Filtros : "ICB" "NUNES, MARIA TEREZA" Removidos: "Universidade de Santo Amaro (UNISA)" "ROSSONI, LUCIANA VENTURINI" "Usp" Limpar

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  • Source: Life Sciences. Unidade: ICB

    Subjects: DEGENERAÇÃO NEURAL, INSULINA, TRIIODOTIRONINA, DIABETES MELLITUS, HIPOCAMPO, HORMÔNIOS TIREOIDIANOS

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      ALMEIDA, Fernanda Prieto de et al. Thyroid hormone improves insulin signaling and reduces the activation of neurodegenerative pathway in the hippocampus of diabetic adult male rats. Life Sciences, v. 192, p. 253-258, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2017.11.013. Acesso em: 03 jun. 2024.
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      Almeida, F. P. de, Costa, A. C. P., Crunfli, F., Teixeira, S. da S., Nunes, M. T., & Torrão, A. da S. (2018). Thyroid hormone improves insulin signaling and reduces the activation of neurodegenerative pathway in the hippocampus of diabetic adult male rats. Life Sciences, 192, 253-258. doi:10.1016/j.lfs.2017.11.013
    • NLM

      Almeida FP de, Costa ACP, Crunfli F, Teixeira S da S, Nunes MT, Torrão A da S. Thyroid hormone improves insulin signaling and reduces the activation of neurodegenerative pathway in the hippocampus of diabetic adult male rats [Internet]. Life Sciences. 2018 ; 192 253-258.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.lfs.2017.11.013
    • Vancouver

      Almeida FP de, Costa ACP, Crunfli F, Teixeira S da S, Nunes MT, Torrão A da S. Thyroid hormone improves insulin signaling and reduces the activation of neurodegenerative pathway in the hippocampus of diabetic adult male rats [Internet]. Life Sciences. 2018 ; 192 253-258.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.lfs.2017.11.013
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: TRIIODOTIRONINA, HORMÔNIOS TIREOIDIANOS, EXPRESSÃO GÊNICA, LINFÓCITOS T, RNA MENSAGEIRO, RIBOSSOMOS, PROTEÍNAS DA MEMBRANA

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      SOUZA, Paula Bargi de e SILVA, Francemilson Goulart da e NUNES, Maria Tereza. Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback. Molecular and Cellular Endocrinology, v. 478, p. 45-52, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2018.07.006. Acesso em: 03 jun. 2024.
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      Souza, P. B. de, Silva, F. G. da, & Nunes, M. T. (2018). Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback. Molecular and Cellular Endocrinology, 478, 45-52. doi:10.1016/j.mce.2018.07.006
    • NLM

      Souza PB de, Silva FG da, Nunes MT. Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback [Internet]. Molecular and Cellular Endocrinology. 2018 ; 478 45-52.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mce.2018.07.006
    • Vancouver

      Souza PB de, Silva FG da, Nunes MT. Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback [Internet]. Molecular and Cellular Endocrinology. 2018 ; 478 45-52.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mce.2018.07.006
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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      SALGUEIRO, Rafael Barrera et al. Exercise training reverses the negative effects of chronic L-arginine supplementation on insulin sensitivity. Life Sciences, v. 191, p. 17-23, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2017.10.001. Acesso em: 03 jun. 2024.
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      Salgueiro, R. B., Gerlinger-Romero, F., Guimarães-Ferreira, L., Barbosa, T. de C., & Nunes, M. T. (2017). Exercise training reverses the negative effects of chronic L-arginine supplementation on insulin sensitivity. Life Sciences, 191, 17-23. doi:10.1016/j.lfs.2017.10.001
    • NLM

      Salgueiro RB, Gerlinger-Romero F, Guimarães-Ferreira L, Barbosa T de C, Nunes MT. Exercise training reverses the negative effects of chronic L-arginine supplementation on insulin sensitivity [Internet]. Life Sciences. 2017 ; 191 17-23.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.lfs.2017.10.001
    • Vancouver

