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Artigo de periodico
Mass spectrometry-based characterization of protein aggregates in tissues and biofluid (2024)
Silva, Janaina Macedo da ; Fernandes, Livia Rosa ; Blanes, Claudia Angeli ; Palmisano, Giuseppe
Fonte: Advances in Experimental Medicine and Biology. Unidade: ICB
Assuntos: PARASITOLOGIA, COMPLEXOS MULTIENZIMÁTICOS, MICRORNAS, PROTEÍNAS SANGUÍNEAS, ESPECTROMETRIA DE MASSAS, MICROSCOPIA ELETRÔNICA ANIMAL, PLASMA, PLASMA DE ANIMAL, WESTERN BLOTTING, DOENÇAS NEURODEGENERATIVAS, CAMUNDONGOS
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SILVA, Janaina Macedo da et al. Mass spectrometry-based characterization of protein aggregates in tissues and biofluid. Advances in Experimental Medicine and Biology, v. 1443, p. 257-267, 2024Tradução . . Disponível em: https://doi.org/10.1007/978-3-031-50624-6_14. Acesso em: 16 ago. 2024.
APA
Silva, J. M. da, Fernandes, L. R., Blanes, C. A., & Palmisano, G. (2024). Mass spectrometry-based characterization of protein aggregates in tissues and biofluid. Advances in Experimental Medicine and Biology, 1443, 257-267. doi:10.1007/978-3-031-50624-6_14
NLM
Silva JM da, Fernandes LR, Blanes CA, Palmisano G. Mass spectrometry-based characterization of protein aggregates in tissues and biofluid [Internet]. Advances in Experimental Medicine and Biology. 2024 ; 1443 257-267.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1007/978-3-031-50624-6_14.
Vancouver
Silva JM da, Fernandes LR, Blanes CA, Palmisano G. Mass spectrometry-based characterization of protein aggregates in tissues and biofluid [Internet]. Advances in Experimental Medicine and Biology. 2024 ; 1443 257-267.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1007/978-3-031-50624-6_14.
Artigo de periodico
MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection (2024)
Kaminski, Valéria de Lima ; Borges, Bruno Montanari ; Santos, Bianca Vieira dos; Preite, Nycolas Willian ; Loures, Flavio Vieira ; Calich, Vera Lúcia Garcia
Fonte: Frontiers in Cellular and Infection Microbiology. Unidade: ICB
Assuntos: IMUNOLOGIA, PARACOCCIDIOIDES, PARACOCCIDIOIDOMICOSE, DERMATOPATIA PARASITÁRIA ANIMAL, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, ANTÍGENOS DE FUNGOS, FÁRMACOS IMUNOSSUPRESSORES, INTERLEUCINA 10, TRANSDUÇÃO DE SINAL CELULAR, LINFÓCITOS T, PULMÃO, LECTINAS, IMUNOLOGIA CELULAR
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KAMINSKI, Valéria de Lima et al. MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection. Frontiers in Cellular and Infection Microbiology, v. 14, p. 20 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2024.1392744. Acesso em: 16 ago. 2024.
APA
Kaminski, V. de L., Borges, B. M., Santos, B. V. dos, Preite, N. W., Loures, F. V., & Calich, V. L. G. (2024). MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection. Frontiers in Cellular and Infection Microbiology, 14, 20 . doi:10.3389/fcimb.2024.1392744
NLM
Kaminski V de L, Borges BM, Santos BV dos, Preite NW, Loures FV, Calich VLG. MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Cellular and Infection Microbiology. 2024 ; 14 20 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fcimb.2024.1392744
Vancouver
Kaminski V de L, Borges BM, Santos BV dos, Preite NW, Loures FV, Calich VLG. MDSCs use a complex molecular network to suppress T-cell immunity in a pulmonary model of fungal infection [Internet]. Frontiers in Cellular and Infection Microbiology. 2024 ; 14 20 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fcimb.2024.1392744
Artigo de periodico
Formoterol reduces muscle wasting in mice undergoing doxorubicin chemotherapy (2024)
Lima Junior, Edson Alves de ; Teixeira, Alexandre Abilio de Souza ; Silveira, Loreana Sanches ; Jové, Queralt; Alvarez Ladron, Natalia; Pereira, Marcelo G; Lopez-Soriano, Francisco Javier; Argiles, Josep M; Brum, Patrícia Chakur ; Busquets, Silvia; Rosa Neto, Jose Cesar
Fonte: Frontiers in Oncology. Unidades: EEFE, ICB
Assuntos: BIOLOGIA CELULAR, CAMUNDONGOS, QUIMIOTERÁPICOS, CAQUEXIA
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LIMA JUNIOR, Edson Alves de et al. Formoterol reduces muscle wasting in mice undergoing doxorubicin chemotherapy. Frontiers in Oncology, v. 13, n. ja 2024, 2024Tradução . . Disponível em: https://doi.org/10.3389/fonc.2023.1237709. Acesso em: 16 ago. 2024.
