Filtros : "CAPELOZZI, VERA LUIZA" "2018" Removido: "Indexado no: Current Contents" Limpar

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  • Source: Pathophysiology. Unidades: FM, FMRP, FFCLRP

    Subjects: ESTENOSE DA VALVA AÓRTICA, APOPTOSE, COLÁGENO

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      SANT’ANA, Paula Grippa et al. Heart remodeling produced by aortic stenosis promotes cardiomyocyte apoptosis mediated by collagen V imbalance. Pathophysiology, v. 25, n. 4, p. 373-379, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.pathophys.2018.07.001. Acesso em: 07 jun. 2024.
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      Sant’Ana, P. G., Batah, S. S., Leão, P. dos S., Teodoro, W. R., Souza, S. L. B. de, Ferreira Mota, G. A., et al. (2018). Heart remodeling produced by aortic stenosis promotes cardiomyocyte apoptosis mediated by collagen V imbalance. Pathophysiology, 25( 4), 373-379. doi:10.1016/j.pathophys.2018.07.001
    • NLM

      Sant’Ana PG, Batah SS, Leão P dos S, Teodoro WR, Souza SLB de, Ferreira Mota GA, Vileigas DF, Silva VL da, Campos DHS de, Okoshi K, Capelozzi VL, Cicogna AC, Fabro AT. Heart remodeling produced by aortic stenosis promotes cardiomyocyte apoptosis mediated by collagen V imbalance [Internet]. Pathophysiology. 2018 ; 25( 4): 373-379.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1016/j.pathophys.2018.07.001
    • Vancouver

      Sant’Ana PG, Batah SS, Leão P dos S, Teodoro WR, Souza SLB de, Ferreira Mota GA, Vileigas DF, Silva VL da, Campos DHS de, Okoshi K, Capelozzi VL, Cicogna AC, Fabro AT. Heart remodeling produced by aortic stenosis promotes cardiomyocyte apoptosis mediated by collagen V imbalance [Internet]. Pathophysiology. 2018 ; 25( 4): 373-379.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1016/j.pathophys.2018.07.001
  • Source: Human pathology. Unidade: FM

    Subjects: TUMOR CARCINOIDE, NEOPLASIAS PULMONARES, MORFOMETRIA, ANTÍGENOS, PROTEÍNAS NUCLEARES, GLICOPROTEÍNAS

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      VILHENA, Alyne Fonseca de et al. Histomorphometric evaluation of the Ki-67 proliferation rate and CD34 microvascular and D2-40 lymphovascular densities drives the pulmonary typical carcinoid outcome. Human pathology, v. 81, p. 201-210, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.humpath.2018.07.007. Acesso em: 07 jun. 2024.
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      Vilhena, A. F. de, Pereira, J. C. das N., Parra, E. R., Balancin, M. L., Ab'saber, A., Martins, V., et al. (2018). Histomorphometric evaluation of the Ki-67 proliferation rate and CD34 microvascular and D2-40 lymphovascular densities drives the pulmonary typical carcinoid outcome. Human pathology, 81, 201-210. doi:10.1016/j.humpath.2018.07.007
    • NLM

      Vilhena AF de, Pereira JC das N, Parra ER, Balancin ML, Ab'saber A, Martins V, Farhat C, Abrantes MM, Campos JRM de, Tedde ML. Histomorphometric evaluation of the Ki-67 proliferation rate and CD34 microvascular and D2-40 lymphovascular densities drives the pulmonary typical carcinoid outcome [Internet]. Human pathology. 2018 ; 81 201-210.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1016/j.humpath.2018.07.007
    • Vancouver

      Vilhena AF de, Pereira JC das N, Parra ER, Balancin ML, Ab'saber A, Martins V, Farhat C, Abrantes MM, Campos JRM de, Tedde ML. Histomorphometric evaluation of the Ki-67 proliferation rate and CD34 microvascular and D2-40 lymphovascular densities drives the pulmonary typical carcinoid outcome [Internet]. Human pathology. 2018 ; 81 201-210.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1016/j.humpath.2018.07.007
  • Source: Histology and histopathology. Unidade: FM