      Salgueiro RB, Gerlinger-Romero F, Guimarães-Ferreira L, Barbosa T de C, Nunes MT. Exercise training reverses the negative effects of chronic L-arginine supplementation on insulin sensitivity [Internet]. Life Sciences. 2017 ; 191 17-23.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.lfs.2017.10.001
  • Source: Scientific Reports. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline et al. Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring. Scientific Reports, v. 7, n. 1, p. 1-12, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-15529-9. Acesso em: 03 jun. 2024.
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      Serrano-Nascimento, C., Salgueiro, R. B., Pantaleão, T., Costa, V. M. C. da, & Nunes, M. T. (2017). Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring. Scientific Reports, 7( 1), 1-12. doi:10.1038/s41598-017-15529-9
    • NLM

      Serrano-Nascimento C, Salgueiro RB, Pantaleão T, Costa VMC da, Nunes MT. Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring [Internet]. Scientific Reports. 2017 ; 7( 1): 1-12.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1038/s41598-017-15529-9
    • Vancouver

      Serrano-Nascimento C, Salgueiro RB, Pantaleão T, Costa VMC da, Nunes MT. Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring [Internet]. Scientific Reports. 2017 ; 7( 1): 1-12.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1038/s41598-017-15529-9
  • Source: Journal of Molecular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      BARGI-SOUZA, P. e SILVA, Francemilson Goulart da e NUNES, Maria Tereza. Novel aspects of T3 actions on GH and TSH synthesis and secretion: physiological implications. Journal of Molecular Endocrinology, v. 59, n. 4, p. R167-R178, 2017Tradução . . Disponível em: https://doi.org/10.1530/JME-17-0068. Acesso em: 03 jun. 2024.
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      Bargi-Souza, P., Silva, F. G. da, & Nunes, M. T. (2017). Novel aspects of T3 actions on GH and TSH synthesis and secretion: physiological implications. Journal of Molecular Endocrinology, 59( 4), R167-R178. doi:10.1530/JME-17-0068
    • NLM

      Bargi-Souza P, Silva FG da, Nunes MT. Novel aspects of T3 actions on GH and TSH synthesis and secretion: physiological implications [Internet]. Journal of Molecular Endocrinology. 2017 ; 59( 4): R167-R178.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1530/JME-17-0068
    • Vancouver

      Bargi-Souza P, Silva FG da, Nunes MT. Novel aspects of T3 actions on GH and TSH synthesis and secretion: physiological implications [Internet]. Journal of Molecular Endocrinology. 2017 ; 59( 4): R167-R178.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1530/JME-17-0068
  • Source: Acta Physiologica. Unidade: ICB

    Assunto: FISIOLOGIA

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      TEIXEIRA, S. da Silva et al. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice. Acta Physiologica, v. 220, n. 2, p. 238-250, 2017Tradução . . Disponível em: https://doi.org/10.1111/apha.12821. Acesso em: 03 jun. 2024.
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      Teixeira, S. da S., Filgueira, C., Sieglaff, D. H., Benod , C., Villagomez, R., Minze, L. J., et al. (2017). 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice. Acta Physiologica, 220( 2), 238-250. doi:10.1111/apha.12821
    • NLM

      Teixeira S da S, Filgueira C, Sieglaff DH, Benod C, Villagomez R, Minze LJ, Zhang A, Webb P, Nunes MT. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice [Internet]. Acta Physiologica. 2017 ; 220( 2): 238-250.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1111/apha.12821
    • Vancouver

      Teixeira S da S, Filgueira C, Sieglaff DH, Benod C, Villagomez R, Minze LJ, Zhang A, Webb P, Nunes MT. 3,5-diiodothyronine (3,5-T2) reduces blood glucose independently of insulin sensitization in obese mice [Internet]. Acta Physiologica. 2017 ; 220( 2): 238-250.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1111/apha.12821
  • Source: Endocrine Connections. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline et al. Iodine excess exposure during pregnancy and lactation impairs maternal thyroid function in rats. Endocrine Connections, v. 6, n. 7, p. 510-521, 2017Tradução . . Disponível em: https://doi.org/10.1530/EC-17-0106. Acesso em: 03 jun. 2024.
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      Serrano-Nascimento, C., Salgueiro, R. B., Vitzel, K. F., Pantaleão, T., Costa, V. M. C. da, & Nunes, M. T. (2017). Iodine excess exposure during pregnancy and lactation impairs maternal thyroid function in rats. Endocrine Connections, 6( 7), 510-521. doi:10.1530/EC-17-0106
    • NLM