APA
Lima Junior, E. A. de, Teixeira, A. A. de S., Silveira, L. S., Jové, Q., Alvarez Ladron, N., Pereira, M. G., et al. (2024). Formoterol reduces muscle wasting in mice undergoing doxorubicin chemotherapy. Frontiers in Oncology, 13( ja 2024). doi:10.3389/fonc.2023.1237709
NLM
Lima Junior EA de, Teixeira AA de S, Silveira LS, Jové Q, Alvarez Ladron N, Pereira MG, Lopez-Soriano FJ, Argiles JM, Brum PC, Busquets S, Rosa Neto JC. Formoterol reduces muscle wasting in mice undergoing doxorubicin chemotherapy [Internet]. Frontiers in Oncology. 2024 ; 13( ja 2024):[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fonc.2023.1237709
Vancouver
Lima Junior EA de, Teixeira AA de S, Silveira LS, Jové Q, Alvarez Ladron N, Pereira MG, Lopez-Soriano FJ, Argiles JM, Brum PC, Busquets S, Rosa Neto JC. Formoterol reduces muscle wasting in mice undergoing doxorubicin chemotherapy [Internet]. Frontiers in Oncology. 2024 ; 13( ja 2024):[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fonc.2023.1237709
Artigo de periodico
Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome (2024)
Cesaro, Aline Cristina Parletta ; Cerri, Gabriela Cavazza ; Gasparini, Claudia Ribeiro Borba; Panico, Karine ; Vieira Júnior, Denival Nascimento ; Rodrigues, Larissa Maria Zacarias ; Senger, Nathalia ; Silva, Amanda de Almeida; Fevereiro, Marina Reingruber ; Diniz, Gabriela Placoná ; Irigoyen, Maria Cláudia Costa ; Chaves, Maria Luiza Morais Barreto de
Fonte: Pflügers Archiv - European Journal of Physiology. Unidades: ICB, FM
Assuntos: ANATOMIA, HIPERTROFIA VENTRICULAR ESQUERDA, INSUFICIÊNCIA CARDÍACA ANIMAL, CARDIOMEGALIA ANIMAL, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, FREQUÊNCIA CARDÍACA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS
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CESARO, Aline Cristina Parletta et al. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome. Pflügers Archiv - European Journal of Physiology, v. 476, p. 1065-1075, 2024Tradução . . Disponível em: https://doi.org/10.1007/s00424-024-02965-6. Acesso em: 16 ago. 2024.
APA
Cesaro, A. C. P., Cerri, G. C., Gasparini, C. R. B., Panico, K., Vieira Júnior, D. N., Rodrigues, L. M. Z., et al. (2024). Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome. Pflügers Archiv - European Journal of Physiology, 476, 1065-1075. doi:10.1007/s00424-024-02965-6
NLM
Cesaro ACP, Cerri GC, Gasparini CRB, Panico K, Vieira Júnior DN, Rodrigues LMZ, Senger N, Silva A de A, Fevereiro MR, Diniz GP, Irigoyen MCC, Chaves MLMB de. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome [Internet]. Pflügers Archiv - European Journal of Physiology. 2024 ; 476 1065-1075.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1007/s00424-024-02965-6
Vancouver
Cesaro ACP, Cerri GC, Gasparini CRB, Panico K, Vieira Júnior DN, Rodrigues LMZ, Senger N, Silva A de A, Fevereiro MR, Diniz GP, Irigoyen MCC, Chaves MLMB de. Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome [Internet]. Pflügers Archiv - European Journal of Physiology. 2024 ; 476 1065-1075.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1007/s00424-024-02965-6
Artigo de periodico
Lisinopril increases lung ACE2 levels and SARS-CoV-2 viral load and decreases inflammation but not disease severity in experimental COVID-19 (2024)
Santos, Yasmin Silva ; Gamon, Thaís Helena Martins ; Azevedo, Marcela Santiago Pacheco de ; Bielavsky, Mônica ; Darido, Maria Laura Goussain ; Oliveira, Danielle Bruna Leal de ; Souza, Edmarcia Elisa de ; Luvizotto, Maria Cecília Rui ; Ackerman, Hans Christian; Carvalho, Leonardo José Moura; Epiphanio, Sabrina ; Wrenger, Carsten ; Durigon, Edison Luiz ; Marinho, Claudio Romero Farias
Fonte: Frontiers in Pharmacology. Unidades: FCF, ICB, BIOTECNOLOGIA
Assuntos: PARASITOLOGIA, MICROBIOLOGIA, COVID-19, INIBIDORES DE ENZIMAS, DOENÇAS RESPIRATÓRIAS, ANGIOTENSINA II, SISTEMA RENINA-ANGIOTENSINA, ANTIVIRAIS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS
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SANTOS, Yasmin Silva et al. Lisinopril increases lung ACE2 levels and SARS-CoV-2 viral load and decreases inflammation but not disease severity in experimental COVID-19. Frontiers in Pharmacology, v. 15, 2024Tradução . . Disponível em: https://doi.org/10.3389/fphar.2024.1414406. Acesso em: 16 ago. 2024.