    Subjects: TENDINITE, METALOPROTEINASES, COLÁGENO

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      FERNANDES, Tulio Diniz e TEODORO, Walcy Rosolia e CAPELOZZI, Vera Luiza. Matrix metalloproteinase-1 (MMP-1) and (MMP-8) gene polymorphisms promote increase and remodeling of the collagen III and V in posterior tibial tendinopathy. Histology and histopathology, v. 33, n. 9, p. 929-936, 2018Tradução . . Disponível em: https://doi.org/10.14670/HH-11-982. Acesso em: 07 jun. 2024.
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      Fernandes, T. D., Teodoro, W. R., & Capelozzi, V. L. (2018). Matrix metalloproteinase-1 (MMP-1) and (MMP-8) gene polymorphisms promote increase and remodeling of the collagen III and V in posterior tibial tendinopathy. Histology and histopathology, 33( 9), 929-936. doi:10.14670/HH-11-982
    • NLM

      Fernandes TD, Teodoro WR, Capelozzi VL. Matrix metalloproteinase-1 (MMP-1) and (MMP-8) gene polymorphisms promote increase and remodeling of the collagen III and V in posterior tibial tendinopathy [Internet]. Histology and histopathology. 2018 ; 33( 9): 929-936.[citado 2024 jun. 07 ] Available from: https://doi.org/10.14670/HH-11-982
    • Vancouver

      Fernandes TD, Teodoro WR, Capelozzi VL. Matrix metalloproteinase-1 (MMP-1) and (MMP-8) gene polymorphisms promote increase and remodeling of the collagen III and V in posterior tibial tendinopathy [Internet]. Histology and histopathology. 2018 ; 33( 9): 929-936.[citado 2024 jun. 07 ] Available from: https://doi.org/10.14670/HH-11-982
  • Source: European journal of anaesthesiology. Unidade: FM

    Subjects: INFLAMAÇÃO, DOENÇA PULMONAR OBSTRUTIVA CRÔNICA, BIOMARCADORES, INFLAMAÇÃO

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      MAGALHAES, Paulo A. F. e CAPELLOZZI, Vera L. Effects of pressure support ventilation on ventilator-induced lung injury in mild acute respiratory distress syndrome depend on level of positive end-expiratory pressure: A randomised animal study. European journal of anaesthesiology, v. 35, n. 4, p. 298-306, 2018Tradução . . Disponível em: https://doi.org/10.1097/EJA.0000000000000763. Acesso em: 07 jun. 2024.
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      Magalhaes, P. A. F., & Capellozzi, V. L. (2018). Effects of pressure support ventilation on ventilator-induced lung injury in mild acute respiratory distress syndrome depend on level of positive end-expiratory pressure: A randomised animal study. European journal of anaesthesiology, 35( 4), 298-306. doi:10.1097/EJA.0000000000000763
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      Magalhaes PAF, Capellozzi VL. Effects of pressure support ventilation on ventilator-induced lung injury in mild acute respiratory distress syndrome depend on level of positive end-expiratory pressure: A randomised animal study [Internet]. European journal of anaesthesiology. 2018 ; 35( 4): 298-306.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1097/EJA.0000000000000763
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      Magalhaes PAF, Capellozzi VL. Effects of pressure support ventilation on ventilator-induced lung injury in mild acute respiratory distress syndrome depend on level of positive end-expiratory pressure: A randomised animal study [Internet]. European journal of anaesthesiology. 2018 ; 35( 4): 298-306.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1097/EJA.0000000000000763
  • Source: Archives of pathology & laboratory medicine. Unidade: FM