      Serrano-Nascimento C, Salgueiro RB, Vitzel KF, Pantaleão T, Costa VMC da, Nunes MT. Iodine excess exposure during pregnancy and lactation impairs maternal thyroid function in rats [Internet]. Endocrine Connections. 2017 ; 6( 7): 510-521.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1530/EC-17-0106
    • Vancouver

      Serrano-Nascimento C, Salgueiro RB, Vitzel KF, Pantaleão T, Costa VMC da, Nunes MT. Iodine excess exposure during pregnancy and lactation impairs maternal thyroid function in rats [Internet]. Endocrine Connections. 2017 ; 6( 7): 510-521.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1530/EC-17-0106
  • Source: American Journal of Physiology. Cell Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      CALIL-SILVEIRA, Jamile et al. Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, v. 310, n. 7, p. C576-C582, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00210.2015. Acesso em: 03 jun. 2024.
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      Calil-Silveira, J., Serrano-Nascimento, C., Kopp, P. A., & Nunes, M. T. (2016). Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, 310( 7), C576-C582. doi:10.1152/ajpcell.00210.2015
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1152/ajpcell.00210.2015
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1152/ajpcell.00210.2015
  • Source: Anais da Academia Brasileira de Ciências. Unidade: ICB

    Subjects: FERRO, CORAÇÃO, MÚSCULO ESQUELÉTICO

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      SOUZA, Janaina Sena de e RACT, Erika Lia Brunetto e NUNES, Maria Tereza. Iron restriction increases myoglobin gene and protein expression in Soleus muscle of rats. Anais da Academia Brasileira de Ciências, v. 88, n. 4, p. 2277-2290, 2016Tradução . . Disponível em: https://doi.org/10.1590/0001-3765201620160173. Acesso em: 03 jun. 2024.
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      Souza, J. S. de, Ract, E. L. B., & Nunes, M. T. (2016). Iron restriction increases myoglobin gene and protein expression in Soleus muscle of rats. Anais da Academia Brasileira de Ciências, 88( 4), 2277-2290. doi:10.1590/0001-3765201620160173
    • NLM

      Souza JS de, Ract ELB, Nunes MT. Iron restriction increases myoglobin gene and protein expression in Soleus muscle of rats [Internet]. Anais da Academia Brasileira de Ciências. 2016 ;88( 4): 2277-2290.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1590/0001-3765201620160173
    • Vancouver

      Souza JS de, Ract ELB, Nunes MT. Iron restriction increases myoglobin gene and protein expression in Soleus muscle of rats [Internet]. Anais da Academia Brasileira de Ciências. 2016 ;88( 4): 2277-2290.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1590/0001-3765201620160173
  • Source: Physiological Reports. Unidade: ICB

    Assunto: FISIOLOGIA

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      TEIXEIRA, Silvania da Silva et al. Thyroid hormone treatment decreases hepatic glucose production and renal reabsorption of glucose in alloxan-induced diabetic Wistar rats. Physiological Reports, v. 4, n. 18, p. 1-9, 2016Tradução . . Disponível em: https://doi.org/10.14814/phy2.12961. Acesso em: 03 jun. 2024.
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      Teixeira, S. da S., Panveloski-Costa, A. C., Carvalho, A., Schiavon, F. P. M., Marque, A. de C. R., Campello, R. S., et al. (2016). Thyroid hormone treatment decreases hepatic glucose production and renal reabsorption of glucose in alloxan-induced diabetic Wistar rats. Physiological Reports, 4( 18), 1-9. doi:10.14814/phy2.12961
    • NLM

      Teixeira S da S, Panveloski-Costa AC, Carvalho A, Schiavon FPM, Marque A de CR, Campello RS, Bazotte RB, Nunes MT. Thyroid hormone treatment decreases hepatic glucose production and renal reabsorption of glucose in alloxan-induced diabetic Wistar rats [Internet]. Physiological Reports. 2016 ; 4( 18): 1-9.[citado 2024 jun. 03 ] Available from: https://doi.org/10.14814/phy2.12961
    • Vancouver