APA
Santos, Y. S., Gamon, T. H. M., Azevedo, M. S. P. de, Bielavsky, M., Darido, M. L. G., Oliveira, D. B. L. de, et al. (2024). Lisinopril increases lung ACE2 levels and SARS-CoV-2 viral load and decreases inflammation but not disease severity in experimental COVID-19. Frontiers in Pharmacology, 15. doi:10.3389/fphar.2024.1414406
NLM
Santos YS, Gamon THM, Azevedo MSP de, Bielavsky M, Darido MLG, Oliveira DBL de, Souza EE de, Luvizotto MCR, Ackerman HC, Carvalho LJM, Epiphanio S, Wrenger C, Durigon EL, Marinho CRF. Lisinopril increases lung ACE2 levels and SARS-CoV-2 viral load and decreases inflammation but not disease severity in experimental COVID-19 [Internet]. Frontiers in Pharmacology. 2024 ; 15[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fphar.2024.1414406
Vancouver
Santos YS, Gamon THM, Azevedo MSP de, Bielavsky M, Darido MLG, Oliveira DBL de, Souza EE de, Luvizotto MCR, Ackerman HC, Carvalho LJM, Epiphanio S, Wrenger C, Durigon EL, Marinho CRF. Lisinopril increases lung ACE2 levels and SARS-CoV-2 viral load and decreases inflammation but not disease severity in experimental COVID-19 [Internet]. Frontiers in Pharmacology. 2024 ; 15[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fphar.2024.1414406
Artigo de periodico
A subiculum-hypothalamic pathway functions in dynamic threat detection and memory updating (2024)
Viellard, Juliette Marguerite Alix ; Melleu, Fernando Falkenburger ; Tamais, Alicia Moraes ; Almeida, Alisson Pinto de ; Zerbini, Carolina ; Ikebara, Juliane Midori ; Domingues, Karolina; Lima, Miguel Antonio Xavier de ; Oliveira, Fernando Augusto de ; Motta, Simone Cristina ; Canteras, Newton Sabino
Fonte: Current Biology. Unidade: ICB
Assuntos: ANATOMIA, COMPORTAMENTO ANIMAL, MEMÓRIA ANIMAL, HIPOTÁLAMO, SUBSTÂNCIA CINZENTA PERIAQUEDUTAL, HIPOCAMPU DE ANIMAL, VIAS NEURAIS, CIRCUITOS ELÉTRICOS, RECEPTORES SENSORIAIS, MEDO, CAMUNDONGOS
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VIELLARD, Juliette Marguerite Alix et al. A subiculum-hypothalamic pathway functions in dynamic threat detection and memory updating. Current Biology, v. 34, n. 12, p. 2657-2671, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.cub.2024.05.006. Acesso em: 16 ago. 2024.
APA
Viellard, J. M. A., Melleu, F. F., Tamais, A. M., Almeida, A. P. de, Zerbini, C., Ikebara, J. M., et al. (2024). A subiculum-hypothalamic pathway functions in dynamic threat detection and memory updating. Current Biology, 34( 12), 2657-2671. doi:10.1016/j.cub.2024.05.006
NLM
Viellard JMA, Melleu FF, Tamais AM, Almeida AP de, Zerbini C, Ikebara JM, Domingues K, Lima MAX de, Oliveira FA de, Motta SC, Canteras NS. A subiculum-hypothalamic pathway functions in dynamic threat detection and memory updating [Internet]. Current Biology. 2024 ; 34( 12): 2657-2671.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.cub.2024.05.006
Vancouver
Viellard JMA, Melleu FF, Tamais AM, Almeida AP de, Zerbini C, Ikebara JM, Domingues K, Lima MAX de, Oliveira FA de, Motta SC, Canteras NS. A subiculum-hypothalamic pathway functions in dynamic threat detection and memory updating [Internet]. Current Biology. 2024 ; 34( 12): 2657-2671.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.cub.2024.05.006
Artigo de periodico
Sex-dependent changes in AMPAR expression and Na, K-ATPase activity in the cerebellum and hippocampus of α-Klotho-Hypomorphic mice (2024)
Santos, Geovane Ribeiro Dos ; Cararo-Lopes, Marina Lopes ; Possebon, Isabela Ribeiro ; Lima, Larissa de Sá ; Scavone, Cristoforo ; Kawamoto, Elisa Mitiko
Fonte: Neuropharmacology. Unidade: ICB
Assuntos: FARMACOLOGIA, CAMUNDONGOS, ENVELHECIMENTO, PROTEÍNAS, HIPOCAMPU DE ANIMAL, CEREBELO, COMPORTAMENTO SEXUAL ANIMAL, PROCESSOS COGNITIVOS
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SANTOS, Geovane Ribeiro Dos et al. Sex-dependent changes in AMPAR expression and Na, K-ATPase activity in the cerebellum and hippocampus of α-Klotho-Hypomorphic mice. Neuropharmacology, v. 258, p. 11 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2024.110097. Acesso em: 16 ago. 2024.
APA
Santos, G. R. D., Cararo-Lopes, M. L., Possebon, I. R., Lima, L. de S., Scavone, C., & Kawamoto, E. M. (2024). Sex-dependent changes in AMPAR expression and Na, K-ATPase activity in the cerebellum and hippocampus of α-Klotho-Hypomorphic mice. Neuropharmacology, 258, 11 . doi:10.1016/j.neuropharm.2024.110097
NLM
Santos GRD, Cararo-Lopes ML, Possebon IR, Lima L de S, Scavone C, Kawamoto EM. Sex-dependent changes in AMPAR expression and Na, K-ATPase activity in the cerebellum and hippocampus of α-Klotho-Hypomorphic mice [Internet]. Neuropharmacology. 2024 ; 258 11 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.neuropharm.2024.110097
Vancouver
Santos GRD, Cararo-Lopes ML, Possebon IR, Lima L de S, Scavone C, Kawamoto EM. Sex-dependent changes in AMPAR expression and Na, K-ATPase activity in the cerebellum and hippocampus of α-Klotho-Hypomorphic mice [Internet]. Neuropharmacology. 2024 ; 258 11 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.neuropharm.2024.110097
Artigo de periodico
Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice (2024)
Franco, Thaina Maquedo ; Tavares, Mariana Rosolen ; Novaes, Leonardo Santana ; Peixoto-Santos, Jose Eduardo ; Araujo, Ronaldo Carvalho ; Bader, Michael ; Wasinski, Frederick ; Munhoz, Carolina Demarchi ; Donato Junior, José
Fonte: International Journal of Molecular Sciences. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA, BRADICININA, INFLAMAÇÃO, SISTEMA NERVOSO CENTRAL, PRESSÃO SANGUÍNEA, PROTEÍNAS G, RECEPTORES DE CATECOLAMINA, RECEPTORES DE DOPAMINA, CAMUNDONGOS, PESOS E MEDIDAS CORPORAIS, AMINOÁCIDOS
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FRANCO, Thaina Maquedo et al. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice. International Journal of Molecular Sciences, v. 25, n. 3, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.3390/ijms25031490. Acesso em: 16 ago. 2024.