    Subjects: NEOPLASIAS PULMONARES, IMUNOHISTOQUÍMICA

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      THUNNISSEN, Erik e CAPELOZZI, Vera Luiza. Immunohistochemistry of pulmonary biomarkers a perspective from members of the pulmonary pathology society. Archives of pathology & laboratory medicine, v. 142, n. 3, p. 408-419, 2018Tradução . . Disponível em: https://doi.org/10.5858/arpa.2017-0106-SA. Acesso em: 07 jun. 2024.
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      Thunnissen, E., & Capelozzi, V. L. (2018). Immunohistochemistry of pulmonary biomarkers a perspective from members of the pulmonary pathology society. Archives of pathology & laboratory medicine, 142( 3), 408-419. doi:10.5858/arpa.2017-0106-SA
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      Thunnissen E, Capelozzi VL. Immunohistochemistry of pulmonary biomarkers a perspective from members of the pulmonary pathology society [Internet]. Archives of pathology & laboratory medicine. 2018 ; 142( 3): 408-419.[citado 2024 jun. 07 ] Available from: https://doi.org/10.5858/arpa.2017-0106-SA
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      Thunnissen E, Capelozzi VL. Immunohistochemistry of pulmonary biomarkers a perspective from members of the pulmonary pathology society [Internet]. Archives of pathology & laboratory medicine. 2018 ; 142( 3): 408-419.[citado 2024 jun. 07 ] Available from: https://doi.org/10.5858/arpa.2017-0106-SA
  • Source: Archives of pathology & laboratory medicine. Unidade: FM

    Subjects: NEOPLASIAS PULMONARES, TESTES RESPIRATÓRIOS, INFLAMAÇÃO

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      JAIN, Deepali e CAPELOZZI, Vera Luiza. Rapid on-site evaluation of endobronchial ultrasound-guided transbronchial needle aspirations for the diagnosis of lung cancer a perspective from members of the pulmonary pathology society. Archives of pathology & laboratory medicine, v. 142, n. 2, p. 253-262, 2018Tradução . . Disponível em: https://doi.org/10.5858/arpa.2017-0114-SA. Acesso em: 07 jun. 2024.
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      Jain, D., & Capelozzi, V. L. (2018). Rapid on-site evaluation of endobronchial ultrasound-guided transbronchial needle aspirations for the diagnosis of lung cancer a perspective from members of the pulmonary pathology society. Archives of pathology & laboratory medicine, 142( 2), 253-262. doi:10.5858/arpa.2017-0114-SA
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      Jain D, Capelozzi VL. Rapid on-site evaluation of endobronchial ultrasound-guided transbronchial needle aspirations for the diagnosis of lung cancer a perspective from members of the pulmonary pathology society [Internet]. Archives of pathology & laboratory medicine. 2018 ; 142( 2): 253-262.[citado 2024 jun. 07 ] Available from: https://doi.org/10.5858/arpa.2017-0114-SA
    • Vancouver

      Jain D, Capelozzi VL. Rapid on-site evaluation of endobronchial ultrasound-guided transbronchial needle aspirations for the diagnosis of lung cancer a perspective from members of the pulmonary pathology society [Internet]. Archives of pathology & laboratory medicine. 2018 ; 142( 2): 253-262.[citado 2024 jun. 07 ] Available from: https://doi.org/10.5858/arpa.2017-0114-SA
  • Source: Anesthesiology. Unidade: FM

    Subjects: RATOS WISTAR, VENTILAÇÃO, PULMÃO, PNEUMOPATIAS

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      SANTOS, Raquel S. e CAPELOZZI, Vera Luiza. Biologic Impact of Mechanical Power at High and Low Tidal Volumes in Experimental Mild Acute Respiratory Distress Syndrome. Anesthesiology, v. 128, n. 6, p. 1193-1206, 2018Tradução . . Disponível em: https://doi.org/10.1097/ALN.0000000000002143. Acesso em: 07 jun. 2024.
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      Santos, R. S., & Capelozzi, V. L. (2018). Biologic Impact of Mechanical Power at High and Low Tidal Volumes in Experimental Mild Acute Respiratory Distress Syndrome. Anesthesiology, 128( 6), 1193-1206. doi:10.1097/ALN.0000000000002143
    • NLM