      Teixeira S da S, Panveloski-Costa AC, Carvalho A, Schiavon FPM, Marque A de CR, Campello RS, Bazotte RB, Nunes MT. Thyroid hormone treatment decreases hepatic glucose production and renal reabsorption of glucose in alloxan-induced diabetic Wistar rats [Internet]. Physiological Reports. 2016 ; 4( 18): 1-9.[citado 2024 jun. 03 ] Available from: https://doi.org/10.14814/phy2.12961
  • Source: Advances in Physiology Education. Unidade: ICB

    Assunto: FISIOLOGIA

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      CAFÉ-MENDES, Cecília C. et al. Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil. Advances in Physiology Education, v. 40, n. 4, p. 491-498, 2016Tradução . . Disponível em: https://doi.org/10.1152/advan.00031.2016. Acesso em: 03 jun. 2024.
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      Café-Mendes, C. C., Righi, L. L., Calil-Silveira, J., Nunes, M. T., & Abdulkader, F. R. de M. (2016). Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil. Advances in Physiology Education, 40( 4), 491-498. doi:10.1152/advan.00031.2016
    • NLM

      Café-Mendes CC, Righi LL, Calil-Silveira J, Nunes MT, Abdulkader FR de M. Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil [Internet]. Advances in Physiology Education. 2016 ; 40( 4): 491-498.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1152/advan.00031.2016
    • Vancouver

      Café-Mendes CC, Righi LL, Calil-Silveira J, Nunes MT, Abdulkader FR de M. Winter course in physiology: a successful example of continuing education for secondary school teachers in Brazil [Internet]. Advances in Physiology Education. 2016 ; 40( 4): 491-498.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1152/advan.00031.2016
  • Source: Thyroid. Unidade: ICB

    Assunto: FISIOLOGIA

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      CALIL-SILVEIRA, Jamile et al. Underlying mechanisms of pituitary-thyroid axis function disruption by chronic iodine excess in rats. Thyroid, v. 26, n. 10, p. 1488-1498, 2016Tradução . . Disponível em: https://doi.org/10.1089/thy.20150338. Acesso em: 03 jun. 2024.
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      Calil-Silveira, J., Serrano-Nascimento, C., Laconca, R. C., Schmiedecke, L., Salgueiro, R. B., Kondo, A. K., & Nunes, M. T. (2016). Underlying mechanisms of pituitary-thyroid axis function disruption by chronic iodine excess in rats. Thyroid, 26( 10), 1488-1498. doi:10.1089/thy.20150338
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Laconca RC, Schmiedecke L, Salgueiro RB, Kondo AK, Nunes MT. Underlying mechanisms of pituitary-thyroid axis function disruption by chronic iodine excess in rats [Internet]. Thyroid. 2016 ; 26( 10): 1488-1498.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1089/thy.20150338
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Laconca RC, Schmiedecke L, Salgueiro RB, Kondo AK, Nunes MT. Underlying mechanisms of pituitary-thyroid axis function disruption by chronic iodine excess in rats [Internet]. Thyroid. 2016 ; 26( 10): 1488-1498.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1089/thy.20150338
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline et al. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, v. 426, p. 73-90, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.02.006. Acesso em: 03 jun. 2024.
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      Serrano-Nascimento, C., Nicola, J. P., Teixeira, S. da S., Poyares, L. L., Lellis-Santos, C., Bordin, S., et al. (2016). Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, 426, 73-90. doi:10.1016/j.mce.2016.02.006
    • NLM

      Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
    • Vancouver

      Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
  • Source: Acta Physiologica. Unidade: ICB

    Subjects: CITOCINAS, DIABETES MELLITUS, TRIIODOTIRONINA

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      COSTA, Ana Carolina Panveloski et al. Thyroid hormone reduces inflammatory cytokinesimproving glycaemia control in alloxan-induced diabeticwistar rats. Acta Physiologica, v. 217, n. 2, p. 130-140, 2016Tradução . . Disponível em: https://doi.org/10.1111/apha.12647. Acesso em: 03 jun. 2024.
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      Costa, A. C. P., Teixeira, S. da S., Ribeiro, I. M. R., Nascimento, C. S. do, Neves, R. X. das, Ribeiro, R. R., et al. (2016). Thyroid hormone reduces inflammatory cytokinesimproving glycaemia control in alloxan-induced diabeticwistar rats. Acta Physiologica, 217( 2), 130-140. doi:10.1111/apha.12647
    • NLM