APA
Franco, T. M., Tavares, M. R., Novaes, L. S., Peixoto-Santos, J. E., Araujo, R. C., Bader, M., et al. (2024). Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice. International Journal of Molecular Sciences, 25( 3), 14 . doi:10.3390/ijms25031490
NLM
Franco TM, Tavares MR, Novaes LS, Peixoto-Santos JE, Araujo RC, Bader M, Wasinski F, Munhoz CD, Donato Junior J. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 3): 14 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3390/ijms25031490
Vancouver
Franco TM, Tavares MR, Novaes LS, Peixoto-Santos JE, Araujo RC, Bader M, Wasinski F, Munhoz CD, Donato Junior J. Effects of bradykinin B2 receptor ablation from tyrosine hydroxylase cells on behavioral and motor aspects in male and female mice [Internet]. International Journal of Molecular Sciences. 2024 ; 25( 3): 14 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3390/ijms25031490
Artigo de periodico
FVB/NJ strain as a mouse model for cutaneous leishmaniasis by Leishmania (L.) amazonensis (2024)
Carrara, Guilherme Moreira Paiva ; Stolf, Beatriz Simonsen
Fonte: Memórias do Instituto Oswaldo Cruz. Unidade: ICB
Assuntos: PARASITOLOGIA, LEISHMANIA, INFECÇÕES POR PROTOZOÁRIOS, LEISHMANIOSE VISCERAL ANIMAL, LEISHMANIOSES ANIMAL, CAMUNDONGOS, MACRÓFAGOS
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CARRARA, Guilherme Moreira Paiva e STOLF, Beatriz Simonsen. FVB/NJ strain as a mouse model for cutaneous leishmaniasis by Leishmania (L.) amazonensis. Memórias do Instituto Oswaldo Cruz, v. 119, p. 7 , 2024Tradução . . Disponível em: https://doi.org/10.1590/0074-02760230182. Acesso em: 16 ago. 2024.
APA
Carrara, G. M. P., & Stolf, B. S. (2024). FVB/NJ strain as a mouse model for cutaneous leishmaniasis by Leishmania (L.) amazonensis. Memórias do Instituto Oswaldo Cruz, 119, 7 . doi:10.1590/0074-02760230182
NLM
Carrara GMP, Stolf BS. FVB/NJ strain as a mouse model for cutaneous leishmaniasis by Leishmania (L.) amazonensis [Internet]. Memórias do Instituto Oswaldo Cruz. 2024 ; 119 7 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1590/0074-02760230182
Vancouver
Carrara GMP, Stolf BS. FVB/NJ strain as a mouse model for cutaneous leishmaniasis by Leishmania (L.) amazonensis [Internet]. Memórias do Instituto Oswaldo Cruz. 2024 ; 119 7 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1590/0074-02760230182
Artigo de periodico
Zika virus infection impairs synaptogenesis, induces neuroinflammation, and could be an environmental risk factor for autism spectrum disorder outcome (2024)
Ohki, Cristine Marie Yde ; Benazzato, Cecília ; Linden, Vanessa van der
Fonte: Biochimica et Biophysica Acta. Molecular Basis of Disease. Unidade: ICB
Assuntos: MICROBIOLOGIA, ZIKA VÍRUS, INFECÇÕES POR VÍRUS DE RNA, ASTRÓCITOS, TRANSTORNO DO ESPECTRO AUTISTA, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, ESTUDOS DE COORTES, CRIANÇAS, APOPTOSE, GLUTAMATOS, MICROCEFALIA
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OHKI, Cristine Marie Yde e BENAZZATO, Cecília e LINDEN, Vanessa van der. Zika virus infection impairs synaptogenesis, induces neuroinflammation, and could be an environmental risk factor for autism spectrum disorder outcome. Biochimica et Biophysica Acta. Molecular Basis of Disease, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2024.167097. Acesso em: 16 ago. 2024.