      Santos RS, Capelozzi VL. Biologic Impact of Mechanical Power at High and Low Tidal Volumes in Experimental Mild Acute Respiratory Distress Syndrome [Internet]. Anesthesiology. 2018 ; 128( 6): 1193-1206.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1097/ALN.0000000000002143
    • Vancouver

      Santos RS, Capelozzi VL. Biologic Impact of Mechanical Power at High and Low Tidal Volumes in Experimental Mild Acute Respiratory Distress Syndrome [Internet]. Anesthesiology. 2018 ; 128( 6): 1193-1206.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1097/ALN.0000000000002143
  • Source: Homeopathy. Unidade: FM

    Subjects: HOMEOPATIA, NANOPARTÍCULAS, ÁGUA, TRIIODOTIRONINA

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      GUEDES, Jose Roberto Pereira e BONAMIN, Leoni Villano e CAPELOZZI, Vera Luiza. Water-related mechanisms proposed for storing and transmitting homeopathic information: putative links with biological responses. Homeopathy, v. 107, n. 3, p. 172-180, 2018Tradução . . Disponível em: https://doi.org/10.1055/s-0038-1642055. Acesso em: 07 jun. 2024.
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      Guedes, J. R. P., Bonamin, L. V., & Capelozzi, V. L. (2018). Water-related mechanisms proposed for storing and transmitting homeopathic information: putative links with biological responses. Homeopathy, 107( 3), 172-180. doi:10.1055/s-0038-1642055
    • NLM

      Guedes JRP, Bonamin LV, Capelozzi VL. Water-related mechanisms proposed for storing and transmitting homeopathic information: putative links with biological responses [Internet]. Homeopathy. 2018 ; 107( 3): 172-180.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1055/s-0038-1642055
    • Vancouver

      Guedes JRP, Bonamin LV, Capelozzi VL. Water-related mechanisms proposed for storing and transmitting homeopathic information: putative links with biological responses [Internet]. Homeopathy. 2018 ; 107( 3): 172-180.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1055/s-0038-1642055
  • Source: Histology and histopathology. Unidades: FM, FMRP

    Subjects: FIBROSE PULMONAR, COLÁGENO, EXPRESSÃO GÊNICA, EPIDEMIOLOGIA EXPERIMENTAL, ANTIOXIDANTES

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      MARTINS, Vanessa et al. Butylated hydroxytoluene induces type-V collagen and overexpression of remodeling genes/proteins in experimental lung fibrosis. Histology and histopathology, v. 33, n. 10, p. 1111-1123, 2018Tradução . . Disponível em: https://doi.org/10.14670/HH-18-010. Acesso em: 07 jun. 2024.
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      Martins, V., Teodoro, W. R., Velosa, A. P. P., Andrade, P., Farhat, C., Fabro, A. T., & Capelozzi, V. L. (2018). Butylated hydroxytoluene induces type-V collagen and overexpression of remodeling genes/proteins in experimental lung fibrosis. Histology and histopathology, 33( 10), 1111-1123. doi:10.14670/HH-18-010
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      Martins V, Teodoro WR, Velosa APP, Andrade P, Farhat C, Fabro AT, Capelozzi VL. Butylated hydroxytoluene induces type-V collagen and overexpression of remodeling genes/proteins in experimental lung fibrosis [Internet]. Histology and histopathology. 2018 ; 33( 10): 1111-1123.[citado 2024 jun. 07 ] Available from: https://doi.org/10.14670/HH-18-010
    • Vancouver

