. 2015 ; 125( 11): 4135-4148.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1172/JCI80477
Vancouver
Walter JE, Rosen LB, Csomos K, Rosenberg JM, Mathew D, Keszei M, Ujhazi B, Chen K, Lee YN, Tirosh I, Dobbs K, Al-Herz W, Cowan MJ, Puck J, Blessing JJ, Grimley MS, Malech H, Ravin SSD, Gennery AR, Abraham RS, Joshi AY, Boyce TG, Butte MJ, Nadeau KC, Balboni I, Sullivan KE, Akhter J, Adeli M, El-Feky RA, El-Ghoneimy DH, Dbaibo G, Wakim R, Azzari C, Palma P, Cancrini C, Capuder K, Condino-Neto A, Costa-Carvalho BT, Oliveira JB, Roifman C, Buchbinder D, Kumanovics A, Franco JL, Niehues T, Schuetz C, Kuijpers T, Yee C, Chou J, Masaad MJ, Geha R, Uzel G, Gelman R, Holland SM, Recher M, Utz PJ, Browne SK, Notarangelo LD. Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency [Internet]. Journal of Clinical Investigation. 2015 ; 125( 11): 4135-4148.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1172/JCI80477
Artigo de periodico
Therapeutic applications of TRAIL receptor agonists in cancer and beyond (2015)
Amarante-Mendes, João Gustavo Pessini ; Griffith, Thomas S.
Fonte: Pharmacology & Therapeutics. Unidade: ICB
Assuntos: CITOCINAS, NEOPLASIAS, APOPTOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMARANTE-MENDES, João Gustavo Pessini e GRIFFITH, Thomas S. Therapeutic applications of TRAIL receptor agonists in cancer and beyond. Pharmacology & Therapeutics, v. 155, p. 117-131, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.pharmthera.2015.09.001. Acesso em: 10 jun. 2024.
APA
Amarante-Mendes, J. G. P., & Griffith, T. S. (2015). Therapeutic applications of TRAIL receptor agonists in cancer and beyond. Pharmacology & Therapeutics, 155, 117-131. doi:10.1016/j.pharmthera.2015.09.001
NLM
Amarante-Mendes JGP, Griffith TS. Therapeutic applications of TRAIL receptor agonists in cancer and beyond [Internet]. Pharmacology & Therapeutics. 2015 ; 155 117-131.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.pharmthera.2015.09.001
Vancouver
Amarante-Mendes JGP, Griffith TS. Therapeutic applications of TRAIL receptor agonists in cancer and beyond [Internet]. Pharmacology & Therapeutics. 2015 ; 155 117-131.[citado 2024 jun. 10 ] Available from: https://doi.org/10.1016/j.pharmthera.2015.09.001

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