APA
Ohki, C. M. Y., Benazzato, C., & Linden, V. van der. (2024). Zika virus infection impairs synaptogenesis, induces neuroinflammation, and could be an environmental risk factor for autism spectrum disorder outcome. Biochimica et Biophysica Acta. Molecular Basis of Disease. doi:10.1016/j.bbadis.2024.167097
NLM
Ohki CMY, Benazzato C, Linden V van der. Zika virus infection impairs synaptogenesis, induces neuroinflammation, and could be an environmental risk factor for autism spectrum disorder outcome [Internet]. Biochimica et Biophysica Acta. Molecular Basis of Disease. 2024 ;[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.bbadis.2024.167097
Vancouver
Ohki CMY, Benazzato C, Linden V van der. Zika virus infection impairs synaptogenesis, induces neuroinflammation, and could be an environmental risk factor for autism spectrum disorder outcome [Internet]. Biochimica et Biophysica Acta. Molecular Basis of Disease. 2024 ;[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.bbadis.2024.167097
Artigo de periodico
Effects of low-dose rapamycin on lymphoid organs of mice prone and resistant to accelerated senescence (2024)
Barros, Rafael dos Santos ; Queiroz, Luiz Adriano Damasceno de ; Pantoja, Kamilla da Costa ; Bustia, Sofia Xavier ; Sousa, Emanuella Sarmento Alho de ; Assis, Josiane Betim de ; Rodrigues, Stephen Fernandes de Paula ; Akamine, Eliana Hiromi ; Nunes, Anderson de Sá ; Martins, Joilson de Oliveira
Fonte: Frontiers in Immunology. Unidades: ICB, FCF
Assuntos: IMUNOLOGIA, FARMACOLOGIA, LINFÓCITOS T, ENVELHECIMENTO, CITOCINAS, MEDULA ÓSSEA DE ANIMAL, TIMO (GLÂNDULA ENDÓCRINA), FÁRMACOS IMUNOSSUPRESSORES, CAMUNDONGOS
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BARROS, Rafael dos Santos et al. Effects of low-dose rapamycin on lymphoid organs of mice prone and resistant to accelerated senescence. Frontiers in Immunology, v. 15, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2024.1310505. Acesso em: 16 ago. 2024.
APA
Barros, R. dos S., Queiroz, L. A. D. de, Pantoja, K. da C., Bustia, S. X., Sousa, E. S. A. de, Assis, J. B. de, et al. (2024). Effects of low-dose rapamycin on lymphoid organs of mice prone and resistant to accelerated senescence. Frontiers in Immunology, 15, 14 . doi:10.3389/fimmu.2024.1310505
NLM
Barros R dos S, Queiroz LAD de, Pantoja K da C, Bustia SX, Sousa ESA de, Assis JB de, Rodrigues SF de P, Akamine EH, Nunes A de S, Martins J de O. Effects of low-dose rapamycin on lymphoid organs of mice prone and resistant to accelerated senescence [Internet]. Frontiers in Immunology. 2024 ; 15 14 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fimmu.2024.1310505
Vancouver
Barros R dos S, Queiroz LAD de, Pantoja K da C, Bustia SX, Sousa ESA de, Assis JB de, Rodrigues SF de P, Akamine EH, Nunes A de S, Martins J de O. Effects of low-dose rapamycin on lymphoid organs of mice prone and resistant to accelerated senescence [Internet]. Frontiers in Immunology. 2024 ; 15 14 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fimmu.2024.1310505
Artigo de periodico
RANKL signaling drives skeletal muscle into the oxidative profile (2024)
Araujo, Paulo Henrique Cavalcanti de ; Cezine, Maria Eduarda Ramos ; Vulczak, Anderson ; Vieira, Luiz Carlos; Matsuo, Flávia Sayuri ; Remoto, Júlia Maranghetti; Santos, Audrei dos Reis ; Alberici, Luciane Carla ; Osako, Mariana Kiomy ; Miyabara, Elen Haruka
Fonte: Journal of Bone and Mineral Research. Unidades: FCFRP, ICB, FMRP
Assuntos: ANATOMIA, MITOCÔNDRIAS, FIBRAS MUSCULARES, MÚSCULO ESQUELÉTICO, ORGANELAS CELULARES, TRANSDUÇÃO DE SINAL CELULAR, WESTERN BLOTTING, CAMUNDONGOS, RESPIRAÇÃO ANIMAL
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ARAUJO, Paulo Henrique Cavalcanti de et al. RANKL signaling drives skeletal muscle into the oxidative profile. Journal of Bone and Mineral Research, v. 39, n. 6, p. 753-764, 2024Tradução . . Disponível em: https://doi.org/10.1093/jbmr/zjae058. Acesso em: 16 ago. 2024.
APA
Araujo, P. H. C. de, Cezine, M. E. R., Vulczak, A., Vieira, L. C., Matsuo, F. S., Remoto, J. M., et al. (2024). RANKL signaling drives skeletal muscle into the oxidative profile. Journal of Bone and Mineral Research, 39( 6), 753-764. doi:10.1093/jbmr/zjae058
NLM
Araujo PHC de, Cezine MER, Vulczak A, Vieira LC, Matsuo FS, Remoto JM, Santos A dos R, Alberici LC, Osako MK, Miyabara EH. RANKL signaling drives skeletal muscle into the oxidative profile [Internet]. Journal of Bone and Mineral Research. 2024 ; 39( 6): 753-764.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1093/jbmr/zjae058
Vancouver
Araujo PHC de, Cezine MER, Vulczak A, Vieira LC, Matsuo FS, Remoto JM, Santos A dos R, Alberici LC, Osako MK, Miyabara EH. RANKL signaling drives skeletal muscle into the oxidative profile [Internet]. Journal of Bone and Mineral Research. 2024 ; 39( 6): 753-764.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1093/jbmr/zjae058
Artigo de periodico
Sex-specific regulation of miR-22 and ERα in white adipose tissue of obese dam's female offspring impairs the early postnatal development of functional beige adipocytes in mice (2024)
Sousa, Érica de; Bolin, Anaysa Paola ; Oliveira, Nayara Preste de; Real, Caroline Cristiano ; Hu, Xiaoyun ; Huang, Zhan-Peng ; Wang, Da Zhi ; Rodrigues, Alice Cristina
Fonte: Biochimica et Biophysica Acta. Molecular Basis of Disease. Unidades: ICB, FCF
Assuntos: FARMACOLOGIA, ONTOGENIA, ÁCIDOS GRAXOS, OXIDAÇÃO, OBESIDADE, ADIPOSIDADE, TECIDO ADIPOSO, ESTRÓGENOS, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUSA, Érica de et al. Sex-specific regulation of miR-22 and ERα in white adipose tissue of obese dam's female offspring impairs the early postnatal development of functional beige adipocytes in mice. Biochimica et Biophysica Acta. Molecular Basis of Disease, v. 1870, n. 4, p. 15 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2024.167057. Acesso em: 16 ago. 2024.