      Martins V, Teodoro WR, Velosa APP, Andrade P, Farhat C, Fabro AT, Capelozzi VL. Butylated hydroxytoluene induces type-V collagen and overexpression of remodeling genes/proteins in experimental lung fibrosis [Internet]. Histology and histopathology. 2018 ; 33( 10): 1111-1123.[citado 2024 jun. 07 ] Available from: https://doi.org/10.14670/HH-18-010
  • Source: Critical care medicine. Unidade: FM

    Subjects: SÍNDROME DO DESCONFORTO RESPIRATÓRIO EM ADULTOS, PNEUMONIA, CÉLULAS ESTROMAIS, FATORES DE CRESCIMENTO, RATOS WISTAR

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      SILVA, Johnatas D. e CAPELOZZI, Vera Luiza. Mesenchymal Stem Cells From Bone Marrow, Adipose Tissue, and Lung Tissue Differentially Mitigate Lung and Distal Organ Damage in Experimental Acute Respiratory Distress Syndrome. Critical care medicine, v. 46, n. 2, p. E132-E140, 2018Tradução . . Disponível em: https://doi.org/10.1097/CCM.0000000000002833. Acesso em: 07 jun. 2024.
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      Silva, J. D., & Capelozzi, V. L. (2018). Mesenchymal Stem Cells From Bone Marrow, Adipose Tissue, and Lung Tissue Differentially Mitigate Lung and Distal Organ Damage in Experimental Acute Respiratory Distress Syndrome. Critical care medicine, 46( 2), E132-E140. doi:10.1097/CCM.0000000000002833
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      Silva JD, Capelozzi VL. Mesenchymal Stem Cells From Bone Marrow, Adipose Tissue, and Lung Tissue Differentially Mitigate Lung and Distal Organ Damage in Experimental Acute Respiratory Distress Syndrome [Internet]. Critical care medicine. 2018 ; 46( 2): E132-E140.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1097/CCM.0000000000002833
    • Vancouver

      Silva JD, Capelozzi VL. Mesenchymal Stem Cells From Bone Marrow, Adipose Tissue, and Lung Tissue Differentially Mitigate Lung and Distal Organ Damage in Experimental Acute Respiratory Distress Syndrome [Internet]. Critical care medicine. 2018 ; 46( 2): E132-E140.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1097/CCM.0000000000002833
  • Source: Human pathology. Unidade: FM

    Subjects: GRANULOMA, FIBROSE PULMONAR, BIÓPSIA, BIOMARCADORES

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      SOUZA, Paola da Costa e TEODORO, Walcy Rosolia e CAPELOZZI, Vera Luiza. Comprehensive analysis of immune, extracellular matrices and pathogens profile in lung granulomatosis of unexplained etiology. Human pathology, v. 75, p. 104-115, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.humpath.2018.01.018. Acesso em: 07 jun. 2024.
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      Souza, P. da C., Teodoro, W. R., & Capelozzi, V. L. (2018). Comprehensive analysis of immune, extracellular matrices and pathogens profile in lung granulomatosis of unexplained etiology. Human pathology, 75, 104-115. doi:10.1016/j.humpath.2018.01.018
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      Souza P da C, Teodoro WR, Capelozzi VL. Comprehensive analysis of immune, extracellular matrices and pathogens profile in lung granulomatosis of unexplained etiology [Internet]. Human pathology. 2018 ; 75 104-115.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1016/j.humpath.2018.01.018
    • Vancouver

      Souza P da C, Teodoro WR, Capelozzi VL. Comprehensive analysis of immune, extracellular matrices and pathogens profile in lung granulomatosis of unexplained etiology [Internet]. Human pathology. 2018 ; 75 104-115.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1016/j.humpath.2018.01.018
  • Source: PLOS ONE. Unidade: FM