      Costa ACP, Teixeira S da S, Ribeiro IMR, Nascimento CS do, Neves RX das, Ribeiro RR, Seelaender MCL, Antunes VR, Nunes MT. Thyroid hormone reduces inflammatory cytokinesimproving glycaemia control in alloxan-induced diabeticwistar rats [Internet]. Acta Physiologica. 2016 ; 217( 2): 130-140.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1111/apha.12647
    • Vancouver

      Costa ACP, Teixeira S da S, Ribeiro IMR, Nascimento CS do, Neves RX das, Ribeiro RR, Seelaender MCL, Antunes VR, Nunes MT. Thyroid hormone reduces inflammatory cytokinesimproving glycaemia control in alloxan-induced diabeticwistar rats [Internet]. Acta Physiologica. 2016 ; 217( 2): 130-140.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1111/apha.12647
  • Source: Chronobiology International. Unidade: ICB

    Assunto: FISIOLOGIA

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      GARCIA, Rodrigo Antonio Peliciari et al. Interrelationship between 3,5,3´-triiodothyronine and the circadian clock in the rodent heart. Chronobiology International, v. 33, n. 10, p. 1444-1454, 2016Tradução . . Disponível em: https://doi.org/10.1080/07420528.2016.1229673. Acesso em: 03 jun. 2024.
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      Garcia, R. A. P., Prévide, R. M., Nunes, M. T., & Young, M. E. (2016). Interrelationship between 3,5,3´-triiodothyronine and the circadian clock in the rodent heart. Chronobiology International, 33( 10), 1444-1454. doi:10.1080/07420528.2016.1229673
    • NLM

      Garcia RAP, Prévide RM, Nunes MT, Young ME. Interrelationship between 3,5,3´-triiodothyronine and the circadian clock in the rodent heart [Internet]. Chronobiology International. 2016 ; 33( 10): 1444-1454.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1080/07420528.2016.1229673
    • Vancouver

      Garcia RAP, Prévide RM, Nunes MT, Young ME. Interrelationship between 3,5,3´-triiodothyronine and the circadian clock in the rodent heart [Internet]. Chronobiology International. 2016 ; 33( 10): 1444-1454.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1080/07420528.2016.1229673
  • Source: Cell Calcium. Unidade: ICB

    Subjects: FISIOLOGIA, HORMÔNIO TIREOTROFICO, CAMUNDONGOS, CALCIO

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      TOMIC, Melanija et al. Calcium signaling properties of a thyrotroph cell line, mouse TαT1 cells. Cell Calcium, v. 58, p. 598-605, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ceca.2015.09.003. Acesso em: 03 jun. 2024.
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      Tomic, M., Bargi-Souza, P., Leiva-Salcedo, E., Nunes, M. T., & Stojilkovic, S. S. (2015). Calcium signaling properties of a thyrotroph cell line, mouse TαT1 cells. Cell Calcium, 58, 598-605. doi:10.1016/j.ceca.2015.09.003
    • NLM

      Tomic M, Bargi-Souza P, Leiva-Salcedo E, Nunes MT, Stojilkovic SS. Calcium signaling properties of a thyrotroph cell line, mouse TαT1 cells [Internet]. Cell Calcium. 2015 ; 58 598-605.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.ceca.2015.09.003
    • Vancouver

      Tomic M, Bargi-Souza P, Leiva-Salcedo E, Nunes MT, Stojilkovic SS. Calcium signaling properties of a thyrotroph cell line, mouse TαT1 cells [Internet]. Cell Calcium. 2015 ; 58 598-605.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.ceca.2015.09.003
  • Source: Growth Hormone & IGF Research. Unidade: ICB

    Subjects: FISIOLOGIA, RATOS, LACTATOS

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      SALGUEIRO, Rafael Barrera et al. Lactate activates the somatotropic axis in rats. Growth Hormone & IGF Research, v. 6, p. 268-270, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ghir.2014.09.003. Acesso em: 03 jun. 2024.
    • APA