APA
Sousa, É. de, Bolin, A. P., Oliveira, N. P. de, Real, C. C., Hu, X., Huang, Z. -P., et al. (2024). Sex-specific regulation of miR-22 and ERα in white adipose tissue of obese dam's female offspring impairs the early postnatal development of functional beige adipocytes in mice. Biochimica et Biophysica Acta. Molecular Basis of Disease, 1870( 4), 15 . doi:10.1016/j.bbadis.2024.167057
NLM
Sousa É de, Bolin AP, Oliveira NP de, Real CC, Hu X, Huang Z-P, Wang DZ, Rodrigues AC. Sex-specific regulation of miR-22 and ERα in white adipose tissue of obese dam's female offspring impairs the early postnatal development of functional beige adipocytes in mice [Internet]. Biochimica et Biophysica Acta. Molecular Basis of Disease. 2024 ; 1870( 4): 15 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.bbadis.2024.167057
Vancouver
Sousa É de, Bolin AP, Oliveira NP de, Real CC, Hu X, Huang Z-P, Wang DZ, Rodrigues AC. Sex-specific regulation of miR-22 and ERα in white adipose tissue of obese dam's female offspring impairs the early postnatal development of functional beige adipocytes in mice [Internet]. Biochimica et Biophysica Acta. Molecular Basis of Disease. 2024 ; 1870( 4): 15 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.bbadis.2024.167057
Artigo de periodico
Lactobacillus acidophilus LA-5 ameliorates inflammation and alveolar bone loss promoted by A. Actinomycetemcomitans and S. gordonii in mice and impacts oral and gut microbiomes (2024)
Bueno, Manuela Rocha ; Martins, Fernando Henrique ; Rocha, Catarina Medeiros ; Kawamoto, Dione ; Ishikawa, Karin Hitomi ; Ando-Suguimoto, Ellen Sayuri; Carlucci, Aline Ramos ; Arroteia, Leticia S.; Casarin, Renato V. ; Mayer, Marcia Pinto Alves
Fonte: Microorganisms. Unidades: ICB, FO
Assuntos: MICROBIOLOGIA, GENÉTICA ODONTOLÓGICA, LACTOBACILLUS, ACTINOBACILLUS ACTINOMYCETEMCOMITANS, BACTÉRIAS GRAM-NEGATIVAS, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS, INFLAMAÇÃO, CAVIDADE DENTÁRIA, OSSO E OSSOS, PERIODONTITE, PROBIÓTICOS, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BUENO, Manuela Rocha et al. Lactobacillus acidophilus LA-5 ameliorates inflammation and alveolar bone loss promoted by A. Actinomycetemcomitans and S. gordonii in mice and impacts oral and gut microbiomes. Microorganisms, v. 12, n. 4, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.3390/microorganisms12040836. Acesso em: 16 ago. 2024.
APA
Bueno, M. R., Martins, F. H., Rocha, C. M., Kawamoto, D., Ishikawa, K. H., Ando-Suguimoto, E. S., et al. (2024). Lactobacillus acidophilus LA-5 ameliorates inflammation and alveolar bone loss promoted by A. Actinomycetemcomitans and S. gordonii in mice and impacts oral and gut microbiomes. Microorganisms, 12( 4), 14 . doi:10.3390/microorganisms12040836
NLM
Bueno MR, Martins FH, Rocha CM, Kawamoto D, Ishikawa KH, Ando-Suguimoto ES, Carlucci AR, Arroteia LS, Casarin RV, Mayer MPA. Lactobacillus acidophilus LA-5 ameliorates inflammation and alveolar bone loss promoted by A. Actinomycetemcomitans and S. gordonii in mice and impacts oral and gut microbiomes [Internet]. Microorganisms. 2024 ; 12( 4): 14 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3390/microorganisms12040836
Vancouver
Bueno MR, Martins FH, Rocha CM, Kawamoto D, Ishikawa KH, Ando-Suguimoto ES, Carlucci AR, Arroteia LS, Casarin RV, Mayer MPA. Lactobacillus acidophilus LA-5 ameliorates inflammation and alveolar bone loss promoted by A. Actinomycetemcomitans and S. gordonii in mice and impacts oral and gut microbiomes [Internet]. Microorganisms. 2024 ; 12( 4): 14 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3390/microorganisms12040836
Artigo de periodico
Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model (2024)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Fonte: Experimental Neurology. Unidades: ICB, IQ
Assuntos: FISIOLOGIA, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON, MODELOS ANIMAIS DE DOENÇAS, CÉLULAS MORTAS, ASTRÓCITOS, NEURÓGLIA, CAMUNDONGOS, RATOS, TÉCNICAS ESTEREOTÁXICAS, IMUNOHISTOQUÍMICA, REAÇÃO EM CADEIA POR POLIMERASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Ana Flávia Fernandes et al. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, v. 377, p. 15 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2024.114780. Acesso em: 16 ago. 2024.