    Subjects: ARTRITE REUMATOIDE, TOLERÂNCIA, ANTÍGENOS, IMUNIZAÇÃO, ARTRITE, PULMÃO

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      ATAYDE, Silvana Ramos et al. Collagen V oral administration decreases inflammation and remodeling of synovial membrane in experimental arthritis. PLOS ONE, v. 13, n. 7, 2018Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0201106. Acesso em: 07 jun. 2024.
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      Atayde, S. R., Catanozi, S., Antonangelo, L., Mello, S. B. V. de, Capelozzi, V. L., & Teodoro, W. R. (2018). Collagen V oral administration decreases inflammation and remodeling of synovial membrane in experimental arthritis. PLOS ONE, 13( 7). doi:10.1371/journal.pone.0201106
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      Atayde SR, Catanozi S, Antonangelo L, Mello SBV de, Capelozzi VL, Teodoro WR. Collagen V oral administration decreases inflammation and remodeling of synovial membrane in experimental arthritis [Internet]. PLOS ONE. 2018 ; 13( 7):[citado 2024 jun. 07 ] Available from: https://doi.org/10.1371/journal.pone.0201106
    • Vancouver

      Atayde SR, Catanozi S, Antonangelo L, Mello SBV de, Capelozzi VL, Teodoro WR. Collagen V oral administration decreases inflammation and remodeling of synovial membrane in experimental arthritis [Internet]. PLOS ONE. 2018 ; 13( 7):[citado 2024 jun. 07 ] Available from: https://doi.org/10.1371/journal.pone.0201106
  • Source: Critical care. Unidade: FM

    Subjects: ACIDENTE VASCULAR CEREBRAL, ISQUEMIA CEREBRAL, MACRÓFAGOS, PNEUMOPATIAS, RATOS

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      SAMARY, Cynthia S. e CAPELOZZI, Vera Luiza. Focal ischemic stroke leads to lung injury and reduces alveolar macrophage phagocytic capability in rats. Critical care, v. 22, 2018Tradução . . Disponível em: https://doi.org/10.1186/s13054-018-2164-0. Acesso em: 07 jun. 2024.
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      Samary, C. S., & Capelozzi, V. L. (2018). Focal ischemic stroke leads to lung injury and reduces alveolar macrophage phagocytic capability in rats. Critical care, 22. doi:10.1186/s13054-018-2164-0
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      Samary CS, Capelozzi VL. Focal ischemic stroke leads to lung injury and reduces alveolar macrophage phagocytic capability in rats [Internet]. Critical care. 2018 ; 22[citado 2024 jun. 07 ] Available from: https://doi.org/10.1186/s13054-018-2164-0
    • Vancouver

      Samary CS, Capelozzi VL. Focal ischemic stroke leads to lung injury and reduces alveolar macrophage phagocytic capability in rats [Internet]. Critical care. 2018 ; 22[citado 2024 jun. 07 ] Available from: https://doi.org/10.1186/s13054-018-2164-0
  • Source: Frontiers in physiology. Unidade: FM

    Subjects: INTERLEUCINA 6, METALOPROTEINASES, SISTEMA RESPIRATÓRIO, EXPRESSÃO GÊNICA

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      MORAES, Lillian e CAPELOZZI, Vera Luiza. Impact of Different Tidal Volume Levels at Low Mechanical Power on Ventilator-Induced Lung Injury in Rats. Frontiers in physiology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fphys.2018.00318. Acesso em: 07 jun. 2024.
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      Moraes, L., & Capelozzi, V. L. (2018). Impact of Different Tidal Volume Levels at Low Mechanical Power on Ventilator-Induced Lung Injury in Rats. Frontiers in physiology, 9. doi:10.3389/fphys.2018.00318
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      Moraes L, Capelozzi VL. Impact of Different Tidal Volume Levels at Low Mechanical Power on Ventilator-Induced Lung Injury in Rats [Internet]. Frontiers in physiology. 2018 ; 9[citado 2024 jun. 07 ] Available from: https://doi.org/10.3389/fphys.2018.00318
    • Vancouver