      Salgueiro, R. B., Garcia, R. A. P., Buonfiglio, D. do C., Peroni, C. N., & Nunes, M. T. (2015). Lactate activates the somatotropic axis in rats. Growth Hormone & IGF Research, 6, 268-270. doi:10.1016/j.ghir.2014.09.003
    • NLM

      Salgueiro RB, Garcia RAP, Buonfiglio D do C, Peroni CN, Nunes MT. Lactate activates the somatotropic axis in rats [Internet]. Growth Hormone & IGF Research. 2015 ; 6 268-270.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.ghir.2014.09.003
    • Vancouver

      Salgueiro RB, Garcia RAP, Buonfiglio D do C, Peroni CN, Nunes MT. Lactate activates the somatotropic axis in rats [Internet]. Growth Hormone & IGF Research. 2015 ; 6 268-270.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.ghir.2014.09.003
  • Source: Endocrinology. Unidade: ICB

    Subjects: FISIOLOGIA, GLÂNDULA PITUITÁRIA, EXPRESSÃO GÊNICA

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    • ABNT

      BARGI-SOUZA, Paula et al. Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells. Endocrinology, v. 156, n. 1, p. 242-254, 2015Tradução . . Disponível em: https://doi.org/10.1210/en.2014-1281. Acesso em: 03 jun. 2024.
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      Bargi-Souza, P., Kucka, M., Bjelobaba, I., Tomic, M., Janjic, M. M., Nunes, M. T., & Stojikovic, S. S. (2015). Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells. Endocrinology, 156( 1), 242-254. doi:10.1210/en.2014-1281
    • NLM

      Bargi-Souza P, Kucka M, Bjelobaba I, Tomic M, Janjic MM, Nunes MT, Stojikovic SS. Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells [Internet]. Endocrinology. 2015 ; 156( 1): 242-254.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1210/en.2014-1281
    • Vancouver

      Bargi-Souza P, Kucka M, Bjelobaba I, Tomic M, Janjic MM, Nunes MT, Stojikovic SS. Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells [Internet]. Endocrinology. 2015 ; 156( 1): 242-254.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1210/en.2014-1281
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: FISIOLOGIA, GLICOPROTEINAS, GLÂNDULA PITUITÁRIA, RATOS, SECREÇÃO ANIMAL

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    • ABNT

      SOUZA, Paula Bargi de et al. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion. Molecular and Cellular Endocrinology, v. 409, p. 73-81, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.04.002. Acesso em: 03 jun. 2024.
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      Souza, P. B. de, Romano, R. M., Silva, F. G. da, Brunetto, E. L., & Nunes, M. T. (2015). T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion. Molecular and Cellular Endocrinology, 409, 73-81. doi:10.1016/j.mce.2015.04.002
    • NLM

      Souza PB de, Romano RM, Silva FG da, Brunetto EL, Nunes MT. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion [Internet]. Molecular and Cellular Endocrinology. 2015 ; 409 73-81.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mce.2015.04.002
    • Vancouver

      Souza PB de, Romano RM, Silva FG da, Brunetto EL, Nunes MT. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion [Internet]. Molecular and Cellular Endocrinology. 2015 ; 409 73-81.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1016/j.mce.2015.04.002
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Subjects: FISIOLOGIA, HIPERTIREOIDISMO, EXPRESSÃO GÊNICA

    Acesso à fonteDOIHow to cite
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    • ABNT

      SOUZA, K. de Picoli e NUNES, Maria Tereza. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood. Brazilian Journal of Medical and Biological Research, v. 47, n. 8, p. 670-678, 2014Tradução . . Disponível em: https://doi.org/10.1590/1414-431X20142875. Acesso em: 03 jun. 2024.
    • APA

      Souza, K. de P., & Nunes, M. T. (2014). Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood. Brazilian Journal of Medical and Biological Research, 47( 8), 670-678. doi:10.1590/1414-431X20142875
    • NLM

      Souza K de P, Nunes MT. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood [Internet]. Brazilian Journal of Medical and Biological Research. 2014 ; 47( 8): 670-678.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1590/1414-431X20142875
    • Vancouver

      Souza K de P, Nunes MT. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood [Internet]. Brazilian Journal of Medical and Biological Research. 2014 ; 47( 8): 670-678.[citado 2024 jun. 03 ] Available from: https://doi.org/10.1590/1414-431X20142875

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