APA
Ferreira, A. F. F., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2024). Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, 377, 15 . doi:10.1016/j.expneurol.2024.114780
NLM
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Vancouver
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Artigo de periodico
TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease (2024)
Cabral, Laís M. ; Oliveira, Luis M. ; Miranda, Nicole C.; Kawamoto, Elisa Mitiko ; Costa, Soraia Katia Pereira ; Moreira, Thiago S. ; Takakura, Ana Carolina
Fonte: Brain Research. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, INFLAMAÇÃO, CITOCINAS, NECROSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CABRAL, Laís M. et al. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease. Brain Research, v. 1822, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2023.148586. Acesso em: 16 ago. 2024.
APA
Cabral, L. M., Oliveira, L. M., Miranda, N. C., Kawamoto, E. M., Costa, S. K. P., Moreira, T. S., & Takakura, A. C. (2024). TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease. Brain Research, 1822. doi:10.1016/j.brainres.2023.148586
NLM
Cabral LM, Oliveira LM, Miranda NC, Kawamoto EM, Costa SKP, Moreira TS, Takakura AC. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease [Internet]. Brain Research. 2024 ; 1822[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.brainres.2023.148586
Vancouver
Cabral LM, Oliveira LM, Miranda NC, Kawamoto EM, Costa SKP, Moreira TS, Takakura AC. TNFR1-mediated neuroinflammation is necessary for respiratory deficits observed in 6-hydroxydopamine mouse model of Parkinsońs Disease [Internet]. Brain Research. 2024 ; 1822[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.brainres.2023.148586
Artigo de periodico
Ironing out obesity (2024)
Souza, Gabriel Orefice de ; Santos, Willian Oliveira dos ; Donato Junior, José
Fonte: Trends in Endocrinology and Metabolism. Unidade: ICB
Assuntos: FISIOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, OBESIDADE, RESISTÊNCIA À INSULINA, LEPTINA, HORMÔNIOS DO TECIDO ADIPOSO, FERRO
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ABNT
SOUZA, Gabriel Orefice de e SANTOS, Willian Oliveira dos e DONATO JUNIOR, José. Ironing out obesity. Trends in Endocrinology and Metabolism, v. 35, n. 6, p. 456-458, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.tem.2024.04.001. Acesso em: 16 ago. 2024.
APA
Souza, G. O. de, Santos, W. O. dos, & Donato Junior, J. (2024). Ironing out obesity. Trends in Endocrinology and Metabolism, 35( 6), 456-458. doi:10.1016/j.tem.2024.04.001
NLM
Souza GO de, Santos WO dos, Donato Junior J. Ironing out obesity [Internet]. Trends in Endocrinology and Metabolism. 2024 ; 35( 6): 456-458.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.tem.2024.04.001
Vancouver
Souza GO de, Santos WO dos, Donato Junior J. Ironing out obesity [Internet]. Trends in Endocrinology and Metabolism. 2024 ; 35( 6): 456-458.[citado 2024 ago. 16 ] Available from: https://doi.org/10.1016/j.tem.2024.04.001
Artigo de periodico
Platelet-activating factor receptor (PAFR) regulates neuronal maturation and synaptic transmission during postnatal retinal development (2024)
Dalmaso, Barbara ; Liber, André Maurício Passos ; Ventura, Dora Selma Fix ; Jancar, Sonia ; Del Debbio, Carolina Beltrame
Fonte: Frontiers in Cellular Neuroscience. Unidades: IP, ICB
Assuntos: BIOLOGIA CELULAR, IMUNOLOGIA, CAMUNDONGOS, RETINA, PLAQUETAS SANGUÍNEAS, NEUROBLASTOMA, PROLIFERAÇÃO CELULAR, RETINOBLASTOMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DALMASO, Barbara et al. Platelet-activating factor receptor (PAFR) regulates neuronal maturation and synaptic transmission during postnatal retinal development. Frontiers in Cellular Neuroscience, v. 18, p. 17 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fncel.2024.1343745. Acesso em: 16 ago. 2024.