      Moraes L, Capelozzi VL. Impact of Different Tidal Volume Levels at Low Mechanical Power on Ventilator-Induced Lung Injury in Rats [Internet]. Frontiers in physiology. 2018 ; 9[citado 2024 jun. 07 ] Available from: https://doi.org/10.3389/fphys.2018.00318
  • Source: Frontiers in immunology. Unidade: FM

    Subjects: INFLAMAÇÃO, LEUCÓCITOS, SEPSE, CHOQUE SÉPTICO, ESTADOS UNIDOS

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      GONCALVES-DE-ALBUQUERQUE, Cassiano Felippe e CAPELOZZI, Vera Luiza. The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis. Frontiers in immunology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00901. Acesso em: 07 jun. 2024.
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      Goncalves-de-albuquerque, C. F., & Capelozzi, V. L. (2018). The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis. Frontiers in immunology, 9. doi:10.3389/fimmu.2018.00901
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      Goncalves-de-albuquerque CF, Capelozzi VL. The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis [Internet]. Frontiers in immunology. 2018 ; 9[citado 2024 jun. 07 ] Available from: https://doi.org/10.3389/fimmu.2018.00901
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      Goncalves-de-albuquerque CF, Capelozzi VL. The Yin and Yang of Tyrosine Kinase inhibition During experimental Polymicrobial sepsis [Internet]. Frontiers in immunology. 2018 ; 9[citado 2024 jun. 07 ] Available from: https://doi.org/10.3389/fimmu.2018.00901
  • Source: Brazilian journal of medical and biological research. Unidades: FM, FMRP

    Subjects: BIOMARCADORES, PROTO-ONCOGENES, NEOPLASIAS PULMONARES

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      RANGEL, M.P. et al. Detection of sputum cofilin-1 as indicator of malignancy. Brazilian journal of medical and biological research, v. 51, n. 8, p. e7138, 2018Tradução . . Disponível em: https://doi.org/10.1590/1414-431x20187138. Acesso em: 07 jun. 2024.
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      Rangel, M. P., Antonangelo, L., Capelozzi, V. L., & Fabro, A. T. (2018). Detection of sputum cofilin-1 as indicator of malignancy. Brazilian journal of medical and biological research, 51( 8), e7138. doi:10.1590/1414-431x20187138
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      Rangel MP, Antonangelo L, Capelozzi VL, Fabro AT. Detection of sputum cofilin-1 as indicator of malignancy [Internet]. Brazilian journal of medical and biological research. 2018 ; 51( 8): e7138.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1590/1414-431x20187138
    • Vancouver

      Rangel MP, Antonangelo L, Capelozzi VL, Fabro AT. Detection of sputum cofilin-1 as indicator of malignancy [Internet]. Brazilian journal of medical and biological research. 2018 ; 51( 8): e7138.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1590/1414-431x20187138
  • Source: Critical care medicine. Unidade: FM

    Subjects: PULMÃO, MATRIZ EXTRACELULAR, PROTEOGLICANAS, SÍNDROME DO DESCONFORTO RESPIRATÓRIO EM ADULTOS, EXPRESSÃO GÊNICA

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      SILVA, Pedro Leme e CAPELOZZI, Vera Luiza. Biological response to time-controlled adaptive ventilation depends on acute respiratory distress syndrome etiology. Critical care medicine, v. 46, n. 6, p. E609-E617, 2018Tradução . . Disponível em: https://doi.org/10.1097/CCM.0000000000003078. Acesso em: 07 jun. 2024.
    • APA

      Silva, P. L., & Capelozzi, V. L. (2018). Biological response to time-controlled adaptive ventilation depends on acute respiratory distress syndrome etiology. Critical care medicine, 46( 6), E609-E617. doi:10.1097/CCM.0000000000003078
    • NLM