APA
Dalmaso, B., Liber, A. M. P., Ventura, D. S. F., Jancar, S., & Del Debbio, C. B. (2024). Platelet-activating factor receptor (PAFR) regulates neuronal maturation and synaptic transmission during postnatal retinal development. Frontiers in Cellular Neuroscience, 18, 17 . doi:10.3389/fncel.2024.1343745
NLM
Dalmaso B, Liber AMP, Ventura DSF, Jancar S, Del Debbio CB. Platelet-activating factor receptor (PAFR) regulates neuronal maturation and synaptic transmission during postnatal retinal development [Internet]. Frontiers in Cellular Neuroscience. 2024 ; 18 17 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fncel.2024.1343745
Vancouver
Dalmaso B, Liber AMP, Ventura DSF, Jancar S, Del Debbio CB. Platelet-activating factor receptor (PAFR) regulates neuronal maturation and synaptic transmission during postnatal retinal development [Internet]. Frontiers in Cellular Neuroscience. 2024 ; 18 17 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.3389/fncel.2024.1343745
Artigo de periodico
Specialized Tfh cell subsets driving type-1 and type-2 humoral responses in lymphoid tissue (2024)
Kumar, Saumya ; Basto, Afonso P. ; Ribeiro, Filipa ; Almeida, Silvia C. P. ; Campos, Patricia; Peres, Carina ; Pulvirenti, Nadia ; Al-Khalidi, Sarwah ; Kilbey, Anna; Tosello, Jimena ; Piaggio, Eliane ; Gama-Carvalho, Margarida ; Coffelt, Seth B. ; Roberts, Edward W. ; Geginat, Jens ; Florindo, Helena F. ; Graca, Luis ; Russo, Momtchilo
Fonte: Cell Discovery. Unidade: ICB
Assuntos: IMUNOLOGIA, CAMUNDONGOS, TECIDO LINFOIDE, IMUNOLOGIA CELULAR, IMUNOGENÉTICA, FENÓTIPOS, HELMINTHES, HELMINTÍASE ANIMAL, ANTICORPOS, LINFONODOS DE ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KUMAR, Saumya et al. Specialized Tfh cell subsets driving type-1 and type-2 humoral responses in lymphoid tissue. Cell Discovery, v. 10, n. 1, p. 21 , 2024Tradução . . Disponível em: https://doi.org/10.1038/s41421-024-00681-0. Acesso em: 16 ago. 2024.
APA
Kumar, S., Basto, A. P., Ribeiro, F., Almeida, S. C. P., Campos, P., Peres, C., et al. (2024). Specialized Tfh cell subsets driving type-1 and type-2 humoral responses in lymphoid tissue. Cell Discovery, 10( 1), 21 . doi:10.1038/s41421-024-00681-0
NLM
Kumar S, Basto AP, Ribeiro F, Almeida SCP, Campos P, Peres C, Pulvirenti N, Al-Khalidi S, Kilbey A, Tosello J, Piaggio E, Gama-Carvalho M, Coffelt SB, Roberts EW, Geginat J, Florindo HF, Graca L, Russo M. Specialized Tfh cell subsets driving type-1 and type-2 humoral responses in lymphoid tissue [Internet]. Cell Discovery. 2024 ; 10( 1): 21 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1038/s41421-024-00681-0
Vancouver
Kumar S, Basto AP, Ribeiro F, Almeida SCP, Campos P, Peres C, Pulvirenti N, Al-Khalidi S, Kilbey A, Tosello J, Piaggio E, Gama-Carvalho M, Coffelt SB, Roberts EW, Geginat J, Florindo HF, Graca L, Russo M. Specialized Tfh cell subsets driving type-1 and type-2 humoral responses in lymphoid tissue [Internet]. Cell Discovery. 2024 ; 10( 1): 21 .[citado 2024 ago. 16 ] Available from: https://doi.org/10.1038/s41421-024-00681-0
Artigo de periodico
Environmental enrichment enhances ethanol preference over social reward in male swiss mice: involvement of oxytocin-dopamine interactions (2024)
Rae, Mariana Beu ; Gomes, Ivone ; Spelta, Lídia Emmanuela Wiazowski ; Bailey, Alexis ; Marcourakis, Tania ; Devi, Lakshmi ; Camarini, Rosana
Fonte: Neuropharmacology. Unidades: FCF, ICB, FM
Assuntos: ETANOL, FOSFOLIPASES, HIPOTÁLAMO, RECEPTORES DE DOPAMINA, OCITOCINA, CAMUNDONGOS, SISTEMA NERVOSO CENTRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RAE, Mariana Beu et al. Environmental enrichment enhances ethanol preference over social reward in male swiss mice: involvement of oxytocin-dopamine interactions. Neuropharmacology, v. 253, p. 1-12 art. 109971, 2024Tradução . . Disponível em: https://dx.doi.org/10.1016/j.neuropharm.2024.109971. Acesso em: 16 ago. 2024.
APA
Rae, M. B., Gomes, I., Spelta, L. E. W., Bailey, A., Marcourakis, T., Devi, L., & Camarini, R. (2024). Environmental enrichment enhances ethanol preference over social reward in male swiss mice: involvement of oxytocin-dopamine interactions. Neuropharmacology, 253, 1-12 art. 109971. doi:10.1016/j.neuropharm.2024.109971
NLM
Rae MB, Gomes I, Spelta LEW, Bailey A, Marcourakis T, Devi L, Camarini R. Environmental enrichment enhances ethanol preference over social reward in male swiss mice: involvement of oxytocin-dopamine interactions [Internet]. Neuropharmacology. 2024 ; 253 1-12 art. 109971.[citado 2024 ago. 16 ] Available from: https://dx.doi.org/10.1016/j.neuropharm.2024.109971
Vancouver
Rae MB, Gomes I, Spelta LEW, Bailey A, Marcourakis T, Devi L, Camarini R. Environmental enrichment enhances ethanol preference over social reward in male swiss mice: involvement of oxytocin-dopamine interactions [Internet]. Neuropharmacology. 2024 ; 253 1-12 art. 109971.[citado 2024 ago. 16 ] Available from: https://dx.doi.org/10.1016/j.neuropharm.2024.109971

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