      Silva PL, Capelozzi VL. Biological response to time-controlled adaptive ventilation depends on acute respiratory distress syndrome etiology [Internet]. Critical care medicine. 2018 ; 46( 6): E609-E617.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1097/CCM.0000000000003078
    • Vancouver

      Silva PL, Capelozzi VL. Biological response to time-controlled adaptive ventilation depends on acute respiratory distress syndrome etiology [Internet]. Critical care medicine. 2018 ; 46( 6): E609-E617.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1097/CCM.0000000000003078
  • Source: Glycoconjugate journal. Unidade: FM

    Subjects: PROTEOGLICANAS, NEOPLASIAS PULMONARES, IMUNOHISTOQUÍMICA

    Acesso à fonteDOIHow to cite
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      RANGEL, Maristela P. et al. Biomolecular analysis of matrix proteoglycans as biomarkers in non small cell lung cancer. Glycoconjugate journal, v. 35, n. 2, p. 233-242, 2018Tradução . . Disponível em: https://doi.org/10.1007/s10719-018-9815-x. Acesso em: 07 jun. 2024.
    • APA

      Rangel, M. P., Sa, V. K. de, Prieto, T., Martins, J. R. M., Olivieri, E. H. R., Carraro, D., et al. (2018). Biomolecular analysis of matrix proteoglycans as biomarkers in non small cell lung cancer. Glycoconjugate journal, 35( 2), 233-242. doi:10.1007/s10719-018-9815-x
    • NLM

      Rangel MP, Sa VK de, Prieto T, Martins JRM, Olivieri EHR, Carraro D, Takagaki T, Capelozzi VL. Biomolecular analysis of matrix proteoglycans as biomarkers in non small cell lung cancer [Internet]. Glycoconjugate journal. 2018 ; 35( 2): 233-242.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1007/s10719-018-9815-x
    • Vancouver

      Rangel MP, Sa VK de, Prieto T, Martins JRM, Olivieri EHR, Carraro D, Takagaki T, Capelozzi VL. Biomolecular analysis of matrix proteoglycans as biomarkers in non small cell lung cancer [Internet]. Glycoconjugate journal. 2018 ; 35( 2): 233-242.[citado 2024 jun. 07 ] Available from: https://doi.org/10.1007/s10719-018-9815-x
  • Source: Archives of pathology & laboratory medicine. Unidade: FM

    Subjects: PNEUMOPATIAS, HIPERSENSIBILIDADE, SISTEMA IMUNE, BIÓPSIA

    Acesso à fonteDOIHow to cite
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      MILLER, Ross e CAPELOZZI, Vera Luiza. Hypersensitivity pneumonitis a perspectiv from Members of the Pulmonary Pathology Society. Archives of pathology & laboratory medicine, v. 142, n. 1, p. 120-126, 2018Tradução . . Disponível em: https://doi.org/10.5858/arpa.2017-0138-SA. Acesso em: 07 jun. 2024.
    • APA

      Miller, R., & Capelozzi, V. L. (2018). Hypersensitivity pneumonitis a perspectiv from Members of the Pulmonary Pathology Society. Archives of pathology & laboratory medicine, 142( 1), 120-126. doi:10.5858/arpa.2017-0138-SA
    • NLM

      Miller R, Capelozzi VL. Hypersensitivity pneumonitis a perspectiv from Members of the Pulmonary Pathology Society [Internet]. Archives of pathology & laboratory medicine. 2018 ; 142( 1): 120-126.[citado 2024 jun. 07 ] Available from: https://doi.org/10.5858/arpa.2017-0138-SA
    • Vancouver

      Miller R, Capelozzi VL. Hypersensitivity pneumonitis a perspectiv from Members of the Pulmonary Pathology Society [Internet]. Archives of pathology & laboratory medicine. 2018 ; 142( 1): 120-126.[citado 2024 jun. 07 ] Available from: https://doi.org/10.5858/arpa.2017-0138-SA

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